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Senescence in premalignant tumours
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1016/s0092-8674(00)81902-9openalex-snapshot
Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4a
inferredcites →https://doi.org/10.1016/s0092-8674(03)00401-xopenalex-snapshot
Rb-Mediated Heterochromatin Formation and Silencing of E2F Target Genes during Cellular Senescence
inferredcites →https://doi.org/10.1016/s1535-6108(03)00191-0openalex-snapshot
Tumor induction by an endogenous K-ras oncogene is highly dependent on cellular context
inferredcites →https://doi.org/10.1016/s1535-6108(04)00085-6openalex-snapshot
Endogenous oncogenic K-rasG12D stimulates proliferation and widespread neoplastic and developmental defects
inferredcites →https://doi.org/10.1038/25870openalex-snapshot
p19ARF links the tumour suppressor p53 to Ras
inferredcites →https://doi.org/10.1073/pnas.92.20.9363openalex-snapshot
A biomarker that identifies senescent human cells in culture and in aging skin in vivo.
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4808568openalex-extracted
Constructing a Risk Scoring Model for Breast Cancer: Focusing on Mitochondrial Energy Metabolism and Cellular Senescence Gene Features
inferred← citeshttps://doi.org/10.1002/1878-0261.12807openalex-snapshot
Cellular senescence in cancer: from mechanisms to detection
inferred← citeshttps://doi.org/10.1002/1878-0261.13268openalex-snapshot
Cellular senescence and the tumour microenvironment
inferred← citeshttps://doi.org/10.1002/1878-0261.13269openalex-snapshot
Autophagy at the intersection of aging, senescence, and cancer
inferred← citeshttps://doi.org/10.1002/1878-0261.13312openalex-snapshot
Targeting senescence as an anticancer therapy
inferred← citeshttps://doi.org/10.1002/1878-0261.70039openalex-snapshot
Determination of ADP / ATP translocase isoform ratios in malignancy and cellular senescence
inferred← citeshttps://doi.org/10.1002/2211-5463.70134openalex-snapshot
Overview of molecular signatures of senescence and associated resources: pros and cons
inferred← citeshttps://doi.org/10.1002/9780470015902.a0025372openalex-snapshot
Cancer and the Ageing Process
inferred← citeshttps://doi.org/10.1002/9780470696330.ch5openalex-snapshot
Molecular Biology of Preneoplastic Lesions of the Lung
inferred← citeshttps://doi.org/10.1002/9781118300312.ch4openalex-snapshot
Regulation of Senescence by microRNAs
inferred← citeshttps://doi.org/10.1002/9783527627523.ch7openalex-snapshot
Retinoblastoma: The Prototypic Hereditary Tumor
inferred← citeshttps://doi.org/10.1002/aac2.70027openalex-snapshot
Chronobiology of Cancer: How Aging Fuels Oncogenesis at the Molecular Level
inferred← citeshttps://doi.org/10.1002/adtp.202100149openalex-snapshot
Senolysis Reduces Senescence in Veins and Cancer Cell Migration
inferred← citeshttps://doi.org/10.1002/bies.201000055openalex-snapshot
Replication stress, a source of epigenetic aberrations in cancer?
