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Formation of NHEJ-derived reciprocal chromosomal translocations does not require Ku70
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/1521-4141(200203)32:3<701::aid-immu701>3.0.co;2-topenalex-snapshot
Different types of V(D)J recombination and end-joining defects in DNA double-strand break repair mutant mammalian cells
inferredcites →https://doi.org/10.1016/j.dnarep.2006.05.013openalex-snapshot
Roles of nonhomologous DNA end joining, V(D)J recombination, and class switch recombination in chromosomal translocations
inferredcites →https://doi.org/10.1016/j.dnarep.2006.05.015openalex-snapshot
Recombinase, chromosomal translocations and lymphoid neoplasia: Targeting mistakes and repair failures
inferredcites →https://doi.org/10.1016/s0092-8674(02)00675-xopenalex-snapshot
The Mechanism and Regulation of Chromosomal V(D)J Recombination
inferredcites →https://doi.org/10.1016/s0092-8674(02)00770-5openalex-snapshot
Unrepaired DNA Breaks in p53-Deficient Cells Lead to Oncogenic Gene Amplification Subsequent to Translocations
inferredcites →https://doi.org/10.1038/ncb1591openalex-snapshot
Positional stability of single double-strand breaks in mammalian cells
inferredcites →https://doi.org/10.1038/nrm1202openalex-snapshot
Mechanism and regulation of human non-homologous DNA end-joining
inferredcites →https://doi.org/10.1073/pnas.94.15.8076openalex-snapshot
Ku70-deficient embryonic stem cells have increased ionizing radiosensitivity, defective DNA end-binding activity, and inability to support V(D)J recombination
inferredcites →https://doi.org/10.1074/jbc.271.24.14405openalex-snapshot
Ku80-deficient Cells Exhibit Excess Degradation of Extrachromosomal DNA
inferredcites →https://doi.org/10.1084/jem.20020851openalex-snapshot
Evidence for Replicative Repair of DNA Double-Strand Breaks Leading to Oncogenic Translocation and Gene Amplification
inferredcites →https://doi.org/10.1182/blood-2005-06-2437openalex-snapshot
A model of oncogenic rearrangements: differences between chromosomal translocation mechanisms and simple double-strand break repair
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4685388openalex-extracted
Epidermal Growth Factor Rejuvenates Aged Hematopoietic Stem Cells
inferred← citeshttps://doi.org/10.1002/9780470015902.a0026853openalex-snapshot
Mechanisms of Chromosome Translocations in Cancer
inferred← citeshttps://doi.org/10.1002/gcc.23141openalex-snapshot
Modeling sarcoma relevant translocations using CRISPR‐Cas9 in human embryonic stem derived mesenchymal precursors
inferred← citeshttps://doi.org/10.1002/jctb.1900openalex-snapshot
Expression proteomics of olfactory ensheathing cells
inferred← citeshttps://doi.org/10.1007/978-1-4614-4741-2_2openalex-snapshot
DNA-PK, a Pharmacological Target in Cancer Chemotherapy and Radiotherapy?
