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Functional role of L-type Ca v 1.3 Ca 2+ channels in cardiac pacemaker activity
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Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/bf00656997openalex-snapshot
Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patches
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Slow inward current and cardiac arrhythmias
inferredcites →https://doi.org/10.1016/s0008-6363(01)00232-2openalex-snapshot
Transgenic mice overexpressing human KvLQT1 dominant-negative isoform Part II: Pharmacological profile
inferredcites →https://doi.org/10.1016/s0008-6363(01)00370-4openalex-snapshot
Properties of the hyperpolarization-activated current (If) in isolated mouse sino-atrial cells
inferredcites →https://doi.org/10.1016/s0014-5793(00)01979-7openalex-snapshot
Expression of T‐ and L‐type calcium channel mRNA in murine sinoatrial node
inferredcites →https://doi.org/10.1016/s0092-8674(00)00013-1openalex-snapshot
Congenital Deafness and Sinoatrial Node Dysfunction in Mice Lacking Class D L-Type Ca2+ Channels
inferredcites →https://doi.org/10.1016/s0140-6736(00)04440-8openalex-snapshot
Targeting voltage-gated Ca2+ channels
inferredcites →https://doi.org/10.1074/jbc.m006467200openalex-snapshot
Functional Embryonic Cardiomyocytes after Disruption of the L-type α1C (Ca 1.2) Calcium Channel Gene in the Mouse
inferredcites →https://doi.org/10.1074/jbc.m101469200openalex-snapshot
α1D (Cav1.3) Subunits Can Form L-type Ca2+ Channels Activating at Negative Voltages
inferredcites →https://doi.org/10.1111/j.1469-7793.2000.00051.xopenalex-snapshot
Properties of the delayed rectifier potassium current in porcine sino‐atrial node cells
inferredcites →https://doi.org/10.1113/jphysiol.1988.sp016916openalex-snapshot
Contribution of two types of calcium currents to the pacemaker potentials of rabbit sino‐atrial node cells.
inferredcites →https://doi.org/10.1146/annurev.ph.55.030193.002323openalex-snapshot
Pacemaker Mechanisms in Cardiac Tissue
inferredcites →https://doi.org/10.1152/ajpheart.1997.272.6.h2793openalex-snapshot
Regional differences in the role of the Ca2+ and Na+ currents in pacemaker activity in the sinoatrial node
inferredcites →https://doi.org/10.1152/ajpheart.1999.276.3.h1064openalex-snapshot
Contribution of L-type Ca2+current to electrical activity in sinoatrial nodal myocytes of rabbits
inferredcites →https://doi.org/10.1152/physrev.1993.73.1.197openalex-snapshot
Cardiac pacemaking in the sinoatrial node
inferredcites →https://doi.org/10.1159/000016320openalex-snapshot
Pharmacology, Structure and Function of Cardiac L-Type Ca2+ Channels
inferredcites →https://doi.org/10.1159/000350083openalex-snapshot
Down-Regulation of Na+/K+ATPase Activity by Human Parvovirus B19 Capsid Protein VP1
inferredcites →https://doi.org/10.1159/000368472openalex-snapshot
Regulation of the Voltage Gated K+ Channel Kv1.3 by Recombinant Human Klotho Protein
inferredcites →https://doi.org/10.1161/01.res.0000018003.14304.e2openalex-snapshot
Functional Roles of Ca v 1.3 (α 1D ) Calcium Channel in Sinoatrial Nodes
inferredcites →https://doi.org/10.1523/jneurosci.21-16-05944.2001openalex-snapshot
Neuronal Ca(V)1.3alpha(1) L-type channels activate at relatively hyperpolarized membrane potentials and are incompletely inhibited by dihydropyridines.
inferredcites →https://doi.org/10.1536/ihj.37.673openalex-snapshot
Ionic Mechanisms of the Cardiac Pacemaker Potential.
