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Receptor for Advanced Glycation End Products (RAGE) Deficiency Attenuates the Development of Atherosclerosis in Diabetes
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/s00109-005-0688-7openalex-snapshot
Understanding RAGE, the receptor for advanced glycation end products
inferredcites →https://doi.org/10.1016/j.biocel.2007.03.012openalex-snapshot
CD36: Implications in cardiovascular disease
inferredcites →https://doi.org/10.1016/s0002-9440(10)63909-0openalex-snapshot
RAGE Drives the Development of Glomerulosclerosis and Implicates Podocyte Activation in the Pathogenesis of Diabetic Nephropathy
inferredcites →https://doi.org/10.1038/2012openalex-snapshot
Suppression of accelerated diabetic atherosclerosis by the soluble receptor for advanced glycation endproducts
inferredcites →https://doi.org/10.1042/bj20021371openalex-snapshot
Novel splice variants of the receptor for advanced glycation end-products expressed in human vascular endothelial cells and pericytes, and their putative roles in diabetes-induced vascular injury
inferredcites →https://doi.org/10.1074/jbc.272.26.16498openalex-snapshot
Characterization and Functional Analysis of the Promoter of RAGE, the Receptor for Advanced Glycation End Products
inferredcites →https://doi.org/10.1093/clinchem/40.7.1317openalex-snapshot
Glycohemoglobin measured by automated affinity HPLC correlates with both short-term and long-term antecedent glycemia
inferredcites →https://doi.org/10.1111/j.1600-065x.2007.00515.xopenalex-snapshot
Mast cells: multipotent local effector cells in atherothrombosis
inferredcites →https://doi.org/10.1161/01.atv.0000148007.06370.68openalex-snapshot
Stem Cell Transplantation Reveals That Absence of Macrophage CD36 Is Protective Against Atherosclerosis
inferredcites →https://doi.org/10.1161/01.atv.0000177813.99577.6bopenalex-snapshot
Rosiglitazone Attenuates Atherosclerosis in a Model of Insulin Insufficiency Independent of Its Metabolic Effects
inferredcites →https://doi.org/10.1161/01.cir.0000021122.63813.32openalex-snapshot
Prevention of Accelerated Atherosclerosis by Angiotensin-Converting Enzyme Inhibition in Diabetic Apolipoprotein E–Deficient Mice
inferredcites →https://doi.org/10.1161/01.cir.0000039325.03698.36openalex-snapshot
RAGE Blockade Stabilizes Established Atherosclerosis in Diabetic Apolipoprotein E–Null Mice
inferredcites →https://doi.org/10.1161/01.cir.0000124061.78478.94openalex-snapshot
Irbesartan but Not Amlodipine Suppresses Diabetes-Associated Atherosclerosis
inferredcites →https://doi.org/10.1161/01.res.74.6.1141openalex-snapshot
Angiotensin II stimulates NADH and NADPH oxidase activity in cultured vascular smooth muscle cells.
inferredcites →https://doi.org/10.1161/circulationaha.106.621854openalex-snapshot
Advanced Glycation End Products
inferredcites →https://doi.org/10.1161/circulationaha.106.664250openalex-snapshot
Lack of the Antioxidant Enzyme Glutathione Peroxidase-1 Accelerates Atherosclerosis in Diabetic Apolipoprotein E–Deficient Mice
inferredcites →https://doi.org/10.1172/jci11771openalex-snapshot
Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice
inferredcites →https://doi.org/10.1172/jci17115openalex-snapshot
Central role of RAGE-dependent neointimal expansion in arterial restenosis
inferredcites →https://doi.org/10.1172/jci18058openalex-snapshot
Loss of pain perception in diabetes is dependent on a receptor of the immunoglobulin superfamily
inferredcites →https://doi.org/10.1172/jci18704openalex-snapshot
Receptor for advanced glycation end products (RAGE) regulates sepsis but not the adaptive immune response
inferredcites →https://doi.org/10.1172/jci32703openalex-snapshot
Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE–/– mice
inferredcites →https://doi.org/10.1681/asn.2005010013openalex-snapshot
Interactions between Renin Angiotensin System and Advanced Glycation in the Kidney
inferredcites →https://doi.org/10.2337/db05-1375openalex-snapshot
Receptor for Advanced Glycation End Products Is Involved in Impaired Angiogenic Response in Diabetes
inferredcites →https://doi.org/10.2337/db06-0221openalex-snapshot
RAGE Control of Diabetic Nephropathy in a Mouse Model
inferredcites →https://doi.org/10.2337/diab.43.5.676openalex-snapshot
Glycation, Glycoxidation, and Cross-Linking of Collagen by Glucose: Kinetics, Mechanisms, and Inhibition of Late Stages of the Maillard Reaction
inferredcites →https://doi.org/10.2337/diabetes.50.12.2792openalex-snapshot
Diabetes-Associated Sustained Activation of the Transcription Factor Nuclear Factor-κB
inferredcites →https://doi.org/10.2337/diabetes.53.7.1813openalex-snapshot
Advanced Glycation End Product Interventions Reduce Diabetes-Accelerated Atherosclerosis
inferredcites →https://doi.org/10.2353/ajpath.2006.050713openalex-snapshot
Posttranslationally Modified Proteins as Mediators of Sustained Intestinal Inflammation
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4638793openalex-extracted
