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Functional Genomics of Brain Aging and Alzheimers Disease: Focus on Selective Neuronal Vulnerability

https://doi.org/10.2174/138920210793360943
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Neuron Number in the Entorhinal Cortex and CA1 in Preclinical Alzheimer Disease
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PGC-1α Expression Decreases in the Alzheimer Disease Brain as a Function of Dementia
inferredcites →https://doi.org/10.1002/1531-8249(200007)48:1<77::aid-ana12>3.0.co;2-aopenalex-snapshot
Expression profile of transcripts in Alzheimer's disease tangle-bearing CA1 neurons
inferredcites →https://doi.org/10.1002/1531-8249(20010201)49:2<202::aid-ana40>3.0.co;2-3openalex-snapshot
Loss and atrophy of layer II entorhinal cortex neurons in elderly people with mild cognitive impairment
inferredcites →https://doi.org/10.1002/9780470513422.ch3openalex-snapshot
Dysfunction and Death of Neurons in Human Degenerative Neurological Diseases and in Animal Models
inferredcites →https://doi.org/10.1002/ana.20667openalex-snapshot
Model‐guided microarray implicates the retromer complex in Alzheimer's disease
inferredcites →https://doi.org/10.1002/ana.410210603openalex-snapshot
Neocortical cell counts in normal human adult aging
inferredcites →https://doi.org/10.1002/ana.410300410openalex-snapshot
Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment
inferredcites →https://doi.org/10.1002/ana.410340416openalex-snapshot
Oxidative damage to mitochondrial DNA shows marked age‐dependent increases in human brain
inferredcites →https://doi.org/10.1002/ana.410410106openalex-snapshot
Neuronal loss correlates with but exceeds neurofibrillary tangles in Alzheimer's disease
inferredcites →https://doi.org/10.1002/ana.410440706openalex-snapshot
Determinants of neuronal vulnerability in neurodegenerative diseases
inferredcites →https://doi.org/10.1002/cne.903010105openalex-snapshot
Quantitative analysis of a vulnerable subset of pyramidal neurons in Alzheimer's disease: I. Superior frontal and inferior temporal cortex
inferredcites →https://doi.org/10.1002/hipo.20555openalex-snapshot
A molecular blueprint of gene expression in hippocampal subregions CA1, CA3, and DG is conserved in the brain of the common marmoset
inferredcites →https://doi.org/10.1002/jnr.10351openalex-snapshot
Gene expression profiling of 12633 genes in Alzheimer hippocampal CA1: Transcription and neurotrophic factor down‐regulation and up‐regulation of apoptotic and pro‐inflammatory signaling
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Genome wide profiling of altered gene expression in the neocortex of Alzheimer's disease
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Regional neuronal loss in aging and Alzheimer's disease: a brief review
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Alzheimer's disease affects limbic nuclei of the thalamus
inferredcites →https://doi.org/10.1007/bf00307624openalex-snapshot
Laminar distribution of neuron degeneration in posterior cingulate cortex in Alzheimer's disease
inferredcites →https://doi.org/10.1007/bf00308809openalex-snapshot
Neuropathological stageing of Alzheimer-related changes
inferredcites →https://doi.org/10.1007/bf00687883openalex-snapshot
Demonstration of microglial cells in and around senile (neuritic) plaques in the Alzheimer brain
inferredcites →https://doi.org/10.1007/bf01245030openalex-snapshot
Synapsin I (protein I) in different brain regions in senile dementia of Alzheimer type and in multiinfarct dementia
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Complement activation in amyloid plaques in Alzheimer’s dementia
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Role of microglia in plaque formation in senile dementia of the Alzheimer type
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Age-related decline in metabolic competence of small and medium-sized synaptic mitochondria
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Reactive microglia in aging and dementia: an immunohistochemical study of postmortem human brain tissue
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The significance of neuroinflammation in understanding Alzheimer’s disease
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Sub-regional hippocampal vulnerability in various animal models leading to cognitive dysfunction
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Oligonucleotide-based Analysis of Differentially Expressed Genes in Hippocampus of Transgenic Mice Expressing NSE-controlled APPsw
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Aging modifies brain region-specific vulnerability to experimental oxidative stress induced by low dose hydrogen peroxide
