On the public citation graph
Mitochondrial Energetics in the Heart in Obesity-Related Diabetes
0 signed · 619 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/978-1-4615-5653-4_7openalex-snapshot
Permeabilized cell and skinned fiber techniques in studies of mitochondrial function in vivo
inferredcites →https://doi.org/10.1016/0005-2728(87)90174-5openalex-snapshot
Mitochondrial respiratory parameters in cardiac tissue: A novel method of assessment by using saponin-skinned fibers
inferredcites →https://doi.org/10.1016/0006-2944(83)90054-6openalex-snapshot
Carnitine content of skeletal and cardiac muscle from genetically diabetic () and control mice
inferredcites →https://doi.org/10.1016/0014-5793(90)80259-lopenalex-snapshot
Does the function of adenine nucleotide translocase in fatty acid uncoupling depend on the type of mitochondria?
inferredcites →https://doi.org/10.1016/j.bbabio.2005.06.002openalex-snapshot
The reactions catalysed by the mitochondrial uncoupling proteins UCP2 and UCP3
inferredcites →https://doi.org/10.1016/j.bbrc.2003.12.121openalex-snapshot
Hypoxia-induced decrease of UCP3 gene expression in rat heart parallels metabolic gene switching but fails to affect mitochondrial respiratory coupling
inferredcites →https://doi.org/10.1016/j.cell.2006.09.024openalex-snapshot
Suppression of Reactive Oxygen Species and Neurodegeneration by the PGC-1 Transcriptional Coactivators
inferredcites →https://doi.org/10.1016/j.ejheart.2006.03.001openalex-snapshot
Type II Diabetic Mice Exhibit Contractile Dysfunction but Maintain Cardiac Output by Favourable Loading Conditions
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2004.05.034openalex-snapshot
Mitochondrial superoxide: production, biological effects, and activation of uncoupling proteins
inferredcites →https://doi.org/10.1016/s0005-2736(98)00232-6openalex-snapshot
Structure and function of the uncoupling protein from brown adipose tissue
inferredcites →https://doi.org/10.1023/a:1005427919188openalex-snapshot
Mitochondrial Oxygen Radical Generation and Leak: Sites of Production in States 4 and 3, Organ Specificity, and Relation to Aging and Longevity
inferredcites →https://doi.org/10.1038/415096aopenalex-snapshot
Superoxide activates mitochondrial uncoupling proteins
inferredcites →https://doi.org/10.1042/bj20050890openalex-snapshot
The basal proton conductance of mitochondria depends on adenine nucleotide translocase content
inferredcites →https://doi.org/10.1042/bj2410883openalex-snapshot
Reactions of electron-transfer flavoprotein and electron-transfer flavoprotein: ubiquinone oxidoreductase
inferredcites →https://doi.org/10.1055/s-2006-950397openalex-snapshot
Oxidative Stress in Young Zucker Rats with Impaired Glucose Tolerance is Diminished by Acarbose
inferredcites →https://doi.org/10.1073/pnas.0336724100openalex-snapshot
A critical role for PPARα-mediated lipotoxicity in the pathogenesis of diabetic cardiomyopathy: Modulation by dietary fat content
inferredcites →https://doi.org/10.1073/pnas.0604567103openalex-snapshot
Structure of electron transfer flavoprotein-ubiquinone oxidoreductase and electron transfer to the mitochondrial ubiquinone pool
inferredcites →https://doi.org/10.1074/jbc.m308529200openalex-snapshot
Superoxide Activates Uncoupling Proteins by Generating Carbon-centered Radicals and Initiating Lipid Peroxidation
inferredcites →https://doi.org/10.1074/jbc.m405339200openalex-snapshot
Hydroperoxy Fatty Acid Cycling Mediated by Mitochondrial Uncoupling Protein UCP2
inferredcites →https://doi.org/10.1074/jbc.m505258200openalex-snapshot
Uncoupling Proteins 2 and 3 Function in Concert to Augment Tolerance to Cardiac Ischemia
inferredcites →https://doi.org/10.1074/jbc.m601387200openalex-snapshot
UCP1 and Defense against Oxidative Stress
inferredcites →https://doi.org/10.1093/emboj/cdg412openalex-snapshot
A signalling role for 4‐hydroxy‐2‐nonenal in regulation of mitochondrial uncoupling
inferredcites →https://doi.org/10.1096/fasebj.11.10.9271366openalex-snapshot
A role for uncoupling protein‐2 as a regulator of mitochondrial hydrogen peroxide generation
inferredcites →https://doi.org/10.1096/fj.03-0159hypopenalex-snapshot
A function for novel uncoupling proteins: antioxidant defense of mitochondrial matrix by translocating fatty acid peroxides from the inner to the outer membrane leaflet
inferredcites →https://doi.org/10.1124/jpet.106.110460openalex-snapshot
Calorie Restriction Improves Cardiovascular Risk Factors via Reduction of Mitochondrial Reactive Oxygen Species in Type II Diabetic Rats
inferredcites →https://doi.org/10.1139/y04-065openalex-snapshot
Diabetic cardiomyopathy: recent evidence from mouse models of type 1 and type 2 diabetes
inferredcites →https://doi.org/10.1152/ajpendo.00047.2004openalex-snapshot
Cardiac mitochondrial damage and biogenesis in a chronic model of type 1 diabetes
inferredcites →https://doi.org/10.1152/ajpendo.00233.2005openalex-snapshot
Cardiac metabolism in mice: tracer method developments and in vivo application revealing profound metabolic inflexibility in diabetes
inferredcites →https://doi.org/10.1152/ajpheart.00088.2002openalex-snapshot
Echocardiographic assessment of cardiac function in diabeticdb/dband transgenicdb/db-hGLUT4 mice
inferredcites →https://doi.org/10.1152/ajpheart.00406.2005openalex-snapshot
The intrinsic circadian clock within the cardiomyocyte
inferredcites →https://doi.org/10.1152/ajpheart.00856.2006openalex-snapshot
Magnetic resonance imaging of progressive cardiomyopathic changes in the db/db mouse
inferredcites →https://doi.org/10.1152/ajpheart.00925.2003openalex-snapshot
Regular exercise is associated with a protective metabolic phenotype in the rat heart
inferredcites →https://doi.org/10.1152/ajpheart.2001.280.3.h977openalex-snapshot
Increased uncoupling proteins and decreased efficiency in palmitate-perfused hyperthyroid rat heart
inferredcites →https://doi.org/10.1152/physiol.00008.2006openalex-snapshot
Mitochondrial Uncoupling: A Key Contributor to Reduced Cardiac Efficiency in Diabetes
inferredcites →https://doi.org/10.1152/physrev.1984.64.1.1openalex-snapshot
Thermogenic mechanisms in brown fat.
