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Regulation of vascular smooth muscle phenotype by cross-regulation of krüppel-like factors

https://doi.org/10.4196/kjpp.2017.21.1.37

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Disruption of tumor necrosis factor-α gene diminishes the development of atherosclerosis in ApoE-deficient mice

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MRI follow-up of TNF-dependent differential progression of atherosclerotic wall-thickening in mouse aortic arch from early to advanced stages

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Akt1 isoform modulates phenotypic conversion of vascular smooth muscle cells

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All-trans retinoic acid increases KLF4 acetylation by inducing HDAC2 phosphorylation and its dissociation from KLF4 in vascular smooth muscle cells

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Platelet-derived growth factor regulates vascular smooth muscle phenotype via mammalian target of rapamycin complex 1

inferredcites →https://doi.org/10.1016/j.bbrc.2015.06.123openalex-snapshot

Essential role of krüppel-like factor 5 during tumor necrosis factor α-induced phenotypic conversion of vascular smooth muscle cells

inferredcites →https://doi.org/10.1016/j.jash.2008.09.002openalex-snapshot

Vascular smooth muscle cell signaling mechanisms for contraction to angiotensin II and endothelin-1

inferredcites →https://doi.org/10.1016/j.surg.2006.04.004openalex-snapshot

Angiotensin II and tumor necrosis factor-alpha upregulate survivin and Kruppel-like factor 5 in smooth muscle cells: Potential relevance to vein graft hyperplasia

inferredcites →https://doi.org/10.1016/j.yjmcc.2007.04.005openalex-snapshot

Kruppel-like Factors (KLFs) in muscle biology

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Inducible expression of BTEB2, a member of the zinc-finger family of transcription factors, in cardiac allograft arteriosclerosis

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Activation of Cardiac Gene Expression by Myocardin, a Transcriptional Cofactor for Serum Response Factor

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SUMOylation of Krüppel-like transcription factor 5 acts as a molecular switch in transcriptional programs of lipid metabolism involving PPAR-δ

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The AKT/IκB kinase pathway promotes angiogenic/metastatic gene expression in colorectal cancer by activating nuclear factor-κB and β-catenin

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Ubiquitin–proteasome degradation of KLF5 transcription factor in cancer and untransformed epithelial cells

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Interferon α/β Promotes Cell Survival by Activating Nuclear Factor κB through Phosphatidylinositol 3-Kinase and Akt

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Internalin B Activates Nuclear Factor-κB via Ras, Phosphoinositide 3-Kinase, and Akt

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Krüppel-like Factor 4 Promotes Differentiation by Transforming Growth Factor-β Receptor-mediated Smad and p38 MAPK Signaling in Vascular Smooth Muscle Cells

inferredcites →https://doi.org/10.1074/jbc.m405100200openalex-snapshot

Insulin Receptor Substrate-1/SHP-2 Interaction, a Phenotype-dependent Switching Machinery of Insulin-like Growth Factor-I Signaling in Vascular Smooth Muscle Cells

inferredcites →https://doi.org/10.1074/jbc.m410578200openalex-snapshot

The Deacetylase HDAC1 Negatively Regulates the Cardiovascular Transcription Factor Krüppel-like Factor 5 through Direct Interaction

inferredcites →https://doi.org/10.1074/jbc.m412862200openalex-snapshot

Kruppel-like Factor 4 Abrogates Myocardin-induced Activation of Smooth Muscle Gene Expression

inferredcites →https://doi.org/10.1074/jbc.m603413200openalex-snapshot

Protein Inhibitor of Activated STAT1 Interacts with and Up-regulates Activities of the Pro-proliferative Transcription Factor Krüppel-like Factor 5

inferredcites →https://doi.org/10.1074/jbc.m701847200openalex-snapshot

Krüppel-like Factor 4 Is Acetylated by p300 and Regulates Gene Transcription via Modulation of Histone Acetylation

inferredcites →https://doi.org/10.1074/jbc.m709300200openalex-snapshot

Activation of KLF8 Transcription by Focal Adhesion Kinase in Human Ovarian Epithelial and Cancer Cells

inferredcites →https://doi.org/10.1074/jbc.m803612200openalex-snapshot

SUMOylation Regulates Nuclear Localization of Krüppel-like Factor 5

inferredcites →https://doi.org/10.1074/jbc.m806230200openalex-snapshot

Krüppel-like Factor 5 Shows Proliferation-specific Roles in Vascular Remodeling, Direct Stimulation of Cell Growth, and Inhibition of Apoptosis

inferredcites →https://doi.org/10.1089/ten.teb.2009.0630openalex-snapshot

Molecular Regulation of Contractile Smooth Muscle Cell Phenotype: Implications for Vascular Tissue Engineering

inferredcites →https://doi.org/10.1093/jb/mvp115openalex-snapshot

Angiotensin II Stimulates KLF5 Phosphorylation and its Interaction with c-Jun Leading to Suppression of p21 Expression in Vascular Smooth Muscle Cells