inferred← citeshttps://doi.org/10.1007/978-3-319-14151-0_3openalex-snapshot
The Role of PARPs in DNA Strand Break Repair
inferred← citeshttps://doi.org/10.1007/978-94-017-9421-3_2openalex-snapshot
DNA Repair Dysregulation in Cancer: From Molecular Mechanisms to Synthetic Lethal Opportunities
inferred← citeshttps://doi.org/10.1007/978-981-13-0593-1_2openalex-snapshot
Induction of Chromosomal Translocations with CRISPR-Cas9 and Other Nucleases: Understanding the Repair Mechanisms That Give Rise to Translocations
inferred← citeshttps://doi.org/10.1007/s00018-009-0068-5openalex-snapshot
Faithful after break-up: suppression of chromosomal translocations
inferred← citeshttps://doi.org/10.1016/b978-0-12-397933-9.00018-7openalex-snapshot
Molecular Mechanisms of AID Function
inferred← citeshttps://doi.org/10.1016/b978-0-12-803309-8.00019-7openalex-snapshot
Repair of Double-Strand Breaks by Nonhomologous End Joining
inferred← citeshttps://doi.org/10.1016/b978-0-12-803582-5.00009-7openalex-snapshot
DNA double-strand repair by nonhomologous end joining and its clinical relevance
inferred← citeshttps://doi.org/10.1016/b978-0-12-803582-5.00011-5openalex-snapshot
Radiation DNA damage and use in cancer/therapeutics-translation of radiation modifiers
inferred← citeshttps://doi.org/10.1016/b978-0-323-85679-9.00019-2openalex-snapshot
Repair of double-strand breaks by nonhomologous end joining; Its components and their function
inferred← citeshttps://doi.org/10.1016/b978-0-323-95895-0.00001-5openalex-snapshot
Molecular Mechanism of Activation-Induced Cytidine Deaminase
inferred← citeshttps://doi.org/10.1016/b978-0-444-53632-7.00803-0openalex-snapshot
Molecular Radiation Biology
inferred← citeshttps://doi.org/10.1016/j.bbrc.2011.11.006openalex-snapshot
Mode of ATM-dependent suppression of chromosome translocation
inferred← citeshttps://doi.org/10.1016/j.cancergen.2014.05.008openalex-snapshot
Identifying molecular markers for the sensitive detection of residual atypical teratoid rhabdoid tumor cells
inferred← citeshttps://doi.org/10.1016/j.cancergencyto.2010.04.011openalex-snapshot
The transposase domain protein Metnase/SETMAR suppresses chromosomal translocations
inferred← citeshttps://doi.org/10.1016/j.canrad.2011.05.004openalex-snapshot
Réparation des cassures double-brin de l’ADN, un mécanisme peut en cacher un autre : la ligature d’extrémités non homologues alternative
inferred← citeshttps://doi.org/10.1016/j.cell.2010.03.016openalex-snapshot
Origin of Chromosomal Translocations in Lymphoid Cancer
inferred← citeshttps://doi.org/10.1016/j.celrep.2013.08.034openalex-snapshot
A Role for BLM in Double-Strand Break Repair Pathway Choice: Prevention of CtIP/Mre11-Mediated Alternative Nonhomologous End-Joining
inferred← citeshttps://doi.org/10.1016/j.dnarep.2009.04.007openalex-snapshot
Choreography of recombination proteins during the DNA damage response
inferred← citeshttps://doi.org/10.1016/j.dnarep.2011.03.001openalex-snapshot
Subtelomeric regions in mammalian cells are deficient in DNA double-strand break repair
inferred← citeshttps://doi.org/10.1016/j.dnarep.2011.09.002openalex-snapshot
PIF1 disruption or NBS1 hypomorphism does not affect chromosome healing or fusion resulting from double-strand breaks near telomeres in murine embryonic stem cells
inferred← citeshttps://doi.org/10.1016/j.dnarep.2013.10.005openalex-snapshot
The absence of Ku but not defects in classical non-homologous end-joining is required to trigger PARP1-dependent end-joining
inferred← citeshttps://doi.org/10.1016/j.dnarep.2014.02.007openalex-snapshot
Alternative end-joining pathway(s): Bricolage at DNA breaks
inferred← citeshttps://doi.org/10.1016/j.dnarep.2014.03.021openalex-snapshot
Protein ADP-ribosylation and the cellular response to DNA strand breaks
inferred← citeshttps://doi.org/10.1016/j.dnarep.2014.12.002openalex-snapshot
Development of a novel method to create double-strand break repair fingerprints using next-generation sequencing
inferred← citeshttps://doi.org/10.1016/j.isci.2024.110306openalex-snapshot
Epidermal growth factor augments the self-renewal capacity of aged hematopoietic stem cells
inferred← citeshttps://doi.org/10.1016/j.molcel.2008.05.029openalex-snapshot
