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TL1A produced by lamina propria macrophages induces Th1 and Th17 immune responses in cooperation with IL-23 in patients with Crohn's disease

https://doi.org/10.1002/ibd.21124
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/ibd.20223openalex-snapshot
TNFSF15 is an ethnic-specific IBD gene
inferredcites →https://doi.org/10.1002/ibd.20399openalex-snapshot
Contribution of TNFSF15 gene variants to Crohnʼs disease susceptibility confirmed in UK population
inferredcites →https://doi.org/10.1002/ibd.20475openalex-snapshot
IL-23/IL-17 immunity as a hallmark of Crohnʼs disease
inferredcites →https://doi.org/10.1007/s00535-005-1744-3openalex-snapshot
Novel pathophysiological concepts of inflammatory bowel disease
inferredcites →https://doi.org/10.1016/j.clim.2004.02.007openalex-snapshot
Potential role for TL1A, the new TNF-family member and potent costimulator of IFN-γ, in mucosal inflammation
inferredcites →https://doi.org/10.1016/j.clim.2008.07.014openalex-snapshot
Circulating levels of TNF-like cytokine 1A (TL1A) and its decoy receptor 3 (DcR3) in rheumatoid arthritis
inferredcites →https://doi.org/10.1016/j.immuni.2008.04.021openalex-snapshot
The TNF-Family Receptor DR3 is Essential for Diverse T Cell-Mediated Inflammatory Diseases
inferredcites →https://doi.org/10.1016/s1074-7613(02)00283-2openalex-snapshot
TL1A Is a TNF-like Ligand for DR3 and TR6/DcR3 and Functions as a T Cell Costimulator
inferredcites →https://doi.org/10.1038/nature01355openalex-snapshot
Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain
inferredcites →https://doi.org/10.1038/nature06005openalex-snapshot
Unravelling the pathogenesis of inflammatory bowel disease
inferredcites →https://doi.org/10.1038/nm0107-26openalex-snapshot
IL-23: a master regulator in Crohn disease
inferredcites →https://doi.org/10.1038/nri2340openalex-snapshot
The genetics and immunopathogenesis of inflammatory bowel disease
inferredcites →https://doi.org/10.1053/gast.2001.28021openalex-snapshot
Macrophage-derived IL-18–mediated intestinal inflammation in the murine model of Crohn's disease
inferredcites →https://doi.org/10.1053/j.gastro.2008.04.037openalex-snapshot
TL1A (TNFSF15) Regulates the Development of Chronic Colitis by Modulating Both T-Helper 1 and T-Helper 17 Activation
inferredcites →https://doi.org/10.1084/jem.20071364openalex-snapshot
TL1A–DR3 interaction regulates Th17 cell function and Th17-mediated autoimmune disease
inferredcites →https://doi.org/10.1084/jem.20072378openalex-snapshot
The Death Receptor 3–TNF-like protein 1A pathway drives adverse bone pathology in inflammatory arthritis
inferredcites →https://doi.org/10.1093/hmg/ddi379openalex-snapshot
Single nucleotide polymorphisms in TNFSF15 confer susceptibility to Crohn's disease
inferredcites →https://doi.org/10.1097/00054725-200501000-00003openalex-snapshot
Expression of Interleukin-12-Related Cytokine Transcripts in Inflammatory Bowel Disease: Elevated Interleukin-23p19 and Interleukin-27p28 in Crohnʼs Disease But Not in Ulcerative Colitis
inferredcites →https://doi.org/10.1097/01.mib.0000194183.92671.b6openalex-snapshot
Both IL-12p70 and IL-23 are synthesized during active Crohnʼs disease and are down-regulated by treatment with anti-IL-12 p40 monoclonal antibody
inferredcites →https://doi.org/10.1136/gut.2003.024190openalex-snapshot
T-bet upregulation and subsequent interleukin 12 stimulation are essential for induction of Th1 mediated immunopathology in Crohn’s disease
inferredcites →https://doi.org/10.1136/gut.2007.135053openalex-snapshot
IL23 differentially regulates the Th1/Th17 balance in ulcerative colitis and Crohn’s disease
