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Astrogliopathy in Tauopathies

https://doi.org/10.3390/neuroglia1010010

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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1001/archneur.62.9.1444openalex-snapshot

A New Mutation of the τ Gene, G303V, in Early-Onset Familial Progressive Supranuclear Palsy

inferredcites →https://doi.org/10.1001/jamaneurol.2015.2711openalex-snapshot

Globular Glial Tauopathy Presenting as Semantic Variant Primary Progressive Aphasia

inferredcites →https://doi.org/10.1002/9781444341256openalex-snapshot

Neurodegeneration : the molecular pathology of dementia and movement disorders

inferredcites →https://doi.org/10.1002/9781444341256.ch14openalex-snapshot

Frontotemporal Dementia and Parkinsonism Linked to Chromosome 17

inferredcites →https://doi.org/10.1002/9781444341256.ch15openalex-snapshot

Progressive Supranuclear Palsy and Corticobasal Degeneration

inferredcites →https://doi.org/10.1002/9781444341256.ch46openalex-snapshot

Inherited Amyloidoses and Neurodegeneration: Familial British Dementia and Familial Danish Dementia

inferredcites →https://doi.org/10.1002/ana.10163openalex-snapshot

Late‐onset frontotemporal dementia with a novel exon 1 (Arg5His) tau gene mutation

inferredcites →https://doi.org/10.1002/ana.10340openalex-snapshot

An R5L τ mutation in a subject with a progressive supranuclear palsy phenotype

inferredcites →https://doi.org/10.1002/ana.10447openalex-snapshot

A novel L266V mutation of the tau gene causes frontotemporal dementia with a unique tau pathology

inferredcites →https://doi.org/10.1002/ana.10721openalex-snapshot

Variable phenotypic expression and extensive tau pathology in two families with the novel tau mutation L315R

inferredcites →https://doi.org/10.1002/ana.24559openalex-snapshot

Deep clinical and neuropathological phenotyping of P ick disease

inferredcites →https://doi.org/10.1002/cne.21744openalex-snapshot

Tauopathy with paired helical filaments in an aged chimpanzee

inferredcites →https://doi.org/10.1002/cne.23428openalex-snapshot

Alzheimer's disease pathology in the neocortex and hippocampus of the western lowland gorilla (Gorilla gorilla gorilla)

inferredcites →https://doi.org/10.1002/glia.20207openalex-snapshot

Astrocyte activation and reactive gliosis

inferredcites →https://doi.org/10.1002/glia.23163openalex-snapshot

Reduced gliotransmitter release from astrocytes mediates tau‐induced synaptic dysfunction in cultured hippocampal neurons

inferredcites →https://doi.org/10.1007/978-1-4615-4885-0openalex-snapshot

Cerebral Cortex

inferredcites →https://doi.org/10.1007/978-1-61779-452-0_3openalex-snapshot

Heterogeneity of Astrocytic Form and Function

inferredcites →https://doi.org/10.1007/978-3-319-57193-5_17openalex-snapshot

Neuroglia: Functional Paralysis and Reactivity in Alzheimer’s Disease and Other Neurodegenerative Pathologies

inferredcites →https://doi.org/10.1007/bf00294178openalex-snapshot

Expression of small heat-shock protein hsp 27 in reactive gliosis in Alzheimer disease and other types of dementia

inferredcites →https://doi.org/10.1007/bf00308809openalex-snapshot

Neuropathological stageing of Alzheimer-related changes

inferredcites →https://doi.org/10.1007/bf00318575openalex-snapshot

Thorn-shaped astrocytes: possibly secondarily induced tau-positive glial fibrillary tangles

inferredcites →https://doi.org/10.1007/s00401-002-0569-xopenalex-snapshot

Tau accumulation in astrocytes in progressive supranuclear palsy is a degenerative rather than a reactive process

inferredcites →https://doi.org/10.1007/s00401-002-0587-8openalex-snapshot

Glycogen synthase kinase-3 is associated with neuronal and glial hyperphosphorylated tau deposits in Alzheimer's disease, Pick's disease, progressive supranuclear palsy and corticobasal degeneration