A Biochemically Defined System for Coding Joint Formation in V(D)J Recombination
inferred← citeshttps://doi.org/10.1016/j.molcel.2014.08.002openalex-snapshot
Chromosomal Translocations in Human Cells Are Generated by Canonical Nonhomologous End-Joining
inferred← citeshttps://doi.org/10.1016/j.molcel.2016.06.020openalex-snapshot
Essential Roles for Polymerase θ-Mediated End Joining in the Repair of Chromosome Breaks
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2011.02.005openalex-snapshot
Induction and repair of DNA double strand breaks: The increasing spectrum of non-homologous end joining pathways
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2017.07.008openalex-snapshot
Paths from DNA damage and signaling to genome rearrangements via homologous recombination
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2015.07.001openalex-snapshot
Alternative end-joining repair pathways are the ultimate backup for abrogated classical non-homologous end-joining and homologous recombination repair: Implications for the formation of chromosome translocations
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2015.07.002openalex-snapshot
Marked contribution of alternative end-joining to chromosome-translocation-formation by stochastically induced DNA double-strand-breaks in G2-phase human cells
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2021.503372openalex-snapshot
Analysis of chromatid-break-repair detects a homologous recombination to non-homologous end-joining switch with increasing load of DNA double-strand breaks
inferred← citeshttps://doi.org/10.1016/j.mrrev.2012.06.002openalex-snapshot
Recognition, signaling, and repair of DNA double-strand breaks produced by ionizing radiation in mammalian cells: The molecular choreography
inferred← citeshttps://doi.org/10.1016/j.semcdb.2011.10.007openalex-snapshot
DNA double-strand break repair pathways, chromosomal rearrangements and cancer
inferred← citeshttps://doi.org/10.1016/j.stem.2012.10.012openalex-snapshot
Thrombopoietin-Increased DNA-PK-Dependent DNA Repair Limits Hematopoietic Stem and Progenitor Cell Mutagenesis in Response to DNA Damage
inferred← citeshttps://doi.org/10.1016/j.tig.2008.08.007openalex-snapshot
MMEJ repair of double-strand breaks (director’s cut): deleted sequences and alternative endings
inferred← citeshttps://doi.org/10.1016/j.tig.2019.06.001openalex-snapshot
Templated Insertions: A Smoking Gun for Polymerase Theta-Mediated End Joining
inferred← citeshttps://doi.org/10.1038/emboj.2010.142openalex-snapshot
The function of classical and alternative non‐homologous end‐joining pathways in the fusion of dysfunctional telomeres
inferred← citeshttps://doi.org/10.1038/embor.2009.173openalex-snapshot
Dealing with dangerous accidents: DNA double‐strand breaks take centre stage
inferred← citeshttps://doi.org/10.1038/leu.2010.174openalex-snapshot
Analysis of genomic breakpoints in p190 and p210 BCR–ABL indicate distinct mechanisms of formation
inferred← citeshttps://doi.org/10.1038/ncb2941openalex-snapshot
The biogenesis of chromosome translocations
inferred← citeshttps://doi.org/10.1038/ncomms15110openalex-snapshot
PARP3 is a promoter of chromosomal rearrangements and limits G4 DNA
inferred← citeshttps://doi.org/10.1038/nrg3729openalex-snapshot
Mechanisms underlying mutational signatures in human cancers
inferred← citeshttps://doi.org/10.1038/nrm2851openalex-snapshot
Mitotic homologous recombination maintains genomic stability and suppresses tumorigenesis
inferred← citeshttps://doi.org/10.1038/nrm3523openalex-snapshot
Charity begins at home: non-coding RNA functions in DNA repair
inferred← citeshttps://doi.org/10.1038/nsmb.1641openalex-snapshot
Role of Mre11 in chromosomal nonhomologous end joining in mammalian cells
inferred← citeshttps://doi.org/10.1038/nsmb.1773openalex-snapshot
Alternative end-joining is suppressed by the canonical NHEJ component Xrcc4–ligase IV during chromosomal translocation formation
inferred← citeshttps://doi.org/10.1038/nsmb.1940openalex-snapshot
An essential role for CtIP in chromosomal translocation formation through an alternative end-joining pathway
inferred← citeshttps://doi.org/10.1038/nsmb.1942openalex-snapshot
