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Valproate protects dopaminergic neurons in midbrain neuron/glia cultures by stimulating the release of neurotrophic factors from astrocytes

https://doi.org/10.1038/sj.mp.4001893

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1001/archpsyc.57.1.90openalex-snapshot

Mind Glue

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Tumor necrosis factor-? regulation of the Id gene family in astrocytes and microglia during CNS inflammatory injury

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Clinicopathological findings following intraventricular glial-derived neurotrophic factor treatment in a patient with Parkinson's disease

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Neuroprotective role of astrocytes in cerebral ischemia: Focus on ischemic preconditioning

inferredcites →https://doi.org/10.1006/exnr.1994.1218openalex-snapshot

Multiple Astrocyte Transcripts Encode Nigral Trophic Factors in Rat and Human

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CNTF regulation of astrogliosis and the activation of microglia in the developing rat central nervous system

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Receptor-coupled uptake of valproate in rat astroglial primary cultures

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Valproate pretreatment protects dopaminergic neurons from LPS-induced neurotoxicity in rat primary midbrain cultures: role of microglia

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New roles for astrocytes (stars at last)

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New roles for astrocytes: Redefining the functional architecture of the brain

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Postmortem studies in mood disorders indicate altered numbers of neurons and glial cells

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Clinical and preclinical evidence for the neurotrophic effects of mood stabilizers: implications for the pathophysiology and treatment of manic–depressive illness

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Neuroimaging studies of mood disorders

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Regulation of Akt and glycogen synthase kinase-3β phosphorylation by sodium valproate and lithium

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Protection and repair of the nigrostriatal dopaminergic system by GDNF in vivo

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Mesencephalic dopaminergic neurons protected by GDNF from axotomy-induced degeneration in the adult brain

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Functional recovery in parkinsonian monkeys treated with GDNF

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Valproic acid inhibits histone deacetylase activity and suppresses excitotoxicity-induced GAPDH nuclear accumulation and apoptotic death in neurons

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Protracted lithium treatment protects against the ER stress elicited by thapsigargin in rat PC12 cells: roles of intracellular calcium, GRP78 and Bcl-2

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Lithium protection against glutamate excitotoxicity in rat cerebral cortical neurons: involvement of NMDA receptor inhibition possibly by decreasing NR2B tyrosine phosphorylation

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GDNF enhances the synaptic efficacy of dopaminergic neurons in culture

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Purification and Initial Characterization of Rat B49 Glial Cell Line‐Derived Neurotrophic Factor

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Glial Cell Line‐Derived Neurotrophic Growth Factor Inhibits Apoptotic Death of Postnatal Substantia Nigra Dopamine Neurons in Primary Culture

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Differential Effects of Glial Cell Line‐Derived Neurotrophic Factor and Neurturin on Developing and Adult Substantia Nigra Dopaminergic Neurons

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The Mood‐Stabilizing Agents Lithium and Valproate RobustlIncrease the Levels of the Neuroprotective Protein bcl‐2 in the CNS

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Molecular consequences of activated microglia in the brain: overactivation induces apoptosis

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Treatment of spinal muscular atrophy by sodium butyrate

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Histone Deacetylase Is a Direct Target of Valproic Acid, a Potent Anticonvulsant, Mood Stabilizer, and Teratogen

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The Mood Stabilizer Valproic Acid Activates Mitogen-activated Protein Kinases and Promotes Neurite Growth

inferredcites →https://doi.org/10.1074/jbc.m307657200openalex-snapshot

NADPH Oxidase Mediates Lipopolysaccharide-induced Neurotoxicity and Proinflammatory Gene Expression in Activated Microglia

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The Role of Microglia in Paraquat-Induced Dopaminergic Neurotoxicity

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The neuropathology of primary mood disorder

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Valproic acid defines a novel class of HDAC inhibitors inducing differentiation of transformed cells

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Sodium butyrate ameliorates phenotypic expression in a transgenic mouse model of spinal and bulbar muscular atrophy

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Synergistic dopaminergic neurotoxicity of MPTP and inflammogen lipopolysaccharide: relevance to the etiology of Parkinson's disease

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Control of Synapse Number by Glia

