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Cross-disorder risk gene CACNA1C differentially modulates susceptibility to psychiatric disorders during development and adulthood
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Outgoing claims: the papers this work cites, builds on, or argues with.
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Aging and cognition: Facilitation of recent memory in aged nonhuman primates by nimodipine
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Nimodipine in the treatment of old age dementias
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Mood Disorder Susceptibility Gene CACNA1C Modifies Mood-Related Behaviors in Mice and Interacts with Sex to Influence Behavior in Mice and Diagnosis in Humans
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CaV1.2 Calcium Channel Dysfunction Causes a Multisystem Disorder Including Arrhythmia and Autism
inferredcites →https://doi.org/10.1016/j.celrep.2014.10.015openalex-snapshot
A Role for Noncoding Variation in Schizophrenia
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Trauma exposure and stress-related disorders in inner city primary care patients
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CACNA1C rs1006737 genotype and bipolar disorder: Focus on intermediate phenotypes and cardiovascular comorbidity
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Use of calcium antagonists and cognitive decline in old age
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Genetic Differences in the Immediate Transcriptome Response to Stress Predict Risk-Related Brain Function and Psychiatric Disorders
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inferredcites →https://doi.org/10.1016/j.pneurobio.2012.06.001openalex-snapshot
CACNA1C (Cav1.2) in the pathophysiology of psychiatric disease
inferredcites →https://doi.org/10.1016/j.psyneuen.2010.09.007openalex-snapshot
Pituitary glucocorticoid receptor deletion reduces vulnerability to chronic stress
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Fkbp52 heterozygosity alters behavioral, endocrine and neurogenetic parameters under basal and chronic stress conditions in mice
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Ca v 1.2 channels mediate persistent chronic stress-induced behavioral deficits that are associated with prefrontal cortex activation of the p25/Cdk5-glucocorticoid receptor pathway
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Effects of combined chronic nimodipine and acute metrifonate treatment on spatial and avoidance behavior
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Subregion- and Cell Type–Restricted Gene Knockout in Mouse Brain
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Two components of long-term potentiation induced by different patterns of afferent activation
inferredcites →https://doi.org/10.1038/mp.2008.133openalex-snapshot
Gene-wide analyses of genome-wide association data sets: evidence for multiple common risk alleles for schizophrenia and bipolar disorder and for overlap in genetic risk
inferredcites →https://doi.org/10.1038/mp.2009.107openalex-snapshot
Meta-analysis of genome-wide association data of bipolar disorder and major depressive disorder
inferredcites →https://doi.org/10.1038/mp.2009.142openalex-snapshot
Potential diagnostic markers for postpartum depression point out to altered immune signaling
inferredcites →https://doi.org/10.1038/mp.2009.49openalex-snapshot
The bipolar disorder risk allele at CACNA1C also confers risk of recurrent major depression and of schizophrenia
inferredcites →https://doi.org/10.1038/mp.2009.69openalex-snapshot
CACNA1C (rs1006737) is associated with schizophrenia