inferred← citeshttps://doi.org/10.1002/bies.20606openalex-snapshot
The evolving concept of tumor microenvironments
inferred← citeshttps://doi.org/10.1002/cbic.202200364openalex-snapshot
Recent Advances in Strategies for Imaging Detection and Intervention of Cellular Senescence
inferred← citeshttps://doi.org/10.1002/cnr2.70682openalex-snapshot
A Review of Senescence‐Associated Secretory Phenotype‐Mediated Remodeling of the Tumor Microenvironment: Implications for Cancer Progression and Therapy
inferred← citeshttps://doi.org/10.1002/cpcy.16openalex-snapshot
Quantitative Analysis of Cellular Senescence in Culture and In Vivo
inferred← citeshttps://doi.org/10.1002/emmm.201000099openalex-snapshot
V600EBraf induces gastrointestinal crypt senescence and promotes tumour progression through enhanced CpG methylation of p16INK4a
inferred← citeshttps://doi.org/10.1002/emmm.201000101openalex-snapshot
p21 loss blocks senescence following Apc loss and provokes tumourigenesis in the renal but not the intestinal epithelium
inferred← citeshttps://doi.org/10.1002/gcc.20846openalex-snapshot
Expression status of the autophagy‐regulatory gene ATG6/BECN1 in ERBB2‐positive breast carcinomas: Bypassing ERBB2‐induced oncogenic senescence to regulate the efficacy of ERBB2‐targeted therapies
inferred← citeshttps://doi.org/10.1002/hep.21552openalex-snapshot
Telomere shortening and inactivation of cell cycle checkpoints characterize human hepatocarcinogenesis†
inferred← citeshttps://doi.org/10.1002/hep.23072openalex-snapshot
Large liver cell change in hepatitis B virus–related liver cirrhosis #
inferred← citeshttps://doi.org/10.1002/hep.28411openalex-snapshot
Aspartate β‐hydroxylase modulates cellular senescence through glycogen synthase kinase 3β in hepatocellular carcinoma
inferred← citeshttps://doi.org/10.1002/ijc.23222openalex-snapshot
Evidence that senescent human prostate epithelial cells enhance tumorigenicity: Cell fusion as a potential mechanism and inhibition by p16INK4a and hTERT
inferred← citeshttps://doi.org/10.1002/ijc.24049openalex-snapshot
p53 dependent cell‐cycle arrest triggered by chemotherapy in xenografted breast tumors
inferred← citeshttps://doi.org/10.1002/ijc.29130openalex-snapshot
p16INK4a/Ki‐67 co‐expression specifically identifies transformed cells in the head and neck region
inferred← citeshttps://doi.org/10.1002/ijc.30833openalex-snapshot
The role of cold‐inducible RNA binding protein in cell stress response
inferred← citeshttps://doi.org/10.1002/ijc.32900openalex-snapshot
Cdc6 as a novel target in cancer: Oncogenic potential, senescence and subcellular localisation
inferred← citeshttps://doi.org/10.1002/ijc.35456openalex-snapshot
Decoding the crossroads of aging and cancer through single‐cell analysis: Implications for precision oncology
inferred← citeshttps://doi.org/10.1002/jcb.25898openalex-snapshot
Balancing Between Aging and Cancer: Molecular Genetics Meets Traditional Chinese Medicine
inferred← citeshttps://doi.org/10.1002/jcb.26443openalex-snapshot
RUNX‐mediated growth arrest and senescence are attenuated by diverse mechanisms in cells expressing RUNX1 fusion oncoproteins
inferred← citeshttps://doi.org/10.1002/jcp.20919openalex-snapshot
Molecular signaling and genetic pathways of senescence: Its role in tumorigenesis and aging
inferred← citeshttps://doi.org/10.1002/jcp.21337openalex-snapshot
Suppression of human tumor cell proliferation by Smurf2‐induced senescence
inferred← citeshttps://doi.org/10.1002/jcp.22786openalex-snapshot
The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence
inferred← citeshttps://doi.org/10.1002/jcp.24420openalex-snapshot
Autophagy and senescence in cancer therapy
inferred← citeshttps://doi.org/10.1002/jcp.24521openalex-snapshot
MEK1/2 Overactivation Can Promote Growth Arrest by Mediating ERK1/2-Dependent Phosphorylation of p70S6K
inferred← citeshttps://doi.org/10.1002/jcp.31327openalex-snapshot
