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RETRACTED: Cyclosporin A inhibits Al-induced cytochrome c release from mitochondria in aged rabbits

https://doi.org/10.3233/jad-2001-3404
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Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Retraction Watch lists this work as retracted (export 2026-10-21; notice 2024-03-05, 10.3233/jad-239015). OpenCitations Index CSV, figshare 24356626 v8 (2026-09-08) records 18 citing works; 0 have dates after the notice under the dataset’s date-resolution rule.

Dataset join, best-effort; this page does not check the publisher record. Retraction Watch data via Crossref Labs, used with credit; OpenCitations Index (CC0) and Meta (CC BY 4.0); dataset doi:10.7910/DVN/WT8VOA (CC BY 4.0).

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/(sici)1097-4547(19990601)56:5<457::aid-jnr2>3.0.co;2-popenalex-snapshot
Superoxide mediates the cell-death-enhancing action of presenilin-1 mutations
inferredcites →https://doi.org/10.1007/bf02110643openalex-snapshot
The permeability transition pore as a mitochondrial calcium release channel: A critical appraisal
inferredcites →https://doi.org/10.1016/0006-8993(95)01264-8openalex-snapshot
Quantitative image analysis of temporal changes in tau and neurofilament proteins during the course of acute experimental neurofibrillary degeneration; non-phosphorylated epitopes precede phosphorylation
inferredcites →https://doi.org/10.1016/0304-4157(95)00003-aopenalex-snapshot
The mitochondrial permeability transition
inferredcites →https://doi.org/10.1016/s0006-8993(97)00780-4openalex-snapshot
Neurofibrillary lesions in experimental aluminum-induced encephalopathy and Alzheimer's disease share immunoreactivity for amyloid precursor protein, Aβ, α1-antichymotrypsin and ubiquitin-protein conjugates
inferredcites →https://doi.org/10.1016/s0006-8993(99)02047-8openalex-snapshot
Free radicals and lipid peroxidation do not mediate β-amyloid-induced neuronal cell death
inferredcites →https://doi.org/10.1016/s0021-9258(20)80631-0openalex-snapshot
Physiological effectors modify voltage sensing by the cyclosporin A-sensitive permeability transition pore of mitochondria.
inferredcites →https://doi.org/10.1016/s0165-0173(98)00046-0openalex-snapshot
An evaluation of the role of mitochondria in neurodegenerative diseases: mitochondrial mutations and oxidative pathology, protective nuclear responses, and cell death in neurodegeneration
inferredcites →https://doi.org/10.1016/s0304-3940(99)00327-4openalex-snapshot
Release of mitochondrial cytochrome c and DNA fragmentation after cold injury-induced brain trauma in mice: possible role in neuronal apoptosis
inferredcites →https://doi.org/10.1042/bj2680153openalex-snapshot
Inhibition of Ca2+-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adenine nucleotide translocase
inferredcites →https://doi.org/10.1046/j.1471-4159.1999.0722323.xopenalex-snapshot
The Lipid Peroxidation Product 4‐Hydroxynonenal Inhibits Neurite Outgrowth, Disrupts Neuronal Microtubules, and Modifies Cellular Tubulin
inferredcites →https://doi.org/10.1073/pnas.94.8.3668openalex-snapshot
Cytosol-to-membrane redistribution of Bax and Bcl-X L during apoptosis
inferredcites →https://doi.org/10.1084/jem.183.4.1533openalex-snapshot
Mitochondrial control of nuclear apoptosis.
inferredcites →https://doi.org/10.1093/brain/122.8.1519openalex-snapshot
Neocortical and hippocampal glucose hypometabolism following neurotoxic lesions of the entorhinal and perirhinal cortices in the non-human primate as shown by PET
inferredcites →https://doi.org/10.1097/00001756-199902050-00026openalex-snapshot
Cyclosporin A limits calcium-induced axonal damage following traumatic brain injury
inferredcites →https://doi.org/10.1111/j.1365-2990.1990.tb01290.xopenalex-snapshot
Neuronal cytoskeletal lesions induced in the CNS by intraventricular and intravenous aluminium maltol in rabbits
inferredcites →https://doi.org/10.1128/mcb.18.11.6353openalex-snapshot
The Mitochondrial Permeability Transition Is Required for Tumor Necrosis Factor Alpha-Mediated Apoptosis and CytochromecRelease