inferredcites →https://doi.org/10.1038/mp.2012.71openalex-snapshot
Forebrain elimination of cacna1c mediates anxiety-like behavior in mice
inferredcites →https://doi.org/10.1038/mp.2013.107openalex-snapshot
A rare mutation of CACNA1C in a patient with bipolar disorder, and decreased gene expression associated with a bipolar-associated common SNP of CACNA1C in brain
inferredcites →https://doi.org/10.1038/mp.2014.143openalex-snapshot
Functional implications of a psychiatric risk variant within CACNA1C in induced human neurons
inferredcites →https://doi.org/10.1038/mp.2016.87openalex-snapshot
Forebrain glutamatergic, but not GABAergic, neurons mediate anxiogenic effects of the glucocorticoid receptor
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The environment and schizophrenia
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Biological insights from 108 schizophrenia-associated genetic loci
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Collaborative genome-wide association analysis supports a role for ANK3 and CACNA1C in bipolar disorder
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inferredcites →https://doi.org/10.1038/ng.943openalex-snapshot
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inferredcites →https://doi.org/10.1038/nn.2504openalex-snapshot
Observational fear learning involves affective pain system and Cav1.2 Ca2+ channels in ACC
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Animal models of neuropsychiatric disorders
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Mouse model of Timothy syndrome recapitulates triad of autistic traits
inferredcites →https://doi.org/10.1073/pnas.1217750110openalex-snapshot
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inferredcites →https://doi.org/10.1074/jbc.m006467200openalex-snapshot
Functional Embryonic Cardiomyocytes after Disruption of the L-type α1C (Ca 1.2) Calcium Channel Gene in the Mouse
inferredcites →https://doi.org/10.1080/19336950.2015.1048936openalex-snapshot
L-type calcium channels as drug targets in CNS disorders
inferredcites →https://doi.org/10.1093/ijnp/pyw112openalex-snapshot
Decreased Nucleus Accumbens Expression of Psychiatric Disorder Risk Gene Cacna1c Promotes Susceptibility to Social Stress
inferredcites →https://doi.org/10.1093/schbul/7.1.12openalex-snapshot
Recent Stressful Life Events and Episodes of Schizophrenia
inferredcites →https://doi.org/10.1093/schbul/sbj033openalex-snapshot
Genes for Schizophrenia and Bipolar Disorder? Implications for Psychiatric Nosology
inferredcites →https://doi.org/10.1093/schbul/sbu016openalex-snapshot
Genetic Relationships Between Schizophrenia, Bipolar Disorder, and Schizoaffective Disorder
inferredcites →https://doi.org/10.1101/cshperspect.a003947openalex-snapshot
Voltage-Gated Calcium Channels
inferredcites →https://doi.org/10.1101/lm.773208openalex-snapshot
Conditional forebrain deletion of the L-type calcium channel CaV1.2 disrupts remote spatial memories in mice
inferredcites →https://doi.org/10.1101/lm.808608openalex-snapshot
Selective and protracted effect of nifedipine on fear memory extinction correlates with induced stress response
inferredcites →https://doi.org/10.1101/lm.886208openalex-snapshot
Role of L-type Ca2+channel isoforms in the extinction of conditioned fear
inferredcites →https://doi.org/10.1101/lm.893808openalex-snapshot
L-type voltage-gated calcium channels in conditioned fear: A genetic and pharmacological analysis
inferredcites →https://doi.org/10.1111/ejn.12952openalex-snapshot
Sex‐dependent modulation of age‐related cognitive decline by the L‐type calcium channel gene Cacna1c (Cav1.2)