SATB1, senescence and senescence‐related diseases
inferred← citeshttps://doi.org/10.1002/jsfa.12088openalex-snapshot
An in vitro study: prebiotic effects of edible palm hearts in batch human fecal fermentation system
inferred← citeshttps://doi.org/10.1002/med.20246openalex-snapshot
Stem cell MicroRNAs in senescence and immortalization: novel players in cancer therapy
inferred← citeshttps://doi.org/10.1002/path.2165openalex-snapshot
Disproportionate representation of KRAS gene mutation in atypical adenomatous hyperplasia, but even distribution of EGFR gene mutation from preinvasive to invasive adenocarcinomas
inferred← citeshttps://doi.org/10.1002/path.2585openalex-snapshot
The epigenetic regulators Bmi1 and Ring1B are differentially regulated in pancreatitis and pancreatic ductal adenocarcinoma
inferred← citeshttps://doi.org/10.1002/path.2783openalex-snapshot
Regulation of tissue‐ and stimulus‐specific cell fate decisions by p53 in vivo
inferred← citeshttps://doi.org/10.1002/path.5883openalex-snapshot
Clinical significance of pancreatic ductal metaplasia
inferred← citeshttps://doi.org/10.1002/pros.21201openalex-snapshot
1,25‐dihydroxyvitamin D3 and PI3K/AKT inhibitors synergistically inhibit growth and induce senescence in prostate cancer cells
inferred← citeshttps://doi.org/10.1002/wrna.1297openalex-snapshot
Noncoding RNA control of cellular senescence
inferred← citeshttps://doi.org/10.1007/978-0-387-69805-2_25openalex-snapshot
Oncogene Addiction: Mouse Models and Clinical Relevance for Molecularly Targeted Therapies
inferred← citeshttps://doi.org/10.1007/978-0-387-89072-2_4openalex-snapshot
Genetics of Retinoblastoma and Genetic Counseling
inferred← citeshttps://doi.org/10.1007/978-1-0716-2659-7_8openalex-snapshot
Induction and Characterization of Cellular Senescence in Salamanders
inferred← citeshttps://doi.org/10.1007/978-1-0716-4426-3_2openalex-snapshot
Current Methodologies to Assess Cellular Senescence in Cancer
inferred← citeshttps://doi.org/10.1007/978-1-0716-4426-3_8openalex-snapshot
Detecting Telomeric DNA Damage by Immuno-Telo FISH
inferred← citeshttps://doi.org/10.1007/978-1-4020-9063-9_66openalex-snapshot
Role of Zinc and Selenium in Oxidative Stress and Immunosenescence: Implications for Healthy Ageing and Longevity
inferred← citeshttps://doi.org/10.1007/978-1-4419-0639-7_6openalex-snapshot
Chromatin in Senescent Cells: A Conduit for the Anti-Aging Effects of Wnt Signaling?
inferred← citeshttps://doi.org/10.1007/978-1-4419-0639-7_8openalex-snapshot
Polycomb Group of Genes and the Epigenetics of Aging
inferred← citeshttps://doi.org/10.1007/978-1-4419-1075-2_3openalex-snapshot
Oncogene-Induced Senescence (OIS) as a Cellular Response to Oncogenic Stresses
inferred← citeshttps://doi.org/10.1007/978-1-4419-1075-2_7openalex-snapshot
A Comparison of Senescence in Mouse and Human Cells
inferred← citeshttps://doi.org/10.1007/978-1-4419-1770-6_9openalex-snapshot
The Senescence Secretome and Its Impact on Tumor Suppression and Cancer
inferred← citeshttps://doi.org/10.1007/978-1-4419-8231-5_2openalex-snapshot
Lessons on p53 from Mouse Models
inferred← citeshttps://doi.org/10.1007/978-1-4614-1445-2_6openalex-snapshot
Tumor Dormancy, Oncogene Addiction, Cellular Senescence, and Self-Renewal Programs
inferred← citeshttps://doi.org/10.1007/978-1-4614-3676-8_10openalex-snapshot
TP53 Germline Mutations: Genetics of Li–Fraumeni Syndrome
inferred← citeshttps://doi.org/10.1007/978-1-4614-6220-0_11openalex-snapshot
Hippo and Mouse Models for Cancer
inferred← citeshttps://doi.org/10.1007/978-1-4614-8039-6_15openalex-snapshot
Histone Demethylases in Prostate Cancer
inferred← citeshttps://doi.org/10.1007/978-1-4614-8815-6_12openalex-snapshot
p53 in Head and Neck Cancer
inferred← citeshttps://doi.org/10.1007/978-1-4939-6631-8_66-1openalex-snapshot
Animal Modeling of Pancreatitis-to-Cancer Progression
