On the public citation graph

Mitochondrial Dysfunction and Therapeutic Perspectives in Cardiovascular Diseases

https://doi.org/10.3390/ijms232416053
0 signed · 286 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

1 of the 141 references listed on this page is in the Retraction Watch database as retracted (export 2026-10-21): 10.1155/2022/9530007 (notice 2024-01-09).

Dataset join, best-effort; this page does not check the publisher record. Retraction Watch data via Crossref Labs, used with credit; OpenCitations Index (CC0) and Meta (CC BY 4.0); dataset doi:10.7910/DVN/WT8VOA (CC BY 4.0).

Is this your work? Sign in with ORCID and put your name on the claims that are yours.
Claim with ORCID
Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.
See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/adma.202006570openalex-snapshot
Biomimetic Design of Mitochondria‐Targeted Hybrid Nanozymes as Superoxide Scavengers
inferredcites →https://doi.org/10.1002/anie.201915826openalex-snapshot
Smart Design of Nanomaterials for Mitochondria‐Targeted Nanotherapeutics
inferredcites →https://doi.org/10.1002/emmm.201303672openalex-snapshot
Mitochondrially targeted ZFNs for selective degradation of pathogenic mitochondrial genomes bearing large‐scale deletions or point mutations
inferredcites →https://doi.org/10.1002/jimd.12281openalex-snapshot
Moving towards clinical trials for mitochondrial diseases
inferredcites →https://doi.org/10.1002/med.21732openalex-snapshot
Mitochondrial dysfunction in cardiovascular disease: Current status of translational research/clinical and therapeutic implications
inferredcites →https://doi.org/10.1002/rmb2.12230openalex-snapshot
Mitochondrial replacement therapy and assisted reproductive technology: A paradigm shift toward treatment of genetic diseases in gametes or in early embryos
inferredcites →https://doi.org/10.1007/978-981-10-6674-0_15openalex-snapshot
Significance of Mitochondria DNA Mutations in Diseases
inferredcites →https://doi.org/10.1007/s10565-020-09549-xopenalex-snapshot
New potentials of mitochondrial DNA editing
inferredcites →https://doi.org/10.1007/s11010-022-04570-6openalex-snapshot
METTL3-modified lncRNA-SNHG8 binds to PTBP1 to regulate ALAS2 expression to increase oxidative stress and promote myocardial infarction
inferredcites →https://doi.org/10.1007/s11427-020-1819-8openalex-snapshot
CRISPR/Cas9-mediated mutagenesis at microhomologous regions of human mitochondrial genome
inferredcites →https://doi.org/10.1007/s13238-020-00713-xopenalex-snapshot
BMAL1 regulates mitochondrial fission and mitophagy through mitochondrial protein BNIP3 and is critical in the development of dilated cardiomyopathy
inferredcites →https://doi.org/10.1007/s43657-022-00060-3openalex-snapshot
Mitochondria as the Essence of Yang Qi in the Human Body
inferredcites →https://doi.org/10.1016/j.atherosclerosis.2013.01.006openalex-snapshot
Changes of mitochondria in atherosclerosis: Possible determinant in the pathogenesis of the disease
inferredcites →https://doi.org/10.1016/j.atherosclerosis.2017.10.019openalex-snapshot
Network analysis reveals a causal role of mitochondrial gene activity in atherosclerotic lesion formation
inferredcites →https://doi.org/10.1016/j.bbadis.2016.09.003openalex-snapshot
Polydatin protects cardiomyocytes against myocardial infarction injury by activating Sirt3
inferredcites →https://doi.org/10.1016/j.ceb.2017.03.013openalex-snapshot
PINK1 and Parkin: emerging themes in mitochondrial homeostasis
inferredcites →https://doi.org/10.1016/j.cellsig.2007.04.009openalex-snapshot
Oxyl radicals, redox-sensitive signalling cascades and antioxidants
inferredcites →https://doi.org/10.1016/j.cmet.2017.11.002openalex-snapshot
NAD+ Intermediates: The Biology and Therapeutic Potential of NMN and NR
inferredcites →https://doi.org/10.1016/j.colsurfb.2017.11.048openalex-snapshot
Negatively charged polypeptide-peptide nanoparticles showing efficient drug delivery to the mitochondria
inferredcites →https://doi.org/10.1016/j.csbj.2021.06.003openalex-snapshot
Strategies for mitochondrial gene editing
inferredcites →https://doi.org/10.1016/j.devcel.2007.11.019openalex-snapshot
Chemical Inhibition of the Mitochondrial Division Dynamin Reveals Its Role in Bax/Bak-Dependent Mitochondrial Outer Membrane Permeabilization
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2016.04.001openalex-snapshot
Mitochondrial generation of superoxide and hydrogen peroxide as the source of mitochondrial redox signaling
inferredcites →https://doi.org/10.1016/j.heliyon.2020.e04643openalex-snapshot
Development of mitochondrial replacement therapy: A review
inferredcites →https://doi.org/10.1016/j.hfc.2021.07.003openalex-snapshot
Cardiovascular Involvement in mtDNA Disease
inferredcites →https://doi.org/10.1016/j.ihj.2018.01.031openalex-snapshot
Coenzyme Q10 in the treatment of heart failure: A systematic review of systematic reviews
inferredcites →https://doi.org/10.1016/j.ijcard.2013.01.278openalex-snapshot
Mechanisms of oxidative stress in human aortic aneurysms — Association with clinical risk factors for atherosclerosis and disease severity
inferredcites →https://doi.org/10.1016/j.jacc.2019.03.009openalex-snapshot
2019 ACC/AHA Guideline on the Primary Prevention of Cardiovascular Disease: Executive Summary
inferredcites →https://doi.org/10.1016/j.mito.2018.06.002openalex-snapshot
Gene therapy for the mitochondrial genome: Purging mutations, pacifying ailments
inferredcites →https://doi.org/10.1016/j.omtn.2021.11.016openalex-snapshot
DdCBE mediates efficient and inheritable modifications in mouse mitochondrial genome
inferredcites →https://doi.org/10.1016/j.phrs.2021.106038openalex-snapshot
Mitochondrial dysfunction and mitochondrial therapies in heart failure
inferredcites →https://doi.org/10.1016/j.phrs.2022.106366openalex-snapshot
Iron deficiency promotes aortic media degeneration by activating endoplasmic reticulum stress-mediated IRE1 signaling pathway
inferredcites →https://doi.org/10.1016/j.redox.2019.101192openalex-snapshot
Nicotinamide mononucleotide (NMN) supplementation rescues cerebromicrovascular endothelial function and neurovascular coupling responses and improves cognitive function in aged mice
inferredcites →https://doi.org/10.1016/j.redox.2020.101660openalex-snapshot
Pleiotropic effects of mdivi-1 in altering mitochondrial dynamics, respiration, and autophagy in cardiomyocytes
inferredcites →https://doi.org/10.1016/j.tox.2022.153275openalex-snapshot
Ophiopogonin D′-induced mitophagy and mitochondrial damage are associated with dysregulation of the PINK1/Parkin signaling pathway in AC16 cells
inferredcites →https://doi.org/10.1016/j.xhgg.2021.100057openalex-snapshot
Identification of a common polymorphism in COQ8B acting as a modifier of thoracic aortic aneurysm severity
inferredcites →https://doi.org/10.1016/j.ymgme.2021.06.001openalex-snapshot
Pathogenic variants in MRPL44 cause infantile cardiomyopathy due to a mitochondrial translation defect