inferredcites →https://doi.org/10.1161/01.cir.0000127959.28627.f8openalex-snapshot
Effect of Obesity and Insulin Resistance on Myocardial Substrate Metabolism and Efficiency in Young Women
inferredcites →https://doi.org/10.1161/01.res.0000117088.36577.ebopenalex-snapshot
Cardiac-Specific Induction of the Transcriptional Coactivator Peroxisome Proliferator-Activated Receptor γ Coactivator-1α Promotes Mitochondrial Biogenesis and Reversible Cardiomyopathy in a Developmental Stage-Dependent Manner
inferredcites →https://doi.org/10.1161/circulationaha.105.554360openalex-snapshot
Reduced Mitochondrial Oxidative Capacity and Increased Mitochondrial Uncoupling Impair Myocardial Energetics in Obesity
inferredcites →https://doi.org/10.1161/circulationaha.106.662296openalex-snapshot
Insulin-Resistant Heart Exhibits a Mitochondrial Biogenic Response Driven by the Peroxisome Proliferator-Activated Receptor-α/PGC-1α Gene Regulatory Pathway
inferredcites →https://doi.org/10.1161/circulationaha.106.679597openalex-snapshot
Diabetic Cardiomyopathy Revisited
inferredcites →https://doi.org/10.1172/jci0214080openalex-snapshot
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
inferredcites →https://doi.org/10.1172/jci10268openalex-snapshot
Peroxisome proliferator–activated receptor γ coactivator-1 promotes cardiac mitochondrial biogenesis
inferredcites →https://doi.org/10.1172/jci14080openalex-snapshot
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
inferredcites →https://doi.org/10.1172/jci200524405openalex-snapshot
Mitochondrial energy metabolism in heart failure: a question of balance
inferredcites →https://doi.org/10.1196/annals.1338.061openalex-snapshot
Alleviation of Oxidative Damage in Multiple Tissues in Rats with Streptozotocin‐Induced Diabetes by Rice Bran Oil Supplementation
inferredcites →https://doi.org/10.1210/en.2005-0938openalex-snapshot
Reduced Cardiac Efficiency and Altered Substrate Metabolism Precedes the Onset of Hyperglycemia and Contractile Dysfunction in Two Mouse Models of Insulin Resistance and Obesity
inferredcites →https://doi.org/10.2337/diabetes.51.8.2587openalex-snapshot
Impaired Long-Chain Fatty Acid Oxidation and Contractile Dysfunction in the Obese Zucker Rat Heart
inferredcites →https://doi.org/10.2337/diabetes.53.5.1336openalex-snapshot
Catalase Protects Cardiomyocyte Function in Models of Type 1 and Type 2 Diabetes
inferredcites →https://doi.org/10.2337/diabetes.53.9.2366openalex-snapshot
Impaired Cardiac Efficiency and Increased Fatty Acid Oxidation in Insulin-Resistant ob/ob Mouse Hearts
inferredcites →https://doi.org/10.2337/diabetes.54.12.3496openalex-snapshot
Plasma Free Fatty Acids and Peroxisome Proliferator–Activated Receptor α in the Control of Myocardial Uncoupling Protein Levels
inferredcites →https://doi.org/10.2337/diabetes.55.02.06.db05-1164openalex-snapshot
Increased Myocardial Oxygen Consumption Reduces Cardiac Efficiency in Diabetic Mice
inferredcites →https://doi.org/10.2337/diabetes.55.03.06.db05-1039openalex-snapshot
Protection of Cardiac Mitochondria by Overexpression of MnSOD Reduces Diabetic Cardiomyopathy
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4641193openalex-extracted
Visualizing the Role of ER Stress in Diabetic Cardiomyopathy: A Bibliometric Analysis
inferred← citeshttps://doi.org/10.1002/cbf.3195openalex-snapshot
Increased 4‐hydroxy‐2‐nonenal‐induced proteasome dysfunction is correlated with cardiac damage in streptozotocin‐injected rats with isoproterenol infusion
inferred← citeshttps://doi.org/10.1002/chem.202404517openalex-snapshot
Discovery of 1,3,4‐Oxadiazin‐5‐One Derivative CJ1‐34 as a Partial ATP Synthase Inhibitor for CNS Applications
inferred← citeshttps://doi.org/10.1002/ctm2.736openalex-snapshot
BSCL2/Seipin deficiency in hearts causes cardiac energy deficit and dysfunction via inducing excessive lipid catabolism
inferred← citeshttps://doi.org/10.1002/ehf2.14224openalex-snapshot
Diabetic Cardiomyopathy: A Brief Summary on Lipid Toxicity
inferred← citeshttps://doi.org/10.1002/ejhf.3347openalex-snapshot
Diabetic Myocardial Disorder. A Clinical Consensus Statement of the Heart Failure Association of the ESC and the ESC Working Group on Myocardial & Pericardial Diseases
inferred← citeshttps://doi.org/10.1002/jcp.31293openalex-snapshot
Ablation of Sam50 is associated with fragmentation and alterations in metabolism in murine and human myotubes