CtIP promotes microhomology-mediated alternative end joining during class-switch recombination
inferred← citeshttps://doi.org/10.1038/nsmb.2172openalex-snapshot
The E3 ubiquitin ligase Rnf8 stabilizes Tpp1 to promote telomere end protection
inferred← citeshttps://doi.org/10.1038/nsmb0410-393openalex-snapshot
NHEJ and its backup pathways in chromosomal translocations
inferred← citeshttps://doi.org/10.1038/onc.2012.491openalex-snapshot
The Forkhead Box M1 protein regulates BRIP1 expression and DNA damage repair in epirubicin treatment
inferred← citeshttps://doi.org/10.1038/onc.2013.457openalex-snapshot
FOXM1 targets NBS1 to regulate DNA damage-induced senescence and epirubicin resistance
inferred← citeshttps://doi.org/10.1038/s41388-021-01856-9openalex-snapshot
Mechanisms driving chromosomal translocations: lost in time and space
inferred← citeshttps://doi.org/10.1038/s41418-024-01296-4openalex-snapshot
The cGAS-Ku80 complex regulates the balance between two end joining subpathways
inferred← citeshttps://doi.org/10.1038/s41467-020-19060-wopenalex-snapshot
Repair of G1 induced DNA double-strand breaks in S-G2/M by alternative NHEJ
inferred← citeshttps://doi.org/10.1038/s41556-019-0388-0openalex-snapshot
The nucleoskeleton protein IFFO1 immobilizes broken DNA and suppresses chromosome translocation during tumorigenesis
inferred← citeshttps://doi.org/10.1038/srep41812openalex-snapshot
Regulation of pairing between broken DNA-containing chromatin regions by Ku80, DNA-PKcs, ATM, and 53BP1
inferred← citeshttps://doi.org/10.1073/pnas.0708541104openalex-snapshot
Defects in XRCC4 and KU80 differentially affect the joining of distal nonhomologous ends
inferred← citeshttps://doi.org/10.1073/pnas.0902076106openalex-snapshot
Chromosomal translocations induced at specified loci in human stem cells
inferred← citeshttps://doi.org/10.1073/pnas.0902545106openalex-snapshot
Aberrantly resolved RAG-mediated DNA breaks in Atm-deficient lymphocytes target chromosomal breakpoints in cis
inferred← citeshttps://doi.org/10.1073/pnas.0906355107openalex-snapshot
An end-joining repair mechanism in Escherichia coli
inferred← citeshttps://doi.org/10.1073/pnas.1801591115openalex-snapshot
Parp3 promotes long-range end joining in murine cells
inferred← citeshttps://doi.org/10.1073/pnas.1818595116openalex-snapshot
Frequency of DNA end joining in trans is not determined by the predamage spatial proximity of double-strand breaks in yeast
inferred← citeshttps://doi.org/10.1073/pnas.2014176117openalex-snapshot
Polymerase δ promotes chromosomal rearrangements and imprecise double-strand break repair
inferred← citeshttps://doi.org/10.1074/jbc.r700039200openalex-snapshot
The Mechanism of Human Nonhomologous DNA End Joining
inferred← citeshttps://doi.org/10.1083/jcb.201401146openalex-snapshot
Polo-like kinase 3 regulates CtIP during DNA double-strand break repair in G1
inferred← citeshttps://doi.org/10.1093/jnci/djx059openalex-snapshot
Drugging the Cancers Addicted to DNA Repair
inferred← citeshttps://doi.org/10.1093/jncimonographs/lgn009openalex-snapshot
Induction of Chromosomal Translocations in Mouse and Human Cells Using Site-Specific Endonucleases
inferred← citeshttps://doi.org/10.1093/nar/gkac073openalex-snapshot
RAD51 protects against nonconservative DNA double-strand break repair through a nonenzymatic function
inferred← citeshttps://doi.org/10.1093/nar/gkaf1354openalex-snapshot
A nucleoskeleton network preserves genomic integrity by promoting NHEJ and restraining chromosome translocations
inferred← citeshttps://doi.org/10.1093/nar/gkaf468openalex-snapshot
Genome rearrangements induced by the stimulation of end-joining of DNA double strand breaks through multiple phosphorylation of MRE11 by the kinase PKB/AKT1
inferred← citeshttps://doi.org/10.1093/nar/gkn572openalex-snapshot
Evidence for an inositol hexakisphosphate-dependent role for Ku in mammalian nonhomologous end joining that is independent of its role in the DNA-dependent protein kinase
inferred← citeshttps://doi.org/10.1093/nar/gkr1272openalex-snapshot
MRE11 and RAD50, but not NBS1, are essential for gene targeting in the moss Physcomitrella patens
inferred← citeshttps://doi.org/10.1093/nar/gkr223openalex-snapshot
How does DNA break during chromosomal translocations?
