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Role of voltage-gated L-type Ca2+ channel isoforms for brain function

https://doi.org/10.1042/bst0340903
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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/s00210-004-0940-7openalex-snapshot
Disturbed atrio-ventricular conduction and normal contractile function in isolated hearts from Ca v 1.3-knockout mice
inferredcites →https://doi.org/10.1007/s10162-003-4020-3openalex-snapshot
Null Mutation of ?1D Ca2+ Channel Gene Results in Deafness but No Vestibular Defect in Mice
inferredcites →https://doi.org/10.1016/0014-2999(88)90813-8openalex-snapshot
BAY K 8644-enhances immobility in the mouse behavioral despair test, an effect blocked by nifedipine
inferredcites →https://doi.org/10.1016/0896-6273(93)90332-lopenalex-snapshot
Functional diversity of L-type calcium channels in rat cerebellar neurons
inferredcites →https://doi.org/10.1016/j.neuropharm.2006.02.010openalex-snapshot
L-type calcium channel ligands block nicotine-induced signaling to CREB by inhibiting nicotinic receptors
inferredcites →https://doi.org/10.1016/j.neuroscience.2006.01.059openalex-snapshot
Brain activation pattern induced by stimulation of L-type Ca2+-channels: Contribution of CaV1.3 and CaV1.2 isoforms
inferredcites →https://doi.org/10.1016/s0006-3495(01)75964-4openalex-snapshot
Fast Exocytosis with Few Ca2+ Channels in Insulin-Secreting Mouse Pancreatic B Cells
inferredcites →https://doi.org/10.1016/s0006-8993(00)02868-7openalex-snapshot
L-type calcium channels modulate glutamate-driven bursting activity in the nucleus accumbens in vivo
inferredcites →https://doi.org/10.1016/s0092-8674(00)00013-1openalex-snapshot
Congenital Deafness and Sinoatrial Node Dysfunction in Mice Lacking Class D L-Type Ca2+ Channels
inferredcites →https://doi.org/10.1016/s0165-6147(98)01171-7openalex-snapshot
Structural basis of drug binding to L Ca2+ channels
inferredcites →https://doi.org/10.1016/s0169-328x(02)00643-5openalex-snapshot
Age-related working memory impairment is correlated with increases in the L-type calcium channel protein α1D (Cav1.3) in area CA1 of the hippocampus and both are ameliorated by chronic nimodipine treatment
inferredcites →https://doi.org/10.1016/s0306-4522(03)00329-4openalex-snapshot
Neurological phenotype and synaptic function in mice lacking the CaV1.3 α subunit of neuronal L-type voltage-dependent Ca2+ channels
inferredcites →https://doi.org/10.1016/s0378-5955(03)00054-6openalex-snapshot
Role of class D L-type Ca2+ channels for cochlear morphology
inferredcites →https://doi.org/10.1016/s0959-4388(03)00076-xopenalex-snapshot
Signaling from synapse to nucleus: the logic behind the mechanisms
inferredcites →https://doi.org/10.1038/87865openalex-snapshot
The cellular neurobiology of depression
inferredcites →https://doi.org/10.1073/pnas.0935295100openalex-snapshot
Functional role of L-type Ca v 1.3 Ca 2+ channels in cardiac pacemaker activity
inferredcites →https://doi.org/10.1073/pnas.96.18.10164openalex-snapshot
Syntaxin 1 interacts with the L D subtype of voltage-gated Ca 2+ channels in pancreatic β cells
inferredcites →https://doi.org/10.1073/pnas.96.26.15228openalex-snapshot
Calcium channel activation and self-biting in mice
inferredcites →https://doi.org/10.1074/jbc.m006467200openalex-snapshot
Functional Embryonic Cardiomyocytes after Disruption of the L-type α1C (Ca 1.2) Calcium Channel Gene in the Mouse
inferredcites →https://doi.org/10.1074/jbc.m101469200openalex-snapshot
α1D (Cav1.3) Subunits Can Form L-type Ca2+ Channels Activating at Negative Voltages
inferredcites →https://doi.org/10.1074/jbc.m103724200openalex-snapshot
Functional Properties of CaV1.3 (α1D) L-type Ca2+ Channel Splice Variants Expressed by Rat Brain and Neuroendocrine GH3 Cells
inferredcites →https://doi.org/10.1074/jbc.m307598200openalex-snapshot
Enhanced Expression of L-type Cav1.3 Calcium Channels in Murine Embryonic Hearts from Cav1.2-deficient Mice
inferredcites →https://doi.org/10.1074/jbc.m409008200openalex-snapshot
Opposite Effects of a Single IIIS5 Mutation on Phenylalkylamine and Dihydropyridine Interaction with L-type Ca2+ Channels
inferredcites →https://doi.org/10.1085/jgp.113.5.679openalex-snapshot
Anomalous L-Type Calcium Channels of Rat Spinal Motoneurons
inferredcites →https://doi.org/10.1093/emboj/cdg389openalex-snapshot
Impaired insulin secretion and glucose tolerance in β cell‐selective CaV1.2 Ca2+ channel null mice
inferredcites →https://doi.org/10.1093/emboj/cdg583openalex-snapshot
Dominant role of smooth muscle L‐type calcium channel Cav1.2 for blood pressure regulation
inferredcites →https://doi.org/10.1096/fj.05-5292fjeopenalex-snapshot
Control of intestinal motility by the Cav1.2 L‐type calcium channel in mice
inferredcites →https://doi.org/10.1111/j.1460-9568.2005.04177.xopenalex-snapshot
Distribution and regulation of L‐type calcium channels in deep dorsal horn neurons after sciatic nerve injury in rats
inferredcites →https://doi.org/10.1111/j.1460-9568.2006.04734.xopenalex-snapshot
CaV1.2 and CaV1.3 neuronal L‐type calcium channels: differential targeting and signaling to pCREB
inferredcites →https://doi.org/10.1111/j.1471-4159.2005.03318.xopenalex-snapshot
Androgens activate mitogen‐activated protein kinase signaling: Role in neuroprotection
inferredcites →https://doi.org/10.1111/j.1472-8206.2006.00393.xopenalex-snapshot
Nifedipine inhibits picrotoxin‐induced seizure activity: further evidence on the involvement of L‐type calcium channel blockers in epilepsy
