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Escherichia coli Cytotoxic Necrotizing Factor 1 (CNF1): Toxin Biology, in Vivo Applications and Therapeutic Potential
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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An Escherichia coli Cytotoxin Increases Superoxide Anion Generation via Rac in Epithelial Cells
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Toxin-Induced Activation of Rho GTP-Binding Protein Increases Bcl-2 Expression and Influences Mitochondrial Homeostasis
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Triggered Phagocytosis by Salmonella: Bacterial Molecular Mimicry of RhoGTPase Activation/Deactivation
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A cell division-active protein from E. coli
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Cytoskeletal changes induced in HEp-2 cells by the cytotoxic necrotizing factor of Escherichia coli
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Spatial and temporal aspects of spinal cord and brainstem activation in the formalin pain model
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Induction of Spine Growth and Synapse Formation by Regulation of the Spine Actin Cytoskeleton
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The Rac GTPase-activating bacterial protein toxin CNF1 induces analgesia up-regulating μ-opioid receptors
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GDIs: central regulatory molecules in Rho GTPase activation
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The Rho GTPase activators CNF1 and DNT bacterial toxins have mucosal adjuvant properties
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Intranasal immunization with tetanus toxoid and CNF1 as a new mucosal adjuvant protects BALB/c mice against lethal challenge
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The cytotoxic necrotizing factor 1 (CNF1) from Escherichia coli
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CNF1 Exploits the Ubiquitin-Proteasome Machinery to Restrict Rho GTPase Activation for Bacterial Host Cell Invasion
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Rho and Rac Take Center Stage
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Long-term persistence of cellular immunity to Oka vaccine virus induced by pernasal co-administration with Escherichia coli enterotoxin in mice
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Hippocampal LTP Is Accompanied by Enhanced F-Actin Content within the Dendritic Spine that Is Essential for Late LTP Maintenance In Vivo
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Role of the Sensory Neuron Cytoskeleton in Second Messenger Signaling for Inflammatory Pain
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Rho GTPase-activating proteins in cell regulation
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Crystal structure of a small G protein in complex with the GTPase-activating protein rhoGAP
inferredcites →https://doi.org/10.1038/42735openalex-snapshot
Gln 63 of Rho is deamidated by Escherichia coli cytotoxic necrotizing factor-1
inferredcites →https://doi.org/10.1038/42743openalex-snapshot
Toxin-induced activation of the G protein p21 Rho by deamidation of glutamine
inferredcites →https://doi.org/10.1038/45829openalex-snapshot
A Salmonella protein antagonizes Rac-1 and Cdc42 to mediate host-cell recovery after bacterial invasion
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Structure of the Rho-activating domain of Escherichia coli cytotoxic necrotizing factor 1.
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Bacterial toxins and the Rho GTP-binding protein: what microbes teach us about cell regulation
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Cytotoxic necrotizing factor 1 hinders skeletal muscle differentiation in vitro by perturbing the activation/deactivation balance of Rho GTPases
inferredcites →https://doi.org/10.1046/j.1365-2958.1997.4151781.xopenalex-snapshot
