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Syndromes of neurodegeneration with brain iron accumulation (NBIA): An update on clinical presentations, histological and genetic underpinnings, and treatment considerations
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Outgoing claims: the papers this work cites, builds on, or argues with.
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Recessively Inherited Parkinsonism
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Kufor Rakeb Disease: Autosomal recessive, levodopa‐responsive parkinsonism with pyramidal degeneration, supranuclear gaze palsy, and dementia
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Hereditary spastic paraplegia associated with dopa‐responsive parkinsonism
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Focal hand dystonia in a patient with PANK2 mutation
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A novel ferritin light chain gene mutation in a Japanese family with neuroferritinopathy: Description of clinical features and implications for genotype–phenotype correlations
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Novel PANK2 gene mutations in Korean patient with pantothenate kinase‐associated neurodegeneration presenting unilateral dystonic tremor
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Clinical spectrum of Kufor‐Rakeb syndrome in the Chilean kindred with ATP13A2 mutations
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Indian‐subcontinent NBIA: Unusual phenotypes, novel PANK2 mutations, and undetermined genetic forms
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Early‐onset L‐dopa‐responsive parkinsonism with pyramidal signs due to ATP13A2, PLA2G6, FBXO7 and spatacsin mutations
inferredcites →https://doi.org/10.1002/mds.23352openalex-snapshot
Eye movement disorders in ATP13A2 mutation carriers (PARK9)
inferredcites →https://doi.org/10.1002/mds.23751openalex-snapshot
Iron‐related MRI images in patients with pantothenate kinase–associated neurodegeneration (PKAN) treated with deferiprone: Results of a phase II pilot trial
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Proton magnetic resonance spectroscopy in steele‐richardson‐olszewski syndrome
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High‐field magnetic resonance imaging of brain iron: birth of a biomarker?
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Über den eisennachweis im gehirn, besonders in zentren des extrapyramidal-motorischen systems. I. Teil
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α-Synuclein accumulation in a case of neurodegeneration with brain iron accumulation type 1 (NBIA-1, formerly Hallervorden-Spatz syndrome) with widespread cortical and brainstem-type Lewy bodies
inferredcites →https://doi.org/10.1007/s004010050049openalex-snapshot
Juvenile-onset generalized neuroaxonal dystrophy (Hallervorden-Spatz disease) with diffuse neurofibrillary and Lewy body pathology
inferredcites →https://doi.org/10.1007/s00702-009-0234-9openalex-snapshot
Eye of the tiger-like MRI in parkinsonian variant of multiple system atrophy
inferredcites →https://doi.org/10.1007/s12311-010-0235-0openalex-snapshot
The Neuropathology of Late-Onset Friedreich’s Ataxia
inferredcites →https://doi.org/10.1016/j.ajhg.2008.10.010openalex-snapshot
Mutations in the Fatty Acid 2-Hydroxylase Gene Are Associated with Leukodystrophy with Spastic Paraparesis and Dystonia
inferredcites →https://doi.org/10.1016/j.ajo.2005.03.024openalex-snapshot
Neuro-Ophthalmologic and Electroretinographic Findings in Pantothenate Kinase-Associated Neurodegeneration (formerly Hallervorden-Spatz Syndrome)
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Preliminary observation of elevated levels of nanocrystalline iron oxide in the basal ganglia of neuroferritinopathy patients
inferredcites →https://doi.org/10.1016/j.clineuro.2009.04.007openalex-snapshot
A patient with pantothenate kinase-associated neurodegeneration and supranuclear gaze palsy
inferredcites →https://doi.org/10.1016/j.ejpn.2008.01.006openalex-snapshot
Deep brain stimulation as a mode of treatment of early onset pantothenate kinase-associated neurodegeneration
inferredcites →https://doi.org/10.1016/j.jns.2009.11.008openalex-snapshot
Siblings with the adult-onset slowly progressive type of pantothenate kinase-associated neurodegeneration and a novel mutation, Ile346Ser, in PANK2: Clinical features and 99mTc-ECD brain perfusion SPECT findings
inferredcites →https://doi.org/10.1016/j.jns.2011.01.010openalex-snapshot
Friedreich's ataxia: Pathology, pathogenesis, and molecular genetics
inferredcites →https://doi.org/10.1016/j.nbd.2010.04.009openalex-snapshot