inferredcites →https://doi.org/10.1111/j.1476-5381.2011.01318.xopenalex-snapshot
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inferredcites →https://doi.org/10.1124/mol.108.049981openalex-snapshot
Expression and 1,4-Dihydropyridine-Binding Properties of Brain L-Type Calcium Channel Isoforms
inferredcites →https://doi.org/10.1126/science.1067020openalex-snapshot
The Molecular Biology of Memory Storage: A Dialogue Between Genes and Synapses
inferredcites →https://doi.org/10.1126/science.1202107openalex-snapshot
Glutamatergic and Dopaminergic Neurons Mediate Anxiogenic and Anxiolytic Effects of CRHR1
inferredcites →https://doi.org/10.1152/jn.01148.2003openalex-snapshot
Enhanced Long-Term Potentiation During Aging Is Masked by Processes Involving Intracellular Calcium Stores
inferredcites →https://doi.org/10.1152/jn.1999.82.2.736openalex-snapshot
VDCCs and NMDARs Underlie Two Forms of LTP in CA1 Hippocampus In Vivo
inferredcites →https://doi.org/10.1186/1471-2202-8-63openalex-snapshot
Inducible gene inactivation in neurons of the adult mouse forebrain
inferredcites →https://doi.org/10.1523/eneuro.0006-16.2016openalex-snapshot
The Neuropsychiatric Disease-Associated Genecacna1cMediates Survival of Young Hippocampal Neurons
inferredcites →https://doi.org/10.1523/eneuro.0118-16.2016openalex-snapshot
Deletion of the Mouse Homolog ofCACNA1CDisrupts Discrete Forms of Hippocampal-Dependent Memory and Neurogenesis within the Dentate Gyrus
inferredcites →https://doi.org/10.1523/jneurosci.1531-05.2005openalex-snapshot
Role of Hippocampal Cav1.2 Ca2+Channels in NMDA Receptor-Independent Synaptic Plasticity and Spatial Memory
inferredcites →https://doi.org/10.1523/jneurosci.18-09-03171.1998openalex-snapshot
Reversal of Age-Related Alterations in Synaptic Plasticity by Blockade of L-Type Ca2+Channels
inferredcites →https://doi.org/10.1523/jneurosci.2255-10.2010openalex-snapshot
Molecular Switch from L-Type Cav1.3 to Cav1.2 Ca2+Channel Signaling Underlies Long-Term Psychostimulant-Induced Behavioral and Molecular Plasticity
inferredcites →https://doi.org/10.1523/jneurosci.2315-11.2011openalex-snapshot
Cav1.2 L-Type Ca2+Channels Mediate Cocaine-Induced GluA1 Trafficking in the Nucleus Accumbens, a Long-Term Adaptation Dependent on Ventral Tegmental Area Cav1.3 Channels
inferredcites →https://doi.org/10.1523/jneurosci.4164-08.2010openalex-snapshot
Homeostatic Switch in Hebbian Plasticity and Fear Learning after Sustained Loss of Cav1.2 Calcium Channels
inferredcites →https://doi.org/10.3389/fnbeh.2012.00087openalex-snapshot
Hippocampus-dependent place learning enables spatial flexibility in C57BL6/N mice
inferredcites →https://doi.org/10.3389/fncel.2013.00091openalex-snapshot
Activation of CRH receptor type 1 expressed on glutamatergic neurons increases excitability of CA1 pyramidal neurons by the modulation of voltage-gated ion channels
inferredcites →https://doi.org/10.3389/fnins.2014.00331openalex-snapshot
Neuroinformatic analyses of common and distinct genetic components associated with major neuropsychiatric disorders
inferredcites →https://doi.org/10.5665/sleep.4972openalex-snapshot
Cacna1c(Cav1.2) Modulates Electroencephalographic Rhythm and Rapid Eye Movement Sleep Recovery
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Incoming claims: where this work appears in other papers’ reference lists and arguments.
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inferred← citeshttps://doi.org/10.1038/s41386-021-01045-yopenalex-snapshot
Investigation of genetic loci shared between bipolar disorder and risk-taking propensity: potential implications for pharmacological interventions
inferred← citeshttps://doi.org/10.1038/s41398-018-0337-xopenalex-snapshot