Advanced Glycation Endproducts Induce Overexpression of Receptor of Advanced Glycation Endproducts/Calmodulin-Dependent Protein Kinase 4 and Secrete Interferon-y in Human CD4 Lymphocytes
inferred← citeshttps://doi.org/10.1002/9781118148143.ch11openalex-snapshot
Oxidative Stress in Cardiovascular Diseases
inferred← citeshttps://doi.org/10.1002/9781118828533.ch3openalex-snapshot
Glycation, Inflammation and RAGE
inferred← citeshttps://doi.org/10.1002/9781118924853.ch41openalex-snapshot
Pathogenesis of Macrovascular Complications in Diabetes
inferred← citeshttps://doi.org/10.1002/9781119697473.ch46openalex-snapshot
Pathogenesis of Macrovascular Complications in Diabetes
inferred← citeshttps://doi.org/10.1002/9781444324808.ch35openalex-snapshot
Pathogenesis of Microvascular Complications
inferred← citeshttps://doi.org/10.1002/9781444324808.ch39openalex-snapshot
The Pathogenesis of Macrovascular Complications Including Atherosclerosis in Diabetes
inferred← citeshttps://doi.org/10.1002/biof.1018openalex-snapshot
An update on advanced glycation endproducts and atherosclerosis
inferred← citeshttps://doi.org/10.1002/cpt.148openalex-snapshot
Emerging Targets for Therapeutic Development in Diabetes and Its Complications: The RAGE Signaling Pathway
inferred← citeshttps://doi.org/10.1002/dmrr.1076openalex-snapshot
Resveratrol ameliorates vasculopathy in STZ‐induced diabetic rats: role of AGE–RAGE signalling
inferred← citeshttps://doi.org/10.1002/jcb.27524openalex-snapshot
Retracted: Advanced glycation end‐products induce oxidative stress through the Sirt1/Nrf2 axis by interacting with the receptor of AGEs under diabetic conditions
inferred← citeshttps://doi.org/10.1002/jcp.22492openalex-snapshot
Involvement of membrane type 1‐matrix metalloproteinase (MT1‐MMP) in RAGE activation signaling pathways
inferred← citeshttps://doi.org/10.1002/jcp.24860openalex-snapshot
In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1‐Mediated Cholesterol Efflux
inferred← citeshttps://doi.org/10.1002/path.4045openalex-snapshot
Advanced glycation end product Nε‐carboxymethyllysine induces endothelial cell injury: the involvement of SHP‐1‐regulated VEGFR‐2 dephosphorylation
inferred← citeshttps://doi.org/10.1002/prca.201400052openalex-snapshot
The role of post‐translational modifications in acute and chronic cardiovascular disease
inferred← citeshttps://doi.org/10.1007/164_2020_391openalex-snapshot
Inhibitors of Advanced Glycation End Product (AGE) Formation and Accumulation
inferred← citeshttps://doi.org/10.1007/978-1-4899-8035-9_11openalex-snapshot
Oxidative Stress and Cardiovascular Disease in Diabetes
inferred← citeshttps://doi.org/10.1007/978-1-60761-857-7_13openalex-snapshot
Cardiovascular Complications in Renal Failure: Implications of Advanced Glycation End Products and Their Receptor RAGE
inferred← citeshttps://doi.org/10.1007/978-1-60761-956-7_1openalex-snapshot
Role of Oxidative Stress and Targeted Antioxidant Therapies in Experimental Models of Diabetic Complications
inferred← citeshttps://doi.org/10.1007/978-1-61779-606-7_3openalex-snapshot
Oxidative Modifications as Triggers of AMD Pathology
inferred← citeshttps://doi.org/10.1007/978-3-0346-0168-9_9openalex-snapshot
AGE-RAGE signalling in endothelial dysfunction and atherosclerosis in diabetes
inferred← citeshttps://doi.org/10.1007/978-3-642-30018-9_155openalex-snapshot
Reactive Oxygen Species and Diabetes-Associated Atherosclerosis – Evidence from Experimental Models and Targeted Antioxidant Therapy
inferred← citeshttps://doi.org/10.1007/978-3-642-30018-9_186openalex-snapshot
Reactive Oxygen Species (ROS) and Diabetic Nephropathy
inferred← citeshttps://doi.org/10.1007/978-3-642-30018-9_22openalex-snapshot
Vascular Reactive Oxygen Species Biology – Insights from Transgenic and Knockout Mouse Models
inferred← citeshttps://doi.org/10.1007/978-4-431-55720-3_5openalex-snapshot
Mechanism for the Development of Bone Disease in Diabetes: Increased Oxidative Stress and Advanced Glycation End Products
inferred← citeshttps://doi.org/10.1007/978-981-10-4376-5_16openalex-snapshot
Diabetes and Advanced Glycation End Products
inferred← citeshttps://doi.org/10.1007/s00011-014-0735-3openalex-snapshot
Expression of leukocyte adhesion-related glycosyltransferase genes in acute coronary syndrome patients
inferred← citeshttps://doi.org/10.1007/s00109-009-0439-2openalex-snapshot
Receptor for AGE (RAGE) and its ligands—cast into leading roles in diabetes and the inflammatory response
inferred← citeshttps://doi.org/10.1007/s00109-014-1146-1openalex-snapshot
The pathobiology of diabetic vascular complications—cardiovascular and kidney disease
inferred← citeshttps://doi.org/10.1007/s00125-009-1378-8openalex-snapshot
Apolipoprotein E deficiency abrogates insulin resistance in a mouse model of type 2 diabetes mellitus
inferred← citeshttps://doi.org/10.1007/s00125-009-1458-9openalex-snapshot
Multiple levels of regulation determine the role of the receptor for AGE (RAGE) as common soil in inflammation, immune responses and diabetes mellitus and its complications
inferred← citeshttps://doi.org/10.1007/s00125-009-1540-3openalex-snapshot