inferredcites →https://doi.org/10.1113/jphysiol.2003.053256openalex-snapshot
Cav1.3 (α1D) Ca2+ Currents in Neonatal Outer Hair Cells of Mice
inferredcites →https://doi.org/10.1124/pr.57.4.4openalex-snapshot
International Union of Pharmacology. XLVII. Nomenclature and Structure-Function Relationships of Voltage-Gated Sodium Channels
inferredcites →https://doi.org/10.1124/pr.57.4.5openalex-snapshot
International Union of Pharmacology. XLVIII. Nomenclature and Structure-Function Relationships of Voltage-Gated Calcium Channels
inferredcites →https://doi.org/10.1152/jn.00788.2003openalex-snapshot
Role of Persistent Sodium and Calcium Currents in Motoneuron Firing and Spasticity in Chronic Spinal Rats
inferredcites →https://doi.org/10.1152/jn.1998.79.6.2847openalex-snapshot
Ionic Basis for Serotonin-Induced Bistable Membrane Properties in Guinea Pig Trigeminal Motoneurons
inferredcites →https://doi.org/10.1161/01.res.0000018003.14304.e2openalex-snapshot
Functional Roles of Ca v 1.3 (α 1D ) Calcium Channel in Sinoatrial Nodes
inferredcites →https://doi.org/10.1161/01.res.73.5.974openalex-snapshot
Subunit-dependent modulation of recombinant L-type calcium channels. Molecular basis for dihydropyridine tissue selectivity.
inferredcites →https://doi.org/10.1172/jci13310openalex-snapshot
Requirement for the L-type Ca2+ channel α1D subunit in postnatal pancreatic β cell generation
inferredcites →https://doi.org/10.1172/jci200113310openalex-snapshot
Requirement for the L-type Ca2+ channel α1D subunit in postnatal pancreatic β cell generation
inferredcites →https://doi.org/10.1172/jci200420208openalex-snapshot
Isoform-specific regulation of mood behavior and pancreatic β cell and cardiovascular function by L-type Ca2+ channels
inferredcites →https://doi.org/10.1177/1073858405278236openalex-snapshot
Psychostimulants, L-type Calcium Channels, Kinases, and Phosphatases
inferredcites →https://doi.org/10.1523/jneurosci.0481-06.2006openalex-snapshot
Insulin-Like Growth Factor-1 Modulation of CaV1.3 Calcium Channels Depends on Ca2+ Release from IP3-Sensitive Stores and Calcium/Calmodulin Kinase II Phosphorylation of the 1 Subunit EF Hand
inferredcites →https://doi.org/10.1523/jneurosci.1089-05.2005openalex-snapshot
Neuronal L-Type Calcium Channels Open Quickly and Are Inhibited Slowly
inferredcites →https://doi.org/10.1523/jneurosci.1531-05.2005openalex-snapshot
Role of Hippocampal Cav1.2 Ca2+Channels in NMDA Receptor-Independent Synaptic Plasticity and Spatial Memory
inferredcites →https://doi.org/10.1523/jneurosci.16-18-05567.1996openalex-snapshot
Multiple Channel Types Contribute to the Low-Voltage-Activated Calcium Current in Hippocampal CA3 Pyramidal Neurons
inferredcites →https://doi.org/10.1523/jneurosci.20-13-04786.2000openalex-snapshot
Sustained Activation of Hippocampal Lp-Type Voltage-Gated Calcium Channels by Tetanic Stimulation
inferredcites →https://doi.org/10.1523/jneurosci.21-16-05944.2001openalex-snapshot
Neuronal Ca(V)1.3alpha(1) L-type channels activate at relatively hyperpolarized membrane potentials and are incompletely inhibited by dihydropyridines.
inferredcites →https://doi.org/10.1523/jneurosci.21-20-j0006.2001openalex-snapshot
Somatic Colocalization of Rat SK1 and D class (Cav 1.2) L-type Calcium Channels in Rat CA1 Hippocampal Pyramidal Neurons
inferredcites →https://doi.org/10.1523/jneurosci.22-20-09113.2002openalex-snapshot
L-Type Voltage-Gated Calcium Channels Are Required for Extinction, But Not for Acquisition or Expression, of Conditional Fear in Mice
inferredcites →https://doi.org/10.1523/jneurosci.23-34-10832.2003openalex-snapshot
CaV1.3 Channels Are Essential for Development and Presynaptic Activity of Cochlear Inner Hair Cells
inferredcites →https://doi.org/10.1523/jneurosci.2734-05.2005openalex-snapshot
Control of Spontaneous Firing Patterns by the Selective Coupling of Calcium Currents to Calcium-Activated Potassium Currents in Striatal Cholinergic Interneurons
inferredcites →https://doi.org/10.1523/jneurosci.3327-04.2005openalex-snapshot
G-Protein-Coupled Receptor Modulation of Striatal CaV1.3 L-Type Ca2+Channels Is Dependent on a Shank-Binding Domain
inferredcites →https://doi.org/10.4049/jimmunol.172.9.5206openalex-snapshot
Dihydropyridine Receptors Are Selective Markers of Th2 Cells and Can Be Targeted to Prevent Th2-Dependent Immunopathological Disorders

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Association of Hydroxylmethyl Glutaryl Coenzyme A Reductase Inhibitors, L-Type Calcium Channel Antagonists, and Biguanides With Rates of Psychiatric Hospitalization and Self-Harm in Individuals With Serious Mental Illness
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Calmodulin Interactions with C av1 and C av2 Voltage‐Gated Calcium Channel IQ Domains
inferred← citeshttps://doi.org/10.1002/9780470429907.ch1openalex-snapshot
Neuronal L‐Type Voltage‐Gated Calcium Channels
inferred← citeshttps://doi.org/10.1002/9781119951438.eibc0697openalex-snapshot
Calmodulin Interactions with C av1 and C av2 Voltage‐Gated Calcium Channel IQ Domains
inferred← citeshttps://doi.org/10.1002/ajmg.b.31122openalex-snapshot
L‐type calcium channels and psychiatric disorders: A brief review
inferred← citeshttps://doi.org/10.1002/ajmg.b.32348openalex-snapshot
Genetic analysis of SNPs in CACNA1C and ANK3 gene with schizophrenia: A comprehensive meta‐analysis
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Common variants in CACNA1C and MDD susceptibility: A comprehensive meta‐analysis
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What causes cell death in Parkinson's disease?
