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Increased amyloid beta-peptide deposition in cerebral cortex as a consequence of apolipoprotein E genotype in late-onset Alzheimer disease.
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/archneur.1985.04060100083029openalex-snapshot
Diagnosis of Alzheimer's Disease
inferredcites →https://doi.org/10.1002/ana.410240110openalex-snapshot
Clinically diagnosed Alzheimer's disease: Autopsy results in 150 cases
inferredcites →https://doi.org/10.1002/ana.410270502openalex-snapshot
Synapse loss in frontal cortex biopsies in Alzheimer's disease: Correlation with cognitive severity
inferredcites →https://doi.org/10.1002/ana.410300410openalex-snapshot
Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment
inferredcites →https://doi.org/10.1006/bbrc.1993.1423openalex-snapshot
Apolipoprotein E: Binding to Soluble Alzheimer′s β-Amyloid
inferredcites →https://doi.org/10.1007/bf00308476openalex-snapshot
A comparative study of histological and immunohistochemical methods for neurofibrillary tangles and senile plaques in Alzheimer's disease
inferredcites →https://doi.org/10.1016/0006-8993(91)91092-fopenalex-snapshot
Apolipoprotein E immunoreactivity in cerebral amyloid deposits and neurofibrillary tangles in Alzheimer's disease and kuru plaque amyloid in Creutzfeldt-Jakob disease
inferredcites →https://doi.org/10.1016/0014-4886(88)90220-8openalex-snapshot
Genetic linkage studies in Alzheimer's disease families
inferredcites →https://doi.org/10.1016/0140-6736(91)92823-kopenalex-snapshot
Apolipoprotein E genotyping by one-stage PCR
inferredcites →https://doi.org/10.1016/0304-3940(86)90152-7openalex-snapshot
Monoclonal antibodies raised against a subsequence of senile plaque core protein react with plaque cores, plaque periphery and cerebrovascular amyloid in Alzheimer's disease
inferredcites →https://doi.org/10.1016/0304-3940(92)90444-copenalex-snapshot
Apolipoprotein E: A pathological chaperone protein in patients with cerebral and systemic amyloid
inferredcites →https://doi.org/10.1016/s0022-2275(20)43176-1openalex-snapshot
Restriction isotyping of human apolipoprotein E by gene amplification and cleavage with HhaI.
inferredcites →https://doi.org/10.1038/325733a0openalex-snapshot
The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor
inferredcites →https://doi.org/10.1038/341144a0openalex-snapshot
The secreted form of the Alzheimer's amyloid precursor protein with the Kunitz domain is protease nexin-II
inferredcites →https://doi.org/10.1038/341546a0openalex-snapshot
Protease nexin-II, a potent anti-chymotrypsin, shows identity to amyloid β-protein precursor
inferredcites →https://doi.org/10.1038/349704a0openalex-snapshot
Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease
inferredcites →https://doi.org/10.1038/353844a0openalex-snapshot
Early-onset Alzheimer's disease caused by mutations at codon 717 of the β-amyloid precursor protein gene
inferredcites →https://doi.org/10.1038/ng0692-218openalex-snapshot
Presenile dementia and cerebral haemorrhage linked to a mutation at codon 692 of the β–amyloid precursor protein gene
inferredcites →https://doi.org/10.1038/ng0892-345openalex-snapshot
A pathogenic mutation for probable Alzheimer's disease in the APP gene at the N–terminus of β–amyloid
inferredcites →https://doi.org/10.1073/pnas.90.17.8098openalex-snapshot
Binding of human apolipoprotein E to synthetic amyloid beta peptide: isoform-specific effects and implications for late-onset Alzheimer disease.
inferredcites →https://doi.org/10.1073/pnas.90.5.1977openalex-snapshot
Apolipoprotein E: high-avidity binding to beta-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer disease.
inferredcites →https://doi.org/10.1097/00005072-199107000-00001openalex-snapshot
Macrophage Apolipoprotein Synthesis and Endoneurial Distribution as a Response to Segmental Demyelination
inferredcites →https://doi.org/10.1097/00005072-199201000-00009openalex-snapshot
Kunitz Protease Inhibitor-Containing Amyloid β Protein Precursor Immunoreactivity in Alzheimer's Disease
inferredcites →https://doi.org/10.1126/science.1925564openalex-snapshot
A mutation in the Amyloid Precursor Protein Associated with Hereditary Alzheimer's Disease
inferredcites →https://doi.org/10.1126/science.2949367openalex-snapshot
Amyloid β Protein Gene: cDNA, mRNA Distribution, and Genetic Linkage Near the Alzheimer Locus
inferredcites →https://doi.org/10.1126/science.3544219openalex-snapshot
Conservation of Brain Amyloid Proteins in Aged Mammals and Humans with Alzheimer's Disease
inferredcites →https://doi.org/10.1126/science.3810169openalex-snapshot
Characterization and Chromosomal Localization of a cDNA Encoding Brain Amyloid of Alzheimer's Disease
inferredcites →https://doi.org/10.1126/science.8346443openalex-snapshot
Gene Dose of Apolipoprotein E Type 4 Allele and the Risk of Alzheimer's Disease in Late Onset Families
inferredcites →https://doi.org/10.1128/jvi.65.9.4759-4768.1991openalex-snapshot
Neuropathological changes in scrapie and Alzheimer's disease are associated with increased expression of apolipoprotein E and cathepsin D in astrocytes
inferredcites →https://doi.org/10.1172/jci113568openalex-snapshot
Immunohistochemical localization of apolipoprotein E in human glial neoplasms.