Pantothenate kinase-2 (Pank2) silencing causes cell growth reduction, cell-specific ferroportin upregulation and iron deregulation
inferredcites →https://doi.org/10.1016/j.nbd.2011.02.009openalex-snapshot
A truncating mutation in ATP13A2 is responsible for adult-onset neuronal ceroid lipofuscinosis in Tibetan terriers
inferredcites →https://doi.org/10.1016/j.neucli.2008.12.003openalex-snapshot
Low-frequency rTMS of the premotor cortex reduces complex movement patterns in a patient with pantothenate kinase-associated neurodegenerative disease (PKAN)
inferredcites →https://doi.org/10.1016/j.neulet.2010.05.025openalex-snapshot
Accumulation of oxidative DNA damage in brain mitochondria in mouse model of hereditary ferritinopathy
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2010.05.009openalex-snapshot
Widespread Lewy body and tau accumulation in childhood and adult onset dystonia-parkinsonism cases with PLA2G6 mutations
inferredcites →https://doi.org/10.1016/j.nurt.2007.05.006openalex-snapshot
Iron in Chronic Brain Disorders: Imaging and Neurotherapeutic Implications
inferredcites →https://doi.org/10.1016/j.parkreldis.2004.05.003openalex-snapshot
Levodopa-responsive parkinsonism in hereditary spastic paraplegia with thin corpus callosum
inferredcites →https://doi.org/10.1016/j.spen.2006.08.007openalex-snapshot
Neurodegeneration With Brain Iron Accumulation: From Genes to Pathogenesis
inferredcites →https://doi.org/10.1016/s0002-9440(10)64548-8openalex-snapshot
Neurodegeneration with Brain Iron Accumulation, Type 1 Is Characterized by α-, β-, and γ-Synuclein Neuropathology
inferredcites →https://doi.org/10.1016/s0022-510x(00)00337-3openalex-snapshot
Widespread expression of α-synuclein and τ immunoreactivity in Hallervorden–Spatz syndrome with protracted clinical course
inferredcites →https://doi.org/10.1016/s0065-2571(01)00026-7openalex-snapshot
Mitochondria and ceramide: intertwined roles in regulation of apoptosis
inferredcites →https://doi.org/10.1016/s0887-8994(00)00243-5openalex-snapshot
Julius Hallervorden’s wartime activities: Implications for science under dictatorship
inferredcites →https://doi.org/10.1016/s0887-8994(01)00296-xopenalex-snapshot
Cranial MRI changes may precede symptoms in Hallervorden-Spatz syndrome
inferredcites →https://doi.org/10.1038/nature05292openalex-snapshot
Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases
inferredcites →https://doi.org/10.1038/ncpneuro0356openalex-snapshot
Superficial siderosis: a case report and review of the literature
inferredcites →https://doi.org/10.1038/ng1826openalex-snapshot
PLA2G6, encoding a phospholipase A2, is mutated in neurodegenerative disorders with high brain iron
inferredcites →https://doi.org/10.1038/ng1884openalex-snapshot
Hereditary parkinsonism with dementia is caused by mutations in ATP13A2, encoding a lysosomal type 5 P-type ATPase
inferredcites →https://doi.org/10.1038/nrc1411openalex-snapshot
Biologically active sphingolipids in cancer pathogenesis and treatment
inferredcites →https://doi.org/10.1055/s-0031-1274101openalex-snapshot
Identification of a second major locus for neurodegeneration with brain iron accumulation
inferredcites →https://doi.org/10.1056/nejmoa020817openalex-snapshot
Genetic, Clinical, and Radiographic Delineation of Hallervorden–Spatz Syndrome
inferredcites →https://doi.org/10.1073/pnas.0912105107openalex-snapshot
Pantethine rescues a Drosophila model for pantothenate kinase–associated neurodegeneration
inferredcites →https://doi.org/10.1089/ars.2009.2668openalex-snapshot
Lipid Oxidation and Peroxidation in CNS Health and Disease: From Molecular Mechanisms to Therapeutic Opportunities
inferredcites →https://doi.org/10.1093/brain/110.4.993openalex-snapshot
ADULT ONSET HALLERVORDEN-SPATZ DISEASE WITH NEUROFIBRILLARY PATHOLOGY
inferredcites →https://doi.org/10.1093/brain/awl319openalex-snapshot
Clinical features and natural history of neuroferritinopathy caused by the FTL1 460InsA mutation
inferredcites →https://doi.org/10.1093/brain/awq022openalex-snapshot
Dystonia in neurodegeneration with brain iron accumulation: outcome of bilateral pallidal stimulation
inferredcites →https://doi.org/10.1093/brain/awr042openalex-snapshot
Novel histopathologic findings in molecularly-confirmed pantothenate kinase-associated neurodegeneration
inferredcites →https://doi.org/10.1093/hmg/ddp314openalex-snapshot
Pantothenate kinase-associated neurodegeneration: insights from a Drosophila model
inferredcites →https://doi.org/10.1093/jnen/64.4.280openalex-snapshot
Hereditary Ferritinopathy: A Novel Mutation, Its Cellular Pathology, and Pathogenetic Insights