inferred← citeshttps://doi.org/10.1002/mrm.29908openalex-snapshot
Quantitative 17O MRS I of myocardial oxygen metabolic rate, blood flow, and oxygen extraction fraction under normal and high workload conditions
inferred← citeshttps://doi.org/10.1002/oby.21072openalex-snapshot
Earlier onset of diabesity‐Induced adverse cardiac remodeling in female compared to male mice
inferred← citeshttps://doi.org/10.1007/0-387-29553-4_18openalex-snapshot
Oxidative Stress in Heart Failure
inferred← citeshttps://doi.org/10.1007/978-1-4419-1607-5_14openalex-snapshot
Obesity and Cardiac Dysfunction
inferred← citeshttps://doi.org/10.1007/978-1-4419-7130-2_12openalex-snapshot
Molecular Changes in Fatty Acid Oxidation in the Failing Heart
inferred← citeshttps://doi.org/10.1007/978-1-4614-4599-9_20openalex-snapshot
Diabetes and Cardiac Mitochondria
inferred← citeshttps://doi.org/10.1007/978-1-4899-8035-9_5openalex-snapshot
Mitochondria and Oxidative Stress in Diabetes
inferred← citeshttps://doi.org/10.1007/978-1-4939-1227-8_11openalex-snapshot
Fuel Metabolism Plasticity in Pathological Cardiac Hypertrophy and Failure
inferred← citeshttps://doi.org/10.1007/978-1-4939-1227-8_5openalex-snapshot
Cardiac Energy Metabolism in Heart Failure Associated with Obesity and Diabetes
inferred← citeshttps://doi.org/10.1007/978-1-4939-1227-8_7openalex-snapshot
Relationship Between Substrate Metabolism and Cardiac Efficiency
inferred← citeshttps://doi.org/10.1007/978-1-4939-6890-9_4openalex-snapshot
Techniques to Investigate Bioenergetics of Mitochondria
inferred← citeshttps://doi.org/10.1007/978-1-61779-891-7_3openalex-snapshot
Mitochondria in Cardiac Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-47336-5_7openalex-snapshot
Role of Sodium-Glucose Co-transporters on Cardiac Function in Metabolic Syndrome Mammalians
inferred← citeshttps://doi.org/10.1007/978-3-030-84763-0_15openalex-snapshot
Sympathetic Nervous System and Cardiovascular Alterations Due to Food Restrictions
inferred← citeshttps://doi.org/10.1007/978-3-031-14952-8_15openalex-snapshot
Renin Angiotensin System in the Pathophysiology of Diabetic Cardiomyopathy in Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-3-319-16018-4_3openalex-snapshot
Diabetes Mellitus and Metabolic Syndrome
inferred← citeshttps://doi.org/10.1007/978-3-319-31251-4_4openalex-snapshot
Cardiac Metabolism and Energetic Control
inferred← citeshttps://doi.org/10.1007/978-3-319-55330-6_6openalex-snapshot
Mitochondrial Function in Non-ischemic Heart Failure
inferred← citeshttps://doi.org/10.1007/978-3-642-17214-4_14openalex-snapshot
Mitochondria as Potential Targets in Antidiabetic Therapy
inferred← citeshttps://doi.org/10.1007/978-3-642-30018-9_44openalex-snapshot
Dietary Fatty Acids, Redox Signaling, and the Heart
inferred← citeshttps://doi.org/10.1007/978-981-10-4307-9_12openalex-snapshot
Exercise Amaliorates Metabolic Disturbances and Oxidative Stress in Diabetic Cardiomyopathy: Possible Underlying Mechanisms
inferred← citeshttps://doi.org/10.1007/978-981-19-8342-9_7openalex-snapshot
Mitochondrial Dysfunction as a Trigger of Inflammation in Cardiomyopathies
inferred← citeshttps://doi.org/10.1007/978-981-96-6831-1_6openalex-snapshot
Role of SDH in Cardiovascular Disorders
inferred← citeshttps://doi.org/10.1007/s00125-014-3171-6openalex-snapshot
Molecular mechanisms of diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s00125-021-05637-7openalex-snapshot
Metabolic, structural and biochemical changes in diabetes and the development of heart failure
inferred← citeshttps://doi.org/10.1007/s00395-011-0156-1openalex-snapshot
A high fat diet increases mitochondrial fatty acid oxidation and uncoupling to decrease efficiency in rat heart
inferred← citeshttps://doi.org/10.1007/s00395-013-0329-1openalex-snapshot
Impaired mitochondrial biogenesis due to dysfunctional adiponectin-AMPK-PGC-1α signaling contributing to increased vulnerability in diabetic heart
inferred← citeshttps://doi.org/10.1007/s00395-015-0493-6openalex-snapshot
SIRT3 deficiency impairs mitochondrial and contractile function in the heart
inferred← citeshttps://doi.org/10.1007/s00395-015-0495-4openalex-snapshot
Myocardial mitochondrial dysfunction in mice lacking adiponectin receptor 1
inferred← citeshttps://doi.org/10.1007/s00395-018-0707-9openalex-snapshot
Uncoupling protein 3 deficiency impairs myocardial fatty acid oxidation and contractile recovery following ischemia/reperfusion