The endothelin receptor antagonist avosentan ameliorates nephropathy and atherosclerosis in diabetic apolipoprotein E knockout mice
inferred← citeshttps://doi.org/10.1007/s00125-009-1555-9openalex-snapshot
Cell division autoantigen 1 plays a profibrotic role by modulating downstream signalling of TGF-β in a murine diabetic model of atherosclerosis
inferred← citeshttps://doi.org/10.1007/s00125-009-1619-xopenalex-snapshot
Angiotensin II subtype 2 receptor blockade and deficiency attenuate the development of atherosclerosis in an apolipoprotein E-deficient mouse model of diabetes
inferred← citeshttps://doi.org/10.1007/s00125-010-2000-9openalex-snapshot
Delayed intervention with AGE inhibitors attenuates the progression of diabetes-accelerated atherosclerosis in diabetic apolipoprotein E knockout mice
inferred← citeshttps://doi.org/10.1007/s00125-011-2186-5openalex-snapshot
Soluble receptor for AGE (RAGE) is a novel independent predictor of all-cause and cardiovascular mortality in type 1 diabetes
inferred← citeshttps://doi.org/10.1007/s00125-013-2837-9openalex-snapshot
Urotensin II receptor antagonism confers vasoprotective effects in diabetes associated atherosclerosis: studies in humans and in a mouse model of diabetes
inferred← citeshttps://doi.org/10.1007/s00125-013-3118-3openalex-snapshot
Pharmacological inhibition of NOX reduces atherosclerotic lesions, vascular ROS and immune–inflammatory responses in diabetic Apoe −/− mice
inferred← citeshttps://doi.org/10.1007/s00198-013-2511-6openalex-snapshot
Neuropathy and the vascular-bone axis in diabetes: lessons from Charcot osteoarthropathy
inferred← citeshttps://doi.org/10.1007/s00223-017-0243-xopenalex-snapshot
Advanced Glycation End Product 3 (AGE3) Increases Apoptosis and the Expression of Sclerostin by Stimulating TGF-β Expression and Secretion in Osteocyte-Like MLO-Y4-A2 Cells
inferred← citeshttps://doi.org/10.1007/s00281-009-0144-9openalex-snapshot
Advanced glycation end products and C-peptide—modulators in diabetic vasculopathy and atherogenesis
inferred← citeshttps://doi.org/10.1007/s00391-011-0279-xopenalex-snapshot
RAGE influences obesity in mice
inferred← citeshttps://doi.org/10.1007/s00395-012-0306-0openalex-snapshot
Pioglitazone attenuates progression of aortic valve calcification via down-regulating receptor for advanced glycation end products
inferred← citeshttps://doi.org/10.1007/s00430-009-0130-9openalex-snapshot
Possible hidden hazards of mass vaccination against new influenza A/H1N1: have the cardiovascular risks been adequately weighed?
inferred← citeshttps://doi.org/10.1007/s00726-010-0773-2openalex-snapshot
Advanced glycation endproducts: from precursors to RAGE: round and round we go
inferred← citeshttps://doi.org/10.1007/s00726-010-0779-9openalex-snapshot
Early- and advanced non-enzymatic glycation in diabetic vascular complications: the search for therapeutics
inferred← citeshttps://doi.org/10.1007/s00774-016-0800-9openalex-snapshot
Impaired osteogenic differentiation and enhanced cellular receptor of advanced glycation end products sensitivity in patients with type 2 diabetes
inferred← citeshttps://doi.org/10.1007/s10616-025-00885-6openalex-snapshot
KLF2 inhibits keloid progression by targeting PI3K/AKT-mediated MMP-2/9 signaling
inferred← citeshttps://doi.org/10.1007/s10719-016-9682-2openalex-snapshot
DNA-aptamers raised against AGEs as a blocker of various aging-related disorders
inferred← citeshttps://doi.org/10.1007/s10753-013-9704-1openalex-snapshot
High Mobility Group Box 1 (HMGB1) Mediates High-Glucose-Induced Calcification in Vascular Smooth Muscle Cells of Saphenous Veins
inferred← citeshttps://doi.org/10.1007/s11010-016-2763-5openalex-snapshot
Soluble receptor for advanced glycation end products mitigates vascular dysfunction in spontaneously hypertensive rats
inferred← citeshttps://doi.org/10.1007/s11033-020-06130-xopenalex-snapshot
Expatiating the molecular approaches of HMGB1 in diabetes mellitus: Highlighting signalling pathways via RAGE and TLRs
inferred← citeshttps://doi.org/10.1007/s11154-021-09680-yopenalex-snapshot
Diabetes complications and extracellular vesicle therapy
inferred← citeshttps://doi.org/10.1007/s11325-020-02029-wopenalex-snapshot
TLRs and RAGE are elevated in carotid plaques from patients with moderate-to-severe obstructive sleep apnea syndrome
inferred← citeshttps://doi.org/10.1007/s11596-010-0349-0openalex-snapshot
A possible mechanism linking hyperglycemia and reduced high-density lipoprotein cholesterol levels in diabetes
inferred← citeshttps://doi.org/10.1007/s11892-009-0005-xopenalex-snapshot
Lipids versus glucose in inflammation and the pathogenesis of macrovascular disease in diabetes
inferred← citeshttps://doi.org/10.1007/s11897-012-0089-5openalex-snapshot
Receptor for Advanced Glycation End Products (RAGE) and Implications for the Pathophysiology of Heart Failure
inferred← citeshttps://doi.org/10.1007/s12015-024-10826-zopenalex-snapshot
Erroneous Differentiation of Tendon Stem/Progenitor Cells in the Pathogenesis of Tendinopathy: Current Evidence and Future Perspectives