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A quantitative morphometric analysis of the neuronal and synaptic content of the frontal and temporal cortex in patients with Alzheimer's disease
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The human entorhinal cortex: normal morphology and lamina-specific pathology in various diseases
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Hypothesis on the Regulation of Cytosol Calcium Concentration and the Aging Brain
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Regional synaptic pathology in Alzheimer's disease
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The role of cortical connectivity in Alzheimer's disease pathogenesis: A review and model system
inferredcites →https://doi.org/10.1016/0301-0082(89)90009-9openalex-snapshot
Functional organization of the extrinsic and intrinsic circuitry of the parahippocampal region
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Selective vulnerability of the hippocampus in brain ischemia
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Soluble oligomers of the amyloid β-protein impair synaptic plasticity and behavior
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Gene expression profiles of metabolic enzyme transcripts in Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.molbrainres.2005.07.018openalex-snapshot
High intrinsic oxidative stress may underlie selective vulnerability of the hippocampal CA1 region
inferredcites →https://doi.org/10.1016/j.nbd.2005.09.004openalex-snapshot
Alzheimer's disease: mRNA expression profiles of multiple patients show alterations of genes involved with calcium signaling
inferredcites →https://doi.org/10.1016/j.neubiorev.2006.07.001openalex-snapshot
Differential aging of the brain: Patterns, cognitive correlates and modifiers
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2005.02.010openalex-snapshot
Comparative analysis of cortical gene expression in mouse models of Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2006.03.007openalex-snapshot
Measurement of hippocampal subfields and age-related changes with high resolution MRI at 4T
inferredcites →https://doi.org/10.1016/j.neuroimage.2009.07.025openalex-snapshot
Higher serum total cholesterol levels in late middle age are associated with glucose hypometabolism in brain regions affected by Alzheimer's disease and normal aging
inferredcites →https://doi.org/10.1016/j.neuroscience.2006.09.060openalex-snapshot
Genome dynamics and transcriptional deregulation in aging
inferredcites →https://doi.org/10.1016/j.neuroscience.2007.03.015openalex-snapshot
Genome instability and DNA repair in brain, ageing and neurological disease
inferredcites →https://doi.org/10.1016/j.neuroscience.2009.10.003openalex-snapshot
Selective vulnerability of brain regions to oxidative stress in a non-coma model of insulin-induced hypoglycemia
inferredcites →https://doi.org/10.1016/j.tins.2008.10.003openalex-snapshot
Deciphering complex mechanisms in neurodegenerative diseases: the advent of systems biology
inferredcites →https://doi.org/10.1016/j.yexcr.2006.10.028openalex-snapshot
Alterations in immunological and neurological gene expression patterns in Alzheimer's disease tissues
inferredcites →https://doi.org/10.1016/s0006-8993(02)02558-1openalex-snapshot
Age-related changes in cerebral blood flow and glucose metabolism in conscious rhesus monkeys
inferredcites →https://doi.org/10.1016/s0022-510x(00)00285-9openalex-snapshot
Selective regional loss of exocytotic presynaptic vesicle proteins in Alzheimer’s disease brains
inferredcites →https://doi.org/10.1016/s0022-510x(00)00396-8openalex-snapshot
Comparison of regional cerebral blood flow and glucose metabolism in the normal brain: effect of aging
inferredcites →https://doi.org/10.1016/s0022-510x(99)00286-5openalex-snapshot
Low cerebral glucose extraction rates in the human medial temporal cortex and cerebellum
inferredcites →https://doi.org/10.1016/s0074-7742(06)81004-4openalex-snapshot
Life and Death of Neurons in The Aging Cerebral Cortex
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Chapter 37 Selective vulnerability of corticocortical and hippocampal circuits in aging and Alzheimer's disease
inferredcites →https://doi.org/10.1016/s0140-6736(94)92338-8openalex-snapshot
Differences in the pattern of hippocampal neuronal loss in normal ageing and Alzheimer's disease
inferredcites →https://doi.org/10.1016/s0197-4580(00)00124-xopenalex-snapshot
Inflammation and Alzheimer's disease
inferredcites →https://doi.org/10.1016/s0197-4580(01)00230-5openalex-snapshot
Hippocampal formation glucose metabolism and volume losses in MCI and AD