inferredcites →https://doi.org/10.1016/s0140-6736(18)32203-7openalex-snapshot
Global, regional, and national age-sex-specific mortality for 282 causes of death in 195 countries and territories, 1980–2017: a systematic analysis for the Global Burden of Disease Study 2017
inferredcites →https://doi.org/10.1021/acsami.9b10645openalex-snapshot
Cardioprotection of Tetrahedral DNA Nanostructures in Myocardial Ischemia-Reperfusion Injury
inferredcites →https://doi.org/10.1021/acsnano.7b00221openalex-snapshot
Injectable Fullerenol/Alginate Hydrogel for Suppression of Oxidative Stress Damage in Brown Adipose-Derived Stem Cells and Cardiac Repair
inferredcites →https://doi.org/10.1021/acssynbio.8b00411openalex-snapshot
Knock-In Strategy for Editing Human and Zebrafish Mitochondrial DNA Using Mito-CRISPR/Cas9 System
inferredcites →https://doi.org/10.1038/cdd.2012.81openalex-snapshot
The pathways of mitophagy for quality control and clearance of mitochondria
inferredcites →https://doi.org/10.1038/gt.2010.25openalex-snapshot
Organ-specific shifts in mtDNA heteroplasmy following systemic delivery of a mitochondria-targeted restriction endonuclease
inferredcites →https://doi.org/10.1038/gt.2011.196openalex-snapshot
Manipulation of mtDNA heteroplasmy in all striated muscles of newborn mice by AAV9-mediated delivery of a mitochondria-targeted restriction endonuclease
inferredcites →https://doi.org/10.1038/nature18303openalex-snapshot
Towards clinical application of pronuclear transfer to prevent mitochondrial DNA disease
inferredcites →https://doi.org/10.1038/nature20592openalex-snapshot
Mitochondrial replacement in human oocytes carrying pathogenic mitochondrial DNA mutations
inferredcites →https://doi.org/10.1038/ncomms15750openalex-snapshot
Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis
inferredcites →https://doi.org/10.1038/ncomms9245openalex-snapshot
Increased COUP-TFII expression in adult hearts induces mitochondrial dysfunction resulting in heart failure
inferredcites →https://doi.org/10.1038/nm.3261openalex-snapshot
Specific elimination of mutant mitochondrial genomes in patient-derived cells by mitoTALENs
inferredcites →https://doi.org/10.1038/s12276-019-0355-7openalex-snapshot
Mitochondrial dysfunction and oxidative stress in heart disease
inferredcites →https://doi.org/10.1038/s41401-020-00518-yopenalex-snapshot
Drp1-dependent mitochondrial fission in cardiovascular disease
inferredcites →https://doi.org/10.1038/s41401-020-0459-6openalex-snapshot
GJA1-20k attenuates Ang II-induced pathological cardiac hypertrophy by regulating gap junction formation and mitochondrial function
inferredcites →https://doi.org/10.1038/s41418-018-0086-7openalex-snapshot
Pathogenesis of cardiac ischemia reperfusion injury is associated with CK2α-disturbed mitochondrial homeostasis via suppression of FUNDC1-related mitophagy
inferredcites →https://doi.org/10.1038/s41419-019-1400-0openalex-snapshot
Altered mitochondrial quality control in Atg7-deficient VSMCs promotes enhanced apoptosis and is linked to unstable atherosclerotic plaque phenotype
inferredcites →https://doi.org/10.1038/s41420-022-01046-wopenalex-snapshot
Mitochondrial damage and activation of the cytosolic DNA sensor cGAS–STING pathway lead to cardiac pyroptosis and hypertrophy in diabetic cardiomyopathy mice
inferredcites →https://doi.org/10.1038/s41467-018-04131-wopenalex-snapshot
Linear mitochondrial DNA is rapidly degraded by components of the replication machinery
inferredcites →https://doi.org/10.1038/s41467-022-28358-wopenalex-snapshot
In vivo mitochondrial base editing via adeno-associated viral delivery to mouse post-mitotic tissue
inferredcites →https://doi.org/10.1038/s41556-018-0176-2openalex-snapshot
Mechanisms of mitophagy in cellular homeostasis, physiology and pathology
inferredcites →https://doi.org/10.1038/s41569-021-00610-8openalex-snapshot
Boosting mitochondrial metabolism with dietary supplements in heart failure
inferredcites →https://doi.org/10.1038/s41569-022-00703-yopenalex-snapshot
The role of mitochondrial fission in cardiovascular health and disease
inferredcites →https://doi.org/10.1038/s41586-020-2477-4openalex-snapshot
A bacterial cytidine deaminase toxin enables CRISPR-free mitochondrial base editing
inferredcites →https://doi.org/10.1038/s41591-019-0477-4openalex-snapshot
In support of mitochondrial replacement therapy
inferredcites →https://doi.org/10.1038/s41598-019-40419-7openalex-snapshot
Impaired mitochondrial oxidative phosphorylation capacity in epicardial adipose tissue is associated with decreased concentration of adiponectin and severity of coronary atherosclerosis
inferredcites →https://doi.org/10.1038/s41598-019-49666-0openalex-snapshot
Chronic Pressure Overload Results in Deficiency of Mitochondrial Membrane Transporter ABCB7 Which Contributes to Iron Overload, Mitochondrial Dysfunction, Metabolic Shift and Worsens Cardiac Function
inferredcites →https://doi.org/10.1038/srep03844openalex-snapshot
Therapeutic potential of somatic cell nuclear transfer for degenerative disease caused by mitochondrial DNA mutations
inferredcites →https://doi.org/10.1042/ebc20170099openalex-snapshot
Human diseases associated with defects in assembly of OXPHOS complexes
inferredcites →https://doi.org/10.1056/nejmra022567openalex-snapshot
Mitochondrial Respiratory-Chain Diseases
inferredcites →https://doi.org/10.1073/pnas.1002178107openalex-snapshot
NADPH oxidase 4 (Nox4) is a major source of oxidative stress in the failing heart
inferredcites →https://doi.org/10.1073/pnas.2203628119openalex-snapshot
Succinyl-CoA-based energy metabolism dysfunction in chronic heart failure
inferredcites →https://doi.org/10.1074/jbc.m304854200openalex-snapshot
Production of Reactive Oxygen Species by Mitochondria
inferredcites →https://doi.org/10.1080/07853890.2017.1417631openalex-snapshot
The role of mitochondrial dysfunction in cardiovascular disease: a brief review
inferredcites →https://doi.org/10.1080/19420889.2018.1467189openalex-snapshot
Autophagy receptor optineurin promotes autophagosome formation by potentiating LC3-II production and phagophore maturation
inferredcites →https://doi.org/10.1091/mbc.e12-10-0721openalex-snapshot
Fis1, Mff, MiD49, and MiD51 mediate Drp1 recruitment in mitochondrial fission
inferredcites →https://doi.org/10.1093/cvr/cvab248openalex-snapshot
MicroRNA-27b-3p down-regulates FGF1 and aggravates pathological cardiac remodelling
inferredcites →https://doi.org/10.1093/cvr/cvy150openalex-snapshot
Decreased mitochondrial respiration in aneurysmal aortas of Fibulin-4 mutant mice is linked to PGC1A regulation
inferredcites →https://doi.org/10.1093/eurheartj/ehu436openalex-snapshot
Mitochondrial injury and dysfunction in hypertension-induced cardiac damage
inferredcites →https://doi.org/10.1093/gerona/glaa297openalex-snapshot
Cardiovascular Health and Mitochondrial Function: Testing an Association
inferredcites →https://doi.org/10.1096/fj.201902550rrropenalex-snapshot