inferred← citeshttps://doi.org/10.1007/s12272-012-0513-0openalex-snapshot
RETRACTED ARTICLE: Attenuation of aortic injury by ursolic acid through RAGE-Nox-NFκB pathway in streptozocin-induced diabetic rats
inferred← citeshttps://doi.org/10.1007/s12272-020-01262-xopenalex-snapshot
Retraction Note to: Attenuation of aortic injury by ursolic acid through RAGE-Nox-NFκB pathway in streptozocin-induced diabetic rats
inferred← citeshttps://doi.org/10.1007/s13340-013-0148-5openalex-snapshot
Identifying and interpreting novel targets that address more than one diabetic complication: a strategy for optimal end organ protection in diabetes
inferred← citeshttps://doi.org/10.1007/s40200-018-0378-yopenalex-snapshot
Life-style-induced metabolic derangement and epigenetic changes promote diabetes and oxidative stress leading to NASH and atherosclerosis severity
inferred← citeshttps://doi.org/10.1007/s40200-026-01991-8openalex-snapshot
Investigation of advanced glycosylation end products, NF-κB activation, Nrf-2 rs35652124, rs6721961 regions on endoplasmic reticulum stress in peripheral artery disease
inferred← citeshttps://doi.org/10.1007/s40495-023-00328-wopenalex-snapshot
Neutrophil-Derived S100A8/A9 in Cardiovascular Disease and Beyond
inferred← citeshttps://doi.org/10.1007/s43188-021-00114-0openalex-snapshot
Fimbristylis ovata and Artemisia vulgaris extracts inhibited AGE-mediated RAGE expression, ROS generation, and inflammation in THP-1 cells
inferred← citeshttps://doi.org/10.1016/b978-0-323-29738-7.00033-2openalex-snapshot
Complications of Diabetes Mellitus
inferred← citeshttps://doi.org/10.1016/b978-0-323-87862-3.00007-0openalex-snapshot
Pathology and Vascular Biology of Atherosclerosis in Patients With Type 2 Diabetes
inferred← citeshttps://doi.org/10.1016/b978-1-4377-0324-5.00033-xopenalex-snapshot
Complications of Diabetes Mellitus
inferred← citeshttps://doi.org/10.1016/b978-1-4377-1366-4.00008-1openalex-snapshot
Molecular Biology and Genetics of Atherosclerosis
inferred← citeshttps://doi.org/10.1016/bs.acc.2020.02.006openalex-snapshot
S100 family proteins in inflammation and beyond
inferred← citeshttps://doi.org/10.1016/j.arr.2025.102919openalex-snapshot
A receptor for glycation end products (RAGE) is a key transmitter between garb-aging and inflammaging
inferred← citeshttps://doi.org/10.1016/j.asjsur.2021.12.064openalex-snapshot
Change of cardiovascular risk associated serologic biomarkers after gastric bypass: A comparison of diabetic and non-diabetic Asian patients
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2008.12.016openalex-snapshot
Increased serum HMGB1 level is associated with coronary artery disease in nondiabetic and type 2 diabetic patients
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2010.04.006openalex-snapshot
Receptor for advanced glycation end-products (RAGE) regulation of adiposity and adiponectin is associated with atherogenesis in apoE-deficient mouse
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2011.05.023openalex-snapshot
Association of increased S100B, S100A6 and S100P in serum levels with acute coronary syndrome and also with the severity of myocardial infarction in cardiac tissue of rat models with ischemia–reperfusion injury
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2012.01.019openalex-snapshot
Advanced glycation end-product Nɛ-carboxymethyl-Lysine accelerates progression of atherosclerotic calcification in diabetes
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2012.07.020openalex-snapshot
Inhibition of neointimal hyperplasia in the rat carotid artery injury model by a HMGB1 inhibitor
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2014.03.025openalex-snapshot
Comparison of effects of pioglitazone and glimepiride on plasma soluble RAGE and RAGE expression in peripheral mononuclear cells in type 2 diabetes: Randomized controlled trial (PioRAGE)
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2014.05.945openalex-snapshot
Quinapril treatment abolishes diabetes-associated atherosclerosis in RAGE/apolipoprotein E double knockout mice
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2016.06.007openalex-snapshot
Lipidomic approach provides new clues toward solving the mystery of accelerated atherosclerosis in diabetes
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2023.117304openalex-snapshot
RAGE/DIAPH1 and atherosclerosis through an evolving lens: Viewing the cell from the “Inside – Out”
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2024.117609openalex-snapshot
Skin autofluorescence, a measure for accumulation of advanced glycation end products, positively associates with blood neutrophil and monocyte counts in the general population, and particularly in men with prediabetes
inferred← citeshttps://doi.org/10.1016/j.bbadis.2016.05.005openalex-snapshot
Cellular mechanisms and consequences of glycation in atherosclerosis and obesity
inferred← citeshttps://doi.org/10.1016/j.bbagen.2011.03.014openalex-snapshot
Role of advanced glycation end products (AGEs) and oxidative stress in vascular complications in diabetes
