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The Role of Apolipoprotein E in Alzheimer's Disease

https://doi.org/10.1016/j.neuron.2009.06.026

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inferredcites →https://doi.org/10.1001/archneur.60.2.161openalex-snapshot

Apolipoprotein E, Alzheimer Disease, and African Americans

inferredcites →https://doi.org/10.1001/archneur.61.8.1200openalex-snapshot

Loss of Apolipoprotein E Receptor LR11 in Alzheimer Disease

inferredcites →https://doi.org/10.1001/archneur.62.3.454openalex-snapshot

Individual Growth Curve Analysis of APOE ε4–Associated Cognitive Decline in Alzheimer Disease

inferredcites →https://doi.org/10.1001/archpsyc.63.2.168openalex-snapshot

Role of Genes and Environments for Explaining Alzheimer Disease

inferredcites →https://doi.org/10.1001/jama.272.21.1666openalex-snapshot

Apolipoprotein E alleles, dyslipidemia, and coronary heart disease. The Framingham Offspring Study

inferredcites →https://doi.org/10.1001/jama.278.16.1349openalex-snapshot

Effects of age, sex, and ethnicity on the association between apolipoprotein E genotype and Alzheimer disease. A meta-analysis. APOE and Alzheimer Disease Meta Analysis Consortium

inferredcites →https://doi.org/10.1001/jama.279.10.751openalex-snapshot

The APOE -∊4 Allele and the Risk of Alzheimer Disease Among African Americans, Whites, and Hispanics

inferredcites →https://doi.org/10.1002/(sici)1097-4547(19961001)46:1<58::aid-jnr8>3.0.co;2-eopenalex-snapshot

Relative efficacies of amyloid ? peptide (A?) binding proteins in A? aggregation

inferredcites →https://doi.org/10.1002/1097-0029(20000815)50:4<316::aid-jemt11>3.0.co;2-eopenalex-snapshot

Influence of lipoproteins on microglial degradation of Alzheimer's amyloid beta-protein

inferredcites →https://doi.org/10.1002/1097-4547(20001115)62:4<608::aid-jnr16>3.0.co;2-4openalex-snapshot

Effect of apolipoprotein AII on the interaction of apolipoprotein E with ?-amyloid: Some apo(E-AII) complexes inhibit the internalization of ?-amyloid in cultures of neuroblastoma cells

inferredcites →https://doi.org/10.1002/ana.20730openalex-snapshot

Inverse relation between in vivo amyloid imaging load and cerebrospinal fluid Aβ 42 in humans

inferredcites →https://doi.org/10.1002/ana.21190openalex-snapshot

Neuronal LR11/sorLA expression is reduced in mild cognitive impairment

inferredcites →https://doi.org/10.1002/ana.410370509openalex-snapshot

Gene dose of the ε4 allele of apolipoprotein E and disease progression in sporadic late‐onset alzheimer's disease

inferredcites →https://doi.org/10.1002/ana.410390110openalex-snapshot

Clinical and pathological correlates of apolipoprotein E ε4 in Alzheimer's disease

inferredcites →https://doi.org/10.1002/ana.410390320openalex-snapshot

Aβ‐peptide length aid apolipoprotein E genotype in Alzheimer's disease

inferredcites →https://doi.org/10.1002/glia.20822openalex-snapshot

Binding and uptake of Aβ1‐42 by primary human astrocytes in vitro

inferredcites →https://doi.org/10.1002/jnr.10221openalex-snapshot

Defective neuronal sprouting by human apolipoprotein E4 is a gain‐of‐negative function

inferredcites →https://doi.org/10.1006/exnr.1998.6818openalex-snapshot

Evidence for Normal Aging of the Septo-Hippocampal Cholinergic System in apoE (−/−) Mice but Impaired Clearance of Axonal Degeneration Products Following Injury

inferredcites →https://doi.org/10.1006/exnr.1998.6860openalex-snapshot

High Frequency of Apolipoprotein E ϵ4 Allele in Young Individuals with Very Mild Alzheimer's Disease-Related Neurofibrillary Changes

inferredcites →https://doi.org/10.1006/exnr.2001.7715openalex-snapshot

Behavioral Phenotyping of GFAP-ApoE3 and -ApoE4 Transgenic Mice: ApoE4 Mice Show Profound Working Memory Impairments in the Absence of Alzheimer's-like Neuropathology

inferredcites →https://doi.org/10.1006/nbdi.1995.0019openalex-snapshot

Co-expression of β-amyloid precursor protein (βAPP) and apolipoprotein E in cell culture: analysis of βAPP processing

inferredcites →https://doi.org/10.1006/nbdi.1996.0023openalex-snapshot

Human Apolipoprotein E2, E3, and E4 Isoform-Specific Transgenic Mice: Human-like Pattern of GlialandNeuronal Immunoreactivity in Central Nervous System Not Observed in Wild-Type Mice

inferredcites →https://doi.org/10.1006/nbdi.1998.0213openalex-snapshot

Sialylated Human Apolipoprotein E (apoEs) Is Preferentially Associated with Neuron-Enriched Cultures from APOE Transgenic Mice

inferredcites →https://doi.org/10.1006/nbdi.2001.0401openalex-snapshot

Impaired Neuronal Plasticity in Transgenic Mice Expressing Human Apolipoprotein E4 Compared to E3 in a Model of Entorhinal Cortex Lesion

inferredcites →https://doi.org/10.1006/nbdi.2002.0483openalex-snapshot

Human and Murine ApoE Markedly Alters Aβ Metabolism before and after Plaque Formation in a Mouse Model of Alzheimer's Disease

inferredcites →https://doi.org/10.1006/neur.1995.0053openalex-snapshot

Effects of Apolipoprotein E Genotype on Cortical Neuropathology in Senile Dementia of the Lewy Body and Alzheimer's Disease

inferredcites →https://doi.org/10.1007/bf00225071openalex-snapshot

Apolipoprotein E (apoE) polymorphism and its influence on ApoE concentrations in the cerebrospinal fluid in Finnish patients with Alzheimer's disease

inferredcites →https://doi.org/10.1007/s00018-003-3018-7openalex-snapshot

Cholesterol homeostasis and function in neurons of the central nervous system

inferredcites →https://doi.org/10.1007/s004010000263openalex-snapshot

Apolipoprotein E affects the amount, form, and anatomical distribution of amyloid β-peptide deposition in homozygous APP V717F transgenic mice

inferredcites →https://doi.org/10.1007/s11064-008-9588-xopenalex-snapshot

Structural and Quantitative Comparison of Cerebrospinal Fluid Glycoproteins in Alzheimer’s Disease Patients and Healthy Individuals

inferredcites →https://doi.org/10.1016/0002-9343(65)90081-1openalex-snapshot

Tangier disease (familial high density lipoprotein deficiency)

inferredcites →https://doi.org/10.1016/0006-8993(91)91092-fopenalex-snapshot

Apolipoprotein E immunoreactivity in cerebral amyloid deposits and neurofibrillary tangles in Alzheimer's disease and kuru plaque amyloid in Creutzfeldt-Jakob disease

inferredcites →https://doi.org/10.1016/0006-8993(96)00183-7openalex-snapshot

Apolipoprotein E (apoE) levels in brains from Alzheimer disease patients and controls

inferredcites →https://doi.org/10.1016/0169-328x(94)00233-5openalex-snapshot

Apolipoprotein E genotype, Alzheimer's pathologies and related gene expression in the aged population

inferredcites →https://doi.org/10.1016/0169-328x(95)00097-copenalex-snapshot

Association of apolipoprotein E genotype with brain levels of apolipoprotein E and apolipoprotein J (clusterin) in Alzheimer disease

inferredcites →https://doi.org/10.1016/0197-4580(95)00076-qopenalex-snapshot

Alzheimer pathology of patients carrying apoliprotein E ε4 allele

inferredcites →https://doi.org/10.1016/0197-4580(96)00112-1openalex-snapshot

Alzheimer Aβ neurotoxicity: Promotion by antichymotrypsin, ApoE4; inhibition by Aβ-related peptides

inferredcites →https://doi.org/10.1016/0304-3940(92)90444-copenalex-snapshot

Apolipoprotein E: A pathological chaperone protein in patients with cerebral and systemic amyloid

inferredcites →https://doi.org/10.1016/0304-3940(94)12126-bopenalex-snapshot

Effect of apolipoprotein E genotype on Alzheimer's disease neuropathology in a cohort of elderly Norwegians

inferredcites →https://doi.org/10.1016/0304-3940(96)12774-9openalex-snapshot

Relevance of the quantification of apolipoprotein E in the cerebrospinal fluid in Alzheimer's disease