inferred← citeshttps://doi.org/10.1002/glia.23723openalex-snapshot
The voltage‐gated calcium channel CaV1.2 promotes adult oligodendrocyte progenitor cell survival in the mouse corpus callosum but not motor cortex
inferred← citeshttps://doi.org/10.1002/hipo.22811openalex-snapshot
Type‐1 astrocyte‐like stem cells harboring Cacna1d gene deletion exhibit reduced proliferation and decreased neuronal fate choice
inferred← citeshttps://doi.org/10.1002/jnr.22222openalex-snapshot
p38 mitogen‐activated protein kinase and calcium channels mediate signaling in depolarization‐induced activation of peroxisome proliferator‐activated receptor gamma coactivator‐1α in neurons
inferred← citeshttps://doi.org/10.1002/jnr.24508openalex-snapshot
Dementia associated with disorders of the basal ganglia
inferred← citeshttps://doi.org/10.1002/mds.22801openalex-snapshot
A molecular basis for the increased vulnerability of substantia nigra dopamine neurons in aging and Parkinson's disease
inferred← citeshttps://doi.org/10.1002/wmts.38openalex-snapshot
Calcium channels as therapeutic targets
inferred← citeshttps://doi.org/10.1007/164_2022_629openalex-snapshot
The Role of Striatal Cav1.3 Calcium Channels in Therapeutics for Parkinson’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-031-08881-0_21openalex-snapshot
Novel Insights into the Role of Voltage-Gated Calcium Channel Genes in Psychiatric Disorders
inferred← citeshttps://doi.org/10.1007/978-3-319-28637-2_6openalex-snapshot
Life on the Edge: Determinants of Selective Neuronal Vulnerability in Parkinson’s Disease
inferred← citeshttps://doi.org/10.1007/s00109-026-02700-xopenalex-snapshot
Non-transferrin-bound iron uptake by cardiomyocytes
inferred← citeshttps://doi.org/10.1007/s00424-010-0800-xopenalex-snapshot
Channelopathies in Cav1.1, Cav1.3, and Cav1.4 voltage-gated L-type Ca2+ channels
inferred← citeshttps://doi.org/10.1007/s00424-013-1314-0openalex-snapshot
G protein modulation of K2P potassium channel TASK-2
inferred← citeshttps://doi.org/10.1007/s00424-014-1627-7openalex-snapshot
TASK-2 K2P K+ channel: thoughts about gating and its fitness to physiological function
inferred← citeshttps://doi.org/10.1007/s00441-014-1936-3openalex-snapshot
The role of L-type voltage-gated calcium channels Cav1.2 and Cav1.3 in normal and pathological brain function
inferred← citeshttps://doi.org/10.1007/s00702-015-1398-0openalex-snapshot
Rasagiline and selegiline suppress calcium efflux from mitochondria by PK11195-induced opening of mitochondrial permeability transition pore: a novel anti-apoptotic function for neuroprotection
inferred← citeshttps://doi.org/10.1007/s10072-017-2963-0openalex-snapshot
Role of CACNA1C gene polymorphisms and protein expressions in the pathogenesis of schizophrenia: a case-control study in a Chinese population
inferred← citeshttps://doi.org/10.1007/s12017-013-8234-1openalex-snapshot
Raised Activity of L-Type Calcium Channels Renders Neurons Prone to Form Paroxysmal Depolarization Shifts
inferred← citeshttps://doi.org/10.1007/s12031-016-0803-yopenalex-snapshot
Regulation and the Mechanism of Estrogen on Cav1.2 Gene in Rat-Cultured Cortical Astrocytes
inferred← citeshttps://doi.org/10.1007/s12640-011-9271-xopenalex-snapshot
L-Type Cav1.2 Calcium Channel is Involved in 6-Hydroxydopamine-Induced Neurotoxicity in Rats
inferred← citeshttps://doi.org/10.1016/b978-0-12-802206-1.00041-6openalex-snapshot
Determinants of Selective Vulnerability of Dopamine Neurons in Parkinson's Disease
inferred← citeshttps://doi.org/10.1016/b978-1-4160-6641-5.00010-6openalex-snapshot
Pathogenesis of Parkinson's Disease
inferred← citeshttps://doi.org/10.1016/j.arr.2023.101899openalex-snapshot
Inhibition of ferroptosis through regulating neuronal calcium homeostasis: An emerging therapeutic target for Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.bbi.2025.07.004openalex-snapshot
The role of the risk gene CACNA1C in neuroinflammation and peripheral immunity in autism spectrum disorder
inferred← citeshttps://doi.org/10.1016/j.bbr.2019.112119openalex-snapshot
Influence of magnesium supplementation and L-type calcium channel blocker on haloperidol-induced movement disturbances
inferred← citeshttps://doi.org/10.1016/j.bbrc.2016.08.168openalex-snapshot
Calcium and Parkinson's disease
inferred← citeshttps://doi.org/10.1016/j.bcp.2022.115071openalex-snapshot
Potential role of IP3/Ca2+ signaling and phosphodiesterases: Relevance to neurodegeneration in Alzheimer’s disease and possible therapeutic strategies
inferred← citeshttps://doi.org/10.1016/j.bmc.2010.03.038openalex-snapshot
Antagonism of 4-substituted 1,4-dihydropyridine-3,5-dicarboxylates toward voltage-dependent L-type Ca2+ channels CaV1.3 and CaV1.2
inferred← citeshttps://doi.org/10.1016/j.bmc.2013.04.054openalex-snapshot
Antagonism of L-type Ca2+ channels CaV1.3 and CaV1.2 by 1,4-dihydropyrimidines and 4H-pyrans as dihydropyridine mimics