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Wnt/β-Catenin Signaling Causes Sinoatrial Node Dysfunction by Inhibition of T-type Calcium Current
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Identification and detection of the isolated sinus venosus from the Asian toad
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Expression of calcium channel CaV1.3 in cat spinal cord: Light and electron microscopic immunohistochemical study
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Ion Channels in the Heart
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Interactions with PDZ proteins diversify voltage‐gated calcium channel signaling
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Same-Single-Cell Analysis of Pacemaker-Specific Markers in Human Induced Pluripotent Stem Cell-Derived Cardiomyocyte Subtypes Classified by Electrophysiology
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L‐type Ca2+ channels in heart and brain
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Exploring the Function and Pharmacotherapeutic of Potential Voltage-Gated Ca2+ Channels with Gene-Knockout Models
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Cardiac Functions of Voltage-Gated Ca2+ Channels: Role of the Pharmacoresistant Type (E-/R-Type) in Cardiac Modulation and Putative Implication in Sudden Unexpected Death in Epilepsy (SUDEP)
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Pacemaker Current and Automatic Rhythms: Toward a Molecular Understanding
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Inherited Ventricular Arrhythmias: The Role of the Multi-Subunit Structure of the L-Type Calcium Channel Complex
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26 Sinus and Atrial Arrhythmias
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Splicing and Editing to Fine-Tune Activity of High Voltage-Activated Calcium Channels
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L-Type Ca2+ Channels and Cardiac Arrhythmias
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Defects in Ankyrin-based Protein Targeting Pathways in Human Arrhythmia
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Cardiac Pacemaking Is an Emergent Property of Complex Synchronized Signaling on Multiple Scales
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From early clinical studies to randomized controlled clinical trials
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Distribution and Regulation of L-Type Ca2+ Channels in Cardiomyocyte Microdomains
inferred← citeshttps://doi.org/10.1007/978-3-319-66816-1_1280-1openalex-snapshot
Sinoatrial Node Dysfunction and Deafness (SANDD)
inferred← citeshttps://doi.org/10.1007/978-3-540-35109-2_19openalex-snapshot
Analysis of Calcium Channels by Conditional Mutagenesis
inferred← citeshttps://doi.org/10.1007/978-3-540-72110-9_4openalex-snapshot
Creating a Cardiac Pacemaker by Gene Therapy
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Modeling the Action Potential in Mouse Sinoatrial Node Myocytes
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Molecular Basis of the Electrical Activity of the Atrioventricular Junction and Purkinje Fibres
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Defects in Ankyrin-Based Protein Targeting Pathways in Human Arrhythmia
inferred← citeshttps://doi.org/10.1007/978-3-642-40282-1_13openalex-snapshot
Cav1.3 Channels and Sino-Atrial Node Dysfunction
inferred← citeshttps://doi.org/10.1007/978-3-642-40282-1_16openalex-snapshot
Pharmacology of Voltage-Gated Calcium Channels in Clinic
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Role of L-Type Ca2+ Channels in Sensory Cells
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Splicing and Editing to Customize CaV Channel Structures for Optimal Neural Function
inferred← citeshttps://doi.org/10.1007/978-94-007-6334-0_7openalex-snapshot
Protein Interaction Partners of Cav2.3 R-Type Voltage-Gated Calcium Channels
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Electrophysiological Features of Telocytes
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Calcium Channel Splice Variants and Their Effects in Brain and Cardiovascular Function
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Disturbed atrio-ventricular conduction and normal contractile function in isolated hearts from Ca v 1.3-knockout mice
inferred← citeshttps://doi.org/10.1007/s00395-012-0265-5openalex-snapshot
Genetic suppression of Gαs protein provides rate control in atrial fibrillation
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Protein kinase C activation inhibits α1D L-type Ca channel: A single-channel analysis
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PDE type-4 inhibition increases L-type Ca2+ currents, action potential firing, and quantal size of exocytosis in mouse chromaffin cells
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Channelopathies in Cav1.1, Cav1.3, and Cav1.4 voltage-gated L-type Ca2+ channels
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T-type channels in the sino-atrial and atrioventricular pacemaker mechanism
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Heterogeneous functional expression of the sustained inward Na+ current in guinea pig sinoatrial node cells
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RBP2 stabilizes slow Cav1.3 Ca2+ channel inactivation properties of cochlear inner hair cells
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Regulation of cardiovascular calcium channel activity by post-translational modifications or interacting proteins
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Low voltage-activated channels in rat pulmonary vein cardiomyocytes: coexistence of a non-selective cationic channel and of T-type Ca channels
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De novo CACAN1D Ca2+ channelopathies: clinical phenotypes and molecular mechanism
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Channelopathies of voltage-gated L-type Cav1.3/α1D and T-type Cav3.1/α1G Ca2+ channels in dysfunction of heart automaticity
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Paradigm shift: new concepts for HCN4 function in cardiac pacemaking
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Are Cav1.3 calcium channels possible targets to modulate the in vivo activity of DA SN neurons in Parkinson’s disease?