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INFANTILE NEUROANONAL DYSTROPHY
inferredcites →https://doi.org/10.1097/00005072-199809000-00010openalex-snapshot
Diagnostic Neuropathology
inferredcites →https://doi.org/10.1097/wad.0b013e3181a2b76bopenalex-snapshot
Frontotemporal Dementia-amyotrophic Lateral Sclerosis Complex is Simulated by Neurodegeneration With Brain Iron Accumulation
inferredcites →https://doi.org/10.1111/j.1399-0004.2011.01745.xopenalex-snapshot
Novel mutation in ATP13A2 widens the spectrum of Kufor‐Rakeb syndrome (PARK9)
inferredcites →https://doi.org/10.1111/j.1468-1331.2010.03298.xopenalex-snapshot
Idiopathic NBIA - clinical spectrum and transcranial sonography findings
inferredcites →https://doi.org/10.1111/j.1469-8749.2011.03993.xopenalex-snapshot
FA2H-related disorders: a novel c.270+3A>T splice-site mutation leads to a complex neurodegenerative phenotype
inferredcites →https://doi.org/10.1111/j.1600-0404.1994.tb02645.xopenalex-snapshot
Pallido-pyramidal degeneration, supranuclear upgaze paresis and dementia: Kufor-Rakeb syndrome
inferredcites →https://doi.org/10.1111/j.1742-4658.2008.06709.xopenalex-snapshot
Emerging pathways in genetic Parkinson's disease: Potential role of ceramide metabolism in Lewy body disease
inferredcites →https://doi.org/10.1136/jmg.2008.061929openalex-snapshot
Clinical and genetic delineation of neurodegeneration with brain iron accumulation
inferredcites →https://doi.org/10.1136/jmg.40.5.e69openalex-snapshot
Neuroferritinopathy in a French family with late onset dominant dystonia
inferredcites →https://doi.org/10.1136/jnnp.2008.169953openalex-snapshot
Iron accumulation in syndromes of neurodegeneration with brain iron accumulation 1 and 2: causative or consequential?
inferredcites →https://doi.org/10.1136/jnnp.60.1.98openalex-snapshot
Signal changes on MRI and increases in reactive microgliosis, astrogliosis, and iron in the putamen of two patients with multiple system atrophy.
inferredcites →https://doi.org/10.1146/annurev.neuro.30.051606.094232openalex-snapshot
Copper and Iron Disorders of the Brain
inferredcites →https://doi.org/10.1159/000148691openalex-snapshot
The Neurological Presentation of Ceruloplasmin Gene Mutations
inferredcites →https://doi.org/10.1212/01.wnl.0000148577.62644.77openalex-snapshot
[ 123 I]FP-CIT SPECT findings in two patients with Hallervorden–Spatz disease with homozygous mutation in PANK2 gene
inferredcites →https://doi.org/10.1212/01.wnl.0000237446.06971.72openalex-snapshot
Severe tongue protrusion dystonia
inferredcites →https://doi.org/10.1212/01.wnl.0000260963.08711.08openalex-snapshot
ATP13A2 missense mutations in juvenile parkinsonism and young onset Parkinson disease
inferredcites →https://doi.org/10.1212/01.wnl.0000310985.40011.d6openalex-snapshot
T2* and FSE MRI distinguishes four subtypes of neurodegeneration with brain iron accumulation
inferredcites →https://doi.org/10.1212/01.wnl.0000327094.67726.28openalex-snapshot
Neurodegeneration associated with genetic defects in phospholipase A 2
inferredcites →https://doi.org/10.1212/wnl.55.1.89openalex-snapshot
Clinical and pathologic findings in hereditary spastic paraparesis with spastin mutation
inferredcites →https://doi.org/10.1371/journal.pone.0012897openalex-snapshot
Catalytic Function of PLA2G6 Is Impaired by Mutations Associated with Infantile Neuroaxonal Dystrophy but Not Dystonia-Parkinsonism
inferredcites →https://doi.org/10.1523/jneurosci.4265-04.2005openalex-snapshot
Altered Neuronal Mitochondrial Coenzyme A Synthesis in Neurodegeneration with Brain Iron Accumulation Caused by Abnormal Processing, Stability, and Catalytic Activity of Mutant Pantothenate Kinase 2
inferredcites →https://doi.org/10.2353/ajpath.2008.070823openalex-snapshot
Disrupted Membrane Homeostasis and Accumulation of Ubiquitinated Proteins in a Mouse Model of Infantile Neuroaxonal Dystrophy Caused by PLA2G6 Mutations
inferredcites →https://doi.org/10.3174/ajnr.a1483openalex-snapshot
MR Imaging Findings in Autosomal Recessive Hereditary Spastic Paraplegia
inferredcites →https://doi.org/10.3174/ajnr.a1849openalex-snapshot
Diffusion Tensor MR Imaging in Children with Pantothenate Kinase−Associated Neurodegeneration with Brain Iron Accumulation and Their Siblings
inferredcites →https://doi.org/10.4061/2010/342834openalex-snapshot
Sleep in Genetically Confirmed Pantothenate Kinase-Associated Neurodegeneration: A Video-Polysomnographic Study
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Phospholipase A2, reactive oxygen species, and lipid peroxidation in CNS pathologies
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Dystonia and Parkinson’s disease: Do they have a shared biology?
