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Molecular Features Underlying Neurodegeneration Identified through In Vitro Modeling of Genetically Diverse Parkinson’s Disease Patients
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/jamaneurol.2014.2704openalex-snapshot
Clinical Correlations With Lewy Body Pathology inLRRK2-Related Parkinson Disease
inferredcites →https://doi.org/10.1002/ajmg.b.30530openalex-snapshot
Cytogenetic and molecular characterization of A2BP1/FOX1 as a candidate gene for autism
inferredcites →https://doi.org/10.1002/ana.1132openalex-snapshot
Lewy bodies and parkinsonism in families with parkin mutations
inferredcites →https://doi.org/10.1002/emmm.201200215openalex-snapshot
Disease‐specific phenotypes in dopamine neurons from human iPS‐based models of genetic and sporadic Parkinson's disease
inferredcites →https://doi.org/10.1006/nbdi.2002.0507openalex-snapshot
Environmental Risk Factors and Parkinson's Disease: Selective Degeneration of Nigral Dopaminergic Neurons Caused by the Herbicide Paraquat
inferredcites →https://doi.org/10.1007/s10038-004-0145-4openalex-snapshot
The de novo chromosome 16 translocations of two patients with abnormal phenotypes (mental retardation and epilepsy) disrupt the A2BP1 gene
inferredcites →https://doi.org/10.1016/j.brainres.2006.02.052openalex-snapshot
Electrophysiological characterization of substantia nigra dopaminergic neurons in partially lesioned rats: Effects of subthalamotomy and levodopa treatment
inferredcites →https://doi.org/10.1016/j.celrep.2014.02.005openalex-snapshot
HITS-CLIP and Integrative Modeling Define the Rbfox Splicing-Regulatory Network Linked to Brain Development and Autism
inferredcites →https://doi.org/10.1016/j.celrep.2014.10.023openalex-snapshot
iPSC-Derived Dopamine Neurons Reveal Differences between Monozygotic Twins Discordant for Parkinson’s Disease
inferredcites →https://doi.org/10.1016/j.neuro.2014.08.009openalex-snapshot
Effects of the Parkinsonian toxin MPP+ on electrophysiological properties of nigral dopaminergic neurons
inferredcites →https://doi.org/10.1016/j.neuron.2004.10.023openalex-snapshot
Cloning of the Gene Containing Mutations that Cause PARK8-Linked Parkinson's Disease
inferredcites →https://doi.org/10.1016/j.neuron.2004.11.005openalex-snapshot
Mutations in LRRK2 Cause Autosomal-Dominant Parkinsonism with Pleomorphic Pathology
inferredcites →https://doi.org/10.1016/j.neuroscience.2011.10.057openalex-snapshot
Neurotoxin-based models of Parkinson's disease
inferredcites →https://doi.org/10.1016/j.stem.2011.01.013openalex-snapshot
LRRK2 Mutant iPSC-Derived DA Neurons Demonstrate Increased Susceptibility to Oxidative Stress
inferredcites →https://doi.org/10.1016/j.stem.2011.06.007openalex-snapshot
Epigenetic Memory and Preferential Lineage-Specific Differentiation in Induced Pluripotent Stem Cells Derived from Human Pancreatic Islet Beta Cells
inferredcites →https://doi.org/10.1016/j.stem.2013.01.008openalex-snapshot
Genetic Correction of a LRRK2 Mutation in Human iPSCs Links Parkinsonian Neurodegeneration to ERK-Dependent Changes in Gene Expression
inferredcites →https://doi.org/10.1016/j.stemcr.2014.01.013openalex-snapshot
Isolation of Human Induced Pluripotent Stem Cell-Derived Dopaminergic Progenitors by Cell Sorting for Successful Transplantation
inferredcites →https://doi.org/10.1016/s0092-8674(03)00477-xopenalex-snapshot
Differential Control of the Releasable Vesicle Pools by SNAP-25 Splice Variants and SNAP-23
inferredcites →https://doi.org/10.1038/33416openalex-snapshot
Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism
inferredcites →https://doi.org/10.1038/35049061openalex-snapshot
The splice of life: Alternative splicing and neurological disease
inferredcites →https://doi.org/10.1038/nature09798openalex-snapshot
Hotspots of aberrant epigenomic reprogramming in human induced pluripotent stem cells