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The genetic basis for inherited forms of sinoatrial dysfunction and atrioventricular node dysfunction
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Genetic isolation of stem cell-derived pacemaker-nodal cardiac myocytes
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Silencing of the T-type voltage-gated calcium channel α1 subunit by fungus-mediated RNAi altered the structure of F-actin and caused defective behaviors in Ditylenchus destructor
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Creating a cardiac pacemaker by gene therapy
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A modified method for isolating sinoatrial node myocytes from adult mice
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NALCN: A Regulator of Pacemaker Activity
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What keeps us ticking? Sinoatrial node mechano-sensitivity: the grandfather clock of cardiac rhythm
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The mechanism of increased postnatal heart rate and sinoatrial node pacemaker activity in mice
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Sinoatrial node pacemaker cells share dominant biological properties with glutamatergic neurons
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In Vitro Cultured Cardiomyocytes for Evaluating Cardiotoxicity
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Cardiac Action Potentials
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Cellular and Molecular Pathobiology of the Cardiac Conduction System
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In vitro Cultured Cardiomyocytes for Evaluating Cardiotoxicity
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OBSOLETE: Cellular Sinoatrial Node and Atrioventricular Node Activity in the Heart
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Cellular Sinoatrial Node and Atrioventricular Node Activity in the Heart
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Genomic basis of heart rate and rhythm generation
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Regulation of Cardiac Calcium Channels
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Cellular Sinoatrial Node and Atrioventricular Node Activity in the Heart
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Cav1.3 Voltage-Gated Calcium Channel
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Principles of Cellular Architecture and Physiology with Applications in Electrophysiology
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Calcium Channels in the Heart
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Regulation of Cardiac Calcium Channels
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Electrical and mechanical stimulation of cardiac cells and tissue constructs
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Altered function of neuronal L-type calcium channels in ageing and neuroinflammation: Implications in age-related synaptic dysfunction and cognitive decline
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Ca2+-dependent modulation of voltage-gated Ca2+ channels
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Differential splicing patterns of L-Type calcium channel Cav1.2 subunit in hearts of Spontaneously Hypertensive Rats and Wistar Kyoto rats
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Gradual differentiation and confinement of the cardiac conduction system as indicated by marker gene expression
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Molecular cloning and characterization of a hamster Cav1.3 Ca2+ channel variant with a long carboxyl terminus
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Cav1.3 and Cav1.2 channels of adrenal chromaffin cells: Emerging views on cAMP/cGMP-mediated phosphorylation and role in pacemaking
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Silencing of Cav1.2 gene in neonatal cardiomyocytes by lentiviral delivered shRNA
inferred← citeshttps://doi.org/10.1016/j.bbrc.2013.06.023openalex-snapshot
Calreticulin negatively regulates the surface expression of Cav1.3 L-type calcium channel
inferred← citeshttps://doi.org/10.1016/j.bbrc.2013.08.044openalex-snapshot
Potent inhibition of L-type Ca2+ currents by a Rad variant associated with congestive heart failure
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Novel function of α 1D L-type calcium channel in the atria
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Calcium signaling affects migration and proliferation differently in individual cancer cells due to nifedipine treatment
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Hallmarks of the channelopathies associated with L-type calcium channels : A focus on the Timothy mutations in Cav1.2 channels
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Beta-cardiotoxin, a funny current inhibitor, and its effect on ion channels involved in heart rate regulation and on calcium transient profile in mouse sinoatrial node cells