Molecular localization of the Escherichia coli cytotoxic necrotizing factor CNF1 cell‐binding and catalytic domains
inferredcites →https://doi.org/10.1046/j.1365-2958.1999.01453.xopenalex-snapshot
The 5′ region of cnf1 harbours a translational regulatory mechanism for CNF1 synthesis and encodes the cell‐binding domain of the toxin
inferredcites →https://doi.org/10.1046/j.1365-2958.2001.02596.xopenalex-snapshot
Mutation of specific acidic residues of the CNF1 T domain into lysine alters cell membrane translocation of the toxin
inferredcites →https://doi.org/10.1046/j.1472-765x.2000.00698.xopenalex-snapshot
Frequency of Escherichia coli strains producing the cytotoxic necrotizing factor (CNF1) in nosocomial urinary tract infections
inferredcites →https://doi.org/10.1073/pnas.0610059104openalex-snapshot
Enhancement of learning and memory after activation of cerebral Rho GTPases
inferredcites →https://doi.org/10.1074/jbc.272.14.9587openalex-snapshot
Cytotoxic Necrotizing Factor 1 from Escherichia coli and Dermonecrotic Toxin from Bordetella bronchiseptica Induce p21 -dependent Tyrosine Phosphorylation of Focal Adhesion Kinase and Paxillin in Swiss 3T3 Cells
inferredcites →https://doi.org/10.1074/jbc.272.31.19532openalex-snapshot
Escherichia coli Cytotoxic Necrotizing Factor 1 (CNF1), a Toxin That Activates the Rho GTPase
inferredcites →https://doi.org/10.1074/jbc.273.22.13669openalex-snapshot
The Rho-deamidating Cytotoxic Necrotizing Factor 1 fromEscherichia coli Possesses Transglutaminase Activity
inferredcites →https://doi.org/10.1074/jbc.m301028200openalex-snapshot
37-kDa Laminin Receptor Precursor Modulates Cytotoxic Necrotizing Factor 1–mediated RhoA Activation and Bacterial Uptake
inferredcites →https://doi.org/10.1074/jbc.m401580200openalex-snapshot
Activation and Proteasomal Degradation of Rho GTPases by Cytotoxic Necrotizing Factor-1 Elicit a Controlled Inflammatory Response
inferredcites →https://doi.org/10.1074/jbc.m410176200openalex-snapshot
67-kDa Laminin Receptor Promotes Internalization of Cytotoxic Necrotizing Factor 1-expressing Escherichia coli K1 into Human Brain Microvascular Endothelial Cells
inferredcites →https://doi.org/10.1091/mbc.11.5.1775openalex-snapshot
The p21 Rho-activating Toxin Cytotoxic Necrotizing Factor 1 Is Endocytosed by a Clathrin-independent Mechanism and Enters the Cytosol by an Acidic-dependent Membrane Translocation Step
inferredcites →https://doi.org/10.1091/mbc.12.7.2061openalex-snapshot
Activation of Rho GTPases by Cytotoxic Necrotizing Factor 1 Induces Macropinocytosis and Scavenging Activity in Epithelial Cells
inferredcites →https://doi.org/10.1091/mbc.e03-05-0301openalex-snapshot
Rac GTPase Instructs Nuclear Factor-κB Activation by Conveying the SCF Complex and IkBα to the Ruffling Membranes
inferredcites →https://doi.org/10.1091/mbc.e06-10-0910openalex-snapshot
Cytotoxic Necrotizing Factor 1 Prevents Apoptosis via the Akt/IκB Kinase Pathway: Role of Nuclear Factor-κB and Bcl-2
inferredcites →https://doi.org/10.1099/0022-1317-51-7-548openalex-snapshot
Virulence properties of Escherichia coli faecal strains isolated in Poland from healthy children and strains belonging to serogroups O18, O26, O44, O86, O126 and O127 isolated from children with diarrhoea
inferredcites →https://doi.org/10.1099/00222615-31-3-175openalex-snapshot
Virulence factors of bacteraemic Escherichia coli with particular reference to production of cytotoxic necrotising factor (CNF) by P-fimbriate strains
inferredcites →https://doi.org/10.1101/gad.1003302openalex-snapshot
Guanine nucleotide exchange factors for Rho GTPases: turning on the switch
inferredcites →https://doi.org/10.1111/j.1365-2958.1993.tb01254.xopenalex-snapshot
Induction of phagocytic behaviour in human epithelial cells by Escherichia coli cytotoxic necrotizing factor type1
inferredcites →https://doi.org/10.1111/j.1471-4159.2005.03551.xopenalex-snapshot
Rapid disassembly of dynamic microtubules upon activation of the capsaicin receptor TRPV1