inferredcites →https://doi.org/10.1038/nature10110openalex-snapshot
Transcriptomic analysis of autistic brain reveals convergent molecular pathology
inferredcites →https://doi.org/10.1038/nature10648openalex-snapshot
Dopamine neurons derived from human ES cells efficiently engraft in animal models of Parkinson’s disease
inferredcites →https://doi.org/10.1038/nature11557openalex-snapshot
Progressive degeneration of human neural stem cells caused by pathogenic LRRK2
inferredcites →https://doi.org/10.1038/ncb2839openalex-snapshot
SON connects the splicing-regulatory network with pluripotency in human embryonic stem cells
inferredcites →https://doi.org/10.1038/ncb748openalex-snapshot
α-Synuclein is phosphorylated in synucleinopathy lesions
inferredcites →https://doi.org/10.1038/ncomms1453openalex-snapshot
Parkinson's disease induced pluripotent stem cells with triplication of the α-synuclein locus
inferredcites →https://doi.org/10.1038/ng.841openalex-snapshot
The splicing regulator Rbfox1 (A2BP1) controls neuronal excitation in the mammalian brain
inferredcites →https://doi.org/10.1038/nm1001-1144openalex-snapshot
Parkin ubiquitinates the α-synuclein–interacting protein, synphilin-1: implications for Lewy-body formation in Parkinson disease
inferredcites →https://doi.org/10.1038/nrn3504openalex-snapshot
NMDA receptor subunit diversity: impact on receptor properties, synaptic plasticity and disease
inferredcites →https://doi.org/10.1073/pnas.1014041108openalex-snapshot
Rapid induction and long-term self-renewal of primitive neural precursors from human embryonic stem cells by small molecule inhibitors
inferredcites →https://doi.org/10.1074/jbc.m802223200openalex-snapshot
Serine 129 Phosphorylation of α-Synuclein Induces Unfolded Protein Response-mediated Cell Death
inferredcites →https://doi.org/10.1091/mbc.e05-07-0595openalex-snapshot
Alternative Splicing of SNAP-25 Regulates Secretion through Nonconservative Substitutions in the SNARE Domain
inferredcites →https://doi.org/10.1093/emboj/cdg089openalex-snapshot
A vertebrate RNA‐binding protein Fox‐1 regulates tissue‐specific splicing via the pentanucleotide GCAUG
inferredcites →https://doi.org/10.1093/hmg/9.9.1303openalex-snapshot
A novel protein with RNA-binding motifs interacts with ataxin-2
inferredcites →https://doi.org/10.1093/hmg/dds240openalex-snapshot
RBFOX1 regulates both splicing and transcriptional networks in human neuronal development
inferredcites →https://doi.org/10.1101/gad.1837009openalex-snapshot
An inducible change in Fox-1/A2BP1 splicing modulates the alternative splicing of downstream neuronal target exons
inferredcites →https://doi.org/10.1113/jphysiol.2012.234062openalex-snapshot
GluN1 splice variant control of GluN1/GluN2D NMDA receptors
inferredcites →https://doi.org/10.1126/science.1077209openalex-snapshot
Mutations in the DJ-1 Gene Associated with Autosomal Recessive Early-Onset Parkinsonism
inferredcites →https://doi.org/10.1126/science.1096284openalex-snapshot
Hereditary Early-Onset Parkinson's Disease Caused by Mutations in PINK1
inferredcites →https://doi.org/10.1126/science.276.5321.2045openalex-snapshot
Mutation in the α-Synuclein Gene Identified in Families with Parkinson's Disease
inferredcites →https://doi.org/10.1126/scitranslmed.3003985openalex-snapshot
Pharmacological Rescue of Mitochondrial Deficits in iPSC-Derived Neural Cells from Patients with Familial Parkinson’s Disease
inferredcites →https://doi.org/10.1146/annurev-pathol-011110-130242openalex-snapshot
Parkinson's Disease: Genetics and Pathogenesis
inferredcites →https://doi.org/10.1152/jn.2000.83.3.1300openalex-snapshot
Exon 5 and Spermine Regulate Deactivation of NMDA Receptor Subtypes
inferredcites →https://doi.org/10.1186/1756-6606-5-35openalex-snapshot
Mitochondrial dysfunction associated with increased oxidative stress and α-synuclein accumulation in PARK2 iPSC-derived neurons and postmortem brain tissue