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CACNA1D De Novo Mutations in Autism Spectrum Disorders Activate Cav1.3 L-Type Calcium Channels
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C-Terminal Modulatory Domain Controls Coupling of Voltage-Sensing to Pore Opening in Cav1.3 L-type Ca2+ Channels
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Positive Feedback Mechanisms among Local Ca Releases, NCX, and ICaL Ignite Pacemaker Action Potentials
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Micro graphite-patterned diamond sensors: Towards the simultaneous in vitro detection of molecular release and action potentials generation from excitable cells
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Catecholamines and development of cardiac pacemaking: An intrinsically intimate relationship
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L-type calcium channels in adrenal chromaffin cells: Role in pace-making and secretion
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Physical and functional interaction of Snapin with Cav1.3 calcium channel impacts channel protein trafficking in atrial myocytes
inferred← citeshttps://doi.org/10.1016/j.ejphar.2007.06.050openalex-snapshot
Effects of phenylalkylamines and benzothiazepines on Cav1.3-mediated Ca2+ currents in neonatal mouse inner hair cells
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Effects of S(+)-efonidipine on the rabbit sinus node action potential and calcium channel subunits CaV1.2, CaV1.3 and CaV3.1
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Human native Cav1 channels in chromaffin cells: contribution to exocytosis and firing of spontaneous action potentials
inferred← citeshttps://doi.org/10.1016/j.ejphar.2017.08.006openalex-snapshot
Ionic mechanisms of the action of anaesthetics on sinoatrial node automaticity
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Alectinib causes sinus bradycardia by suppressing L-type calcium current in sinus node
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Differential gene expression during placentation in pregnancies conceived with different fertility treatments compared with spontaneous pregnancies
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Ionic mechanisms underlying region-specific remodeling of rabbit atrial action potentials caused by intermittent burst stimulation
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The case for the funny current and the calcium clock
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TRPM4 modulates sinus node diastolic depolarization
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Novel re-expression of L-type calcium channel Cav1.3 in left ventricles of failing human heart
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Natriuretic Peptide Receptor-C Protects Against Angiotensin II-Mediated Sinoatrial Node Disease in Mice
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Dysfunction in Ankyrin-B Disrupts Heart Rate Regulation by Altering the Membrane Abundance of GIRK Channels
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Molecular and Functional Remodeling of Superior and Inferior SAN in a Rat Model of HCM
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Congenital heart block: Identification of autoantibody binding site on the extracellular loop (domain I, S5–S6) of α1D L-type Ca channel
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The molecular basis of the inhibition of CaV1 calcium-dependent inactivation by the distal carboxy tail
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Modulation of Cav1.3 Ca2+ channel gating by Rab3 interacting molecule
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Relevance of tissue specific subunit expression in channelopathies
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Ca2+ currents in cardiac myocytes: Old story, new insights
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Voltage-dependent calcium channels and cardiac pacemaker activity: From ionic currents to genes
inferred← citeshttps://doi.org/10.1016/j.pbiomolbio.2005.05.010openalex-snapshot
Recent advances in understanding sex differences in cardiac repolarization
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Cell-specific Dynamic Clamp analysis of the role of funny If current in cardiac pacemaking
inferred← citeshttps://doi.org/10.1016/j.pbiomolbio.2020.08.004openalex-snapshot
Autonomic modulation of sinoatrial node: Role of pacemaker current and calcium sensitive adenylyl cyclase isoforms
inferred← citeshttps://doi.org/10.1016/j.pbiomolbio.2021.06.003openalex-snapshot
The funny current in genetically modified mice
inferred← citeshttps://doi.org/10.1016/j.pbiomolbio.2021.07.007openalex-snapshot
Cyclic nucleotide signaling and pacemaker activity
inferred← citeshttps://doi.org/10.1016/j.pbiomolbio.2022.10.008openalex-snapshot
The virtual sinoatrial node: What did computational models tell us about cardiac pacemaking?