inferred← citeshttps://doi.org/10.1016/j.braindev.2012.07.008openalex-snapshot
MRI, MR spectroscopy, and diffusion tensor imaging findings in patient with static encephalopathy of childhood with neurodegeneration in adulthood (SENDA)
inferred← citeshttps://doi.org/10.1016/j.braindev.2016.02.010openalex-snapshot
A novel gene mutation in PANK2 in a patient with severe jaw-opening dystonia
inferred← citeshttps://doi.org/10.1016/j.braindev.2016.08.012openalex-snapshot
Infantile neuroaxonal dystrophy and PLA2G6-associated neurodegeneration: An update for the diagnosis
inferred← citeshttps://doi.org/10.1016/j.braindev.2021.06.010openalex-snapshot
Neurodegeneration with brain iron accumulation: Characterization of clinical, radiological, and genetic features of pediatric patients from Southern India
inferred← citeshttps://doi.org/10.1016/j.brainres.2020.147066openalex-snapshot
Loss of tyrosine hydroxylase, motor deficits and elevated iron in a mouse model of phospholipase A2G6-associated neurodegeneration (PLAN)
inferred← citeshttps://doi.org/10.1016/j.clineuro.2017.01.011openalex-snapshot
Clinical and genetic features of PKAN patients in a tertiary centre in Turkey
inferred← citeshttps://doi.org/10.1016/j.ejpn.2014.02.005openalex-snapshot
Late diagnosis of fucosidosis in a child with progressive fixed dystonia, bilateral pallidal lesions and red spots on the skin
inferred← citeshttps://doi.org/10.1016/j.lfs.2016.07.021openalex-snapshot
Polyphenols in dementia: From molecular basis to clinical trials
inferred← citeshttps://doi.org/10.1016/j.nbd.2011.12.054openalex-snapshot
Iron dysregulation in movement disorders
inferred← citeshttps://doi.org/10.1016/j.ncl.2014.09.015openalex-snapshot
Movement Disorders in Systemic Diseases
inferred← citeshttps://doi.org/10.1016/j.neuint.2012.11.015openalex-snapshot
Genetics and iron in the systems biology of Parkinson’s disease and some related disorders
inferred← citeshttps://doi.org/10.1016/j.neuint.2012.12.006openalex-snapshot
Metals, oxidative stress and neurodegeneration: A focus on iron, manganese and mercury
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2015.10.034openalex-snapshot
Identification of mutation in GTPBP2 in patients of a family with neurodegeneration accompanied by iron deposition in the brain
inferred← citeshttps://doi.org/10.1016/j.parkreldis.2013.06.020openalex-snapshot
Transcranial sonography in mitochondrial membrane protein-associated neurodegeneration
inferred← citeshttps://doi.org/10.1016/j.parkreldis.2015.02.006openalex-snapshot
Pattern of disease progression in atypical form of pantothenate-kinase-associated neurodegeneration (PKAN) – Prospective study
inferred← citeshttps://doi.org/10.1016/j.parkreldis.2016.01.022openalex-snapshot
The clinical syndrome of dystonia with anarthria/aphonia
inferred← citeshttps://doi.org/10.1016/j.parkreldis.2017.02.025openalex-snapshot
Parkinsonian-Pyramidal syndromes: A systematic review
inferred← citeshttps://doi.org/10.1016/j.parkreldis.2017.03.013openalex-snapshot
Evolution and novel radiological changes of neurodegeneration associated with mutations in C19orf12
inferred← citeshttps://doi.org/10.1016/j.parkreldis.2019.06.025openalex-snapshot
Juvenile parkinsonism: Differential diagnosis, genetics, and treatment
inferred← citeshttps://doi.org/10.1016/j.pjnns.2013.09.002openalex-snapshot
Mitochondrial protein associated neurodegeneration – Case report
inferred← citeshttps://doi.org/10.1016/j.pjnns.2014.05.001openalex-snapshot
Update on neurodegeneration with brain iron accumulation
inferred← citeshttps://doi.org/10.1016/j.praneu.2015.01.005openalex-snapshot
Syndromes parkinsoniens rares de causes héréditaires
inferred← citeshttps://doi.org/10.1016/j.revmed.2017.09.001openalex-snapshot
Les mouvements anormaux : mise au point
inferred← citeshttps://doi.org/10.1016/j.spen.2012.03.004openalex-snapshot
Current Concepts and Controversies in Neurodegeneration with Brain Iron Accumulation
inferred← citeshttps://doi.org/10.1016/j.spen.2012.03.007openalex-snapshot
Therapeutic Advances in Neurodegeneration With Brain Iron Accumulation