inferredcites →https://doi.org/10.1212/wnl.25.2.120openalex-snapshot
The incidence of cerebral amyloid angiopathy in Alzheimer's disease
inferredcites →https://doi.org/10.1212/wnl.41.4.479openalex-snapshot
The Consortium to Establish a Registry for Alzheimer's Disease (CERAD)
inferredcites →https://doi.org/10.1212/wnl.43.8.1467openalex-snapshot
Association of apolipoprotein E allele ϵ4 with late‐onset familial and sporadic Alzheimer's disease
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4741157openalex-extracted
Synthesis and Biological Evaluation of Tacrine-Diterpenoid Alkaloid Hybrids as Multifunctional Agents Against Alzheimer's Disease
inferred← citeshttps://doi.org/10.1001/archinte.162.17.1934openalex-snapshot
The Role of Hormone Replacement Therapy in the Prevention of Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archneur.1995.00540350068018openalex-snapshot
Apolipoprotein E ε4 Allele and the Lifetime Risk of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1001/archneur.1996.00550040085017openalex-snapshot
Apolipoprotein E Genotype and Rate of Decline in Probable Alzheimer's Disease
inferred← citeshttps://doi.org/10.1001/archneur.1996.00550120041014openalex-snapshot
Apolipoprotein E Phenotype Frequency and Cerebrospinal Fluid Concentration Are Not Associated With Creutzfeldt-Jakob Disease
inferred← citeshttps://doi.org/10.1001/archneur.1997.00550210044011openalex-snapshot
Apolipoprotein E Phenotype and Cognitive Decline in a Prospective Study of Elderly Community Women
inferred← citeshttps://doi.org/10.1001/archneur.55.11.1460openalex-snapshot
Absence of an Apolipoprotein E ϵ4 Allele Is Associated With Increased Parietal Regional Cerebral Blood Flow Asymmetry in Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archneur.55.3.326openalex-snapshot
Clinicopathologic Studies in Cognitively Healthy Aging and Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archneur.55.7.1001openalex-snapshot
Apolipoprotein E Genotype and Deposits of Aβ40 and Aβ42 in Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archneur.55.7.937openalex-snapshot
High Cerebrospinal Fluid Tau and Low Amyloid β42 Levels in the Clinical Diagnosis of Alzheimer Disease and Relation to Apolipoprotein E Genotype
inferred← citeshttps://doi.org/10.1001/archneur.56.9.1111openalex-snapshot
Deposition of β-Amyloid Subtypes 40 and 42 Differentiates Dementia With Lewy Bodies From Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archneur.57.1.100openalex-snapshot
Plasma and Cerebrospinal Fluid Levels of Amyloid β Proteins 1-40 and 1-42 in Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archneur.57.5.646openalex-snapshot
Role of the Low-density Lipoprotein Receptor–Related Protein in β-Amyloid Metabolism and Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/archneur.60.5.729openalex-snapshot
Neurofibrillary Tangles, Amyloid, and Memory in Aging and Mild Cognitive Impairment
inferred← citeshttps://doi.org/10.1001/archneur.61.3.341openalex-snapshot
Increased Risk for Alzheimer Disease With the Interaction of MPO and A2M Polymorphisms
inferred← citeshttps://doi.org/10.1001/archneur.63.2.189openalex-snapshot
Apolipoprotein E and Dementia in Parkinson Disease
inferred← citeshttps://doi.org/10.1001/archneur.65.7.875openalex-snapshot
Abeta Predictor of Alzheimer Disease Symptoms
inferred← citeshttps://doi.org/10.1001/archneurol.2009.224openalex-snapshot
Codistribution of Amyloid β Plaques and Spongiform Degeneration in Familial Creutzfeldt-Jakob Disease With the E200K-129M Haplotype
inferred← citeshttps://doi.org/10.1001/archneurol.2009.237openalex-snapshot
Effects of Family History and Apolipoprotein E ε4 Status on Cognitive Decline in the Absence of Alzheimer Dementia
inferred← citeshttps://doi.org/10.1001/jamaneurol.2013.5200openalex-snapshot
APOEGenotype and Brain Development
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.0834openalex-snapshot
Association of Cortical β-Amyloid Protein in the Absence of Insoluble Deposits With Alzheimer Disease
inferred← citeshttps://doi.org/10.1002/(sici)1096-8628(19980207)81:1<49::aid-ajmg10>3.0.co;2-wopenalex-snapshot
Alzheimer diseases: a model of gene mutations and susceptibility polymorphisms for complex psychiatric diseases
inferred← citeshttps://doi.org/10.1002/(sici)1096-8628(19980207)81:1<98::aid-ajmg17>3.0.co;2-ropenalex-snapshot
Initial results of a genome survey for novel alzheimer's disease risk genes: association with a locus on the X chromosome
inferred← citeshttps://doi.org/10.1002/(sici)1096-8628(19980328)81:2<196::aid-ajmg14>3.0.co;2-copenalex-snapshot