inferredcites →https://doi.org/10.1007/s00401-003-0711-4openalex-snapshot

Distribution of astrocytic plaques in the corticobasal degeneration brain and comparison with tuft-shaped astrocytes in the progressive supranuclear palsy brain

inferredcites →https://doi.org/10.1007/s00401-003-0719-9openalex-snapshot

A novel leukoencephalopathy associated with tau deposits primarily in white matter glia

inferredcites →https://doi.org/10.1007/s00401-003-0756-4openalex-snapshot

Primary progressive aphasia as the initial manifestation of corticobasal degeneration and unusual tauopathies

inferredcites →https://doi.org/10.1007/s00401-004-0904-5openalex-snapshot

Distribution of tuft-shaped astrocytes in the cerebral cortex in progressive supranuclear palsy

inferredcites →https://doi.org/10.1007/s00401-004-0917-0openalex-snapshot

Regional and cellular pathology in frontotemporal dementia: relationship to stage of disease in cases with and without Pick bodies

inferredcites →https://doi.org/10.1007/s00401-005-1086-5openalex-snapshot

Sporadic four-repeat tauopathy with frontotemporal degeneration, parkinsonism and motor neuron disease

inferredcites →https://doi.org/10.1007/s00401-007-0266-xopenalex-snapshot

Argyrophilic thorny astrocyte clusters in association with Alzheimer’s disease pathology in possible primary progressive aphasia

inferredcites →https://doi.org/10.1007/s00401-009-0619-8openalex-snapshot

Astrocytes: biology and pathology

inferredcites →https://doi.org/10.1007/s00401-010-0649-2openalex-snapshot

Sporadic four-repeat tauopathy with frontotemporal lobar degeneration, Parkinsonism, and motor neuron disease: a distinct clinicopathological and biochemical disease entity

inferredcites →https://doi.org/10.1007/s00401-011-0819-xopenalex-snapshot

A peculiar constellation of tau pathology defines a subset of dementia in the elderly

inferredcites →https://doi.org/10.1007/s00401-011-0857-4openalex-snapshot

Globular glial tauopathies (GGT) presenting with motor neuron disease or frontotemporal dementia: an emerging group of 4-repeat tauopathies

inferredcites →https://doi.org/10.1007/s00401-013-1080-2openalex-snapshot

Argyrophilic grain disease differs from other tauopathies by lacking tau acetylation

inferredcites →https://doi.org/10.1007/s00401-013-1171-0openalex-snapshot

Globular glial tauopathies (GGT): consensus recommendations

inferredcites →https://doi.org/10.1007/s00401-014-1349-0openalex-snapshot

Primary age-related tauopathy (PART): a common pathology associated with human aging

inferredcites →https://doi.org/10.1007/s00401-014-1373-0openalex-snapshot

Differential induction and spread of tau pathology in young PS19 tau transgenic mice following intracerebral injections of pathological tau from Alzheimer’s disease or corticobasal degeneration brains

inferredcites →https://doi.org/10.1007/s00401-015-1390-7openalex-snapshot

PART is part of Alzheimer disease

inferredcites →https://doi.org/10.1007/s00401-015-1407-2openalex-snapshot

PART, a distinct tauopathy, different from classical sporadic Alzheimer disease

inferredcites →https://doi.org/10.1007/s00401-015-1425-0openalex-snapshot

A novel tau mutation, p.K317N, causes globular glial tauopathy

inferredcites →https://doi.org/10.1007/s00401-015-1507-zopenalex-snapshot

Propagation of tau pathology: hypotheses, discoveries, and yet unresolved questions from experimental and human brain studies

inferredcites →https://doi.org/10.1007/s00401-015-1509-xopenalex-snapshot

Aging-related tau astrogliopathy (ARTAG): harmonized evaluation strategy

inferredcites →https://doi.org/10.1007/s00401-015-1513-1openalex-snapshot

Astrocytes: a central element in neurological diseases

inferredcites →https://doi.org/10.1007/s004010000283openalex-snapshot

Distinct isoforms of tau aggregated in neurons and glial cells in brains of patients with Pick's disease, corticobasal degeneration and progressive supranuclear palsy

