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DJ-X-013 Reduces LPS-Induced Inflammation, Modulates Th17/ Myeloid-Derived Suppressor Cells, and Alters NF-kB Expression to Ameliorate Experimental Colitis

https://doi.org/10.2139/ssrn.4834005
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inferredcites →https://doi.org/10.1002/ibd.21124openalex-extracted
TL1A produced by lamina propria macrophages induces Th1 and Th17 immune responses in cooperation with IL-23 in patients with Crohn's disease
inferredcites →https://doi.org/10.1016/j.bcp.2014.12.007openalex-extracted
Transcription factor NF-κB associates with microtubules and stimulates apoptosis in response to suppression of microtubule dynamics in MCF-7 cells
inferredcites →https://doi.org/10.1016/j.biopha.2018.02.060openalex-extracted
Intraperitoneal administration of mesenchymal stem cells ameliorates acute dextran sulfate sodium-induced colitis by suppressing dendritic cells
inferredcites →https://doi.org/10.1016/j.biopha.2023.114514openalex-extracted
Piceatannol induces regulatory T cells and modulates the inflammatory response and adipogenesis
inferredcites →https://doi.org/10.1016/j.cellimm.2021.104364openalex-extracted
Blockade of TLRs-triggered macrophage activation by caffeic acid exerted protective effects on experimental ulcerative colitis
inferredcites →https://doi.org/10.1016/j.clim.2003.09.013openalex-extracted
Neutralization of interleukin-17 aggravates dextran sulfate sodium-induced colitis in mice
inferredcites →https://doi.org/10.1016/j.jalz.2013.02.003openalex-extracted
2013 Alzheimer's disease facts and figures
inferredcites →https://doi.org/10.1016/j.tcb.2013.09.005openalex-extracted
Immune synapse: conductor of orchestrated organelle movement
inferredcites →https://doi.org/10.1016/s0140-6736(17)32448-0openalex-extracted
Worldwide incidence and prevalence of inflammatory bowel disease in the 21st century: a systematic review of population-based studies
inferredcites →https://doi.org/10.1016/s0140-6736(23)00457-9openalex-extracted
Incidence, prevalence, and co-occurrence of autoimmune disorders over time and by age, sex, and socioeconomic status: a population-based cohort study of 22 million individuals in the UK
inferredcites →https://doi.org/10.1016/s0169-409x(00)00129-0openalex-extracted
Experimental and computational approaches to estimate solubility and permeability in drug discovery and development settings 1PII of original article: S0169-409X(96)00423-1. The article was originally published in Advanced Drug Delivery Reviews 23 (1997) 3–25. 1
inferredcites →https://doi.org/10.1016/s2468-1253(19)30333-4openalex-extracted
The global, regional, and national burden of inflammatory bowel disease in 195 countries and territories, 1990–2017: a systematic analysis for the Global Burden of Disease Study 2017
inferredcites →https://doi.org/10.1038/nm0996-998openalex-extracted
Local administration of antisense phosphorothiate olignucleotides to the p65 subunit of NF–κB abrogates established experimental colitis in mice
inferredcites →https://doi.org/10.1038/nri2506openalex-extracted
Myeloid-derived suppressor cells as regulators of the immune system
inferredcites →https://doi.org/10.1038/s41577-020-00490-yopenalex-extracted
Myeloid-derived suppressor cells in the era of increasing myeloid cell diversity
inferredcites →https://doi.org/10.1038/s41591-019-0675-0openalex-extracted
Chronic inflammation in the etiology of disease across the life span
inferredcites →https://doi.org/10.1038/srep42717openalex-extracted
SwissADME: a free web tool to evaluate pharmacokinetics, drug-likeness and medicinal chemistry friendliness of small molecules
inferredcites →https://doi.org/10.1053/j.gastro.2008.06.032openalex-extracted
Myeloid-Derived Suppressor Cells in Inflammatory Bowel Disease: A New Immunoregulatory Pathway
inferredcites →https://doi.org/10.1073/pnas.022649799openalex-extracted
Human macrophage activation programs induced by bacterial pathogens
inferredcites →https://doi.org/10.1073/pnas.0809317106openalex-extracted
Invariant NKT cells inhibit development of the Th 17 lineage
inferredcites →https://doi.org/10.1080/00365521.2018.1447597openalex-extracted
Immunopathogenesis of inflammatory bowel disease and mechanisms of biological therapies
inferredcites →https://doi.org/10.1080/08916934.2017.1283405openalex-extracted