Initial results of a genome survey for novel alzheimer's disease risk genes: Association with a locus on the X chromosome
inferred← citeshttps://doi.org/10.1002/(sici)1096-9861(19980713)396:4<511::aid-cne7>3.0.co;2-4openalex-snapshot
Expression of differential immune factors in temporal cortex and cerebellum: The role of ?-1-antichymotrypsin, apolipoprotein E, and reactive glia in the progression of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/(sici)1097-4547(19970701)49:1<9::aid-jnr2>3.0.co;2-hopenalex-snapshot
Association of human, rat, and rabbit apolipoprotein E with ?-amyloid
inferred← citeshttps://doi.org/10.1002/(sici)1097-4547(19981001)54:1<58::aid-jnr7>3.0.co;2-gopenalex-snapshot
Apolipoprotein E4 induces neuronal cell death under conditions of suppressed de novo cholesterol synthesis
inferred← citeshttps://doi.org/10.1002/(sici)1097-4547(19991015)58:2<301::aid-jnr10>3.0.co;2-9openalex-snapshot
Higher avidity binding of apolipoprotein (E-AII) complex than of apolipoprotein E monomer to ?-amyloid
inferred← citeshttps://doi.org/10.1002/(sici)1097-4547(20000401)60:1<73::aid-jnr8>3.0.co;2-sopenalex-snapshot
Broadly altered expression of the mRNA isoforms of FE65, a facilitator of beta amyloidogenesis, in Alzheimer cerebellum and other brain regions
inferred← citeshttps://doi.org/10.1002/(sici)1097-4598(199612)19:12<1605::aid-mus11>3.0.co;2-qopenalex-snapshot
Apolipoprotein E allele frequencies in sporadic inclusion body myositis
inferred← citeshttps://doi.org/10.1002/(sici)1097-4695(199709)33:3<329::aid-neu10>3.0.co;2-aopenalex-snapshot
Protection against developmental retardation in apolipoprotein E-deficient mice by a fatty neuropeptide: Implications for early treatment of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/(sici)1097-4695(19990605)39:3<371::aid-neu4>3.0.co;2-eopenalex-snapshot
?-sheet breaker peptide inhibitor of Alzheimer's amyloidogenesis with increased blood-brain barrier permeability and resistance to proteolytic degradation in plasma
inferred← citeshttps://doi.org/10.1002/(sici)1099-1077(200001)15:1<1::aid-hup153>3.0.co;2-1openalex-snapshot
A unifying hypothesis of Alzheimer's disease. III. Risk factors
inferred← citeshttps://doi.org/10.1002/(sici)1099-1166(1998110)13:11<767::aid-gps866>3.0.co;2-fopenalex-snapshot
Apolipoprotein E ?4 allele in association with global cognitive performance and CSF markers in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/(sici)1099-1166(199909)14:9<695::aid-gps19>3.0.co;2-ropenalex-snapshot
The current status of Apo E4 as a risk factor for Alzheimer's disease: an epidemiological perspective
inferred← citeshttps://doi.org/10.1002/(sici)1099-1166(200003)15:3<260::aid-gps109>3.0.co;2-mopenalex-snapshot
Association of serum AACT levels and AACT signal polymorphism with late-onset Alzheimer's disease in Northern Ireland
inferred← citeshttps://doi.org/10.1002/0471266949.bmc106openalex-snapshot
Alzheimer's Disease: Search for Therapeutics
inferred← citeshttps://doi.org/10.1002/1097-0029(20000815)50:4<268::aid-jemt3>3.0.co;2-1openalex-snapshot
LDL receptor-related protein (LRP) in Alzheimer's disease: Towards a unified theory of pathogenesis
inferred← citeshttps://doi.org/10.1002/1097-0029(20000815)50:4<278::aid-jemt5>3.0.co;2-topenalex-snapshot
Apolipoprotein E isoforms in Alzheimer's disease pathology and etiology
inferred← citeshttps://doi.org/10.1002/1097-0029(20000815)50:4<291::aid-jemt8>3.0.co;2-lopenalex-snapshot
Roles for lipoprotein lipase in Alzheimer's disease: An association study
inferred← citeshttps://doi.org/10.1002/1097-0029(20000815)50:4<297::aid-jemt9>3.0.co;2-copenalex-snapshot
Astrocyte lipoproteins, effects of apoE on neuronal function, and role of apoE in amyloid-? deposition in vivo
inferred← citeshttps://doi.org/10.1002/1097-0029(20000815)50:4<316::aid-jemt11>3.0.co;2-eopenalex-snapshot
Influence of lipoproteins on microglial degradation of Alzheimer's amyloid beta-protein
inferred← citeshttps://doi.org/10.1002/1531-8249(199909)46:3<382::aid-ana14>3.0.co;2-5openalex-snapshot
Genetic association of ?2-macroglobulin with Alzheimer's disease in a Finnish elderly population
inferred← citeshttps://doi.org/10.1002/1531-8249(200006)47:6<739::aid-ana6>3.0.co;2-8openalex-snapshot
Apolipoprotein E facilitates neuritic and cerebrovascular plaque formation in an Alzheimer's disease model
inferred← citeshttps://doi.org/10.1002/1531-8249(200008)48:2<201::aid-ana10>3.0.co;2-xopenalex-snapshot
Differences in the A?40/A?42 ratio associated with cerebrospinal fluid lipoproteins as a function of apolipoprotein E genotype
inferred← citeshttps://doi.org/10.1002/9780470514924.ch10openalex-snapshot