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4160337openalex-extracted
inferred← citeshttps://doi.org/10.1006/nbdi.2001.0429openalex-snapshot
RETRACTED: GDNF Protects against Aluminum-Induced Apoptosis in Rabbits by Upregulating Bcl-2 and Bcl-XL and Inhibiting Mitochondrial Bax Translocation
inferred← citeshttps://doi.org/10.1007/s10534-014-9803-yopenalex-snapshot
Zinc down regulates Apaf-1-dependent Bax/Bcl-2 mediated caspases activation during aluminium induced neurotoxicity
inferred← citeshttps://doi.org/10.1007/s11064-016-1926-9openalex-snapshot
Folic Acid Protected Neural Cells Against Aluminum-Maltolate-Induced Apoptosis by Preventing miR-19 Downregulation
inferred← citeshttps://doi.org/10.1007/s12011-015-0229-3openalex-snapshot
Protective Effect of Selenium Against Aluminum Chloride-Induced Alzheimer’s Disease: Behavioral and Biochemical Alterations in Rats
inferred← citeshttps://doi.org/10.1016/j.brainresrev.2006.04.003openalex-snapshot
A new insight on Al-maltolate-treated aged rabbit as Alzheimer's animal model
inferred← citeshttps://doi.org/10.1016/j.immuni.2012.01.009openalex-snapshot
Oxidized Mitochondrial DNA Activates the NLRP3 Inflammasome during Apoptosis
inferred← citeshttps://doi.org/10.1016/j.jinorgbio.2022.112032openalex-snapshot
Comparative Study of Aluminum Speciation on Brain-Type Creatine Kinase: Enzyme Kinetic, Molecular Docking, Cellular Experiment, and Mouse Model Study
inferred← citeshttps://doi.org/10.1016/j.jtemb.2015.04.002openalex-snapshot
Mangiferin ameliorates aluminium chloride-induced cognitive dysfunction via alleviation of hippocampal oxido-nitrosative stress, proinflammatory cytokines and acetylcholinesterase level
inferred← citeshttps://doi.org/10.1016/j.neuro.2003.12.004openalex-snapshot
Aluminum Maltolate-Induced Toxicity in NT2 Cells Occurs Through Apoptosis and Includes Cytochrome c Release
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2016.02.055openalex-snapshot
Quercetin attenuates neuronal death against aluminum-induced neurodegeneration in the rat hippocampus
inferred← citeshttps://doi.org/10.1016/s1734-1140(11)70607-7openalex-snapshot
Neuroprotective effect of carvedilol against aluminium induced toxicity: possible behavioral and biochemical alterations in rats
inferred← citeshttps://doi.org/10.1111/j.1600-079x.2011.00861.xopenalex-snapshot
Melatonin preserves the transient mitochondrial permeability transition for protection during mitochondrial Ca2+stress in astrocyte
inferred← citeshttps://doi.org/10.1152/physrev.00013.2006openalex-snapshot
Mitochondrial Membrane Permeabilization in Cell Death
inferred← citeshttps://doi.org/10.22159/ijpps.2017v9i5.17584openalex-snapshot
NEUROPROTECTIVE ACTIVITY OF FRACTIONAL FLOWER EXTRACTS OF MIRABILIS JALAPA AGAINST ALUMINIUM HYDROCHLORIDE INDUCED NEUROTOXICITY IN MALE WISTER RATS
inferred← citeshttps://doi.org/10.22192/ijarbs.2017.04.11.010openalex-snapshot
Potential role of some nutraceuticals in neurotoxicity induced by aluminum oxide in experimental animal model
inferred← citeshttps://doi.org/10.3109/00207451003760080openalex-snapshot
Animal Models of Alzheimer's Disease: An Understanding of Pathology and Therapeutic Avenues
inferred← citeshttps://doi.org/10.3233/jad-2006-9211openalex-snapshot
Peri-nuclear clustering of mitochondria is triggered during aluminum maltolate induced apoptosis
inferred← citeshttps://doi.org/10.4021/jocmr2009.09.1265openalex-snapshot
Studies on Genomic DNA Stability in Aluminium-Maltolate Treated Aged New Zealand Rabbit: Relevance to the Alzheimer’s Animal Model
inferred← citeshttps://doi.org/10.5045/kjh.2005.40.2.75openalex-snapshot
Apoptosis Induction by Curcumin in Human Myelogenous Leukemia Cell Lines
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