inferred← citeshttps://doi.org/10.1016/j.brainres.2014.07.014openalex-snapshot
Nifedipine and nimodipine protect dopaminergic substantia nigra neurons against axotomy-induced cell death in rat vibrosections via modulating inflammatory responses
inferred← citeshttps://doi.org/10.1016/j.ceca.2007.04.015openalex-snapshot
L-type calcium channels in adrenal chromaffin cells: Role in pace-making and secretion
inferred← citeshttps://doi.org/10.1016/j.ceca.2009.12.003openalex-snapshot
Calcium, cellular aging, and selective neuronal vulnerability in Parkinson's disease
inferred← citeshttps://doi.org/10.1016/j.ceca.2015.09.007openalex-snapshot
The L-type calcium channel Cav1.3 is required for proper hippocampal neurogenesis and cognitive functions
inferred← citeshttps://doi.org/10.1016/j.ejphar.2013.06.010openalex-snapshot
Implicating the role of plasma membrane localized calcium channels and exchangers in stress-induced deleterious effects
inferred← citeshttps://doi.org/10.1016/j.febslet.2010.07.014openalex-snapshot
Direct interaction and functional coupling between voltage‐gated CaV1.3 Ca2+ channel and GABAB receptor subunit 2
inferred← citeshttps://doi.org/10.1016/j.jbc.2021.100502openalex-snapshot
The molecular basis of the inhibition of CaV1 calcium-dependent inactivation by the distal carboxy tail
inferred← citeshttps://doi.org/10.1016/j.jchemneu.2022.102170openalex-snapshot
Neuroprotective Effects of Aripiprazole in Stress-Induced Depressive-Like Behavior: Possible Role of Cacna1c
inferred← citeshttps://doi.org/10.1016/j.jpsychires.2014.02.003openalex-snapshot
Genetic variation in CACNA1C affects neural processing in major depression
inferred← citeshttps://doi.org/10.1016/j.neulet.2012.02.014openalex-snapshot
GABAB receptor-mediated ERK1/2 phosphorylation via a direct interaction with CaV1.3 channels
inferred← citeshttps://doi.org/10.1016/j.neulet.2012.03.043openalex-snapshot
Electroconvulsive stimulations prevent chronic stress-induced increases in L-type calcium channel mRNAs in the hippocampus and basolateral amygdala
inferred← citeshttps://doi.org/10.1016/j.neuro.2024.06.016openalex-snapshot
Selective dopaminergic neurotoxicity modulated by inherent cell-type specific neurobiology
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2014.12.027openalex-snapshot
L-type calcium channel blockers and substance P induce angiogenesis of cortical vessels associated with beta-amyloid plaques in an Alzheimer mouse model
inferred← citeshttps://doi.org/10.1016/j.neuroimage.2009.09.028openalex-snapshot
Effect of CACNA1C rs1006737 on neural correlates of verbal fluency in healthy individuals
inferred← citeshttps://doi.org/10.1016/j.neuroimage.2013.11.030openalex-snapshot
The impact of a CACNA1C gene polymorphism on learning and hippocampal formation in healthy individuals: A diffusion tensor imaging study
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Parkinson's Disease: Return of an Old Prime Suspect
inferred← citeshttps://doi.org/10.1016/j.neuron.2008.09.005openalex-snapshot
Calcium Channel Regulation and Presynaptic Plasticity
inferred← citeshttps://doi.org/10.1016/j.neuron.2009.01.033openalex-snapshot
Interplay between Cytosolic Dopamine, Calcium, and α-Synuclein Causes Selective Death of Substantia Nigra Neurons
inferred← citeshttps://doi.org/10.1016/j.neuron.2011.11.022openalex-snapshot
RNA Editing of the IQ Domain in Cav1.3 Channels Modulates Their Ca2+-Dependent Inactivation
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2014.03.021openalex-snapshot
Psychostimulants, antidepressants and neurokinin-1 receptor antagonists (‘motor disinhibitors’) have overlapping, but distinct, effects on monoamine transmission: The involvement of L-type Ca2+ channels and implications for the treatment of ADHD
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2008.01.077openalex-snapshot
Connexin31.1 deficiency in the mouse impairs object memory and modulates open-field exploration, acetylcholine esterase levels in the striatum, and cAMP response element-binding protein levels in the striatum and piriform cortex
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2009.08.006openalex-snapshot
Subcellular distribution of L-type calcium channel subtypes in rat hippocampal neurons
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2011.05.042openalex-snapshot
Ethanol alters calcium signaling in axonal growth cones
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2011.08.045openalex-snapshot
The role of calcium and mitochondrial oxidant stress in the loss of substantia nigra pars compacta dopaminergic neurons in Parkinson's disease
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2014.10.037openalex-snapshot
Dopamine midbrain neurons in health and Parkinson’s disease: Emerging roles of voltage-gated calcium channels and ATP-sensitive potassium channels
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2019.09.041openalex-snapshot