inferred← citeshttps://doi.org/10.1007/978-1-4939-6670-7_14openalex-snapshot
Detection of Senescent Cells by Extracellular Markers Using a Flow Cytometry-Based Approach
inferred← citeshttps://doi.org/10.1007/978-1-4939-6670-7_18openalex-snapshot
Detection of Oncogene-Induced Senescence In Vivo
inferred← citeshttps://doi.org/10.1007/978-1-4939-6670-7_19openalex-snapshot
Detection of Senescence Markers During Mammalian Embryonic Development
inferred← citeshttps://doi.org/10.1007/978-1-4939-6670-7_2openalex-snapshot
Senescence Phenotypes Induced by Ras in Primary Cells
inferred← citeshttps://doi.org/10.1007/978-1-4939-6670-7_5openalex-snapshot
Genome-Wide miRNA Screening for Genes Bypassing Oncogene-Induced Senescence
inferred← citeshttps://doi.org/10.1007/978-1-4939-7193-0_66openalex-snapshot
Animal Modeling of Pancreatitis-to-Cancer Progression
inferred← citeshttps://doi.org/10.1007/978-1-4939-8931-7_9openalex-snapshot
IMR90 ER:RAS: A Cell Model of Oncogene-Induced Senescence
inferred← citeshttps://doi.org/10.1007/978-1-59745-221-2_3openalex-snapshot
Evaluating the Importance of Apoptosis and Other Determinants of Cell Death and Survival
inferred← citeshttps://doi.org/10.1007/978-1-59745-458-2_10openalex-snapshot
Senescence, Apoptosis, and Cancer
inferred← citeshttps://doi.org/10.1007/978-1-59745-458-2_13openalex-snapshot
Simple Sequence Mutations
inferred← citeshttps://doi.org/10.1007/978-1-62703-239-1_1openalex-snapshot
Cell Senescence as Both a Dynamic and a Static Phenotype
inferred← citeshttps://doi.org/10.1007/978-1-62703-239-1_12openalex-snapshot
Detection of Senescence-Associated Heterochromatin Foci (SAHF)
inferred← citeshttps://doi.org/10.1007/978-1-62703-239-1_3openalex-snapshot
Senescence Regulation by the p53 Protein Family
inferred← citeshttps://doi.org/10.1007/978-1-62703-239-1_8openalex-snapshot
Colorimetric Detection of Senescence-Associated β Galactosidase
inferred← citeshttps://doi.org/10.1007/978-1-62703-429-6_8openalex-snapshot
Methods to Study MYC-Regulated Cellular Senescence
inferred← citeshttps://doi.org/10.1007/978-1-84882-644-1_5openalex-snapshot
The Role of Histone Demethylases in Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-37482-2_30-1openalex-snapshot
Pathology of Pancreatic Ductal Adenocarcinoma
inferred← citeshttps://doi.org/10.1007/978-3-030-41683-6_30openalex-snapshot
Pathology of Pancreatic Ductal Adenocarcinoma
inferred← citeshttps://doi.org/10.1007/978-3-030-44903-2_3openalex-snapshot
Interconnection Between Cellular Senescence, Regeneration and Ageing in Salamanders
inferred← citeshttps://doi.org/10.1007/978-3-030-44903-2_6openalex-snapshot
Targeted Senolytic Strategies Based on the Senescent Surfaceome
inferred← citeshttps://doi.org/10.1007/978-3-030-44903-2_7openalex-snapshot
Senolysis and Senostasis Through the Plasma Membrane
inferred← citeshttps://doi.org/10.1007/978-3-030-90475-3_3openalex-snapshot
Pathogenesis of Pituitary Adenomas
inferred← citeshttps://doi.org/10.1007/978-3-031-47375-3_15openalex-snapshot
The Role of the Smallest Molecule Hydrogen Overcoming Ageing-Related Disease
inferred← citeshttps://doi.org/10.1007/978-3-031-76469-1_2openalex-snapshot
Biomedical Research in Aging
inferred← citeshttps://doi.org/10.1007/978-3-319-06103-0_5openalex-snapshot
The Biology of Aging: Role in Cancer, Metabolic Dysfunction, and Health Disparities
inferred← citeshttps://doi.org/10.1007/978-3-319-12136-9_4openalex-snapshot
Mathematical Modeling for DNA Repair, Carcinogenesis and Cancer Detection
inferred← citeshttps://doi.org/10.1007/978-3-319-22330-8_5openalex-snapshot
Pathobiology of Hepatitis B Virus-Induced Carcinogenesis
inferred← citeshttps://doi.org/10.1007/978-3-319-34214-6_3openalex-snapshot
Molecular Mechanisms of Hepatocellular Carcinoma
inferred← citeshttps://doi.org/10.1007/978-3-319-44870-1_79-1openalex-snapshot
Cellular Senescence and Tumor Promotion
