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Autophagy Contributes to Metabolic Reprogramming and Therapeutic Resistance in Pancreatic Tumors
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Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/jamaoncol.2019.0684openalex-snapshot
Effect of Gemcitabine and nab-Paclitaxel With or Without Hydroxychloroquine on Patients With Advanced Pancreatic Cancer
inferredcites →https://doi.org/10.1002/adma.202007778openalex-snapshot
Mito‐Bomb: Targeting Mitochondria for Cancer Therapy
inferredcites →https://doi.org/10.1002/ags3.12225openalex-snapshot
Role of the tumor microenvironment in pancreatic cancer
inferredcites →https://doi.org/10.1002/jcsm.12225openalex-snapshot
Establishment and characterization of a novel murine model of pancreatic cancer cachexia
inferredcites →https://doi.org/10.1002/jso.23194openalex-snapshot
Histopathologic assessment of pancreatic cancer: Does one size fit all?
inferredcites →https://doi.org/10.1007/s00018-021-03866-yopenalex-snapshot
Overcoming chemoresistance by targeting reprogrammed metabolism: the Achilles' heel of pancreatic ductal adenocarcinoma
inferredcites →https://doi.org/10.1007/s00280-017-3446-yopenalex-snapshot
Phase I study of a chloroquine–gemcitabine combination in patients with metastatic or unresectable pancreatic cancer
inferredcites →https://doi.org/10.1016/j.ajpath.2013.02.034openalex-snapshot
Oxidative Stress Induced by Inactivation of TP53INP1 Cooperates with KrasG12D to Initiate and Promote Pancreatic Carcinogenesis in the Murine Pancreas
inferredcites →https://doi.org/10.1016/j.bbabio.2017.01.012openalex-snapshot
Mitochondria and cancer chemoresistance
inferredcites →https://doi.org/10.1016/j.biochi.2015.07.024openalex-snapshot
The stress protein TP53INP1 plays a tumor suppressive role by regulating metabolic homeostasis
inferredcites →https://doi.org/10.1016/j.ceb.2009.12.008openalex-snapshot
Role of autophagy in suppression of inflammation and cancer
inferredcites →https://doi.org/10.1016/j.cell.2007.12.018openalex-snapshot
Autophagy in the Pathogenesis of Disease
inferredcites →https://doi.org/10.1016/j.cell.2013.11.019openalex-snapshot
Autophagy-Mediated Tumor Promotion
inferredcites →https://doi.org/10.1016/j.celrep.2021.109528openalex-snapshot
Autophagy is a major metabolic regulator involved in cancer therapy resistance
inferredcites →https://doi.org/10.1016/j.cgh.2012.11.016openalex-snapshot
Mutant TP53 in Duodenal Samples of Pancreatic Juice From Patients With Pancreatic Cancer or High-Grade Dysplasia
inferredcites →https://doi.org/10.1016/j.cmet.2008.08.013openalex-snapshot
Loss of Autophagy Diminishes Pancreatic β Cell Mass and Function with Resultant Hyperglycemia
inferredcites →https://doi.org/10.1016/j.cmet.2015.12.006openalex-snapshot
The Emerging Hallmarks of Cancer Metabolism
inferredcites →https://doi.org/10.1016/j.cmet.2017.04.004openalex-snapshot
Autophagy and Tumor Metabolism
inferredcites →https://doi.org/10.1016/j.cmet.2019.08.013openalex-snapshot
Mechanisms and Implications of Metabolic Heterogeneity in Cancer
inferredcites →https://doi.org/10.1016/j.cmet.2020.10.017openalex-snapshot
Functional Genomics In Vivo Reveal Metabolic Dependencies of Pancreatic Cancer Cells
inferredcites →https://doi.org/10.1016/j.ejso.2020.02.037openalex-snapshot
Borderline or locally advanced pancreatic adenocarcinoma: A single center experience on the FOLFIRINOX induction regimen
inferredcites →https://doi.org/10.1016/j.gde.2010.12.008openalex-snapshot
Autophagy in tumorigenesis and energy metabolism: friend by day, foe by night
inferredcites →https://doi.org/10.1016/j.jcmgh.2017.05.005openalex-snapshot
Acinar-to-Ductal Metaplasia Induced by Transforming Growth Factor Beta Facilitates KRAS G12D -driven Pancreatic Tumorigenesis