inferredcites →https://doi.org/10.1016/0306-4522(94)00596-wopenalex-snapshot

Apolipoprotein E polymorphism influences not only cerebral senile plaque load but also Alzheimer-type neurofibrillary tangle formation

inferredcites →https://doi.org/10.1016/0306-4522(95)00331-copenalex-snapshot

Influence of the apolipoprotein E genotype on amyloid deposition and neurofibrillary tangle formation in Alzheimer's disease

inferredcites →https://doi.org/10.1016/0735-1097(95)92270-fopenalex-snapshot

750-5 Apolipoprotein E Alleles, Dyslipidemia, and Coronary Heart Disease: The Framingham Offspring Study

inferredcites →https://doi.org/10.1016/0896-6273(93)90070-8openalex-snapshot

Apolipoprotein E in sporadic Alzheimer's disease: Allelic variation and receptor interactions

inferredcites →https://doi.org/10.1016/0896-6273(95)90079-9openalex-snapshot

Characterization of stable complexes involving apolipoprotein E and the amyloid β peptide in Alzheimer's disease brain

inferredcites →https://doi.org/10.1016/j.bbr.2004.09.019openalex-snapshot

Human apoE targeted replacement mouse lines: h-apoE4 and h-apoE3 mice differ on spatial memory performance and avoidance behavior

inferredcites →https://doi.org/10.1016/j.bbr.2008.05.008openalex-snapshot

Middle-aged human apoE4 targeted-replacement mice show retention deficits on a wide range of spatial memory tasks

inferredcites →https://doi.org/10.1016/j.biocel.2008.12.003openalex-snapshot

Human apolipoprotein A–I binds amyloid-β and prevents Aβ-induced neurotoxicity

inferredcites →https://doi.org/10.1016/j.biopsych.2004.07.021openalex-snapshot

Cerebrospinal fluid β-amyloid1–42 and tau in control subjects at risk for Alzheimer’s disease: The effect of APOE ε4 allele

inferredcites →https://doi.org/10.1016/j.cell.2005.02.008openalex-snapshot

Twenty Years of the Alzheimer’s Disease Amyloid Hypothesis: A Genetic Perspective

inferredcites →https://doi.org/10.1016/j.jneuroim.2003.10.015openalex-snapshot

APOE genotype-specific differences in human and mouse macrophage nitric oxide production

inferredcites →https://doi.org/10.1016/j.nbd.2004.10.013openalex-snapshot

Human apoE4-targeted replacement mice display synaptic deficits in the absence of neuropathology

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2005.09.011openalex-snapshot

Lack of LDL receptor aggravates learning deficits and amyloid deposits in Alzheimer transgenic mice

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2006.03.016openalex-snapshot

Apolipoprotein E polymorphism and dendritic shape in hippocampal interneurons

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2006.12.009openalex-snapshot

Genetic study evaluating LDLR polymorphisms and Alzheimer's disease

inferredcites →https://doi.org/10.1016/j.neurobiolaging.2007.11.014openalex-snapshot

APOE genotype-specific differences in the innate immune response

inferredcites →https://doi.org/10.1016/j.neuron.2005.06.030openalex-snapshot

Aβ42 Is Essential for Parenchymal and Vascular Amyloid Deposition in Mice

inferredcites →https://doi.org/10.1016/j.neuron.2007.08.008openalex-snapshot

Amyloid Precursor Protein Regulates Brain Apolipoprotein E and Cholesterol Metabolism through Lipoprotein Receptor LRP1

inferredcites →https://doi.org/10.1016/j.neuron.2008.01.003openalex-snapshot

The Blood-Brain Barrier in Health and Chronic Neurodegenerative Disorders

inferredcites →https://doi.org/10.1016/j.neuron.2008.02.003openalex-snapshot

Endocytosis Is Required for Synaptic Activity-Dependent Release of Amyloid-β In Vivo

inferredcites →https://doi.org/10.1016/j.neuron.2008.04.010openalex-snapshot

ApoE Promotes the Proteolytic Degradation of Aβ

inferredcites →https://doi.org/10.1016/j.neuroscience.2003.10.011openalex-snapshot

Marked regional differences of brain human apolipoprotein e expression in targeted replacement mice

inferredcites →https://doi.org/10.1016/j.neuroscience.2008.06.022openalex-snapshot

Clusterin expression during fetal and postnatal CNS development in mouse

inferredcites →https://doi.org/10.1016/j.numecd.2007.01.003openalex-snapshot

Apolipoprotein E polymorphism is associated with both carotid and coronary atherosclerosis in patients with coronary artery disease

inferredcites →https://doi.org/10.1016/j.sbi.2007.08.017openalex-snapshot

Versatility in ligand recognition by LDL receptor family proteins: advances and frontiers

inferredcites →https://doi.org/10.1016/j.tins.2003.08.005openalex-snapshot

New roles for astrocytes: Regulation of CNS synaptogenesis

inferredcites →https://doi.org/10.1016/s0002-9440(10)63399-8openalex-snapshot

ApoAI Deficiency Results in Marked Reductions in Plasma Cholesterol But No Alterations in Amyloid-β Pathology in a Mouse Model of Alzheimer's Disease-Like Cerebral Amyloidosis

inferredcites →https://doi.org/10.1016/s0006-8993(01)03367-4openalex-snapshot

Apolipoprotein E4 inhibits, and apolipoprotein E3 promotes neurite outgrowth in cultured adult mouse cortical neurons through the low-density lipoprotein receptor-related protein

inferredcites →https://doi.org/10.1016/s0006-8993(02)03437-6openalex-snapshot

Apolipoprotein E alters the processing of the β-amyloid precursor protein in APPV717F transgenic mice

inferredcites →https://doi.org/10.1016/s0006-8993(96)01363-7openalex-snapshot

Aβ1–40 but not Aβ1–42 levels in cortex correlate with apolipoprotein E ϵ4 allele dosage in sporadic Alzheimer's disease

inferredcites →https://doi.org/10.1016/s0006-8993(96)01420-5openalex-snapshot

Neurodegeneration and cognitive impairment in apoE-deficient mice is ameliorated by infusion of recombinant apoE

inferredcites →https://doi.org/10.1016/s0006-8993(97)01318-8openalex-snapshot

Amyloid-β peptide activates cultured astrocytes: morphological alterations, cytokine induction and nitric oxide release

inferredcites →https://doi.org/10.1016/s0006-8993(99)01542-5openalex-snapshot

Neuronal localization of a novel mosaic apolipoprotein E receptor, LR11, in rat and human brain

inferredcites →https://doi.org/10.1016/s0006-8993(99)01894-6openalex-snapshot

Apolipoprotein E and β-amyloid levels in the hippocampus and frontal cortex of Alzheimer's disease subjects are disease-related and apolipoprotein E genotype dependent

inferredcites →https://doi.org/10.1016/s0014-4886(03)00088-8openalex-snapshot

APOE ε3/ε4 heterozygotes have an elevated proportion of apolipoprotein E4 in cerebrospinal fluid relative to plasma, independent of Alzheimer’s disease diagnosis

inferredcites →https://doi.org/10.1016/s0021-9258(17)31529-6openalex-snapshot

Isoform-specific binding of apolipoprotein E to beta-amyloid.

inferredcites →https://doi.org/10.1016/s0021-9258(17)32449-3openalex-snapshot

Evidence that production and release of amyloid beta-protein involves the endocytic pathway

inferredcites →https://doi.org/10.1016/s0021-9258(18)47945-8openalex-snapshot

Lipoproteins and their receptors in the central nervous system. Characterization of the lipoproteins in cerebrospinal fluid and identification of apolipoprotein B,E(LDL) receptors in the brain.

