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Meta-analysis and imputation refines the association of 15q25 with smoking quantity
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Genome-wide association study of recurrent major depressive disorder in two European case–control cohorts
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Genetic variation in the CHRNA5 gene affects mRNA levels and is associated with risk for alcohol dependence
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Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls
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A second generation human haplotype map of over 3.1 million SNPs
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A variant associated with nicotine dependence, lung cancer and peripheral arterial disease
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A susceptibility locus for lung cancer maps to nicotinic acetylcholine receptor subunit genes on 15q25
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SLC2A9 is a newly identified urate transporter influencing serum urate concentration, urate excretion and gout
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Genome-wide association scan of tag SNPs identifies a susceptibility locus for lung cancer at 15q25.1
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Genome-wide association of early-onset myocardial infarction with single nucleotide polymorphisms and copy number variants
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Genome-wide meta-analyses identify multiple loci associated with smoking behavior
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Sequence variants at CHRNB3–CHRNA6 and CYP2A6 affect smoking behavior
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A new multipoint method for genome-wide association studies by imputation of genotypes
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Validating, augmenting and refining genome-wide association signals
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α-5/α-3 nicotinic receptor subunit alleles increase risk for heavy smoking
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KORA-gen - Resource for Population Genetics, Controls and a Broad Spectrum of Disease Phenotypes
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Genome-wide association and meta-analysis of bipolar disorder in individuals of European ancestry
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Novel genes identified in a high-density genome wide association study for nicotine dependence
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Practical aspects of imputation-driven meta-analysis of genome-wide association studies
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Risk for nicotine dependence and lung cancer is conferred by mRNA expression levels and amino acid change in CHRNA5
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Ethnicity-related differences in left ventricular function, structure and geometry: a population study of UK Indian Asian and European white subjects
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Transcription Deregulation at the 15q25 Locus in Association with Lung Adenocarcinoma Risk
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Estimating the Dimension of a Model
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Heritability of Cardiovascular and Personality Traits in 6,148 Sardinians
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A Genome-Wide Investigation of SNPs and CNVs in Schizophrenia
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A Genome-Wide Association Study in Chronic Obstructive Pulmonary Disease (COPD): Identification of Two Major Susceptibility Loci
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What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Increased Genetic Vulnerability to Smoking at CHRNA5 in Early-Onset Smokers
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Association ofCHRNA4polymorphisms with smoking behavior in two populations
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Genome‐wide search for replicable risk gene regions in alcohol and nicotine co‐dependence
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Associations of rare nicotinic cholinergic receptor gene variants to nicotine and alcohol dependence
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Genome‐wide meta‐analysis identifies a novel susceptibility signal at CACNA2D3 for nicotine dependence
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Methods for the Analysis and Interpretation for Rare Variants Associated with Complex Traits
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The Role of Pharmacogenetics in Smoking
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Transethnic meta‐analysis of genomewide association studies
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Smoking and Genetic Risk Variation Across Populations of E uropean, A sian, and A frican A merican Ancestry—A Meta‐Analysis of Chromosome 15q25
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Genotype Imputation for A frican A mericans Using Data From H ap M ap Phase II Versus 1000 G enomes P rojects
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The Case‐Only Test for Gene–Environment Interaction is Not Uniformly Powerful: An Empirical Example
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Distinct Loci in the CHRNA5/CHRNA3/CHRNB4 Gene Cluster Are Associated With Onset of Regular Smoking
inferred← citeshttps://doi.org/10.1002/gepi.22258openalex-snapshot
Bivariate logistic Bayesian LASSO for detecting rare haplotype association with two correlated phenotypes
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TheCHRNA5/CHRNA3/CHRNB4Nicotinic Receptor Regulome: Genomic Architecture, Regulatory Variants, and Clinical Associations
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BMI1 enhancer polymorphism underlies chromosome 10p12.31 association with childhood acute lymphoblastic leukemia
inferred← citeshttps://doi.org/10.1002/ijc.32318openalex-snapshot
Glioma risk associated with extent of estimated European genetic ancestry in African Americans and Hispanics
inferred← citeshttps://doi.org/10.1002/ijc.33616openalex-snapshot
Differential genetic influences over colorectal cancer risk and gene expression in large bowel mucosa
inferred← citeshttps://doi.org/10.1002/mco2.415openalex-snapshot
Genome‐wide meta‐analysis and fine‐mapping prioritize potential causal variants and genes related to leprosy
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Re‐defininG AddiCCH3Tion: genomics and epigenomics on substance use disorders
inferred← citeshttps://doi.org/10.1002/wps.20250openalex-snapshot
At risk or not at risk? A meta‐analysis of the prognostic accuracy of psychometric interviews for psychosis prediction
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Molecular Mechanisms Associated with Nicotine Pharmacology and Dependence
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Genetic Susceptibility to Lung Cancer
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Genetics of Substance Use Disorders
inferred← citeshttps://doi.org/10.1007/978-1-4939-0653-6_5openalex-snapshot
Have the Genetics of Cannabis Involvement Gone to Pot?
