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Single‐Vesicle Electrochemistry Following Repetitive Stimulation Reveals a Mechanism for Plasticity Changes with Iron Deficiency

https://doi.org/10.1002/anie.202200716

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Nanoelectrode for Amperometric Monitoring of Individual Vesicular Exocytosis Inside Single Synapses

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Nanoelectrode for Amperometric Monitoring of Individual Vesicular Exocytosis Inside Single Synapses

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Quantitative Measurement of Transmitters in Individual Vesicles in the Cytoplasm of Single Cells with Nanotip Electrodes

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Single‐Vesicle Electrochemistry Following Repetitive Stimulation Reveals a Mechanism for Plasticity Changes with Iron Deficiency

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Electrochemical Quantification of Neurotransmitters in Single Live Cell Vesicles Shows Exocytosis is Predominantly Partial

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Iron deficiency on neuronal function

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Actions of (±)-7-hydroxy-N,N-dipropylaminotetralin (7-OH-DPAT) on dopamine synthesis in limbic and extrapyramidal regions of rat brain

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Motor cortex excitability in restless legs syndrome

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Abnormally increased CSF 3-Ortho-methyldopa (3-OMD) in untreated restless legs syndrome (RLS) patients indicates more severe disease and possibly abnormally increased dopamine synthesis

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Analytical Techniques: Shedding Light upon Nanometer-Sized Secretory Vesicles

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Iron chelation down-regulates dopamine transporter expression by decreasing mRNA stability and increasing endocytosis in N2a cells

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The role of iron in brain ageing and neurodegenerative disorders

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The evidence for open and closed exocytosis as the primary release mechanism

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Early life nutrition and neural plasticity

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Quantitative Chemical Measurements of Vesicular Transmitters with Electrochemical Cytometry

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Actin Controls the Vesicular Fraction of Dopamine Released During Extended Kiss and Run Exocytosis

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Only a Fraction of Quantal Content is Released During Exocytosis as Revealed by Electrochemical Cytometry of Secretory Vesicles

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Is plasticity of synapses the mechanism of long-term memory storage?

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Synaptic Plasticity: Multiple Forms, Functions, and Mechanisms

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Protein kinase C activation decreases peripheral actin network density and increases central nonmuscle myosin II contractility in neuronal growth cones

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Altered dopaminergic profile in the putamen and substantia nigra in restless leg syndrome

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Absence of Postexercise and Delayed Facilitation of Motor Cortex Excitability in Restless Legs Syndrome: Evidence of Altered Cortical Plasticity?

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Brain iron deficiency and affected contextual fear memory in mice with conditional Ferroportin1 ablation in the brain

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Regulation of striatal tyrosine hydroxylase phosphorylation by acute and chronic haloperidol

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Down‐regulation of dopamine transporter by iron chelationin vitrois mediated by altered trafficking, not synthesis

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The PC12 cell as model for neurosecretion

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Dopamine Autoreceptorsa Biochemical, Pharmacological, and Morphological Studies

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Changes in Synaptic Morphology Accompany Actin Signaling during LTP

inferredcites →https://doi.org/10.1523/jneurosci.18-15-05575.1998openalex-snapshot

D 2 -Like Dopamine Autoreceptor Activation Reduces Quantal Size in PC12 Cells

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cAMP-Mediated Stabilization of Fusion Pores in Cultured Rat Pituitary Lactotrophs

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Mass Spectrometric Imaging of Plasma Membrane Lipid Alteration Correlated with Amperometrically Measured Activity-Dependent Plasticity in Exocytosis

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A useful electroencephalography (EEG) marker of brain plasticity: delta waves

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