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Nitric Oxide Synthase Expressions in ADR-induced Cardiomyopathy in Rats
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Nicorandil Protects Against Lethal Ischemic Ventricular Arrhythmias and Up-regulates Endothelial Nitric Oxide Synthase Expression and Sulfonylurea Receptor 2 mRNA in Conscious Rats with Acute Myocardial Infarction
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Up‐regulation of cardiac nitric oxide synthase 1‐derived nitric oxide after myocardial infarction in senescent rats
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Alterations in Myocardial Cytoskeletal and Regulatory Protein Expression Following a Single Doxorubicin Injection
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Modulation of cardiac contraction, relaxation and rate by the endothelial nitric oxide synthase (eNOS): lessons from genetically modified mice
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Cardiomyocyte-Restricted Overexpression of Endothelial Nitric Oxide Synthase ( NOS3 ) Attenuates β-Adrenergic Stimulation and Reinforces Vagal Inhibition of Cardiac Contraction
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β-Adrenergic Blockade in Developing Heart Failure
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Fas-Mediated Apoptosis in Adriamycin-Induced Cardiomyopathy in Rats
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Augmented Expression of Neuronal Nitric Oxide Synthase in the Atria Parasympathetically Decreases Heart Rate During Acute Myocardial Infarction in Rats
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Cardiac-Specific Overexpression of Inducible Nitric Oxide Synthase Does Not Result in Severe Cardiac Dysfunction
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Catechin protects against oxidative stress and inflammatory-mediated cardiotoxicity in adriamycin-treated rats
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Anthracycline-Related Heart Failure: Certain Knowledge and Open Questions
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Esculetin attenuates doxorubicin-induced cardiac toxicity: Evidence from gene expression, enzyme activity, and molecular docking analyses
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Anti-inflammatory activity and selective inhibition of iNOS gene expression by a polyherbal formulation
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Recent progress in doxorubicin-induced cardiotoxicity and protective potential of natural products
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Beneficial effects of carbon monoxide-releasing molecule-2 (CORM-2) on acute doxorubicin cardiotoxicity in mice: Role of oxidative stress and apoptosis
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Iron chelation-afforded cardioprotection against chronic anthracycline cardiotoxicity: A study of salicylaldehyde isonicotinoyl hydrazone (SIH)
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Doxorubicin-induced cardiomyopathy: From molecular mechanisms to therapeutic strategies
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Anthracycline-induced cardiotoxicity: Overview of studies examining the roles of oxidative stress and free cellular iron
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How to Protect Doxorubicin-Induced Cardiomyopathy in Male Albino Rats?
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Protection from Doxorubicin-Induced Cardiomyopathy Using the Modified Anthracycline N-Benzyladriamycin-14-valerate (AD 198)
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Activation of Nuclear Factor-κB Pathway by Simvastatin and RhoA Silencing Increases Doxorubicin Cytotoxicity in Human Colon Cancer HT29 Cells
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Role of superoxide, nitric oxide, and peroxynitrite in doxorubicin-induced cell death in vivo and in vitro
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Chemotherapy-Induced Cardiotoxicity: Overview of the Roles of Oxidative Stress
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Oxidative Stress and Cellular Response to Doxorubicin: A Common Factor in the Complex Milieu of Anthracycline Cardiotoxicity
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Role of Oxidative Stress and Inflammation in Doxorubicin-Induced Cardiotoxicity: A Brief Account
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Structural and Functional Remodeling of Rat Cardiac Tissue in Doxorubicin-Induced Chronic Heart Failure and Its Pharmacological Adjustment by a Newly Synthesized 4-Amino-1,2,4-Triazole Compound
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Doxorubicin-Induced Cardiotoxicity: From Mechanisms to Development of Efficient Therapy
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Doxorubicin Cardiotoxicity: Multiple Targets and Translational Perspectives
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