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Tim-3 Induces Il-17 Production Via Transcription Factor Pu.1 on Cd4+T Cells in Primary Ebv Infection

https://doi.org/10.2139/ssrn.4645308
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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/s00109-016-1402-7openalex-extracted
SIRT1 inhibits differentiation of monocytes to macrophages: amelioration of synovial inflammation in rheumatoid arthritis
inferredcites →https://doi.org/10.1016/b978-0-323-39255-6.50066-1openalex-extracted
Infectious Mononucleosis
inferredcites →https://doi.org/10.1016/j.autrev.2019.102429openalex-extracted
Critical role of interleukin (IL)-17 in inflammatory and immune disorders: An updated review of the evidence focusing in controversies
inferredcites →https://doi.org/10.1016/j.bbadis.2019.165626openalex-extracted
Minor C allele of the SNP rs7873784 associated with rheumatoid arthritis and type-2 diabetes mellitus binds PU.1 and enhances TLR4 expression.
inferredcites →https://doi.org/10.1016/j.ccell.2018.05.004openalex-extracted
Th9 Cells Represent a Unique Subset of CD4+ T Cells Endowed with the Ability to Eradicate Advanced Tumors
inferredcites →https://doi.org/10.1016/j.clim.2008.01.006openalex-extracted
Galectin-9 suppresses the generation of Th17, promotes the induction of regulatory T cells, and regulates experimental autoimmune arthritis
inferredcites →https://doi.org/10.1016/j.ymthe.2021.12.013openalex-extracted
Tim-3 regulates sepsis-induced immunosuppression by inhibiting the NF-κB signaling pathway in CD4 T cells
inferredcites →https://doi.org/10.1038/nature11868openalex-extracted
Sodium chloride drives autoimmune disease by the induction of pathogenic TH17 cells
inferredcites →https://doi.org/10.1038/ni.1798openalex-extracted
MicroRNA miR-326 regulates TH-17 differentiation and is associated with the pathogenesis of multiple sclerosis
inferredcites →https://doi.org/10.1038/ni987openalex-extracted
Tim-3 inhibits T helper type 1–mediated auto- and alloimmune responses and promotes immunological tolerance
inferredcites →https://doi.org/10.1038/onc.2017.79openalex-extracted
PU.1 acts as tumor suppressor for myeloma cells through direct transcriptional repression of IRF4
inferredcites →https://doi.org/10.1038/s41467-022-28172-4openalex-extracted
Neutrophil extracellular traps and their histones promote Th17 cell differentiation directly via TLR2
inferredcites →https://doi.org/10.1084/jem.20100703openalex-extracted
Modulation of experimental autoimmune encephalomyelitis through TRAF3-mediated suppression of interleukin 17 receptor signaling
inferredcites →https://doi.org/10.1089/vim.2014.0129openalex-extracted
Epstein-Barr Virus and Human herpes virus 6 Type A DNA Enhance IL-17 Production in Mice
inferredcites →https://doi.org/10.1136/jitc-2020-001895openalex-extracted
Blocking IL-17A enhances tumor response to anti-PD-1 immunotherapy in microsatellite stable colorectal cancer
inferredcites →https://doi.org/10.1182/blood-2004-03-0842openalex-extracted
Interleukin-15 induces IL-12 receptor β1 gene expression through PU.1 and IRF 3 by targeting chromatin remodeling
inferredcites →https://doi.org/10.1182/bloodadvances.2017007260openalex-extracted
Th17 immune microenvironment in Epstein-Barr virus–negative Hodgkin lymphoma: implications for immunotherapy
inferredcites →https://doi.org/10.1371/journal.pone.0200546openalex-extracted
Epstein-Barr virus DNA modulates regulatory T-cell programming in addition to enhancing interleukin-17A production via Toll-like receptor 9
inferredcites →https://doi.org/10.1371/journal.ppat.1007748openalex-extracted
CD8+ T cells retain protective functions despite sustained inhibitory receptor expression during Epstein-Barr virus infection in vivo
inferredcites →https://doi.org/10.15252/embj.2021109803openalex-extracted
Batf stabilizes Th17 cell development via impaired Stat5 recruitment of Ets1‐Runx1 complexes
inferredcites →https://doi.org/10.3389/fimmu.2021.668095openalex-extracted
Induction of IL-9 in Peripheral Lymphocytes of Rheumatoid Arthritis Patients and Healthy Donors by Th17-Inducing Cytokine Conditions
inferredcites →https://doi.org/10.3389/fimmu.2022.1001201openalex-extracted
The role of a key transcription factor PU.1 in autoimmune diseases
inferredcites →https://doi.org/10.4049/jimmunol.1502383openalex-extracted
The ETS Family Transcription Factors Etv5 and PU.1 Function in Parallel To Promote Th9 Cell Development
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