Aldehyde dehydrogenase 2 protects against abdominal aortic aneurysm formation by reducing reactive oxygen species, vascular inflammation, and apoptosis of vascular smooth muscle cells
inferredcites →https://doi.org/10.1097/hco.0000000000000383openalex-snapshot
Mitochondrial DNA mutations and cardiovascular disease
inferredcites →https://doi.org/10.1111/acel.12461openalex-snapshot
Nicotinamide mononucleotide supplementation reverses vascular dysfunction and oxidative stress with aging in mice
inferredcites →https://doi.org/10.1111/acel.13329openalex-snapshot
NAD + supplementation prevents STING‐induced senescence in ataxia telangiectasia by improving mitophagy
inferredcites →https://doi.org/10.1111/joim.13055openalex-snapshot
DNA‐editing enzymes as potential treatments for heteroplasmic mtDNA diseases
inferredcites →https://doi.org/10.1111/jpi.12661openalex-snapshot
Melatonin protects against thoracic aortic aneurysm and dissection through SIRT1‐dependent regulation of oxidative stress and vascular smooth muscle cell loss
inferredcites →https://doi.org/10.1113/jphysiol.2013.268680openalex-snapshot
Mitochondria‐targeted antioxidant (MitoQ) ameliorates age‐related arterial endothelial dysfunction in mice
inferredcites →https://doi.org/10.1155/2018/5835072openalex-snapshot
Reactive Oxygen Species from NADPH Oxidase and Mitochondria Participate in the Proliferation of Aortic Smooth Muscle Cells from a Model of Metabolic Syndrome
inferredcites →https://doi.org/10.1155/2018/7293861openalex-snapshot
SIRT3: A New Regulator of Cardiovascular Diseases
inferredcites →https://doi.org/10.1155/2021/5545261openalex-snapshot
Ndufs1 Deficiency Aggravates the Mitochondrial Membrane Potential Dysfunction in Pressure Overload‐Induced Myocardial Hypertrophy
inferredcites →https://doi.org/10.1155/2021/9972413openalex-snapshot
BTK Promotes Atherosclerosis by Regulating Oxidative Stress, Mitochondrial Injury, and ER Stress of Macrophages
inferredcites →https://doi.org/10.1155/2022/7906091openalex-snapshot
Reactive Oxygen Species and Oxidative Stress in Vascular‐Related Diseases
inferredcites →https://doi.org/10.1155/2022/9530007openalex-snapshot
[Retracted] Mitochondrial Dysfunction and Cardiovascular Disease: Pathophysiology and Emerging Therapies
inferredcites →https://doi.org/10.1159/000443082openalex-snapshot
Ursodeoxycholic Acid Attenuates Acute Aortic Dissection Formation in Angiotensin II-Infused Apolipoprotein E-Deficient Mice Associated with Reduced ROS and Increased Nrf2 Levels
inferredcites →https://doi.org/10.1161/01.res.0000258450.44413.96openalex-snapshot
Mitochondrial Dysfunction in Atherosclerosis
inferredcites →https://doi.org/10.1161/atvbaha.117.310042openalex-snapshot
Mitochondrial Respiration Is Reduced in Atherosclerosis, Promoting Necrotic Core Formation and Reducing Relative Fibrous Cap Thickness
inferredcites →https://doi.org/10.1161/cir.0000000000000677openalex-snapshot
2019 ACC/AHA Guideline on the Primary Prevention of Cardiovascular Disease: Executive Summary: A Report of the American College of Cardiology/American Heart Association Task Force on Clinical Practice Guidelines
inferredcites →https://doi.org/10.1161/circresaha.110.232306openalex-snapshot
Mitochondrial Oxidative Stress Mediates Angiotensin II–Induced Cardiac Hypertrophy and Gαq Overexpression–Induced Heart Failure
inferredcites →https://doi.org/10.1161/circresaha.111.243964openalex-snapshot
PGC-1β Deficiency Accelerates the Transition to Heart Failure in Pressure Overload Hypertrophy
inferredcites →https://doi.org/10.1161/circresaha.112.279760openalex-snapshot
Increased Oxidative Stress in the Nucleus Caused by Nox4 Mediates Oxidation of HDAC4 and Cardiac Hypertrophy
inferredcites →https://doi.org/10.1161/circresaha.114.302153openalex-snapshot
Macrophage Mitochondrial Oxidative Stress Promotes Atherosclerosis and Nuclear Factor-κB–Mediated Inflammation in Macrophages
inferredcites →https://doi.org/10.1161/circresaha.117.311082openalex-snapshot
Autophagy and Mitophagy in Cardiovascular Disease
inferredcites →https://doi.org/10.1161/circresaha.117.312317openalex-snapshot
AMPKα2 Protects Against the Development of Heart Failure by Enhancing Mitophagy via PINK1 Phosphorylation
inferredcites →https://doi.org/10.1161/circresaha.118.314607openalex-snapshot
Mitophagy Is Essential for Maintaining Cardiac Function During High Fat Diet-Induced Diabetic Cardiomyopathy
inferredcites →https://doi.org/10.1161/circresaha.121.318241openalex-snapshot
Cardiac Energy Metabolism in Heart Failure
inferredcites →https://doi.org/10.1161/circresaha.121.319377openalex-snapshot
Alternative Mitophagy Protects the Heart Against Obesity-Associated Cardiomyopathy
inferredcites →https://doi.org/10.1161/circulationaha.105.607168openalex-snapshot
Deletion of p47 phox Attenuates Angiotensin II–Induced Abdominal Aortic Aneurysm Formation in Apolipoprotein E–Deficient Mice
inferredcites →https://doi.org/10.1161/circulationaha.118.036259openalex-snapshot
Mitochondrial Dysfunction in Heart Failure With Preserved Ejection Fraction
inferredcites →https://doi.org/10.1161/circulationaha.120.046528openalex-snapshot
Single-Cell Transcriptome Analysis Reveals Dynamic Cell Populations and Differential Gene Expression Patterns in Control and Aneurysmal Human Aortic Tissue
inferredcites →https://doi.org/10.1161/circulationaha.121.053575openalex-snapshot
Altered Cardiac Energetics and Mitochondrial Dysfunction in Hypertrophic Cardiomyopathy
inferredcites →https://doi.org/10.1172/jci138538openalex-snapshot
Boosting NAD level suppresses inflammatory activation of PBMCs in heart failure
inferredcites →https://doi.org/10.1186/s12881-020-01045-7openalex-snapshot
Associations of mitochondrial DNA 3777–4679 region mutations with maternally inherited essential hypertensive subjects in China
inferredcites →https://doi.org/10.1186/s12920-018-0408-0openalex-snapshot
Mitochondrial DNA 7908–8816 region mutations in maternally inherited essential hypertensive subjects in China
inferredcites →https://doi.org/10.1186/s12967-022-03642-xopenalex-snapshot
Omentin1 ameliorates myocardial ischemia-induced heart failure via SIRT3/FOXO3a-dependent mitochondrial dynamical homeostasis and mitophagy
inferredcites →https://doi.org/10.1186/s40659-018-0182-7openalex-snapshot
Increased ROS production and DNA damage in monocytes are biomarkers of aging and atherosclerosis
inferredcites →https://doi.org/10.1517/14728222.2016.1101068openalex-snapshot
Targeting autophagy and mitophagy for mitochondrial diseases treatment
inferredcites →https://doi.org/10.15252/emmm.201708084openalex-snapshot
mitoTev‐TALE: a monomeric DNA editing enzyme to reduce mutant mitochondrial DNA levels
inferredcites →https://doi.org/10.2174/156720205774962656openalex-snapshot
The Emerging Role of Coenzyme Q-10 in Aging, Neurodegeneration, Cardiovascular Disease, Cancer and Diabetes Mellitus
inferredcites →https://doi.org/10.2174/1573399818666220429101336openalex-snapshot
Coenzyme Q10 for Diabetes and Cardiovascular Disease: Useful or Useless?