Calcium Signaling in Neurons and Glial Cells: Role of Cav1 channels
inferred← citeshttps://doi.org/10.1016/j.nicl.2019.101774openalex-snapshot
CACNA1C risk variant affects microstructural connectivity of the amygdala
inferred← citeshttps://doi.org/10.1016/j.nlm.2017.08.011openalex-snapshot
L-type VDCCs participate in behavioral-LTP and memory retention
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2011.10.001openalex-snapshot
Genetic approaches to a better understanding of bipolar disorder
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2014.12.004openalex-snapshot
Pharmacology of cognitive enhancers for exposure-based therapy of fear, anxiety and trauma-related disorders
inferred← citeshttps://doi.org/10.1016/j.pneurobio.2011.09.010openalex-snapshot
Quantitative aspects of L-type Ca2+ currents
inferred← citeshttps://doi.org/10.1016/j.pnpbp.2017.02.004openalex-snapshot
Ca v 1.2, but not Ca v 1.3, L-type calcium channel subtype mediates nicotine-induced conditioned place preference in mice
inferred← citeshttps://doi.org/10.1016/j.tins.2009.01.006openalex-snapshot
Calcium homeostasis, selective vulnerability and Parkinson's disease
inferred← citeshttps://doi.org/10.1016/j.transci.2015.04.007openalex-snapshot
Neurodegeneration in the elderly – When the blood type matters: An overview of the McLeod syndrome with focus on hematological features
inferred← citeshttps://doi.org/10.1016/s0079-6123(10)83004-3openalex-snapshot
What causes the death of dopaminergic neurons in Parkinson’s disease?
inferred← citeshttps://doi.org/10.1016/s1474-4422(07)70246-6openalex-snapshot
Calcium, ageing, and neuronal vulnerability in Parkinson's disease
inferred← citeshttps://doi.org/10.1017/cbo9781139003872.011openalex-snapshot
Genetic and biological contributions to cognitive impairment
inferred← citeshttps://doi.org/10.1017/s0033291710002217openalex-snapshot
Effects of aCACNA1Cgenotype on attention networks in healthy individuals
inferred← citeshttps://doi.org/10.1021/acschembio.0c00577openalex-snapshot
A Single Amino Acid Determines the Selectivity and Efficacy of Selective Negative Allosteric Modulators of CaV1.3 L‑Type Calcium Channels
inferred← citeshttps://doi.org/10.1021/jm3015926openalex-snapshot
Target- and Mechanism-Based Therapeutics for Neurodegenerative Diseases: Strength in Numbers
inferred← citeshttps://doi.org/10.1021/jm301839qopenalex-snapshot
Ligand Based Approach to L-Type Calcium Channel by Imidazo[2,1-b]thiazole-1,4-Dihydropyridines: from Heart Activity to Brain Affinity
inferred← citeshttps://doi.org/10.1021/jm500263vopenalex-snapshot
New 5-Unsubstituted Dihydropyridines with Improved CaV1.3 Selectivity as Potential Neuroprotective Agents against Ischemic Injury
inferred← citeshttps://doi.org/10.1021/jm800151nopenalex-snapshot
Discovery of Novel and Cardioselective Diltiazem-like Calcium Channel Blockers via Virtual Screening
inferred← citeshttps://doi.org/10.1038/s41598-025-29536-8openalex-snapshot
L-type calcium channel modulation reveals the relationship between neuronal synchrony and hyperactivity in the inferior colliculus following noise-induced hearing loss
inferred← citeshttps://doi.org/10.1038/srep17349openalex-snapshot
CACNA1C risk variant affects facial emotion recognition in healthy individuals
inferred← citeshttps://doi.org/10.1038/srep38602openalex-snapshot
A high-throughput fluorimetric microarray with enhanced fluorescence and suppressed “coffee-ring” effects for the detection of calcium ions in blood
inferred← citeshttps://doi.org/10.1073/pnas.0606173104openalex-snapshot
Neutralization of a single arginine residue gates open a two-pore domain, alkali-activated K + channel
inferred← citeshttps://doi.org/10.1080/15287394.2011.606796openalex-snapshot
Neurotoxicity Following Acute Inhalation Exposure to the Oil Dispersant COREXIT EC9500A
inferred← citeshttps://doi.org/10.1085/jgp.202313531openalex-snapshot
Inactivation of CaV1 and CaV2 channels
inferred← citeshttps://doi.org/10.1085/jgp.202513879openalex-snapshot
Inhibition of CaV1.4 channels by CaV3 channel antagonists ML218 and Z944
inferred← citeshttps://doi.org/10.1089/ars.2010.3521openalex-snapshot
The Origins of Oxidant Stress in Parkinson's Disease and Therapeutic Strategies
inferred← citeshttps://doi.org/10.1089/ars.2011.4033openalex-snapshot
Toxin Models of Mitochondrial Dysfunction in Parkinson's Disease
inferred← citeshttps://doi.org/10.1093/brain/awu131openalex-snapshot
Cav1.3 channels control D2-autoreceptor responses via NCS-1 in substantia nigra dopamine neurons
inferred← citeshttps://doi.org/10.1093/cercor/bhy189openalex-snapshot
A Common CACNA1C Gene Risk Variant has Sex-Dependent Effects on Behavioral Traits and Brain Functional Activity
inferred← citeshttps://doi.org/10.1093/med/9780197549469.003.0046openalex-snapshot
High-Voltage-Activated Calcium Channels in Epilepsy
inferred← citeshttps://doi.org/10.1093/schbul/sby096openalex-snapshot