inferred← citeshttps://doi.org/10.1093/nar/gkr656openalex-snapshot
Ku counteracts mobilization of PARP1 and MRN in chromatin damaged with DNA double-strand breaks
inferred← citeshttps://doi.org/10.1093/nar/gks585openalex-snapshot
Terminal deoxynucleotidyl transferase requires KU80 and XRCC4 to promote N-addition at non-V(D)J chromosomal breaks in non-lymphoid cells
inferred← citeshttps://doi.org/10.1093/nar/gku298openalex-snapshot
Requirement for Parp-1 and DNA ligases 1 or 3 but not of Xrcc1 in chromosomal translocation formation by backup end joining
inferred← citeshttps://doi.org/10.1093/nar/gkv714openalex-snapshot
Processing by MRE11 is involved in the sensitivity of subtelomeric regions to DNA double-strand breaks
inferred← citeshttps://doi.org/10.1093/narcan/zcac029openalex-snapshot
High-throughput screen to identify compounds that prevent or target telomere loss in human cancer cells
inferred← citeshttps://doi.org/10.1093/ndt/gfs571openalex-snapshot
A possible role of cystatin C in adipose tissue homeostasis may impact kidney function estimation in metabolic syndrome
inferred← citeshttps://doi.org/10.1097/moh.0b013e328302f711openalex-snapshot
Mechanisms of leukemia translocations
inferred← citeshttps://doi.org/10.1101/cshperspect.a012740openalex-snapshot
Repair of Strand Breaks by Homologous Recombination
inferred← citeshttps://doi.org/10.1101/cshperspect.a012757openalex-snapshot
Repair of Double-Strand Breaks by End Joining
inferred← citeshttps://doi.org/10.1101/gr.147314.112openalex-snapshot
Cancer translocations in human cells induced by zinc finger and TALE nucleases
inferred← citeshttps://doi.org/10.1111/febs.14053openalex-snapshot
Genomic rearrangements induced by unscheduled DNA double strand breaks in somatic mammalian cells
inferred← citeshttps://doi.org/10.1111/nyas.12370openalex-snapshot
Mechanisms of oncogenic chromosomal translocations
inferred← citeshttps://doi.org/10.1111/tpj.12793openalex-snapshot
Comprehensive identification of mutations induced by heavy‐ion beam irradiation in A rabidopsis thaliana
inferred← citeshttps://doi.org/10.1126/science.1237150openalex-snapshot
Spatial Dynamics of Chromosome Translocations in Living Cells
inferred← citeshttps://doi.org/10.1128/mcb.01137-09openalex-snapshot
Effect of Telomere Proximity on Telomere Position Effect, Chromosome Healing, and Sensitivity to DNA Double-Strand Breaks in a Human Tumor Cell Line
inferred← citeshttps://doi.org/10.1134/s0006297925602102openalex-snapshot
The Role of Non-Homologous End Joining and Microhomology-Mediated End Joining in Chromosomal Rearrangements
inferred← citeshttps://doi.org/10.1142/9781848165939_0005openalex-snapshot
Resolution of AID Lesions in Class Switch Recombination
inferred← citeshttps://doi.org/10.1146/annurev-cellbio-111822-014426openalex-snapshot
Microhomology-Mediated End-Joining Chronicles: Tracing the Evolutionary Footprints of Genome Protection