inferredcites →https://doi.org/10.2217/nnm-2020-0266openalex-snapshot
Mitochondrial Responses to Organelle-Specific Drug Delivering Nanoparticles Composed of Polypeptide and Peptide Complexes
inferredcites →https://doi.org/10.3389/fcell.2021.628991openalex-snapshot
From Flies to Men: ROS and the NADPH Oxidase in Phagocytes
inferredcites →https://doi.org/10.3389/fcvm.2021.675647openalex-snapshot
Sirt3 Protects Against Thoracic Aortic Dissection Formation by Reducing Reactive Oxygen Species, Vascular Inflammation, and Apoptosis of Smooth Muscle Cells
inferredcites →https://doi.org/10.3389/fgene.2021.627050openalex-snapshot
Adapting CRISPR/Cas9 System for Targeting Mitochondrial Genome
inferredcites →https://doi.org/10.3389/fphar.2019.01404openalex-snapshot
The Role of Myocardial Mitochondrial Quality Control in Heart Failure
inferredcites →https://doi.org/10.3389/fphar.2020.604700openalex-snapshot
Vitamin D Attenuates Ischemia/Reperfusion-Induced Cardiac Injury by Reducing Mitochondrial Fission and Mitophagy
inferredcites →https://doi.org/10.3389/fphys.2020.565751openalex-snapshot
Berberine Promotes Cardiac Function by Upregulating PINK1/Parkin-Mediated Mitophagy in Heart Failure
inferredcites →https://doi.org/10.3390/antiox10111824openalex-snapshot
Mitochondrial Management of Reactive Oxygen Species
inferredcites →https://doi.org/10.3390/antiox9040341openalex-snapshot
Coenzyme Q10: Clinical Applications in Cardiovascular Diseases
inferredcites →https://doi.org/10.3390/cells10020443openalex-snapshot
Autophagy and Mitophagy as Essential Components of Atherosclerosis
inferredcites →https://doi.org/10.3390/cells7120235openalex-snapshot
SIRT-3 Modulation by Resveratrol Improves Mitochondrial Oxidative Phosphorylation in Diabetic Heart through Deacetylation of TFAM
inferredcites →https://doi.org/10.3390/ijms21217870openalex-snapshot
Coenzyme Q10 Supplementation for the Reduction of Oxidative Stress: Clinical Implications in the Treatment of Chronic Diseases
inferredcites →https://doi.org/10.3390/ijms22020551openalex-snapshot
Mitochondrial DNA Replacement Techniques to Prevent Human Mitochondrial Diseases
inferredcites →https://doi.org/10.3390/ijms22083903openalex-snapshot
Mitophagy in Human Diseases
inferredcites →https://doi.org/10.3390/ijms23020952openalex-snapshot
The Role of Mitochondrial DNA Mutations in Cardiovascular Diseases
inferredcites →https://doi.org/10.3390/jcm9051266openalex-snapshot
Clinical Evidence for Q10 Coenzyme Supplementation in Heart Failure: From Energetics to Functional Improvement
inferredcites →https://doi.org/10.3390/life11020165openalex-snapshot
Disturbance of Mitochondrial Dynamics and Mitochondrial Therapies in Atherosclerosis
inferredcites →https://doi.org/10.3390/nu13041165openalex-snapshot
Functional Food and Bioactive Compounds on the Modulation of the Functionality of HDL-C: A Narrative Review
inferredcites →https://doi.org/10.3390/nu14132649openalex-snapshot
Dietary Therapy in Prevention of Cardiovascular Disease (CVD)—Tradition or Modernity? A Review of the Latest Approaches to Nutrition in CVD
inferredcites →https://doi.org/10.3892/mmr.2015.3709openalex-snapshot
A comprehensive analysis of differentially expressed genes and pathways in abdominal aortic aneurysm
inferredcites →https://doi.org/10.5551/jat.27292openalex-snapshot
Angiotensin Ⅱ Activates MCP-1 and Induces Cardiac Hypertrophy and Dysfunction via Toll-like Receptor 4
inferredcites →https://doi.org/10.5603/cj.a2019.0022openalex-snapshot
Effect of coenzyme Q10 in Europeans with chronic heart failure: A sub-group analysis of the Q-SYMBIO randomized double-blind trial
inferredcites →https://doi.org/10.7150/thno.45922openalex-snapshot
Mitochondrial Sirtuin 3: New emerging biological function and therapeutic target
inferredcites →https://doi.org/10.7150/thno.61875openalex-snapshot
Pulmonary circulation-mediated heart targeting for the prevention of heart failure by inhalation of intrinsically bioactive nanoparticles
inferredcites →https://doi.org/10.7554/elife.59828openalex-snapshot
Instability in NAD+ metabolism leads to impaired cardiac mitochondrial function and communication
inferredcites →https://doi.org/10.7554/elife.69310openalex-snapshot
Aorta smooth muscle-on-a-chip reveals impaired mitochondrial dynamics as a therapeutic target for aortic aneurysm in bicuspid aortic valve disease

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4647442openalex-extracted
Mrpl42 Attenuates Myocardial Ischemia/Reperfusion Injury by Regulating Mitochondrial Translation Function
inferred← citeshttps://doi.org/10.1002/adhm.71594openalex-snapshot
Mitochondria‐Targeted Nanotherapies in Aging Neurodegenerative Disorders: Emerging Prospects and Clinical Potential
inferred← citeshttps://doi.org/10.1002/bio.70312openalex-snapshot
Analysis of Active Components and NRF2 Activation in Olive Leaf Pressurized Water Extract
inferred← citeshttps://doi.org/10.1002/ddr.70150openalex-snapshot
Sestrin2 Regulates Mitochondrial Function and Autophagy via Nrf2/SIRT3 Signaling to Ameliorate Hypoxia/Reoxygenation‐Induced Rat's Cardiomyocyte Injury
inferred← citeshttps://doi.org/10.1002/inmd.20250005openalex-snapshot
Mitochondrial dysfunction: Related diseases, influencing factors, and detection
inferred← citeshttps://doi.org/10.1002/jbm.a.37668openalex-snapshot
Nanoparticle‐based therapeutic strategies for mitochondrial dysfunction in cardiovascular disease
inferred← citeshttps://doi.org/10.1002/mco2.70194openalex-snapshot
Molecular Hydrogen Therapy: Mechanisms, Delivery Methods, Preventive, and Therapeutic Application
inferred← citeshttps://doi.org/10.1002/mco2.70268openalex-snapshot
Oxidative Stress: Signaling Pathways, Biological Functions, and Disease
inferred← citeshttps://doi.org/10.1002/mco2.70326openalex-snapshot
Mitochondrial Dysfunction in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1002/mco2.70790openalex-snapshot
Targeting Mitochondria in Aging‐Related Diseases: Therapeutic Potential and Obstacles
inferred← citeshttps://doi.org/10.1002/path.6474openalex-snapshot
Dysregulated mitochondrial energy metabolism drives the progression of mucosal field effects to invasive bladder cancer
inferred← citeshttps://doi.org/10.1007/s00011-025-01994-wopenalex-snapshot
Mitochondrial dysfunction is a major cause of thromboinflammation and inflammatory cell death in critical illnesses