inferred← citeshttps://doi.org/10.1007/978-3-319-56065-6_8openalex-snapshot
Oral Cancer: Recent Developments and Future Challenges
inferred← citeshttps://doi.org/10.1007/978-3-319-57415-8_79openalex-snapshot
Cellular Senescence and Tumor Promotion
inferred← citeshttps://doi.org/10.1007/978-3-319-64597-1_136-1openalex-snapshot
T Cell Senescence and Tumor Immunotherapy
inferred← citeshttps://doi.org/10.1007/978-3-319-64597-1_66-1openalex-snapshot
Role of Zinc and Selenium in Oxidative Stress and Immunosenescence: Implications for Healthy Aging and Longevity
inferred← citeshttps://doi.org/10.1007/978-3-319-95387-8_3openalex-snapshot
Biomedical Research in Aging
inferred← citeshttps://doi.org/10.1007/978-3-319-99375-1_136openalex-snapshot
T Cell Senescence and Tumor Immunotherapy
inferred← citeshttps://doi.org/10.1007/978-3-319-99375-1_66openalex-snapshot
Role of Zinc and Selenium in Oxidative Stress and Immunosenescence: Implications for Healthy Aging and Longevity
inferred← citeshttps://doi.org/10.1007/978-90-481-2561-6_3openalex-snapshot
An Oncogene-Induced DNA Replication Stress Model for Cancer Development
inferred← citeshttps://doi.org/10.1007/978-94-007-5958-9_18openalex-snapshot
Senescence Arrest of Endopolyploid Cells Renders Senescence into One Mechanism for Positive Tumorigenesis
inferred← citeshttps://doi.org/10.1007/978-94-007-5958-9_24openalex-snapshot
Role of Senescence Induction in Cancer Therapy
inferred← citeshttps://doi.org/10.1007/978-94-007-7726-2_19openalex-snapshot
Premalignancy and Cellular Senescence
inferred← citeshttps://doi.org/10.1007/978-94-007-7726-2_27openalex-snapshot
Dormancy and Recurrence of Cancer Stem Cells in Bone: Role of Bone Morphogenetic Proteins
inferred← citeshttps://doi.org/10.1007/978-94-017-9421-3_7openalex-snapshot
Senescence in Oncogenesis: From Molecular Mechanisms to Therapeutic Opportunities
inferred← citeshttps://doi.org/10.1007/978-94-017-9421-3_8openalex-snapshot
Pro-senescence Therapy for Cancer: Time for the Clinic
inferred← citeshttps://doi.org/10.1007/978-981-13-2541-0_2openalex-snapshot
Understanding Cellular and Molecular Events of Skin Aging and Cancer: An Integrative Perspective
inferred← citeshttps://doi.org/10.1007/978-981-16-7977-3_4openalex-snapshot
Cellular Aging and Metabolites in Aging
inferred← citeshttps://doi.org/10.1007/978-981-96-1041-9_1openalex-snapshot
Introduction to Cellular Senescence and Aging
inferred← citeshttps://doi.org/10.1007/978-981-96-1041-9_6openalex-snapshot
Cellular Senescence and Stem Cell Exhaustion: Implications for Aging and Regeneration
inferred← citeshttps://doi.org/10.1007/978-981-99-0313-9_5openalex-snapshot
Hypoxia and Senescence: Role of Oxygen in Modulation of Tumor Suppression
inferred← citeshttps://doi.org/10.1007/s00018-009-0034-2openalex-snapshot
A comparative analysis of the cell biology of senescence and aging
inferred← citeshttps://doi.org/10.1007/s00018-013-1343-zopenalex-snapshot
Histone variants: emerging players in cancer biology
inferred← citeshttps://doi.org/10.1007/s00018-014-1691-3openalex-snapshot
Physiological and pathological consequences of cellular senescence
inferred← citeshttps://doi.org/10.1007/s00018-015-1840-3openalex-snapshot
Dysregulated glycolysis as an oncogenic event
inferred← citeshttps://doi.org/10.1007/s00018-017-2587-9openalex-snapshot
Epithelial cell senescence: an adaptive response to pre-carcinogenic stresses?
inferred← citeshttps://doi.org/10.1007/s00018-019-03261-8openalex-snapshot
Mechanisms and significance of therapy-induced and spontaneous senescence of cancer cells
inferred← citeshttps://doi.org/10.1007/s00018-020-03638-0openalex-snapshot
Induction and transmission of oncogene-induced senescence
inferred← citeshttps://doi.org/10.1007/s00018-022-04605-7openalex-snapshot
Anthracycline-induced cardiotoxicity and cell senescence: new therapeutic option?