inferred← citeshttps://doi.org/10.1007/s00395-018-0711-0openalex-snapshot
Of mice and men: models and mechanisms of diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s00395-023-01019-9openalex-snapshot
Targeting mitochondrial shape: at the heart of cardioprotection
inferred← citeshttps://doi.org/10.1007/s00424-021-02650-yopenalex-snapshot
Ca2+ mishandling and mitochondrial dysfunction: a converging road to prediabetic and diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s10554-012-0174-6openalex-snapshot
Effects of weight loss on myocardial energetics and diastolic function in obesity
inferred← citeshttps://doi.org/10.1007/s10554-014-0548-zopenalex-snapshot
Myocardial tissue phase mapping reveals impaired myocardial tissue velocities in obesity
inferred← citeshttps://doi.org/10.1007/s10557-018-6780-3openalex-snapshot
Pterostilbene Decreases Cardiac Oxidative Stress and Inflammation via Activation of AMPK/Nrf2/HO-1 Pathway in Fructose-Fed Diabetic Rats
inferred← citeshttps://doi.org/10.1007/s10741-010-9208-0openalex-snapshot
Enhancing the metabolic substrate: PPAR-alpha agonists in heart failure
inferred← citeshttps://doi.org/10.1007/s10741-012-9305-3openalex-snapshot
Rodent models of heart failure: an updated review
inferred← citeshttps://doi.org/10.1007/s10741-012-9320-4openalex-snapshot
Obesity cardiomyopathy and systolic function: Obesity is not independently associated with dilated cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s10741-013-9377-8openalex-snapshot
Metabolic dysfunction in diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s10741-018-9733-9openalex-snapshot
Lanosteryl triterpenes from Protorhus longifolia as a cardioprotective agent: a mini review
inferred← citeshttps://doi.org/10.1007/s10741-020-09997-xopenalex-snapshot
The roles of epicardial adipose tissue in heart failure
inferred← citeshttps://doi.org/10.1007/s10863-013-9503-2openalex-snapshot
UCP2 and ANT differently modulate proton-leak in brain mitochondria of long-term hyperglycemic and recurrent hypoglycemic rats
inferred← citeshttps://doi.org/10.1007/s10863-013-9530-zopenalex-snapshot
Dietary fat, fatty acid saturation and mitochondrial bioenergetics
inferred← citeshttps://doi.org/10.1007/s10974-021-09603-zopenalex-snapshot
Changes in myocardial myosin heavy chain isoform composition with exercise and post-exercise cold-water immersion
inferred← citeshttps://doi.org/10.1007/s11010-014-2236-7openalex-snapshot
Enhanced ROS production and oxidative damage in subcutaneous white adipose tissue mitochondria in obese and type 2 diabetes subjects
inferred← citeshttps://doi.org/10.1007/s11010-017-2993-1openalex-snapshot
Methylene blue decreases mitochondrial lysine acetylation in the diabetic heart
inferred← citeshttps://doi.org/10.1007/s11010-021-04108-2openalex-snapshot
Myocardial salvage by succinate dehydrogenase inhibition in ischemia–reperfusion injury depends on diabetes stage in rats
inferred← citeshttps://doi.org/10.1007/s11010-024-04951-zopenalex-snapshot
Role of AMP deaminase in diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s11154-010-9131-7openalex-snapshot
Diabetic cardiomyopathy, causes and effects
inferred← citeshttps://doi.org/10.1007/s11154-023-09850-0openalex-snapshot
An update on the secretory functions of brown, white, and beige adipose tissue: Towards therapeutic applications
inferred← citeshttps://doi.org/10.1007/s11332-022-00924-1openalex-snapshot
Irisin pathways in hearts of Type 1 diabetic adult male rats following 6 weeks of moderate and high-volume aerobic exercise on a treadmill
inferred← citeshttps://doi.org/10.1007/s11892-008-0042-xopenalex-snapshot
The complexities of diabetic cardiomyopathy: Lessons from patients and animal models
inferred← citeshttps://doi.org/10.1007/s12012-019-09514-zopenalex-snapshot
High Intensity Interval Training Ameliorates Mitochondrial Dysfunction in the Left Ventricle of Mice with Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/s12015-018-9858-1openalex-snapshot
Diabetic Cardiomyopathy Modelling Using Induced Pluripotent Stem Cell Derived Cardiomyocytes: Recent Advances and Emerging Models
inferred← citeshttps://doi.org/10.1007/s12020-012-9623-1openalex-snapshot
A new insight of mechanisms, diagnosis and treatment of diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s12170-014-0417-2openalex-snapshot
Diabetic Cardiomyopathy: Distinct and Preventable Entity or Inevitable Consequence?