Replicated associations of FADS1, MAD1L1, and a rare variant at 10q26.13 with bipolar disorder in Chinese population
inferred← citeshttps://doi.org/10.1038/s41398-019-0626-zopenalex-snapshot
1,25-Dihydroxyvitamin D modulates L-type voltage-gated calcium channels in a subset of neurons in the developing mouse prefrontal cortex
inferred← citeshttps://doi.org/10.1038/s41398-021-01683-2openalex-snapshot
Enhanced long-term potentiation and impaired learning in mice lacking alternative exon 33 of CaV1.2 calcium channel
inferred← citeshttps://doi.org/10.1038/s41398-024-03140-2openalex-snapshot
Cacna1c deficiency in forebrain glutamatergic neurons alters behavior and hippocampal plasticity in female mice
inferred← citeshttps://doi.org/10.1038/s41398-026-04038-xopenalex-snapshot
Cross-Species evidence for hippocampal CACNA1C as a therapeutic target for alcohol use disorder
inferred← citeshttps://doi.org/10.1038/s41419-018-0676-9openalex-snapshot
Psychiatric risk gene Cacna1c determines mitochondrial resilience against oxidative stress in neurons
inferred← citeshttps://doi.org/10.1038/s41431-020-00711-xopenalex-snapshot
Genetic modifiers in rare disorders: the case of fragile X syndrome
inferred← citeshttps://doi.org/10.1038/s41467-019-10704-0openalex-snapshot
Chromatin organization in the female mouse brain fluctuates across the oestrous cycle
inferred← citeshttps://doi.org/10.1038/s41582-019-0246-5openalex-snapshot
Early-life adversity and neurological disease: age-old questions and novel answers
inferred← citeshttps://doi.org/10.1038/s41593-018-0151-zopenalex-snapshot
Chronic CRH depletion from GABAergic, long-range projection neurons in the extended amygdala reduces dopamine release and increases anxiety
inferred← citeshttps://doi.org/10.1038/s41598-022-26504-4openalex-snapshot
Neuronal deletion of CaV1.2 is associated with sex-specific behavioral phenotypes in mice
inferred← citeshttps://doi.org/10.1038/s41598-026-48841-4openalex-snapshot
Cacna1c deficiency in parvalbumin-expressing neurons promotes anxiety and passive stress-coping behavior
inferred← citeshttps://doi.org/10.1038/s44319-025-00410-yopenalex-snapshot
miR-708-5p is elevated in bipolar patients and can induce mood disorder-associated behavior in mice
inferred← citeshttps://doi.org/10.1073/pnas.2108768119openalex-snapshot
Disrupted Cacna1c gene expression perturbs spontaneous Ca 2+ activity causing abnormal brain development and increased anxiety
inferred← citeshttps://doi.org/10.1073/pnas.2110094118openalex-snapshot
Regulation of neuronal excitation–transcription coupling by Kv2.1-induced clustering of somatic L-type Ca 2+ channels at ER-PM junctions
inferred← citeshttps://doi.org/10.1073/pnas.2415650122openalex-snapshot
Structure of a polymorphic repeat at the CACNA1C schizophrenia locus
inferred← citeshttps://doi.org/10.1073/pnas.2606900123openalex-snapshot
Ank3 loss in adult forebrain excitatory neurons disrupts behavior, neuronal activity, membrane proteome, and myelination
inferred← citeshttps://doi.org/10.1080/15592294.2021.1896983openalex-snapshot
Cell type-specific chromatin accessibility analysis in the mouse and human brain
inferred← citeshttps://doi.org/10.1080/19336950.2020.1807851openalex-snapshot
Loss of Cav1.2 channels impairs hippocampal theta burst stimulation-induced long-term potentiation
inferred← citeshttps://doi.org/10.1080/19336950.2023.2176984openalex-snapshot
L-type calcium channels and neuropsychiatric diseases: Insights into genetic risk variant-associated genomic regulation and impact on brain development
inferred← citeshttps://doi.org/10.1080/19336950.2025.2523788openalex-snapshot