inferred← citeshttps://doi.org/10.1016/j.pbiomolbio.2022.11.005openalex-snapshot
Pacemaker activity and ion channels in the sinoatrial node cells: MicroRNAs and arrhythmia
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2004.12.003openalex-snapshot
Mouse models to study L-type calcium channel function
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2013.04.010openalex-snapshot
Structure, function and clinical relevance of the cardiac conduction system, including the atrioventricular ring and outflow tract tissues
inferred← citeshttps://doi.org/10.1016/j.pneurobio.2011.09.010openalex-snapshot
Quantitative aspects of L-type Ca2+ currents
inferred← citeshttps://doi.org/10.1016/j.pneurobio.2014.04.001openalex-snapshot
Computational modeling of spike generation in serotonergic neurons of the dorsal raphe nucleus
inferred← citeshttps://doi.org/10.1016/j.pneurobio.2015.09.002openalex-snapshot
Genetic disruption of voltage-gated calcium channels in psychiatric and neurological disorders
inferred← citeshttps://doi.org/10.1016/j.taap.2016.08.006openalex-snapshot
Interaction between amiodarone and hepatitis-C virus nucleotide inhibitors in human induced pluripotent stem cell-derived cardiomyocytes and HEK-293 Cav 1.2 over-expressing cells
inferred← citeshttps://doi.org/10.1016/j.tcm.2003.09.006openalex-snapshot
Pacemaker Channels and Sinus Node Arrhythmia
inferred← citeshttps://doi.org/10.1016/j.tria.2021.100131openalex-snapshot
Do human sinoatrial node cells have t-tubules?
inferred← citeshttps://doi.org/10.1016/j.vascn.2007.03.003openalex-snapshot
Evaluation of the guinea pig monophasic action potential (MAP) assay in predicting drug-induced delay of ventricular repolarisation using 12 clinically documented drugs
inferred← citeshttps://doi.org/10.1016/j.vph.2026.107582openalex-snapshot
Ion channels of the human heart: A comprehensive four-chamber analysis
inferred← citeshttps://doi.org/10.1016/j.yexcr.2014.01.003openalex-snapshot
Molecular mechanisms of congenital heart block
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2003.10.007openalex-snapshot
Functional expression of L- and T-type Ca2+ channels in murine HL-1 cells
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2009.07.007openalex-snapshot
Local control of Ca2+-induced Ca2+ release in mouse sinoatrial node cells
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2010.10.002openalex-snapshot
Expression and roles of Cav1.3 (α1D) L-Type Ca2+ Channel in atrioventricular node automaticity
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2010.12.017openalex-snapshot
Molecular architecture of the human specialised atrioventricular conduction axis
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2011.08.014openalex-snapshot
Different subcellular populations of L-type Ca2+ channels exhibit unique regulation and functional roles in cardiomyocytes
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2012.01.018openalex-snapshot
The natriuretic peptides BNP and CNP increase heart rate and electrical conduction by stimulating ionic currents in the sinoatrial node and atrial myocardium following activation of guanylyl cyclase-linked natriuretic peptide receptors
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2012.08.020openalex-snapshot
Natriuretic peptides regulate heart rate and sinoatrial node function by activating multiple natriuretic peptide receptors
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2012.11.022openalex-snapshot
Ca2+ channel and Na+/Ca2+ exchange localization in cardiac myocytes
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2015.02.003openalex-snapshot
Sick sinus syndrome and atrial fibrillation in older persons — A view from the sinoatrial nodal myocyte
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2021.01.005openalex-snapshot
Rad-GTPase contributes to heart rate via L-type calcium channel regulation