inferred← citeshttps://doi.org/10.1016/j.bbagen.2016.11.040openalex-snapshot
The shedding-derived soluble receptor for advanced glycation endproducts sustains inflammation during acute Pseudomonas aeruginosa lung infection
inferred← citeshttps://doi.org/10.1016/j.bbrc.2019.10.037openalex-snapshot
Inhibition of core fucosylation limits progression of diabetic kidney disease
inferred← citeshttps://doi.org/10.1016/j.bbrc.2022.07.031openalex-snapshot
A novel mouse model of diabetes, atherosclerosis and fatty liver disease using an AAV8-PCSK9-D377Y injection and dietary manipulation in db/db mice
inferred← citeshttps://doi.org/10.1016/j.bcp.2010.01.013openalex-snapshot
Soluble RAGE: Therapy and biomarker in unraveling the RAGE axis in chronic disease and aging
inferred← citeshttps://doi.org/10.1016/j.bioactmat.2026.05.033openalex-snapshot
Development of a vascularized multi-organoid-on-a-chip to model the heart-islet axis in diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.biocel.2016.06.016openalex-snapshot
Advanced glycation end products and their receptor in age-related, non-communicable chronic inflammatory diseases; Overview of clinical evidence and potential contributions to disease
inferred← citeshttps://doi.org/10.1016/j.biopha.2025.118088openalex-snapshot
Cryo-EM structure of HMGB1-RAGE complex and its inhibitory effect on lung cancer
inferred← citeshttps://doi.org/10.1016/j.carpath.2012.06.007openalex-snapshot
Diabetes mellitus accelerates cartilaginous metaplasia and calcification in atherosclerotic vessels of LDLr mutant mice
inferred← citeshttps://doi.org/10.1016/j.carpath.2013.03.006openalex-snapshot
Insulin-degrading enzyme deficiency in bone marrow cells increases atherosclerosis in LDL receptor-deficient mice
inferred← citeshttps://doi.org/10.1016/j.cbi.2016.03.015openalex-snapshot
Chrysin, a PPAR-γ agonist improves myocardial injury in diabetic rats through inhibiting AGE-RAGE mediated oxidative stress and inflammation
inferred← citeshttps://doi.org/10.1016/j.cellimm.2021.104379openalex-snapshot
The receptor for advanced glycation endproducts (RAGE) decreases survival of tumor-bearing mice by enhancing the generation of lung metastasis-associated myeloid-derived suppressor cells
inferred← citeshttps://doi.org/10.1016/j.cellsig.2013.06.013openalex-snapshot
Cellular signalling of the receptor for advanced glycation end products (RAGE)
inferred← citeshttps://doi.org/10.1016/j.clinbiochem.2014.12.022openalex-snapshot
Dynamic changes in sRAGE levels and relationship with cardiac function in STEMI patients
inferred← citeshttps://doi.org/10.1016/j.cmet.2011.07.015openalex-snapshot
Insulin Resistance, Hyperglycemia, and Atherosclerosis
inferred← citeshttps://doi.org/10.1016/j.cmet.2013.04.001openalex-snapshot
Hyperglycemia Promotes Myelopoiesis and Impairs the Resolution of Atherosclerosis
inferred← citeshttps://doi.org/10.1016/j.coph.2013.06.009openalex-snapshot
Targeting advanced glycation endproducts and mitochondrial dysfunction in cardiovascular disease
inferred← citeshttps://doi.org/10.1016/j.coph.2024.102464openalex-snapshot
Effects of protein glycation and protective mechanisms against glycative stress
inferred← citeshttps://doi.org/10.1016/j.diabet.2015.01.006openalex-snapshot
Linking RAGE and Nox in diabetic micro- and macrovascular complications
inferred← citeshttps://doi.org/10.1016/j.eclnm.2010.02.001openalex-snapshot
Supplemental dietary arginine reduces renal RAGE expression and oxidative damage in rats with streptozotocin-induced type 2 diabetes
inferred← citeshttps://doi.org/10.1016/j.ekir.2018.04.002openalex-snapshot
Summary of the 2018 ISN Frontiers Meeting: Kidney Disease and Cardiovascular Disease
inferred← citeshttps://doi.org/10.1016/j.exger.2010.11.007openalex-snapshot
Role of advanced glycation end products (AGEs) and receptor for AGEs (RAGE) in vascular damage in diabetes
inferred← citeshttps://doi.org/10.1016/j.fct.2013.06.052openalex-snapshot
Advanced glycation endproducts in food and their effects on health
inferred← citeshttps://doi.org/10.1016/j.fct.2014.04.033openalex-snapshot
Purple sweet potato color ameliorates kidney damage via inhibiting oxidative stress mediated NLRP3 inflammasome activation in high fat diet mice
inferred← citeshttps://doi.org/10.1016/j.ijcard.2013.05.013openalex-snapshot
Receptor for advanced glycation end products (RAGE) in vascular and inflammatory diseases
inferred← citeshttps://doi.org/10.1016/j.ijcard.2013.12.143openalex-snapshot
DNA aptamer raised against advanced glycation end products inhibits neointimal hyperplasia in balloon-injured rat carotid arteries
inferred← citeshttps://doi.org/10.1016/j.ijcard.2016.06.137openalex-snapshot
Atrial tissue expression of receptor for advanced glycation end-products (RAGE) and atrial fibrosis in patients with mitral valve disease
inferred← citeshttps://doi.org/10.1016/j.ijcme.2014.07.002openalex-snapshot
Effect of statins on the serum soluble form of receptor for advanced glycation end-products and its association with coronary atherosclerosis in patients with angina pectoris