Astrocytic abnormalities in brain-specific Cacna1c -deficient mice: Implications for BBB impairment in neuropsychiatric diseases associated with CACNA1C mutations
inferred← citeshttps://doi.org/10.1093/hmg/ddz235openalex-snapshot
Long-term environmental impact on object recognition, spatial memory, and reversal learning capabilities in Cacna1c haploinsufficient rats
inferred← citeshttps://doi.org/10.1093/schbul/sbad073openalex-snapshot
Pleiotropic Association of CACNA1C Variants With Neuropsychiatric Disorders
inferred← citeshttps://doi.org/10.1093/schbul/sby096openalex-snapshot
CACNA1C: Association With Psychiatric Disorders, Behavior, and Neurogenesis
inferred← citeshttps://doi.org/10.1097/jxx.0000000000000968openalex-snapshot
Pharmacogenetic testing may benefit people receiving low-dose lithium in clinical practice
inferred← citeshttps://doi.org/10.1097/ypg.0000000000000325openalex-snapshot
Mapping, clustering, and analysis of research in psychiatric genomics
inferred← citeshttps://doi.org/10.1101/2020.05.26.117903openalex-snapshot
Language impairment with a microduplication in 1q42.3q43
inferred← citeshttps://doi.org/10.1101/2021.06.09.447782openalex-snapshot
Bipolar-associated miR-499-5p controls neuroplasticity by downregulating the Ca v 1.2 L-type voltage gated calcium channel subunit CACNB2
inferred← citeshttps://doi.org/10.1101/2021.09.30.462600openalex-snapshot
Functional characterisation of single nucleotide variants of the psychiatric risk gene cacna1c in the zebrafish
inferred← citeshttps://doi.org/10.1101/2024.01.15.575709openalex-snapshot
Sex-specific mechanisms underlie long-term potentiation at hippocampus-nucleus accumbens synapses
inferred← citeshttps://doi.org/10.1101/2024.03.05.24303780openalex-snapshot
Structural characterization of a polymorphic repeat at the CACNA1C schizophrenia locus
inferred← citeshttps://doi.org/10.1101/2024.03.14.584977openalex-snapshot
Early life stress-induced miR-708-5p regulates mood disorder-associated behavioural phenotypes in mice and is a potential diagnostic biomarker for bipolar disorder
inferred← citeshttps://doi.org/10.1101/2025.02.09.637126openalex-snapshot
Calcium channel-coupled transcription factors facilitate direct nuclear signaling
inferred← citeshttps://doi.org/10.1101/2025.08.25.672202openalex-snapshot
Genetic and structural evidence links Ca 2+ dysregulation and ATP2B2 to neuropsychiatric illness
inferred← citeshttps://doi.org/10.1101/2025.09.15.676436openalex-snapshot
Human-specific tandem repeat in CACNA1C modulates responses to neuronal stimulation
inferred← citeshttps://doi.org/10.1101/744540openalex-snapshot
Robust Hi-C chromatin loop maps in human neurogenesis and brain tissues at high-resolution
inferred← citeshttps://doi.org/10.1111/gbb.12552openalex-snapshot
Sex‐dependent effects of Cacna1c haploinsufficiency on juvenile social play behavior and pro‐social 50‐kHz ultrasonic communication in rats
inferred← citeshttps://doi.org/10.1111/gbb.12620openalex-snapshot
The effects of tamoxifen on mouse behavior
inferred← citeshttps://doi.org/10.1111/gbb.70021openalex-snapshot
Reduced Cacna1c Expression Produces Anhedonic Reactions to Palatable Sucrose in Rats: No Interactions With Juvenile or Adult Stress
inferred← citeshttps://doi.org/10.1111/jnc.70229openalex-snapshot
Clozapine's Molecular Signature: Selective 5‐ HT2A Receptor Downregulation and Signalling in Rat Brain Cortical Regions
inferred← citeshttps://doi.org/10.1111/pcn.13494openalex-snapshot
Clinical manifestations of schizophrenia in four patients with variants in voltage‐gated calcium channel‐encoding genes: a case series
inferred← citeshttps://doi.org/10.1124/pharmrev.120.000245openalex-snapshot