inferredcites →https://doi.org/10.1146/annurev.med.60.051407.123757openalex-snapshot
IL-23 and Autoimmunity: New Insights into the Pathogenesis of Inflammatory Bowel Disease
inferredcites →https://doi.org/10.1172/jci34610openalex-snapshot
Unique CD14+ intestinal macrophages contribute to the pathogenesis of Crohn disease via IL-23/IFN-γ axis
inferredcites →https://doi.org/10.4049/jimmunol.0804369openalex-snapshot
Human CD14+ Macrophages in Intestinal Lamina Propria Exhibit Potent Antigen-Presenting Ability
inferredcites →https://doi.org/10.4049/jimmunol.157.3.1261openalex-snapshot
Disparate CD4+ lamina propria (LP) lymphokine secretion profiles in inflammatory bowel disease. Crohn’s disease LP cells manifest increased secretion of IFN-γ, whereas ulcerative colitis LP cells manifest increased secretion of IL-5
inferredcites →https://doi.org/10.4049/jimmunol.171.9.4868openalex-snapshot
Expression, Localization, and Functional Activity of TL1A, a Novel Th1-Polarizing Cytokine in Inflammatory Bowel Disease
inferredcites →https://doi.org/10.4049/jimmunol.172.11.7002openalex-snapshot
TL1A Synergizes with IL-12 and IL-18 to Enhance IFN-γ Production in Human T Cells and NK Cells
inferredcites →https://doi.org/10.4049/jimmunol.174.8.4985openalex-snapshot
Dominant Role for TL1A/DR3 Pathway in IL-12 plus IL-18-Induced IFN-γ Production by Peripheral Blood and Mucosal CCR9+ T Lymphocytes
inferredcites →https://doi.org/10.4049/jimmunol.178.7.4033openalex-snapshot
The T Cell Costimulator TL1A Is Induced by FcγR Signaling in Human Monocytes and Dendritic Cells

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4834005openalex-extracted
DJ-X-013 Reduces LPS-Induced Inflammation, Modulates Th17/ Myeloid-Derived Suppressor Cells, and Alters NF-kB Expression to Ameliorate Experimental Colitis
inferred← citeshttps://doi.org/10.1002/1873-3468.12747openalex-snapshot
Multifaceted death receptor 3 signaling—promoting survival and triggering death
inferred← citeshttps://doi.org/10.1002/cyto.b.21372openalex-snapshot
Expression of death receptor 3 on peripheral blood mononuclear cells differes in adult IBD patients and children with newly diagnosed IBD
inferred← citeshttps://doi.org/10.1002/ibd.21248openalex-snapshot
IL-23/IL-17 axis in IBD
inferred← citeshttps://doi.org/10.1002/jcp.27032openalex-snapshot
mTOR inhibitor INK128 attenuates dextran sodium sulfate‐induced colitis by promotion of MDSCs on Treg cell expansion
inferred← citeshttps://doi.org/10.1002/mdr2.70007openalex-snapshot
Breaking Boundaries: Chronic Diseases and the Frontiers of Immune Microenvironments
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Cytokines and Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1007/978-1-4614-8256-7_15openalex-snapshot
Cytokines in Inflammatory Bowel Disease
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Intestinal Macrophages: Specialized Antigen-Presenting Cells at the Front Line
inferred← citeshttps://doi.org/10.1007/978-3-030-84164-5_4openalex-snapshot
Macrophage-Associated Disorders: Pathophysiology, Treatment Challenges, and Possible Solutions
inferred← citeshttps://doi.org/10.1007/978-3-031-14744-9_3openalex-snapshot
Cytokines and Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1007/978-3-0348-0620-6_124-1openalex-snapshot
Inflammatory Bowel Disease Models in Animals
inferred← citeshttps://doi.org/10.1007/978-3-319-49215-5_3openalex-snapshot
Cytokines and Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1007/978-3-7643-8550-7_124openalex-snapshot
Inflammatory Bowel Disease Models in Animals
inferred← citeshttps://doi.org/10.1007/s00228-013-1559-8openalex-snapshot
Elevated risk of opportunistic viral infection in patients with Crohn’s disease during biological therapies: a meta analysis of randomized controlled trials
