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High-Mobility Group Box 1 Disrupts Metabolic Function with Cigarette Smoke Exposure in a Ceramide-Dependent Manner
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/s00018-012-0917-5openalex-snapshot
A role for sphingolipids in the pathophysiology of obesity-induced inflammation
inferredcites →https://doi.org/10.1007/s00408-014-9561-1openalex-snapshot
Overexpression of RAGE Contributes to Cigarette Smoke-Induced Nitric Oxide Generation in COPD
inferredcites →https://doi.org/10.1007/s00432-011-1067-0openalex-snapshot
Receptor for advanced glycation end products (RAGE) partially mediates HMGB1-ERKs activation in clear cell renal cell carcinoma
inferredcites →https://doi.org/10.1007/s11010-014-1978-6openalex-snapshot
The role of HMGB1-RAGE axis in migration and invasion of hepatocellular carcinoma cell lines
inferredcites →https://doi.org/10.1016/j.cmet.2011.04.008openalex-snapshot
High-Mobility Group Box 1 Is Essential for Mitochondrial Quality Control
inferredcites →https://doi.org/10.1016/j.cmet.2015.06.007openalex-snapshot
Targeted Induction of Ceramide Degradation Leads to Improved Systemic Metabolism and Reduced Hepatic Steatosis
inferredcites →https://doi.org/10.1016/j.neulet.2006.10.063openalex-snapshot
Distribution and cellular localization of high mobility group box protein 1 (HMGB1) in the spinal cord of a transgenic mouse model of ALS
inferredcites →https://doi.org/10.1016/j.pharmthera.2013.11.001openalex-snapshot
An overview on HMGB1 inhibitors as potential therapeutic agents in HMGB1-related pathologies
inferredcites →https://doi.org/10.1016/j.plipres.2005.11.002openalex-snapshot
Ceramides in insulin resistance and lipotoxicity
inferredcites →https://doi.org/10.1016/s0140-6736(10)61388-8openalex-snapshot
Worldwide burden of disease from exposure to second-hand smoke: a retrospective analysis of data from 192 countries
inferredcites →https://doi.org/10.1038/onc.2010.261openalex-snapshot
HMGB1 release and redox regulates autophagy and apoptosis in cancer cells
inferredcites →https://doi.org/10.1038/onc.2012.631openalex-snapshot
The HMGB1/RAGE inflammatory pathway promotes pancreatic tumor growth by regulating mitochondrial bioenergetics
inferredcites →https://doi.org/10.1042/bj20130807openalex-snapshot
Mitochondrial fission mediates ceramide-induced metabolic disruption in skeletal muscle
inferredcites →https://doi.org/10.1055/s-0030-1251503openalex-snapshot
The Role of HMGB1/RAGE in Inflammatory Cardiomyopathy
inferredcites →https://doi.org/10.1074/jbc.m112.359950openalex-snapshot
Fenretinide Prevents Lipid-induced Insulin Resistance by Blocking Ceramide Biosynthesis
inferredcites →https://doi.org/10.1089/ars.2010.3356openalex-snapshot
High-Mobility Group Box 1, Oxidative Stress, and Disease
inferredcites →https://doi.org/10.1093/nar/29.9.e45openalex-snapshot
A new mathematical model for relative quantification in real-time RT-PCR
inferredcites →https://doi.org/10.1097/shk.0000000000000468openalex-snapshot
Lipopolysaccharide Disrupts Mitochondrial Physiology in Skeletal Muscle via Disparate Effects on Sphingolipid Metabolism
inferredcites →https://doi.org/10.1097/shk.0b013e3181997173openalex-snapshot
HMGB1 IS MARKEDLY ELEVATED WITHIN 6 HOURS OF MECHANICAL TRAUMA IN HUMANS
inferredcites →https://doi.org/10.1111/j.1365-2796.2003.01302.xopenalex-snapshot
Extracellular role of HMGB1 in inflammation and sepsis
inferredcites →https://doi.org/10.1136/bmj.309.6959.901openalex-snapshot
Mortality in relation to smoking: 40 years' observations on male British doctors
inferredcites →https://doi.org/10.1136/bmj.38142.554479.aeopenalex-snapshot
Mortality in relation to smoking: 50 years' observations on male British doctors
inferredcites →https://doi.org/10.1146/annurev.bi.65.070196.003023openalex-snapshot
CROSSTALK BETWEEN NUCLEAR AND MITOCHONDRIAL GENOMES
inferredcites →https://doi.org/10.1152/ajpcell.00009.2010openalex-snapshot
Low anticoagulant heparin targets multiple sites of inflammation, suppresses heparin-induced thrombocytopenia, and inhibits interaction of RAGE with its ligands