L-Type Calcium Channel Blockers: A Potential Novel Therapeutic Approach to Drug Dependence
inferred← citeshttps://doi.org/10.1134/s0026893323020176openalex-snapshot
Changes in Expression of Genes Associated with Calcium Processes in the Hippocampus in Mice Exposed to Chronic Social Stress
inferred← citeshttps://doi.org/10.1155/2022/1886658openalex-snapshot
Genetic Polymorphisms in miR-137 and Its Target Genes, TCF4 and CACNA1C, Contribute to the Risk of Bipolar Disorder: A Preliminary Case-Control Study and Bioinformatics Analysis
inferred← citeshttps://doi.org/10.11606/t.5.2023.tde-22082023-162443openalex-snapshot
Influência de polimorfismos de genes funcionais (GAD1 rs1978340, CACNA1C rs100737 e BDNF rs6265) nos neurometabólitos cerebrais em pacientes com Transtorno Afetivo Bipolar e controles
inferred← citeshttps://doi.org/10.1177/00048674211009595openalex-snapshot
An independent, replicable, functional and significant risk variant block at intron 3 of CACNA1C for schizophrenia
inferred← citeshttps://doi.org/10.1186/s12014-024-09485-6openalex-snapshot
Mapping dynamic molecular changes in hippocampal subregions after traumatic brain injury through spatial proteomics
inferred← citeshttps://doi.org/10.1186/s12881-019-0916-6openalex-snapshot
Genetic polymorphisms associated with sleep-related phenotypes; relationships with individual nocturnal symptoms of insomnia in the HUNT study
inferred← citeshttps://doi.org/10.1186/s12883-019-1381-8openalex-snapshot
Identification of a novel mutation in the CACNA1C gene in a Chinese family with autosomal dominant cerebellar ataxia
inferred← citeshttps://doi.org/10.1186/s12916-023-03218-6openalex-snapshot
Genetic proxies for antihypertensive drugs and mental disorders: Mendelian randomization study in European and East Asian populations
inferred← citeshttps://doi.org/10.1186/s12993-022-00198-0openalex-snapshot
A family-based study of genetic and epigenetic effects across multiple neurocognitive, motor, social-cognitive and social-behavioral functions
inferred← citeshttps://doi.org/10.1186/s13023-021-01850-0openalex-snapshot
Calcium channelopathies and intellectual disability: a systematic review
inferred← citeshttps://doi.org/10.1186/s13073-018-0526-5openalex-snapshot
Endophenotypes in psychiatric disease: prospects and challenges
inferred← citeshttps://doi.org/10.1192/bjp.2019.157openalex-snapshot
Voltage-gated calcium channel blockers for psychiatric disorders: genomic reappraisal
inferred← citeshttps://doi.org/10.1192/bjp.2020.36openalex-snapshot
100 words on delusions – 100 words
inferred← citeshttps://doi.org/10.1192/j.eurpsy.2023.2408openalex-snapshot
Cortical morphological heterogeneity of schizophrenia and its relationship with glutamatergic receptor variations
inferred← citeshttps://doi.org/10.1242/dmm.034116openalex-snapshot
Cacna1c haploinsufficiency leads to pro-social 50-kHz ultrasonic communication deficits in rats
inferred← citeshttps://doi.org/10.1371/journal.pgen.1008488openalex-snapshot
An autism-causing calcium channel variant functions with selective autophagy to alter axon targeting and behavior
inferred← citeshttps://doi.org/10.1523/eneuro.0282-21.2022openalex-snapshot
Late-Onset Behavioral and Synaptic Consequences of L-Type Ca2+Channel Activation in the Basolateral Amygdala of Developing Rats
inferred← citeshttps://doi.org/10.1523/jneurosci.0100-24.2024openalex-snapshot
Sex-Specific Mechanisms Underlie Long-Term Potentiation at Hippocampus→Medium Spiny Neuron Synapses in the Medial Shell of the Nucleus Accumbens
inferred← citeshttps://doi.org/10.1523/jneurosci.2397-17.2017openalex-snapshot