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Reactive Astrocytes in Neural Repair and Protection

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Randomized, double-blind trial of glial cell line-derived neurotrophic factor (GDNF) in PD

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Primary Rat Mesencephalic Neuron-Glia, Neuron-Enriched, Microglia-Enriched, and Astroglia-Enriched Cultures

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MAP Kinase Activation by Fluoxetine and Its Relation to Gene Expression in Cultured Rat Astrocytes

inferredcites →https://doi.org/10.1385/mn:25:3:245openalex-snapshot

The Role of Astroglia on the Survival of Dopamine Neurons

inferredcites →https://doi.org/10.1523/jneurosci.18-23-09822.1998openalex-snapshot

Glial Cell Line-Derived Neurotrophic Factor Requires Transforming Growth Factor-β for Exerting Its Full Neurotrophic Potential on Peripheral and CNS Neurons

inferredcites →https://doi.org/10.1523/jneurosci.21-02-00581.2001openalex-snapshot

Brain-Derived Neurotrophic Factor and Glial Cell Line-Derived Neurotrophic Factor Are Required Simultaneously for Survival of Dopaminergic Primary Sensory NeuronsIn Vivo

inferredcites →https://doi.org/10.1523/jneurosci.23-12-05141.2003openalex-snapshot

Regulation of Natural Cell Death in Dopaminergic Neurons of the Substantia Nigra by Striatal Glial Cell Line-Derived Neurotrophic FactorIn Vivo

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Critical Role for Microglial NADPH Oxidase in Rotenone-Induced Degeneration of Dopaminergic Neurons

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Histone Deacetylase Inhibition by Sodium Butyrate Chemotherapy Ameliorates the Neurodegenerative Phenotype in Huntington's Disease Mice

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Glia as a Therapeutic Target: Selective Suppression of Human Amyloid-β-Induced Upregulation of Brain Proinflammatory Cytokine Production Attenuates Neurodegeneration

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Effects of acute ethanol exposure on class I HDACs family enzymes in wild-type and BDNF+/− mice

inferred← citeshttps://doi.org/10.1016/j.ejphar.2008.04.058openalex-snapshot

Clinically relevant concentrations of valproic acid modulate melatonin MT1 receptor, HDAC and MeCP2 mRNA expression in C6 glioma cells

inferred← citeshttps://doi.org/10.1016/j.ejphar.2009.10.018openalex-snapshot

Neurocognition in bipolar disorders—A closer look at comorbidities and medications

inferred← citeshttps://doi.org/10.1016/j.ejphar.2012.05.012openalex-snapshot

Zonisamide up-regulated the mRNAs encoding astrocytic anti-oxidative and neurotrophic factors

inferred← citeshttps://doi.org/10.1016/j.ejphar.2013.06.034openalex-snapshot

Intracellular pathways of antipsychotic combined therapies: Implication for psychiatric disorders treatment

inferred← citeshttps://doi.org/10.1016/j.eplepsyres.2014.02.005openalex-snapshot

Valproic acid in normal therapeutic concentration has no neuroprotective or differentiation influencing effects on long term expanded murine neural stem cells

inferred← citeshttps://doi.org/10.1016/j.expneurol.2011.11.042openalex-snapshot

Valproic acid improves locomotion in vivo after SCI and axonal growth of neurons in vitro

inferred← citeshttps://doi.org/10.1016/j.expneurol.2014.01.018openalex-snapshot

Hippocampal dysfunction during the chronic phase following a single exposure to cranial irradiation

inferred← citeshttps://doi.org/10.1016/j.expneurol.2015.11.003openalex-snapshot

Anti-high mobility group box 1 antibody exerts neuroprotection in a rat model of Parkinson's disease

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2011.12.006openalex-snapshot