Aberrant function of myeloid-derived suppressor cells (MDSCs) in experimental colitis and in inflammatory bowel disease (IBD) immune responses
inferredcites →https://doi.org/10.1089/jir.2007.0059openalex-extracted
CXCL10-Producing Mucosal CD4 + T Cells, NK Cells, and NKT Cells Are Associated with Chronic Colitis in IL-10 −/− Mice, Which Can Be Abrogated by Anti-CXCL10 Antibody Inhibition
inferredcites →https://doi.org/10.1093/ecco-jcc/jjad119openalex-extracted
An Intestinal Th17 Subset is Associated with Inflammation in Crohn’s Disease and Activated by Adherent-invasive Escherichia coli
inferredcites →https://doi.org/10.1093/ecco-jcc/jjx160openalex-extracted
Intestinal T Cell Profiling in Inflammatory Bowel Disease: Linking T Cell Subsets to Disease Activity and Disease Course
inferredcites →https://doi.org/10.1093/ibd/izab029openalex-extracted
Human Intestinal Macrophages Are Involved in the Pathology of Both Ulcerative Colitis and Crohn Disease
inferredcites →https://doi.org/10.1093/pcmedi/pbab025openalex-extracted
Dichotomous roles of neutrophils in modulating pathogenic and repair processes of inflammatory bowel diseases
inferredcites →https://doi.org/10.1097/01.mib.0000218764.06959.91openalex-extracted
Critical role of IL-17 receptor signaling in acute TNBS-induced colitis
inferredcites →https://doi.org/10.1097/mog.0b013e32833d4b71openalex-extracted
The role of the macrophage in sentinel responses in intestinal immunity
inferredcites →https://doi.org/10.1101/cshperspect.a022608openalex-extracted
Microtubules and Microtubule-Associated Proteins
inferredcites →https://doi.org/10.1111/febs.15238openalex-extracted
NKT cells and the regulation of intestinal immunity: a two‐way street
inferredcites →https://doi.org/10.1111/imm.12328openalex-extracted
miR‐155 deficiency protects mice from experimental colitis by reducing T helper type 1/type 17 responses
inferredcites →https://doi.org/10.1111/imr.12107openalex-extracted
The central role of the cytoskeleton in mechanisms and functions of the NK cell immune synapse
inferredcites →https://doi.org/10.1111/j.1365-2036.2010.04499.xopenalex-extracted
Increased proportion of CD16+ NK cells in the colonic lamina propria of inflammatory bowel disease patients, but not after azathioprine treatment
inferredcites →https://doi.org/10.1111/j.1365-2249.2011.04374.xopenalex-extracted
Dextran sulphate sodium increases splenic Gr1+CD11b+ cells which accelerate recovery from colitis following intravenous transplantation
inferredcites →https://doi.org/10.1111/j.1365-2796.2012.02588.xopenalex-extracted
Targeting T ‐cell migration in inflammatory bowel disease
inferredcites →https://doi.org/10.1124/jpet.109.160838openalex-extracted
Resveratrol (Trans-3,5,4′-trihydroxystilbene) Induces Silent Mating Type Information Regulation-1 and Down-Regulates Nuclear Transcription Factor-κB Activation to Abrogate Dextran Sulfate Sodium-Induced Colitis
inferredcites →https://doi.org/10.1126/science.aas8995openalex-extracted
HDAC6 mediates an aggresome-like mechanism for NLRP3 and pyrin inflammasome activation
inferredcites →https://doi.org/10.1136/gut.42.4.477openalex-extracted
Activation of nuclear factor κB in inflammatory bowel disease
inferredcites →https://doi.org/10.1136/gut.52.5.720openalex-extracted
Increases in free radicals and cytoskeletal protein oxidation and nitration in the colon of patients with inflammatory bowel disease
inferredcites →https://doi.org/10.1136/jitc-2020-000609openalex-extracted
Role of myeloid-derived suppressor cells in the promotion and immunotherapy of colitis-associated cancer
inferredcites →https://doi.org/10.1155/2019/7247238openalex-extracted
A Comprehensive Review and Update on the Pathogenesis of Inflammatory Bowel Disease
inferredcites →https://doi.org/10.1155/2021/8816041openalex-extracted
Th17 Cells in Inflammatory Bowel Disease: Cytokines, Plasticity, and Therapies
inferredcites →https://doi.org/10.1155/2022/1623181openalex-extracted
Microtubule Organization Is Essential for Maintaining Cellular Morphology and Function
inferredcites →https://doi.org/10.1155/2022/7025634openalex-extracted
Emerging Role of Dendritic Cell Intervention in the Treatment of Inflammatory Bowel Disease
inferredcites →https://doi.org/10.1172/jci118861openalex-extracted
Enterocolitis and colon cancer in interleukin-10-deficient mice are associated with aberrant cytokine production and CD4(+) TH1-like responses.