inferred← citeshttps://doi.org/10.1016/s0035-3787(04)71000-0openalex-snapshot
Diversité génétique des canaux calciques activés par la dépolarisation membranaire
inferred← citeshttps://doi.org/10.1016/s1574-3349(07)18002-9openalex-snapshot
Morphogenesis of the Vertebrate Heart
inferred← citeshttps://doi.org/10.1017/s1047951114001358openalex-snapshot
Congenital heart block: current thoughts on management, morphologic spectrum, and role of intervention
inferred← citeshttps://doi.org/10.1038/aps.2015.135openalex-snapshot
Mechanisms underlying the cardiac pacemaker: the role of SK4 calcium-activated potassium channels
inferred← citeshttps://doi.org/10.1038/emboj.2008.3openalex-snapshot
Cardiac pacemaker function of HCN4 channels in mice is confined to embryonic development and requires cyclic AMP
inferred← citeshttps://doi.org/10.1038/ncomms5664openalex-snapshot
Cardiac arrhythmia induced by genetic silencing of ‘funny’ (f) channels is rescued by GIRK4 inactivation
inferred← citeshttps://doi.org/10.1038/nn.2694openalex-snapshot
Loss of Cav1.3 (CACNA1D) function in a human channelopathy with bradycardia and congenital deafness
inferred← citeshttps://doi.org/10.1038/nn1751openalex-snapshot
C-terminal modulator controls Ca2+-dependent gating of Cav1.4 L-type Ca2+ channels
inferred← citeshttps://doi.org/10.1038/nrcardio.2017.165openalex-snapshot
Next-generation pacemakers: from small devices to biological pacemakers
inferred← citeshttps://doi.org/10.1038/nrn3311openalex-snapshot
Calcium channel auxiliary α2δ and β subunits: trafficking and one step beyond
inferred← citeshttps://doi.org/10.1038/s10038-018-0542-8openalex-snapshot
Identification of CACNA1D variants associated with sinoatrial node dysfunction and deafness in additional Pakistani families reveals a clinical significance
inferred← citeshttps://doi.org/10.1038/s41467-020-20448-xopenalex-snapshot
Cellular and molecular landscape of mammalian sinoatrial node revealed by single-cell RNA sequencing
inferred← citeshttps://doi.org/10.1038/s41467-023-43502-wopenalex-snapshot
Selective blockade of Cav1.2 (α1C) versus Cav1.3 (α1D) L-type calcium channels by the black mamba toxin calciseptine
inferred← citeshttps://doi.org/10.1038/s41467-024-54296-wopenalex-snapshot
Large-scale single-nuclei profiling identifies role for ATRNL1 in atrial fibrillation
inferred← citeshttps://doi.org/10.1038/s41586-020-1947-zopenalex-snapshot
Mechanism of adrenergic CaV1.2 stimulation revealed by proximity proteomics
inferred← citeshttps://doi.org/10.1038/s41598-017-08191-8openalex-snapshot
CaV1.3 L-type Ca2+ channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na+ current
inferred← citeshttps://doi.org/10.1038/s41598-017-14230-1openalex-snapshot
Ca2+ protein alpha 1D of CaV1.3 regulates intracellular calcium concentration and migration of colon cancer cells through a non-canonical activity
inferred← citeshttps://doi.org/10.1038/s41598-020-76049-7openalex-snapshot
Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity
inferred← citeshttps://doi.org/10.1038/s41598-023-29723-5openalex-snapshot
Characterization of sinoatrial automaticity in Microcebus murinus to study the effect of aging on cardiac activity and the correlation with longevity
inferred← citeshttps://doi.org/10.1038/s42003-024-06962-4openalex-snapshot
Sexual dimorphism in a mouse model of Friedreich’s ataxia with severe cardiomyopathy
inferred← citeshttps://doi.org/10.1038/sj.bjp.0706101openalex-snapshot
Responses of the sustained inward current to autonomic agonists in guinea‐pig sino‐atrial node pacemaker cells
inferred← citeshttps://doi.org/10.1038/srep13688openalex-snapshot
Compensatory T-type Ca2+ channel activity alters D2-autoreceptor responses of Substantia nigra dopamine neurons from Cav1.3 L-type Ca2+ channel KO mice
inferred← citeshttps://doi.org/10.1038/srep21538openalex-snapshot