inferredcites →https://doi.org/10.1186/s13059-014-0550-8openalex-snapshot
Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2
inferredcites →https://doi.org/10.1371/journal.pone.0026159openalex-snapshot
SNCA Triplication Parkinson's Patient's iPSC-derived DA Neurons Accumulate α-Synuclein and Are Susceptible to Oxidative Stress
inferredcites →https://doi.org/10.1515/revneuro-2012-0071openalex-snapshot
The role of Ser129 phosphorylation of α-synuclein in neurodegeneration of Parkinson’s disease: a review of in vivo models
inferredcites →https://doi.org/10.1523/jneurosci.4441-10.2011openalex-snapshot
Mitochondrial Parkin Recruitment Is Impaired in Neurons Derived from Mutant PINK1 Induced Pluripotent Stem Cells
inferredcites →https://doi.org/10.3892/ijmm.2015.2061openalex-snapshot
Upregulation of RBFOX1 in the malformed cortex of patients with intractable epilepsy and in cultured rat neurons
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Roles of Ipsc in Advancing Treatments for Neurological Disorders
inferred← citeshttps://doi.org/10.1002/adbi.202000223openalex-snapshot
Electrophysiological Phenotype Characterization of Human iPSC‐Derived Neuronal Cell Lines by Means of High‐Density Microelectrode Arrays
inferred← citeshttps://doi.org/10.1002/adhm.202002119openalex-snapshot
Emerging Brain‐Pathophysiology‐Mimetic Platforms for Studying Neurodegenerative Diseases: Brain Organoids and Brains‐on‐a‐Chip
inferred← citeshttps://doi.org/10.1002/jcp.29800openalex-snapshot
Induced pluripotent stem cells (iPSCs) as game‐changing tools in the treatment of neurodegenerative disease: Mirage or reality?
inferred← citeshttps://doi.org/10.1002/wrna.1398openalex-snapshot
Developmental regulation of RNA processing by Rbfox proteins
inferred← citeshttps://doi.org/10.1007/5584_2021_643openalex-snapshot
Pluripotent Stem Cell Derived Neurons as In Vitro Models for Studying Autosomal Recessive Parkinson’s Disease (ARPD): PLA2G6 and Other Gene Loci
inferred← citeshttps://doi.org/10.1007/978-3-032-13729-6_18openalex-snapshot
Dopaminergic Cell Types Analysis in Parkinson’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-15-8061-1_20openalex-snapshot
Discovering Biomarkers in Parkinson’s Disease Using Module Correspondence and Pathway Information
inferred← citeshttps://doi.org/10.1007/978-981-97-4288-2_9openalex-snapshot
Altered RNA Metabolism in Neurodegenerative Disorders
inferred← citeshttps://doi.org/10.1007/s00018-020-03700-xopenalex-snapshot
Dissecting the non-neuronal cell contribution to Parkinson’s disease pathogenesis using induced pluripotent stem cells
inferred← citeshttps://doi.org/10.1007/s00441-017-2749-yopenalex-snapshot
iPS cells in the study of PD molecular pathogenesis
inferred← citeshttps://doi.org/10.1007/s00702-021-02348-6openalex-snapshot
Na+ leak-current channel (NALCN) at the junction of motor and neuropsychiatric symptoms in Parkinson’s disease
inferred← citeshttps://doi.org/10.1007/s11064-019-02781-wopenalex-snapshot
In Vivo Phenotyping of Familial Parkinson’s Disease with Human Induced Pluripotent Stem Cells: A Proof-of-Concept Study
inferred← citeshttps://doi.org/10.1007/s11910-018-0893-8openalex-snapshot
Using Patient-Derived Induced Pluripotent Stem Cells to Identify Parkinson’s Disease-Relevant Phenotypes
inferred← citeshttps://doi.org/10.1007/s12041-018-0960-6openalex-snapshot
Parkinson’s disease: what the model systems have taught us so far
inferred← citeshttps://doi.org/10.1007/s12640-022-00564-wopenalex-snapshot
CRISPR and iPSCs: Recent Developments and Future Perspectives in Neurodegenerative Disease Modelling, Research, and Therapeutics
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Engineered neural circuits for modeling brain physiology and neuropathology
inferred← citeshttps://doi.org/10.1016/j.bbadis.2019.03.004openalex-snapshot