inferred← citeshttps://doi.org/10.1007/s12192-023-01361-wopenalex-snapshot
Novel insights into the role of mitochondria in diabetic cardiomyopathy: molecular mechanisms and potential treatments
inferred← citeshttps://doi.org/10.1007/s12265-010-9197-3openalex-snapshot
Risk Factors Preceding Type 2 Diabetes and Cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s12265-013-9453-4openalex-snapshot
Beneficial Cardiac Effects of Caloric Restriction Are Lost with Age in a Murine Model of Obesity
inferred← citeshttps://doi.org/10.1007/s12265-025-10630-1openalex-snapshot
Exercise Empowerment: A Multifaceted Anatomy in Managing Diabetic Myocardial Disorder
inferred← citeshttps://doi.org/10.1007/s12265-025-10655-6openalex-snapshot
A Molecular Perspective on the Intricate Interplay Among Exosomes, Bioenergetic Metabolism, and the Pathogenesis of Diabetic Cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s12350-011-9362-3openalex-snapshot
Potentiation of abnormalities in myocardial metabolism with the development of diabetes in women with obesity and insulin resistance
inferred← citeshttps://doi.org/10.1007/s13105-018-0653-xopenalex-snapshot
Cardiac mitochondrial respiration following a low-carbohydrate, high-fat diet in apolipoprotein E-deficient mice
inferred← citeshttps://doi.org/10.1007/s13238-012-2043-4openalex-snapshot
Mitochondria in the pathogenesis of diabetes: a proteomic view
inferred← citeshttps://doi.org/10.1016/b978-0-12-800093-9.00012-0openalex-snapshot
Glucose Uptake and Its Consequence on Cardiomyocyte Function
inferred← citeshttps://doi.org/10.1016/b978-0-12-801238-3.99831-3openalex-snapshot
OBSOLETE: Mitochondria Bioenergetics in the Heart
inferred← citeshttps://doi.org/10.1016/b978-0-12-802394-5.00012-1openalex-snapshot
Metabolic Remodeling in the Development of Heart Failure
inferred← citeshttps://doi.org/10.1016/b978-0-12-802733-2.00003-7openalex-snapshot
Role of mTOR Signaling in Cardioprotection
inferred← citeshttps://doi.org/10.1016/b978-0-12-809657-4.99831-4openalex-snapshot
Mitochondrial Bioenergetics in the Heart
inferred← citeshttps://doi.org/10.1016/b978-0-12-811752-1.00011-0openalex-snapshot
Role of Mitochondria in Cardiovascular Comorbidities Associated with Obesity and Type 2 Diabetes
inferred← citeshttps://doi.org/10.1016/b978-0-12-822258-4.00011-0openalex-snapshot
Cardiac transcriptomic remodeling in metabolic syndrome
inferred← citeshttps://doi.org/10.1016/b978-0-12-824412-8.00019-9openalex-snapshot
Nutrigenomics of type 2 diabetes: Gene–diet interactions
inferred← citeshttps://doi.org/10.1016/b978-0-323-60987-6.00008-9openalex-snapshot
Oxidative Stress in Heart Failure
inferred← citeshttps://doi.org/10.1016/b978-0-323-60987-6.00017-xopenalex-snapshot
Alterations in Cardiac Metabolism in Heart Failure
inferred← citeshttps://doi.org/10.1016/b978-0-323-88426-6.00009-9openalex-snapshot
Overview on chronic complications of diabetes mellitus
inferred← citeshttps://doi.org/10.1016/b978-0-443-15734-9.00076-5openalex-snapshot
Bioenergetics in the Heart
inferred← citeshttps://doi.org/10.1016/b978-0-443-18858-9.00022-6openalex-snapshot
Diabetes and mitochondrial transplantation
inferred← citeshttps://doi.org/10.1016/b978-0-444-53480-4.00027-8openalex-snapshot
Mechanisms of disease
inferred← citeshttps://doi.org/10.1016/j.addr.2021.113904openalex-snapshot
Fibrosis of the diabetic heart: Clinical significance, molecular mechanisms, and therapeutic opportunities
inferred← citeshttps://doi.org/10.1016/j.bbabio.2008.04.038openalex-snapshot
Within brown-fat cells, UCP1-mediated fatty acid-induced uncoupling is independent of fatty acid metabolism
inferred← citeshttps://doi.org/10.1016/j.bbadis.2016.07.020openalex-snapshot
Acetylation control of cardiac fatty acid β-oxidation and energy metabolism in obesity, diabetes, and heart failure
inferred← citeshttps://doi.org/10.1016/j.bbadis.2016.08.021openalex-snapshot
Mitochondrial dysfunction and its impact on diabetic heart
inferred← citeshttps://doi.org/10.1016/j.bbadis.2017.10.013openalex-snapshot
MG53 and disordered metabolism in striated muscle
inferred← citeshttps://doi.org/10.1016/j.bbagen.2013.09.019openalex-snapshot
The role of mitochondria in the aetiology of insulin resistance and type 2 diabetes
inferred← citeshttps://doi.org/10.1016/j.bbalip.2009.09.023openalex-snapshot
Lipotoxicity in the heart
inferred← citeshttps://doi.org/10.1016/j.bbalip.2014.07.016openalex-snapshot
Cardiac diastolic dysfunction in high-fat diet fed mice is associated with lipotoxicity without impairment of cardiac energetics in vivo
inferred← citeshttps://doi.org/10.1016/j.bbalip.2014.11.007openalex-snapshot
Intrinsic and extrinsic regulation of cardiac lipoprotein lipase following diabetes
inferred← citeshttps://doi.org/10.1016/j.bbalip.2016.03.016openalex-snapshot
Cardiomyocyte-endothelial cell control of lipoprotein lipase
inferred← citeshttps://doi.org/10.1016/j.bbalip.2016.03.018openalex-snapshot
The role of CD36 in the regulation of myocardial lipid metabolism
inferred← citeshttps://doi.org/10.1016/j.bbalip.2016.03.020openalex-snapshot
Cardiac fatty acid oxidation in heart failure associated with obesity and diabetes
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2011.01.009openalex-snapshot
Nuclear-encoded mitochondrial proteins and their role in cardioprotection
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2011.01.014openalex-snapshot