inferred← citeshttps://doi.org/10.1007/s00281-014-0444-6openalex-snapshot
Use of animal models in elucidating disease pathogenesis in IBD
inferred← citeshttps://doi.org/10.1007/s10620-012-2304-3openalex-snapshot
Myosin Light Chain Kinase Inhibitor Inhibits Dextran Sulfate Sodium-Induced Colitis in Mice
inferred← citeshttps://doi.org/10.1007/s11427-020-1720-9openalex-snapshot
MAP3K2 augments Th1 cell differentiation via IL-18 to promote T cell-mediated colitis
inferred← citeshttps://doi.org/10.1016/b978-0-12-384929-8.00060-5openalex-snapshot
Inflammatory Bowel Diseases
inferred← citeshttps://doi.org/10.1016/b978-0-12-394596-9.00009-3openalex-snapshot
Animal Models of Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-415847-4.00082-3openalex-snapshot
Crohn’s Disease
inferred← citeshttps://doi.org/10.1016/j.addr.2019.12.001openalex-snapshot
Drug delivery to macrophages: A review of targeting drugs and drug carriers to macrophages for inflammatory diseases
inferred← citeshttps://doi.org/10.1016/j.ajpath.2015.02.024openalex-snapshot
Intestinal Antigen-Presenting Cells
inferred← citeshttps://doi.org/10.1016/j.bbi.2011.07.236openalex-snapshot
Role of resveratrol-induced CD11b+ Gr-1+ myeloid derived suppressor cells (MDSCs) in the reduction of CXCR3+ T cells and amelioration of chronic colitis in IL-10−/− mice
inferred← citeshttps://doi.org/10.1016/j.biopha.2024.117379openalex-snapshot
DJ-X-013 reduces LPS-induced inflammation, modulates Th17/ myeloid-derived suppressor cells, and alters NF-κB expression to ameliorate experimental colitis
inferred← citeshttps://doi.org/10.1016/j.biopha.2025.117939openalex-snapshot
Modulation of occludin, NF-κB, p-STAT3, and Th17 response by DJ-X-025 decreases inflammation and ameliorates experimental colitis
inferred← citeshttps://doi.org/10.1016/j.clim.2010.07.001openalex-snapshot
High intestinal and systemic levels of decoy receptor 3 (DcR3) and its ligand TL1A in active ulcerative colitis
inferred← citeshttps://doi.org/10.1016/j.csbj.2020.10.008openalex-snapshot
Functional roles in cell signaling of adaptor protein TRADD from a structural perspective
inferred← citeshttps://doi.org/10.1016/j.cyto.2013.04.004openalex-snapshot
Multiple activating and repressive cis -promoter regions regulate TNFSF15 expression in human primary mononuclear cells
inferred← citeshttps://doi.org/10.1016/j.cyto.2014.04.007openalex-snapshot
TL1A increased the differentiation of peripheral Th17 in rheumatoid arthritis
inferred← citeshttps://doi.org/10.1016/j.cyto.2015.10.008openalex-snapshot
Chemokine and cytokine levels in inflammatory bowel disease patients
inferred← citeshttps://doi.org/10.1016/j.cytogfr.2018.12.002openalex-snapshot
IL-23 in inflammatory bowel diseases and colon cancer
inferred← citeshttps://doi.org/10.1016/j.dci.2020.103685openalex-snapshot
Cloning and characterization of tumor necrosis factor superfamily 15 in rock bream, Oplegnathus fasciatus; phylogenetic, in silico, and expressional analysis
inferred← citeshttps://doi.org/10.1016/j.dld.2011.09.002openalex-snapshot
Differential expression of the TL1A/DcR3 system of TNF/TNFR-like proteins in large vs. small intestinal Crohn's disease
inferred← citeshttps://doi.org/10.1016/j.febslet.2010.04.030openalex-snapshot
Functional signaling of membrane‐bound TL1A induces IFN‐γ expression
inferred← citeshttps://doi.org/10.1016/j.humimm.2013.11.006openalex-snapshot
Genetic risk and protective factors of TNFSF15 gene variants detected using single nucleotide polymorphisms in Hungarians with psoriasis and psoriatic arthritis
inferred← citeshttps://doi.org/10.1016/j.isci.2026.117795openalex-snapshot