inferredcites →https://doi.org/10.1016/j.molmet.2019.10.002openalex-snapshot
Metabolic functions of the tumor suppressor p53: Implications in normal physiology, metabolic disorders, and cancer
inferredcites →https://doi.org/10.1016/j.redox.2014.12.003openalex-snapshot
Interplay between ROS and autophagy in cancer cells, from tumor initiation to cancer therapy
inferredcites →https://doi.org/10.1016/j.semcdb.2019.05.029openalex-snapshot
Autophagy and mitophagy in cancer metabolic remodelling
inferredcites →https://doi.org/10.1016/j.xcrm.2020.100143openalex-snapshot
Targeting Mitochondrial Complex I Overcomes Chemoresistance in High OXPHOS Pancreatic Cancer
inferredcites →https://doi.org/10.1038/cdd.2009.149openalex-snapshot
The receptor for advanced glycation end products (RAGE) sustains autophagy and limits apoptosis, promoting pancreatic tumor cell survival
inferredcites →https://doi.org/10.1038/cdd.2012.30openalex-snapshot
TP53INP1, a tumor suppressor, interacts with LC3 and ATG8-family proteins through the LC3-interacting region (LIR) and promotes autophagy-dependent cell death
inferredcites →https://doi.org/10.1038/modpathol.3800685openalex-snapshot
Pancreatic adenocarcinoma: update on the surgical pathology of carcinomas of ductal origin and PanINs
inferredcites →https://doi.org/10.1038/nature12865openalex-snapshot
p53 status determines the role of autophagy in pancreatic tumour development
inferredcites →https://doi.org/10.1038/nature13611openalex-snapshot
Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function
inferredcites →https://doi.org/10.1038/nature16965openalex-snapshot
Genomic analyses identify molecular subtypes of pancreatic cancer
inferredcites →https://doi.org/10.1038/nature19084openalex-snapshot
Pancreatic stellate cells support tumour metabolism through autophagic alanine secretion
inferredcites →https://doi.org/10.1038/nchembio.1712openalex-snapshot
Targeting mitochondria metabolism for cancer therapy
inferredcites →https://doi.org/10.1038/nrc3262openalex-snapshot
Deconvoluting the context-dependent role for autophagy in cancer
inferredcites →https://doi.org/10.1038/nrd4002openalex-snapshot
Modulation of oxidative stress as an anticancer strategy
inferredcites →https://doi.org/10.1038/onc.2012.210openalex-snapshot
Maintenance of acinar cell organization is critical to preventing Kras-induced acinar-ductal metaplasia
inferredcites →https://doi.org/10.1038/s41388-017-0046-6openalex-snapshot
Dual role of autophagy in hallmarks of cancer
inferredcites →https://doi.org/10.1038/s41416-020-01039-5openalex-snapshot
Autophagy as a therapeutic target in pancreatic cancer
inferredcites →https://doi.org/10.1038/s41419-021-03742-4openalex-snapshot
TP53INP1 exerts neuroprotection under ageing and Parkinson’s disease-related stress condition
inferredcites →https://doi.org/10.1038/s41467-020-17882-2openalex-snapshot
Autophagy regulates fatty acid availability for oxidative phosphorylation through mitochondria-endoplasmic reticulum contact sites
inferredcites →https://doi.org/10.1038/s41467-020-18954-zopenalex-snapshot
Exploiting oxidative phosphorylation to promote the stem and immunoevasive properties of pancreatic cancer stem cells
inferredcites →https://doi.org/10.1038/s41556-018-0037-zopenalex-snapshot
Cargo recognition and degradation by selective autophagy
inferredcites →https://doi.org/10.1038/s41568-021-00344-2openalex-snapshot
Autophagy in tumour immunity and therapy
inferredcites →https://doi.org/10.1038/s41568-021-00378-6openalex-snapshot
Cancer metabolism: looking forward
inferredcites →https://doi.org/10.1038/s41571-019-0281-6openalex-snapshot
From state-of-the-art treatments to novel therapies for advanced-stage pancreatic cancer
inferredcites →https://doi.org/10.1038/s41575-019-0109-yopenalex-snapshot