Interaction of Transthyretin with Amyloid β‐Protein: Binding and Inhibition of Amyloid Formation
inferred← citeshttps://doi.org/10.1002/9780470514924.ch8openalex-snapshot
Alzheimer's Disease: Genesis of Amyloid
inferred← citeshttps://doi.org/10.1002/9780470514924.ch9openalex-snapshot
Apolipoprotein E and Amyloidogenesis
inferred← citeshttps://doi.org/10.1002/9780470571224.pse456openalex-snapshot
Overview of CNS Neuropharmaceuticals
inferred← citeshttps://doi.org/10.1002/9780470572702.ch27openalex-snapshot
Role of Oxidatively Stressed Lipids in Amyloid Formation and Toxicity
inferred← citeshttps://doi.org/10.1002/9781444344110.ch6openalex-snapshot
Genetic Risk Factors for Dementia
inferred← citeshttps://doi.org/10.1002/9783527619344.ch10openalex-snapshot
Apolipoprotein E: Structural and Functional Interactions with Amyloid β
inferred← citeshttps://doi.org/10.1002/ajmg.1320600519openalex-snapshot
Apolipoprotein E in the genetics and epidemiology of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/ajmg.b.30087openalex-snapshot
APOE and other loci affect age‐at‐onset in Alzheimer's disease families with PS2 mutation
inferred← citeshttps://doi.org/10.1002/ajmg.b.30896openalex-snapshot
Evidence supporting a role for the calcium‐sensing receptor in Alzheimer disease
inferred← citeshttps://doi.org/10.1002/ajmg.b.32499openalex-snapshot
Genomic variants, genes, and pathways of Alzheimer's disease: An overview
inferred← citeshttps://doi.org/10.1002/alz.14392openalex-snapshot
APOE ε4–associated heterogeneity of neuroimaging biomarkers across the Alzheimer's disease continuum
inferred← citeshttps://doi.org/10.1002/alz.14498openalex-snapshot
Long non‐coding RNAs as key regulators of neurodegenerative protein aggregation
inferred← citeshttps://doi.org/10.1002/alz.70594openalex-snapshot
Prevalence and risk factors of cerebral microhemorrhages and superficial siderosis in cognitively unimpaired older adults: analysis from the CHARIOT‐PRO SubStudy
inferred← citeshttps://doi.org/10.1002/alz.71586openalex-snapshot
Association of cognitive impairment and APOE ε4 with Centiloids in Hispanic and non‐Hispanic White cohorts
inferred← citeshttps://doi.org/10.1002/ana.10810openalex-snapshot
Clinical manifestations of cerebral amyloid angiopathy–related inflammation
inferred← citeshttps://doi.org/10.1002/ana.1134openalex-snapshot
Human apolipoprotein E4 accelerates β‐amyloid deposition in APPsw transgenic mouse brain
inferred← citeshttps://doi.org/10.1002/ana.1159openalex-snapshot
Relationship of the extended tau haplotype to tau biochemistry and neuropathology in progressive supranuclear palsy
inferred← citeshttps://doi.org/10.1002/ana.21843openalex-snapshot
APOE predicts amyloid‐beta but not tau Alzheimer pathology in cognitively normal aging
inferred← citeshttps://doi.org/10.1002/ana.22516openalex-snapshot
Cerebral amyloid angiopathy in the elderly
inferred← citeshttps://doi.org/10.1002/ana.24369openalex-snapshot
APOE ε2 is associated with milder clinical and pathological A lzheimer disease
inferred← citeshttps://doi.org/10.1002/ana.24992openalex-snapshot
Brain microvascular injury and white matter disease provoked by diabetes‐associated hyperamylinemia
inferred← citeshttps://doi.org/10.1002/ana.410360315openalex-snapshot
The apolipoprotein E ε4 allele does not influence the clinical expression of the amyloid precursor protein gene codon 693 or 692 mutations
inferred← citeshttps://doi.org/10.1002/ana.410370212openalex-snapshot
Multiple, diverse senile plaque–associated proteins are ligands of an apolipoprotein e receptor, the α2‐macroglobulin receptor/low‐density‐lipoprotein receptor—related protein
inferred← citeshttps://doi.org/10.1002/ana.410370215openalex-snapshot
Apolipoprotein E genotypes in Parkinson's disease with and without dementia
inferred← citeshttps://doi.org/10.1002/ana.410370414openalex-snapshot
Cerebrospinal fluid levels of amyloid β‐protein in alzheimer's disease: Inverse correlation with severity of dementia and effect of apolipoprotein e genotype
inferred← citeshttps://doi.org/10.1002/ana.410370509openalex-snapshot
Gene dose of the ε4 allele of apolipoprotein E and disease progression in sporadic late‐onset alzheimer's disease
inferred← citeshttps://doi.org/10.1002/ana.410370510openalex-snapshot
The apolipoprotein E ε2 allele is associated with an increased risk of early‐onset alzheimer's disease and a reduced survival
inferred← citeshttps://doi.org/10.1002/ana.410380120openalex-snapshot
Epistatic effect of APP717 mutation and apolipoprotein E genotype in familial Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/ana.410380122openalex-snapshot