inferred← citeshttps://doi.org/10.1017/9781139924771.018openalex-snapshot
Early clinical features of the parkinsonian-related dementias
inferred← citeshttps://doi.org/10.1017/9781316105139.008openalex-snapshot
Asim K. Bag, Aparna Singhal, Lázaro Luís Faria do Amaral
inferred← citeshttps://doi.org/10.1017/9781316145050.016openalex-snapshot
Parkinsonism Due to CSF1R Mutation
inferred← citeshttps://doi.org/10.1017/9781316145050.024openalex-snapshot
PKAN Misdiagnosed as ‘Progressive Delayed-Onset Postanoxic Dystonia’
inferred← citeshttps://doi.org/10.1017/cbo9781139175845.009openalex-snapshot
Movement disorders in metabolic diseases in adulthood
inferred← citeshttps://doi.org/10.1017/cjn.2019.244openalex-snapshot
Myoclonus, Hydrocephalus in Mitochondrial Protein-Associated Neurodegeneration
inferred← citeshttps://doi.org/10.1017/cjn.2020.279openalex-snapshot
Iron Chelation in Movement Disorders: Logical or Ironical
inferred← citeshttps://doi.org/10.1017/s0317167100013354openalex-snapshot
Iron - Too Much of a Good Thing
inferred← citeshttps://doi.org/10.1021/acs.jmedchem.0c01531openalex-snapshot
Cyclic Phosphopantothenic Acid Prodrugs for Treatment of Pantothenate Kinase-Associated Neurodegeneration
inferred← citeshttps://doi.org/10.1038/hgv.2015.48openalex-snapshot
Novel PLA2G6 mutations associated with an exonic deletion due to non-allelic homologous recombination in a patient with infantile neuroaxonal dystrophy
inferred← citeshttps://doi.org/10.1038/nrneurol.2015.198openalex-snapshot
A novel diagnostic approach to patients with myoclonus
inferred← citeshttps://doi.org/10.1038/s41380-019-0375-7openalex-snapshot
Brain iron is associated with accelerated cognitive decline in people with Alzheimer pathology
inferred← citeshttps://doi.org/10.1038/s41598-025-94838-wopenalex-snapshot
Serum metabolomics indicates ferroptosis in patients with pantothenate kinase associated neurodegeneration
inferred← citeshttps://doi.org/10.1080/00207454.2019.1694924openalex-snapshot
Neuropsychological functions and psychiatric symptoms in late-onset manifestation of pantothenate kinase-associated neurodegeneration: a clinical case report
inferred← citeshttps://doi.org/10.1093/med/9780190930868.003.0008openalex-snapshot
Movement Disorders
inferred← citeshttps://doi.org/10.1093/med/9780197699461.003.0009openalex-snapshot
Hyperkinetic Movement Disorders
inferred← citeshttps://doi.org/10.1093/med/9780199361953.003.0008openalex-snapshot
Movement Disorders
inferred← citeshttps://doi.org/10.1097/md.0000000000034853openalex-snapshot
The first Vietnamese patient who presented late onset of pantothenate kinase-associated neurodegeneration diagnosed by whole exome sequencing: A case report
inferred← citeshttps://doi.org/10.1097/wco.0b013e3283550cacopenalex-snapshot
Neurodegeneration with brain iron accumulation
inferred← citeshttps://doi.org/10.1097/wco.0b013e3283629bebopenalex-snapshot
Metals and movement disorders
inferred← citeshttps://doi.org/10.1097/wco.0b013e3283632e83openalex-snapshot
The pallidopyramidal syndromes
inferred← citeshttps://doi.org/10.1101/2024.02.06.579103openalex-snapshot
A pharmacological toolkit for human microglia identifies Topoisomerase I inhibitors as immunomodulators for Alzheimer’s disease
inferred← citeshttps://doi.org/10.1111/ane.12616openalex-snapshot
An updated review of Parkinson's disease genetics and clinicopathological correlations
inferred← citeshttps://doi.org/10.1111/cge.12137openalex-snapshot
A novel frameshift mutation of C19ORF12 causes NBIA4 with cerebellar atrophy and manifests with severe peripheral motor axonal neuropathy
inferred← citeshttps://doi.org/10.1111/j.1468-1331.2012.03856.xopenalex-snapshot
Follow‐up study of 25 Chinese children with PLA 2G6‐associated neurodegeneration
inferred← citeshttps://doi.org/10.1111/jon.12195openalex-snapshot
Neurodegeneration with Brain Iron Accumulation: Clinicoradiological Approach to Diagnosis
inferred← citeshttps://doi.org/10.1111/nan.12242openalex-snapshot
Review: Insights into molecular mechanisms of disease in neurodegeneration with brain iron accumulation: unifying theories