inferred← citeshttps://doi.org/10.1007/s00210-025-03897-3openalex-snapshot
Natural and synthetic potential drug leads for rheumatoid arthritis probing innovative target: mitochondrial dysfunction and NLRP3 inflammasome activation
inferred← citeshttps://doi.org/10.1007/s00415-025-13101-4openalex-snapshot
Advances in research on mitochondrial dysfunction in neurodegenerative diseases
inferred← citeshttps://doi.org/10.1007/s10528-025-11104-4openalex-snapshot
Exploring the Interconnections Between Mitochondrial Dysfunction and Polycystic Ovary Syndrome: A Comprehensive Integrated Analysis
inferred← citeshttps://doi.org/10.1007/s10616-025-00762-2openalex-snapshot
Naringin regulates the cGAS-STING pathway to improve mitochondrial dysfunction and ferroptosis after myocardial ischemia–reperfusion injury
inferred← citeshttps://doi.org/10.1007/s11010-024-05074-1openalex-snapshot
Mitochondrial complex-1 as a therapeutic target for cardiac diseases
inferred← citeshttps://doi.org/10.1007/s11626-024-01000-3openalex-snapshot
OPA3 inhibits the cGAS-STING pathway mediated by mtDNA stress to promote colorectal cancer progression
inferred← citeshttps://doi.org/10.1007/s11626-026-01156-0openalex-snapshot
A preliminary study on the mechanism of stromal interaction molecule 1 (STIM1) involvement in Adriamycin-induced podocyte injury
inferred← citeshttps://doi.org/10.1007/s12012-023-09800-xopenalex-snapshot
Mitochondrial Dysfunction in Cardiotoxicity Induced by BCR-ABL1 Tyrosine Kinase Inhibitors -Underlying Mechanisms, Detection, Potential Therapies
inferred← citeshttps://doi.org/10.1007/s12265-026-10793-5openalex-snapshot
Targeting Ferroptosis and Pyroptosis in Cardiovascular Diseases: Mechanisms and Therapeutic Implication
inferred← citeshttps://doi.org/10.1007/s13105-024-01039-6openalex-snapshot
Role of ROS and autophagy in the pathological process of atherosclerosis
inferred← citeshttps://doi.org/10.1016/b978-0-443-36312-2.00011-5openalex-snapshot
Metabolic disorders associated with mitochondrial dysfunction
inferred← citeshttps://doi.org/10.1016/j.addr.2024.115195openalex-snapshot
Unlocking the Mitochondria for Nanomedicine-based Treatments: Overcoming Biological Barriers, Improving Designs, and Selecting Verification Techniques
inferred← citeshttps://doi.org/10.1016/j.anireprosci.2025.107797openalex-snapshot
Nicotinamide mononucleotide enhances porcine sperm quality by activating the SIRT3-SOD2/ROS pathway and promoting oxidative phosphorylation
inferred← citeshttps://doi.org/10.1016/j.arr.2023.101917openalex-snapshot
The role of mitochondrial quality surveillance in skin aging: Focus on mitochondrial dynamics, biogenesis and mitophagy
inferred← citeshttps://doi.org/10.1016/j.arr.2025.102920openalex-snapshot
Targeting mitochondrial microRNAs in cardiovascular pathologies: A new frontier in precision cardiology
inferred← citeshttps://doi.org/10.1016/j.arr.2026.103135openalex-snapshot
Nanomedicine guiding mitochondrial function in the treatment of cardiovascular diseases: Mechanisms, latest developments, and clinical translation
inferred← citeshttps://doi.org/10.1016/j.arr.2026.103137openalex-snapshot
The multi-target cardiac protection mechanism of butylphthalide and AI-driven precision medicine: From molecular basis to clinical translation
inferred← citeshttps://doi.org/10.1016/j.bbrc.2026.154574openalex-snapshot
Cardiac-specific Lmpt knockdown induces heart dysfunction via mitochondrial quality decline and metabolic disturbance in Drosophila
inferred← citeshttps://doi.org/10.1016/j.bcp.2025.117577openalex-snapshot
Hydrogen sulfide revisited: a stealthy ally in the unseen war against cardiovascular disease
inferred← citeshttps://doi.org/10.1016/j.biopha.2024.116673openalex-snapshot
The interplay between mitochondrial dysfunction and NLRP3 inflammasome in multiple sclerosis: Therapeutic implications and animal model studies
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2023.115472openalex-snapshot
Inhibition of mitochondrial calcium uptake by Ru360 enhances the effect of 1800 MHz radio-frequency electromagnetic fields on DNA damage
inferred← citeshttps://doi.org/10.1016/j.ejmech.2024.116471openalex-snapshot
Recent advances of honokiol:pharmacological activities, manmade derivatives and structure-activity relationship
inferred← citeshttps://doi.org/10.1016/j.exer.2025.110632openalex-snapshot
Imbalanced mitochondrial homeostasis in ocular diseases: unique pathogenesis and targeted therapy
inferred← citeshttps://doi.org/10.1016/j.fitote.2025.106627openalex-snapshot
Ganoderma lucidum extract reduces skin aging by reducing mitochondrial stress and controlling mitochondrial numbers
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2025.12.019openalex-snapshot
Mitochondrial uncoupling sensitizes gastric cancer cells to elesclomol-induced cuproptosis via FDX1/DLAT upregulation
inferred← citeshttps://doi.org/10.1016/j.gendis.2025.101856openalex-snapshot
Mitochondrial transplantation in lung diseases: From mechanisms to application prospects
inferred← citeshttps://doi.org/10.1016/j.heliyon.2023.e22273openalex-snapshot
Cardiac damage following renal ischemia reperfusion injury increased with excessive consumption of high fat diet but enhanced the cardiac resistance to reperfusion stress in rat
inferred← citeshttps://doi.org/10.1016/j.intimp.2024.112616openalex-snapshot
Spotlight on necroptosis: Role in pathogenesis and therapeutic potential of intervertebral disc degeneration
inferred← citeshttps://doi.org/10.1016/j.intimp.2024.113722openalex-snapshot
Mitochondria dysfunction: A trigger for cardiovascular diseases in systemic lupus erythematosus
inferred← citeshttps://doi.org/10.1016/j.intimp.2025.115529openalex-snapshot
Mitochondrial targeting of coenzyme Q10 in inflammatory bowel disease: mechanisms, challenges, and prospects for clinical application
inferred← citeshttps://doi.org/10.1016/j.intimp.2025.116108openalex-snapshot
Anemoside B4 ameliorates septic cardiomyopathy by activating SRC-mediated PI3K/AKT/mTOR pathway to alleviate mitochondrial dysfunction and cardiomyocyte senescence
inferred← citeshttps://doi.org/10.1016/j.intimp.2026.116251openalex-snapshot
Role of reactive oxygen species in polycystic ovary syndrome: signalling pathways, mechanisms, and traditional Chinese medicine treatment strategies