Apolipoprotein E genotype in diverse neurodegenerative disorders
inferred← citeshttps://doi.org/10.1002/ana.410380219openalex-snapshot
Apolipoprotein E ϵ4 and cerebral hemorrhage associated with amyloid angiopathy
inferred← citeshttps://doi.org/10.1002/ana.410380318openalex-snapshot
Apolipoprotein E ϵ4 allele is not associated with earlier age at onset in amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1002/ana.410380415openalex-snapshot
The CYP2D6B allele is associated with a milder synaptic pathology in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/ana.410380619openalex-snapshot
Apolipoprotein E ϵ4 in inclusion body myositis
inferred← citeshttps://doi.org/10.1002/ana.410390110openalex-snapshot
Clinical and pathological correlates of apolipoprotein E ε4 in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/ana.410390320openalex-snapshot
Aβ‐peptide length aid apolipoprotein E genotype in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/ana.410390521openalex-snapshot
High frequency of apolipoprotein E ε2 in patients with cerebral hemorrhage due to cerebral amyloid angiopathy
inferred← citeshttps://doi.org/10.1002/ana.410400212openalex-snapshot
Genetic studies in Alzheimer's disease with an NACP/α‐synuclein polymorphism
inferred← citeshttps://doi.org/10.1002/ana.410400604openalex-snapshot
Genetic factors in Alzheimer's disease: A review of recent advances
inferred← citeshttps://doi.org/10.1002/ana.410410317openalex-snapshot
Increased apolipoprotein E ϵ4 in epilepsy with senile plaques
inferred← citeshttps://doi.org/10.1002/ana.410410604openalex-snapshot
Cerebral amyloid angiopathy and apolipoprotein E: Bad news for the good allele?
inferred← citeshttps://doi.org/10.1002/ana.410410607openalex-snapshot
High frequency of apolipoprotein E ϵ2 Allele in hemorrhage due to cerebral amyloid angiopathy
inferred← citeshttps://doi.org/10.1002/anbr.202000040openalex-snapshot
The Membrane Axis of Alzheimer's Nanomedicine
inferred← citeshttps://doi.org/10.1002/ange.202210675openalex-snapshot
pH Dependence of Amyloid‐β Fibril Assembly Kinetics: Unravelling the Microscopic Molecular Processes
inferred← citeshttps://doi.org/10.1002/ange.202215785openalex-snapshot
Imaging Amyloid‐β Membrane Interactions: Ion‐Channel Pores and Lipid‐Bilayer Permeability in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/anie.202210675openalex-snapshot
pH Dependence of Amyloid‐β Fibril Assembly Kinetics: Unravelling the Microscopic Molecular Processes
inferred← citeshttps://doi.org/10.1002/anie.202215785openalex-snapshot
Imaging Amyloid‐β Membrane Interactions: Ion‐Channel Pores and Lipid‐Bilayer Permeability in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/cbic.202400224openalex-snapshot
Role of Amyloidogenic and Non‐Amyloidogenic Protein Spaces in Neurodegenerative Diseases and their Mitigation Using Theranostic Agents
inferred← citeshttps://doi.org/10.1002/cm.20388openalex-snapshot
Cytoskeletal pathologies of Alzheimer disease
inferred← citeshttps://doi.org/10.1002/cm.21282openalex-snapshot
Actin dynamics and cofilin‐actin rods in alzheimer disease
inferred← citeshttps://doi.org/10.1002/ddr.10063openalex-snapshot
Biological markers of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/dneu.22715openalex-snapshot
Plasma amyloid and tau as dementia biomarkers in Down syndrome: Systematic review and meta‐analyses
inferred← citeshttps://doi.org/10.1002/glia.22289openalex-snapshot
APOE genotype alters glial activation and loss of synaptic markers in mice
inferred← citeshttps://doi.org/10.1002/glia.24105openalex-snapshot
Microglia‐specific ApoE knock‐out does not alter Alzheimer's disease plaque pathogenesis or gene expression
inferred← citeshttps://doi.org/10.1002/gps.1550openalex-snapshot
Cerebral amyloid angiopathy and cortical microinfarcts as putative substrates of vascular dementia
inferred← citeshttps://doi.org/10.1002/gps.2628openalex-snapshot
Alzheimer's disease genetics: current knowledge and future challenges
inferred← citeshttps://doi.org/10.1002/hbm.23036openalex-snapshot
Altered memory-related functional connectivity of the anterior and posterior hippocampus in older adults at increased genetic risk for Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/hipo.22422openalex-snapshot
APOE ɛ4 constrains engagement of encoding-related compensatory networks in amnestic mild cognitive impairment
inferred← citeshttps://doi.org/10.1002/humu.20180openalex-snapshot
TNF polymorphisms in Alzheimer disease and functional implications on CSF beta-amyloid levels
inferred← citeshttps://doi.org/10.1002/humu.23872openalex-snapshot