inferredcites →https://doi.org/10.1007/s004010000333openalex-snapshot

Familial frontotemporal dementia and parkinsonism with a novel N296H mutation in exon 10 of the tau gene and a widespread tau accumulation in the glial cells

inferredcites →https://doi.org/10.1007/s004010050642openalex-snapshot

Argyrophilic grain disease: widespread hyperphosphorylation of tau protein in limbic neurons

inferredcites →https://doi.org/10.1007/s004010050911openalex-snapshot

Astrocytic plaques and tufts of abnormal fibers do not coexist in corticobasal degeneration and progressive supranuclear palsy

inferredcites →https://doi.org/10.1007/s004010051077openalex-snapshot

Astrocytes expressing hyperphosphorylated tau protein without glial fibrillary tangles in argyrophilic grain disease

inferredcites →https://doi.org/10.1007/s00441-014-1814-zopenalex-snapshot

Neuroglia in ageing and disease

inferredcites →https://doi.org/10.1007/s00702-014-1360-6openalex-snapshot

Frontotemporal lobar degeneration FTLD-tau: preclinical lesions, vascular, and Alzheimer-related co-pathologies

inferredcites →https://doi.org/10.1007/s007020100016openalex-snapshot

Phosphorylated mitogen-activated protein kinase (MAPK/ERK-P), protein kinase of 38 kDa (p38-P), stress-activated protein kinase (SAPK/JNK-P), and calcium/calmodulin-dependent kinase II (CaM kinase II) are differentially expressed in tau deposits in neurons and glial cells in tauopathies

inferredcites →https://doi.org/10.1007/s10072-012-1006-0openalex-snapshot

Spatial patterns of the tau pathology in progressive supranuclear palsy

inferredcites →https://doi.org/10.1016/0304-3940(92)90145-wopenalex-snapshot

Appearance of paired nucleated, Tau-positive glia in patients with progressive supranuclear palsy brain tissue

inferredcites →https://doi.org/10.1016/0304-3940(92)90227-xopenalex-snapshot

Glial fibrillary tangles with straight tubules in the brains of patients with progressive supranuclear palsy

inferredcites →https://doi.org/10.1016/0304-3940(95)12151-xopenalex-snapshot

Immunohistochemical investigation of tau-positive structures in the cerebral cortex of patients with progressive supranuclear palsy

inferredcites →https://doi.org/10.1016/j.ajpath.2013.04.025openalex-snapshot

Acetylated Tau Neuropathology in Sporadic and Hereditary Tauopathies

inferredcites →https://doi.org/10.1016/j.bbadis.2015.03.003openalex-snapshot

Intraneuronal accumulation of misfolded tau protein induces overexpression of Hsp27 in activated astrocytes

inferredcites →https://doi.org/10.1016/j.bbadis.2015.11.014openalex-snapshot

Reactive gliosis in the pathogenesis of CNS diseases

inferredcites →https://doi.org/10.1016/j.brainres.2009.12.005openalex-snapshot

Alzheimer-type tau pathology in advanced aged nonhuman primate brains harboring substantial amyloid deposition

inferredcites →https://doi.org/10.1016/j.brainres.2011.06.014openalex-snapshot

Perivascular orientation of astrocytic plaques and tuft-shaped astrocytes

inferredcites →https://doi.org/10.1016/j.celrep.2015.04.043openalex-snapshot

Synaptic Contacts Enhance Cell-to-Cell Tau Pathology Propagation

inferredcites →https://doi.org/10.1016/j.celrep.2016.12.066openalex-snapshot

Primary Cell Culture of Live Neurosurgically Resected Aged Adult Human Brain Cells and Single Cell Transcriptomics

inferredcites →https://doi.org/10.1016/j.coph.2015.09.011openalex-snapshot

Astroglia dynamics in ageing and Alzheimer's disease

inferredcites →https://doi.org/10.1016/j.gene.2004.07.013openalex-snapshot

Tau gene (MAPT) sequence variation among primates

inferredcites →https://doi.org/10.1016/j.jns.2009.11.002openalex-snapshot

Neuronal and glial tau pathology in early frontotemporal lobar degeneration-tau, Pick's disease subtype

inferredcites →https://doi.org/10.1016/j.jprot.2011.03.033openalex-snapshot

Quantitative proteomics analysis of phosphorylated proteins in the hippocampus of Alzheimer's disease subjects

inferredcites →https://doi.org/10.1016/j.nbd.2005.12.001openalex-snapshot

A MAPT mutation in a regulatory element upstream of exon 10 causes frontotemporal dementia

inferredcites →https://doi.org/10.1016/j.neuint.2003.08.015openalex-snapshot

The problem of astrocyte identity

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2011.04.013openalex-snapshot