CACNA1C: Association With Psychiatric Disorders, Behavior, and Neurogenesis
inferred← citeshttps://doi.org/10.1096/fj.06-6380comopenalex-snapshot
A role for presenilin in post‐stress regulation: effects of presenilin mutations on Ca 2+ currents in Drosophila
inferred← citeshttps://doi.org/10.1101/2021.01.06.425618openalex-snapshot
Resolving the molecular fingerprint of the distal carboxy tail in modulating Ca V 1 calcium dependent inactivation
inferred← citeshttps://doi.org/10.1101/lm.808608openalex-snapshot
Selective and protracted effect of nifedipine on fear memory extinction correlates with induced stress response
inferred← citeshttps://doi.org/10.1101/lm.996908openalex-snapshot
Rethinking the role of L-type voltage-gated calcium channels in fear memory extinction
inferred← citeshttps://doi.org/10.1111/bpa.12905openalex-snapshot
Cerebellar developmental deficits underlie neurodegenerative disorder spinocerebellar ataxia type 23
inferred← citeshttps://doi.org/10.1111/bph.13747openalex-snapshot
Calcium signalling through L‐type calcium channels: role in pathophysiology of spinal nociceptive transmission
inferred← citeshttps://doi.org/10.1111/ejn.70206openalex-snapshot
Shank3 Regulates L‐Type Voltage‐Gated Ca 2+ Channels in the Mouse Striatum and Hippocampus at the Early Developmental Stage
inferred← citeshttps://doi.org/10.1111/fcp.12174openalex-snapshot
Investigating the role of nisoldipine in foot‐shock‐induced post‐traumatic stress disorder in mice
inferred← citeshttps://doi.org/10.1111/j.1471-4159.2008.05596.xopenalex-snapshot
Modulation of basal and stress‐induced amygdaloid substance P release by the potent and selective NK1 receptor antagonist L‐822429
inferred← citeshttps://doi.org/10.1111/j.1526-4610.2008.01298.xopenalex-snapshot
Verapamil for Cluster Headache. Clinical Pharmacology and Possible Mode of Action
inferred← citeshttps://doi.org/10.1111/jcmm.70123openalex-snapshot
The L‐type calcium channel CaV1.3: A potential target for cancer therapy
inferred← citeshttps://doi.org/10.1111/jnc.13572openalex-snapshot
Converging roles of ion channels, calcium, metabolic stress, and activity pattern of Substantia nigra dopaminergic neurons in health and Parkinson's disease
inferred← citeshttps://doi.org/10.1111/jne.70268openalex-snapshot
Voltage‐gated calcium channel activity of gonadotropin‐releasing hormone ( GnRH ) neurons is altered by age and by prenatal androgen exposure in female mice
inferred← citeshttps://doi.org/10.1124/jpet.107.128215openalex-snapshot
Design of Mutant β2 Subunits as Decoy Molecules to Reduce the Expression of Functional Ca2+ Channels in Cardiac Cells
inferred← citeshttps://doi.org/10.1124/jpet.110.178244openalex-snapshot
Inhibition of Recombinant L-Type Voltage-Gated Calcium Channels by Positive Allosteric Modulators of GABAA Receptors
inferred← citeshttps://doi.org/10.1124/mol.108.049981openalex-snapshot
Expression and 1,4-Dihydropyridine-Binding Properties of Brain L-Type Calcium Channel Isoforms
inferred← citeshttps://doi.org/10.1126/sciadv.abm4560openalex-snapshot
Ca v 1.3 calcium channels are full-range linear amplifiers of firing frequencies in lateral DA SN neurons
inferred← citeshttps://doi.org/10.1126/sciadv.ady6978openalex-snapshot
α-Synuclein expression is required for somatodendritic dopamine release and immediate early gene induction
inferred← citeshttps://doi.org/10.1152/ajpcell.00288.2010openalex-snapshot
Small GTPase Rab11b regulates degradation of surface membrane L-type Cav1.2 channels
inferred← citeshttps://doi.org/10.1152/ajpcell.00329.2013openalex-snapshot
Cav1.2 and Cav1.3 L-type calcium channels operate in a similar voltage range but show different coupling to Ca2+-dependent conductances in hippocampal neurons
inferred← citeshttps://doi.org/10.1152/jn.00757.2013openalex-snapshot
Cav1.2 and Cav1.3 L-type calcium channels regulate dopaminergic firing activity in the mouse ventral tegmental area
inferred← citeshttps://doi.org/10.1152/jn.00890.2007openalex-snapshot
Neurotensin Enhances GABAergic Activity in Rat Hippocampus CA1 Region by Modulating L-Type Calcium Channels
inferred← citeshttps://doi.org/10.1152/jn.00948.2011openalex-snapshot
Repertoire of high voltage-activated Ca2+ channels in the lateral superior olive: functional analysis in wild-type, Cav1.3−/−, and Cav1.2DHP−/− mice
inferred← citeshttps://doi.org/10.1152/physrev.00016.2014openalex-snapshot
Molecular Aspects of Structure, Gating, and Physiology of pH-Sensitive Background K 2P and Kir K + -Transport Channels
inferred← citeshttps://doi.org/10.1152/physrev.00022.2021openalex-snapshot
Mechanisms and physiological implications of cooperative gating of clustered ion channels
inferred← citeshttps://doi.org/10.1152/physrev.00029.2007openalex-snapshot
Supramolecular Assemblies and Localized Regulation of Voltage-Gated Ion Channels
inferred← citeshttps://doi.org/10.1155/2012/597214openalex-snapshot