Histone deacetylase inhibition activates transcription factor Nrf2 and protects against cerebral ischemic damage

inferred← citeshttps://doi.org/10.1016/j.gene.2023.147898openalex-snapshot

Astrocytic transcription factors REST, YY1, and putative microRNAs in Parkinson’s disease and advanced therapeutic strategies

inferred← citeshttps://doi.org/10.1016/j.heliyon.2019.e01951openalex-snapshot

Mechanism of methylation and acetylation of high GDNF transcription in glioma cells: A review

inferred← citeshttps://doi.org/10.1016/j.heliyon.2024.e26650openalex-snapshot

Differential effects of antiseizure medications on neurogenesis: Evidence from cells to animals

inferred← citeshttps://doi.org/10.1016/j.it.2008.05.002openalex-snapshot

Why neurodegenerative diseases are progressive: uncontrolled inflammation drives disease progression

inferred← citeshttps://doi.org/10.1016/j.jad.2012.01.024openalex-snapshot

The DRD2/ANKK1 gene is associated with response to add-on dextromethorphan treatment in bipolar disorder

inferred← citeshttps://doi.org/10.1016/j.jfma.2018.06.017openalex-snapshot

Valproate is protective against 6-OHDA-induced dopaminergic neurodegeneration in rodent midbrain: A potential role of BDNF up-regulation

inferred← citeshttps://doi.org/10.1016/j.jneuroim.2010.02.002openalex-snapshot

Strain-specific BDNF expression of rat primary astrocytes

inferred← citeshttps://doi.org/10.1016/j.mcn.2016.09.003openalex-snapshot

Mechanism for neurotropic action of vorinostat, a pan histone deacetylase inhibitor

inferred← citeshttps://doi.org/10.1016/j.nbd.2010.08.024openalex-snapshot

Valproic acid induces microglial dysfunction, not apoptosis, in human glial cultures

inferred← citeshttps://doi.org/10.1016/j.nbd.2011.02.011openalex-snapshot

Granulocyte macrophage-colony stimulating factor protects against substantia nigra dopaminergic cell loss in an environmental toxin model of Parkinson's disease

inferred← citeshttps://doi.org/10.1016/j.nbd.2011.06.016openalex-snapshot

Activated microglia decrease histone acetylation and Nrf2-inducible anti-oxidant defence in astrocytes: Restoring effects of inhibitors of HDACs, p38 MAPK and GSK3β

inferred← citeshttps://doi.org/10.1016/j.nbd.2012.04.014openalex-snapshot

Astrocyte-derived GDNF is a potent inhibitor of microglial activation

inferred← citeshttps://doi.org/10.1016/j.nbd.2012.11.018openalex-snapshot

Automated imaging system for fast quantitation of neurons, cell morphology and neurite morphometry in vivo and in vitro

inferred← citeshttps://doi.org/10.1016/j.neubiorev.2007.03.002openalex-snapshot

GSK-3 is a viable potential target for therapeutic intervention in bipolar disorder

inferred← citeshttps://doi.org/10.1016/j.neuint.2013.02.001openalex-snapshot

Effects of sodium butyrate on oxidative stress and behavioral changes induced by administration of d-AMPH

inferred← citeshttps://doi.org/10.1016/j.neuint.2014.01.008openalex-snapshot

Ventral tegmental area/substantia nigra and prefrontal cortex rodent organotypic brain slices as an integrated model to study the cellular changes induced by oxygen/glucose deprivation and reperfusion: Effect of neuroprotective agents

inferred← citeshttps://doi.org/10.1016/j.neuint.2021.105080openalex-snapshot

A1/A2 astrocytes in central nervous system injuries and diseases: Angels or devils?

inferred← citeshttps://doi.org/10.1016/j.neulet.2008.12.001openalex-snapshot

Effects of engrafted neural stem cells in Alzheimer's disease rats

inferred← citeshttps://doi.org/10.1016/j.neulet.2009.10.037openalex-snapshot

HDAC inhibitor trichostatin A-inhibited survival of dopaminergic neuronal cells

inferred← citeshttps://doi.org/10.1016/j.neulet.2010.04.013openalex-snapshot

Potent neuroprotective effects of novel structural derivatives of valproic acid: Potential roles of HDAC inhibition and HSP70 induction

inferred← citeshttps://doi.org/10.1016/j.neulet.2011.07.031openalex-snapshot

Circulating levels of GDNF in bipolar disorder

inferred← citeshttps://doi.org/10.1016/j.neulet.2012.08.015openalex-snapshot

Effects of valproic acid on the expression of trophic factors in human bone marrow mesenchymal stromal cells