inferredcites →https://doi.org/10.1016/s0197-4580(01)00289-5openalex-snapshot
Inflammation, autotoxicity and Alzheimer disease
inferredcites →https://doi.org/10.1016/s0197-4580(88)80079-4openalex-snapshot
Expression of immune system-associated antigens by cells of the human central nervous system: Relationship to the pathology of Alzheimer's disease
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Altered expression of a-type but not b-type synapsin isoform in the brain of patients at high risk for Alzheimer's disease assessed by DNA microarray technique
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Selective age-related loss of CALBINDIN-D28k from basal forebrain cholinergic neurons in the common marmoset (callithrix jacchus)
inferredcites →https://doi.org/10.1016/s0969-9961(02)00009-8openalex-snapshot
Defects in expression of genes related to synaptic vesicle traffickingin frontal cortex of Alzheimer’s disease
inferredcites →https://doi.org/10.1016/s0969-9961(03)00126-8openalex-snapshot
DNA microarray analysis of functionally discrete human brain regions reveals divergent transcriptional profiles
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Laser microdissection in CNS research
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Reliability and Reproducibility of Gene Expression Measurements Using Amplified RNA from Laser-Microdissected Primary Breast Tissue with Oligonucleotide Arrays
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Selective vulnerability in adult and ageing mammalian neurons
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Immune System Response in Alzheimer's Disease
inferredcites →https://doi.org/10.1023/a:1020952704398openalex-snapshot
Gene Expression Profiles of Cholinergic Nucleus Basalis Neurons in Alzheimer's Disease
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Brain development during childhood and adolescence: a longitudinal MRI study
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Melanized dopaminergic neurons are differentially susceptible to degeneration in Parkinson's disease
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Gene regulation and DNA damage in the ageing human brain
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Mitochondrial DNA deletions in human brain: regional variability and increase with advanced age
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Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid β-peptide
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Neural plasticity in the ageing brain
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Ageing and neuronal vulnerability
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A TOMM40 variable-length polymorphism predicts the age of late-onset Alzheimer's disease
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Synaptic memory mechanisms: Alzheimer's disease amyloid β-peptide-induced dysfunction
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β‐Amyloid deposition and neurofibrillary tangle formation in the olfactory bulb in ageing and Alzheimer’s disease
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Differential Vulnerability of the CA1 and CA3 Subfields of the Hippocampus to Superoxide and Hydroxyl Radicals In Vitro
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Incipient Alzheimer's disease: Microarray correlation analyses reveal major transcriptional and tumor suppressor responses
inferredcites →https://doi.org/10.1073/pnas.0709259105openalex-snapshot
Alzheimer's disease is associated with reduced expression of energy metabolism genes in posterior cingulate neurons
inferredcites →https://doi.org/10.1073/pnas.0806883105openalex-snapshot
Gene expression changes in the course of normal brain aging are sexually dimorphic
inferredcites →https://doi.org/10.1073/pnas.090106797openalex-snapshot
Cerebral metabolic and cognitive decline in persons at genetic risk for Alzheimer's disease
inferredcites →https://doi.org/10.1073/pnas.1010459107openalex-snapshot
Regional aerobic glycolysis in the human brain
inferredcites →https://doi.org/10.1073/pnas.1010461107openalex-snapshot
Spatial correlation between brain aerobic glycolysis and amyloid-β (Aβ) deposition
inferredcites →https://doi.org/10.1073/pnas.191044198openalex-snapshot
Prediction of cognitive decline in normal elderly subjects with 2-[ 18 F]fluoro-2-deoxy- d -glucose/positron-emission tomography (FDG/PET)
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Anatomical correlates of the distribution of the pathological changes in the neocortex in Alzheimer disease.
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Preserved neuron number in the hippocampus of aged rats with spatial learning deficits.