inferredcites →https://doi.org/10.1152/ajpendo.00258.2014openalex-snapshot
Ceramides mediate cigarette smoke-induced metabolic disruption in mice
inferredcites →https://doi.org/10.1152/ajpheart.00043.2015openalex-snapshot
Cardiomyocyte mitochondrial respiration is reduced by receptor for advanced glycation end-product signaling in a ceramide-dependent manner
inferredcites →https://doi.org/10.1152/ajplung.00099.2012openalex-snapshot
RAGE signaling by alveolar macrophages influences tobacco smoke-induced inflammation
inferredcites →https://doi.org/10.1152/ajplung.00318.2007openalex-snapshot
RAGE: developmental expression and positive feedback regulation by Egr-1 during cigarette smoke exposure in pulmonary epithelial cells
inferredcites →https://doi.org/10.1155/2014/765784openalex-snapshot
Insulin Increases Ceramide Synthesis in Skeletal Muscle
inferredcites →https://doi.org/10.1155/2016/2763160openalex-snapshot
Oral Gingival Cell Cigarette Smoke Exposure Induces Muscle Cell Metabolic Disruption
inferredcites →https://doi.org/10.1172/jci43378openalex-snapshot
Lipid-induced insulin resistance mediated by the proinflammatory receptor TLR4 requires saturated fatty acid–induced ceramide biosynthesis in mice
inferredcites →https://doi.org/10.1172/jci57144openalex-snapshot
Ceramides as modulators of cellular and whole-body metabolism
inferredcites →https://doi.org/10.1186/1471-2261-14-165openalex-snapshot
Cigarette smoke increases cardiomyocyte ceramide accumulation and inhibits mitochondrial respiration
inferredcites →https://doi.org/10.2174/156652407783220750openalex-snapshot
RAGE as a Receptor of HMGB1 (Amphoterin): Roles in Health and Disease
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/ctm2.1679openalex-snapshot
Roles of glutamic pyruvate transaminase 2 in reprogramming of airway epithelial lipidomic and metabolomic profiles after smoking
inferred← citeshttps://doi.org/10.1002/dvdy.93openalex-snapshot
Ceramide: a novel inducer for neural tube defects
inferred← citeshttps://doi.org/10.1016/j.intimp.2017.10.007openalex-snapshot
HMGB1 mediates Aspergillus fumigatus-induced inflammatory response in alveolar macrophages of COPD mice via activating MyD88/NF-κB and syk/PI3K signalings
inferred← citeshttps://doi.org/10.1038/s41419-025-07563-7openalex-snapshot
Life-threatening risk factors contribute to the development of diseases with the highest mortality through the induction of regulated necrotic cell death
inferred← citeshttps://doi.org/10.1042/bcj20170736openalex-snapshot
Insulin selectively reduces mitochondrial uncoupling in brown adipose tissue in mice
inferred← citeshttps://doi.org/10.11606/d.60.2021.tde-29092021-103246openalex-snapshot
Perfil de esfingolipídios no câncer de cabeça e pescoço
inferred← citeshttps://doi.org/10.1249/mss.0000000000002441openalex-snapshot
Serum Acylglycerols Inversely Associate with Muscle Oxidative Capacity in Severe COPD
inferred← citeshttps://doi.org/10.2174/1573403x14666180913120015openalex-snapshot
Smoking and Endothelial Dysfunction
inferred← citeshttps://doi.org/10.3389/fcell.2020.00111openalex-snapshot
Downregulation of Lysosomal Acid Ceramidase Mediates HMGB1-Induced Migration and Proliferation of Mouse Coronary Arterial Myocytes
inferred← citeshttps://doi.org/10.3390/ijms19082247openalex-snapshot
β-Hydroxybutyrate Elicits Favorable Mitochondrial Changes in Skeletal Muscle
inferred← citeshttps://doi.org/10.3390/ijms20225598openalex-snapshot
Diesel Exhaust Particle Exposure Compromises Alveolar Macrophage Mitochondrial Bioenergetics
inferred← citeshttps://doi.org/10.3390/ijms27208993openalex-snapshot
Cachexia and Sarcopenia in Patients with Coexisting Chronic Obstructive Pulmonary Disease and Pulmonary Tuberculosis
inferred← citeshttps://doi.org/10.3390/metabo16060421openalex-snapshot
Ceramide-Driven Mechanisms in Pulmonary Fibrosis
inferred← citeshttps://doi.org/10.7150/ijms.53500openalex-snapshot
Concentrations of HMGB1 and Hsp70 of healthy subjects in upper and lower airway: Literature Review and Meta-analysis
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