Levels of 1.2 L-Type Channels Peak in the First Two Weeks in Rat Hippocampus Whereas 1.3 Channels Steadily Increase through Development
inferred← citeshttps://doi.org/10.1155/2016/1796715openalex-snapshot
Sleep Spindles as Facilitators of Memory Formation and Learning
inferred← citeshttps://doi.org/10.1159/000322544openalex-snapshot
Pregabalin Antagonizes Copper-Induced Toxicity in the Brain: In vitro and in vivo Studies
inferred← citeshttps://doi.org/10.1167/iovs.18-23829openalex-snapshot
D- cis -Diltiazem Can Produce Oxidative Stress in Healthy Depolarized Rods In Vivo
inferred← citeshttps://doi.org/10.1177/030006051204000507openalex-snapshot
Correlation between Oxidative Stress and L-type Calcium Channel Expression in the Ventricular Myocardia of Selenium-deficient Mice
inferred← citeshttps://doi.org/10.1186/s41983-021-00307-2openalex-snapshot
Reflex memory theory of acquired involuntary motor and sensory disorders
inferred← citeshttps://doi.org/10.1192/bjp.bp.113.126979openalex-snapshot
CACNA1C, schizophrenia and major depressive disorder in the Han Chinese population
inferred← citeshttps://doi.org/10.1242/dmm.006841openalex-snapshot
A translational continuum of model systems for evaluating treatment strategies in Alzheimer’s disease: isradipine as a candidate drug
inferred← citeshttps://doi.org/10.1242/dmm.028738openalex-snapshot
The role of Ca2+ signaling in Parkinson's disease
inferred← citeshttps://doi.org/10.12659/msm.908765openalex-snapshot
Micro-RNA-137 Inhibits Tau Hyperphosphorylation in Alzheimer’s Disease and Targets the CACNA1C Gene in Transgenic Mice and Human Neuroblastoma SH-SY5Y Cells
inferred← citeshttps://doi.org/10.1371/journal.pgen.1009911openalex-snapshot
Ca2+ administration prevents α-synuclein proteotoxicity by stimulating calcineurin-dependent lysosomal proteolysis
inferred← citeshttps://doi.org/10.1371/journal.pone.0016141openalex-snapshot
Gating of a pH-Sensitive K2P Potassium Channel by an Electrostatic Effect of Basic Sensor Residues on the Selectivity Filter
inferred← citeshttps://doi.org/10.1371/journal.pone.0016778openalex-snapshot
A Folding Pathway-Dependent Score to Recognize Membrane Proteins
inferred← citeshttps://doi.org/10.1381/096089297765555359openalex-snapshot
Manuscript Preparation
inferred← citeshttps://doi.org/10.1523/jneurosci.0893-19.2020openalex-snapshot
Neuronal L-Type Calcium Channel Signaling to the Nucleus Requires a Novel CaMKIIα-Shank3 Interaction
inferred← citeshttps://doi.org/10.1523/jneurosci.1720-07.2007openalex-snapshot
Ca2+/Calmodulin Regulates Trafficking of CaV1.2 Ca2+Channels in Cultured Hippocampal Neurons
inferred← citeshttps://doi.org/10.1523/jneurosci.2168-09.2010openalex-snapshot
12-Lipoxygenase Regulates Hippocampal Long-Term Potentiation by Modulating L-Type Ca2+Channels
inferred← citeshttps://doi.org/10.1523/jneurosci.2357-17.2018openalex-snapshot
A Critical Neurodevelopmental Role for L-Type Voltage-Gated Calcium Channels in Neurite Extension and Radial Migration
inferred← citeshttps://doi.org/10.1523/jneurosci.4164-08.2010openalex-snapshot
Homeostatic Switch in Hebbian Plasticity and Fear Learning after Sustained Loss of Cav1.2 Calcium Channels
inferred← citeshttps://doi.org/10.1523/jneurosci.5098-10.2011openalex-snapshot
Cav1.3 Calcium Channels Are Required for Normal Development of the Auditory Brainstem
inferred← citeshttps://doi.org/10.17116/jnevro202312312120openalex-snapshot
Nimodipine in treatment of bipolar disorder
inferred← citeshttps://doi.org/10.18632/oncotarget.17618openalex-snapshot
Isradipine attenuates MPTP-induced dopamine neuron degeneration by inhibiting up-regulation of L-type calcium channels and iron accumulation in the substantia nigra of mice
inferred← citeshttps://doi.org/10.2174/092986708785909094openalex-snapshot
Computational Models of Neuronal Biophysics and the Characterization of Potential Neuropharmacological Targets
inferred← citeshttps://doi.org/10.2174/1570159x20666220327211156openalex-snapshot
The Role of Voltage-Gated Calcium Channels in Basal Ganglia Neurodegenerative Disorders
inferred← citeshttps://doi.org/10.2174/1871527319666200525011110openalex-snapshot
Overview of Therapeutic Drugs and Methods for the Treatment of Parkinson’s Disease
inferred← citeshttps://doi.org/10.3109/15622975.2015.1019360openalex-snapshot
CACNA1CSNP rs1006737 associates with bipolar I disorder independent of theBcl-2SNP rs956572 variant and its associated effect on intracellular calcium homeostasis
inferred← citeshttps://doi.org/10.3233/jad-130560openalex-snapshot
CaV1.2 Calcium Channel Expression in Reactive Astrocytes is associated with the Formation of Amyloid-β Plaques in an Alzheimer's Disease Mouse Model
inferred← citeshttps://doi.org/10.3389/fgene.2012.00200openalex-snapshot
Calcium homeostasis in aging neurons
inferred← citeshttps://doi.org/10.3389/fgene.2016.00107openalex-snapshot
Do Copy Number Changes in CACNA2D2, CACNA2D3, and CACNA1D Constitute a Predisposing Risk Factor for Alzheimer’s Disease?