Molecular subtypes of pancreatic cancer
inferredcites →https://doi.org/10.1038/s41575-021-00486-6openalex-snapshot
Cell death in pancreatic cancer: from pathogenesis to therapy
inferredcites →https://doi.org/10.1038/s41580-019-0120-8openalex-snapshot
Blocking autophagy to starve pancreatic cancer
inferredcites →https://doi.org/10.1038/s41586-018-0697-7openalex-snapshot
Autophagy maintains tumour growth through circulating arginine
inferredcites →https://doi.org/10.1038/s41586-020-2229-5openalex-snapshot
Autophagy promotes immune evasion of pancreatic cancer by degrading MHC-I
inferredcites →https://doi.org/10.1038/s41588-019-0566-9openalex-snapshot
Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution
inferredcites →https://doi.org/10.1038/s41591-019-0367-9openalex-snapshot
Protective autophagy elicited by RAF→MEK→ERK inhibition suggests a treatment strategy for RAS-driven cancers
inferredcites →https://doi.org/10.1038/s41591-019-0368-8openalex-snapshot
Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer
inferredcites →https://doi.org/10.1038/s43018-020-00110-7openalex-snapshot
Autophagy promotes growth of tumors with high mutational burden by inhibiting a T-cell immune response
inferredcites →https://doi.org/10.1038/sj.onc.1208642openalex-snapshot
Loss of BNIP3 expression is a late event in pancreatic cancer contributing to chemoresistance and worsened prognosis
inferredcites →https://doi.org/10.1038/srep30340openalex-snapshot
Autophagy is induced in the skeletal muscle of cachectic cancer patients
inferredcites →https://doi.org/10.1053/j.gastro.2013.07.049openalex-snapshot
Genetic Alterations Associated With Progression From Pancreatic Intraepithelial Neoplasia to Invasive Pancreatic Tumor
inferredcites →https://doi.org/10.1053/j.gastro.2017.01.010openalex-snapshot
Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Promotes Growth of Pancreatic Tumors in Mice
inferredcites →https://doi.org/10.1053/j.gastro.2018.08.033openalex-snapshot
Stratification of Pancreatic Ductal Adenocarcinomas Based on Tumor and Microenvironment Features
inferredcites →https://doi.org/10.1053/j.gastro.2018.09.053openalex-snapshot
Levels of the Autophagy-Related 5 Protein Affect Progression and Metastasis of Pancreatic Tumors in Mice
inferredcites →https://doi.org/10.1053/j.seminoncol.2018.12.003openalex-snapshot
Systemic treatment of pancreatic cancer revisited
inferredcites →https://doi.org/10.1056/nejmoa1011923openalex-snapshot
FOLFIRINOX versus Gemcitabine for Metastatic Pancreatic Cancer
inferredcites →https://doi.org/10.1056/nejmoa1809775openalex-snapshot
FOLFIRINOX or Gemcitabine as Adjuvant Therapy for Pancreatic Cancer
inferredcites →https://doi.org/10.1073/pnas.0703942104openalex-snapshot
Tumor protein 53-induced nuclear protein 1 expression is repressed by miR-155, and its restoration inhibits pancreatic tumor development
inferredcites →https://doi.org/10.1073/pnas.1307237110openalex-snapshot
Hypoxic and Ras-transformed cells support growth by scavenging unsaturated fatty acids from lysophospholipids
inferredcites →https://doi.org/10.1073/pnas.1515617113openalex-snapshot
Macroautophagy is dispensable for growth of KRAS mutant tumors and chloroquine efficacy
inferredcites →https://doi.org/10.1073/pnas.1519384112openalex-snapshot
Basal autophagy maintains pancreatic acinar cell homeostasis and protein synthesis and prevents ER stress
inferredcites →https://doi.org/10.1073/pnas.1809980116openalex-snapshot
TP53INP1 deficiency maintains murine B lymphopoiesis in aged bone marrow through redox-controlled IL-7R/STAT5 signaling
inferredcites →https://doi.org/10.1080/15548627.2018.1474314openalex-snapshot
Chloroquine inhibits autophagic flux by decreasing autophagosome-lysosome fusion