inferred← citeshttps://doi.org/10.1007/s00109-011-0766-yopenalex-snapshot
Novel ARF/p53-independent senescence pathways in cancer repression
inferred← citeshttps://doi.org/10.1007/s00109-016-1401-8openalex-snapshot
Therapy-induced microenvironmental changes in cancer
inferred← citeshttps://doi.org/10.1007/s00204-024-03702-9openalex-snapshot
Cellular senescence gene TACC3 associated with colorectal cancer risk via genetic and DNA methylated alteration
inferred← citeshttps://doi.org/10.1007/s00403-016-1713-0openalex-snapshot
Senescence in the lesional fibroblasts of non-segmental vitiligo patients
inferred← citeshttps://doi.org/10.1007/s00412-007-0115-7openalex-snapshot
Cellular senescence and chromatin structure
inferred← citeshttps://doi.org/10.1007/s00412-009-0238-0openalex-snapshot
pRb, a local chromatin organizer with global possibilities
inferred← citeshttps://doi.org/10.1007/s00412-014-0469-6openalex-snapshot
Chromatin maintenance and dynamics in senescence: a spotlight on SAHF formation and the epigenome of senescent cells
inferred← citeshttps://doi.org/10.1007/s00432-023-04953-2openalex-snapshot
miR-656-3p inhibits melanomas in vitro and in vivo by inducing senescence via inhibiting LMNB2
inferred← citeshttps://doi.org/10.1007/s00709-013-0515-xopenalex-snapshot
Do we age because we have mitochondria?
inferred← citeshttps://doi.org/10.1007/s10330-007-0035-2openalex-snapshot
Pancreatic cancer — Molecular alterations
inferred← citeshttps://doi.org/10.1007/s10353-009-0496-3openalex-snapshot
Molecular biology, models, and histopathology of chronic pancreatitis and pancreatic cancer
inferred← citeshttps://doi.org/10.1007/s10495-009-0316-zopenalex-snapshot
Hallmarks for senescence in carcinogenesis: novel signaling players
inferred← citeshttps://doi.org/10.1007/s10522-008-9134-xopenalex-snapshot
Premature senescence of human endothelial cells induced by inhibition of glutaminase
inferred← citeshttps://doi.org/10.1007/s10522-013-9473-0openalex-snapshot
Immunosurveillance of senescent cells: the bright side of the senescence program
inferred← citeshttps://doi.org/10.1007/s10522-014-9529-9openalex-snapshot
Senescent cells: SASPected drivers of age-related pathologies
inferred← citeshttps://doi.org/10.1007/s10522-015-9593-9openalex-snapshot
Premature aging/senescence in cancer cells facing therapy: good or bad?
inferred← citeshttps://doi.org/10.1007/s10522-022-09972-zopenalex-snapshot
Tackling cellular senescence by targeting miRNAs
inferred← citeshttps://doi.org/10.1007/s10522-026-10449-6openalex-snapshot
Senescent cells in systemic aging: SASP heterogeneity, immune escape, and endocrine modulation
inferred← citeshttps://doi.org/10.1007/s10528-015-9709-6openalex-snapshot
Molecular Mechanisms Underlying Pituitary Pathogenesis
inferred← citeshttps://doi.org/10.1007/s10555-006-8161-9openalex-snapshot
A role for substance P in cancer promotion and progression: a mechanism to counteract intracellular death signals following oncogene activation or DNA damage
inferred← citeshttps://doi.org/10.1007/s10555-010-9209-4openalex-snapshot
Biological and clinical significance of KRAS mutations in lung cancer: an oncogenic driver that contrasts with EGFR mutation
inferred← citeshttps://doi.org/10.1007/s10555-010-9220-9openalex-snapshot
Senescent cells as a source of inflammatory factors for tumor progression
inferred← citeshttps://doi.org/10.1007/s10555-011-9344-6openalex-snapshot
EMT in carcinoma progression and dissemination: Facts, unanswered questions, and clinical considerations
inferred← citeshttps://doi.org/10.1007/s10555-012-9408-2openalex-snapshot
Challenges and advances in mouse modeling for human pancreatic tumorigenesis and metastasis
inferred← citeshttps://doi.org/10.1007/s10555-017-9667-zopenalex-snapshot
Cytokine-induced senescence for cancer surveillance
inferred← citeshttps://doi.org/10.1007/s10555-020-09875-wopenalex-snapshot
Caveolin-1, a master regulator of cellular senescence
inferred← citeshttps://doi.org/10.1007/s10555-022-10073-zopenalex-snapshot
Dormant cancer cells: programmed quiescence, senescence, or both?