inferred← citeshttps://doi.org/10.1136/bjophthalmol-2017-310181openalex-snapshot
Visual and ocular motor function in the atypical form of neurodegeneration with brain iron accumulation type I
inferred← citeshttps://doi.org/10.1155/2012/387095openalex-snapshot
Late Onset Neurodegeneration with Brain-Iron Accumulation Presenting as Parkinsonism
inferred← citeshttps://doi.org/10.1155/2013/860539openalex-snapshot
Phenotypes and Genotypes of Patients with Pantothenate Kinase‐Associated Neurodegeneration in Asian and Caucasian Populations: 2 Cases and Literature Review
inferred← citeshttps://doi.org/10.1155/2013/891748openalex-snapshot
Polyphenols: Multipotent Therapeutic Agents in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1155/2017/3247034openalex-snapshot
Open-Label Fosmetpantotenate, a Phosphopantothenate Replacement Therapy in a Single Patient with Atypical PKAN
inferred← citeshttps://doi.org/10.1155/2018/2850341openalex-snapshot
Unraveling the Burden of Iron in Neurodegeneration: Intersections with Amyloid Beta Peptide Pathology
inferred← citeshttps://doi.org/10.1176/appi.neuropsych.13120375openalex-snapshot
Early Cues to Detect Atypical Panthothenate Kinase-Associated Neurodegeneration
inferred← citeshttps://doi.org/10.1177/08830738261495303openalex-snapshot
Improving Dystonia Management of Children Admitted for Elective Surgical Procedures: A Quality Improvement Project
inferred← citeshttps://doi.org/10.1177/2050313x17720101openalex-snapshot
Novel PANK2 mutation in the first Greek compound heterozygote patient with pantothenate-kinase-associated neurodegeneration
inferred← citeshttps://doi.org/10.1186/2051-5960-1-55openalex-snapshot
The systemic iron-regulatory proteins hepcidin and ferroportin are reduced in the brain in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1212/01.wnl.0000435557.21319.adopenalex-snapshot
Teaching Neuro Images : Neurodegeneration with brain iron accumulation in aceruloplasminemia
inferred← citeshttps://doi.org/10.1212/cpj.0b013e318278bf06openalex-snapshot
Pediatric movement disorders
inferred← citeshttps://doi.org/10.1212/wnl.0000000000001511openalex-snapshot
Cortical pencil lining in neuroferritinopathy: A diagnostic clue
inferred← citeshttps://doi.org/10.1212/wnl.0000000000012756openalex-snapshot
Clinical Reasoning: A Middle-aged Man With Progressive Gait Abnormalities
inferred← citeshttps://doi.org/10.1212/wnl.0000000000206986openalex-snapshot
Teaching Neuro Images : Neurodegeneration with brain iron accumulation in aceruloplasminemia
inferred← citeshttps://doi.org/10.1212/wnl.0000000000213482openalex-snapshot
“Jack-Knife” Dystonia in Pantothenate Kinase–Associated Neurodegeneration
inferred← citeshttps://doi.org/10.1242/dmm.029926openalex-snapshot
A novel high-throughput yeast genetic screen for factors modifying protein levels of the Early-Onset Torsion Dystonia-associated variant torsinAΔE
inferred← citeshttps://doi.org/10.1371/journal.pone.0085271openalex-snapshot
Profound Morphological Changes in the Erythrocytes and Fibrin Networks of Patients with Hemochromatosis or with Hyperferritinemia, and Their Normalization by Iron Chelators and Other Agents
inferred← citeshttps://doi.org/10.1371/journal.pone.0141629openalex-snapshot
Deficiency of Calcium-Independent Phospholipase A2 Beta Induces Brain Iron Accumulation through Upregulation of Divalent Metal Transporter 1
inferred← citeshttps://doi.org/10.1371/journal.pone.0184104openalex-snapshot
iPSC-derived neuronal models of PANK2-associated neurodegeneration reveal mitochondrial dysfunction contributing to early disease
inferred← citeshttps://doi.org/10.1517/17530059.2012.704017openalex-snapshot
Genetic diagnosis of hyperkinetic movement disorders
inferred← citeshttps://doi.org/10.1523/jneurosci.1409-14.2014openalex-snapshot
Abnormal High-Frequency Burst Firing of Cerebellar Neurons in Rapid-Onset Dystonia-Parkinsonism
inferred← citeshttps://doi.org/10.15557/an.2024.0003openalex-snapshot
Assessment of the degree of disability of the Polish NBIA-MPAN population based on a patient survey