Microarray analysis of the astrocyte transcriptome in the aging brain: relationship to Alzheimer’s pathology and APOE genotype

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2013.07.008openalex-snapshot

Acute isolation and transcriptome characterization of cortical astrocytes and microglia from young and aged mice

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2014.06.004openalex-snapshot

Isolation of glia from Alzheimer's mice reveals inflammation and dysfunction

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2015.12.017openalex-snapshot

Early Alzheimer's disease–type pathology in the frontal cortex of wild mountain gorillas ( Gorilla beringei beringei )

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2017.07.006openalex-snapshot

Aged chimpanzees exhibit pathologic hallmarks of Alzheimer's disease

inferredcites →https://doi.org/10.1016/j.neuron.2010.08.044openalex-snapshot

Acetylation of Tau Inhibits Its Degradation and Contributes to Tauopathy

inferredcites →https://doi.org/10.1016/j.neuron.2015.11.013openalex-snapshot

Purification and Characterization of Progenitor and Mature Human Astrocytes Reveals Transcriptional and Functional Differences with Mouse

inferredcites →https://doi.org/10.1016/j.neuron.2016.09.055openalex-snapshot

Tau Prion Strains Dictate Patterns of Cell Pathology, Progression Rate, and Regional Vulnerability In Vivo

inferredcites →https://doi.org/10.1016/j.nicl.2017.09.008openalex-snapshot

Advancing functional dysconnectivity and atrophy in progressive supranuclear palsy

inferredcites →https://doi.org/10.1016/j.pneurobio.2012.03.005openalex-snapshot

Defining Alzheimer as a common age-related neurodegenerative process not inevitably leading to dementia

inferredcites →https://doi.org/10.1016/j.pneurobio.2016.01.001openalex-snapshot

Astrogliosis: An integral player in the pathogenesis of Alzheimer's disease

inferredcites →https://doi.org/10.1016/s0002-9440(10)63106-9openalex-snapshot

Expression of the Small Heat-Shock Protein αB-Crystallin in Tauopathies with Glial Pathology

inferredcites →https://doi.org/10.1016/s0002-9440(10)63812-6openalex-snapshot

Filamentous Tau in Oligodendrocytes and Astrocytes of Transgenic Mice Expressing the Human Tau Isoform with the P301L Mutation

inferredcites →https://doi.org/10.1016/s0072-9752(07)01251-1openalex-snapshot

Argyrophilic grain disease

inferredcites →https://doi.org/10.1016/s0197-0186(01)00054-7openalex-snapshot

Pathological glial tau accumulations in neurodegenerative disease: review and case report

inferredcites →https://doi.org/10.1016/s0197-4580(03)00113-1openalex-snapshot

High prevalence of thorn-shaped astrocytes in the aged human medial temporal lobe

inferredcites →https://doi.org/10.1016/s0197-4580(98)00034-7openalex-snapshot

Glial Tau Pathology in Neurodegenerative Diseases: Their Nature and Comparison with Neuronal Tangles

inferredcites →https://doi.org/10.1016/s0304-3940(97)00161-4openalex-snapshot

Glial tau-positive structures lack the sequence encoded by exon 3 of the tau protein gene

inferredcites →https://doi.org/10.1016/s0896-6273(02)00789-4openalex-snapshot

Transgenic Mouse Model of Tauopathies with Glial Pathology and Nervous System Degeneration

inferredcites →https://doi.org/10.1017/cbo9781107588660openalex-snapshot

Neuropathology of Neurodegenerative Diseases

inferredcites →https://doi.org/10.1038/ncb1901openalex-snapshot

Transmission and spreading of tauopathy in transgenic mouse brain

inferredcites →https://doi.org/10.1038/ncomms1255openalex-snapshot

The acetylation of tau inhibits its function and promotes pathological tau aggregation