inferred← citeshttps://doi.org/10.1016/j.neulet.2016.05.060openalex-snapshot

Histone H3 acetylation in the postmortem Parkinson’s disease primary motor cortex

inferred← citeshttps://doi.org/10.1016/j.neulet.2016.12.017openalex-snapshot

Valproic acid protects against MPP+-mediated neurotoxicity in SH-SY5Y Cells through autophagy

inferred← citeshttps://doi.org/10.1016/j.neulet.2017.12.037openalex-snapshot

Pathological histone acetylation in Parkinson’s disease: Neuroprotection and inhibition of microglial activation through SIRT 2 inhibition

inferred← citeshttps://doi.org/10.1016/j.neulet.2019.03.033openalex-snapshot

Enhancing the expression of neurotrophic factors in epidermal neural crest stem cells by valproic acid: A potential candidate for combinatorial treatment

inferred← citeshttps://doi.org/10.1016/j.neuro.2014.06.002openalex-snapshot

Existence of glia mitigated ketamine-induced neurotoxicity in neuron–glia mixed cultures of neonatal rat cortex and the glia-mediated protective effect of 2-PMPA

inferred← citeshttps://doi.org/10.1016/j.neuro.2017.11.003openalex-snapshot

Manganese chloride induces histone acetylation changes in neuronal cells: Its role in manganese-induced damage

inferred← citeshttps://doi.org/10.1016/j.neuro.2018.05.001openalex-snapshot

Valproic acid attenuates manganese-induced reduction in expression of GLT-1 and GLAST with concomitant changes in murine dopaminergic neurotoxicity

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2007.11.020openalex-snapshot

Inflammatory priming of the substantia nigra influences the impact of later paraquat exposure: Neuroimmune sensitization of neurodegeneration

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2009.09.010openalex-snapshot

Urocortin modulates dopaminergic neuronal survival via inhibition of glycogen synthase kinase-3β and histone deacetylase

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2015.10.001openalex-snapshot

A novel histone deacetylase 1 and 2 isoform-specific inhibitor alleviates experimental Parkinson's disease

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2018.02.018openalex-snapshot

Upregulation of histone deacetylase 2 in laser capture nigral microglia in Parkinson's disease

inferred← citeshttps://doi.org/10.1016/j.neuroimage.2009.11.025openalex-snapshot

Amygdala volume in depressed patients with bipolar disorder assessed using high resolution 3T MRI: The impact of medication

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2009.11.003openalex-snapshot

The mechanism of heme oxygenase-1 action involved in the enhancement of neurotrophic factor expression

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2010.09.022openalex-snapshot

Histone deacetylase inhibition alters histone methylation associated with heat shock protein 70 promoter modifications in astrocytes and neurons

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2013.07.015openalex-snapshot

Differential regulation of Bdnf expression in cortical neurons by class-selective histone deacetylase inhibitors

inferred← citeshttps://doi.org/10.1016/j.neuropharm.2019.107919openalex-snapshot

Anti-neuroinflammatory, protective effects of the synthetic microneurotrophin BNN-20 in the advanced dopaminergic neurodegeneration of “weaver” mice

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2007.06.053openalex-snapshot

Valproic acid and other histone deacetylase inhibitors induce microglial apoptosis and attenuate lipopolysaccharide-induced dopaminergic neurotoxicity

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2010.04.041openalex-snapshot

Valproic acid enhances microglial phagocytosis of amyloid-β1–42

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2011.01.012openalex-snapshot

Valproate reduces CHOP levels and preserves oligodendrocytes and axons after spinal cord injury

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2011.08.010openalex-snapshot

Protective effects of valproic acid on the nigrostriatal dopamine system in a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine mouse model of Parkinson's disease

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2014.02.028openalex-snapshot

Neuroprotection by valproic acid in an intrastriatal rotenone model of Parkinson’s disease

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2015.05.018openalex-snapshot

