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Biomaterials-based anti-inflammatory treatment strategies for Alzheimer’s disease

https://doi.org/10.4103/1673-5374.374137

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Microglia constitute a barrier that prevents neurotoxic protofibrillar Aβ42 hotspots around plaques

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Blood–brain barrier breakdown in Alzheimer disease and other neurodegenerative disorders

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The Amyloid-β Pathway in Alzheimer’s Disease

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The innate immunity protein IFITM3 modulates γ-secretase in Alzheimer’s disease

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FSH blockade improves cognition in mice with Alzheimer’s disease

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Fibrin-targeting immunotherapy protects against neuroinflammation and neurodegeneration

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Antibiotic-induced perturbations in gut microbial diversity influences neuro-inflammation and amyloidosis in a murine model of Alzheimer’s disease

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Reduction of Abeta amyloid pathology in APPPS1 transgenic mice in the absence of gut microbiota

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Curcumin-primed exosomes potently ameliorate cognitive function in AD mice by inhibiting hyperphosphorylation of the Tau protein through the AKT/GSK-3β pathway

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S-equol, a metabolite of dietary soy isoflavones, alleviates lipopolysaccharide-induced depressive-like behavior in mice by inhibiting neuroinflammation and enhancing synaptic plasticity

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Acute Trem2 reduction triggers increased microglial phagocytosis, slowing amyloid deposition in mice

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Tumor Necrosis Factor-α, Interleukin-1β, and Interferon-γ Stimulate γ-Secretase-mediated Cleavage of Amyloid Precursor Protein through a JNK-dependent MAPK Pathway

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Grape seed proanthocyanidins improves depression-like behavior by alleviating oxidative stress and NLRP3 activation in the hippocampus of prenatally-stressed female offspring rats

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Indomethacin attenuates neuroinflammation and memory impairment in an STZ-induced model of Alzheimer’s like disease

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Dual functional liposomes carrying antioxidants against tau hyperphosphorylation and apoptosis of neurons

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