inferredcites →https://doi.org/10.1038/nm.3457openalex-snapshot

Cell-to-cell transmission of pathogenic proteins in neurodegenerative diseases

inferredcites →https://doi.org/10.1038/nmeth.4407openalex-snapshot

Massively parallel single-nucleus RNA-seq with DroNc-seq

inferredcites →https://doi.org/10.1038/nn.3639openalex-snapshot

Tau promotes neurodegeneration through global chromatin relaxation

inferredcites →https://doi.org/10.1038/nn.4493openalex-snapshot

Identification of diverse astrocyte populations and their malignant analogs

inferredcites →https://doi.org/10.1038/nrn1870openalex-snapshot

Astrocyte dysfunction in neurological disorders: a molecular perspective

inferredcites →https://doi.org/10.1038/srep20762openalex-snapshot

Astrocyte-to-neuron intercellular prion transfer is mediated by cell-cell contact

inferredcites →https://doi.org/10.1038/srep42991openalex-snapshot

Astrocyte pathology in a human neural stem cell model of frontotemporal dementia caused by mutant TAU protein

inferredcites →https://doi.org/10.1046/j.1365-2990.2003.00435.xopenalex-snapshot

Tau phosphorylation and kinase activation in familial tauopathy linked to deln296 mutation

inferredcites →https://doi.org/10.1073/pnas.1301175110openalex-snapshot

Brain homogenates from human tauopathies induce tau inclusions in mouse brain

inferredcites →https://doi.org/10.1073/pnas.1507125112openalex-snapshot

A survey of human brain transcriptome diversity at the single cell level

inferredcites →https://doi.org/10.1073/pnas.94.8.4113openalex-snapshot

Familial multiple system tauopathy with presenile dementia: A disease with abundant neuronal and glial tau filaments

inferredcites →https://doi.org/10.1074/jbc.m115.709485openalex-snapshot

Extracellular Vesicles Isolated from the Brains of rTg4510 Mice Seed Tau Protein Aggregation in a Threshold-dependent Manner

inferredcites →https://doi.org/10.1074/jbc.r114.549295openalex-snapshot

Prion-like Properties of Tau Protein: The Importance of Extracellular Tau as a Therapeutic Target

inferredcites →https://doi.org/10.1084/jem.20160833openalex-snapshot

Unique pathological tau conformers from Alzheimer’s brains transmit tau pathology in nontransgenic mice

inferredcites →https://doi.org/10.1089/rej.2008.0677openalex-snapshot

Cerebral Amyloid-Beta Protein Accumulation with Aging in Cotton-Top Tamarins: A Model of Early Alzheimer's Disease?

inferredcites →https://doi.org/10.1093/brain/123.5.880openalex-snapshot

Progressive supranuclear palsy pathology caused by a novel silent mutation in exon 10 of the tau gene

inferredcites →https://doi.org/10.1093/brain/awh720openalex-snapshot

Neuropathology in the S305S tau gene mutation

inferredcites →https://doi.org/10.1093/brain/awm280openalex-snapshot

The tauopathy associated with mutation +3 in intron 10 of Tau: characterization of the MSTD family

inferredcites →https://doi.org/10.1093/brain/awm305openalex-snapshot

Argyrophilic grain disease

inferredcites →https://doi.org/10.1093/brain/awv236openalex-snapshot

The preclinical phase of the pathological process underlying sporadic Alzheimer’s disease

inferredcites →https://doi.org/10.1093/brain/aww256openalex-snapshot

Astrogliopathy predominates the earliest stage of corticobasal degeneration pathology

inferredcites →https://doi.org/10.1093/hmg/ddu475openalex-snapshot

Comprehensive phosphoproteome analysis unravels the core signaling network that initiates the earliest synapse pathology in preclinical Alzheimer's disease brain

inferredcites →https://doi.org/10.1093/jnen/59.1.39openalex-snapshot

Filamentous Tau Pathology in Nerve Cells, Astrocytes, and Oligodendrocytes of Aged Baboons

inferredcites →https://doi.org/10.1093/jnen/60.12.1190openalex-snapshot

Activation of the JNK/p38 Pathway Occurs in Diseases Characterized by Tau Protein Pathology and Is Related to Tau Phosphorylation But Not to Apoptosis