inferred← citeshttps://doi.org/10.1007/s10555-023-10082-6openalex-snapshot
Insights into the role of senescence in tumor dormancy: mechanisms and applications
inferred← citeshttps://doi.org/10.1007/s10555-025-10302-1openalex-snapshot
The intricate interplay between circadian rhythm, androgen signaling, hormone therapy, and cellular senescence in prostate cancer
inferred← citeshttps://doi.org/10.1007/s10637-017-0493-5openalex-snapshot
Induction of accelerated senescence by the microtubule-stabilizing agent peloruside A
inferred← citeshttps://doi.org/10.1007/s10911-011-9221-5openalex-snapshot
Oncogene-Induced Senescence and its Role in Tumor Suppression
inferred← citeshttps://doi.org/10.1007/s11010-020-03996-0openalex-snapshot
Roles of ARF tumour suppressor protein in lung cancer: time to hit the nail on the head!
inferred← citeshttps://doi.org/10.1007/s11060-023-04387-3openalex-snapshot
Cellular senescence in glioma
inferred← citeshttps://doi.org/10.1007/s11302-024-10036-9openalex-snapshot
P2Y11 receptor is a critical regulator of extracellular ATP-mediated premature senescence in lung fibroblasts: Implications of ER-Ca+2 release/mitochondrial ROS production signaling pathway
inferred← citeshttps://doi.org/10.1007/s11357-012-9452-4openalex-snapshot
A role for c-Abl in cell senescence and spontaneous immortalization
inferred← citeshttps://doi.org/10.1007/s11357-014-9637-0openalex-snapshot
A comparison of oncogene-induced senescence and replicative senescence: implications for tumor suppression and aging
inferred← citeshttps://doi.org/10.1007/s11357-015-9764-2openalex-snapshot
Cellular senescence: from growth arrest to immunogenic conversion
inferred← citeshttps://doi.org/10.1007/s11515-014-1299-xopenalex-snapshot
Growth arrest signaling of the Raf/MEK/ERK pathway in cancer
inferred← citeshttps://doi.org/10.1007/s11596-012-0057-zopenalex-snapshot
Lower phosphorylation of p38 MAPK blocks the oxidative stress-induced senescence in myeloid leukemic CD34+CD38− cells
inferred← citeshttps://doi.org/10.1007/s11596-015-1457-7openalex-snapshot
Resveratrol-downregulated phosphorylated liver kinase B1 is involved in senescence of acute myeloid leukemia stem cells
inferred← citeshttps://doi.org/10.1007/s11684-014-0327-1openalex-snapshot
Reactive oxygen species generation is essential for cisplatininduced accelerated senescence in hepatocellular carcinoma
inferred← citeshttps://doi.org/10.1007/s12013-007-0003-zopenalex-snapshot
Standardizing cancer biomarkers criteria: data elements as a foundation for a database. Inflammatory mediator/M-CSF as model marker
inferred← citeshttps://doi.org/10.1007/s12013-008-9006-7openalex-snapshot
MRGing Chromatin Dynamics and Cellular Senescence
inferred← citeshttps://doi.org/10.1007/s12026-009-8128-3openalex-snapshot
MHC class II regulation by epigenetic agents and microRNAs
inferred← citeshttps://doi.org/10.1007/s12094-007-0042-3openalex-snapshot
Genomic instability: on the birth and death of cancer
inferred← citeshttps://doi.org/10.1007/s12672-020-00391-5openalex-snapshot
Interleukin-23 Represses the Level of Cell Senescence Induced by the Androgen Receptor Antagonists Enzalutamide and Darolutamide in Castration-Resistant Prostate Cancer Cells
inferred← citeshttps://doi.org/10.1007/s12672-025-03078-xopenalex-snapshot
Unveiling the therapeutic potential of senescence-related IQGAP2 in pancreatic Cancer through post-GWAS genomic and scRNA-seq analyses
inferred← citeshttps://doi.org/10.1007/s13277-014-1639-0openalex-snapshot