The role of inflammation and microglial activation in the pathophysiology of psychiatric disorders

inferred← citeshttps://doi.org/10.1016/j.nurt.2010.07.003openalex-snapshot

Reactive Astrocytes As Therapeutic Targets for CNS Disorders

inferred← citeshttps://doi.org/10.1016/j.pharmthera.2013.05.010openalex-snapshot

Epigenetic targeting of histone deacetylase: Therapeutic potential in Parkinson's disease?

inferred← citeshttps://doi.org/10.1016/j.pharmthera.2016.07.001openalex-snapshot

Modulating epigenetic HAT activity for reinstating acetylation homeostasis: A promising therapeutic strategy for neurological disorders

inferred← citeshttps://doi.org/10.1016/j.phrs.2010.01.011openalex-snapshot

HDAC inhibitors and neurodegeneration: At the edge between protection and damage

inferred← citeshttps://doi.org/10.1016/j.phrs.2015.11.024openalex-snapshot

Pharmacological modulation of HDAC1 and HDAC6 in vivo in a zebrafish model: Therapeutic implications for Parkinson’s disease

inferred← citeshttps://doi.org/10.1016/j.phrs.2015.12.033openalex-snapshot

Long-term effects of early life stress exposure: Role of epigenetic mechanisms

inferred← citeshttps://doi.org/10.1016/j.pmip.2016.11.003openalex-snapshot

Putative biological predictors of treatment response in bipolar disorders

inferred← citeshttps://doi.org/10.1016/j.pneurobio.2008.09.006openalex-snapshot

Driving GDNF expression: The green and the red traffic lights

inferred← citeshttps://doi.org/10.1016/j.pneurobio.2009.08.001openalex-snapshot

The influence of microglia on the pathogenesis of Parkinson's disease

inferred← citeshttps://doi.org/10.1016/j.pneurobio.2015.05.002openalex-snapshot

The epigenetics of aging and neurodegeneration

inferred← citeshttps://doi.org/10.1016/j.pnpbp.2008.03.022openalex-snapshot

Valproate-induced parkinsonism, glial cells and Alexander's disease

inferred← citeshttps://doi.org/10.1016/j.pnpbp.2012.01.017openalex-snapshot

Valproic acid mediates the synaptic excitatory/inhibitory balance through astrocytes — A preliminary study

inferred← citeshttps://doi.org/10.1016/j.pnpbp.2016.09.002openalex-snapshot

Histone deacetylases inhibitors (HDACis) as novel therapeutic application in various clinical diseases

inferred← citeshttps://doi.org/10.1016/j.psyneuen.2014.05.011openalex-snapshot

Elevated levels of plasma brain derived neurotrophic factor in rapid cycling bipolar disorder patients

inferred← citeshttps://doi.org/10.1016/j.semcancer.2018.07.005openalex-snapshot

The reversible effects of glial cell line–derived neurotrophic factor (GDNF) in the human brain

inferred← citeshttps://doi.org/10.1016/j.tice.2019.01.002openalex-snapshot

Modeling traumatic injury in organotypic spinal cord slice culture obtained from adult rat

inferred← citeshttps://doi.org/10.1016/j.tins.2009.06.002openalex-snapshot

Multiple roles of HDAC inhibition in neurodegenerative conditions

inferred← citeshttps://doi.org/10.1016/j.vascn.2012.08.001openalex-snapshot

Epigenetic histone acetylation and deacetylation mechanisms in experimental models of neurodegenerative disorders

inferred← citeshttps://doi.org/10.1016/j.wneu.2023.10.135openalex-snapshot

Effects of Valproic Acid Therapy on Rats with Spinal Cord Injury: A Systematic Review and Meta-Analysis

inferred← citeshttps://doi.org/10.1016/j.ygcen.2016.02.011openalex-snapshot

Role of aromatase and radial glial cells in neurotoxin-induced dopamine neuron degeneration and regeneration

inferred← citeshttps://doi.org/10.1016/s1734-1140(11)70723-xopenalex-snapshot

Kynurenic acid: a new effector of valproate action?