Bispecific antibodies targeting TL1A and IL-12/23p40 suppress pathogenic Th1/Th17 responses and ameliorate colitis in mice
inferred← citeshttps://doi.org/10.1016/j.jff.2016.04.038openalex-snapshot
γ-Glutamyl valine supplementation-induced mitigation of gut inflammation in a porcine model of colitis
inferred← citeshttps://doi.org/10.1016/j.lfs.2020.118220openalex-snapshot
TL1A primed dendritic cells activation exacerbated chronic murine colitis
inferred← citeshttps://doi.org/10.1016/j.medj.2024.03.010openalex-snapshot
TL1A inhibition for inflammatory bowel disease treatment: From inflammation to fibrosis
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2012.10.008openalex-snapshot
Immune aspects of the pathogenesis of inflammatory bowel disease
inferred← citeshttps://doi.org/10.1016/j.rmclc.2019.06.004openalex-snapshot
ETIOLOGÍA Y FISIOPATOLOGÍA DE LA ENFERMEDAD INFLAMATORIA INTESTINAL
inferred← citeshttps://doi.org/10.1016/j.trsl.2016.08.007openalex-snapshot
Crohn's disease-associated mucosal factors regulate the expression of TNF-like cytokine 1A and its receptors in primary subepithelial intestinal myofibroblasts and intestinal epithelial cells
inferred← citeshttps://doi.org/10.1016/j.vaccine.2011.06.032openalex-snapshot
Strong viremia control in vaccinated macaques does not prevent gradual Th17 cell loss from central memory
inferred← citeshttps://doi.org/10.1016/s2468-1253(25)00071-8openalex-snapshot
Safety and efficacy of the anti-TL1A monoclonal antibody tulisokibart for Crohn's disease: a phase 2a induction trial
inferred← citeshttps://doi.org/10.1038/mi.2014.87openalex-snapshot
A major population of mucosal memory CD4+ T cells, coexpressing IL-18Rα and DR3, display innate lymphocyte functionality
inferred← citeshttps://doi.org/10.1038/nrgastro.2015.206openalex-snapshot
Lessons learned — resolving the enigma of genetic factors in IBS
inferred← citeshttps://doi.org/10.1038/nrgastro.2015.52openalex-snapshot
The unusual suspects—innate lymphoid cells as novel therapeutic targets in IBD
inferred← citeshttps://doi.org/10.1038/nri3661openalex-snapshot
Cytokines in inflammatory bowel disease
inferred← citeshttps://doi.org/10.1038/srep37531openalex-snapshot
A Novel Role of Spred2 in the Colonic Epithelial Cell Homeostasis and Inflammation
inferred← citeshttps://doi.org/10.1053/j.gastro.2010.05.040openalex-snapshot
Imbalance of NKp44+NKp46− and NKp44−NKp46+ Natural Killer Cells in the Intestinal Mucosa of Patients With Crohn's Disease
inferred← citeshttps://doi.org/10.1053/j.gastro.2011.02.016openalex-snapshot
Proinflammatory Cytokines in the Pathogenesis of Inflammatory Bowel Diseases
inferred← citeshttps://doi.org/10.1084/jem.20140678openalex-snapshot
CX3CR1+ mononuclear phagocytes support colitis-associated innate lymphoid cell production of IL-22
inferred← citeshttps://doi.org/10.1093/bfgp/elr009openalex-snapshot
Genome-wide association studies and Crohn's disease
inferred← citeshttps://doi.org/10.1093/ecco-jcc/jjaa165openalex-snapshot
Gut Microbial Transcytosis Induced by Tumour Necrosis Factor-like 1A-dependent Activation of a Myosin Light Chain Kinase Splice Variant Contributes to Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1093/ecco-jcc/jjag039openalex-snapshot
TL1A as a therapeutic renaissance in inflammatory bowel disease: a systematic review from molecular mechanisms to clinical translation
inferred← citeshttps://doi.org/10.1093/ibd/izag172openalex-snapshot
Targeting the TL1A pathway in inflammatory bowel disease: mechanistic insights and emerging therapeutic pipeline
inferred← citeshttps://doi.org/10.1096/fj.11-200618openalex-snapshot
The death receptor 3/TL1A pathway is essential for efficient development of antiviral CD4 + and CD8 + T‐cell immunity