inferredcites →https://doi.org/10.1016/s0021-9258(18)81907-xopenalex-snapshot

Glycosylation of Human Apolipoprotein E

inferredcites →https://doi.org/10.1016/s0165-5728(00)00459-8openalex-snapshot

Apolipoprotein E modulates glial activation and the endogenous central nervous system inflammatory response

inferredcites →https://doi.org/10.1016/s0169-328x(99)00073-xopenalex-snapshot

Apolipoprotein E isoform-specific reduction of extracellular amyloid in neuronal cultures

inferredcites →https://doi.org/10.1016/s0197-0186(00)00128-5openalex-snapshot

Effects of apolipoprotein E (apoE) isoforms, β-amyloid (Aβ) and apoE/Aβ complexes on Protein Kinase C-α (PKC-α) translocation and amyloid precursor protein (APP) processing in human SH-SY5Y neuroblastoma cells and fibroblasts

inferredcites →https://doi.org/10.1016/s0197-0186(01)00050-xopenalex-snapshot

Apolipoprotein E and apolipoprotein E receptors modulate Aβ-induced glial neuroinflammatory responses

inferredcites →https://doi.org/10.1016/s0197-4580(00)00143-3openalex-snapshot

Neuroinflammation and Alzheimer's disease: critical roles for cytokine/Aβ-induced glial activation, NF-κB, and apolipoprotein E

inferredcites →https://doi.org/10.1016/s0304-3940(03)00064-8openalex-snapshot

Apolipoprotein E (apoE) uptake and distribution in mammalian cell lines is dependent upon source of apoE and can be monitored in living cells

inferredcites →https://doi.org/10.1016/s0304-3940(96)13294-8openalex-snapshot

Preferential deposition of amyloid β protein (Aβ) in the form Aβ40 in Alzheimer's disease is associated with a gene dosage effect of the apolipoprotein E E4 allele

inferredcites →https://doi.org/10.1016/s0304-3940(97)00429-1openalex-snapshot

Cerebrospinal fluid apolipoprotein E (apoE) levels in Alzheimer's disease patients are increased at follow up and show a correlation with levels of tau protein

inferredcites →https://doi.org/10.1016/s0304-3940(99)00243-8openalex-snapshot

Differential neurotrophic effects of apolipoprotein E in aged transgenic mice

inferredcites →https://doi.org/10.1016/s0304-3940(99)00557-1openalex-snapshot

Lack of allelic imbalance in APOE ϵ 3/4 brain mRNA expression in Alzheimer's disease

inferredcites →https://doi.org/10.1016/s0306-4522(00)00065-8openalex-snapshot

Synaptic loss is accompanied by an increase in synaptic area in the dentate gyrus of aged human Apolipoprotein E4 transgenic mice

inferredcites →https://doi.org/10.1016/s0306-4522(97)00598-8openalex-snapshot

Behavioural, physiological and morphological analysis of a line of apolipoprotein E knockout mouse

inferredcites →https://doi.org/10.1016/s0306-4522(98)00561-2openalex-snapshot

Apolipoprotein E promotes the binding and uptake of β-amyloid into Chinese hamster ovary cells in an isoform-specific manner

inferredcites →https://doi.org/10.1016/s0896-6273(01)00475-5openalex-snapshot

Clearing the Brain's Amyloid Cobwebs

inferredcites →https://doi.org/10.1016/s0896-6273(03)00850-xopenalex-snapshot

ApoE and Clusterin Cooperatively Suppress Aβ Levels and Deposition

inferredcites →https://doi.org/10.1016/s0969-9961(03)00079-2openalex-snapshot

Dendritic spine loss in the hippocampus of young PDAPP and Tg2576 mice and its prevention by the ApoE2 genotype

inferredcites →https://doi.org/10.1016/s1388-1981(99)00196-1openalex-snapshot

Conformation of apolipoprotein E both in free and in lipid-bound form may determine the avidity of triglyceride-rich lipoproteins to the LDL receptor: structural and kinetic study

inferredcites →https://doi.org/10.1016/s1474-4422(08)70169-8openalex-snapshot

Alzheimer's disease and vascular dementia in developing countries: prevalence, management, and risk factors

inferredcites →https://doi.org/10.1021/bi002186kopenalex-snapshot

Apolipoprotein A-I Directly Interacts with Amyloid Precursor Protein and Inhibits Aβ Aggregation and Toxicity

inferredcites →https://doi.org/10.1021/bi961074jopenalex-snapshot

Seeding of Aβ Fibril Formation Is Inhibited by All Three Isotypes of Apolipoprotein E

inferredcites →https://doi.org/10.1021/bi9626362openalex-snapshot

Interaction of Nascent ApoE2, ApoE3, and ApoE4 Isoforms Expressed in Mammalian Cells with Amyloid Peptide β (1−40). Relevance to Alzheimer's Disease

inferredcites →https://doi.org/10.1038/11905openalex-snapshot

Mutations in ABC1 in Tangier disease and familial high-density lipoprotein deficiency

inferredcites →https://doi.org/10.1038/11914openalex-snapshot

The gene encoding ATP-binding cassette transporter 1 is mutated in Tangier disease

inferredcites →https://doi.org/10.1038/11921openalex-snapshot

Tangier disease is caused by mutations in the gene encoding ATP-binding cassette transporter 1

inferredcites →https://doi.org/10.1038/35006165openalex-snapshot

Apolipoprotein E and cognitive performance

inferredcites →https://doi.org/10.1038/35043065openalex-snapshot

Intramembrane proteolysis by presenilins

inferredcites →https://doi.org/10.1038/372092a0openalex-snapshot

Amyloid-associated proteins α1-antichymotrypsin and apolipoprotein E promote assembly of Alzheimer β-protein into filaments

inferredcites →https://doi.org/10.1038/42257openalex-snapshot

Microglial activation by Alzheimer amyloid precursor protein and modulation by apolipoprotein E

inferredcites →https://doi.org/10.1038/nature04001openalex-snapshot

Apolipoprotein-mediated pathways of lipid antigen presentation

inferredcites →https://doi.org/10.1038/ng0694-180openalex-snapshot

Protective effect of apolipoprotein E type 2 allele for late onset Alzheimer disease

inferredcites →https://doi.org/10.1038/ng1197-263openalex-snapshot

Lack of apolipoprotein E dramatically reduces amyloid β-peptide deposition

inferredcites →https://doi.org/10.1038/ng1934openalex-snapshot

Systematic meta-analyses of Alzheimer disease genetic association studies: the AlzGene database

inferredcites →https://doi.org/10.1038/ng1943openalex-snapshot

The neuronal sortilin-related receptor SORL1 is genetically associated with Alzheimer disease

inferredcites →https://doi.org/10.1038/nm1058openalex-snapshot

Apolipoprotein E promotes astrocyte colocalization and degradation of deposited amyloid-β peptides

inferredcites →https://doi.org/10.1038/nrm2101openalex-snapshot

Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid β-peptide

inferredcites →https://doi.org/10.1038/nrn2009openalex-snapshot

Reelin, lipoprotein receptors and synaptic plasticity

inferredcites →https://doi.org/10.1038/sj.emboj.7601930openalex-snapshot

The two faces of protein misfolding: gain‐ and loss‐of‐function in neurodegenerative diseases

inferredcites →https://doi.org/10.1038/sj.jcbfm.9600419openalex-snapshot

Transport Pathways for Clearance of Human Alzheimer's Amyloid β-Peptide and Apolipoproteins E and J in the Mouse Central Nervous System

inferredcites →https://doi.org/10.1042/0264-6021:3480359openalex-snapshot

Lipidation of apolipoprotein E influences its isoform-specific interaction with Alzheimer's amyloid β peptides

inferredcites →https://doi.org/10.1042/bj2930027openalex-snapshot

The cerebrospinal-fluid soluble form of Alzheimer's amyloid β is complexed to SP-40,40 (apolipoprotein J), an inhibitor of the complement membrane-attack complex

inferredcites →https://doi.org/10.1042/bj3060599openalex-snapshot

Fibrillogenesis in Alzheimer's disease of amyloid β peptides and apolipoprotein E

inferredcites →https://doi.org/10.1046/j.1471-4159.1996.66062429.xopenalex-snapshot

Brain Expression of Apolipoproteins E, J, and A‐I in Alzheimer's Disease

inferredcites →https://doi.org/10.1046/j.1471-4159.1997.68020587.xopenalex-snapshot

The Low‐Density Lipoprotein Receptor‐Related Protein, a Multifunctional Apolipoprotein E Receptor, Modulates Hippocampal Neurite Development

inferredcites →https://doi.org/10.1046/j.1471-4159.1997.68020721.xopenalex-snapshot

Characterization of the Binding of Amyloid‐β Peptide to Cell Culture‐Derived Native Apolipoprotein E2, E3, and E4 Isoforms and to Isoforms from Human Plasma

inferredcites →https://doi.org/10.1046/j.1471-4159.1997.68020760.xopenalex-snapshot

Effect of Apolipoprotein E on Neurite Outgrowth and β‐Amyloid‐Induced Toxicity in Developing Rat Primary Hippocampal Cultures

inferredcites →https://doi.org/10.1046/j.1471-4159.1997.69051995.xopenalex-snapshot

Isoform‐Specific Effects of Apolipoproteins E2, E3, and E4 on Cerebral Capillary Sequestration and Blood‐Brain Barrier Transport of Circulating Alzheimer's Amyloid β

inferredcites →https://doi.org/10.1046/j.1471-4159.1998.70041458.xopenalex-snapshot

β‐Amyloid Peptides Increase the Binding and Internalization of Apolipoprotein E to Hippocampal Neurons

inferredcites →https://doi.org/10.1046/j.1471-4159.1999.0732613.xopenalex-snapshot