Brain tumor senescence might be mediated by downregulation of S-phase kinase-associated protein 2 via butylidenephthalide leading to decreased cell viability
inferred← citeshttps://doi.org/10.1007/s13402-017-0341-9openalex-snapshot
A step-by-step microRNA guide to cancer development and metastasis
inferred← citeshttps://doi.org/10.1007/s13402-025-01126-wopenalex-snapshot
Elimination of docetaxel-induced senescence attenuates malignant progression in RB1-deficient CRPC
inferred← citeshttps://doi.org/10.1007/s13577-015-0110-xopenalex-snapshot
Cellular senescence: a hitchhiker’s guide
inferred← citeshttps://doi.org/10.1007/s44258-026-00093-7openalex-snapshot
Modulating senescence for therapeutic delivery
inferred← citeshttps://doi.org/10.1016/b978-0-12-374145-5.00271-0openalex-snapshot
Oncogenic Stress Responses
inferred← citeshttps://doi.org/10.1016/b978-0-12-394447-4.30066-9openalex-snapshot
Cellular Senescence
inferred← citeshttps://doi.org/10.1016/b978-0-12-397803-5.00017-4openalex-snapshot
Cellular Senescence and Premature Aging in Lung Diseases
inferred← citeshttps://doi.org/10.1016/b978-0-12-405943-6.00009-9openalex-snapshot
Stra13 and Sharp-1, the Non-Grouchy Regulators of Development and Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-802389-1.00016-2openalex-snapshot
Chromatin Signaling in Aging and Cellular Senescence
inferred← citeshttps://doi.org/10.1016/b978-0-12-803678-5.00045-xopenalex-snapshot
Cancer and Senescence
inferred← citeshttps://doi.org/10.1016/b978-0-12-814527-2.00007-xopenalex-snapshot
Cancer-targeted chemotherapy: Emerging role of the folate anchored dendrimer as drug delivery nanocarrier
inferred← citeshttps://doi.org/10.1016/b978-0-12-820472-6.00102-xopenalex-snapshot
E2F Transcription Factors in Cancer, More than the Cell Cycle
inferred← citeshttps://doi.org/10.1016/b978-0-12-821618-7.00219-4openalex-snapshot
Cellular Senescence
inferred← citeshttps://doi.org/10.1016/b978-0-12-822514-1.00001-8openalex-snapshot
Premalignant lesions and cellular senescence
inferred← citeshttps://doi.org/10.1016/b978-0-12-822514-1.00010-9openalex-snapshot
A framework for addressing senescent cell burden in the osteoarthritic knee
inferred← citeshttps://doi.org/10.1016/b978-0-12-822514-1.00014-6openalex-snapshot
Senescence as a therapeutic target
inferred← citeshttps://doi.org/10.1016/b978-0-12-822514-1.00016-xopenalex-snapshot
Cellular senescence
inferred← citeshttps://doi.org/10.1016/b978-0-323-26339-9.00010-xopenalex-snapshot
10.1016/b978-0-323-26339-9.00010-x
inferred← citeshttps://doi.org/10.1016/b978-0-443-19001-8.00018-0openalex-snapshot
Chromatin signaling in aging and cellular senescence
inferred← citeshttps://doi.org/10.1016/b978-012373960-5.00106-4openalex-snapshot
Cancer and Senescence
inferred← citeshttps://doi.org/10.1016/b978-1-4160-0016-7.50374-xopenalex-snapshot
Genetic Progression from Retina to Retinoblastoma
inferred← citeshttps://doi.org/10.1016/b978-1-4557-4066-6.00015-9openalex-snapshot
Cellular Senescence
inferred← citeshttps://doi.org/10.1016/bs.acr.2016.05.006openalex-snapshot
The Dual Role of Senescence in Pancreatic Ductal Adenocarcinoma
inferred← citeshttps://doi.org/10.1016/bs.acr.2021.01.002openalex-snapshot
Autophagy and senescence in cancer therapy
inferred← citeshttps://doi.org/10.1016/bs.acr.2021.01.004openalex-snapshot
The role of autophagy in escaping therapy-induced polyploidy/senescence
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