inferred← citeshttps://doi.org/10.1016/j.intimp.2026.116375openalex-snapshot
Corrigendum “Role of reactive oxygen species in polycystic ovary syndrome: Signalling pathways, mechanisms, and traditional Chinese medicine treatment strategies” [Int. Immunopharmacol. 173 (2026) 116251]
inferred← citeshttps://doi.org/10.1016/j.jare.2025.07.021openalex-snapshot
Mitochondrial cardiovascular diseases: molecular mechanisms, multi-omics exploration and therapeutic strategies
inferred← citeshttps://doi.org/10.1016/j.jare.2025.09.046openalex-snapshot
Cross-regulatory mechanisms linking ferroptosis, epigenetics, and circadian rhythm to mitochondrial quality control in diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.jare.2026.02.023openalex-snapshot
Mitochondrial oxidative stress to PANoptosis: Current evidence and therapeutic implications for neurological diseases
inferred← citeshttps://doi.org/10.1016/j.jconrel.2026.115050openalex-snapshot
Nanomedicines in ischemic cardiovascular and cerebrovascular diseases: Theranostic technologies based on reactive oxygen species
inferred← citeshttps://doi.org/10.1016/j.jgr.2025.08.008openalex-snapshot
The role of ginseng and ginsenosides in ROS and cardiovascular disease
inferred← citeshttps://doi.org/10.1016/j.jot.2026.101089openalex-snapshot
Dysregulation of the glycolysis–mitochondrial metabolism axis in osteoarthritis: mechanisms and therapeutic implications
inferred← citeshttps://doi.org/10.1016/j.jpet.2025.103729openalex-snapshot
Targeting mitochondrial RNA polymerase for triple-negative breast cancer
inferred← citeshttps://doi.org/10.1016/j.jsbmb.2024.106595openalex-snapshot
Cross-sex hormonal replacement: Some effects over mitochondria
inferred← citeshttps://doi.org/10.1016/j.jtemb.2024.127553openalex-snapshot
Nephropathy induced by cisplatin results from mitochondrial disruption, impaired energy metabolism, altered expression of renal transporters, and accumulation of urinary toxins
inferred← citeshttps://doi.org/10.1016/j.lfs.2023.122257openalex-snapshot
Mitochondrial dysfunction and neurological disorders: A narrative review and treatment overview
inferred← citeshttps://doi.org/10.1016/j.mad.2024.112009openalex-snapshot
Role of chemokines in aging and age-related diseases
inferred← citeshttps://doi.org/10.1016/j.mito.2024.101957openalex-snapshot
The multifaceted effects of mitochondria in kidney diseases
inferred← citeshttps://doi.org/10.1016/j.mrrev.2026.108587openalex-snapshot
Mitochondrial DNA: Bridging cellular senescence and chronic inflammation in aging and beyond
inferred← citeshttps://doi.org/10.1016/j.phrs.2024.107164openalex-snapshot
Mitofilin in cardiovascular diseases: Insights into the pathogenesis and potential pharmacological interventions
inferred← citeshttps://doi.org/10.1016/j.phrs.2026.108106openalex-snapshot
Mitochondrial semi-autonomous inheritance in cardiovascular pathology: Emerging mechanisms and therapeutic opportunities
inferred← citeshttps://doi.org/10.1016/j.phymed.2023.154955openalex-snapshot
Xinmaikang-mediated mitophagy attenuates atherosclerosis via the PINK1/Parkin signaling pathway
inferred← citeshttps://doi.org/10.1016/j.phymed.2025.156555openalex-snapshot
Natural products alleviate atrial fibrillation by modulating mitochondrial quality control
inferred← citeshttps://doi.org/10.1016/j.phymed.2025.157466openalex-snapshot
Galangin alleviates vascular aging and endothelial cell senescence by targeting SMAD3 to initiate mitophagy
inferred← citeshttps://doi.org/10.1016/j.phymed.2026.158145openalex-snapshot
Allosteric activation of pyruvate carboxylase by Dracoflavan B2 enhances endothelial energy metabolism to attenuate myocardial infarction
inferred← citeshttps://doi.org/10.1016/j.redox.2025.103787openalex-snapshot
Decoding Parkinson's Disease: The interplay of cell death pathways, oxidative stress, and therapeutic innovations
inferred← citeshttps://doi.org/10.1016/j.redox.2026.104190openalex-snapshot
Protein glycoxidation in neuropsychiatric disorders—from basic research to clinical practice
inferred← citeshttps://doi.org/10.1016/j.tox.2024.153880openalex-snapshot
Lycopene alleviates Deoxynivalenol-induced toxicity in Porcine intestinal epithelial cells by mediating mitochondrial function
inferred← citeshttps://doi.org/10.1016/j.tranon.2025.102355openalex-snapshot
Tubeimoside I induces mitophagy by activating the PINK1/Parkin/Mfn2 signaling pathway in acute myeloid leukemia cells
inferred← citeshttps://doi.org/10.1017/s0029665124007559openalex-snapshot
Exploring the association between sarcopenic obesity and cardiovascular risk: a summary of findings from longitudinal studies and potential mechanisms
inferred← citeshttps://doi.org/10.1021/acs.jafc.5c09133openalex-snapshot
From Molecular Structure to Food Safety: Mechanistic Analysis of Strobilurin-Induced Cardiotoxicity in Zebrafish and LC 50 -Based QSAR Modeling for Toxicity Prediction
inferred← citeshttps://doi.org/10.1021/acsami.5c18947openalex-snapshot
A Photosensitizer-Engineered Nanoplatform Exposed to NIR Laser for Mitochondria Targeting and Chemo/Photodynamic Synergistic Therapy against Glioma
inferred← citeshttps://doi.org/10.1038/s41401-024-01424-3openalex-snapshot
Nicotinamide mononucleotide protects septic hearts in mice via preventing cyclophilin F modification and lysosomal dysfunction
inferred← citeshttps://doi.org/10.1038/s41420-024-02159-0openalex-snapshot
Multifaceted mitochondrial as a novel therapeutic target in dry eye: insights and interventions
inferred← citeshttps://doi.org/10.1038/s41598-024-61495-4openalex-snapshot
Inflammatory corpuscle AIM2 facilitates macrophage foam cell formation by inhibiting cholesterol efflux protein ABCA1
inferred← citeshttps://doi.org/10.1038/s41598-026-46809-yopenalex-snapshot
PAK5 drives vascular remodeling in hypoxic pulmonary hypertension via Drp1-dependent mitochondrial midzone division
inferred← citeshttps://doi.org/10.1080/1061186x.2024.2309577openalex-snapshot
Insight into modulators of sphingosine-1-phosphate receptor and implications for cardiovascular therapeutics
inferred← citeshttps://doi.org/10.1080/10641963.2025.2503805openalex-snapshot
Anthocyanin attenuates pulmonary arterial hypertension and associated heart failure via improving mitochondrial function through Nrf2-dependent mechanism
inferred← citeshttps://doi.org/10.1080/14786419.2026.2686978openalex-snapshot