Human Apolipoprotein E Isoform‐Specific Differences in Neuronal Sprouting in Organotypic Hippocampal Culture

inferredcites →https://doi.org/10.1056/nejm199511093331902openalex-snapshot

Apolipoprotein E, Dementia, and Cortical Deposition of β-Amyloid Protein

inferredcites →https://doi.org/10.1056/nejm199602293340913openalex-snapshot

Apolipoprotein E and the Neuropathology of Dementia

inferredcites →https://doi.org/10.1056/nejm199603213341202openalex-snapshot

Preclinical Evidence of Alzheimer's Disease in Persons Homozygous for the ε4 Allele for Apolipoprotein E

inferredcites →https://doi.org/10.1073/pnas.0308655100openalex-snapshot

Binding of F-spondin to amyloid-β precursor protein: A candidate amyloid-β precursor protein ligand that modulates amyloid-β precursor protein cleavage

inferredcites →https://doi.org/10.1073/pnas.0404458101openalex-snapshot

A nontransgenic mouse model shows inducible amyloid-β (Aβ) peptide deposition and elucidates the role of apolipoprotein E in the amyloid cascade

inferredcites →https://doi.org/10.1073/pnas.0409072102openalex-snapshot

Gene delivery of human apolipoprotein E alters brain Aβ burden in a mouse model of Alzheimer's disease

inferredcites →https://doi.org/10.1073/pnas.050004797openalex-snapshot

Apolipoprotein E isoform-dependent amyloid deposition and neuritic degeneration in a mouse model of Alzheimer's disease

inferredcites →https://doi.org/10.1073/pnas.0500579102openalex-snapshot

Correlations between apolipoprotein E ε4 gene dose and brain-imaging measurements of regional hypometabolism

inferredcites →https://doi.org/10.1073/pnas.0503689102openalex-snapshot

Neuronal sorting protein-related receptor sorLA/LR11 regulates processing of the amyloid precursor protein

inferredcites →https://doi.org/10.1073/pnas.0508254102openalex-snapshot

Lipid- and receptor-binding regions of apolipoprotein E4 fragments act in concert to cause mitochondrial dysfunction and neurotoxicity

inferredcites →https://doi.org/10.1073/pnas.0508693102openalex-snapshot

Apolipoprotein (apo) E4 enhances amyloid β peptide production in cultured neuronal cells: ApoE structure as a potential therapeutic target

inferredcites →https://doi.org/10.1073/pnas.0600549103openalex-snapshot

Apolipoprotein E4: A causative factor and therapeutic target in neuropathology, including Alzheimer’s disease

inferredcites →https://doi.org/10.1073/pnas.0604011103openalex-snapshot

Blocking the apolipoprotein E/amyloid-β interaction as a potential therapeutic approach for Alzheimer's disease

inferredcites →https://doi.org/10.1073/pnas.0608332104openalex-snapshot

The presenilin hypothesis of Alzheimer's disease: Evidence for a loss-of-function pathogenic mechanism

inferredcites →https://doi.org/10.1073/pnas.0900345106openalex-snapshot

Fibrillar amyloid-β burden in cognitively normal people at 3 levels of genetic risk for Alzheimer's disease

inferredcites →https://doi.org/10.1073/pnas.090106797openalex-snapshot

Cerebral metabolic and cognitive decline in persons at genetic risk for Alzheimer's disease

inferredcites →https://doi.org/10.1073/pnas.162228299openalex-snapshot

Clusterin promotes amyloid plaque formation and is critical for neuritic toxicity in a mouse model of Alzheimer's disease

inferredcites →https://doi.org/10.1073/pnas.2635903100openalex-snapshot

Functional brain abnormalities in young adults at genetic risk for late-onset Alzheimer's dementia

inferredcites →https://doi.org/10.1073/pnas.90.17.8098openalex-snapshot

Binding of human apolipoprotein E to synthetic amyloid beta peptide: isoform-specific effects and implications for late-onset Alzheimer disease.

inferredcites →https://doi.org/10.1073/pnas.90.20.9649openalex-snapshot

Increased amyloid beta-peptide deposition in cerebral cortex as a consequence of apolipoprotein E genotype in late-onset Alzheimer disease.

inferredcites →https://doi.org/10.1073/pnas.90.5.1977openalex-snapshot

Apolipoprotein E: high-avidity binding to beta-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer disease.

inferredcites →https://doi.org/10.1073/pnas.91.23.11183openalex-snapshot

Isoform-specific interactions of apolipoprotein E with microtubule-associated protein tau: implications for Alzheimer disease.

inferredcites →https://doi.org/10.1073/pnas.92.21.9480openalex-snapshot

Low density lipoprotein receptor-related protein mediates apolipoprotein E-dependent neurite outgrowth in a central nervous system-derived neuronal cell line.

inferredcites →https://doi.org/10.1073/pnas.92.3.763openalex-snapshot

Apolipoprotein E is a kinetic but not a thermodynamic inhibitor of amyloid formation: implications for the pathogenesis and treatment of Alzheimer disease.

inferredcites →https://doi.org/10.1073/pnas.95.18.10914openalex-snapshot

Isoform-specific effects of human apolipoprotein E on brain function revealed in ApoE knockout mice: Increased susceptibility of females

inferredcites →https://doi.org/10.1073/pnas.96.26.15233openalex-snapshot

Apolipoprotein E is essential for amyloid deposition in the APP V717F transgenic mouse model of Alzheimer's disease

inferredcites →https://doi.org/10.1074/jbc.270.34.19791openalex-snapshot

The Inhibitory Effect of Apolipoprotein E4 on Neurite Outgrowth Is Associated with Microtubule Depolymerization

inferredcites →https://doi.org/10.1074/jbc.270.45.27063openalex-snapshot

Stable Expression and Secretion of Apolipoproteins E3 and E4 in Mouse Neuroblastoma Cells Produces Differential Effects on Neurite Outgrowth

inferredcites →https://doi.org/10.1074/jbc.271.47.30121openalex-snapshot

Apolipoprotein E-containing High Density Lipoprotein Promotes Neurite Outgrowth and Is a Ligand for the Low Density Lipoprotein Receptor-related Protein

inferredcites →https://doi.org/10.1074/jbc.271.49.31379openalex-snapshot

Molecular Characterization of a Novel Human Hybrid-type Receptor That Binds the α2-Macroglobulin Receptor-associated Protein

inferredcites →https://doi.org/10.1074/jbc.272.29.17972openalex-snapshot

Targeted Replacement of the Mouse Apolipoprotein E Gene with the Common Human APOE3 Allele Enhances Diet-induced Hypercholesterolemia and Atherosclerosis

inferredcites →https://doi.org/10.1074/jbc.273.7.4206openalex-snapshot

A Minimally Lipidated Form of Cell-derived Apolipoprotein E Exhibits Isoform-specific Stimulation of Neurite Outgrowth in the Absence of Exogenous Lipids or Lipoproteins

inferredcites →https://doi.org/10.1074/jbc.274.42.30001openalex-snapshot

Unique Lipoproteins Secreted by Primary Astrocytes From Wild Type, apoE (−/−), and Human apoE Transgenic Mice

inferredcites →https://doi.org/10.1074/jbc.275.12.8564openalex-snapshot

Apolipoprotein E Is Resistant to Intracellular Degradation in Vitro and in Vivo

inferredcites →https://doi.org/10.1074/jbc.m000602200openalex-snapshot

Apolipoprotein E Receptors Mediate the Effects of β-Amyloid on Astrocyte Cultures

inferredcites →https://doi.org/10.1074/jbc.m306923200openalex-snapshot

APOE Genotype and an ApoE-mimetic Peptide Modify the Systemic and Central Nervous System Inflammatory Response

inferredcites →https://doi.org/10.1074/jbc.m407962200openalex-snapshot

Deficiency of ABCA1 Impairs Apolipoprotein E Metabolism in Brain

inferredcites →https://doi.org/10.1074/jbc.m407963200openalex-snapshot

ABCA1 Is Required for Normal Central Nervous System ApoE Levels and for Lipidation of Astrocyte-secreted apoE

inferredcites →https://doi.org/10.1074/jbc.m408127200openalex-snapshot

Increased tau Phosphorylation in Apolipoprotein E4 Transgenic Mice Is Associated with Activation of Extracellular Signal-regulated Kinase

inferredcites →https://doi.org/10.1074/jbc.m502143200openalex-snapshot

The Low Density Lipoprotein Receptor Regulates the Level of CentralNervous System Human and Murine Apolipoprotein E but Does Not Modify AmyloidPlaque Pathology in PDAPPMice