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The DNA Methylation Signature of Smoking: An Archetype for the Identification of Biomarkers for Behavioral Illness
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Genetic Contributions of the α5 Nicotinic Receptor Subunit to Smoking Behavior
inferred← citeshttps://doi.org/10.1007/978-1-4939-1167-7_17openalex-snapshot
Smoking-Related Genes and Functional Consequences
inferred← citeshttps://doi.org/10.1007/978-1-4939-2824-8_15openalex-snapshot
Meta-Analysis of Rare Variants
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Meta-Analysis of Common and Rare Variants
inferred← citeshttps://doi.org/10.1007/978-3-030-31507-8_24openalex-snapshot
Precision Medicine for Cigarette Smoking Addiction
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Lung Cancer in Developing Countries
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Linkage and Association Studies of Substance Use Disorders
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Genetics of Smoking Behaviour
inferred← citeshttps://doi.org/10.1007/978-3-319-13665-3_3openalex-snapshot
Pharmacogenetics of Nicotine and Associated Smoking Behaviors
inferred← citeshttps://doi.org/10.1007/978-3-319-62941-4_3openalex-snapshot
Genetic Susceptibility to Lung Cancer
inferred← citeshttps://doi.org/10.1007/978-3-642-45028-0_5-1openalex-snapshot
Genetik und Gen-Umwelt-Interaktionen bei psychischen Erkrankungen
inferred← citeshttps://doi.org/10.1007/978-3-642-45028-0_5-2openalex-snapshot
Genetik und Gen-Umwelt-Interaktionen bei psychischen Erkrankungen
inferred← citeshttps://doi.org/10.1007/978-3-642-45378-6_5openalex-snapshot
Genetic Vulnerability to Eating Disorders and Substance Use Disorders
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Genetik und Gen-Umwelt-Interaktionen bei psychischen Erkrankungen
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Genetic Variability in Molecular Responses to Chemical Exposure
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The Genetics of Addiction: A Global Problem with Global Opportunities
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inferred← citeshttps://doi.org/10.1007/978-981-10-5717-5_8openalex-snapshot
Interactions Between Genetics, Lifestyle, and Environmental Factors for Healthcare
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Converging Findings from Linkage and Association Analyses on Susceptibility Genes for Smoking Addiction
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Involvement of Variants in Gene Clusters CHRNA5/A3/B4 on Chromosome 15 to Smoking Behaviors and Lung Cancer
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Biochemical Diagnosis in Substance and Non-substance Addiction
inferred← citeshttps://doi.org/10.1007/s00018-013-1553-4openalex-snapshot
Transcription factor 4 (TCF4) and schizophrenia: integrating the animal and the human perspective
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Extraglottische Atemwegshilfen auf der Intensivstation
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Exome sequence genotype imputation in globally diverse hexaploid wheat accessions
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Multi-omics analysis identifies CpGs near G6PC2 mediating the effects of genetic variants on fasting glucose
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Neuropeptide systems and new treatments for nicotine addiction
inferred← citeshttps://doi.org/10.1007/s00401-018-1825-zopenalex-snapshot
Diffuse gliomas classified by 1p/19q co-deletion, TERT promoter and IDH mutation status are associated with specific genetic risk loci
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An association study on the CHRNA5/A3/B4 gene cluster, smoking and psoriasis vulgaris
inferred← citeshttps://doi.org/10.1007/s00439-010-0911-7openalex-snapshot
Uncovering hidden variance: pair-wise SNP analysis accounts for additional variance in nicotine dependence
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Large-scale genome-wide association study of Asian population reveals genetic factors in FRMD4A and other loci influencing smoking initiation and nicotine dependence
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Translational genetic approaches to substance use disorders: bridging the gap between mice and humans
