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Antimicrobial Peptides Human Beta-Defensin-2 and -3 Protect the Gut During Candida albicans Infections Enhancing the Intestinal Barrier Integrity: In Vitro Study
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Effect of 1064-nm Q-switched Nd:YAG laser on invasiveness and innate immune response in keratinocytes infected with Candida albicans
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The Caco-2 cell line as a model of the intestinal barrier: influence of cell and culture-related factors on Caco-2 cell functional characteristics
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Interaction of Candida albicans with host cells: virulence factors, host defense, escape strategies, and the microbiota
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Human Intestinal Microbiota: Interaction Between Parasites and the Host Immune Response
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AMPed up immunity: how antimicrobial peptides have multiple roles in immune defense
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The Intestinal Epithelium: Central Coordinator of Mucosal Immunity
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The Intestinal Epithelium: Central Coordinator of Mucosal Immunity
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Induction of human defensins by intestinal Caco-2 cells after interactions with opportunistic Candida species
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The Claudins: From Tight Junctions to Biological Systems
inferredcites →https://doi.org/10.1016/j.tim.2004.05.008openalex-snapshot
The distinct morphogenic states of Candida albicans
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Colonic MUC2 mucin regulates the expression and antimicrobial activity of β-defensin 2
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The immunology of host defence peptides: beyond antimicrobial activity
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Zonulin, a regulator of epithelial and endothelial barrier functions, and its involvement in chronic inflammatory diseases
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Intestinal barrier and gut microbiota: Shaping our immune responses throughout life
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“ Candida Albicans Interactions With The Host: Crossing The Intestinal Epithelial Barrier”
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Candida albicans induces early apoptosis followed by secondary necrosis in oral epithelial cells
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Induction of apoptosis in oral epithelial cells by Candida albicans
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Candida albicans-Induced Epithelial Damage Mediates Translocation through Intestinal Barriers
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Increased Intestinal Permeability and Decreased Barrier Function: Does It Really Influence the Risk of Inflammation?
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The expression of the β-defensins hBD-2 and hBD-3 is differentially regulated by NF-κB and MAPK/AP-1 pathways in an in vitro model of Candida esophagitis
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The cross-talk between enterocytes and intraepithelial lymphocytes
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Glycosylation of Candida albicans Cell Wall Proteins Is Critical for Induction of Innate Immune Responses and Apoptosis of Epithelial Cells
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Intestinal Cell Tight Junctions Limit Invasion of Candida albicans through Active Penetration and Endocytosis in the Early Stages of the Interaction of the Fungus with the Intestinal Barrier
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Candida albicans induces mucosal bacterial dysbiosis that promotes invasive infection
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Induction of Different Apoptosis Pathways by Two Proteusmirabilis Clinical Isolates Strains in Prostatic Epithelial Cells
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Claudin1 promotes the proliferation, invasion and migration of nasopharyngeal carcinoma cells by upregulating the expression and nuclear entry of β‑catenin
inferredcites →https://doi.org/10.4236/ojmm.2013.33028openalex-snapshot
Effect of Growth Media, pH and Temperature on Yeast to Hyphal Transition in Candida albicans
inferredcites →https://doi.org/10.5402/2012/345791openalex-snapshot
Modulation of Neutrophil Apoptosis by Antimicrobial Peptides
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Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Additional file 1 of Intestinal Piezo1 aggravates intestinal barrier dysfunction during sepsis by mediating Ca2+ influx
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