Extinction of Contextual Cocaine Memories Requires Ca v 1.2 within D1R-Expressing Cells and Recruits Hippocampal Ca v 1.2-Dependent Signaling Mechanisms
inferred← citeshttps://doi.org/10.15252/embr.202154420openalex-snapshot
Bipolar‐associated miR‐499‐5p controls neuroplasticity by downregulating the Cav1.2 subunit CACNB2
inferred← citeshttps://doi.org/10.18632/aging.102473openalex-snapshot
Association of genes with phenotype in autism spectrum disorder
inferred← citeshttps://doi.org/10.2139/ssrn.5243800openalex-snapshot
Dysregulation of Mir-335-5p, Mir-30d-5p, and Mir-774-5p in Extracellular Vesicles from First-Episode Psychosis Patients: Implications for Biomarker Discovery
inferred← citeshttps://doi.org/10.2217/epi-2020-0034openalex-snapshot
CACNA1C Methylation: Association with Cortisol, Perceived Stress, rs1006737 and Childhood Trauma in Males
inferred← citeshttps://doi.org/10.25557/0031-2991.2020.04.106-117openalex-snapshot
The influence of calcium signaling on the development of autism spectrum disorders
inferred← citeshttps://doi.org/10.31234/osf.io/6mkx7openalex-snapshot
Dysmodulation of Synapse Formation and Pruning Hypothesis of Schizophrenia
inferred← citeshttps://doi.org/10.31857/s0026898423020192openalex-snapshot
Changes in the Expression of Genes, Associated with Calcium Processes, in the Hippocampus of Mice under the Influence of Chronic Social Defeat Stress
inferred← citeshttps://doi.org/10.3389/fcell.2026.1761785openalex-snapshot
Dysregulation of neurodevelopmental regulatory networks in Anorexia Nervosa: an integrated multi-layered omics analysis
inferred← citeshttps://doi.org/10.3389/fendo.2024.1411343openalex-snapshot
The causal relationship between antihypertensive drugs and depression: a Mendelian randomization study of drug targets
inferred← citeshttps://doi.org/10.3389/fncel.2019.00483openalex-snapshot
Interaction of the Psychiatric Risk Gene Cacna1c With Post-weaning Social Isolation or Environmental Enrichment Does Not Affect Brain Mitochondrial Bioenergetics in Rats
inferred← citeshttps://doi.org/10.3389/fncel.2026.1776165openalex-snapshot
Deletion of Cacna1c (CaV1.2) in D1-expressing cells elicits divergent sex-specific effects on aversive and spatial memories
inferred← citeshttps://doi.org/10.3389/fnins.2018.00982openalex-snapshot
Somato-Dendritic Regulation of Raphe Serotonin Neurons; A Key to Antidepressant Action
inferred← citeshttps://doi.org/10.3389/fnins.2019.00986openalex-snapshot
Deletion of CRH From GABAergic Forebrain Neurons Promotes Stress Resilience and Dampens Stress-Induced Changes in Neuronal Activity
inferred← citeshttps://doi.org/10.3389/fnmol.2019.00074openalex-snapshot
Sex and Estrous Cycle Effects on Anxiety- and Depression-Related Phenotypes in a Two-Hit Developmental Stress Model
inferred← citeshttps://doi.org/10.3389/fphar.2021.777607openalex-snapshot
Ion Channel Dysfunction and Neuroinflammation in Migraine and Depression
inferred← citeshttps://doi.org/10.3389/fpsyt.2019.00982openalex-snapshot
CACNA1C rs1006737, Threatening Life Events, and Gene–Environment Interaction Predict Major Depressive Disorder
inferred← citeshttps://doi.org/10.3389/fpsyt.2022.840120openalex-snapshot
Genetic Variants Associated With Resilience in Human and Animal Studies
inferred← citeshttps://doi.org/10.3389/fpsyt.2023.1075250openalex-snapshot
DNA methylation in cocaine use disorder–An epigenome-wide approach in the human prefrontal cortex
inferred← citeshttps://doi.org/10.3389/fpsyt.2023.1183696openalex-snapshot
Evidence for increased DNA damage repair in the postmortem brain of the high stress-response group of schizophrenia
inferred← citeshttps://doi.org/10.3389/fpsyt.2024.1414776openalex-snapshot