inferred← citeshttps://doi.org/10.1146/annurev.biochem.052308.093131openalex-snapshot
The Mechanism of Double-Strand DNA Break Repair by the Nonhomologous DNA End-Joining Pathway
inferred← citeshttps://doi.org/10.1158/0008-5472.can-08-4833openalex-snapshot
Suppression of Nonhomologous End Joining Repair by Overexpression of HMGA2
inferred← citeshttps://doi.org/10.11606/t.46.2015.tde-23092015-142853openalex-snapshot
Estudo dos mecanismos moleculares do reparo de quebra de duplas fitas no DNA mitocondrial
inferred← citeshttps://doi.org/10.1182/blood-2009-11-255109openalex-snapshot
In search of the original leukemic clone in chronic myeloid leukemia patients in complete molecular remission after stem cell transplantation or imatinib
inferred← citeshttps://doi.org/10.1182/blood-2011-05-352252openalex-snapshot
Chronic myelogenous leukemia stem and progenitor cells demonstrate chromosomal instability related to repeated breakage-fusion-bridge cycles mediated by increased nonhomologous end joining
inferred← citeshttps://doi.org/10.1182/blood-2012-10-460527openalex-snapshot
PARP1 is required for chromosomal translocations
inferred← citeshttps://doi.org/10.1182/blood-2013-07-515874openalex-snapshot
Thrombopoietin promotes NHEJ DNA repair in hematopoietic stem cells through specific activation of Erk and NF-κB pathways and their target, IEX-1
inferred← citeshttps://doi.org/10.1182/blood.2020005895openalex-snapshot
Epidermal growth factor receptor–dependent DNA repair promotes murine and human hematopoietic regeneration
inferred← citeshttps://doi.org/10.1186/s13014-016-0750-3openalex-snapshot
Biology of high single doses of IORT: RBE, 5 R’s, and other biological aspects
inferred← citeshttps://doi.org/10.1186/s13014-022-02113-3openalex-snapshot
Ku70 affects the frequency of chromosome translocation in human lymphocytes after radiation and T-cell acute lymphoblastic leukemia
inferred← citeshttps://doi.org/10.1371/journal.pgen.1000855openalex-snapshot
Ku Regulates the Non-Homologous End Joining Pathway Choice of DNA Double-Strand Break Repair in Human Somatic Cells
inferred← citeshttps://doi.org/10.1371/journal.pgen.1001005openalex-snapshot
Dual Roles for DNA Polymerase Theta in Alternative End-Joining Repair of Double-Strand Breaks in Drosophila
inferred← citeshttps://doi.org/10.1371/journal.pgen.1001194openalex-snapshot
ATM Limits Incorrect End Utilization during Non-Homologous End Joining of Multiple Chromosome Breaks
inferred← citeshttps://doi.org/10.1371/journal.pgen.1002080openalex-snapshot
DNA Ligase III Promotes Alternative Nonhomologous End-Joining during Chromosomal Translocation Formation
inferred← citeshttps://doi.org/10.1371/journal.pgen.1003386openalex-snapshot
The Role of ATM in the Deficiency in Nonhomologous End-Joining near Telomeres in a Human Cancer Cell Line
inferred← citeshttps://doi.org/10.1371/journal.pgen.1004086openalex-snapshot
Is Non-Homologous End-Joining Really an Inherently Error-Prone Process?