inferredcites →https://doi.org/10.1093/jnen/60.4.328openalex-snapshot

Frontal Lobe Dementia With Novel Tauopathy: Sporadic Multiple System Tauopathy With Dementia

inferredcites →https://doi.org/10.1093/jnen/63.9.911openalex-snapshot

Staging of Argyrophilic Grains: An Age-Associated Tauopathy

inferredcites →https://doi.org/10.1093/jnen/nlx007openalex-snapshot

Evaluating the Patterns of Aging-Related Tau Astrogliopathy Unravels Novel Insights Into Brain Aging and Neurodegenerative Diseases

inferredcites →https://doi.org/10.1097/00001756-199902250-00010openalex-snapshot

A distinct familial presenile dementia with a novel missense mutation in the tau gene

inferredcites →https://doi.org/10.1097/00001756-200104170-00014openalex-snapshot

Intracellular processing of aggregated tau differs between corticobasal degeneration and progressive supranuclear palsy

inferredcites →https://doi.org/10.1097/01.jnen.0000218446.38158.61openalex-snapshot

Atypical Progressive Supranuclear Palsy With Corticospinal Tract Degeneration

inferredcites →https://doi.org/10.1097/nen.0000000000000030openalex-snapshot

Glial and Neuronal Tau Pathology in Tauopathies

inferredcites →https://doi.org/10.1097/nen.0000000000000180openalex-snapshot

Familial Behavioral Variant Frontotemporal Dementia Associated With Astrocyte-Predominant Tauopathy

inferredcites →https://doi.org/10.1097/nen.0000000000000241openalex-snapshot

Neuroinflammatory Gene Regulation, Mitochondrial Function, Oxidative Stress, and Brain Lipid Modifications With Disease Progression in Tau P301S Transgenic Mice as a Model of Frontotemporal Lobar Degeneration-Tau

inferredcites →https://doi.org/10.1097/nen.0b013e318187a80fopenalex-snapshot

White Matter Tauopathy With Globular Glial Inclusions: A Distinct Sporadic Frontotemporal Lobar Degeneration

inferredcites →https://doi.org/10.1097/nen.0b013e3181b66f1bopenalex-snapshot

Degeneration in Different Parkinsonian Syndromes Relates to Astrocyte Type and Astrocyte Protein Expression

inferredcites →https://doi.org/10.1097/nen.0b013e318232a379openalex-snapshot

Stages of the Pathologic Process in Alzheimer Disease: Age Categories From 1 to 100 Years

inferredcites →https://doi.org/10.1097/wnn.0000000000000060openalex-snapshot

Temporal Variant Frontotemporal Dementia Is Associated with Globular Glial Tauopathy

inferredcites →https://doi.org/10.1111/bpa.12044openalex-snapshot

“Prion‐Like” Templated Misfolding in Tauopathies

inferredcites →https://doi.org/10.1111/bpa.12335openalex-snapshot

Altered Machinery of Protein Synthesis in Alzheimer's: From the Nucleolus to the Ribosome

inferredcites →https://doi.org/10.1111/bpa.12536openalex-snapshot

Protein astrogliopathies in human neurodegenerative diseases and aging

inferredcites →https://doi.org/10.1111/bpa.12537openalex-snapshot

Stratification of astrocytes in healthy and diseased brain

inferredcites →https://doi.org/10.1111/bpa.12538openalex-snapshot

Diversity of astroglial responses across human neurodegenerative disorders and brain aging

inferredcites →https://doi.org/10.1111/bpa.12593openalex-snapshot

Aging‐related tau astrogliopathy (ARTAG): not only tau phosphorylation in astrocytes

inferredcites →https://doi.org/10.1111/j.1365-2990.2007.00927.xopenalex-snapshot

Atypical tauopathy with massive involvement of the white matter

inferredcites →https://doi.org/10.1111/j.1440-1789.2006.00669.xopenalex-snapshot

Ultrastructural characteristics of tau filaments in tauopathies: Immuno‐electron microscopic demonstration of tau filaments in tauopathies