The genetic association between bipolar disorder and dementia: a qualitative review
inferred← citeshttps://doi.org/10.3390/biom13040695openalex-snapshot
Connecting Dots between Mitochondrial Dysfunction and Depression
inferred← citeshttps://doi.org/10.3390/biom16101459openalex-snapshot
Dual Role of CACNA1C/CaV1.2 in Cardiac and Neuropsychiatric Disease
inferred← citeshttps://doi.org/10.3390/cells10020209openalex-snapshot
Using iPSC Models to Understand the Role of Estrogen in Neuron–Glia Interactions in Schizophrenia and Bipolar Disorder
inferred← citeshttps://doi.org/10.3390/cells13141194openalex-snapshot
Human-Induced Pluripotent Stem Cell (iPSC)-Derived GABAergic Neuron Differentiation in Bipolar Disorder
inferred← citeshttps://doi.org/10.3390/genes12111773openalex-snapshot
Prenatal Adversity Alters the Epigenetic Profile of the Prefrontal Cortex: Sexually Dimorphic Effects of Prenatal Alcohol Exposure and Food-Related Stress
inferred← citeshttps://doi.org/10.3390/genes12121850openalex-snapshot
Gene-Environment Interactions in Schizophrenia: A Literature Review
inferred← citeshttps://doi.org/10.3390/ijms19010219openalex-snapshot
Mapping the Schizophrenia Genes by Neuroimaging: The Opportunities and the Challenges
inferred← citeshttps://doi.org/10.3390/ijms20143537openalex-snapshot
Genetic Associations between Voltage-Gated Calcium Channels and Psychiatric Disorders
inferred← citeshttps://doi.org/10.3390/ijms24129795openalex-snapshot
Calcium Handling Remodeling Underlies Impaired Sympathetic Stress Response in Ventricular Myocardium from Cacna1c Haploinsufficient Rats
inferred← citeshttps://doi.org/10.3390/ijms26125547openalex-snapshot
Reduced Gene Dosage of the Psychiatric Risk Gene Cacna1c Is Associated with Impairments in Hypothalamic–Pituitary–Adrenal Axis Activity in Rats
inferred← citeshttps://doi.org/10.35376/10324/39211openalex-snapshot
Biologically relevant subgroups within the schizophrenia syndrome
inferred← citeshttps://doi.org/10.6084/m9.figshare.14594112openalex-snapshot
Additional file 1 of Calcium channelopathies and intellectual disability: a systematic review
inferred← citeshttps://doi.org/10.6084/m9.figshare.14594115openalex-snapshot
Additional file 2 of Calcium channelopathies and intellectual disability: a systematic review
inferred← citeshttps://doi.org/10.6084/m9.figshare.14594118openalex-snapshot
Additional file 3 of Calcium channelopathies and intellectual disability: a systematic review
inferred← citeshttps://doi.org/10.6084/m9.figshare.24934427openalex-snapshot
Additional file 1 of Genetic proxies for antihypertensive drugs and mental disorders: Mendelian randomization study in European and East Asian populations
inferred← citeshttps://doi.org/10.6084/m9.figshare.26719560openalex-snapshot
Additional file 1 of Mapping dynamic molecular changes in hippocampal subregions after traumatic brain injury through spatial proteomics
inferred← citeshttps://doi.org/10.6084/m9.figshare.26719563openalex-snapshot
Additional file 2 of Mapping dynamic molecular changes in hippocampal subregions after traumatic brain injury through spatial proteomics
inferred← citeshttps://doi.org/10.70121/001c.139041openalex-snapshot
Genetic mutations of voltage-gated ion channels associated with channelopathies
inferred← citeshttps://doi.org/10.9758/cpn.2019.17.3.364openalex-snapshot
Genes Involved in Neurodevelopment, Neuroplasticity and Major Depression: No Association for CACNA1C, CHRNA7 and MAPK1
inferred← citeshttps://doi.org/10.9758/cpn.2020.18.2.231openalex-snapshot
ZNF804A Gene Variants Have a Cross-diagnostic Influence on Psychosis and Treatment Improvement in Mood Disorders
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