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Pharmacogenetics of smoking cessation: role of nicotine target and metabolism genes
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Dissecting direct and indirect genetic effects on chronic obstructive pulmonary disease (COPD) susceptibility
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Atlas of human diseases influenced by genetic variants with extreme allele frequency differences
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Genotype imputation for Han Chinese population using Haplotype Reference Consortium as reference
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A Twin Association Study of Nicotine Dependence with Markers in the CHRNA3 and CHRNA5 Genes
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Externalizing Behaviors are Associated with SNPs in the CHRNA5/CHRNA3/CHRNB4 Gene Cluster
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Genome-Wide Association Study of Behavioral Disinhibition in a Selected Adolescent Sample
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Association of the OPRM1 Variant rs1799971 (A118G) with Non-Specific Liability to Substance Dependence in a Collaborative de novo Meta-Analysis of European-Ancestry Cohorts
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The Long-Term Consequences of Vietnam-Era Conscription and Genotype on Smoking Behavior and Health
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GW-SEM: A Statistical Package to Conduct Genome-Wide Structural Equation Modeling
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Clarifying the Genetic Influences on Nicotine Dependence and Quantity of Use in Cigarette Smokers
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Genes as instruments for studying risk behavior effects: an application to maternal smoking and orofacial clefts
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A Genetic Lung Cancer Susceptibility Test may have a Positive Effect on Smoking Cessation
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The impact of cigarette quitting during pregnancy on other prenatal health behaviors
inferred← citeshttps://doi.org/10.1007/s11886-015-0584-xopenalex-snapshot
Genetic Risk Factors and Mendelian Randomization in Cardiovascular Disease
inferred← citeshttps://doi.org/10.1007/s11920-011-0176-4openalex-snapshot
Genome-Wide Association Studies of Alcohol Dependence and Substance Use Disorders
inferred← citeshttps://doi.org/10.1007/s11920-014-0477-5openalex-snapshot
Determining the Causes and Consequences of Nicotine Dependence: Emerging Genetic Research Methods
inferred← citeshttps://doi.org/10.1007/s11920-018-0873-3openalex-snapshot
Human Genetics of Addiction: New Insights and Future Directions
inferred← citeshttps://doi.org/10.1007/s12017-013-8286-2openalex-snapshot
ADHD Diagnosis May Influence the Association between Polymorphisms in Nicotinic Acetylcholine Receptor Genes and Tobacco Smoking
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Introduction to Deep Sequencing and Its Application to Drug Addiction Research with a Focus on Rare Variants
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Contribution of Variants in CHRNA5/A3/B4 Gene Cluster on Chromosome 15 to Tobacco Smoking: From Genetic Association to Mechanism
inferred← citeshttps://doi.org/10.1007/s12501-016-0089-3openalex-snapshot
Zit middelengebruik en verslavingsgedrag in de familie?
inferred← citeshttps://doi.org/10.1007/s13167-022-00308-yopenalex-snapshot
Global burden of cancers attributable to tobacco smoking, 1990–2019: an ecological study
inferred← citeshttps://doi.org/10.1007/s13258-012-0054-4openalex-snapshot
Characterization of functional variants in 33 blood pressure loci using 1000 genomes project data
inferred← citeshttps://doi.org/10.1007/s13577-022-00709-1openalex-snapshot
The α5-nAChR/PD-L1 axis facilitates lung adenocarcinoma cell migration and invasion
inferred← citeshttps://doi.org/10.1007/s40263-015-0243-1openalex-snapshot
Biomarkers of Response to Smoking Cessation Pharmacotherapies: Progress to Date
inferred← citeshttps://doi.org/10.1007/s40429-015-0042-2openalex-snapshot