Mitochondrial dysfunction in diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2011.01.015openalex-snapshot
Targeting fatty acid and carbohydrate oxidation — A novel therapeutic intervention in the ischemic and failing heart
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2011.01.032openalex-snapshot
Caloric excess or restriction mediated modulation of metabolic enzyme acetylation—proposed effects on cardiac growth and function
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2024.119814openalex-snapshot
The role of protein kinase D (PKD) in obesity: Lessons from the heart and other tissues
inferred← citeshttps://doi.org/10.1016/j.bbrc.2013.10.039openalex-snapshot
Ginsenoside Rg3 improves cardiac mitochondrial population quality: Mimetic exercise training
inferred← citeshttps://doi.org/10.1016/j.bbrc.2019.05.084openalex-snapshot
Ablation of TMEM126B protects against heart injury via improving mitochondrial function in high fat diet (HFD)-induced mice
inferred← citeshttps://doi.org/10.1016/j.bcp.2024.116185openalex-snapshot
Transcriptional control of cardiac energy metabolism in health and disease: Lessons from animal models
inferred← citeshttps://doi.org/10.1016/j.biopha.2018.11.086openalex-snapshot
Pathophysiological mechanisms of diabetic cardiomyopathy and the therapeutic potential of epigallocatechin-3-gallate
inferred← citeshttps://doi.org/10.1016/j.cbi.2025.111717openalex-snapshot
Palmitate-induced hepatic insulin resistance as an in vitro model for natural and synthetic drug screening: A scoping review of therapeutic candidates and mechanisms
inferred← citeshttps://doi.org/10.1016/j.cjca.2025.01.029openalex-snapshot
The Impact of Obesity on Cardiac Energy Metabolism and Efficiency in Heart Failure With Preserved Ejection Fraction
inferred← citeshttps://doi.org/10.1016/j.cmet.2007.09.001openalex-snapshot
A Conserved Role for Phosphatidylinositol 3-Kinase but Not Akt Signaling in Mitochondrial Adaptations that Accompany Physiological Cardiac Hypertrophy
inferred← citeshttps://doi.org/10.1016/j.cmet.2010.07.003openalex-snapshot
Cardiolipin Remodeling by ALCAT1 Links Oxidative Stress and Mitochondrial Dysfunction to Obesity
inferred← citeshttps://doi.org/10.1016/j.cmet.2017.04.008openalex-snapshot
DNA-PK Promotes the Mitochondrial, Metabolic, and Physical Decline that Occurs During Aging
inferred← citeshttps://doi.org/10.1016/j.cophys.2022.100559openalex-snapshot
Remodeling of cardiac metabolism in heart failure with preserved ejection fraction
inferred← citeshttps://doi.org/10.1016/j.ddmec.2013.05.004openalex-snapshot
Obesity and cardiac function – the role of caloric excess and its reversal
inferred← citeshttps://doi.org/10.1016/j.ddstr.2008.07.001openalex-snapshot
Cardiovascular consequences of obesity and targets for treatment
inferred← citeshttps://doi.org/10.1016/j.diabres.2013.05.010openalex-snapshot
Left ventricular dysfunction and outcome at two-year follow-up in patients with type 2 diabetes: The DYDA study
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2024.117559openalex-snapshot
Association between glyphosate exposure and cardiovascular health using “Life’s Essential 8” metrics in US adults
inferred← citeshttps://doi.org/10.1016/j.ejcts.2008.02.015openalex-snapshot
Three good reasons for heart surgeons to understand cardiac metabolism☆
inferred← citeshttps://doi.org/10.1016/j.ejphar.2017.10.043openalex-snapshot
Molecular mechanism of doxorubicin-induced cardiomyopathy – An update
inferred← citeshttps://doi.org/10.1016/j.endmts.2020.100076openalex-snapshot
Effects of obesity on the cardiac proteome
inferred← citeshttps://doi.org/10.1016/j.exger.2014.08.008openalex-snapshot
The mitochondrial O-linked N-acetylglucosamine transferase (mOGT) in the diabetic patient could be the initial trigger to develop Alzheimer disease
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2015.11.013openalex-snapshot
Therapeutic inhibition of mitochondrial reactive oxygen species with mito-TEMPO reduces diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2018.08.034openalex-snapshot
Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2018.10.445openalex-snapshot
GCN2 deficiency ameliorates cardiac dysfunction in diabetic mice by reducing lipotoxicity and oxidative stress
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2021.03.046openalex-snapshot
Therapeutic potential of targeting oxidative stress in diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2023.08.009openalex-snapshot
The role of mitochondria in myocardial damage caused by energy metabolism disorders: From mechanisms to therapeutics
inferred← citeshttps://doi.org/10.1016/j.hfc.2012.06.001openalex-snapshot
Diabetes Mellitus and Myocardial Mitochondrial Dysfunction
inferred← citeshttps://doi.org/10.1016/j.hfc.2012.06.008openalex-snapshot
Myocardial Fatty Acid Metabolism and Lipotoxicity in the Setting of Insulin Resistance
inferred← citeshttps://doi.org/10.1016/j.ijcme.2016.10.002openalex-snapshot
Short-term caloric restriction in db/db mice improves myocardial function and increases high molecular weight (HMW) adiponectin
inferred← citeshttps://doi.org/10.1016/j.jacbts.2016.06.009openalex-snapshot
Evidence of Glycolysis Up-Regulation and Pyruvate Mitochondrial Oxidation Mismatch During Mechanical Unloading of the Failing Human Heart
inferred← citeshttps://doi.org/10.1016/j.jacbts.2025.04.009openalex-snapshot
What Is Cardiometabolic HFpEF and How Can We Study it Preclinically?