inferred← citeshttps://doi.org/10.1017/9781911623465.014openalex-snapshot

Anticonvulsants for Mental Disorders: Valproate, Lamotrigine, Carbamazepine and Oxcarbazepine

inferred← citeshttps://doi.org/10.1017/neu.2016.56openalex-snapshot

Glial cells as key elements in the pathophysiology and treatment of bipolar disorder

inferred← citeshttps://doi.org/10.1017/s1461145708009024openalex-snapshot

Histone deacetylase inhibitors up-regulate astrocyte GDNF and BDNF gene transcription and protect dopaminergic neurons

inferred← citeshttps://doi.org/10.1021/acsomega.9b01950openalex-snapshot

Crafting Carbazole-Based Vorinostat and Tubastatin-A-like Histone Deacetylase (HDAC) Inhibitors with Potent in Vitro and in Vivo Neuroactive Functions

inferred← citeshttps://doi.org/10.1021/cn500117aopenalex-snapshot

Modulation of Neurogenesis by Targeting Epigenetic Enzymes Using Small Molecules: An Overview

inferred← citeshttps://doi.org/10.1021/jf903294xopenalex-snapshot

The Plant Phenolic Diterpene Carnosol Suppresses Sodium Nitroprusside-Induced Toxicity in C6 Glial Cells

inferred← citeshttps://doi.org/10.1038/aps.2011.140openalex-snapshot

Beneficial effects of mood stabilizers lithium, valproate and lamotrigine in experimental stroke models

inferred← citeshttps://doi.org/10.1038/ncomms6627openalex-snapshot

Activated astrocytes enhance the dopaminergic differentiation of stem cells and promote brain repair through bFGF

inferred← citeshttps://doi.org/10.1038/npp.2008.15openalex-snapshot

Drug-Induced Activation of Dopamine D1 Receptor Signaling and Inhibition of Class I/II Histone Deacetylase Induce Chromatin Remodeling in Reward Circuitry and Modulate Cocaine-Related Behaviors

inferred← citeshttps://doi.org/10.1038/npp.2009.188openalex-snapshot

Valproate and Amitriptyline Exert Common and Divergent Influences on Global and Gene Promoter-Specific Chromatin Modifications in Rat Primary Astrocytes

inferred← citeshttps://doi.org/10.1038/npp.2009.64openalex-snapshot

Novel Neuroprotective Mechanisms of Memantine: Increase in Neurotrophic Factor Release from Astroglia and Anti-Inflammation by Preventing Microglial Activation

inferred← citeshttps://doi.org/10.1038/s41392-024-01918-wopenalex-snapshot

Crossing epigenetic frontiers: the intersection of novel histone modifications and diseases

inferred← citeshttps://doi.org/10.1038/s41397-021-00215-xopenalex-snapshot

Transcriptional analysis of sodium valproate in a serotonergic cell line reveals gene regulation through both HDAC inhibition-dependent and independent mechanisms

inferred← citeshttps://doi.org/10.1038/s41401-020-00536-wopenalex-snapshot

Low corticosterone levels attenuate late life depression and enhance glutamatergic neurotransmission in female rats

inferred← citeshttps://doi.org/10.1038/s41598-022-12937-4openalex-snapshot

Histone deacetylase 2 inhibitor valproic acid attenuates bisphenol A-induced liver pathology in male mice

inferred← citeshttps://doi.org/10.1038/tp.2013.119openalex-snapshot

Glia and immune cell signaling in bipolar disorder: insights from neuropharmacology and molecular imaging to clinical application

inferred← citeshttps://doi.org/10.1039/9781782622888-00027openalex-snapshot

Oxidative Stress and Redox Signalling in the Parkinson’s Disease Brain

inferred← citeshttps://doi.org/10.1055/s-0034-1382607openalex-snapshot

Evaluation of inhibitors of histone deacetylases as potential neurotrophic agents

inferred← citeshttps://doi.org/10.1089/scd.2014.0218openalex-snapshot

Epigenetic Activation of the Foxa2 Gene Is Required for Maintaining the Potential of Neural Precursor Cells to Differentiate into Dopaminergic Neurons After Expansion

inferred← citeshttps://doi.org/10.1093/abbs/gmw085openalex-snapshot

Gender difference in valproic acid-induced neuroprotective effects on APP/PS1 double transgenic mice modeling Alzheimer's disease

inferred← citeshttps://doi.org/10.1093/biohorizons/hzs003openalex-snapshot

Novel histone deacetylase (HDAC) inhibitors with improved selectivity for HDAC2 and 3 protect against neural cell death

inferred← citeshttps://doi.org/10.1093/jnen/nly073openalex-snapshot

Histone Deacetylase Inhibitors: A Therapeutic Key in Neurological Disorders?