inferredcites →https://doi.org/10.1074/jbc.m504513200openalex-snapshot

Lack of ABCA1 Considerably Decreases Brain ApoE Level and Increases Amyloid Deposition in APP23 Mice

inferredcites →https://doi.org/10.1074/jbc.m508780200openalex-snapshot

Deletion of Abca1 Increases Aβ Deposition in the PDAPP Transgenic Mouse Model of Alzheimer Disease

inferredcites →https://doi.org/10.1074/jbc.m508781200openalex-snapshot

The Absence of ABCA1 Decreases Soluble ApoE Levels but Does Not Diminish Amyloid Deposition in Two Murine Models of Alzheimer Disease

inferredcites →https://doi.org/10.1074/jbc.m705073200openalex-snapshot

SorLA/LR11 Regulates Processing of Amyloid Precursor Protein via Interaction with Adaptors GGA and PACS-1

inferredcites →https://doi.org/10.1093/hmg/6.12.2151openalex-snapshot

Distortion of Allelic Expression of Apolipoprotein E in Alzheimer's Disease

inferredcites →https://doi.org/10.1093/hmg/9.3.353openalex-snapshot

Altered cholesterol metabolism in human apolipoprotein E4 knock-in mice

inferredcites →https://doi.org/10.1093/hmg/ddh199openalex-snapshot

Independent effects of APOE on cholesterol metabolism and brain A levels in an Alzheimer disease mouse model

inferredcites →https://doi.org/10.1093/hmg/ddh299openalex-snapshot

Allelic expression of APOE in human brain: effects of epsilon status and promoter haplotypes

inferredcites →https://doi.org/10.1093/hmg/ddm365openalex-snapshot

Sex-dependent association of a common low-density lipoprotein receptor polymorphism with RNA splicing efficiency in the brain and Alzheimer's disease

inferredcites →https://doi.org/10.1097/00001756-199810050-00031openalex-snapshot

ApoE associated with lipid has a reduced capacity to inhibit β-amyloid fibril formation

inferredcites →https://doi.org/10.1097/00001756-200412030-00020openalex-snapshot

ApoE isoform affects LTP in human targeted replacement mice

inferredcites →https://doi.org/10.1097/00002093-199809000-00012openalex-snapshot

Reduced Levels of Cholesterol, Phospholipids, and Fatty Acids in Cerebrospinal Fluid of Alzheimer Disease Patients Are Not Related to Apolipoprotein E4

inferredcites →https://doi.org/10.1101/gr.146900openalex-snapshot

Sequence Diversity and Large-Scale Typing of SNPs in the Human Apolipoprotein E Gene

inferredcites →https://doi.org/10.1111/j.1471-4159.2004.02581.xopenalex-snapshot

Apolipoprotein E modulates γ‐secretase cleavage of the amyloid precursor protein

inferredcites →https://doi.org/10.1111/j.1471-4159.2004.03007.xopenalex-snapshot

Multiple pathways of apolipoprotein E signaling in primary neurons

inferredcites →https://doi.org/10.1111/j.1471-4159.2005.03578.xopenalex-snapshot

Presenilin clinical mutations can affect γ‐secretase activity by different mechanisms

inferredcites →https://doi.org/10.1111/j.1471-4159.2007.04938.xopenalex-snapshot

Cerebral clearance of human amyloid‐β peptide (1–40) across the blood–brain barrier is reduced by self‐aggregation and formation of low‐density lipoprotein receptor‐related protein‐1 ligand complexes

inferredcites →https://doi.org/10.1111/j.1749-6632.2000.tb06365.xopenalex-snapshot

Lipoproteins in the Central Nervous System

inferredcites →https://doi.org/10.1126/science.1072994openalex-snapshot

The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics

inferredcites →https://doi.org/10.1126/science.3513311openalex-snapshot

A Receptor-Mediated Pathway for Cholesterol Homeostasis

inferredcites →https://doi.org/10.1126/science.8171342openalex-snapshot

Differential Effects of Apolipoproteins E3 and E4 on Neuronal Growth in Vitro

inferredcites →https://doi.org/10.1126/science.8346443openalex-snapshot

Gene Dose of Apolipoprotein E Type 4 Allele and the Risk of Alzheimer's Disease in Late Onset Families

inferredcites →https://doi.org/10.1128/mcb.25.21.9259-9268.2005openalex-snapshot

F-Spondin Interaction with the Apolipoprotein E Receptor ApoEr2 Affects Processing of Amyloid Precursor Protein

inferredcites →https://doi.org/10.1136/jnnp.61.4.352openalex-snapshot

The apolipoprotein E allele epsilon 4 does not correlate with the number of senile plaques or neurofibrillary tangles in patients with Alzheimer's disease.

inferredcites →https://doi.org/10.1146/annurev.biochem.71.110601.135342openalex-snapshot

Lipoprotein Receptors in the Nervous System

inferredcites →https://doi.org/10.1146/annurev.genom.1.1.507openalex-snapshot

Apolipoprotein E: Far More Than a Lipid Transport Protein

inferredcites →https://doi.org/10.1146/annurev.med.47.1.387openalex-snapshot

APOLIPOPROTEIN E ALLELES AS RISK FACTORS IN ALZHEIMER'S DISEASE

inferredcites →https://doi.org/10.1159/000106892openalex-snapshot

Apolipoprotein E in Cerebrospinal Fluid from Patients with Alzheimer's Disease and Other Forms of Dementia Is Reduced but without Any Correlation to the apoE4 Isoform

inferredcites →https://doi.org/10.1159/000337230openalex-snapshot

Amyloid Oligomers Exacerbate Tau Pathology in a Mouse Model of Tauopathy

inferredcites →https://doi.org/10.1172/jci117407openalex-snapshot

Apolipoprotein E associates with beta amyloid peptide of Alzheimer's disease to form novel monofibrils. Isoform apoE4 associates more efficiently than apoE3.

inferredcites →https://doi.org/10.1172/jci25247openalex-snapshot

P-glycoprotein deficiency at the blood-brain barrier increases amyloid- deposition in an Alzheimer disease mouse model

inferredcites →https://doi.org/10.1172/jci2673openalex-snapshot

Type III hyperlipoproteinemia and spontaneous atherosclerosis in mice resulting from gene replacement of mouse Apoe with human Apoe*2.

inferredcites →https://doi.org/10.1172/jci33622openalex-snapshot

Overexpression of ABCA1 reduces amyloid deposition in the PDAPP mouse model of Alzheimer disease

showing the first 200 of 242 · page through all 242

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Identification of Predictive Protein Signature in Mci and Ad in Body Fluids (Blood and Csf)

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Utility of Combinations of Biomarkers, Cognitive Markers, and Risk Factors to Predict Conversion From Mild Cognitive Impairment to Alzheimer Disease in Patients in the Alzheimer's Disease Neuroimaging Initiative

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Genetic Variation and Neuroimaging Measures in Alzheimer Disease

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New Perspectives on Amyloid-β Dynamics After Acute Brain Injury

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11C-PiB Imaging of Human Immunodeficiency Virus–Associated Neurocognitive Disorder

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Plasma Signaling Proteins in Persons at Genetic Risk for Alzheimer Disease

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Cerebrovascular Effects of Apolipoprotein E

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Longitudinal Cerebrospinal Fluid Biomarker Changes in Preclinical Alzheimer Disease During Middle Age

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Apolipoprotein E ε4 and Risk Factors for Alzheimer Disease—Let’s Talk About Sex

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Associations of the Top 20 Alzheimer Disease Risk Variants With Brain Amyloidosis

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Non‐amyloid Approaches To Alzheimer's Disease

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Generation of novel anti‐apo E monoclonal antibodies that selectively recognize apo E isoforms

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The Role of Genetics in Alzheimer's Disease and Parkinson's Disease

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Delivering the Promise of Gene Therapy with Nanomedicines in Treating Central Nervous System Diseases

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Therapeutic Gene Editing of APOE4 in Sporadic Alzheimer's Disease via Prime Editor 7

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A Robust Deep Temporal Causal Discovery Platform for Single‐Cell Gene Regulatory Network Reconstruction

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APOE and dementia – resequencing and genotyping in 105,597 individuals

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Large‐scale plasma proteomic profiling identifies a high‐performance biomarker panel for Alzheimer's disease screening and staging

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Protein phosphatase 2A and complement component 4 are linked to the protective effect of APOE ɛ2 for Alzheimer's disease

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Blood levels of MCP‐1 modulate the genetic risks of Alzheimer's disease mediated by HLA‐DRB1 and APOE for Alzheimer's disease

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AD and non‐AD mediators of the pathway between the APOE genotype and cognition

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Silencing Apoe with divalent‐siRNAs improves amyloid burden and activates immune response pathways in Alzheimer's disease