inferred← citeshttps://doi.org/10.1016/j.jacc.2009.07.031openalex-snapshot
Substrate-Specific Derangements in Mitochondrial Metabolism and Redox Balance in the Atrium of the Type 2 Diabetic Human Heart
inferred← citeshttps://doi.org/10.1016/j.jacc.2010.09.039openalex-snapshot
Obesity Stresses Cardiac Mitochondria Even When You Are Young
inferred← citeshttps://doi.org/10.1016/j.jacc.2017.05.043openalex-snapshot
Oxidative Stress and Cardiovascular Risk: Obesity, Diabetes, Smoking, and Pollution
inferred← citeshttps://doi.org/10.1016/j.jep.2020.113328openalex-snapshot
Protective effects of medicinal plant against diabetes induced cardiac disorder: A review
inferred← citeshttps://doi.org/10.1016/j.jfda.2017.02.003openalex-snapshot
Smog induces oxidative stress and microbiota disruption
inferred← citeshttps://doi.org/10.1016/j.jnutbio.2021.108816openalex-snapshot
Consuming oxidative frying oil impairs cardiac energy production and calcium recycling, causing cardiac hypertrophy, fibrosis and diastolic dysfunction in male Sprague Dawley rats
inferred← citeshttps://doi.org/10.1016/j.jshs.2025.101095openalex-snapshot
Influence of diet-induced obesity and voluntary exercise training on cardiac lipids and mitochondrial function in mice
inferred← citeshttps://doi.org/10.1016/j.lfs.2012.10.028openalex-snapshot
Molecular and metabolic mechanisms of cardiac dysfunction in diabetes
inferred← citeshttps://doi.org/10.1016/j.lfs.2020.117843openalex-snapshot
The role of metabolic diseases in cardiotoxicity associated with cancer therapy: What we know, what we would know
inferred← citeshttps://doi.org/10.1016/j.mad.2016.09.009openalex-snapshot
Interventions for age-related diseases: Shifting the paradigm
inferred← citeshttps://doi.org/10.1016/j.mad.2023.111870openalex-snapshot
The link between obesity and aging - insights into cardiac energy metabolism
inferred← citeshttps://doi.org/10.1016/j.mce.2016.04.004openalex-snapshot
Gene expression profiling in hearts of diabetic mice uncovers a potential role of estrogen-related receptor γ in diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.mce.2019.110692openalex-snapshot
Severe hypoglycemia exacerbates myocardial dysfunction and metabolic remodeling in diabetic mice
inferred← citeshttps://doi.org/10.1016/j.metabol.2009.07.021openalex-snapshot
Superoxide production by mitochondria of insulin-sensitive tissues: mechanistic differences and effect of early diabetes
inferred← citeshttps://doi.org/10.1016/j.metabol.2012.02.013openalex-snapshot
Over-nutrition and metabolic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.mgene.2018.10.004openalex-snapshot
Genetic association study of C5178A and G10398A mitochondrial DNA variants with type 2 diabetes in Bangladeshi population
inferred← citeshttps://doi.org/10.1016/j.mito.2019.03.005openalex-snapshot
Cardiomyocyte mitochondrial dysfunction in diabetes and its contribution in cardiac arrhythmogenesis
inferred← citeshttps://doi.org/10.1016/j.mito.2025.102023openalex-snapshot
Drp1 knockdown aggravates obesity-induced cardiac dysfunction and remodeling
inferred← citeshttps://doi.org/10.1016/j.mmm.2026.07.005openalex-snapshot
Diabète et cœur : comprendre la glucotoxicité cardiaque
inferred← citeshttps://doi.org/10.1016/j.niox.2014.08.002openalex-snapshot
Are nitric oxide-mediated protein modifications of functional significance in diabetic heart? ye'S, -NO’, wh‘Y-NO’t?
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2014.01.003openalex-snapshot
Diabetic cardiomyopathy: Mechanisms and new treatment strategies targeting antioxidant signaling pathways
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2015.03.002openalex-snapshot
Targeting caveolin-3 for the treatment of diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.phrs.2018.09.013openalex-snapshot
Uncoupling proteins as a therapeutic target to protect the diabetic heart
inferred← citeshttps://doi.org/10.1016/j.rccar.2019.12.006openalex-snapshot
De la diabetes a la insuficiencia cardiaca ¿Existe la miocardiopatía diabética?
inferred← citeshttps://doi.org/10.1016/j.rec.2011.05.022openalex-snapshot
Mitochondrial Dynamics: a Potential New Therapeutic Target for Heart Failure
inferred← citeshttps://doi.org/10.1016/j.recesp.2011.05.018openalex-snapshot
Dinámica mitocondrial: un potencial nuevo blanco terapéutico para la insuficiencia cardiaca
inferred← citeshttps://doi.org/10.1016/j.redox.2018.07.007openalex-snapshot
Melatonin improves cardiac function in a mouse model of heart failure with preserved ejection fraction
inferred← citeshttps://doi.org/10.1016/j.redox.2022.102320openalex-snapshot
Increased fatty acid metabolism attenuates cardiac resistance to β-adrenoceptor activation via mitochondrial reactive oxygen species: A potential mechanism of hypoglycemia-induced myocardial injury in diabetes
inferred← citeshttps://doi.org/10.1016/j.redox.2022.102485openalex-snapshot
Isoginkgetin, a bioactive constituent from Ginkgo Biloba, protects against obesity-induced cardiomyopathy via enhancing Nrf2/ARE signaling
inferred← citeshttps://doi.org/10.1016/j.taap.2019.03.005openalex-snapshot
Blockage of ROS and MAPKs-mediated inflammation via restoring SIRT1 by a new compound LF10 prevents type 1 diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.tcm.2009.12.005openalex-snapshot
Metabolic Therapy at the Crossroad: How to Optimize Myocardial Substrate Utilization?
