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Camk2g1 Regulates Notch Signaling and Histone Methylation to Specify Hematopoietic Stem Cells

https://doi.org/10.2139/ssrn.6238530
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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/dvdy.21005openalex-snapshot
Differential expression of CaMK‐II genes during early zebrafish embryogenesis
inferredcites →https://doi.org/10.1002/dvdy.24632openalex-snapshot
Calcium signals act through histone deacetylase to mediate pronephric kidney morphogenesis
inferredcites →https://doi.org/10.1002/sctm.20-0247openalex-snapshot
Hematopoietic stem cells from pluripotent stem cells: Clinical potential, challenges, and future perspectives
inferredcites →https://doi.org/10.1006/dbio.2002.0711openalex-snapshot
In Vivo Imaging of Embryonic Vascular Development Using Transgenic Zebrafish
inferredcites →https://doi.org/10.1007/978-3-030-12457-1_21openalex-snapshot
Widespread Roles of CaMK-II in Developmental Pathways
inferredcites →https://doi.org/10.1016/j.ccell.2017.06.001openalex-snapshot
Stabilization of the c-Myc Protein by CAMKIIγ Promotes T Cell Lymphoma
inferredcites →https://doi.org/10.1016/j.cell.2008.02.008openalex-snapshot
Differentiation of Embryonic Stem Cells to Clinically Relevant Populations: Lessons from Embryonic Development
inferredcites →https://doi.org/10.1016/j.cellsig.2014.11.007openalex-snapshot
A novel crosstalk between calcium/calmodulin kinases II and IV regulates cell proliferation in myeloid leukemia cells
inferredcites →https://doi.org/10.1016/j.devcel.2016.06.024openalex-snapshot
Transforming Growth Factor β Drives Hemogenic Endothelium Programming and the Transition to Hematopoietic Stem Cells
inferredcites →https://doi.org/10.1016/j.exphem.2024.104280openalex-snapshot
The molecular and cellular hematopoietic stem cell specification niche
inferredcites →https://doi.org/10.1016/j.gene.2003.08.023openalex-snapshot
Organization and evolution of multifunctional Ca2+/CaM-dependent protein kinase genes
inferredcites →https://doi.org/10.1016/j.gene.2020.144567openalex-snapshot
Genetic compensation of γ CaMKII, an evolutionarily conserved gene
inferredcites →https://doi.org/10.1016/j.mex.2018.10.009openalex-snapshot
An efficient dissociation protocol for generation of single cell suspension from zebrafish embryos and larvae
inferredcites →https://doi.org/10.1016/j.mod.2009.07.002openalex-snapshot
Notch-responsive cells initiate the secondary transition in larval zebrafish pancreas
inferredcites →https://doi.org/10.1016/j.stemcr.2021.05.014openalex-snapshot
Sequential regulation of hemogenic fate and hematopoietic stem and progenitor cell formation from arterial endothelium by Ezh1/2
inferredcites →https://doi.org/10.1016/j.ydbio.2009.03.024openalex-snapshot
Tbx5-mediated expression of Ca2+/calmodulin-dependent protein kinase II is necessary for zebrafish cardiac and pectoral fin morphogenesis
inferredcites →https://doi.org/10.1016/j.ydbio.2013.05.028openalex-snapshot
CaMK-II activation is essential for zebrafish inner ear development and acts through Delta–Notch signaling
inferredcites →https://doi.org/10.1016/j.ydbio.2015.11.008openalex-snapshot
Complex regulation of HSC emergence by the Notch signaling pathway
inferredcites →https://doi.org/10.1038/cr.2015.109openalex-snapshot
G protein-coupled receptor 183 facilitates endothelial-to-hematopoietic transition via Notch1 inhibition
inferredcites →https://doi.org/10.1038/leu.2015.74openalex-snapshot
The Notch ligand DLL4 specifically marks human hematoendothelial progenitors and regulates their hematopoietic fate
inferredcites →https://doi.org/10.1038/nature08738openalex-snapshot
Haematopoietic stem cells derive directly from aortic endothelium during development
inferredcites →https://doi.org/10.1038/nature08761openalex-snapshot
Blood stem cells emerge from aortic endothelium by a novel type of cell transition
inferredcites →https://doi.org/10.1038/nature10107openalex-snapshot
A somitic Wnt16/Notch pathway specifies haematopoietic stem cells
inferredcites →https://doi.org/10.1038/nature13623openalex-snapshot