inferred← citeshttps://doi.org/10.1016/j.ijcme.2014.07.006openalex-snapshot
Vascular injury is improved by pre-meal glulisine-based bolus insulin therapy in type 2 diabetic patients
inferred← citeshttps://doi.org/10.1016/j.intimp.2025.114316openalex-snapshot
RAGE deficiency obstructs high uric acid-induced oxidative stress and inflammatory response
inferred← citeshttps://doi.org/10.1016/j.jcf.2015.12.011openalex-snapshot
Advanced glycation end products are elevated in cystic fibrosis-related diabetes and correlate with worse lung function
inferred← citeshttps://doi.org/10.1016/j.jcjd.2013.06.001openalex-snapshot
Molecular Mechanisms Linking Diabetes to the Accelerated Development of Atherosclerosis
inferred← citeshttps://doi.org/10.1016/j.jconrel.2021.09.012openalex-snapshot
P-selectin targeted RAGE-shRNA lipoplexes alleviate atherosclerosis-associated inflammation
inferred← citeshttps://doi.org/10.1016/j.jdiacomp.2016.07.001openalex-snapshot
Glycated albumin induces lipid infiltration in mice aorta independently of DM and RAS local modulation by inducing lipid peroxidation and inflammation
inferred← citeshttps://doi.org/10.1016/j.jdiacomp.2017.09.012openalex-snapshot
AGE-albumin enhances ABCA1 degradation by ubiquitin-proteasome and lysosomal pathways in macrophages
inferred← citeshttps://doi.org/10.1016/j.jdiacomp.2020.107613openalex-snapshot
Molecular complexities underlying the vascular complications of diabetes mellitus – A comprehensive review
inferred← citeshttps://doi.org/10.1016/j.jff.2016.12.007openalex-snapshot
Punica granatum L. mesocarp suppresses advanced glycation end products (AGEs)- and H 2 O 2 -induced oxidative stress and pro-inflammatory biomarkers
inferred← citeshttps://doi.org/10.1016/j.jss.2011.06.069openalex-snapshot
Role of High Mobility Group Box 1 (HMGB1) in Wound Healing
inferred← citeshttps://doi.org/10.1016/j.lfs.2020.117344openalex-snapshot
An update on the potential role of advanced glycation end products in glycolipid metabolism
inferred← citeshttps://doi.org/10.1016/j.lfs.2022.120860openalex-snapshot
Aftermath of AGE-RAGE Cascade in the pathophysiology of cardiovascular ailments
inferred← citeshttps://doi.org/10.1016/j.lfs.2023.121713openalex-snapshot
RAGE pathways play an important role in regulation of organ fibrosis
inferred← citeshttps://doi.org/10.1016/j.mad.2010.09.005openalex-snapshot
Advanced glycation end products induce T cell apoptosis: Involvement of oxidative stress, caspase and the mitochondrial pathway
inferred← citeshttps://doi.org/10.1016/j.mad.2015.01.001openalex-snapshot
Age and genetic determinants of variation of circulating levels of the receptor for advanced glycation end products (RAGE) in the general human population
inferred← citeshttps://doi.org/10.1016/j.mam.2014.05.001openalex-snapshot
HMGB1 in health and disease
inferred← citeshttps://doi.org/10.1016/j.mce.2012.02.010openalex-snapshot
A meta-analysis of receptor for advanced glycation end products gene: Four well-evaluated polymorphisms with diabetes mellitus
inferred← citeshttps://doi.org/10.1016/j.metabol.2009.05.024openalex-snapshot
Factors associated with serum high mobility group box 1 (HMGB1) levels in a general population
inferred← citeshttps://doi.org/10.1016/j.molmet.2013.06.001openalex-snapshot
Targeting oxidized LDL improves insulin sensitivity and immune cell function in obese Rhesus macaques
inferred← citeshttps://doi.org/10.1016/j.nano.2017.06.022openalex-snapshot
Enhanced neuroprotection of anthocyanin-loaded PEG-gold nanoparticles against Aβ1-42-induced neuroinflammation and neurodegeneration via the NF-KB /JNK/GSK3β signaling pathway
inferred← citeshttps://doi.org/10.1016/j.phrs.2011.02.003openalex-snapshot
Vildagliptin blocks vascular injury in thoracic aorta of diabetic rats by suppressing advanced glycation end product–receptor axis
inferred← citeshttps://doi.org/10.1016/j.phrs.2020.104633openalex-snapshot
Oral administration of spa-derived green alga improves insulin resistance in overweight subjects: Mechanistic insights from fructose-fed rats
inferred← citeshttps://doi.org/10.1016/j.phrs.2020.105212openalex-snapshot
Emerging roles of neutrophil-borne S100A8/A9 in cardiovascular inflammation
inferred← citeshttps://doi.org/10.1016/j.physbeh.2020.113255openalex-snapshot
RAGE signaling is required for AMPA receptor dysfunction in the hippocampus of hyperglycemic mice
inferred← citeshttps://doi.org/10.1016/j.reprotox.2016.04.015openalex-snapshot
Receptor for advanced glycation end products (RAGE) knockout reduces fetal dysmorphogenesis in murine diabetic pregnancy
inferred← citeshttps://doi.org/10.1016/j.semcancer.2017.06.005openalex-snapshot
An insight on the association of glycation with hepatocellular carcinoma
inferred← citeshttps://doi.org/10.1016/j.sexol.2018.01.011openalex-snapshot
La dysfonction érectile du diabétique
inferred← citeshttps://doi.org/10.1016/j.toxrep.2015.07.007openalex-snapshot
Protective activity of gallic acid against glyoxal -induced renal fibrosis in experimental rats
inferred← citeshttps://doi.org/10.1016/j.vph.2012.02.004openalex-snapshot