inferredcites →https://doi.org/10.1111/j.1445-5994.2006.01153.xopenalex-snapshot

Tauopathies: classification and clinical update on neurodegenerative diseases associated with microtubule‐associated protein tau

inferredcites →https://doi.org/10.1111/j.1471-4159.2006.04032.xopenalex-snapshot

Glial fibrillary acidic protein is a major target of glycoxidative and lipoxidative damage in Pick's disease

inferredcites →https://doi.org/10.1111/j.1471-4159.2012.07737.xopenalex-snapshot

Phosphoproteome profiling of substantia nigra and cortex regions of Alzheimer’s disease patients

inferredcites →https://doi.org/10.1111/j.1750-3639.1998.tb00161.xopenalex-snapshot

Dementia with Grains (Argyrophilic Grain Disease)

inferredcites →https://doi.org/10.1111/j.1750-3639.1999.tb00549.xopenalex-snapshot

Tau‐positive dial Inclusions in Progressive Supranuclear Palsy, Corticobasal Degeneration and Pick's Disease

inferredcites →https://doi.org/10.1111/j.1750-3639.2001.tb00387.xopenalex-snapshot

Phosphorylated Map Kinase (ERK1, ERK2) Expression is Associated with Early Tau Deposition in Neurones and Glial Cells, but not with Increased Nuclear DNA Vulnerability and Cell Death, in Alzheimer Disease, Pick's Disease, Progressive Supranuclear Palsy and Corticobasal Degeneration

inferredcites →https://doi.org/10.1111/j.1750-3639.2003.tb00007.xopenalex-snapshot

Phosphorylated Protein Kinases Associated with Neuronal and Glial Tau Deposits in Argyrophilic Grain Disease

inferredcites →https://doi.org/10.1111/j.1750-3639.2008.00173.xopenalex-snapshot

Delineation of Early Changes in Cases with Progressive Supranuclear Palsy‐Like Pathology. Astrocytes in Striatum are Primary Targets of Tau Phosphorylation and GFAP Oxidation

inferredcites →https://doi.org/10.1111/j.1750-3639.2009.00326.xopenalex-snapshot

Protein Targets of Oxidative Damage in Human Neurodegenerative Diseases with Abnormal Protein Aggregates

inferredcites →https://doi.org/10.1111/j.1750-3639.2012.00627.xopenalex-snapshot

Characterization of Thorn‐Shaped Astrocytes in White Matter of Temporal Lobe in A lzheimer's Disease Brains

inferredcites →https://doi.org/10.1111/nan.12197openalex-snapshot

Invited review: Prion‐like transmission and spreading of tau pathology

inferredcites →https://doi.org/10.1111/nan.12208openalex-snapshot

Invited review: Neuropathology of tauopathies: principles and practice

inferredcites →https://doi.org/10.1111/nan.12213openalex-snapshot

Invited review: Frontotemporal dementia caused by microtubule‐associated protein tau gene ( MAPT ) mutations: a chameleon for neuropathology and neuroimaging

inferredcites →https://doi.org/10.1111/nan.12339openalex-snapshot

Incidental corticobasal degeneration

inferredcites →https://doi.org/10.1111/nan.12425openalex-snapshot

Evolving concepts of chronic traumatic encephalopathy as a neuropathological entity

inferredcites →https://doi.org/10.1111/neup.12134openalex-snapshot

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Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Aging and high-fat diet feeding lead to peripheral insulin resistance and sex-dependent changes in brain of mouse model of tau pathology THY-Tau22

inferred← citeshttps://doi.org/10.1186/s13024-021-00487-8openalex-snapshot

Human iPSC-derived astrocytes transplanted into the mouse brain undergo morphological changes in response to amyloid-β plaques

inferred← citeshttps://doi.org/10.21203/rs.3.rs-112937/v1openalex-snapshot

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Additional file 1 of Aging and high-fat diet feeding lead to peripheral insulin resistance and sex-dependent changes in brain of mouse model of tau pathology THY-Tau22

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Additional file 1 of Human iPSC-derived astrocytes transplanted into the mouse brain undergo morphological changes in response to amyloid-β plaques

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Study of the Neuron-Astrocyte Relationship in a Rodent Model of Tauopathy

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