inferred← citeshttps://doi.org/10.1371/journal.pgen.1006230openalex-snapshot
53BP1 Protects against CtIP-Dependent Capture of Ectopic Chromosomal Sequences at the Junction of Distant Double-Strand Breaks
inferred← citeshttps://doi.org/10.1371/journal.pgen.1008101openalex-snapshot
DSB structure impacts DNA recombination leading to class switching and chromosomal translocations in human B cells
inferred← citeshttps://doi.org/10.1371/journal.pone.0049301openalex-snapshot
Hyperactivation of PARP Triggers Nonhomologous End-Joining in Repair-Deficient Mouse Fibroblasts
inferred← citeshttps://doi.org/10.1371/journal.pone.0062481openalex-snapshot
Human RECQ1 Interacts with Ku70/80 and Modulates DNA End-Joining of Double-Strand Breaks
inferred← citeshttps://doi.org/10.15252/embj.201796948openalex-snapshot
Mutational signatures of non‐homologous and polymerase theta‐mediated end‐joining in embryonic stem cells
inferred← citeshttps://doi.org/10.1667/rade-24-00038.1openalex-snapshot
The Central Role of Cytogenetics in Radiation Biology
inferred← citeshttps://doi.org/10.1667/rr13723.1openalex-snapshot
Effect of Radiation Quality on Mutagenic Joining of Enzymatically-Induced DNA Double-Strand Breaks in Previously Irradiated Human Cells
inferred← citeshttps://doi.org/10.1667/rr15053.1openalex-snapshot
Molecular Cytogenetics Guides Massively Parallel Sequencing of a Radiation-Induced Chromosome Translocation in Human Cells
inferred← citeshttps://doi.org/10.1667/rr3332.1openalex-snapshot
Increased Mutagenic Joining of Enzymatically-Induced DNA Double-Strand Breaks in High-Charge and Energy Particle Irradiated Human Cells
inferred← citeshttps://doi.org/10.17615/5jdw-e446openalex-snapshot
Essential Roles for Polymerase Theta-Mediated End Joining in Repair of Chromosome Breaks
inferred← citeshttps://doi.org/10.18632/oncoscience.178openalex-snapshot
Whole genome sequence analysis links chromothripsis to EGFR, MDM2, MDM4, and CDK4 amplification in glioblastoma
inferred← citeshttps://doi.org/10.21037/1152openalex-snapshot
Alternative pathways of non-homologous end joining (NHEJ) in genomic instability and cancer
inferred← citeshttps://doi.org/10.21037/tcr.2017.06.02openalex-snapshot
Translational research in radiation-induced DNA damage signaling and repair
inferred← citeshttps://doi.org/10.21203/rs.3.rs-2879043/v1openalex-snapshot
Chromatin Structural Characteristics as an Evidence of Nonhomologous End Joining Repair Pathway Promoting TMPRSS2-ERG Gene Fusion in Prostatic Cancer
inferred← citeshttps://doi.org/10.22074/cellj.2018.5210openalex-snapshot
The Association between PARP1 and LIG3 Expression Levels and Chromosomal Translocations in Acute Myeloid Leukemia Patients.
inferred← citeshttps://doi.org/10.3389/fonc.2012.00135openalex-snapshot
Mechanisms of telomere loss and their consequences for chromosome instability
inferred← citeshttps://doi.org/10.3389/fpls.2021.752108openalex-snapshot
Mutagenic Effect of Proton Beams Characterized by Phenotypic Analysis and Whole Genome Sequencing in Arabidopsis
inferred← citeshttps://doi.org/10.3390/genes11010099openalex-snapshot
Clustered DNA Double-Strand Breaks: Biological Effects and Relevance to Cancer Radiotherapy
inferred← citeshttps://doi.org/10.3390/genes12091310openalex-snapshot
Polymerase θ Coordinates Multiple Intrinsic Enzymatic Activities during DNA Repair
inferred← citeshttps://doi.org/10.3390/ijms24054741openalex-snapshot
DNA Damage and Its Role in Cancer Therapeutics
inferred← citeshttps://doi.org/10.3390/ijms27135905openalex-snapshot
CRISPR/Cas9-Based Genome Editing: Understanding Differences in DNA Repair Pathways, Profiles, and Outcomes
inferred← citeshttps://doi.org/10.3791/56617openalex-snapshot
Immunofluorescence Microscopy of γH2AX and 53BP1 for Analyzing the Formation and Repair of DNA Double-strand Breaks
inferred← citeshttps://doi.org/10.3978/j.issn.2218-676x.2013.04.02openalex-snapshot
DNA double strand break repair via non-homologous end-joining.