A functional variant in the miR‐142 promoter modulating its expression and conferring risk of Alzheimer disease
inferred← citeshttps://doi.org/10.1002/jnr.20887openalex-snapshot
Chronic thrombin exposure results in an increase in apolipoprotein-E levels
inferred← citeshttps://doi.org/10.1002/jnr.22391openalex-snapshot
Diverse roles of extracellular calcium‐sensing receptor in the central nervous system
inferred← citeshttps://doi.org/10.1002/jnr.22615openalex-snapshot
Dissociation of β-amyloid from lipoprotein in cerebrospinal fluid from Alzheimer's disease accelerates β-amyloid-42 assembly
inferred← citeshttps://doi.org/10.1002/jnr.23341openalex-snapshot
Influence ofAPOEgenotype and the presence of Alzheimer's pathology on synaptic membrane lipids of human brains
inferred← citeshttps://doi.org/10.1002/mds.27198openalex-snapshot
Cognitive impairment in progressive supranuclear palsy is associated with tau burden
inferred← citeshttps://doi.org/10.1002/med.21440openalex-snapshot
Bridging Type 2 Diabetes and Alzheimer's Disease: Assembling the Puzzle Pieces in the Quest for the Molecules With Therapeutic and Preventive Potential
inferred← citeshttps://doi.org/10.1002/path.1320openalex-snapshot
Cholesterol storage and tau pathology in Niemann–Pick type C disease in the brain
inferred← citeshttps://doi.org/10.1002/yd.2330247605openalex-snapshot
Early diagnosis of alzheimer's disease
inferred← citeshttps://doi.org/10.1006/bbrc.1997.7727openalex-snapshot
Detection of Apolipoprotein E/Dimeric Soluble Amyloid β Complexes in Alzheimer's Disease Brain Supernatants
inferred← citeshttps://doi.org/10.1006/bbrc.1998.9652openalex-snapshot
Elevated Low-Density Lipoprotein in Alzheimer's Disease Correlates with Brain Aβ 1–42 Levels
inferred← citeshttps://doi.org/10.1006/exnr.1997.6420openalex-snapshot
Possible Mechanism for Resistance to Alzheimer's Disease (AD) in Mice Suggests a New Approach to Generate a Mouse Model for Sporadic AD and May Explain Familial Resistance to AD in Man
inferred← citeshttps://doi.org/10.1006/exnr.1997.6710openalex-snapshot
Structure and Functions of Human Cerebrospinal Fluid Lipoproteins from Individuals of Different APOE Genotypes
inferred← citeshttps://doi.org/10.1006/exnr.1998.6852openalex-snapshot
Multivariate Analysis of Laminar Patterns of Neurodegeneration in Posterior Cingulate Cortex in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1006/exnr.1998.6977openalex-snapshot
Myeloperoxidase Polymorphism Is Associated with Gender Specific Risk for Alzheimer's Disease
inferred← citeshttps://doi.org/10.1006/geno.1998.5306openalex-snapshot
A Genome Survey for Novel Alzheimer Disease Risk Loci: Results at 10-cM Resolution
inferred← citeshttps://doi.org/10.1006/geno.1999.6057openalex-snapshot
Analysis of Association at Single Nucleotide Polymorphisms in the APOE Region
inferred← citeshttps://doi.org/10.1006/nbdi.1997.0131openalex-snapshot
Apolipoprotein E Uptake and Low-Density Lipoprotein Receptor-Related Protein Expression by the NTera2/D1 Cell Line: A Cell Culture Model of Relevance for Late-Onset Alzheimer's Disease
inferred← citeshttps://doi.org/10.1006/nbdi.1997.0147openalex-snapshot
Biology of Aβ Amyloid in Alzheimer's Disease
inferred← citeshttps://doi.org/10.1006/nbdi.1997.0155openalex-snapshot
ApoE Synthesis in Human Neuroblastoma Cells
inferred← citeshttps://doi.org/10.1006/nbdi.1999.0273openalex-snapshot
Oxidative Insults Are Associated with Apolipoprotein E Genotype in Alzheimer's Disease Brain
inferred← citeshttps://doi.org/10.1006/nbdi.2002.0483openalex-snapshot
Human and Murine ApoE Markedly Alters Aβ Metabolism before and after Plaque Formation in a Mouse Model of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1007/0-306-47377-1_30openalex-snapshot
Cognitive Approaches to the Memory Disorders of Demented Patients
inferred← citeshttps://doi.org/10.1007/0-306-48529-x_15openalex-snapshot
Pathogenesis of Alzheimer Disease: Metabolic Factors
inferred← citeshttps://doi.org/10.1007/0-387-22621-4_76openalex-snapshot
Neurobiologic Basis of Age-Related Dementing Disorders
inferred← citeshttps://doi.org/10.1007/0-387-23226-5_13openalex-snapshot
The Interaction of Amyloid-β with ApoE
inferred← citeshttps://doi.org/10.1007/0-387-25919-8_6openalex-snapshot
Apolipoproteins in Different Amyloidoses
inferred← citeshttps://doi.org/10.1007/978-0-387-35135-3_11openalex-snapshot
Role of A β Transport and Clearance in the Pathogenesis and Treatment of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-0-387-71522-3_13openalex-snapshot
Apolipoprotein E: A Potent Gene-Based Therapeutic Target for the Treatment of Sporadic Alzheimer's Disease