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The Topographical and Neuroanatomical Distribution of Neurofibrillary Tangles and Neuritic Plaques in the Cerebral Cortex of Patients with Alzheimer's Disease
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Life and Death of Neurons in the Aging Cerebral Cortex
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Human Fascia Dentata Anatomy and Hippocampal Neuron Densities Differ Depending on the Epileptic Syndrome and Age at First Seizure
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Amyotrophic lateral sclerosis
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Microglia and Alzheimer’s disease
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Calcium Hypothesis of Alzheimer's Disease and Brain Aging a
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Life and Death of Neurons in the Aging Brain
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Alzheimer's Disease: Cell-Specific Pathology Isolates the Hippocampal Formation
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Interleukin-10 and the Interleukin-10 Receptor
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Gene expression profiles in anatomically and functionally distinct regions of the normal aged human brain
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Altered neuronal gene expression in brain regions differentially affected by Alzheimer's disease: a reference data set
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Greater Metabolic Rate Decreases in Hippocampal Formation and Proisocortex than in Neocortex in Alzheimer’s Disease
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Longitudinal PET Evaluation of Cerebral Metabolic Decline in Dementia: A Potential Outcome Measure in Alzheimer’s Disease Treatment Studies
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Cerebral Metabolic Correlates of the Clinical Dementia Rating Scale in Mild Cognitive Impairment
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Reviews: Current Concepts in Alzheimer's Disease: A Multidisciplinary Review
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Transcriptomic responses in mouse brain exposed to chronic excess of the neurotransmitter glutamate
inferredcites →https://doi.org/10.1186/1471-2202-10-12openalex-snapshot
Genomic and biochemical approaches in the discovery of mechanisms for selective neuronal vulnerability to oxidative stress
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Molecular signatures of neurodegeneration in the cortex of PS1/PS2 double knockout mice
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Synaptic alterations in CA1 in mild Alzheimer disease and mild cognitive impairment
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Aging and Gene Expression in the Primate Brain
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Profound Loss of Layer II Entorhinal Cortex Neurons Occurs in Very Mild Alzheimer’s Disease
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Selectively Reduced Expression of Synaptic Plasticity-Related Genes in Amyloid Precursor Protein + Presenilin-1 Transgenic Mice
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The Alzheimer's Disease Mitochondrial Cascade Hypothesis
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Selective neuronal vulnerability to oxidative stress in the brain

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Spatial Topography of Alzheimerrs Degeneration Reflects the Cholinergic Projection System
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Transcriptional signatures mediated by acetylation overlap with early-stage Alzheimer’s disease
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Aerobic glycolysis in the primate brain: reconsidering the implications for growth and maintenance
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Metabolism Changes During Aging in the Hippocampus and Striatum of Glud1 (Glutamate Dehydrogenase 1) Transgenic Mice
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The role for DNA/RNA methylation on neurocognitive dysfunctions
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Molecular pathways
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Lipidomics of Human Brain Aging and Alzheimer's Disease Pathology
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Altered function of neuronal L-type calcium channels in ageing and neuroinflammation: Implications in age-related synaptic dysfunction and cognitive decline
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Aging, microglia and cytoskeletal regulation are key factors in the pathological evolution of the APP23 mouse model for Alzheimer's disease
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Ad-derived bone marrow transplant induces proinflammatory immune peripheral mechanisms accompanied by decreased neuroplasticity and reduced gut microbiome diversity affecting AD-like phenotype in the absence of Aβ neuropathology
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Behavioral and hypothalamic transcriptome analyses reveal sex-specific responses to phenanthrene exposure in the fathead minnow (Pimephales promelas)
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Longitudinal Alzheimer’s Degeneration Reflects the Spatial Topography of Cholinergic Basal Forebrain Projections
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FK506-binding protein 1b/12.6: A key to aging-related hippocampal Ca2+ dysregulation?