inferred← citeshttps://doi.org/10.15586/codonpublications.parkinsonsdisease.2018.ch6openalex-snapshot
The Differential Diagnosis of Parkinson’s Disease
inferred← citeshttps://doi.org/10.1586/ern.12.128openalex-snapshot
Brain iron accumulation in aging and neurodegenerative disorders
inferred← citeshttps://doi.org/10.1590/0004-282x20140122openalex-snapshot
Retinitis pigmentosa in pantothenate kinase-associated neurodegeneration
inferred← citeshttps://doi.org/10.1590/s1980-5764-2016dn1002014openalex-snapshot
Neurodegeneration with brain iron accumulation: A case report
inferred← citeshttps://doi.org/10.18632/aging.100695openalex-snapshot
Eryptosis as a marker of Parkinson's disease
inferred← citeshttps://doi.org/10.2174/157015913804999469openalex-snapshot
Genetics and Pathophysiology of Neurodegeneration with Brain Iron Accumulation (NBIA)
inferred← citeshttps://doi.org/10.21767/2171-6625.1000275openalex-snapshot
Molecular Analysis of PANK2 Gene in Two Thai Classic Pantothenate Kinase- Associated Neurodegeneration (PKAN) Patients
inferred← citeshttps://doi.org/10.2991/efood.k.200203.001openalex-snapshot
Dietary Flavonoids in the Management of Huntington’s Disease: Mechanism and Clinical Perspective
inferred← citeshttps://doi.org/10.3389/fnagi.2013.00034openalex-snapshot
A delicate balance: Iron metabolism and diseases of the brain
inferred← citeshttps://doi.org/10.3389/fnagi.2017.00073openalex-snapshot
Blood Transcriptomic Meta-analysis Identifies Dysregulation of Hemoglobin and Iron Metabolism in Parkinson’ Disease
inferred← citeshttps://doi.org/10.3389/fnagi.2023.1298166openalex-snapshot
Research progress on the cannabinoid type-2 receptor and Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fncel.2022.1005182openalex-snapshot
Insight into the potential role of ferroptosis in neurodegenerative diseases
inferred← citeshttps://doi.org/10.3389/fneur.2018.01100openalex-snapshot
PLA2G6-Associated Neurodegeneration (PLAN): Review of Clinical Phenotypes and Genotypes
inferred← citeshttps://doi.org/10.3389/fneur.2020.01024openalex-snapshot
Brain MRI Pattern Recognition in Neurodegeneration With Brain Iron Accumulation
inferred← citeshttps://doi.org/10.3390/antiox9080700openalex-snapshot
Exploring the Use of Dimethyl Fumarate as Microglia Modulator for Neurodegenerative Diseases Treatment
inferred← citeshttps://doi.org/10.3390/biom11010030openalex-snapshot
Iron Dysregulation and Inflammagens Related to Oral and Gut Health Are Central to the Development of Parkinson’s Disease
inferred← citeshttps://doi.org/10.3390/cells10061446openalex-snapshot
iPLA2β Contributes to ER Stress-Induced Apoptosis during Myocardial Ischemia/Reperfusion Injury
inferred← citeshttps://doi.org/10.3390/genes17091132openalex-snapshot
Clinical, Neuroimaging, and Molecular Characterization of a Juvenile PLAN-like Phenotype in Two Siblings Carrying p.Arg645Gln and an Unresolved Exon 4–7 Copy-Number Gain: A Case Report
inferred← citeshttps://doi.org/10.3390/ijms222111998openalex-snapshot
Neurodegenerative Disorders: Spotlight on Sphingolipids
inferred← citeshttps://doi.org/10.3390/ijms23147806openalex-snapshot
Ceramide and Sphingosine-1-Phosphate in Neurodegenerative Disorders and Their Potential Involvement in Therapy
inferred← citeshttps://doi.org/10.3390/ijms241411523openalex-snapshot
Lipids in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.3390/ijms26136477openalex-snapshot
Polyphenols in the Central Nervous System: Cellular Effects and Liposomal Delivery Approaches
inferred← citeshttps://doi.org/10.3390/molecules26216639openalex-snapshot
Metals and Metal-Nanoparticles in Human Pathologies: From Exposure to Therapy
inferred← citeshttps://doi.org/10.3390/molecules28145415openalex-snapshot
Polyphenols and Neurodegenerative Diseases: Potential Effects and Mechanisms of Neuroprotection
inferred← citeshttps://doi.org/10.3410/f.717968396.793468174openalex-snapshot
Faculty Opinions recommendation of Dystonia with brain manganese accumulation resulting from SLC30A10 mutations: a new treatable disorder.