inferred← citeshttps://doi.org/10.3978/j.issn.2218-676x.2013.05.01openalex-snapshot
Inhibition of the non homologous end joining process in the context of hypoxic tumor cells
inferred← citeshttps://doi.org/10.4161/cc.8.15.9085openalex-snapshot
Chimeric IgH-TCRα/δ translocations in T lymphocytes mediated by RAG
inferred← citeshttps://doi.org/10.4172/1948-5956.s8-001openalex-snapshot
DNA-PK, a Pharmacological Target in Cancer Chemotherapy and Radiotherapy?
inferred← citeshttps://doi.org/10.48496/vqfx-t640openalex-snapshot
The Extent and Nature Of Chromosomal Rearrangements in B Lymphocytes
inferred← citeshttps://doi.org/10.6083/m41n7z38openalex-snapshot
Cellular response pathways to formaldehyde
inferred← citeshttps://doi.org/10.6084/m9.figshare.20521358openalex-snapshot
Additional file 1 of Ku70 affects the frequency of chromosome translocation in human lymphocytes after radiation and T-cell acute lymphoblastic leukemia
inferred← citeshttps://doi.org/10.70675/02d2d5f8z36aaz4d04z9d37z5384351bf9cfopenalex-snapshot
Réparation des cassures double brin de l'adn chez les mammifères : rôle des protéines MRE11 et BLM dans l’initiation de la ligature d’extrémités non homologues (NHEJ )
inferred← citeshttps://doi.org/10.70675/2db5ae5fzd2c0z4df3zb852zec2a10a44314openalex-snapshot
RAD51 Protects Against RAD52-Dependent Non-Conservative Double-Strand Break Repair Processes, by Impeding the Annealing Step
inferred← citeshttps://doi.org/10.70675/359bd204z134fz4c92z85e1zaef54e2e7826openalex-snapshot
Role of thrombopoietin in DNA repair an genomic integrity in hematopoietic stem cells
inferred← citeshttps://doi.org/10.70675/4ade78c4z0bf6z4533zb4a6z084522268290openalex-snapshot
Rôle des virus EBV et VIH dans la formation de la translocation chromosomique t(8;14) du Lymphome de Burkitt
inferred← citeshttps://doi.org/10.70675/6096400azb435z40e9zab82zebc5026b539bopenalex-snapshot
Etudes des translocations chromosomiques en utilisant les méthodes d'édition du génome : des mécanismes moléculaires à l’oncogenèse
inferred← citeshttps://doi.org/10.70675/80bdaa99zed42z4eb6zb888z9489e31eec93openalex-snapshot
Dynamics of Rad51 during homologous recombination in living yeast
inferred← citeshttps://doi.org/10.70675/848566f0z81b5z4b78z9817z58941b02acd2openalex-snapshot
Drosophila intestinal stem cell response to DNA damage and replication stress
inferred← citeshttps://doi.org/10.70675/89719cadza51cz4509zbd9fz3da99c280491openalex-snapshot
DNA double-strand break formation and signalling in response to transcription-blocking topoisomerase I complexes
inferred← citeshttps://doi.org/10.70675/8f8f5907z32edz4a84zb3c8zc042507f5b0dopenalex-snapshot
Recombinaison homologue protège contre les défauts de la mitose et la ségrégation des chromosomes déséquilibre causé par le stress de réplication spontanée
inferred← citeshttps://doi.org/10.70675/9f331f7az9151z4c95zbf47zbc6ed65a5896openalex-snapshot
Factors That Affect Chromosomal Translocations in Cells
inferred← citeshttps://doi.org/10.70675/a26d5e5fzf79az42bczae57zc19deae4d7e0openalex-snapshot
Etapes précoces de la lymphomagenèse folliculaire
inferred← citeshttps://doi.org/10.70675/b21b4cebze26cz412bz8d39z1c24f5f3c7b3openalex-snapshot
Mécanismes de contrôle de l’endonucléase Mus81- Eme1 et maintien de la stabilité du génome dans la levure Schizosaccharomyces pombe
inferred← citeshttps://doi.org/10.70675/e3a4a46bzed37z4f21zb647z0313e5cd5811openalex-snapshot
Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome
inferred← citeshttps://doi.org/10.7936/k7k07291openalex-snapshot
ATM Deficiency -- A Multifaceted Defect in Lymphocyte Development
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