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Cognitive function and cerebellar morphometric changes relate to abnormal intra-cerebellar and cerebro-cerebellum functional connectivity in old adults
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Redox lipidomics to better understand brain aging and function
inferred← citeshttps://doi.org/10.1016/j.jalz.2016.12.006openalex-snapshot
Calcium Hypothesis of Alzheimer's disease and brain aging: A framework for integrating new evidence into a comprehensive theory of pathogenesis
inferred← citeshttps://doi.org/10.1016/j.jalz.2019.01.011openalex-snapshot
Novel methods for integration and visualization of genomics and genetics data in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.jchemneu.2011.06.007openalex-snapshot
Microarray analyses of laser-captured hippocampus reveal distinct gray and white matter signatures associated with incipient Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.molmed.2017.04.003openalex-snapshot
A CRISPR Approach to Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1016/j.pneurobio.2017.03.006openalex-snapshot
Autophagic flux control in neurodegeneration: Progress and precision targeting—Where do we stand?
inferred← citeshttps://doi.org/10.1016/j.redox.2023.102943openalex-snapshot
Adaptive responses of neuronal cells to chronic endoplasmic reticulum (ER) stress
inferred← citeshttps://doi.org/10.1016/j.tics.2018.02.011openalex-snapshot
Normalization and the Cholinergic Microcircuit: A Unified Basis for Attention
inferred← citeshttps://doi.org/10.1016/j.toxlet.2018.08.020openalex-snapshot
Excessive apoptosis and disordered autophagy flux contribute to the neurotoxicity induced by high iodine in Sprague-Dawley rat
inferred← citeshttps://doi.org/10.1021/acschemneuro.4c00339openalex-snapshot
Alzheimer’s Disease: Exploring the Landscape of Cognitive Decline
inferred← citeshttps://doi.org/10.1021/cn400082dopenalex-snapshot
Electroosmotic Push–Pull Perfusion: Description and Application to Qualitative Analysis of the Hydrolysis of Exogenous Galanin in Organotypic Hippocampal Slice Cultures
inferred← citeshttps://doi.org/10.1038/s41380-026-03556-wopenalex-snapshot
Neuronal HDAC9: A key regulator of cognitive and synaptic aging, rescuing Alzheimer’s disease-related phenotypes
inferred← citeshttps://doi.org/10.1038/s41398-024-02958-0openalex-snapshot
Hippocampal transcriptome-wide association study and pathway analysis of mitochondrial solute carriers in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1038/s41467-017-01444-0openalex-snapshot
Neuronal hyperactivity due to loss of inhibitory tone in APOE4 mice lacking Alzheimer’s disease-like pathology
inferred← citeshttps://doi.org/10.1038/s41467-026-69866-3openalex-snapshot
Selective weakening of population-coupled synaptic activity in vivo in a mouse model of amyloid-beta pathology
inferred← citeshttps://doi.org/10.1038/s41598-017-12603-0openalex-snapshot
Proteomic mapping of differentially vulnerable pre-synaptic populations identifies regulators of neuronal stability in vivo
inferred← citeshttps://doi.org/10.1038/s41598-020-61142-8openalex-snapshot
APOE4 is Associated with Differential Regional Vulnerability to Bioenergetic Deficits in Aged APOE Mice
inferred← citeshttps://doi.org/10.1038/s41598-025-97830-6openalex-snapshot
Anti-inflammatory effects of 64Zn-aspartate is accompanied by cognitive improvements in rats with Aβ1-40-induced alzheimer disease
inferred← citeshttps://doi.org/10.1038/tp.2013.97openalex-snapshot
Latrepirdine: molecular mechanisms underlying potential therapeutic roles in Alzheimer’s and other neurodegenerative diseases
inferred← citeshttps://doi.org/10.1080/14789450.2021.1940142openalex-snapshot
New insights into human prefrontal cortex aging with a lipidomics approach
inferred← citeshttps://doi.org/10.1097/nen.0000000000000091openalex-snapshot