inferred← citeshttps://doi.org/10.36290/neu.2016.068openalex-snapshot
NBIA - neurodegeneration with brain iron accumulation
inferred← citeshttps://doi.org/10.3892/mmr.2017.6386openalex-snapshot
Iron and Parkinson's disease: A systematic review and meta-analysis
inferred← citeshttps://doi.org/10.4045/tidsskr.14.1115openalex-snapshot
A man in his 50s with high ferritin levels and increasing cognitive impairment
inferred← citeshttps://doi.org/10.4081/ni.2019.7959openalex-snapshot
A novel homozygous variation in the PANK2 gene in two Persian siblings with atypical pantothenate kinase associated neurodegeneration
inferred← citeshttps://doi.org/10.4103/0028-3886.237021openalex-snapshot
Acanthocytosis in progressive childhood dystonia
inferred← citeshttps://doi.org/10.4103/0028-3886.329603openalex-snapshot
NBIA Syndromes: A Step Forward from the Previous Knowledge
inferred← citeshttps://doi.org/10.4103/aian.aian_481_18openalex-snapshot
Neurodegeneration with brain iron accumulation
inferred← citeshttps://doi.org/10.4103/neurol-india.neurol-india-d-23-00460openalex-snapshot
An Interesting Case of Young-Onset Parkinsonism Due to PLA2G6 Gene Mutation-Related Dystonia Parkinsonism
inferred← citeshttps://doi.org/10.5001/omj.2017.12openalex-snapshot
A Novel Deletion Mutation of Exon 2 of the C19orf12 Gene in an Omani Family with Mitochondrial Membrane Protein-Associated Neurodegeneration (MPAN)
inferred← citeshttps://doi.org/10.5535/arm.2018.42.2.363openalex-snapshot
Botulinum Toxin-A Injection in the Treatment of Spasticity in a Infantile-Onset Neurodegeneration With Brain Iron Accumulation: A Case Report
inferred← citeshttps://doi.org/10.5582/irdr.2024.01019openalex-snapshot
Functional impairments in NBIA patients: Preliminary results
inferred← citeshttps://doi.org/10.5692/clinicalneurol.cn-001324openalex-snapshot
A case of novel WDR45 mutation with beta-propeller protein-associated neurodegeneration (BPAN) presenting asymmetrical extrapyramidal signs
inferred← citeshttps://doi.org/10.5812/archneurosci.18555openalex-snapshot
Deficits in Striatal Dopamine and Hippocampal Serotonin Following Induction of Anxiety/Depressive-Like Behaviors by Bisphenol A
inferred← citeshttps://doi.org/10.6016/zdravvestn.2935openalex-snapshot
Nuklearnomedicinske preiskave v diagnostiki demence in parkinsonizmov
inferred← citeshttps://doi.org/10.70675/24e9e526z1e46z4304za707z0debcb9a0bdbopenalex-snapshot
Evaluation des réseaux neuronaux vecteurs de comportements par imagerie anatomique et fonctionnelle in vivo chez l'homme
inferred← citeshttps://doi.org/10.7759/cureus.58127openalex-snapshot
A Case of Beta-Propeller Protein-Associated Neurodegeneration With a Unique Truncating Variant in the WDR45 Gene and Uncommon Clinical and Radiologic Findings
inferred← citeshttps://doi.org/10.7762/cnr.2023.12.2.154openalex-snapshot
Lipocalin-2 Secreted by the Liver Regulates Neuronal Cell Function Through AKT-Dependent Signaling in Hepatic Encephalopathy Mouse Model
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