inferred← citeshttps://doi.org/10.1097/md.0000000000000613openalex-snapshot
IL12p40 Regulates Functional Development of Human CD4+ T Cells
inferred← citeshttps://doi.org/10.1097/mib.0000000000000492openalex-snapshot
Tumor Necrosis Factor–like Cytokine TL1A and Its Receptors DR3 and DcR3
inferred← citeshttps://doi.org/10.1097/mib.0000000000000500openalex-snapshot
Genetic Markers Associated with Clinical Outcomes in Patients with Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1097/mog.0b013e328339d099openalex-snapshot
Proinflammatory cytokines underlying the inflammation of Crohnʼs disease
inferred← citeshttps://doi.org/10.1111/ijfs.14077openalex-snapshot
Effects of a synthetic di‐phosphoserine peptide (SS‐2) on gene expression profiling against TNF‐α induced inflammation
inferred← citeshttps://doi.org/10.1111/j.1600-0625.2011.01304.xopenalex-snapshot
Upregulation and nuclear localization of TNF‐like Cytokine 1A (TL1A) and its receptors DR3 and DcR3 in psoriatic skin lesions
inferred← citeshttps://doi.org/10.1111/jgh.12674openalex-snapshot
Smoking behaviour modifies IL23r‐associated disease risk in patients with Crohn's disease
inferred← citeshttps://doi.org/10.1111/sji.12222openalex-snapshot
Soluble CD 163, a Specific Macrophage Activation Marker, is Decreased by Anti‐ TNF ‐ α Antibody Treatment in Active Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1111/sji.12324openalex-snapshot
TL 1A as a Potential Local Inducer of IL 17A Expression in Colon Mucosa of Inflammatory Bowel Disease Patients
inferred← citeshttps://doi.org/10.1128/9781555819194.ch39openalex-snapshot
Intestinal Mononuclear Phagocytes in Health and Disease
inferred← citeshttps://doi.org/10.1128/microbiolspec.mchd-0047-2016openalex-snapshot
Intestinal Mononuclear Phagocytes in Health and Disease
inferred← citeshttps://doi.org/10.1136/annrheumdis-2020-218719openalex-snapshot
Paradoxical gastrointestinal effects of interleukin-17 blockers
inferred← citeshttps://doi.org/10.1136/gutjnl-2011-301213openalex-snapshot
Identifying and testing candidate genetic polymorphisms in the irritable bowel syndrome (IBS): association with TNFSF15 and TNFα
inferred← citeshttps://doi.org/10.1136/gutjnl-2011-301668openalex-snapshot
Secukinumab, a human anti-IL-17A monoclonal antibody, for moderate to severe Crohn's disease: unexpected results of a randomised, double-blind placebo-controlled trial
inferred← citeshttps://doi.org/10.1136/gutjnl-2012-303063openalex-snapshot
Mucosal CXCR4+ IgG plasma cells contribute to the pathogenesis of human ulcerative colitis through FcγR-mediated CD14 macrophage activation
inferred← citeshttps://doi.org/10.1155/2013/258164openalex-snapshot
The Role of TL1A and DR3 in Autoimmune and Inflammatory Diseases
inferred← citeshttps://doi.org/10.1155/2013/425915openalex-snapshot
High Intestinal and Systemic Levels of Interleukin-23/T-Helper 17 Pathway in Chinese Patients with Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1155/2014/325129openalex-snapshot
Tumour Necrosis Factor Superfamily Members in the Pathogenesis of Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1155/2017/6567432openalex-snapshot
Morinda citrifolia(Noni) Fruit Juice Reduces Inflammatory Cytokines Expression and Contributes to the Maintenance of Intestinal Mucosal Integrity in DSS Experimental Colitis
inferred← citeshttps://doi.org/10.1155/2019/7247238openalex-snapshot
A Comprehensive Review and Update on the Pathogenesis of Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1159/000445133openalex-snapshot
The Role of T-Cell Subsets in Chronic Inflammation in Celiac Disease and Inflammatory Bowel Disease Patients: More Common Mechanisms or More Differences?