inferred← citeshttps://doi.org/10.1007/978-1-4612-4116-4_73openalex-snapshot
Apoe Genotype and MRI volumetry: Implication for Therapy
inferred← citeshttps://doi.org/10.1007/978-1-4612-4116-4_8openalex-snapshot
Isoform-Specific Metabolism of Apolipoprotein E: Implications for Alzheimer ’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4613-0513-2_3openalex-snapshot
11. Molecular genetics of Alzheimer's disease
inferred← citeshttps://doi.org/10.1007/978-1-4615-1721-4_15openalex-snapshot
Role of Genetic Background: Influence of Apolipoprotein E Genotype in Alzheimer’s Disease and After Head Injury
inferred← citeshttps://doi.org/10.1007/978-1-4615-4885-0_15openalex-snapshot
Neuropathological Correlates of Dementia in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4615-4885-0_16openalex-snapshot
Multifocal Cortical Neurodegeneration in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4615-4887-4_10openalex-snapshot
Model of Genetic Susceptibility to Late-Onset Alzheimer’s Disease: Mice Transgenic for Human Apolipoprotein E Alleles
inferred← citeshttps://doi.org/10.1007/978-1-4615-4887-4_9openalex-snapshot
Alzheimer%#x2019;s Disease and Genetically Engineered Animal Models
inferred← citeshttps://doi.org/10.1007/978-1-4615-5337-3_19openalex-snapshot
Regulation of APP Metabolism by Protein Phosphorylation
inferred← citeshttps://doi.org/10.1007/978-1-4615-5337-3_27openalex-snapshot
Amyloid-β Hypothesis of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4615-5337-3_3openalex-snapshot
Apoe and Memory in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4615-6001-2_2openalex-snapshot
Pathological Changes in the Elderly Human Brain
inferred← citeshttps://doi.org/10.1007/978-1-4615-6001-2_3openalex-snapshot
Pathological Changes in Neurodegenerative Disease
inferred← citeshttps://doi.org/10.1007/978-1-4615-6001-2_4openalex-snapshot
Etiological Considerations
inferred← citeshttps://doi.org/10.1007/978-1-4615-8149-9_11openalex-snapshot
Apolipoprotein E and Alzheimer’s Disease: Therapeutic Implications
inferred← citeshttps://doi.org/10.1007/978-1-4615-8149-9_12openalex-snapshot
Apolipoprotein E4 and Cholinergic Dysfunction in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4615-8149-9_80openalex-snapshot
Therapeutic Strategies in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4899-0209-2_14openalex-snapshot
Differential Regulation of APP Secretion by Apolipoprotein E3 and E4
inferred← citeshttps://doi.org/10.1007/978-1-4899-0209-2_15openalex-snapshot
Apolipoprotein E Uptake is Increased by Beta-Amyloid Peptides and Reduced by Blockade of the LDL Receptor
inferred← citeshttps://doi.org/10.1007/978-1-4899-1857-4_10openalex-snapshot
The Neuropsychology of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-4939-2550-6_33openalex-snapshot
Amyloid-Binding Proteins: Affinity-Based Separation, Proteomic Identification, and Optical Biosensor Validation
inferred← citeshttps://doi.org/10.1007/978-1-4939-3777-6_7openalex-snapshot
Genetic Models of Alzheimer’s Disease: The Influence of Apolipoprotein E allele Isoforms on Behaviour in Laboratory Animals
inferred← citeshttps://doi.org/10.1007/978-1-59259-005-6_4openalex-snapshot
The Pathophysiology of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-006-3_1openalex-snapshot
Aß Toxicity in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-471-9_2openalex-snapshot
Potential Biological Mechanisms of ApoE in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-471-9_4openalex-snapshot
Mechanism for β-amyloid Overproduction in Alzheimer Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-473-3_1openalex-snapshot
Roles of CNS Macrophages in Neurodegeneration
inferred← citeshttps://doi.org/10.1007/978-1-59259-475-7_11openalex-snapshot
Signaling by β-Amyloid Precursor Protein
inferred← citeshttps://doi.org/10.1007/978-1-59259-661-4_34openalex-snapshot
Secretases as Potential Targets for Treatment of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-59259-963-9_118openalex-snapshot
Genetics and Neurobiology of Alzheimer’s Disease and Frontotemporal Dementias
inferred← citeshttps://doi.org/10.1007/978-1-84628-440-3_1openalex-snapshot
A Brief Introduction to the History of the β-Amyloid Protein (Aβ) of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-1-84628-440-3_2openalex-snapshot
The Aβcentric Pathway of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-22009-9_686openalex-snapshot
Apolipoprotein Epsilon 4
inferred← citeshttps://doi.org/10.1007/978-3-031-31570-1_20openalex-snapshot
Thirty Years After the Launch of the Amyloid Cascade Hypothesis: Is there a Shift of Gears at Last?