Using induced pluripotent stem cell neuronal models to study neurodegenerative diseases
inferred← citeshttps://doi.org/10.1016/j.conb.2017.12.015openalex-snapshot
Reprogramming neurodegeneration in the big data era
inferred← citeshttps://doi.org/10.1016/j.conb.2021.09.001openalex-snapshot
Synaptic proteostasis in Parkinson's disease
inferred← citeshttps://doi.org/10.1016/j.gde.2017.01.014openalex-snapshot
Structural variants in SNCA gene and the implication to synucleinopathies
inferred← citeshttps://doi.org/10.1016/j.gde.2017.06.002openalex-snapshot
Modeling the genetic complexity of Parkinson's disease by targeted genome edition in iPS cells
inferred← citeshttps://doi.org/10.1016/j.jalz.2017.03.001openalex-snapshot
Genetic analysis of α‐synuclein 3′ untranslated region and its corresponding microRNAs in relation to Parkinson's disease compared to dementia with Lewy bodies
inferred← citeshttps://doi.org/10.1016/j.mcn.2020.103501openalex-snapshot
Decoding Parkinson's disease – iPSC-derived models in the OMICs era
inferred← citeshttps://doi.org/10.1016/j.nbd.2018.10.019openalex-snapshot
Phenotypic and molecular features underlying neurodegeneration of motor neurons derived from spinal and bulbar muscular atrophy patients
inferred← citeshttps://doi.org/10.1016/j.nbd.2022.105837openalex-snapshot
The multifaceted role of LRRK2 in Parkinson's disease: From human iPSC to organoids
inferred← citeshttps://doi.org/10.1016/j.neulet.2020.134777openalex-snapshot
Alterations in α-synuclein and PINK1 expression reduce neurite length and induce mitochondrial fission and Golgi fragmentation in midbrain neurons
inferred← citeshttps://doi.org/10.1016/j.omtn.2023.102084openalex-snapshot
Neuronal-type-specific epigenome editing to decrease SNCA expression: Implications for precision medicine in synucleinopathies
inferred← citeshttps://doi.org/10.1016/j.parkreldis.2018.06.025openalex-snapshot
Evidence for dopaminergic axonal degeneration as an early pathological process in Parkinson's disease
inferred← citeshttps://doi.org/10.1016/j.parkreldis.2019.04.008openalex-snapshot
Gene co-expression analysis of the human substantia nigra identifies BMP2 as a neurotrophic factor that can promote neurite growth in cells overexpressing wild-type or A53T α-synuclein
inferred← citeshttps://doi.org/10.1016/j.stem.2016.07.005openalex-snapshot
Midbrain-like Organoids from Human Pluripotent Stem Cells Contain Functional Dopaminergic and Neuromelanin-Producing Neurons
inferred← citeshttps://doi.org/10.1016/j.stemcr.2022.08.010openalex-snapshot
Age-related pathological impairments in directly reprogrammed dopaminergic neurons derived from patients with idiopathic Parkinson’s disease
inferred← citeshttps://doi.org/10.1016/j.ymthe.2018.08.019openalex-snapshot
Downregulation of SNCA Expression by Targeted Editing of DNA Methylation: A Potential Strategy for Precision Therapy in PD
inferred← citeshttps://doi.org/10.1038/mp.2017.66openalex-snapshot
An update on stem cell biology and engineering for brain development
inferred← citeshttps://doi.org/10.1038/s41380-019-0500-7openalex-snapshot
Three-dimensional modeling of human neurodegeneration: brain organoids coming of age
inferred← citeshttps://doi.org/10.1038/s41531-020-0110-8openalex-snapshot
Genetic predispositions of Parkinson’s disease revealed in patient-derived brain cells
inferred← citeshttps://doi.org/10.1038/s41531-022-00335-6openalex-snapshot
Lipase regulation of cellular fatty acid homeostasis as a Parkinson’s disease therapeutic strategy
inferred← citeshttps://doi.org/10.1038/s41531-022-00346-3openalex-snapshot
Genomic, transcriptomic, and metabolomic profiles of hiPSC-derived dopamine neurons from clinically discordant brothers with identical PRKN deletions
inferred← citeshttps://doi.org/10.1038/s41583-020-00416-6openalex-snapshot
Reverse engineering Lewy bodies: how far have we come and how far can we go?