inferred← citeshttps://doi.org/10.1159/000475439openalex-snapshot
Identification of Two Additional Susceptibility Loci for Inflammatory Bowel Disease in a Chinese Populationy
inferred← citeshttps://doi.org/10.1177/0300060520961675openalex-snapshot
Polymorphism rs6478109 in the TNFSF15 gene contributes to the susceptibility to Crohn’s disease but not ulcerative colitis: a meta-analysis
inferred← citeshttps://doi.org/10.1186/s12929-017-0347-7openalex-snapshot
Decoy receptor 3: an endogenous immunomodulator in cancer growth and inflammatory reactions
inferred← citeshttps://doi.org/10.1186/s12948-015-0022-zopenalex-snapshot
TL1A/DR3 axis involvement in the inflammatory cytokine network during pulmonary sarcoidosis
inferred← citeshttps://doi.org/10.1189/jlb.3a0316-129ropenalex-snapshot
The TNF family member TL1A induces IL-22 secretion in committed human T h 17 cells via IL-9 induction
inferred← citeshttps://doi.org/10.1189/jlb.3ri0315-095ropenalex-snapshot
The TNF-family cytokine TL1A: from lymphocyte costimulator to disease co-conspirator
inferred← citeshttps://doi.org/10.1371/journal.pone.0060136openalex-snapshot
The TNF-Family Cytokine TL1A Inhibits Proliferation of Human Activated B Cells
inferred← citeshttps://doi.org/10.1515/rjim-2015-0016openalex-snapshot
Cytokines in inflammatory bowel disease
inferred← citeshttps://doi.org/10.1556/eujmi.1.2011.1.9openalex-snapshot
CD161 defines effector T cells that express light and respond to TL1A-DR3 signaling
inferred← citeshttps://doi.org/10.1556/eujmi.3.2013.1.2openalex-snapshot
Sustained TL1A (TNFSF15) expression on both lymphoid and myeloid cells leads to mild spontaneous intestinal inflammation and fibrosis
inferred← citeshttps://doi.org/10.2147/ceg.s521893openalex-snapshot
Optimising Monoclonal Antibody Drug Development for Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.2147/itt.s282466openalex-snapshot
All are Equal, Some are More Equal: Targeting IL 12 and 23 in IBD – A Clinical Perspective
inferred← citeshttps://doi.org/10.2169/naika.102.2195openalex-snapshot
4. Recent Progress of Research and Clinics in Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.2169/naika.102.92aopenalex-snapshot
4. Recent Progress of Research and Clinics in Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.2174/092986711797535263openalex-snapshot
Polyphenol Supplementation as a Complementary Medicinal Approach to Treating Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.2174/1573395511309020003openalex-snapshot
Interleukins Involved in Inflammatory Bowel Disease as New Therapeutic Targets
inferred← citeshttps://doi.org/10.2492/inflammregen.30.412openalex-snapshot
Intestinal macrophage, "a double-edged sword" for homeostasis and inflammation in the gut
inferred← citeshttps://doi.org/10.2492/inflammregen.34.078openalex-snapshot
Analysis of disease-pathways by susceptibility genes in primary biliary cirrhosis
inferred← citeshttps://doi.org/10.24996/ijs.2021.62.8.5openalex-snapshot
Roles of IL-17A and IL-23 in the Pathogenesis of Ulcerative Colitis and Crohn’s Disease
inferred← citeshttps://doi.org/10.3109/00365521.2015.1134649openalex-snapshot
Reduced numbers of mucosal DRintmacrophages and increased numbers of CD103+dendritic cells during anti-TNF-α treatment in patients with Crohn’s disease
inferred← citeshttps://doi.org/10.3109/07853890.2010.531758openalex-snapshot
Th17-cytokine blockers as a new approach for treating inflammatory bowel disease
inferred← citeshttps://doi.org/10.3389/fimmu.2019.00361openalex-snapshot
Reduced CD27−IgD− B Cells in Blood and Raised CD27−IgD− B Cells in Gut-Associated Lymphoid Tissue in Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.3389/fimmu.2019.00583openalex-snapshot