Nitric oxide synthase uncoupling: A therapeutic target in cardiovascular diseases
inferred← citeshttps://doi.org/10.1016/j.vph.2012.03.005openalex-snapshot
Role of oxidative stress in diabetes-mediated vascular dysfunction: Unifying hypothesis of diabetes revisited
inferred← citeshttps://doi.org/10.1016/j.vph.2012.06.004openalex-snapshot
The diverse ligand repertoire of the receptor for advanced glycation endproducts and pathways to the complications of diabetes
inferred← citeshttps://doi.org/10.1016/j.vph.2019.106565openalex-snapshot
Uric acid: from a biological advantage to a potential danger. A focus on cardiovascular effects
inferred← citeshttps://doi.org/10.1016/j.yhbeh.2012.04.001openalex-snapshot
Ischemia-induced hyperglycemia: Consequences, neuroendocrine regulation, and a role for RAGE
inferred← citeshttps://doi.org/10.1016/s1957-2557(10)70156-5openalex-snapshot
Place des produits de glycation avancés (AGEs) dans les complications du diabète
inferred← citeshttps://doi.org/10.1017/s146239940900101xopenalex-snapshot
The receptor for advanced glycation endproducts (RAGE) and cardiovascular disease
inferred← citeshttps://doi.org/10.1038/cdd.2009.149openalex-snapshot
The receptor for advanced glycation end products (RAGE) sustains autophagy and limits apoptosis, promoting pancreatic tumor cell survival
inferred← citeshttps://doi.org/10.1038/ki.2010.374openalex-snapshot
Cell division autoantigen 1 enhances signaling and the profibrotic effects of transforming growth factor-β in diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/ki.2013.171openalex-snapshot
RAGE-mediated interstitial fibrosis in neonatal obstructive nephropathy is independent of NF-κB activation
inferred← citeshttps://doi.org/10.1038/ki.2013.372openalex-snapshot
Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b
inferred← citeshttps://doi.org/10.1038/labinvest.2012.163openalex-snapshot
KCa3.1 channels mediate the increase of cell migration and proliferation by advanced glycation endproducts in cultured rat vascular smooth muscle cells
inferred← citeshttps://doi.org/10.1038/labinvest.2015.35openalex-snapshot
Dipeptidyl peptidase-4 deficiency protects against experimental diabetic nephropathy partly by blocking the advanced glycation end products-receptor axis
inferred← citeshttps://doi.org/10.1038/nrd2476openalex-snapshot
Therapies for hyperglycaemia-induced diabetic complications: from animal models to clinical trials
inferred← citeshttps://doi.org/10.1038/nrg2468openalex-snapshot
Metabolic syndrome: from epidemiology to systems biology
inferred← citeshttps://doi.org/10.1038/nri2873openalex-snapshot
Sterile inflammation: sensing and reacting to damage
inferred← citeshttps://doi.org/10.1038/onc.2012.150openalex-snapshot
Depletion of the receptor for advanced glycation end products (RAGE) sensitizes towards apoptosis via p53 and p73 posttranslational regulation
inferred← citeshttps://doi.org/10.1038/s41514-020-00049-0openalex-snapshot
Molecular phenotyping of oxidative stress in diabetes mellitus with point-of-care NMR system
inferred← citeshttps://doi.org/10.1038/s41574-026-01309-xopenalex-snapshot
Glycation and receptors: coming of AGE in the midst of danger
inferred← citeshttps://doi.org/10.1038/s41598-018-21176-5openalex-snapshot
RAGE-aptamer attenuates deoxycorticosterone acetate/salt-induced renal injury in mice
inferred← citeshttps://doi.org/10.1038/s41598-018-25942-3openalex-snapshot
Hyperglycemia does not affect tissue repair responses in shear stress-induced atherosclerotic plaques in ApoE−/− mice
inferred← citeshttps://doi.org/10.1038/s41598-018-33661-yopenalex-snapshot
RAGE deficiency does not affect non-alcoholic steatohepatitis and atherosclerosis in Western type diet-fed Ldlr−/− mice
inferred← citeshttps://doi.org/10.1038/s41598-019-56242-zopenalex-snapshot
Advanced glycation end-products regulate extracellular matrix-adipocyte metabolic crosstalk in diabetes
inferred← citeshttps://doi.org/10.1038/s41598-021-03007-2openalex-snapshot
Oxidative stress and soluble receptor for advanced glycation end-products play a role in the pathophysiology of delirium after cardiac surgery
inferred← citeshttps://doi.org/10.1038/s41598-021-96859-7openalex-snapshot
Deletion of RAGE fails to prevent hepatosteatosis in obese mice due to impairment of other AGEs receptors and detoxifying systems
inferred← citeshttps://doi.org/10.1038/s41598-024-62385-5openalex-snapshot
The Gly82Ser polymorphism in the receptor for advanced glycation endproducts increases the risk for coronary events in the general population
inferred← citeshttps://doi.org/10.1038/s42003-023-04643-2openalex-snapshot
DIAPH1 mediates progression of atherosclerosis and regulates hepatic lipid metabolism in mice
inferred← citeshttps://doi.org/10.1038/srep42027openalex-snapshot
Protective effect of mangiferin on myocardial ischemia-reperfusion injury in streptozotocin-induced diabetic rats: role of AGE-RAGE/MAPK pathways
inferred← citeshttps://doi.org/10.1039/c6fo00169fopenalex-snapshot
Glucitol-core containing gallotannins inhibit the formation of advanced glycation end-products mediated by their antioxidant potential