inferredcites →https://doi.org/10.1080/15548627.2019.1569912openalex-snapshot
Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells
inferredcites →https://doi.org/10.1080/15548627.2020.1847462openalex-snapshot
Regulation and function of autophagy in pancreatic cancer
inferredcites →https://doi.org/10.1091/mbc.e10-06-0500openalex-snapshot
Autophagy facilitates glycolysis during Ras-mediated oncogenic transformation
inferredcites →https://doi.org/10.1101/gad.17558811openalex-snapshot
The dynamic nature of autophagy in cancer
inferredcites →https://doi.org/10.1101/gad.2016111openalex-snapshot
Pancreatic cancers require autophagy for tumor growth
inferredcites →https://doi.org/10.1101/gad.2016311openalex-snapshot
Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis
inferredcites →https://doi.org/10.1101/gad.219642.113openalex-snapshot
Autophagy suppresses progression of K-ras-induced lung tumors to oncocytomas and maintains lipid homeostasis
inferredcites →https://doi.org/10.1101/gad.228122.113openalex-snapshot
Exploiting the bad eating habits of Ras-driven cancers
inferredcites →https://doi.org/10.1101/gad.283416.116openalex-snapshot
Autophagy provides metabolic substrates to maintain energy charge and nucleotide pools in Ras-driven lung cancer cells
inferredcites →https://doi.org/10.1101/gad.325514.119openalex-snapshot
Role of tumor and host autophagy in cancer metabolism
inferredcites →https://doi.org/10.1101/gad.348523.121openalex-snapshot
The biological underpinnings of therapeutic resistance in pancreatic cancer
inferredcites →https://doi.org/10.1126/sciadv.1600200openalex-snapshot
Fundamentals of cancer metabolism
inferredcites →https://doi.org/10.1126/science.aaw5473openalex-snapshot
Metabolic reprogramming and cancer progression
inferredcites →https://doi.org/10.1155/2018/6419805openalex-snapshot
Metabolic Alterations in a Slow‐Paced Model of Pancreatic Cancer‐Induced Wasting
inferredcites →https://doi.org/10.1158/0008-5472.can-08-2320openalex-snapshot
Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function
inferredcites →https://doi.org/10.1158/0008-5472.can-14-0155openalex-snapshot
Projecting Cancer Incidence and Deaths to 2030: The Unexpected Burden of Thyroid, Liver, and Pancreas Cancers in the United States
inferredcites →https://doi.org/10.1158/1078-0432.ccr-04-1785openalex-snapshot
Intrinsic Chemoresistance to Gemcitabine Is Associated with Decreased Expression of BNIP3 in Pancreatic Cancer
inferredcites →https://doi.org/10.1158/1078-0432.ccr-19-4042openalex-snapshot
A Randomized Phase II Preoperative Study of Autophagy Inhibition with High-Dose Hydroxychloroquine and Gemcitabine/Nab-Paclitaxel in Pancreatic Cancer Patients
inferredcites →https://doi.org/10.1158/2159-8290.cd-13-0397openalex-snapshot
Autophagy Sustains Mitochondrial Glutamine Metabolism and Growth of Braf V600E–Driven Lung Tumors
inferredcites →https://doi.org/10.1158/2159-8290.cd-14-0196openalex-snapshot
Targeting Mitochondrial Metabolism by Inhibiting Autophagy in BRAF-Driven Cancers
inferredcites →https://doi.org/10.1158/2159-8290.cd-14-0362openalex-snapshot
Autophagy Is Critical for Pancreatic Tumor Growth and Progression in Tumors with p53 Alterations
inferredcites →https://doi.org/10.1158/2159-8290.cd-15-0671openalex-snapshot
Pancreatic Cancer Metabolism: Breaking It Down to Build It Back Up
inferredcites →https://doi.org/10.1158/2159-8290.cd-17-0952openalex-snapshot
Autophagy Sustains Pancreatic Cancer Growth through Both Cell-Autonomous and Nonautonomous Mechanisms
inferredcites →https://doi.org/10.1158/2159-8290.cd-19-0292openalex-snapshot
Targeting Autophagy in Cancer: Recent Advances and Future Directions
inferredcites →https://doi.org/10.1159/000346388openalex-snapshot
Autophagy and Viruses: Adversaries or Allies?