TL1A (TNFSF15) and DR3 (TNFRSF25): A Co-stimulatory System of Cytokines With Diverse Functions in Gut Mucosal Immunity
inferred← citeshttps://doi.org/10.3389/fimmu.2019.01140openalex-snapshot
The Role of Monocytes and Macrophages in Autoimmune Diseases: A Comprehensive Review
inferred← citeshttps://doi.org/10.3389/fimmu.2020.00410openalex-snapshot
Human Intestinal Mononuclear Phagocytes in Health and Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.3389/fimmu.2025.1524058openalex-snapshot
The role of efferocytosis in inflammatory bowel disease
inferred← citeshttps://doi.org/10.3389/fimmu.2026.1873829openalex-snapshot
Drug-associated inflammatory bowel disease: a real-world pharmacovigilance study using the FAERS and JADER databases
inferred← citeshttps://doi.org/10.3390/cells10081847openalex-snapshot
Translating Treg Therapy for Inflammatory Bowel Disease in Humanized Mice
inferred← citeshttps://doi.org/10.3390/ijms21134748openalex-snapshot
Role of Inflammation in Pathophysiology of Colonic Disease: An Update
inferred← citeshttps://doi.org/10.3390/ijms26115017openalex-snapshot
TL1A as a Target in Inflammatory Bowel Disease: Exploring Mechanisms and Therapeutic Potential
inferred← citeshttps://doi.org/10.3390/molecules24030513openalex-snapshot
A Role of Exopolysaccharide Produced by Streptococcus thermophilus in the Intestinal Inflammation and Mucosal Barrier in Caco-2 Monolayer and Dextran Sulphate Sodium-Induced Experimental Murine Colitis
inferred← citeshttps://doi.org/10.3748/wjg.v18.i41.5848openalex-snapshot
Changes of the cytokine profile in inflammatory bowel diseases
inferred← citeshttps://doi.org/10.3748/wjg.v21.i28.8711openalex-snapshot
Interleukin-18 genetic polymorphisms contribute differentially to the susceptibility to Crohn’s disease
inferred← citeshttps://doi.org/10.3892/br.2019.1216openalex-snapshot
Expression profiling of genes in rheumatoid fibroblast‑like synoviocytes regulated by tumor necrosis factor‑like ligand 1A using cDNA microarray analysis
inferred← citeshttps://doi.org/10.3892/ijmm.2013.1294openalex-snapshot
Role of metallothionein in murine experimental colitis
inferred← citeshttps://doi.org/10.3892/ijmm.2013.1461openalex-snapshot
Decoy receptor 3 regulates the expression of various genes in rheumatoid arthritis synovial fibroblasts
inferred← citeshttps://doi.org/10.3892/mco.2025.2878openalex-snapshot
Mendelian randomization analysis provides insights into the relationship between inflammatory bowel disease and skin cancer
inferred← citeshttps://doi.org/10.4049/jimmunol.1002535openalex-snapshot
The Tachykinins Substance P and Hemokinin-1 Favor the Generation of Human Memory Th17 Cells by Inducing IL-1β, IL-23, and TNF-Like 1A Expression by Monocytes
inferred← citeshttps://doi.org/10.4291/wjgp.v9.i4.73openalex-snapshot
Role of TNFSF15 in the intestinal inflammatory response
inferred← citeshttps://doi.org/10.5217/ir.2012.10.4.317openalex-snapshot
Immunological Abnormalities in the Pathogenesis of Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.70675/910c14fdzc720z4a3fza364z6abf25ce9c20openalex-snapshot
Rôle de l’IL-33 et des cellules lymphoïdes innées de type 2 dans l’inflammation allergique pulmonaire
inferred← citeshttps://doi.org/10.70675/af9bbfd9za4f8z4226za81bzecbc4ab31742openalex-snapshot
Rôle des serpines, inhibiteurs de protéases à serine, du microbiote digestif humain dans les maladies inflammatoires de l'intestin
inferred← citeshttps://doi.org/10.70675/c303a161z5e08z4284za4baz590c01ce4043openalex-snapshot
Analyse fonctionnelle de bactéries du microbiote intestinal humain et évaluation de leurs propriétés bénéfiques contre l’obésité et l’inflammation intestinale
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