inferred← citeshttps://doi.org/10.1039/d2nr03756dopenalex-snapshot
Stimuli-responsive therapeutic systems for the treatment of diabetic infected wounds
inferred← citeshttps://doi.org/10.1042/cs20100501openalex-snapshot
RAGE biology, atherosclerosis and diabetes
inferred← citeshttps://doi.org/10.1042/cs20130065openalex-snapshot
Nox as a target for diabetic complications
inferred← citeshttps://doi.org/10.1042/cs20140045openalex-snapshot
Role of bone-marrow- and non-bone-marrow-derived receptor for advanced glycation end-products (RAGE) in a mouse model of diabetes-associated atherosclerosis
inferred← citeshttps://doi.org/10.1042/cs20150427openalex-snapshot
miR-21 promotes renal fibrosis in diabetic nephropathy by targeting PTEN and SMAD7
inferred← citeshttps://doi.org/10.1042/cs20160249openalex-snapshot
Differential effects of NOX4 and NOX1 on immune cell-mediated inflammation in the aortic sinus of diabetic ApoE−/− mice
inferred← citeshttps://doi.org/10.1042/cs20171459openalex-snapshot
A causal link between oxidative stress and inflammation in cardiovascular and renal complications of diabetes
inferred← citeshttps://doi.org/10.1042/cs20190768openalex-snapshot
Nutrient regulation of inflammatory signalling in obesity and vascular disease
inferred← citeshttps://doi.org/10.1055/s-0040-1721813openalex-snapshot
AGE–RAGE Stress and Coronary Artery Disease
inferred← citeshttps://doi.org/10.1080/00036811.2022.2060210openalex-snapshot
Well-posedness of a mathematical model of diabetic atherosclerosis with advanced glycation end-products
inferred← citeshttps://doi.org/10.1080/07315724.2021.1978349openalex-snapshot
Metabolite Profiling of Antioxidant Rich Fractions of Punica granatum L. Mesocarp and CD36 Expression Regulation
inferred← citeshttps://doi.org/10.1080/07853890902806576openalex-snapshot
Tempering the wrath of RAGE: An emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation
inferred← citeshttps://doi.org/10.1089/ars.2010.3356openalex-snapshot
High-Mobility Group Box 1, Oxidative Stress, and Disease
inferred← citeshttps://doi.org/10.1089/ars.2014.6078openalex-snapshot
Isolevuglandin-Type Lipid Aldehydes Induce the Inflammatory Response of Macrophages by Modifying Phosphatidylethanolamines and Activating the Receptor for Advanced Glycation Endproducts
inferred← citeshttps://doi.org/10.1089/ars.2016.6664openalex-snapshot
Diabetes and Kidney Disease: Role of Oxidative Stress
inferred← citeshttps://doi.org/10.1089/ars.2021.0116openalex-snapshot
Oxidative Stress in Neutrophils: Implications for Diabetic Cardiovascular Complications
inferred← citeshttps://doi.org/10.1089/dna.2016.3552openalex-snapshot
Receptor for Advanced Glycation End Products Regulates Leukotriene B 4 Receptor 1 Signaling
inferred← citeshttps://doi.org/10.1089/rej.2010.1091openalex-snapshot
Age of Onset of Myocardial Infarction: Is Promoter Polymorphism of the RAGE Gene Implicated?
inferred← citeshttps://doi.org/10.1089/scd.2016.0067openalex-snapshot
Receptor for Advanced Glycation End Products-Mediated Signaling Impairs the Maintenance of Bone Marrow Mesenchymal Stromal Cells in Diabetic Model Mice
inferred← citeshttps://doi.org/10.1093/ckj/sfaa143openalex-snapshot
A primer on metabolic memory: why existing diabesity treatments fail
inferred← citeshttps://doi.org/10.1093/cvr/cvaa118openalex-snapshot
Ex vivo Ikkβ ablation rescues the immunopotency of mesenchymal stromal cells from diabetics with advanced atherosclerosis
inferred← citeshttps://doi.org/10.1093/cvr/cvp031openalex-snapshot
Vascular NAD(P)H oxidase activation in diabetes: a double-edged sword in redox signalling
inferred← citeshttps://doi.org/10.1093/europace/euv449openalex-snapshot
High plasma level of soluble RAGE is independently associated with a low recurrence of atrial fibrillation after catheter ablation in diabetic patient
inferred← citeshttps://doi.org/10.1093/glycob/cwac060openalex-snapshot
Understanding the role of glycation in the pathology of various non-communicable diseases along with novel therapeutic strategies
inferred← citeshttps://doi.org/10.1096/fj.201500117openalex-snapshot
Modulation of leukotriene B 4 receptor 1 signaling by receptor for advanced glycation end products (RAGE)
inferred← citeshttps://doi.org/10.1096/fj.201701553rropenalex-snapshot
JNK and ATF4 as two important platforms for tumor necrosis factor‐α–stimulated shedding of receptor for advanced glycation end products
inferred← citeshttps://doi.org/10.1096/fj.202002136ropenalex-snapshot
The role of RAGE in host pathology and crosstalk between RAGE and TLR4 in innate immune signal transduction pathways
inferred← citeshttps://doi.org/10.1096/fj.202300173rropenalex-snapshot
Macrophage RAGE deficiency prevents myocardial fibrosis by repressing autophagy‐mediated macrophage alternative activation
inferred← citeshttps://doi.org/10.1097/ccm.0000000000002070openalex-snapshot
Protective Effect of Inflammasome Activation by Hydrogen Peroxide in a Mouse Model of Septic Shock
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