inferred← citeshttps://doi.org/10.1038/s42003-024-07273-4openalex-snapshot
Modeling early phenotypes of Parkinson’s disease by age-induced midbrain-striatum assembloids
inferred← citeshttps://doi.org/10.1042/ns20210021openalex-snapshot
α-synuclein pathogenesis in hiPSC models of Parkinson’s disease
inferred← citeshttps://doi.org/10.1073/pnas.1900576116openalex-snapshot
Axonal pathology in hPSC-based models of Parkinson’s disease results from loss of Nrf2 transcriptional activity at the Map1b gene locus
inferred← citeshttps://doi.org/10.1080/17460441.2020.1703671openalex-snapshot
Organoid and pluripotent stem cells in Parkinson’s disease modeling: an expert view on their value to drug discovery
inferred← citeshttps://doi.org/10.1093/hmg/ddy355openalex-snapshot
Multiplication of theSNCAlocus exacerbates neuronal nuclear aging
inferred← citeshttps://doi.org/10.1093/oons/kvac011openalex-snapshot
RNA and neuronal function: the importance of post-transcriptional regulation
inferred← citeshttps://doi.org/10.1098/rsob.180138openalex-snapshot
Synaptic dysfunction in neurodegenerative and neurodevelopmental diseases: an overview of induced pluripotent stem-cell-based disease models
inferred← citeshttps://doi.org/10.1101/2020.07.24.219121openalex-snapshot
Neurodegeneration induces a developmental RNA processing program by calpain-mediated MBNL2 degradation
inferred← citeshttps://doi.org/10.1101/2021.04.23.441070openalex-snapshot
Age-related pathological impairments in induced neurons derived from patients with idiopathic Parkinson’s disease
inferred← citeshttps://doi.org/10.1101/2022.02.10.479903openalex-snapshot
An isogenic collection of pluripotent stem cell lines with elevated α-synuclein expression
inferred← citeshttps://doi.org/10.1101/2023.10.28.564305openalex-snapshot
Age-induced midbrain-striatum assembloids model early phenotypes of Parkinson’s disease
inferred← citeshttps://doi.org/10.1101/2024.05.28.596248openalex-snapshot
Fully Human Bifunctional Intrabodies Achieve Graded Reduction of Intracellular Tau and Rescue Survival of MAPT Mutation iPSC-derived Neurons
inferred← citeshttps://doi.org/10.1101/2024.11.06.622223openalex-snapshot
Correction of RBFOX1 deficit rescues Huntington’s disease mis-splicing and pathology
inferred← citeshttps://doi.org/10.1111/bpa.12526openalex-snapshot
Modeling Parkinson's disease with induced pluripotent stem cells harboring α‐synuclein mutations
inferred← citeshttps://doi.org/10.1111/ejn.14345openalex-snapshot
Induced pluripotent stem cell‐based modeling of mutant LRRK 2‐associated Parkinson's disease
inferred← citeshttps://doi.org/10.11606/t.41.2020.tde-15062020-162144openalex-snapshot
Análise comparativa de neurônios derivados de células-tronco pluripotentes induzidas (iPSC) para avaliar a associação da Doença de Gaucher e Doença de Parkinson
inferred← citeshttps://doi.org/10.1186/s10020-026-01471-yopenalex-snapshot
Modest rescue of RBFOX1 splicing function attenuates Huntington’s disease features
inferred← citeshttps://doi.org/10.1186/s12885-018-4440-4openalex-snapshot
Chronophin regulates active vitamin B6 levels and transcriptomic features of glioblastoma cell lines cultured under non-adherent, serum-free conditions
inferred← citeshttps://doi.org/10.1186/s13024-020-00385-5openalex-snapshot
Utility of plasma Neurofilament light as a diagnostic and prognostic biomarker of the postural instability gait disorder motor subtype in early Parkinson’s disease
inferred← citeshttps://doi.org/10.1186/s13024-023-00699-0openalex-snapshot