inferred← citeshttps://doi.org/10.1096/fj.202300963rropenalex-snapshot

Treatment with sodium butyrate induces autophagy resulting in therapeutic benefits for spinocerebellar ataxia type 3

inferred← citeshttps://doi.org/10.1097/fbp.0b013e32836546fcopenalex-snapshot

Effects of sodium butyrate in animal models of mania and depression

inferred← citeshttps://doi.org/10.1097/fpc.0b013e32833caa2bopenalex-snapshot

The ALDH2 and DRD2/ANKK1 genes interacted in bipolar II but not bipolar I disorder

inferred← citeshttps://doi.org/10.1097/ta.0b013e318166b822openalex-snapshot

Prevention of Hypoxia-Induced Neuronal Apoptosis Through Histone Deacetylase Inhibition

inferred← citeshttps://doi.org/10.1097/wnr.0000000000000729openalex-snapshot

Spatiotemporal expression of Ski after rat spinal cord injury

inferred← citeshttps://doi.org/10.1097/wnr.0000000000000911openalex-snapshot

Quetiapine prevents Aβ25–35-induced cell death in cultured neuron by enhancing brain-derived neurotrophic factor release from astrocyte

inferred← citeshttps://doi.org/10.1097/wnr.0b013e328308b355openalex-snapshot

Enhancement of glutamate uptake in 1-methyl-4-phenylpyridinium-treated astrocytes by trichostatin A

inferred← citeshttps://doi.org/10.1111/acel.70382openalex-snapshot

Astrocyte Senescence Impairs Synaptogenesis due to Thrombospondin‐1 Loss

inferred← citeshttps://doi.org/10.1111/bcpt.12201openalex-snapshot

Induction of Neurotrophic and Differentiation Factors in Neural Stem Cells by Valproic Acid

inferred← citeshttps://doi.org/10.1111/bcpt.12915openalex-snapshot

Valproate Plays a Protective Role against Migraine by Inhibiting Protein Kinase C Signalling in Nitroglycerin‐treated Mice

inferred← citeshttps://doi.org/10.1111/bdi.12135openalex-snapshot

White matter alterations in bipolar disorder: potential for drug discovery and development

inferred← citeshttps://doi.org/10.1111/bdi.12399openalex-snapshot

Brain‐derived neurotrophic factor, impaired glucose metabolism, and bipolar disorder course

inferred← citeshttps://doi.org/10.1111/bdi.12418openalex-snapshot

Inter‐relation between brain‐derived neurotrophic factor and antioxidant enzymes in bipolar disorder

inferred← citeshttps://doi.org/10.1111/bdi.70131openalex-snapshot

White Matter Changes During Manic and Depressive Episodes and Phase Switching in Bipolar Disorder: A Tractography Study

inferred← citeshttps://doi.org/10.1111/bph.13208openalex-snapshot

Neurorestoration induced by the HDAC inhibitor sodium valproate in the lactacystin model of P arkinson's is associated with histone acetylation and up‐regulation of neurotrophic factors

inferred← citeshttps://doi.org/10.1111/cns.12390openalex-snapshot

Glial Pathology in Bipolar Disorder: Potential Therapeutic Implications

inferred← citeshttps://doi.org/10.1111/cns.12445openalex-snapshot

Valproic Acid Modifies Synaptic Structure and Accelerates Neurite Outgrowth Via the Glycogen Synthase Kinase‐3β Signaling Pathway in an Alzheimer's Disease Model

inferred← citeshttps://doi.org/10.1111/cns.14473openalex-snapshot

Neuroprotective effects of gemfibrozil in neurological disorders: Focus on inflammation and molecular mechanisms

inferred← citeshttps://doi.org/10.1111/ejn.13482openalex-snapshot

l ‐DOPA‐induced dyskinesia and neuroinflammation: do microglia and astrocytes play a role?

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