inferredcites →https://doi.org/10.1111/j.1574-6976.2007.00078.xopenalex-snapshot
Rho GTPase-activating bacterial toxins: from bacterial virulence regulation to eukaryotic cell biology
inferredcites →https://doi.org/10.1126/science.279.5350.509openalex-snapshot
Rho GTPases and the Actin Cytoskeleton
inferredcites →https://doi.org/10.1128/iai.00075-07openalex-snapshot
Two Domains of Cytotoxic Necrotizing Factor Type 1 Bind the Cellular Receptor, Laminin Receptor Precursor Protein
inferredcites →https://doi.org/10.1128/iai.01145-08openalex-snapshot
Cleavage of Escherichia coli Cytotoxic Necrotizing Factor 1 Is Required for Full Biologic Activity
inferredcites →https://doi.org/10.1128/iai.01413-05openalex-snapshot
Escherichia coli Cytotoxic Necrotizing Factor 1 Blocks Cell Cycle G 2 /M Transition in Uroepithelial Cells
inferredcites →https://doi.org/10.1128/iai.01937-06openalex-snapshot
The Cytotoxic Necrotizing Factors from Yersinia pseudotuberculosis and from Escherichia coli Bind to Different Cellular Receptors but Take the Same Route to the Cytosol
inferredcites →https://doi.org/10.1128/iai.39.3.1300-1306.1983openalex-snapshot
Partial purification and characterization of an escherichia coli toxic factor that induces morphological cell alterations
inferredcites →https://doi.org/10.1128/iai.66.5.2040-2051.1998openalex-snapshot
Characterization of the Roles of Hemolysin and Other Toxins in Enteropathy Caused by Alpha-Hemolytic Escherichia coli Linked to Human Diarrhea
inferredcites →https://doi.org/10.1128/iai.67.2.496-503.1999openalex-snapshot
Deamidation of Cdc42 and Rac by Escherichia coli Cytotoxic Necrotizing Factor 1: Activation of c-Jun N-Terminal Kinase in HeLa Cells
inferredcites →https://doi.org/10.1128/iai.69.11.6839-6845.2001openalex-snapshot
Cytotoxic Necrotizing Factor from Escherichia coli Induces RhoA-Dependent Expression of the Cyclooxygenase-2 Gene
inferredcites →https://doi.org/10.1128/iai.70.10.5865-5869.2002openalex-snapshot
A Uropathogenicity Island Contributes to the Pathogenicity of Escherichia coli Strains That Cause Neonatal Meningitis
inferredcites →https://doi.org/10.1128/iai.71.7.4178-4181.2003openalex-snapshot
Cytotoxic Necrotizing Factor 1 Enhances Reactive Oxygen Species-Dependent Transcription and Secretion of Proinflammatory Cytokines in Human Uroepithelial Cells
inferredcites →https://doi.org/10.1146/annurev.cellbio.21.020604.150721openalex-snapshot
RHO GTPASES: Biochemistry and Biology
inferredcites →https://doi.org/10.1152/jn.00249.2005openalex-snapshot
DECONSTRUCTING ENDOGENOUS PAIN MODULATIONS
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Adjuvants in vaccines and for immunisation: current trends
inferredcites →https://doi.org/10.2174/092986708784221430openalex-snapshot
Bacterial Protein Toxins: Current and Potential Clinical Use
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Botulinum Toxin: Pharmacology and Clinical Developments: A Literature Review
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Antibiotic Resistance in Escherichia coli from Nigerian Students, 1986-1998
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Incoming claims: where this work appears in other papers’ reference lists and arguments.
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The bacterial toxin CNF1 as a tool to induce retinal degeneration reminiscent of retinitis pigmentosa
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Modular domain swapping among the bacterial cytotoxic necrotizing factor (CNF) family for efficient cargo delivery into mammalian cells
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Modeling and computational characterization of a Xanthomonas sp. Hypothetical protein identifies a remote ortholog of Burkholderia lethal factor 1
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Cytotoxic necrotizing factor 1 hinders colon tumorigenesis induced by colibactin-producing Escherichia coli in Apc Min/+ mice
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