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Reductive Stress in the Endoplasmic Reticulum Caused by Ero1α S-Nitrosation Accelerates Senescence

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inferredcites →https://doi.org/10.1016/j.freeradbiomed.2013.05.003openalex-extracted
Reductive stress in young healthy individuals at risk of Alzheimer disease
inferredcites →https://doi.org/10.1038/emboj.2008.202openalex-extracted
A novel disulphide switch mechanism in Ero1α balances ER oxidation in human cells
inferredcites →https://doi.org/10.1038/emboj.2008.230openalex-extracted
Low reduction potential of Ero1α regulatory disulphides ensures tight control of substrate oxidation
inferredcites →https://doi.org/10.1038/s41586-019-1814-yopenalex-extracted
Developmental ROS individualizes organismal stress resistance and lifespan
inferredcites →https://doi.org/10.1093/jnci/djk211openalex-extracted
Surviving Antioxidant Supplements
inferredcites →https://doi.org/10.1101/756742openalex-extracted
Developmental Reactive Oxygen Species Target Histone Methylation to Individualize Stress resistance and Lifespan
inferredcites →https://doi.org/10.1242/jcs.117218openalex-extracted
Endoplasmic reticulum: Reduced and oxidized glutathione revisited
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