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Advancements in APOE and dementia research: Highlights from the 2023 AAIC Advancements: APOE conference

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Associations of cerebral amyloid beta and tau with cognition from midlife

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Benefits and risks of FDA‐approved amyloid‐targeting antibodies for treatment of early Alzheimer's disease: Navigating clinician‐patient engagement

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Circulating apoE4 protein levels from dried blood spots predict cognitive function in a large population‐based survey setting

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Deletion of miR‐33, a regulator of the ABCA1–APOE pathway, ameliorates neuropathological phenotypes in APP/PS1 mice

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Inhibiting mtDNA transcript translation alters Alzheimer's disease‐associated biology

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Conversion to Alzheimer's disease dementia from normal cognition directly or with the intermediate mild cognitive impairment stage

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Preclinical Alzheimer's disease shows alterations in circulating neuronal‐derived extracellular vesicle microRNAs in a multiethnic cohort

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Apolipoprotein ε4 exacerbates white matter impairment in a mouse model of Aβ amyloidosis by decreasing actively myelinating oligodendrocytes

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Pregnancy and breastfeeding are associated with less later‐life cognitive decline in a longitudinal, prospective cohort

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Spatially and temporally progressive hypoperfusion in Alzheimer's disease revealed by normative modeling

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Dissecting Alzheimer's disease heritability across populations

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APOE‐ε4 carriership and dementia risk over 10 years among Asian American and White older adults

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APOE ε2 is associated with milder clinical and pathological A lzheimer disease

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The Role of Genetics in Stroke Risk and Outcome: A Review of Current Evidence

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Cyclobutanes in Small‐Molecule Drug Candidates

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Astrocytic functions and lipid metabolism: Correlations and therapeutic targets in Alzheimer's disease and glioblastoma

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Apolipoprotein E gene polymorphism, trauma burden, and posttraumatic stress symptoms in U.S. military veterans: Results from the National Health and Resilience in Veterans Study

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A profile of brain reserve in adults at genetic risk of Alzheimer's disease

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Longitudinal CSF Alzheimer's disease biomarker changes from middle age to late adulthood

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Plasma biomarker trajectories: Impact of AD genetic risk and clinical progression

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Alzheimer Disease Biomarker Detection Through Electrocatalytic Water Oxidation Induced by Iridium Oxide Nanoparticles

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Multiple SNP Set Analysis for Genome‐Wide Association Studies Through Bayesian Latent Variable Selection

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APOE genotype alters glial activation and loss of synaptic markers in mice

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Targeting microglia for the treatment of Alzheimer's Disease

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Fate of microglia during HIV‐1 infection: From activation to senescence?

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Cocaine and HIV‐1 Tat disrupt cholesterol homeostasis in astrocytes: Implications for HIV‐associated neurocognitive disorders in cocaine user patients

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Novel Roles for the Ectoenzyme CD38 in the Maintenance of Transcriptional and Metabolic Homeostasis in Astrocytes

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The microRNA‐1908 up‐regulation in the peripheral blood cells impairs amyloid clearance by targeting ApoE

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The sex‐specific effect of the apolipoprotein E allele and methylenetetrahydrofolate reductase gene polymorphism on the biochemical, anatomical, and cognitive profiles of patients clinically diagnosed with probable Alzheimer's disease

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Relationship of medial temporal lobe atrophy, APOE genotype, and cognitive reserve in preclinical A lzheimer's disease

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Functional phenotyping of successful aging in long‐term memory: Preserved performance in the absence of neural compensation

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APOE ɛ4 constrains engagement of encoding-related compensatory networks in amnestic mild cognitive impairment

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Novel functionalization strategies of polymeric nanoparticles as carriers for brain medications

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Genetic mutations of APOEε4 carriers in cardiovascular patients lead to the development of insulin resistance and risk of Alzheimer's disease

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Retracted: Effect of microRNA‐186 on oxidative stress injury of neuron by targeting interleukin 2 through the janus kinase‐signal transducer and activator of transcription pathway in a rat model of Alzheimer’s disease

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An overview on microglial origin, distribution, and phenotype in Alzheimer's disease

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Animal models of source memory

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Localization of mature neprilysin in lipid rafts

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Alzheimer's Disease and Frontotemporal Dementia: A Review of Pathophysiology and Therapeutic Approaches

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Recent advances in the multitarget‐directed ligands approach for the treatment of Alzheimer's disease

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Discovery and Development of Small Molecule SHIP Phosphatase Modulators

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Structural and resting‐state MRI detects regional brain differences in young and mid‐age healthy APOE‐e4 carriers compared with non‐APOE‐e4 carriers

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Identification and removal of proteins that co‐purify with infectious prion protein improves the analysis of its secondary structure

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Neuroproteomic tools for battling Alzheimer's disease

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Expanding the cerebrospinal fluid endopeptidome

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Apolipoprotein E receptor pathways in Alzheimer disease

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Neurocognitive Impairment and Associated Genetic Aspects in HIV Infection

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Postinjury Issues and Ethics of Return to Play in Pediatric Concussion

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Role of Cellular Prion Protein in the Amyloid-β Oligomer Pathophysiology of Alzheimer’s Disease

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Remedies for Severe Class Imbalance

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An Introduction to Feature Selection

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A Short Tour of the Predictive Modeling Process

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Factors That Can Affect Model Performance

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Linear Regression and Its Cousins

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Functional MRI Studies of Memory in Aging, Mild Cognitive Impairment, and Alzheimer’s Disease

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Genetic Models of Alzheimer’s Disease: The Influence of Apolipoprotein E allele Isoforms on Behaviour in Laboratory Animals

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Neuropathology of HIV-1 Disease

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Morphometry and Genetics

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Genetic Risk Factors for Alzheimer Disease: Emerging Roles of Microglia in Disease Pathomechanisms

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The Impact of Apolipoprotein E Allelic Variants on Alzheimer’s Disease

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Force Spectroscopy in Mechanical Protein Domains Unfolding

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Apolipoprotein E

inferred← citeshttps://doi.org/10.1007/978-3-031-73368-0_155openalex-snapshot

Apolipoprotein E (APOE) Isoforms and Neurobiology of Sporadic Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/978-3-031-80849-4_13openalex-snapshot

Genomics and Transcriptomics of Alzheimer Disease

inferred← citeshttps://doi.org/10.1007/978-3-031-81728-1_28openalex-snapshot

Computational Multi-target Drug Design

inferred← citeshttps://doi.org/10.1007/978-3-031-81787-8_9openalex-snapshot

Biomarkers

inferred← citeshttps://doi.org/10.1007/978-3-031-96089-5_2openalex-snapshot

Apolipoprotein E

inferred← citeshttps://doi.org/10.1007/978-3-319-11355-5_3openalex-snapshot

Infectious Diseases: Need for Targeted Drug Delivery

inferred← citeshttps://doi.org/10.1007/978-3-319-15889-1_1openalex-snapshot

Neurochemical Aspects of Alzheimer Disease

inferred← citeshttps://doi.org/10.1007/978-3-319-17344-3_3openalex-snapshot

Lipids in Amyloid-β Processing, Aggregation, and Toxicity

inferred← citeshttps://doi.org/10.1007/978-3-319-21813-7_13openalex-snapshot

Neuroregeneration: Disease Modeling and Therapeutic Strategies for Alzheimer’s and Parkinson’s Diseases

inferred← citeshttps://doi.org/10.1007/978-3-319-57193-5_1openalex-snapshot

Alzheimer’s Disease: Insights from Genetic Mouse Models and Current Advances in Human IPSC-Derived Neurons

inferred← citeshttps://doi.org/10.1007/978-3-642-03637-8_47openalex-snapshot

Demenz

inferred← citeshttps://doi.org/10.1007/978-3-642-19795-6_5openalex-snapshot

Alzheimer-Demenz mit präsenilem Beginn – Besonderheiten in Diagnostik, Therapie und Management

inferred← citeshttps://doi.org/10.1007/978-3-642-19844-1_22openalex-snapshot

Tiermodelle für neurodegenerative Erkrankungen

inferred← citeshttps://doi.org/10.1007/978-3-642-45028-0_59-1openalex-snapshot

Demenz

inferred← citeshttps://doi.org/10.1007/978-3-662-49295-6_59openalex-snapshot

Demenz

inferred← citeshttps://doi.org/10.1007/978-3-7091-0932-8_25openalex-snapshot

Synaptic Dysfunction in Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/978-3-7091-1334-9_4openalex-snapshot