inferred← citeshttps://doi.org/10.1016/j.tem.2011.05.001openalex-snapshot
New insights into insulin resistance in the diabetic heart
inferred← citeshttps://doi.org/10.1016/j.tem.2019.05.005openalex-snapshot
Reactive Carbonyl Species: Diabetic Complication in the Heart and Lungs
inferred← citeshttps://doi.org/10.1016/j.tice.2022.101763openalex-snapshot
The melatonin MT2 receptor is involved in the anti-apoptotic effects of melatonin in rats with type 2 diabetes mellitus
inferred← citeshttps://doi.org/10.1016/j.tiv.2020.104948openalex-snapshot
Palmitate-induced toxicity is associated with impaired mitochondrial respiration and accelerated oxidative stress in cultured cardiomyocytes: The critical role of coenzyme Q9/10
inferred← citeshttps://doi.org/10.1016/j.tiv.2020.105048openalex-snapshot
Inorganic nitrite alters mitochondrial dynamics without overt changes in cell death and mitochondrial respiration in cardiomyoblasts under hyperglycemia
inferred← citeshttps://doi.org/10.1016/j.toxrep.2019.11.004openalex-snapshot
N-Acetyl cysteine ameliorates hyperglycemia-induced cardiomyocyte toxicity by improving mitochondrial energetics and enhancing endogenous Coenzyme Q9/10 levels
inferred← citeshttps://doi.org/10.1016/j.trsl.2014.05.001openalex-snapshot
Obesity, metabolic dysfunction, and cardiac fibrosis: pathophysiological pathways, molecular mechanisms, and therapeutic opportunities
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2009.02.001openalex-snapshot
Type 2 diabetes, mitochondrial biology and the heart
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2009.02.014openalex-snapshot
Impaired insulin signaling accelerates cardiac mitochondrial dysfunction after myocardial infarction
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2009.02.019openalex-snapshot
Prolonged exposure to high dietary lipids is not associated with lipotoxicity in heart failure
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2011.01.007openalex-snapshot
Metabolism, hypoxia and the diabetic heart
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2011.10.010openalex-snapshot
The transcriptional coactivators, PGC-1α and β, cooperate to maintain cardiac mitochondrial function during the early stages of insulin resistance
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2012.02.001openalex-snapshot
Genetic loss of insulin receptors worsens cardiac efficiency in diabetes
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2012.02.009openalex-snapshot
Mitochondrial permeability transition in the diabetic heart: Contributions of thiol redox state and mitochondrial calcium to augmented reperfusion injury
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2012.03.016openalex-snapshot
Restoration of glucose metabolism in leptin-resistant mouse hearts after acute myocardial infarction through the activation of survival kinase pathways
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2013.03.007openalex-snapshot
Diet-induced obesity causes long QT and reduces transcription of voltage-gated potassium channels
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2014.02.011openalex-snapshot
GLUT1 deficiency in cardiomyocytes does not accelerate the transition from compensated hypertrophy to heart failure
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2014.03.011openalex-snapshot
Mitochondrial reactive oxygen species production and elimination
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2014.07.018openalex-snapshot
High fat, high sucrose diet causes cardiac mitochondrial dysfunction due in part to oxidative post-translational modification of mitochondrial complex II
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2014.10.015openalex-snapshot
Impaired cytosolic NADH shuttling and elevated UCP3 contribute to inefficient citric acid cycle flux support of postischemic cardiac work in diabetic hearts
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2015.05.012openalex-snapshot
Antioxidant treatment normalizes mitochondrial energetics and myocardial insulin sensitivity independently of changes in systemic metabolic homeostasis in a mouse model of the metabolic syndrome
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2015.10.015openalex-snapshot
Phosphoinositide dependent protein kinase 1 is required for exercise-induced cardiac hypertrophy but not the associated mitochondrial adaptations
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2015.12.011openalex-snapshot
Diabetes-associated cardiac fibrosis: Cellular effectors, molecular mechanisms and therapeutic opportunities
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2016.09.001openalex-snapshot
Preservation of myocardial fatty acid oxidation prevents diastolic dysfunction in mice subjected to angiotensin II infusion
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2018.01.017openalex-snapshot
Decreased ATP production and myocardial contractile reserve in metabolic heart disease
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2019.06.001openalex-snapshot
High-fat diet improves tolerance to myocardial ischemia by delaying normalization of intracellular PH at reperfusion
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2021.01.002openalex-snapshot
Xanthine oxidoreductase-mediated injury is amplified by upregulated AMP deaminase in type 2 diabetic rat hearts under the condition of pressure overload
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2022.09.002openalex-snapshot
Diabetes disturbs functional adaptation of the remote myocardium after ischemia/reperfusion
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2025.02.010openalex-snapshot
Lipotoxicity as a therapeutic target in the type 2 diabetic heart
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2026.07.001openalex-snapshot
From antioxidant hype to mitochondrial reality: Quercetin modulates cardiac redox balance but fails to restore mitochondrial respiration parameters in Zucker diabetic fatty rats
inferred← citeshttps://doi.org/10.1016/j.ymgmr.2021.100753openalex-snapshot
Molecular basis of ventricular arrhythmogenicity in a Pgc-1α deficient murine model
inferred← citeshttps://doi.org/10.1016/s1262-3636(08)70096-xopenalex-snapshot
Diabetes-related metabolic perturbations in cardiac myocyte
inferred← citeshttps://doi.org/10.1021/acs.jproteome.5b00128openalex-snapshot
Comprehensive Metabolic Profiling of Age-Related Mitochondrial Dysfunction in the High-Fat-Fed ob/ob Mouse Heart
showing the first 200 of 567 · page through all 567
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.