Nicotinic Receptor Contributions to Smoking: Insights from Human Studies and Animal Models
inferred← citeshttps://doi.org/10.1007/s40429-019-00273-zopenalex-snapshot
The Impact of Genes on Adolescent Substance Use: a Developmental Perspective
inferred← citeshttps://doi.org/10.1007/s40473-013-0004-8openalex-snapshot
The Genetics, Neurogenetics and Pharmacogenetics of Addiction
inferred← citeshttps://doi.org/10.1007/s43440-021-00243-1openalex-snapshot
Nicotinic acetylcholine gene cluster CHRNA5-A3-B4 variants influence smoking status in a Bangladeshi population
inferred← citeshttps://doi.org/10.1016/b978-0-12-380890-5.00002-8openalex-snapshot
Genetic Basis for Susceptibility to Lung Cancer
inferred← citeshttps://doi.org/10.1016/b978-0-12-383834-6.00061-6openalex-snapshot
Genetics of Atherosclerotic Cardiovascular Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-383834-6.00119-1openalex-snapshot
Addictive Disorders
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Applying In Silico Integrative Genomics to Genetic Studies of Human Disease
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Opportunities for Bioinformatics in the Classification of Behavior and Psychiatric Disorders
inferred← citeshttps://doi.org/10.1016/b978-0-12-398335-0.00045-5openalex-snapshot
Genetics of Nicotine Addiction
inferred← citeshttps://doi.org/10.1016/b978-0-12-405941-2.00026-2openalex-snapshot
Complex Genetics of Alcoholism
inferred← citeshttps://doi.org/10.1016/b978-0-12-418679-8.00002-2openalex-snapshot
Genetics of Substance Use, Abuse, Cessation, and Addiction
inferred← citeshttps://doi.org/10.1016/b978-0-12-800105-9.00038-xopenalex-snapshot
Clinical Syndromes of Substance Use Disorder
inferred← citeshttps://doi.org/10.1016/b978-0-12-800213-1.00020-1openalex-snapshot
Genetic Findings on the Relationship between Smoking and the Stress System
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Contribution of Translational Genetic Research to Our Understanding of Nicotine Dependence
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Role of Conventional Risk Factors in Genetic Susceptibility to Cardiovascular Diseases
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Habenular Synapses and Nicotine
inferred← citeshttps://doi.org/10.1016/b978-0-12-813035-3.00032-0openalex-snapshot
The Medial Habenula-Interpeduncular Nucleus Pathway in Nicotine Sensitization: The Role of α3β4 Nicotinic Acetylcholine Receptors and Substance P
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Genetics, imaging, and cognition
inferred← citeshttps://doi.org/10.1016/b978-0-12-820472-6.00136-5openalex-snapshot
GPCR Patient Drug Interaction—Pharmacogenetics: Genome-Wide Association Studies (GWAS)
inferred← citeshttps://doi.org/10.1016/b978-0-323-47674-4.00024-4openalex-snapshot
Nicotine Dependence
inferred← citeshttps://doi.org/10.1016/b978-1-4377-2362-5.00001-3openalex-snapshot
The Etiology of Cancer
inferred← citeshttps://doi.org/10.1016/b978-1-4557-2865-7.00024-2openalex-snapshot
Nicotine Dependence
inferred← citeshttps://doi.org/10.1016/j.addbeh.2013.08.033openalex-snapshot
The association between CHRN genetic variants and dizziness at first inhalation of cigarette smoke
inferred← citeshttps://doi.org/10.1016/j.addbeh.2018.05.001openalex-snapshot
The role of nicotinic receptor genes (CHRN) in the pathways of prenatal tobacco exposure on smoking behavior among young adult light smokers
inferred← citeshttps://doi.org/10.1016/j.ajhg.2011.08.001openalex-snapshot
A Variant in MCF2L Is Associated with Osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.ajhg.2013.04.004openalex-snapshot
Genome-wide Association Analysis for Multiple Continuous Secondary Phenotypes
inferred← citeshttps://doi.org/10.1016/j.ajhg.2014.07.014openalex-snapshot
Gene-Environment Dependence Creates Spurious Gene-Environment Interaction
inferred← citeshttps://doi.org/10.1016/j.ajhg.2021.02.001openalex-snapshot
Whole-genome sequencing of African Americans implicates differential genetic architecture in inflammatory bowel disease
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2016.09.008openalex-snapshot
Genetic analysis of emerging risk factors in coronary artery disease
inferred← citeshttps://doi.org/10.1016/j.avb.2024.101948openalex-snapshot