inferredcites →https://doi.org/10.1159/000373980openalex-snapshot
Repertoires of Autophagy in the Pathogenesis of Ocular Diseases
inferredcites →https://doi.org/10.1186/s12885-018-4610-4openalex-snapshot
Trends in pancreatic adenocarcinoma incidence and mortality in the United States in the last four decades; a SEER-based study
inferredcites →https://doi.org/10.1186/s12943-015-0449-3openalex-snapshot
Blockade of autophagy reduces pancreatic cancer stem cell activity and potentiates the tumoricidal effect of gemcitabine
inferredcites →https://doi.org/10.1186/s12943-020-01169-7openalex-snapshot
Metabolism of pancreatic cancer: paving the way to better anticancer strategies
inferredcites →https://doi.org/10.1245/s10434-015-4566-4openalex-snapshot
Safety and Biologic Response of Pre-operative Autophagy Inhibition in Combination with Gemcitabine in Patients with Pancreatic Adenocarcinoma
inferredcites →https://doi.org/10.1503/cmaj.201450openalex-snapshot
Advances in the management of pancreatic ductal adenocarcinoma
inferredcites →https://doi.org/10.15252/embj.201490784openalex-snapshot
Autophagy in malignant transformation and cancer progression
inferredcites →https://doi.org/10.15252/emmm.201404318openalex-snapshot
Defects in mitophagy promote redox‐driven metabolic syndrome in the absence of TP53INP1
inferredcites →https://doi.org/10.15252/emmm.201505948openalex-snapshot
Pancreatic cancer microenvironment, to target or not to target?
inferredcites →https://doi.org/10.1634/theoncologist.2014-0086openalex-snapshot
Phase II and Pharmacodynamic Study of Autophagy Inhibition Using Hydroxychloroquine in Patients With Metastatic Pancreatic Adenocarcinoma
inferredcites →https://doi.org/10.3390/biomedicines8080270openalex-snapshot
Mitochondrial Metabolism in PDAC: From Better Knowledge to New Targeting Strategies
inferredcites →https://doi.org/10.3390/cancers12103076openalex-snapshot
The Tumor Microenvironment of Pancreatic Cancer
inferredcites →https://doi.org/10.3390/cancers13102285openalex-snapshot
Perioperative Predictors of Early Recurrence for Resectable and Borderline-Resectable Pancreatic Cancer
inferredcites →https://doi.org/10.3390/cancers13174451openalex-snapshot
Genomic Heterogeneity of Pancreatic Ductal Adenocarcinoma and Its Clinical Impact
inferredcites →https://doi.org/10.3390/cancers14010071openalex-snapshot
Genetic Mutations of Pancreatic Cancer and Genetically Engineered Mouse Models
inferredcites →https://doi.org/10.3390/cells9041063openalex-snapshot
The Role of Autophagy in Pancreatic Cancer: From Bench to the Dark Bedside
inferredcites →https://doi.org/10.3390/ijms21093100openalex-snapshot
Redox Homeostasis and Metabolism in Cancer: A Complex Mechanism and Potential Targeted Therapeutics
inferredcites →https://doi.org/10.3390/ijms22041534openalex-snapshot
Targeting Redox Metabolism in Pancreatic Cancer
inferredcites →https://doi.org/10.3748/wjg.v24.i43.4846openalex-snapshot
Pancreatic cancer: A review of clinical diagnosis, epidemiology, treatment and outcomes
inferredcites →https://doi.org/10.4161/auto.29961openalex-snapshot
Inhibition of autophagy attenuates pancreatic cancer growth independent of TP53/TRP53 status
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Alterations in the CTRB2 gene and response to chemotherapy in pancreatic cancer
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Isoprenylated Coumarin Exhibits Anti-proliferative Effects in Pancreatic Cancer Cells Under Nutrient Starvation by Inhibiting Autophagy
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Recent Advances in Cellular Signaling Interplay between Redox Metabolism and Autophagy Modulation in Cancer: An Overview of Molecular Mechanisms and Therapeutic Interventions
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Therapeutic Aspects and Molecular Targets of Autophagy to Control Pancreatic Cancer Management
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Modulating Glycolysis to Improve Cancer Therapy
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