Jam1a–Jam2a interactions regulate haematopoietic stem cell fate through Notch signalling
inferredcites →https://doi.org/10.1038/nature23883openalex-snapshot
m6A modulates haematopoietic stem and progenitor cell specification
inferredcites →https://doi.org/10.1038/ncb3508openalex-snapshot
Pdgf signalling guides neural crest contribution to the haematopoietic stem cell specification niche
inferredcites →https://doi.org/10.1038/ncomms9510openalex-snapshot
Notch signal strength controls cell fate in the haemogenic endothelium
inferredcites →https://doi.org/10.1038/nri3443openalex-snapshot
Signalling pathways that control vertebrate haematopoietic stem cell specification
inferredcites →https://doi.org/10.1038/s41467-019-11423-2openalex-snapshot
Blood stem cell-forming haemogenic endothelium in zebrafish derives from arterial endothelium
inferredcites →https://doi.org/10.1084/jem.20121484openalex-snapshot
Notch pathway activation targets AML-initiating cell homeostasis and differentiation
inferredcites →https://doi.org/10.1146/annurev.immunol.21.120601.141007openalex-snapshot
Biology of Hematopoietic Stem Cells and Progenitors: Implications for Clinical Application
inferredcites →https://doi.org/10.1158/0008-5472.can-07-2509openalex-snapshot
Activated Ca2+/Calmodulin-Dependent Protein Kinase IIγ Is a Critical Regulator of Myeloid Leukemia Cell Proliferation
inferredcites →https://doi.org/10.1172/jci30779openalex-snapshot
CaMKII regulates retinoic acid receptor transcriptional activity and the differentiation of myeloid leukemia cells
inferredcites →https://doi.org/10.1172/jci92309openalex-snapshot
Endothelial jagged-2 sustains hematopoietic stem and progenitor reconstitution after myelosuppression
inferredcites →https://doi.org/10.1172/jci95946openalex-snapshot
Shifts in podocyte histone H3K27me3 regulate mouse and human glomerular disease
inferredcites →https://doi.org/10.1182/blood-2009-09-244590openalex-snapshot
Notch signaling distinguishes 2 waves of definitive hematopoiesis in the zebrafish embryo
inferredcites →https://doi.org/10.1182/blood-2012-06-434894openalex-snapshot
CaMKII γ, a critical regulator of CML stem/progenitor cells, is a target of the natural product berbamine
inferredcites →https://doi.org/10.1182/blood-2013-11-541391openalex-snapshot
Ncor2 is required for hematopoietic stem cell emergence by inhibiting Fos signaling in zebrafish
inferredcites →https://doi.org/10.1182/blood-2014-04-568170openalex-snapshot
Notch1 acts via Foxc2 to promote definitive hematopoiesis via effects on hemogenic endothelium
inferredcites →https://doi.org/10.1186/1471-2199-7-33openalex-snapshot
Selection of housekeeping genes for gene expression studies in human reticulocytes using real-time PCR
inferredcites →https://doi.org/10.1242/dev.02806openalex-snapshot
Notch signaling controls the differentiation of transporting epithelia and multiciliated cells in the zebrafish pronephros
inferredcites →https://doi.org/10.1242/dev.049627openalex-snapshot
The activation of membrane targeted CaMK-II in the zebrafish Kupffer's vesicle is required for left-right asymmetry
inferredcites →https://doi.org/10.1242/dev.066340openalex-snapshot
CaMK-II is a PKD2 target that promotes pronephric kidney development and stabilizes cilia
inferredcites →https://doi.org/10.1371/journal.pgen.1011102openalex-snapshot
Constitutively active CaMKII Drives B lineage acute lymphoblastic leukemia/lymphoma in tp53 mutant zebrafish
inferredcites →https://doi.org/10.15252/embj.201488784openalex-snapshot
Discrete Notch signaling requirements in the specification of hematopoietic stem cells
inferredcites →https://doi.org/10.18632/oncotarget.20504openalex-snapshot
Activation of CaMKIIγ potentiates T-cell acute lymphoblastic leukemia leukemogenesis via phosphorylating FOXO3a
inferredcites →https://doi.org/10.3390/cells11030358openalex-snapshot
Notch Signaling in HSC Emergence: When, Why and How
inferredcites →https://doi.org/10.3390/genes11050495openalex-snapshot
Activation of Steroidogenesis, Anti-Apoptotic Activity, and Proliferation in Porcine Granulosa Cells by RUNX1 Is Negatively Regulated by H3K27me3 Transcriptional Repression
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