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Interaction of PPARα With the Canonic Wnt Pathway in the Regulation of Renal Fibrosis
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/s00125-010-1943-1openalex-snapshot
The role of lipid peroxidation products and oxidative stress in activation of the canonical wingless-type MMTV integration site (WNT) pathway in a rat model of diabetic retinopathy
inferredcites →https://doi.org/10.1007/s00125-011-2314-2openalex-snapshot
Implication of dysregulation of the canonical wingless-type MMTV integration site (WNT) pathway in diabetic nephropathy
inferredcites →https://doi.org/10.1007/s11010-015-2569-xopenalex-snapshot
The wnt/β-catenin signaling pathway participates in rhein ameliorating kidney injury in DN mice
inferredcites →https://doi.org/10.1016/j.ajpath.2014.06.021openalex-snapshot
Peroxisome Proliferator–Activated Receptor α Protects Capillary Pericytes in the Retina
inferredcites →https://doi.org/10.1016/j.febslet.2006.09.011openalex-snapshot
Modulation of LRP6‐mediated Wnt signaling by molecular chaperone Mesd
inferredcites →https://doi.org/10.1016/j.febslet.2011.01.004openalex-snapshot
CTGF/CCN2 activates canonical Wnt signalling in mesangial cells through LRP6: Implications for the pathogenesis of diabetic nephropathy
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2012.06.027openalex-snapshot
Role of Nox4 in murine models of kidney disease
inferredcites →https://doi.org/10.1016/j.phrs.2012.01.004openalex-snapshot
Are PPAR alpha agonists a rational therapeutic strategy for preventing abnormalities of the diabetic kidney?
inferredcites →https://doi.org/10.1016/j.reprotox.2013.08.001openalex-snapshot
Perfluorooctanoate suppresses spheroid attachment on endometrial epithelial cells through peroxisome proliferator-activated receptor alpha and down-regulation of Wnt signaling
inferredcites →https://doi.org/10.1016/s0002-9440(10)62255-9openalex-snapshot
Genetic Difference in Susceptibility to the Blood-Retina Barrier Breakdown in Diabetes and Oxygen-Induced Retinopathy
inferredcites →https://doi.org/10.1016/s0022-2143(97)90180-3openalex-snapshot
Cell lines with extended in vitro growth potential from human renal proximal tubule: Characterization, response to inducers, and comparison with established cell lines
inferredcites →https://doi.org/10.1016/s0140-6736(10)60576-4openalex-snapshot
Effect of intensive treatment of hyperglycaemia on microvascular outcomes in type 2 diabetes: an analysis of the ACCORD randomised trial
inferredcites →https://doi.org/10.1038/eye.2011.62openalex-snapshot
Medical management of diabetic retinopathy: fenofibrate and ACCORD Eye studies
inferredcites →https://doi.org/10.1042/bj20030426openalex-snapshot
Peroxisome-proliferator-activated receptor γ suppresses Wnt/β-catenin signalling during adipogenesis
inferredcites →https://doi.org/10.1046/j.1523-1755.2001.00766.xopenalex-snapshot
Peroxisome proliferator-activated receptors (PPARs): Novel therapeutic targets in renal disease
inferredcites →https://doi.org/10.1055/s-2001-18600openalex-snapshot
Pathogenesis of diabetic nephropathy
inferredcites →https://doi.org/10.1073/pnas.0434590100openalex-snapshot
Mapping Wnt/β-catenin signaling during mouse development and in colorectal tumors
inferredcites →https://doi.org/10.1074/jbc.m111.223164openalex-snapshot
Wnt/β-Catenin Pathway in Podocytes Integrates Cell Adhesion, Differentiation, and Survival
inferredcites →https://doi.org/10.1089/ars.2012.4583openalex-snapshot
Nitrosative Stress Plays an Important Role in Wnt Pathway Activation in Diabetic Retinopathy
inferredcites →https://doi.org/10.1124/mol.114.097402openalex-snapshot
Activating and Inhibitory Functions of WNT/β-Catenin in the Induction of Cytochromes P450 by Nuclear Receptors in HepaRG Cells
inferredcites →https://doi.org/10.1128/mcb.01211-10openalex-snapshot
Identification of a Novel Inhibitor of the Canonical Wnt Pathway
inferredcites →https://doi.org/10.1128/mcb.23.16.5825-5835.2003openalex-snapshot
A Novel Mechanism for Wnt Activation of Canonical Signaling through the LRP6 Receptor
inferredcites →https://doi.org/10.1146/annurev-physiol-020911-153333openalex-snapshot
Renal Function in Diabetic Disease Models: The Tubular System in the Pathophysiology of the Diabetic Kidney
inferredcites →https://doi.org/10.1146/annurev.cellbio.20.010403.113126openalex-snapshot
THE WNT SIGNALING PATHWAY IN DEVELOPMENT AND DISEASE
inferredcites →https://doi.org/10.1152/ajprenal.00064.2008openalex-snapshot
An androgen-inducible proximal tubule-specific Cre recombinase transgenic model
inferredcites →https://doi.org/10.1152/ajprenal.00363.2006openalex-snapshot
A primary culture of mouse proximal tubular cells, established on collagen-coated membranes
inferredcites →https://doi.org/10.1152/ajprenal.00475.2009openalex-snapshot
Troglitazone ameliorates high glucose-induced EMT and dysfunction of SGLTs through PI3K/Akt, GSK-3β, Snail1, and β-catenin in renal proximal tubule cells
inferredcites →https://doi.org/10.1152/ajprenal.00511.2012openalex-snapshot
Role of Nox2 in diabetic kidney disease
inferredcites →https://doi.org/10.1167/iovs.09-4278openalex-snapshot
The Pathogenic Role of the Canonical Wnt Pathway in Age-Related Macular Degeneration
inferredcites →https://doi.org/10.1167/iovs.13-13127openalex-snapshot
Protective and Antioxidant Effects of PPARα in the Ischemic Retina
inferredcites →https://doi.org/10.1210/edrv.20.5.0380openalex-snapshot