Gene Polymorphisms and Signaling Defects

inferred← citeshttps://doi.org/10.1007/978-981-16-3667-7_3openalex-snapshot

Progress of Nanotechnology-Based Detection and Treatment of Alzheimer’s Disease Biomarkers

inferred← citeshttps://doi.org/10.1007/978-981-16-6703-9_2openalex-snapshot

Deciphering the Molecular and Genetic Basis of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/978-981-16-7606-2_1openalex-snapshot

ApoE: A Risk Factor for Dementia

inferred← citeshttps://doi.org/10.1007/978-981-16-9205-5_13openalex-snapshot

Crosstalk Between Autophagy and Nutrigenomics in Neurodegenerative Diseases

inferred← citeshttps://doi.org/10.1007/978-981-19-5399-6_27openalex-snapshot

Repurposing of Minocycline, a Tetracycline Antibiotic, for Neurodegenerative Disorders

inferred← citeshttps://doi.org/10.1007/978-981-96-0259-9_10openalex-snapshot

Traditional Therapies for Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/978-981-96-0259-9_3openalex-snapshot

Alzheimer’s Disease: Epidemiology, Etiology, Risk Factors, and Future Predictions

inferred← citeshttps://doi.org/10.1007/978-981-97-4288-2_1openalex-snapshot

An Overview of Neurodegenerative Disorders

inferred← citeshttps://doi.org/10.1007/978-981-99-3848-3_8openalex-snapshot

Stem Cells Vs Exosomes: Promising Therapeutic Approach and Biomarkers Agent against Neurodegenerative Disorders

inferred← citeshttps://doi.org/10.1007/978-981-99-6038-5_11openalex-snapshot

Flavonoid-Based Derivatives for Modulating Various Targets of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/s00018-012-0977-6openalex-snapshot

PCSK9 regulates neuronal apoptosis by adjusting ApoER2 levels and signaling

inferred← citeshttps://doi.org/10.1007/s00018-014-1634-zopenalex-snapshot

Transient dynamics of Aβ contribute to toxicity in Alzheimer’s disease

inferred← citeshttps://doi.org/10.1007/s00018-020-03546-3openalex-snapshot

Studies of ApoD−/− and ApoD−/−ApoE−/− mice uncover the APOD significance for retinal metabolism, function, and status of chorioretinal blood vessels

inferred← citeshttps://doi.org/10.1007/s00018-021-03810-0openalex-snapshot

Cytokine signaling convergence regulates the microglial state transition in Alzheimer’s disease

inferred← citeshttps://doi.org/10.1007/s00109-016-1427-yopenalex-snapshot

Apolipoprotein E: from cardiovascular disease to neurodegenerative disorders

inferred← citeshttps://doi.org/10.1007/s00115-010-2951-6openalex-snapshot

Amyloiddarstellung mittels Positronenemissionstomographie

inferred← citeshttps://doi.org/10.1007/s00115-015-0041-5openalex-snapshot

Mechanismen der Alzheimer-Krankheit

inferred← citeshttps://doi.org/10.1007/s00115-019-0751-1openalex-snapshot

Hat die Alzheimer-Forschung versagt?

inferred← citeshttps://doi.org/10.1007/s00125-009-1533-2openalex-snapshot

Cognitive decline associated with dementia and type 2 diabetes: the interplay of risk factors

inferred← citeshttps://doi.org/10.1007/s00221-013-3464-xopenalex-snapshot

The number of cysteine residues per mole in apolipoprotein E affects systematically synchronous neural interactions in women’s healthy brains

inferred← citeshttps://doi.org/10.1007/s00232-024-00314-3openalex-snapshot

Aggregation Behavior of Amyloid Beta Peptide Depends Upon the Membrane Lipid Composition

inferred← citeshttps://doi.org/10.1007/s00259-011-1821-1openalex-snapshot

Individualized quantification of brain β-amyloid burden: results of a proof of mechanism phase 0 florbetaben PET trial in patients with Alzheimer’s disease and healthy controls

inferred← citeshttps://doi.org/10.1007/s00259-014-2919-zopenalex-snapshot

Cortical hypermetabolism in MCI subjects: a compensatory mechanism?

inferred← citeshttps://doi.org/10.1007/s00401-010-0679-9openalex-snapshot

Intraneuronal β-amyloid accumulation and synapse pathology in Alzheimer’s disease

inferred← citeshttps://doi.org/10.1007/s00401-011-0892-1openalex-snapshot

Apolipoprotein E level and cholesterol are associated with reduced synaptic amyloid beta in Alzheimer’s disease and apoE TR mouse cortex

inferred← citeshttps://doi.org/10.1007/s00401-012-0996-2openalex-snapshot

The genetics and neuropathology of Alzheimer’s disease

inferred← citeshttps://doi.org/10.1007/s00401-013-1190-xopenalex-snapshot

The neuropathology of obesity: insights from human disease

inferred← citeshttps://doi.org/10.1007/s00401-016-1637-yopenalex-snapshot

Depletion of TDP-43 decreases fibril and plaque β-amyloid and exacerbates neurodegeneration in an Alzheimer’s mouse model

inferred← citeshttps://doi.org/10.1007/s00401-019-02004-0openalex-snapshot

The new genetic landscape of Alzheimer’s disease: from amyloid cascade to genetically driven synaptic failure hypothesis?

inferred← citeshttps://doi.org/10.1007/s00401-020-02200-3openalex-snapshot

APOE and TREM2 regulate amyloid-responsive microglia in Alzheimer’s disease

inferred← citeshttps://doi.org/10.1007/s00403-009-1015-xopenalex-snapshot

Apolipoprotein E4 and psoriasis

inferred← citeshttps://doi.org/10.1007/s00406-012-0362-3openalex-snapshot

Current application of neurochemical biomarkers in the prediction and differential diagnosis of Alzheimer’s disease and other neurodegenerative dementias

inferred← citeshttps://doi.org/10.1007/s00406-023-01749-wopenalex-snapshot

Plasma Apo-E mediated corticospinal tract abnormalities and suicidality in patients with major depressive disorder

inferred← citeshttps://doi.org/10.1007/s00415-013-6908-0openalex-snapshot

Progressive apraxic agraphia with micrographia presenting as corticobasal syndrome showing extensive Pittsburgh compound B uptake

inferred← citeshttps://doi.org/10.1007/s00429-009-0236-2openalex-snapshot

Immunotherapeutic approaches for Alzheimer’s disease in transgenic mouse models

inferred← citeshttps://doi.org/10.1007/s00429-013-0606-7openalex-snapshot

Impact of a multi-nutrient diet on cognition, brain metabolism, hemodynamics, and plasticity in apoE4 carrier and apoE knockout mice

inferred← citeshttps://doi.org/10.1007/s00702-017-1796-6openalex-snapshot

Explorative results from multistep screening for potential genetic risk loci of Alzheimer’s disease in the longitudinal VITA study cohort

inferred← citeshttps://doi.org/10.1007/s00702-018-1934-9openalex-snapshot

Clinical implications of APOE genotyping for late-onset Alzheimer’s disease (LOAD) risk estimation: a review of the literature

inferred← citeshttps://doi.org/10.1007/s10072-010-0369-3openalex-snapshot

Cognitive deficits in experimental autoimmune encephalomyelitis: neuroinflammation and synaptic degeneration

inferred← citeshttps://doi.org/10.1007/s10072-021-05383-zopenalex-snapshot

ApoE4-positive multiple sclerosis patients are more likely to have cognitive impairment: a cross-sectional study

inferred← citeshttps://doi.org/10.1007/s10354-021-00819-9openalex-snapshot

Genetics of Alzheimer’s disease

inferred← citeshttps://doi.org/10.1007/s10571-010-9631-8openalex-snapshot

Gonadal Steroids do not Affect Apolipoprotein E Expression in Aging Mouse Cerebral Cortex

inferred← citeshttps://doi.org/10.1007/s10897-011-9377-6openalex-snapshot

Differences Between African American and White Research Volunteers in Their Attitudes, Beliefs and Knowledge Regarding Genetic Testing for Alzheimer's Disease

inferred← citeshttps://doi.org/10.1007/s11011-011-9252-zopenalex-snapshot

A high cholesterol diet given to apolipoprotein E-knockout mice has a differential effect on the various neurotrophin systems in the hippocampus

inferred← citeshttps://doi.org/10.1007/s11011-012-9310-1openalex-snapshot

Alzheimer’s disease and type 2 diabetes: the association study of polymorphisms in tumor necrosis factor-alpha and apolipoprotein E genes

inferred← citeshttps://doi.org/10.1007/s11011-015-9737-2openalex-snapshot

Apolipoprotein E genotyping and questionnaire-based assessment of lifestyle risk factors in dyslipidemic patients with a family history of Alzheimer’s disease: test development for clinical application