“Weeding out” violence? Translational perspectives on the neuropsychobiological links between cannabis and aggression
inferred← citeshttps://doi.org/10.1016/j.bbr.2016.04.008openalex-snapshot
18-Methoxycoronaridine acts in the medial habenula to attenuate behavioral and neurochemical sensitization to nicotine
inferred← citeshttps://doi.org/10.1016/j.bcp.2015.08.084openalex-snapshot
UBXN2A regulates nicotinic receptor degradation by modulating the E3 ligase activity of CHIP
inferred← citeshttps://doi.org/10.1016/j.biopsych.2010.06.030openalex-snapshot
Genome-Wide Association Scan of Trait Depression
inferred← citeshttps://doi.org/10.1016/j.biopsych.2010.09.055openalex-snapshot
TTC12-ANKK1-DRD2 and CHRNA5-CHRNA3-CHRNB4 Influence Different Pathways Leading to Smoking Behavior from Adolescence to Mid-Adulthood
inferred← citeshttps://doi.org/10.1016/j.biopsych.2011.02.001openalex-snapshot
Developmental Perspective on the Role of Genes in Smoking Risk
inferred← citeshttps://doi.org/10.1016/j.biopsych.2011.04.017openalex-snapshot
Rare Nonsynonymous Variants in Alpha-4 Nicotinic Acetylcholine Receptor Gene Protect Against Nicotine Dependence
inferred← citeshttps://doi.org/10.1016/j.biopsych.2011.07.018openalex-snapshot
New Approaches to Identifying Rare Genetic Variants Associated with Nicotine Dependence
inferred← citeshttps://doi.org/10.1016/j.biopsych.2013.08.027openalex-snapshot
Rare Nonsynonymous Exonic Variants in Addiction and Behavioral Disinhibition
inferred← citeshttps://doi.org/10.1016/j.biopsych.2014.08.005openalex-snapshot
Genetics of Complex Traits in Psychiatry
inferred← citeshttps://doi.org/10.1016/j.biopsych.2014.08.025openalex-snapshot
Genome-Wide Association Study of Nicotine Dependence in American Populations: Identification of Novel Risk Loci in Both African-Americans and European-Americans
inferred← citeshttps://doi.org/10.1016/j.biopsych.2015.03.011openalex-snapshot
Complexities of the Mu Opioid Receptor
inferred← citeshttps://doi.org/10.1016/j.biopsycho.2010.07.018openalex-snapshot
A genome-wide association study of Cloninger's temperament scales: Implications for the evolutionary genetics of personality
inferred← citeshttps://doi.org/10.1016/j.cancergen.2025.11.004openalex-snapshot
Germline lung cancer SNPs dysregulate known (LATS1) and novel (ADCY2) oncogenes through distal, spatially-constrained eQTLs
inferred← citeshttps://doi.org/10.1016/j.canep.2024.102599openalex-snapshot
Genetic variation near GRB10 associated with bone growth and osteosarcoma risk in canine and human populations
inferred← citeshttps://doi.org/10.1016/j.cmi.2021.11.004openalex-snapshot
Risk of lower respiratory tract infections: a genome-wide association study with Mendelian randomization analysis in three independent European populations
inferred← citeshttps://doi.org/10.1016/j.cobeha.2014.08.002openalex-snapshot
Using molecular genetic information to infer causality in observational data: Mendelian randomisation
inferred← citeshttps://doi.org/10.1016/j.coph.2011.08.003openalex-snapshot
From genes to therapeutic targets for psychiatric disorders – what to expect?
inferred← citeshttps://doi.org/10.1016/j.csbj.2025.07.049openalex-snapshot
Role of ferroptosis-related IREB2 in the shared genetic etiology between smoking and facial aging: Insights from large-scale genome-wide cross-trait analysis
inferred← citeshttps://doi.org/10.1016/j.drugalcdep.2012.01.014openalex-snapshot
Nicotine dependence and comorbid psychiatric disorders: Examination of specific genetic variants in the CHRNA5-A3-B4 nicotinic receptor genes
inferred← citeshttps://doi.org/10.1016/j.drugalcdep.2012.02.003openalex-snapshot
ANAPC1 and SLCO3A1 are associated with nicotine dependence: Meta-analysis of genome-wide association studies
inferred← citeshttps://doi.org/10.1016/j.drugalcdep.2014.05.013openalex-snapshot
Scientific overview: 2013 BBC plenary symposium on tobacco addiction
inferred← citeshttps://doi.org/10.1016/j.drugalcdep.2014.06.027openalex-snapshot
Interplay of genetic risk (CHRNA5) and environmental risk (partner smoking) on cigarette smoking reduction
inferred← citeshttps://doi.org/10.1016/j.drugalcdep.2015.02.029openalex-snapshot
Are genetic variants for tobacco smoking associated with cannabis involvement?