Unravelling cell type-specific responses to Parkinson’s Disease at single cell resolution
inferred← citeshttps://doi.org/10.1186/s13041-024-01086-6openalex-snapshot
Mutant α-synuclein causes death of human cortical neurons via ERK1/2 and JNK activation
inferred← citeshttps://doi.org/10.1186/s40478-018-0561-xopenalex-snapshot
Sporadic Parkinson’s disease derived neuronal cells show disease-specific mRNA and small RNA signatures with abundant deregulation of piRNAs
inferred← citeshttps://doi.org/10.1371/journal.pgen.1011855openalex-snapshot
Clusters of deep intronic RbFox motifs embedded in large assembly of splicing regulators sequences regulate alternative splicing
inferred← citeshttps://doi.org/10.15252/embj.2018101196openalex-snapshot
LIN28A loss of function is associated with Parkinson's disease pathogenesis
inferred← citeshttps://doi.org/10.20944/preprints202101.0480.v1openalex-snapshot
Differences between Cerebral Organoids Derived from a non-PD and a Parkinson’s Patient: A Potential Vulnerability for Neuroinvasive Viruses
inferred← citeshttps://doi.org/10.21037/atm.2019.11.16openalex-snapshot
A compendious summary of Parkinson’s disease patient-derived iPSCs in the first decade
inferred← citeshttps://doi.org/10.21203/rs.3.rs-4088206/v1openalex-snapshot
Engineered Nanobodies With Programmable Target Antigen Proteolysis (Ptap) Fusions Regulate Intracellular Alpha-synuclein in Vitro and in Vivo
inferred← citeshttps://doi.org/10.21203/rs.3.rs-7289863/v1openalex-snapshot
Caspase inhibition restores dopaminergic identity through the PKA–CREB–BDNF axis in Parkinson’s disease neurons
inferred← citeshttps://doi.org/10.21203/rs.3.rs-9117500/v1openalex-snapshot
Improving Neuropathological Reconstruction Fidelity via AI Slice Imputation
inferred← citeshttps://doi.org/10.3389/fcell.2019.00191openalex-snapshot
Gene Co-expression Analysis Identifies Histone Deacetylase 5 and 9 Expression in Midbrain Dopamine Neurons and as Regulators of Neurite Growth via Bone Morphogenetic Protein Signaling
inferred← citeshttps://doi.org/10.3389/fcell.2020.00359openalex-snapshot
Midbrain Organoids: A New Tool to Investigate Parkinson’s Disease
inferred← citeshttps://doi.org/10.3389/fcell.2022.898560openalex-snapshot
An Isogenic Collection of Pluripotent Stem Cell Lines With Elevated α-Synuclein Expression Validated for Neural Induction and Cortical Neuron Differentiation
inferred← citeshttps://doi.org/10.3389/fcell.2022.941984openalex-snapshot
Sevoflurane promotes premature differentiation of dopaminergic neurons in hiPSC-derived midbrain organoids
inferred← citeshttps://doi.org/10.3389/fncel.2019.00571openalex-snapshot
Modeling Cell-Cell Interactions in Parkinson’s Disease Using Human Stem Cell-Based Models
inferred← citeshttps://doi.org/10.3389/fneur.2021.786835openalex-snapshot
Targeting α-Synuclein in Parkinson's Disease by Induced Pluripotent Stem Cell Models
inferred← citeshttps://doi.org/10.3389/fnins.2022.847572openalex-snapshot
TrkB Truncated Isoform Receptors as Transducers and Determinants of BDNF Functions
inferred← citeshttps://doi.org/10.3389/fnins.2025.1584555openalex-snapshot
In vitro electrophysiological characterization of Parkinson’s disease: challenges, advances, and future directions
inferred← citeshttps://doi.org/10.3389/fnmol.2024.1498459openalex-snapshot
α-Synuclein ubiquitination – functions in proteostasis and development of Lewy bodies
inferred← citeshttps://doi.org/10.3390/biom12070876openalex-snapshot