inferred← citeshttps://doi.org/10.1007/s11011-020-00584-5openalex-snapshot

Amelioration of motor and non-motor deficits and increased striatal APoE levels highlight the beneficial role of pistachio supplementation in rotenone-induced rat model of PD

inferred← citeshttps://doi.org/10.1007/s11011-026-01824-wopenalex-snapshot

The metabolic roots of memory loss: interlinking the Diabetes-Alzheimer’s hypothesis

inferred← citeshttps://doi.org/10.1007/s11033-024-09445-1openalex-snapshot

Beneficial effects of metformin treatment on memory impairment

inferred← citeshttps://doi.org/10.1007/s11064-021-03415-wopenalex-snapshot

Multifaceted Alzheimer’s Disease: Building a Roadmap for Advancement of Novel Therapies

inferred← citeshttps://doi.org/10.1007/s11065-014-9263-8openalex-snapshot

Imaging Brain Effects of APOE4 in Cognitively Normal Individuals Across the Lifespan

inferred← citeshttps://doi.org/10.1007/s11095-019-2691-7openalex-snapshot

ApoE-2 Brain-Targeted Gene Therapy Through Transferrin and Penetratin Tagged Liposomal Nanoparticles

inferred← citeshttps://doi.org/10.1007/s11222-023-10347-8openalex-snapshot

Doubly robust estimation and robust empirical likelihood in generalized linear models with missing responses

inferred← citeshttps://doi.org/10.1007/s11248-011-9499-5openalex-snapshot

Plasma proteomic profiles of bovine growth hormone transgenic mice as they age

inferred← citeshttps://doi.org/10.1007/s11357-022-00545-zopenalex-snapshot

Early-life educational attainment, APOE ε4 alleles, and incident dementia risk in late life

inferred← citeshttps://doi.org/10.1007/s11357-023-00883-6openalex-snapshot

Involvement of ApoE4 in dementia with Lewy bodies in the prodromal and demented stages: evaluation of the Strasbourg cohort

inferred← citeshttps://doi.org/10.1007/s11357-025-01695-6openalex-snapshot

Association of APOC1 with cortical atrophy during conversion to Alzheimer’s disease

inferred← citeshttps://doi.org/10.1007/s11571-019-09536-xopenalex-snapshot

Regulating effect of CBF on memory in cognitively normal older adults with different ApoE genotype: the Alzheimer’s Disease Neuroimaging Initiative (ADNI)

inferred← citeshttps://doi.org/10.1007/s11604-019-00867-7openalex-snapshot

Neuroimaging of Alzheimer’s disease: focus on amyloid and tau PET

inferred← citeshttps://doi.org/10.1007/s11682-016-9600-zopenalex-snapshot

Intrinsic functional connectivity alterations in cognitively intact elderly APOE ε4 carriers measured by eigenvector centrality mapping are related to cognition and CSF biomarkers: a preliminary study

inferred← citeshttps://doi.org/10.1007/s11745-014-3895-6openalex-snapshot

PCSK9 is Present in Human Cerebrospinal Fluid and is Maintained at Remarkably Constant Concentrations Throughout the Course of the Day

inferred← citeshttps://doi.org/10.1007/s11883-025-01321-yopenalex-snapshot

S-Nitrosylation: Mechanistic Links between Nitric Oxide Signaling and Atherosclerosis

inferred← citeshttps://doi.org/10.1007/s11910-014-0499-8openalex-snapshot

Genetic Heterogeneity in Alzheimer Disease and Implications for Treatment Strategies

inferred← citeshttps://doi.org/10.1007/s11910-018-0874-yopenalex-snapshot

The Genetics of Dementia with Lewy Bodies: Current Understanding and Future Directions

inferred← citeshttps://doi.org/10.1007/s12017-013-8257-7openalex-snapshot

SUMO and Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/s12017-014-8318-6openalex-snapshot

Apolipoprotein E Isoform-Specific Effects on Lipoprotein Receptor Processing

inferred← citeshttps://doi.org/10.1007/s12017-020-08610-6openalex-snapshot

Transcriptomic Heterogeneity of Alzheimer’s Disease Associated with Lipid Genetic Risk

inferred← citeshttps://doi.org/10.1007/s12031-010-9412-3openalex-snapshot

Age-Dependent Expression of Apolipoprotein E in Mouse Cerebral Cortex

inferred← citeshttps://doi.org/10.1007/s12031-012-9866-6openalex-snapshot

Stimulation of the Retinoid X Receptor Facilitates Beta-Amyloid Clearance Across the Blood–Brain Barrier

inferred← citeshttps://doi.org/10.1007/s12031-013-0038-0openalex-snapshot

Influence of Environmental Enrichment and Depleted Uranium on Behaviour, Cholesterol and Acetylcholine in Apolipoprotein E-Deficient Mice

inferred← citeshttps://doi.org/10.1007/s12031-015-0554-1openalex-snapshot

Genetic Variation of ApoE Gene in Ethnic Kashmiri Population and Its Association with Outcome After Traumatic Brain Injury

inferred← citeshttps://doi.org/10.1007/s12031-016-0804-xopenalex-snapshot

The Neurobiology and Age-Related Prevalence of the ε4 Allele of Apolipoprotein E in Alzheimer’s Disease Cohorts

inferred← citeshttps://doi.org/10.1007/s12031-016-0839-zopenalex-snapshot

The Complex Role of Apolipoprotein E in Alzheimer’s Disease: an Overview and Update

inferred← citeshttps://doi.org/10.1007/s12031-019-01381-1openalex-snapshot

Association between Apolipoprotein E Gene Polymorphism and Alzheimer's Disease in an Iranian Population: A Meta-Analysis

inferred← citeshttps://doi.org/10.1007/s12031-024-02224-4openalex-snapshot

Multiple Roles of Apolipoprotein E4 in Oxidative Lipid Metabolism and Ferroptosis During the Pathogenesis of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/s12035-011-8207-zopenalex-snapshot

The Interesting Interplay Between Interneurons and Adult Hippocampal Neurogenesis

inferred← citeshttps://doi.org/10.1007/s12035-012-8246-0openalex-snapshot

Protein Homeostasis, Aging and Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/s12035-012-8322-5openalex-snapshot

Neuroinflammation and Proteostasis are Modulated by Endogenously Biosynthesized Neuroprotectin D1

inferred← citeshttps://doi.org/10.1007/s12035-012-8337-yopenalex-snapshot

Role of Cholesterol in APP Metabolism and Its Significance in Alzheimer’s Disease Pathogenesis

inferred← citeshttps://doi.org/10.1007/s12035-015-9390-0openalex-snapshot

Dissecting Complex and Multifactorial Nature of Alzheimer’s Disease Pathogenesis: a Clinical, Genomic, and Systems Biology Perspective

inferred← citeshttps://doi.org/10.1007/s12035-017-0722-0openalex-snapshot

Co-Ultramicronized Palmitoylethanolamide/Luteolin Facilitates the Development of Differentiating and Undifferentiated Rat Oligodendrocyte Progenitor Cells

inferred← citeshttps://doi.org/10.1007/s12035-018-0892-4openalex-snapshot

The Interplay Between Apolipoprotein E4 and the Autophagic–Endocytic–Lysosomal Axis

inferred← citeshttps://doi.org/10.1007/s12035-018-0955-6openalex-snapshot

Early Presymptomatic Changes in the Proteome of Mitochondria-Associated Membrane in the APP/PS1 Mouse Model of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/s12035-019-01672-zopenalex-snapshot

Influence of Matrix Metallopeptidase 9 on Beta-Amyloid Elimination Across the Blood-Brain Barrier

inferred← citeshttps://doi.org/10.1007/s12035-022-02950-zopenalex-snapshot

Dysregulated miRNAs in Progression and Pathogenesis of Alzheimer’s Disease

inferred← citeshttps://doi.org/10.1007/s12035-025-05051-9openalex-snapshot

Glycation in Alzheimer’s Disease and Type 2 Diabetes: The Prospect of Dual Drug Approaches for Therapeutic Interventions

inferred← citeshttps://doi.org/10.1007/s12035-025-05465-5openalex-snapshot

Role of Mitochondrial Calcium Dysregulation in Alzheimer’s Disease Pathogenesis

inferred← citeshttps://doi.org/10.1007/s12035-026-05996-5openalex-snapshot

APOE4-Expressing Astrocytes Exhibit Parkinson’s Disease–Related Pathology

inferred← citeshttps://doi.org/10.1007/s12264-010-0731-8openalex-snapshot

Role of vitamin D in immune responses and autoimmune diseases, with emphasis on its role in multiple sclerosis

inferred← citeshttps://doi.org/10.1007/s12272-019-01148-7openalex-snapshot

Autophagy at synapses in neurodegenerative diseases

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