Propolis components from stingless bees collected in Gia Lai, Vietnam, and their NRF2 activation
inferred← citeshttps://doi.org/10.1080/17425255.2024.2398628openalex-snapshot
Underlying mechanisms and management strategies for regorafenib-induced toxicity in hepatocellular carcinoma
inferred← citeshttps://doi.org/10.1093/europace/euaf083openalex-snapshot
GSTP1 inhibits angiotensin II-induced atrial fibrillation by regulating ferroptosis
inferred← citeshttps://doi.org/10.1093/jbmr/zjag050openalex-snapshot
Associations of osteoporosis, sarcopenia, and osteosarcopenia with mortality: a comparative analysis for different sarcopenia definitions in the Manitoba and NHANES populations
inferred← citeshttps://doi.org/10.1097/md.0000000000041757openalex-snapshot
Causal role of mitochondrial proteins in aortic aneurysms: Evidence from Mendelian randomization, transcriptomic analysis, and experimental validation
inferred← citeshttps://doi.org/10.1097/ms9.0000000000003365openalex-snapshot
Innovative strategies for mitochondrial dysfunction in myeloproliferative neoplasms a step toward precision medicine
inferred← citeshttps://doi.org/10.1101/2025.09.04.671113openalex-snapshot
Sirtuin 1 Activation Mitigates Murine Vasculitis Severity by Promoting Autophagy and Mitophagy
inferred← citeshttps://doi.org/10.1111/cpr.70136openalex-snapshot
Pulmonary Vascular Endothelial Cells in Lung Diseases: Mechanisms, Therapeutic Strategies, and Future Directions
inferred← citeshttps://doi.org/10.1111/exd.70195openalex-snapshot
Cross Disease Single‐Cell Meta‐Analysis Deciphers Conserved Pan‐Inflammatory Circuits and Divergent Endothelial Rewiring in Inflammatory Skin Pathogenesis
inferred← citeshttps://doi.org/10.1111/jcmm.70959openalex-snapshot
Silencing of Cholesterol 25‐Hydroxylase Attenuates Lipopolysaccharide‐Induced Cardiomyocyte Damage In Vitro
inferred← citeshttps://doi.org/10.1161/circresaha.125.327991openalex-snapshot
Sirtuin 1 Activation Mitigates Murine Vasculitis Severity by Promoting Mitophagy
inferred← citeshttps://doi.org/10.1161/circresaha.126.328769openalex-snapshot
Shifting the Balance: Mitochondrial Heteroplasmy as a Driver of Cardiac Disease
inferred← citeshttps://doi.org/10.1177/10998004261439115openalex-snapshot
The Theoretical Intersection of Plant-Based Diets, Alzheimer’s, and Cardiovascular Disease
inferred← citeshttps://doi.org/10.1186/s11658-024-00669-4openalex-snapshot
Inter- and intracellular mitochondrial communication: signaling hubs in aging and age-related diseases
inferred← citeshttps://doi.org/10.1186/s12931-025-03468-3openalex-snapshot
Mitochondrial quality control mechanisms as molecular targets for impaired lung development: from fetuses to neonates
inferred← citeshttps://doi.org/10.1186/s13020-024-00955-5openalex-snapshot
Liguzinediol potentiates the metabolic remodeling by activating the AMPK/SIRT3 pathway and represses Caspase-3/GSDME-mediated pyroptosis to ameliorate cardiotoxicity
inferred← citeshttps://doi.org/10.1186/s43556-025-00284-5openalex-snapshot
Role of mitochondria in physiological activities, diseases, and therapy
inferred← citeshttps://doi.org/10.12677/acm.2026.1682888openalex-snapshot
Research Progress of Mitophagy in Stemness Maintenance of Mesenchymal Stem Cells and Cardiovascular Diseases
inferred← citeshttps://doi.org/10.1360/ssv-2025-0049openalex-snapshot
The mtDNA-cGAS-STING pathway: linking neuroinflammation and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1515/med-2026-1518openalex-snapshot
ID2 attenuates post-MI ventricular arrhythmias by targeting GATA4 suppression to preserve mitochondrial function and Na + /K + -ATPase activity
inferred← citeshttps://doi.org/10.1515/oncologie-2024-0596openalex-snapshot
Multiple roles of mitochondria in tumorigenesis and treatment: from mechanistic insights to emerging therapeutic strategies
inferred← citeshttps://doi.org/10.20517/and.2024.24openalex-snapshot
Age-related hearing loss: possible associations with Alzheimer’s disease
inferred← citeshttps://doi.org/10.21203/rs.3.rs-3913902/v1openalex-snapshot
Liguzinediol ameliorates doxorubicin-induced cardiotoxicity and potentiates the metabolic remodeling by activating the AMPK/SIRT3 pathway and represses Caspase-3/GSDME-mediated pyroptosis
inferred← citeshttps://doi.org/10.2147/jir.s503033openalex-snapshot
Metformin Ameliorates Ulcerative Colitis Through Inhibiting NLRP3 Inflammasome Activation
inferred← citeshttps://doi.org/10.2174/0115701611312293241220101556openalex-snapshot
Mitochondrial Dysfunction: Potential Therapy For Abdominal Aortic Aneurysms
inferred← citeshttps://doi.org/10.2174/011573403x375709250616134726openalex-snapshot
Mitochondrial-Derived Peptides as Therapeutics and Biomarkers for Combating Vascular Aging and Associated Cardiovascular Diseases
inferred← citeshttps://doi.org/10.2174/011573403x379197250417061904openalex-snapshot
Mitochondrial Dysfunction in Cardiac Diseases: Insights into Pathophysiology and Clinical Outcomes
inferred← citeshttps://doi.org/10.26442/00403660.2026.05.203610openalex-snapshot
Uric acid and mitochondrial dysfunction: a review
inferred← citeshttps://doi.org/10.26599/fshw.2022.9250140openalex-snapshot
Resveratrol combats chronic diseases through enhancing mitochondrial quality
inferred← citeshttps://doi.org/10.31083/j.fbs1601006openalex-snapshot
The Relationship between Mitochondrial Genome Mutations in Monocytes and the Development of Obesity and Coronary Heart Disease
inferred← citeshttps://doi.org/10.31435/ijitss.4(48).2025.4424openalex-snapshot
MITOCHONDRIAL DYSFUNCTION IN DISEASE: PATHOGENIC MECHANISMS AND NOVEL THERAPIES
inferred← citeshttps://doi.org/10.3389/fcvm.2025.1538202openalex-snapshot
The mitochondrial dysfunction, alongside the modifiable burden of traditional risk factors, drives the development of early-onset coronary artery disease
inferred← citeshttps://doi.org/10.3389/fendo.2023.1252141openalex-snapshot
BAM15 as a mitochondrial uncoupler: a promising therapeutic agent for diverse diseases
inferred← citeshttps://doi.org/10.3389/fendo.2024.1401531openalex-snapshot
Mitochondrial dysfunction and onset of type 2 diabetes along with its complications: a multi-omics Mendelian randomization and colocalization study
inferred← citeshttps://doi.org/10.3389/fendo.2025.1573499openalex-snapshot
Endoplasmic reticulum-mitochondria crosstalk: new mechanisms in the development of atherosclerosis