Early Signs of Molecular Defects in iPSC-Derived Neural Stems Cells from Patients with Familial Parkinson’s Disease
inferred← citeshttps://doi.org/10.3390/biomedicines10102649openalex-snapshot
Cellular Models of Alpha-Synuclein Aggregation: What Have We Learned and Implications for Future Study
inferred← citeshttps://doi.org/10.3390/brainsci11030373openalex-snapshot
Modelling Parkinson’s Disease: iPSCs towards Better Understanding of Human Pathology
inferred← citeshttps://doi.org/10.3390/cells10010006openalex-snapshot
Direct Neuronal Reprogramming of Common Marmoset Fibroblasts by ASCL1, microRNA-9/9*, and microRNA-124 Overexpression
inferred← citeshttps://doi.org/10.3390/cells11213401openalex-snapshot
Visual Function and Survival of Injured Retinal Ganglion Cells in Aged Rbfox1 Knockout Animals
inferred← citeshttps://doi.org/10.3390/ijms21197113openalex-snapshot
Patient-Derived Induced Pluripotent Stem Cell-Based Models in Parkinson’s Disease for Drug Identification
inferred← citeshttps://doi.org/10.3390/jcm9010257openalex-snapshot
Regulation of BDNF-TrkB Signaling and Potential Therapeutic Strategies for Parkinson’s Disease
inferred← citeshttps://doi.org/10.3390/life16061007openalex-snapshot
Dysregulation of the Extracellular Matrix in the Synaptic Pathology of Parkinson’s Disease: Molecular Mechanisms and Novel Therapeutic Approaches
inferred← citeshttps://doi.org/10.3390/pathogens10070913openalex-snapshot
Cerebral Organoids Derived from a Parkinson’s Patient Exhibit Unique Pathogenesis from Chikungunya Virus Infection When Compared to a Non-Parkinson’s Patient
inferred← citeshttps://doi.org/10.3410/f.726596310.793560519openalex-snapshot
Faculty Opinions recommendation of Midbrain-like Organoids from Human Pluripotent Stem Cells Contain Functional Dopaminergic and Neuromelanin-Producing Neurons.
inferred← citeshttps://doi.org/10.34719/fhhg1227openalex-snapshot
Defining The Potential Of Gene Therapy With Bone Morphogenetic Proteins As A Novel Therapeutic Approach In Parkinson's Disease
inferred← citeshttps://doi.org/10.4103/nrr.nrr-d-25-00630openalex-snapshot
Advancements in differentiation of induced pluripotent stem cells into specialized neuronal subtypes
inferred← citeshttps://doi.org/10.4252/wjsc.v11.i9.634openalex-snapshot
Using induced pluripotent stem cells for modeling Parkinson’s disease
inferred← citeshttps://doi.org/10.5483/bmbrep.2019.52.6.142openalex-snapshot
Patient-specific pluripotent stem cell-based Parkinson’s disease models showing endogenous alpha-synuclein aggregation
inferred← citeshttps://doi.org/10.6084/m9.figshare.25347181openalex-snapshot
Additional file 1 of Mutant α-synuclein causes death of human cortical neurons via ERK1/2 and JNK activation
inferred← citeshttps://doi.org/10.6084/m9.figshare.25347265openalex-snapshot
Additional file 2 of Mutant α-synuclein causes death of human cortical neurons via ERK1/2 and JNK activation
inferred← citeshttps://doi.org/10.6084/m9.figshare.26689388openalex-snapshot
Additional file 3 of Mutant α-synuclein causes death of human cortical neurons via ERK1/2 and JNK activation
inferred← citeshttps://doi.org/10.6084/m9.figshare.26689391openalex-snapshot
Additional file 4 of Mutant α-synuclein causes death of human cortical neurons via ERK1/2 and JNK activation
inferred← citeshttps://doi.org/10.7554/elife.49673openalex-snapshot
Rbfox1 up-regulation impairs BDNF-dependent hippocampal LTP by dysregulating TrkB isoform expression levels
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