inferred← citeshttps://doi.org/10.1007/978-3-031-52641-1_29-1openalex-snapshot
APOE and Oxidative Stress
inferred← citeshttps://doi.org/10.1007/978-3-0348-8225-5_28openalex-snapshot
Amyloid β-protein accumulation in the human brain during aging
inferred← citeshttps://doi.org/10.1007/978-3-0348-8845-5_4openalex-snapshot
Abnormalities of protein kinases in neurodegenerative diseases
inferred← citeshttps://doi.org/10.1007/978-3-319-11251-0_41openalex-snapshot
Obesity, Metabolic Dysfunction, and Dementia
inferred← citeshttps://doi.org/10.1007/978-3-319-12125-3_41-1openalex-snapshot
Obesity, Metabolic Dysfunction and Dementia
inferred← citeshttps://doi.org/10.1007/978-3-319-17344-3_3openalex-snapshot
Lipids in Amyloid-β Processing, Aggregation, and Toxicity
inferred← citeshttps://doi.org/10.1007/978-3-319-69892-2_686-1openalex-snapshot
Apolipoprotein Epsilon 4
inferred← citeshttps://doi.org/10.1007/978-3-540-85383-1_8openalex-snapshot
Molecular Genetics of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1007/978-3-642-59371-0_5openalex-snapshot
Physiologisches und pathologisches Altern des Gehirns
inferred← citeshttps://doi.org/10.1007/978-3-642-59519-6_72openalex-snapshot
Molecular Genetics and Molecular Biology of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-642-60175-0_7openalex-snapshot
Molekulargenetik und Molekularbiologie der Alzheimer-Krankheit
inferred← citeshttps://doi.org/10.1007/978-3-642-60680-9_9openalex-snapshot
Molecular Mechanisms of Synaptic Disconnection in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-642-79423-0_4openalex-snapshot
Alzheimer’s Disease and Hemorrhagic Stroke: Their Relationship to βA4 Amyloid Deposition
inferred← citeshttps://doi.org/10.1007/978-3-642-79423-0_5openalex-snapshot
Combining In Vitro Cell Biology and In Vivo Mouse Modelling to Study the Mechanisms Underlying Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-642-80109-9_1openalex-snapshot
Apolipoprotein E and Alzheimer’s Disease: State of the Field After Two Years
inferred← citeshttps://doi.org/10.1007/978-3-642-80109-9_11openalex-snapshot
The βA4 Amyloid Peptide Enhances Cellular Uptake of Apolipoprotein E-Containing Lipoproteins by the Low-Density Lipoprotein Receptor-Related Protein/α 2-Macroglobulin Receptor
inferred← citeshttps://doi.org/10.1007/978-3-642-80109-9_12openalex-snapshot
Interactions of Apolipoprotein E with Microtubule-Associated Proteins
inferred← citeshttps://doi.org/10.1007/978-3-642-80109-9_3openalex-snapshot
Apolipoprotein E4, Cholinergic Integrity, Synaptic Plasticity and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-642-80109-9_4openalex-snapshot
Pattern of Apolipoprotein E Immunoreactivity During Brain Aging
inferred← citeshttps://doi.org/10.1007/978-3-642-80109-9_8openalex-snapshot
Apolipoprotein E and Apolipoprotein E Receptors in the Central Nervous System: Relationship with Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-662-03248-0_2openalex-snapshot
The Vascular Dementias and Cerebrovascular Involvement in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-662-04399-8_10openalex-snapshot
ApoE Receptors in the Brain: Novel Signaling Pathways with Potential Relevance for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-662-72210-7_20openalex-snapshot
Gibt es mehr als dreißig Jahre nach Einführung der Amyloid-Kaskaden-Hypothese endlich den Paradigmenwechsel?
inferred← citeshttps://doi.org/10.1007/978-3-7091-6467-9_18openalex-snapshot
Apolipoprotein E4, cholinergic integrity and the pharmacogenetics of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/978-3-7091-6889-9_130openalex-snapshot
Untersuchungen zum Apolipoprotein E-Genotyp an histologisch gesicherten Fällen von Morbus Alzheimer
inferred← citeshttps://doi.org/10.1007/978-3-7091-6889-9_4openalex-snapshot
„Nicht-Amyloid“-Hypothesen zur Ätiopathogenese der Demenz vom Alzheimer Typ (DAT)
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