inferred← citeshttps://doi.org/10.1016/j.drugalcdep.2015.06.022openalex-snapshot
Genetic variation (CHRNA5), medication (combination nicotine replacement therapy vs. varenicline), and smoking cessation
inferred← citeshttps://doi.org/10.1016/j.drugalcdep.2016.07.029openalex-snapshot
Polygenic risk scores for cigarettes smoked per day do not generalize to a Native American population
inferred← citeshttps://doi.org/10.1016/j.drugalcdep.2018.03.026openalex-snapshot
Exploring the role of low-frequency and rare exonic variants in alcohol and tobacco use
inferred← citeshttps://doi.org/10.1016/j.ebiom.2016.08.012openalex-snapshot
Genetic Risk Can Be Decreased: Quitting Smoking Decreases and Delays Lung Cancer for Smokers With High and Low CHRNA5 Risk Genotypes — A Meta-Analysis
inferred← citeshttps://doi.org/10.1016/j.ehb.2011.09.002openalex-snapshot
Smoking and body weight: Evidence using genetic instruments
inferred← citeshttps://doi.org/10.1016/j.ehb.2013.12.002openalex-snapshot
Mendelian randomization in health research: Using appropriate genetic variants and avoiding biased estimates
inferred← citeshttps://doi.org/10.1016/j.envint.2021.107041openalex-snapshot
Genome-wide gene-air pollution interaction analysis of lung function in 300,000 individuals
inferred← citeshttps://doi.org/10.1016/j.euo.2018.08.020openalex-snapshot
Genome-wide Association Study for Tumour Stage, Grade, Size, and Age at Diagnosis of Non–muscle-invasive Bladder Cancer
inferred← citeshttps://doi.org/10.1016/j.euroneuro.2013.09.010openalex-snapshot
Stratified medicine for mental disorders
inferred← citeshttps://doi.org/10.1016/j.fjpsy.2020.09.001openalex-snapshot
Evidence for the association of the nicotinic acetylcholine receptor CHRNA5/A3/B4 gene cluster and nicotine dependence in a young population of students
inferred← citeshttps://doi.org/10.1016/j.heliyon.2022.e09947openalex-snapshot
CHRNA5 rs16969968 and CHRNA3 rs578776 polymorphisms are associated with multiple nicotine dependence phenotypes in Bangladeshi smokers
inferred← citeshttps://doi.org/10.1016/j.jacc.2015.07.037openalex-snapshot
Cardiovascular Effects of Exposure to Cigarette Smoke and Electronic Cigarettes
inferred← citeshttps://doi.org/10.1016/j.jacc.2016.05.092openalex-snapshot
Cystatin C and Cardiovascular Disease
inferred← citeshttps://doi.org/10.1016/j.jacc.2016.11.056openalex-snapshot
Systematic Evaluation of Pleiotropy Identifies 6 Further Loci Associated With Coronary Artery Disease
inferred← citeshttps://doi.org/10.1016/j.jaut.2020.102562openalex-snapshot
The amino acid variants in HLA II molecules explain the major association with adult-onset Still's disease in the Han Chinese population
inferred← citeshttps://doi.org/10.1016/j.jfda.2013.09.041openalex-snapshot
Genomics and personalized medicine: CHRNA5-CHRNA3-CHRNB4 and smoking cessation treatment
inferred← citeshttps://doi.org/10.1016/j.jhep.2021.04.055openalex-snapshot
An international genome-wide meta-analysis of primary biliary cholangitis: Novel risk loci and candidate drugs
inferred← citeshttps://doi.org/10.1016/j.joca.2016.12.021openalex-snapshot
The causal role of smoking on the risk of hip or knee replacement due to primary osteoarthritis: a Mendelian randomisation analysis of the HUNT study
inferred← citeshttps://doi.org/10.1016/j.joep.2023.102636openalex-snapshot
Challenges in studying the interplay of genes and environment. A study of childhood financial distress moderating genetic predisposition for peak smoking
inferred← citeshttps://doi.org/10.1016/j.jpsychires.2016.06.002openalex-snapshot
Pleiotropic effects of Chr15q25 nicotinic gene cluster and the relationship between smoking, cognition and ADHD
inferred← citeshttps://doi.org/10.1016/j.kint.2020.05.029openalex-snapshot
Common risk variants in NPHS1 and TNFSF15 are associated with childhood steroid-sensitive nephrotic syndrome
inferred← citeshttps://doi.org/10.1016/j.lungcan.2016.05.008openalex-snapshot
Genome-wide association study confirms lung cancer susceptibility loci on chromosomes 5p15 and 15q25 in an African-American population
inferred← citeshttps://doi.org/10.1016/j.molmed.2017.12.001openalex-snapshot
Preparing the Way: Exploiting Genomic Medicine to Stop Smoking