inferredcites →https://doi.org/10.1093/nar/28.9.1955openalex-snapshot

Human Kruppel-like Factor 8: a CACCC-box binding protein that associates with CtBP and represses transcription

inferredcites →https://doi.org/10.1111/j.1538-7836.2005.01366.xopenalex-snapshot

Significance of the transcription factor KLF5 in cardiovascular remodeling

inferredcites →https://doi.org/10.1124/jpet.109.163949openalex-snapshot

Cyclosporine Up-Regulates Krüppel-Like Factor-4 (KLF4) in Vascular Smooth Muscle Cells and Drives Phenotypic Modulation In Vivo

inferredcites →https://doi.org/10.1128/mcb.23.23.8528-8541.2003openalex-snapshot

Positive and Negative Regulation of the Cardiovascular Transcription Factor KLF5 by p300 and the Oncogenic Regulator SET through Interaction and Acetylation on the DNA-Binding Domain

inferredcites →https://doi.org/10.1128/mcb.25.18.8009-8023.2005openalex-snapshot

PIAS1 Activates the Expression of Smooth Muscle Cell Differentiation Marker Genes by Interacting with Serum Response Factor and Class I Basic Helix-Loop-Helix Proteins

inferredcites →https://doi.org/10.1152/physrev.00041.2003openalex-snapshot

Molecular Regulation of Vascular Smooth Muscle Cell Differentiation in Development and Disease

inferredcites →https://doi.org/10.1152/physrev.00058.2009openalex-snapshot

Mammalian Krüppel-Like Factors in Health and Diseases

inferredcites →https://doi.org/10.1152/physrev.1995.75.3.487openalex-snapshot

Regulation of differentiation of vascular smooth muscle cells

inferredcites →https://doi.org/10.1158/0008-5472.can-05-2059openalex-snapshot

Destabilization of Kruppel-Like Factor 4 Protein in Response to Serum Stimulation Involves the Ubiquitin-Proteasome Pathway

inferredcites →https://doi.org/10.1161/01.cir.102.20.2528openalex-snapshot

Regulated Expression of the BTEB2 Transcription Factor in Vascular Smooth Muscle Cells

inferredcites →https://doi.org/10.1161/01.res.0000068405.49081.09openalex-snapshot

Myocardin Is a Key Regulator of CArG-Dependent Transcription of Multiple Smooth Muscle Marker Genes

inferredcites →https://doi.org/10.1161/circresaha.108.176974openalex-snapshot

Conditional Deletion of Kruppel-Like Factor 4 Delays Downregulation of Smooth Muscle Cell Differentiation Markers but Accelerates Neointimal Formation Following Vascular Injury

inferredcites →https://doi.org/10.1248/bpb.29.2004openalex-snapshot

Regulation of Angiotensin II-Induced Krueppel-Like Factor 5 Expression in Vascular Smooth Muscle Cells

inferredcites →https://doi.org/10.2337/diabetes.52.10.2562openalex-snapshot

Insulin Affects Vascular Smooth Muscle Cell Phenotype and Migration Via Distinct Signaling Pathways

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Phenotypic switching of vascular smooth muscle cells in atherosclerosis, hypertension, and aortic dissection

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Smooth muscle cell differentiation: Mechanisms and models for vascular diseases

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ZFP36L1 controls KLF16 mRNA stability in vascular smooth muscle cells during restenosis after vascular injury

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Comparative Studies of Fibrin-Based Engineered Vascular Tissues and Notch Signaling from Progenitor Cells

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Current knowledge of Krüppel-like factor 5 and vascular remodeling: providing insights for therapeutic strategies

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MYST1 ‐Mediated Lysine Acetylation Stabilizes KLF4 to Promote Intimal Hyperplasia

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Single-cell transcriptomic analyses reveal the cellular and genetic basis of aquatic locomotion in scyphozoan jellyfish

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Inhibition of lysine‐specific demethylase 1A suppresses neointimal hyperplasia by targeting bone morphogenetic protein 2 and mediating vascular smooth muscle cell phenotype

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Procoagulant effects of extracellular vesicles in patients with livedo reticularis

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Targeting the Crosstalk of Immune Response and Vascular Smooth Muscle Cells Phenotype Switch for Arteriovenous Fistula Maturation

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miR-92a regulates the expression levels of matrix metalloproteinase 9 and tissue inhibitor of metalloproteinase 3 via sirtuin 1 signaling in hydrogen peroxide-induced vascular smooth muscle cells

inferred← citeshttps://doi.org/10.3906/sag-1710-173openalex-snapshot

MiR-9 promotes the phenotypic switch of vascular smooth muscle cells by targeting KLF5

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Biophysically stressed vascular smooth muscle cells express MCP-1 via a PDGFR-β-HMGB1 signaling pathway

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