inferred← citeshttps://doi.org/10.3389/fnana.2010.00140openalex-snapshot
Vulnerability of mesostriatal dopaminergic neurons in Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fncir.2021.642111openalex-snapshot
The Involvement of CaV1.3 Channels in Prolonged Root Reflexes and Its Potential as a Therapeutic Target in Spinal Cord Injury
inferred← citeshttps://doi.org/10.3389/fnins.2011.00021openalex-snapshot
Therapeutic Utility of Phosphodiesterase Type I Inhibitors in Neurological Conditions
inferred← citeshttps://doi.org/10.3389/fnins.2014.00113openalex-snapshot
Advances in non-dopaminergic treatments for Parkinson's disease
inferred← citeshttps://doi.org/10.3389/fnint.2017.00016openalex-snapshot
Impaired Fear Extinction Due to a Deficit in Ca2+ Influx Through L-Type Voltage-Gated Ca2+ Channels in Mice Deficient for Tenascin-C
inferred← citeshttps://doi.org/10.3389/fphys.2021.737291openalex-snapshot
Contributions of CaV1.3 Channels to Ca2+ Current and Ca2+-Activated BK Current in the Suprachiasmatic Nucleus
inferred← citeshttps://doi.org/10.3390/antiox13040389openalex-snapshot
Effects of L-Type Voltage-Gated Calcium Channel (LTCC) Inhibition on Hippocampal Neuronal Death after Pilocarpine-Induced Seizure
inferred← citeshttps://doi.org/10.3390/biomedicines12122851openalex-snapshot
Peptide Lv and Angiogenesis: A Newly Discovered Angiogenic Peptide
inferred← citeshttps://doi.org/10.3390/biomedicines13061376openalex-snapshot
Role of Cav1.3 Channels in Brain–Heart Interactions: An Unexpected Journey
inferred← citeshttps://doi.org/10.3390/genes8120344openalex-snapshot
Alternative Splicing of L-type CaV1.2 Calcium Channels: Implications in Cardiovascular Diseases
inferred← citeshttps://doi.org/10.3390/vaccines9030283openalex-snapshot
Zika E Glycan Loop Region and Guillain–Barré Syndrome-Related Proteins: A Possible Molecular Mimicry to Be Taken in Account for Vaccine Development
inferred← citeshttps://doi.org/10.35537/10915/145078openalex-snapshot
Evaluación del impacto de la coexpresión del receptor de ghrelina (GHSR) y los receptores de dopamina tipo 1 y 2 (D1R y D2R) sobre la modulación de los canales de calcio operados por voltaje presinápticos (CaV2)
inferred← citeshttps://doi.org/10.4061/2011/261791openalex-snapshot
Mitochondria and Parkinson's Disease
inferred← citeshttps://doi.org/10.4061/2011/716871openalex-snapshot
Mitochondrial Dysfunction in Parkinson's Disease
inferred← citeshttps://doi.org/10.4103/0253-7613.121266openalex-snapshot
Zonisamide: A review of the clinical and experimental evidence for its use in Parkinson′s disease
inferred← citeshttps://doi.org/10.4161/chan.4.6.12872openalex-snapshot
Impact of gating modulation in CaV1.3 L-type calcium channels
inferred← citeshttps://doi.org/10.46932/sfjdv2n2-009openalex-snapshot
Development of the L-type CaV / BK Complex Simulator (I): electrophysiological interaction
inferred← citeshttps://doi.org/10.64898/2026.01.13.699343openalex-snapshot
Autism associated mutation in Cacna1d causes perseverative behavioral phenotype in mice
inferred← citeshttps://doi.org/10.7287/peerj.8521v0.1/reviews/1openalex-snapshot
Peer Review #1 of "Association of the CACNA2D2 gene with schizophrenia in Chinese Han population (v0.1)"
inferred← citeshttps://doi.org/10.7287/peerj.8521v0.1/reviews/2openalex-snapshot
Peer Review #2 of "Association of the CACNA2D2 gene with schizophrenia in Chinese Han population (v0.1)"
inferred← citeshttps://doi.org/10.7287/peerj.8521v0.1/reviews/3openalex-snapshot
Peer Review #3 of "Association of the CACNA2D2 gene with schizophrenia in Chinese Han population (v0.1)"
inferred← citeshttps://doi.org/10.7287/peerj.8521v0.1/reviews/4openalex-snapshot
Peer Review #4 of "Association of the CACNA2D2 gene with schizophrenia in Chinese Han population (v0.1)"
inferred← citeshttps://doi.org/10.7287/peerj.8521v0.2/reviews/1openalex-snapshot
Peer Review #1 of "Association of the CACNA2D2 gene with schizophrenia in Chinese Han population (v0.2)"
inferred← citeshttps://doi.org/10.7287/peerj.8521v0.2/reviews/2openalex-snapshot
Peer Review #2 of "Association of the CACNA2D2 gene with schizophrenia in Chinese Han population (v0.2)"
inferred← citeshttps://doi.org/10.7287/peerj.8521v0.2/reviews/3openalex-snapshot
Peer Review #3 of "Association of the CACNA2D2 gene with schizophrenia in Chinese Han population (v0.2)"
inferred← citeshttps://doi.org/10.7287/peerj.8521v0.2/reviews/4openalex-snapshot
Peer Review #4 of "Association of the CACNA2D2 gene with schizophrenia in Chinese Han population (v0.2)"
inferred← citeshttps://doi.org/10.7287/peerj.8521v0.3/reviews/1openalex-snapshot
Peer Review #1 of "Association of the CACNA2D2 gene with schizophrenia in Chinese Han population (v0.3)"
inferred← citeshttps://doi.org/10.7554/elife.15744openalex-snapshot
Ca2+ entry into neurons is facilitated by cooperative gating of clustered CaV1.3 channels
inferred← citeshttps://doi.org/10.7717/peerj.8521openalex-snapshot
Association of the CACNA2D2 gene with schizophrenia in Chinese Han population
Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.