inferredcites →https://doi.org/10.1172/jci200423594openalex-extracted
Going both ways: Immune regulation via CD1d-dependent NKT cells
inferredcites →https://doi.org/10.1189/jlb.0113050openalex-extracted
The role and potential therapeutic application of myeloid-derived suppressor cells in TNBS-induced colitis
inferredcites →https://doi.org/10.12775/jehs.2022.12.08.074openalex-extracted
Molecular biology of the cell
inferredcites →https://doi.org/10.15252/embj.201488466openalex-extracted
Tubulin glycylases are required for primary cilia, control of cell proliferation and tumor development in colon
inferredcites →https://doi.org/10.2174/138945010790309885openalex-extracted
Anti-TNF Antibody Therapy for Inflammatory Bowel Disease During Pregnancy: A Clinical Review
inferredcites →https://doi.org/10.3109/07853890.2010.531758openalex-extracted
Th17-cytokine blockers as a new approach for treating inflammatory bowel disease
inferredcites →https://doi.org/10.3389/fbioe.2021.715408openalex-extracted
Probing Single-Cell Macrophage Polarization and Heterogeneity Using Thermo-Reversible Hydrogels in Droplet-Based Microfluidics
inferredcites →https://doi.org/10.3389/fcell.2021.625423openalex-extracted
Roles of Macrophages in the Development and Treatment of Gut Inflammation
inferredcites →https://doi.org/10.3389/fimmu.2016.00251openalex-extracted
Recognition and Regulation of T Cells by NK Cells
inferredcites →https://doi.org/10.3389/fimmu.2019.02168openalex-extracted
Macrophage-Specific NF-κB Activation Dynamics Can Segregate Inflammatory Bowel Disease Patients
inferredcites →https://doi.org/10.3389/fimmu.2022.1021634openalex-extracted
The paradoxical role of MDSCs in inflammatory bowel diseases: From bench to bedside
inferredcites →https://doi.org/10.3389/fimmu.2022.1055914openalex-extracted
The role of Th17 cells in inflammatory bowel disease and the research progress
inferredcites →https://doi.org/10.3389/fimmu.2023.1095098openalex-extracted
Identifying neutrophil-associated subtypes in ulcerative colitis and confirming neutrophils promote colitis-associated colorectal cancer
inferredcites →https://doi.org/10.3389/fimmu.2023.1103617openalex-extracted
Roles of macrophages on ulcerative colitis and colitis-associated colorectal cancer
inferredcites →https://doi.org/10.3389/fimmu.2023.1267091openalex-extracted
The role of inflammation in autoimmune disease: a therapeutic target
inferredcites →https://doi.org/10.3389/fmed.2014.00024openalex-extracted
Intestinal Epithelium in Inflammatory Bowel Disease
inferredcites →https://doi.org/10.3389/fphar.2018.00642openalex-extracted
Molecular Mechanisms of T Cells Activation by Dendritic Cells in Autoimmune Diseases
inferredcites →https://doi.org/10.3390/cells10030583openalex-extracted
Functions of Dendritic Cells and Its Association with Intestinal Diseases
inferredcites →https://doi.org/10.3390/nu11061398openalex-extracted
Dietary Composition and Effects in Inflammatory Bowel Disease
inferredcites →https://doi.org/10.3390/nu9070747openalex-extracted
Dietary Supplementation of Fermented Rice Bran Effectively Alleviates Dextran Sodium Sulfate-Induced Colitis in Mice
inferredcites →https://doi.org/10.3748/wjg.v20.i12.3255openalex-extracted
Dysregulation of mucosal immune response in pathogenesis of inflammatory bowel disease
inferredcites →https://doi.org/10.3892/etm.2020.8946openalex-extracted
Histological and ultrastructural changes of the colon in dextran sodium sulfate‑induced mouse colitis
inferredcites →https://doi.org/10.5217/ir.2018.16.1.26openalex-extracted
Immunological pathogenesis of inflammatory bowel disease
inferredcites →https://doi.org/10.5411/wji.v4.i1.26openalex-extracted
Role of myeloid-derived suppressor cells in autoimmune disease
inferredcites →https://doi.org/10.7573/dic.212572openalex-extracted
New developments in ulcerative colitis: latest evidence on management, treatment, and maintenance
inferredcites →https://doi.org/10.1172/jci23594openalex-snapshot
Going both ways: Immune regulation via CD1d-dependent NKT cells
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