inferred← citeshttps://doi.org/10.1016/j.neulet.2016.05.033openalex-snapshot
Pathways to precision medicine in smoking cessation treatments
inferred← citeshttps://doi.org/10.1016/j.neuron.2011.02.015openalex-snapshot
Genetic Vulnerability and Susceptibility to Substance Dependence
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2013.01.017openalex-snapshot
The α3β4* nicotinic acetylcholine receptor subtype mediates nicotine reward and physical nicotine withdrawal signs independently of the α5 subunit in the mouse
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2013.09.009openalex-snapshot
A glimpse into the future – Personalized medicine for smoking cessation
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2014.11.019openalex-snapshot
The habenulo-interpeduncular pathway in nicotine aversion and withdrawal
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2020.108234openalex-snapshot
Genetic susceptibility to nicotine addiction: Advances and shortcomings in our understanding of the CHRNA5/A3/B4 gene cluster contribution
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2020.108256openalex-snapshot
Nicotinic acetylcholine receptors and nicotine addiction: A brief introduction
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2021.108480openalex-snapshot
Linking the CHRNA5 SNP to drug abuse liability: From circuitry to cellular mechanisms
inferred← citeshttps://doi.org/10.1016/j.ophtha.2020.05.014openalex-snapshot
Genome-Wide Association Study in Asians Identifies Novel Loci for High Myopia and Highlights a Nervous System Role in Its Pathogenesis
inferred← citeshttps://doi.org/10.1016/j.pbb.2014.01.009openalex-snapshot
Finding genomic function for genetic associations in nicotine addiction research: The ENCODE project's role in future pharmacogenomic analysis
inferred← citeshttps://doi.org/10.1016/j.pbb.2018.08.004openalex-snapshot
Biochemical, demographic, and self-reported tobacco-related predictors of the acute heart rate response to nicotine in smokers
inferred← citeshttps://doi.org/10.1016/j.phrs.2023.106860openalex-snapshot
Central and peripheral actions of nicotine that influence blood glucose homeostasis and the development of diabetes
inferred← citeshttps://doi.org/10.1016/j.prrv.2016.08.005openalex-snapshot
Pulmonary Effects of Maternal Smoking on the Fetus and Child: Effects on Lung Development, Respiratory Morbidities, and Life Long Lung Health
inferred← citeshttps://doi.org/10.1016/j.psychres.2019.06.025openalex-snapshot
Polygenic liability for schizophrenia predicts shifting-specific executive function deficits and tobacco use in a moderate drinking community sample
inferred← citeshttps://doi.org/10.1016/j.sleep.2026.108845openalex-snapshot
Genetic evidence for causality of late chronotype on metabolic syndrome in East Asians and Europeans
inferred← citeshttps://doi.org/10.1016/j.taap.2014.04.023openalex-snapshot
Alpha5 nicotinic acetylcholine receptor mediates nicotine-induced HIF-1α and VEGF expression in non-small cell lung cancer
inferred← citeshttps://doi.org/10.1016/j.tig.2010.10.006openalex-snapshot
The study of eQTL variations by RNA-seq: from SNPs to phenotypes
inferred← citeshttps://doi.org/10.1016/j.tins.2020.06.001openalex-snapshot
Do Nicotinic Receptors Modulate High-Order Cognitive Processing?
inferred← citeshttps://doi.org/10.1016/j.xcrm.2024.101923openalex-snapshot
A genome-wide association study identified PRKCB as a causal gene and therapeutic target for Mycobacterium avium complex disease
inferred← citeshttps://doi.org/10.1016/s1877-1203(18)30009-0openalex-snapshot
Facteurs de risque des cancers bronchiques : tabac, exposition professionnelle et environnementale
inferred← citeshttps://doi.org/10.1016/s2213-2600(15)00283-0openalex-snapshot
Novel insights into the genetics of smoking behaviour, lung function, and chronic obstructive pulmonary disease (UK BiLEVE): a genetic association study in UK Biobank
inferred← citeshttps://doi.org/10.1017/s0033291715001518openalex-snapshot
The role of conduct disorder in the relationship between alcohol, nicotine and cannabis use disorders
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