Metabolomics of Human Brain Aging and Age-Related Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1109/tcds.2023.3254209openalex-snapshot
Deep-Learning-Based Diagnosis and Prognosis of Alzheimer’s Disease: A Comprehensive Review
inferred← citeshttps://doi.org/10.1117/12.2256840openalex-snapshot
Exploring Alzheimer's anatomical patterns through convolutional networks
inferred← citeshttps://doi.org/10.1139/apnm-2021-0133openalex-snapshot
Combination of magnesium supplementation with treadmill exercise improves memory deficit in aged rats by enhancing hippocampal neurogenesis and plasticity: a functional and histological study
inferred← citeshttps://doi.org/10.1155/2015/626019openalex-snapshot
Role of the Insulin-Like Growth Factor Type 1 Receptor in the Pathogenesis of Diabetic Encephalopathy
inferred← citeshttps://doi.org/10.1186/1471-2164-15-199openalex-snapshot
Genomic convergence and network analysis approach to identify candidate genes in Alzheimer's disease
inferred← citeshttps://doi.org/10.1186/s13073-016-0355-3openalex-snapshot
Integrative network analysis of nineteen brain regions identifies molecular signatures and networks underlying selective regional vulnerability to Alzheimer’s disease
inferred← citeshttps://doi.org/10.1186/s40478-022-01452-2openalex-snapshot
Somatic copy number variant load in neurons of healthy controls and Alzheimer’s disease patients
inferred← citeshttps://doi.org/10.1242/dmm.048926openalex-snapshot
Cell competition from development to neurodegeneration
inferred← citeshttps://doi.org/10.12659/msm.919249openalex-snapshot
Microarray Analysis of Differential Gene Expression in Alzheimer’s Disease Identifies Potential Biomarkers with Diagnostic Value
inferred← citeshttps://doi.org/10.1371/journal.pone.0077572openalex-snapshot
Faster Metabolite 1H Transverse Relaxation in the Elder Human Brain
inferred← citeshttps://doi.org/10.1371/journal.pone.0082481openalex-snapshot
The Contribution of Lysosomotropism to Autophagy Perturbation
inferred← citeshttps://doi.org/10.1371/journal.pone.0108807openalex-snapshot
Selective Vulnerability Related to Aging in Large-Scale Resting Brain Networks
inferred← citeshttps://doi.org/10.14283/vme.2023.2openalex-snapshot
The Calcium Signaling System: Aiming for a Comprehensive Explanation of Aging and the Dementia-Alzheimer’s Syndrome
inferred← citeshttps://doi.org/10.14336/ad.2024.0429openalex-snapshot
Systematic Analysis of Biological Processes Reveals Gene Co-expression Modules Driving Pathway Dysregulation in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.14800/ttnd.503openalex-snapshot
Neuropsin is associated with MAP2c dependent dendritic morphology in aging brain
inferred← citeshttps://doi.org/10.17219/acem/175049openalex-snapshot
Unraveling the therapeutic potential of ginsenoside compound Mc1 in Alzheimer’s disease: Exploring the role of AMPK/SIRT1/NF-κB signaling pathway and mitochondrial function
inferred← citeshttps://doi.org/10.18632/oncotarget.4215openalex-snapshot
DNA methylation and cognitive aging
inferred← citeshttps://doi.org/10.2174/0929866530666230419093531openalex-snapshot
Effect of Bromelain on Chronic Unpredictable Stress-induced Behavioral, Biochemical, and Monoamine Changes in Wistar Albino Rat Model of Depression
inferred← citeshttps://doi.org/10.21926/obm.neurobiol.2002059openalex-snapshot
Dynamic Functional Connectivity Fading Analysis and Classification of Alzheimer’s Disease, Mild Cognitive Impairment and Normal Control Subjects based on Resting-State fMRI Data
inferred← citeshttps://doi.org/10.26481/marble.2015.v6.384openalex-snapshot
DNA methylation in the hippocampus in rodent and primate models of aging and Alzheimer’s disease
inferred← citeshttps://doi.org/10.3109/21678421.2013.812660openalex-snapshot
Does variation in neurodegenerative disease susceptibility and phenotype reflect cerebral differences at the network level?