Peroxisome Proliferator-Activated Receptors: Nuclear Control of Metabolism
inferredcites →https://doi.org/10.14814/phy2.12192openalex-snapshot
Nox-4 deletion reduces oxidative stress and injury by PKC-α-associated mechanisms in diabetic nephropathy
inferredcites →https://doi.org/10.1681/asn.2004110949openalex-snapshot
Wnt-Dependent β-Catenin Signaling Is Activated after Unilateral Ureteral Obstruction, and Recombinant Secreted Frizzled-Related Protein 4 Alters the Progression of Renal Fibrosis
inferredcites →https://doi.org/10.1681/asn.2007010128openalex-snapshot
Molecular Basis for the Cell Type–Specific Induction of SnoN Expression by Hepatocyte Growth Factor
inferredcites →https://doi.org/10.1681/asn.2008060566openalex-snapshot
Wnt/β-Catenin Signaling Promotes Renal Interstitial Fibrosis
inferredcites →https://doi.org/10.1681/asn.2009010019openalex-snapshot
Wnt/β-Catenin Signaling Promotes Podocyte Dysfunction and Albuminuria
inferredcites →https://doi.org/10.1681/asn.2013070810openalex-snapshot
Genetic Targeting or Pharmacologic Inhibition of NADPH Oxidase Nox4 Provides Renoprotection in Long-Term Diabetic Nephropathy
inferredcites →https://doi.org/10.2174/092986710793348581openalex-snapshot
Oxidative Stress in Diabetic Nephropathy
inferredcites →https://doi.org/10.2174/138161209787315701openalex-snapshot
Fibrates and Microvascular Complications in Diabetes - Insight from the FIELD Study
inferredcites →https://doi.org/10.2174/1874-470211205020292openalex-snapshot
PPAR-γ Agonists in Polycystic Kidney Disease with Frequent Development of Cardiovascular Disorders
inferredcites →https://doi.org/10.2337/db09-1056openalex-snapshot
Inhibition of Connective Tissue Growth Factor Overexpression in Diabetic Retinopathy by SERPINA3K via Blocking the WNT/β-Catenin Pathway
inferredcites →https://doi.org/10.2337/db11-0300openalex-snapshot
Therapeutic Potential of a Monoclonal Antibody Blocking the Wnt Pathway in Diabetic Retinopathy
inferredcites →https://doi.org/10.2337/dc09-0621openalex-snapshot
Effects of Long-Term Fenofibrate Treatment on Markers of Renal Function in Type 2 Diabetes
inferredcites →https://doi.org/10.2337/diabetes.54.6.1615openalex-snapshot
The Pathobiology of Diabetic Complications
inferredcites →https://doi.org/10.2337/diabetes.55.04.06.db05-1329openalex-snapshot
Accelerated Diabetic Nephropathy in Mice Lacking the Peroxisome Proliferator–Activated Receptor α
inferredcites →https://doi.org/10.3389/fphys.2014.00429openalex-snapshot
Circadian rhythms, Wnt/beta-catenin pathway and PPAR alpha/gamma profiles in diseases with primary or secondary cardiac dysfunction
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Canonical Wnt Signaling Promotes Neovascularization Through Determination of Endothelial Progenitor Cell Fate via Metabolic Profile Regulation
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Lipid Metabolism Disorder and Renal Fibrosis
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Salutary effect of fenofibrate on type 1 diabetic retinopathy via inhibiting oxidative stress–mediated Wnt/β-catenin pathway activation
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Fenofibrate ameliorates diabetic retinopathy by modulating Nrf2 signaling and NLRP3 inflammasome activation
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The PPAR-α agonist oleoyethanolamide (OEA) ameliorates valproic acid-induced steatohepatitis in rats via suppressing Wnt3a/β-catenin and activating PGC-1α: Involvement of network pharmacology and molecular docking
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Nuclear translocation of GPS2 promotes activation of hepatic stellate cells by fatty acid oxidation pathway
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Cardiac-specific β-catenin deletion dysregulates energetic metabolism and mitochondrial function in perinatal cardiomyocytes
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Intracellular lipids are an independent cause of liver injury and chronic kidney disease in non alcoholic fatty liver disease-like context
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Protective or deleterious role of Wnt/beta-catenin signaling in diabetic nephropathy: An unresolved issue
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Wnt Signaling in vascular eye diseases
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Nuclear Receptors and Transcription Factors in Obesity-Related Kidney Disease
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Activation of PPARα by clofibrate sensitizes pancreatic cancer cells to radiation through the Wnt/β-catenin pathway
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SIK2 maintains breast cancer stemness by phosphorylating LRP6 and activating Wnt/β-catenin signaling
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Fenofibrate prevents iron induced activation of canonical Wnt/β-catenin and oxidative stress signaling in the retina
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WNT–β-catenin signalling — a versatile player in kidney injury and repair
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PPARα activation promotes liver progenitor cell-mediated liver regeneration by suppressing YAP signaling in zebrafish
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Key enzyme in charge of ketone reabsorption of renal tubular SMCT1 may be a new target in diabetic kidney disease
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