inferred← citeshttps://doi.org/10.3389/fendo.2025.1617305openalex-snapshot
Energy metabolism in cardiovascular diseases: unlocking the hidden powerhouse of cardiac pathophysiology
inferred← citeshttps://doi.org/10.3389/fendo.2025.1653567openalex-snapshot
Rejuvenating the failing heart: multidimensional insights into young blood-mediated anti-aging pathways
inferred← citeshttps://doi.org/10.3389/fimmu.2026.1771616openalex-snapshot
Mitochondrial dysfunction and immune microenvironment in gestational diabetes mellitus: insights from bioinformatics analysis and experimental validation
inferred← citeshttps://doi.org/10.3389/fmed.2024.1439182openalex-snapshot
Mitochondria: a breakthrough in combating rheumatoid arthritis
inferred← citeshttps://doi.org/10.3389/fphar.2024.1401961openalex-snapshot
Diabetes cardiomyopathy: targeted regulation of mitochondrial dysfunction and therapeutic potential of plant secondary metabolites
inferred← citeshttps://doi.org/10.3389/fphar.2024.1543291openalex-snapshot
Editorial: Revealing the unconventional mechanisms of mitochondria-targeting drugs in heart-related diseases
inferred← citeshttps://doi.org/10.3390/antiox13010121openalex-snapshot
Maintaining the Mitochondrial Quality Control System Was a Key Event of Tanshinone IIA against Deoxynivalenol-Induced Intestinal Toxicity
inferred← citeshttps://doi.org/10.3390/antiox13050507openalex-snapshot
Supplementing Boar Diet with Nicotinamide Mononucleotide Improves Sperm Quality Probably through the Activation of the SIRT3 Signaling Pathway
inferred← citeshttps://doi.org/10.3390/antiox13070870openalex-snapshot
The Impact of Weight Loss on Inflammation, Oxidative Stress, and Mitochondrial Function in Subjects with Obesity
inferred← citeshttps://doi.org/10.3390/antiox14070797openalex-snapshot
Antioxidant Intervention Against Microplastic Hazards
inferred← citeshttps://doi.org/10.3390/antiox15070791openalex-snapshot
The Fly Maggot Antioxidant Peptide (FMP) Alleviates Oxidative Damage in the Intestines of Weaned Piglets by Enhancing Mitochondrial Autophagy Through Activation of the Nrf2 Signaling Pathway
inferred← citeshttps://doi.org/10.3390/cells12121607openalex-snapshot
The Multi-Faceted Nature of Renalase for Mitochondrial Dysfunction Improvement in Cardiac Disease
inferred← citeshttps://doi.org/10.3390/ijms24032918openalex-snapshot
Genetics and Molecular Basis of Congenital Heart Defects in Down Syndrome: Role of Extracellular Matrix Regulation
inferred← citeshttps://doi.org/10.3390/ijms26083624openalex-snapshot
The Role of N6-Methyladenosine in Mitochondrial Dysfunction and Pathology
inferred← citeshttps://doi.org/10.3390/ijms26157650openalex-snapshot
Relationship Between Oxidative Stress and Cardiovascular Risk in Adolescents in Montenegro
inferred← citeshttps://doi.org/10.3390/ijms26178475openalex-snapshot
Mitochondrial Transport Proteins in Cardiovascular Diseases: Metabolic Gatekeepers, Pathogenic Mediators and Therapeutic Targets
inferred← citeshttps://doi.org/10.3390/ijms27083347openalex-snapshot
Coenzyme Q10 Deficiency and Elevated LEAK Mitochondrial Respiration as Potential Heart Failure Markers in Ebstein Anomaly
inferred← citeshttps://doi.org/10.3390/ijms27188385openalex-snapshot
Syringic Acid in Health and Disease: Mechanisms, Biological Activities, and Future Perspectives
inferred← citeshttps://doi.org/10.3390/jcm13061574openalex-snapshot
Cardiovascular Toxicity of Antineoplastic Treatments in Hematological Diseases: Focus on Molecular Mechanisms to Improve Therapeutic Management
inferred← citeshttps://doi.org/10.3390/metabo16050329openalex-snapshot
Regulation of Mitophagy by Low-Intensity Pulsed Ultrasound Attenuates Endothelial Dysfunction
inferred← citeshttps://doi.org/10.3390/molecules28166078openalex-snapshot
Nicotinamide Riboside, a Promising Vitamin B3 Derivative for Healthy Aging and Longevity: Current Research and Perspectives
inferred← citeshttps://doi.org/10.3390/molecules29112691openalex-snapshot
L-Theanine Prolongs the Lifespan by Activating Multiple Molecular Pathways in Ultraviolet C-Exposed Caenorhabditis elegans
inferred← citeshttps://doi.org/10.3390/molecules30193860openalex-snapshot
Mycotoxin Contamination: Occurrence, Biotransformation, Pathogenic Mechanisms, and Strategies for Nutritional Intervention
inferred← citeshttps://doi.org/10.3390/nu18010093openalex-snapshot
Nutritional Regulation of Cardiac Metabolism and Function: Molecular and Epigenetic Mechanisms and Their Role in Cardiovascular Disease Prevention
inferred← citeshttps://doi.org/10.36740/wlek202401119openalex-snapshot
The impact of mitochondrial dysfunction on the pathogenesis of atherosclerosis
inferred← citeshttps://doi.org/10.3724/abbs.2025153openalex-snapshot
Mitochondrial dysfunction in adipocyte differentiation: implications for obesity and metabolic syndrome intervention
inferred← citeshttps://doi.org/10.3923/tmr.2024.112.123openalex-snapshot
Mitophagy in the Pathogenesis of Obesity-Associated Cardiovascular Diseases: New Mechanistic and Therapeutic Insights
inferred← citeshttps://doi.org/10.4070/kcj.2025.0155openalex-snapshot
Role of CR6-Interacting Factor 1 (Crif1) in Cardiac Mitochondrial Structure and Stress-Induced Functional Decline
inferred← citeshttps://doi.org/10.4103/mgmj.mgmj_383_24openalex-snapshot
Mitochondrial dysfunction in cardiovascular disease: investigating therapeutic approaches to enhance patient outcomes
inferred← citeshttps://doi.org/10.46563/2686-8997-2026-7-1-209openalex-snapshot
Dilated cardiomyopathy in the structure of a familial form of TOP3A-related Bloom-like syndrome
inferred← citeshttps://doi.org/10.54033/cadpedv21n9-024openalex-snapshot
Choque cardiogênico e terapias de reparação mitocondrial: potencial de reversão da disfunção cardíaca
inferred← citeshttps://doi.org/10.6084/m9.figshare.26043670openalex-snapshot
Additional file 1 of Liguzinediol potentiates the metabolic remodeling by activating the AMPK/SIRT3 pathway and represses Caspase-3/GSDME-mediated pyroptosis to ameliorate cardiotoxicity
inferred← citeshttps://doi.org/10.70675/ce19657cz09dez41d0z9ffezca0d3b10b914openalex-snapshot
Rôle du remodelage de la matrice extracellulaire dans la reprogrammation des cellules vasculaires pulmonaires au cours de l'hypertension artérielle pulmonaire
Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.