inferred← citeshttps://doi.org/10.3233/jad-170011openalex-snapshot
Analytical Strategy to Prioritize Alzheimer’s Disease Candidate Genes in Gene Regulatory Networks Using Public Expression Data
inferred← citeshttps://doi.org/10.3389/fgene.2023.1114749openalex-snapshot
A protein-coding gene expression atlas from the brain of pregnant and non-pregnant goats
inferred← citeshttps://doi.org/10.3389/fnagi.2022.1058665openalex-snapshot
The expression pattern of GDF15 in human brain changes during aging and in Alzheimer’s disease
inferred← citeshttps://doi.org/10.3389/fnagi.2022.1069606openalex-snapshot
Preliminary exploration of the co-regulation of Alzheimer’s disease pathogenic genes by microRNAs and transcription factors
inferred← citeshttps://doi.org/10.3389/fnbeh.2022.861155openalex-snapshot
The Brilliance of the Zebrafish Model: Perception on Behavior and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3389/fnins.2017.00702openalex-snapshot
The Endosomal–Lysosomal Pathway Is Dysregulated by APOE4 Expression in Vivo
inferred← citeshttps://doi.org/10.3389/fnins.2020.602642openalex-snapshot
Transcriptional, Behavioral and Biochemical Profiling in the 3xTg-AD Mouse Model Reveals a Specific Signature of Amyloid Deposition and Functional Decline in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3389/fnins.2021.666958openalex-snapshot
Reassessment of Pioglitazone for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3390/antiox9090902openalex-snapshot
Antioxidant and Neuroprotective Effects of Caffeine against Alzheimer’s and Parkinson’s Disease: Insight into the Role of Nrf-2 and A2AR Signaling
inferred← citeshttps://doi.org/10.3390/ijms20153810openalex-snapshot
Lipid and Lipid Raft Alteration in Aging and Neurodegenerative Diseases: A Window for the Development of New Biomarkers
inferred← citeshttps://doi.org/10.3390/ijms21207660openalex-snapshot
Copper Toxicity Links to Pathogenesis of Alzheimer’s Disease and Therapeutics Approaches
inferred← citeshttps://doi.org/10.3390/make5020031openalex-snapshot
Alzheimer’s Disease Detection from Fused PET and MRI Modalities Using an Ensemble Classifier
inferred← citeshttps://doi.org/10.3390/molecules30040874openalex-snapshot
11C-Labeling of a Flavanone Extracted from a South American Native Species for Evaluation of Its Interaction with GSK-3β
inferred← citeshttps://doi.org/10.3892/mmr.2018.9190openalex-snapshot
Identification of a noncoding RNA‑mediated gene pair‑based regulatory module in Alzheimer's disease
inferred← citeshttps://doi.org/10.4103/1673-5374.141784openalex-snapshot
Mechanisms underlying attenuation of apoptosis of cortical neurons in the hypoxic brain by flavonoids from the stems and leaves of Scutellaria baicalensis Georgi
inferred← citeshttps://doi.org/10.52711/2321-5836.2024.00013openalex-snapshot
Evaluation of Neuroprotective Activity of Carica papaya Seeds Extract in Okadaic Acid Induced Alzheimer’s Disease in Zebra Fish Model (Danio rerio)
inferred← citeshttps://doi.org/10.52711/2321-5836.2025.00001openalex-snapshot
Neuroprotective Activity of Epiphyllum oxypetallum Leaves Extract on Scopolamine Induced Alzhemier’s Disease Model
inferred← citeshttps://doi.org/10.52711/2321-5836.2025.00020openalex-snapshot
Molecular Targets of Alzheimer’s Disease Treatment
inferred← citeshttps://doi.org/10.7150/thno.79535openalex-snapshot
Role of primary aging hallmarks in Alzheimer´s disease
inferred← citeshttps://doi.org/10.7287/peerj.8682v0.1/reviews/1openalex-snapshot
Peer Review #1 of "Comparative functional genomic analysis of Alzheimer’s affected and naturally aging brains (v0.1)"
inferred← citeshttps://doi.org/10.7287/peerj.8682v0.1/reviews/2openalex-snapshot
Peer Review #2 of "Comparative functional genomic analysis of Alzheimer’s affected and naturally aging brains (v0.1)"
inferred← citeshttps://doi.org/10.7717/peerj.8682openalex-snapshot
Comparative functional genomic analysis of Alzheimer’s affected and naturally aging brains
Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.