Cardiac sodium channel regulator MOG1 regulates cardiac morphogenesis and rhythm
inferred← citeshttps://doi.org/10.1038/srep40377openalex-snapshot
Methylome-wide Association Study of Atrial Fibrillation in Framingham Heart Study
inferred← citeshttps://doi.org/10.1038/srep42870openalex-snapshot
Blood-based omic profiling supports female susceptibility to tobacco smoke-induced cardiovascular diseases
inferred← citeshttps://doi.org/10.1038/srep44820openalex-snapshot
Cardiac drug-drug interaction between HCV-NS5B pronucleotide inhibitors and amiodarone is determined by their specific diastereochemistry
inferred← citeshttps://doi.org/10.1042/bcj20200206openalex-snapshot
Maurocalcin and its analog MCaE12A facilitate Ca2+ mobilization in cardiomyocytes
inferred← citeshttps://doi.org/10.1042/bst0340903openalex-snapshot
Role of voltage-gated L-type Ca2+ channel isoforms for brain function
inferred← citeshttps://doi.org/10.1042/cs20231025openalex-snapshot
Aging and sinus node dysfunction: mechanisms and future directions
inferred← citeshttps://doi.org/10.1051/medsci/2026103openalex-snapshot
Comment l’adrénaline accélère le cœur : un mécanisme reposant sur les canaux ioniques Ca V 1.3 et HCN
inferred← citeshttps://doi.org/10.1063/1.5037968openalex-snapshot
A microscale biomimetic platform for generation and electro-mechanical stimulation of 3D cardiac microtissues
inferred← citeshttps://doi.org/10.1073/pnas.0506520102openalex-snapshot
Inorganic polyphosphate in the social life of Myxococcus xanthus : Motility, development, and predation
inferred← citeshttps://doi.org/10.1073/pnas.0805500105openalex-snapshot
Dysfunction in ankyrin-B-dependent ion channel and transporter targeting causes human sinus node disease
inferred← citeshttps://doi.org/10.1073/pnas.0810332106openalex-snapshot
Control of heart rate by cAMP sensitivity of HCN channels
inferred← citeshttps://doi.org/10.1073/pnas.1308477110openalex-snapshot
Depressed pacemaker activity of sinoatrial node myocytes contributes to the age-dependent decline in maximum heart rate
inferred← citeshttps://doi.org/10.1073/pnas.1505670112openalex-snapshot
Burst pacemaker activity of the sinoatrial node in sodium–calcium exchanger knockout mice
inferred← citeshttps://doi.org/10.1073/pnas.1517181113openalex-snapshot
G protein-gated I KACh channels as therapeutic targets for treatment of sick sinus syndrome and heart block
inferred← citeshttps://doi.org/10.1073/pnas.2104668118openalex-snapshot
Bidirectional flow of the funny current (I f ) during the pacemaking cycle in murine sinoatrial node myocytes
inferred← citeshttps://doi.org/10.1073/pnas.2236970100openalex-snapshot
Increased phosphorylation of the neuronal L-type Ca 2+ channel Ca v 1.2 during aging
inferred← citeshttps://doi.org/10.1074/jbc.m111.265207openalex-snapshot
Functional Characterization of Alternative Splicing in the C Terminus of L-type CaV1.3 Channels
inferred← citeshttps://doi.org/10.1074/jbc.m114.586883openalex-snapshot
Regulation of Gene Transcription by Voltage-gated L-type Calcium Channel, Cav1.3
inferred← citeshttps://doi.org/10.1074/jbc.m307598200openalex-snapshot
Enhanced Expression of L-type Cav1.3 Calcium Channels in Murine Embryonic Hearts from Cav1.2-deficient Mice
inferred← citeshttps://doi.org/10.1074/jbc.m802254200openalex-snapshot
Modulation of Voltage- and Ca2+-dependent Gating of CaV1.3 L-type Calcium Channels by Alternative Splicing of a C-terminal Regulatory Domain
inferred← citeshttps://doi.org/10.1074/jbc.m809132200openalex-snapshot
Phosphorylation of the Consensus Sites of Protein Kinase A on α1D L-type Calcium Channel
inferred← citeshttps://doi.org/10.1080/00913847.2019.1568769openalex-snapshot
Asymptomatic bradycardia amongst endurance athletes
inferred← citeshttps://doi.org/10.1080/10799890801941947openalex-snapshot
Signaling Complexes of Voltage-Gated Calcium Channels and G Protein-Coupled Receptors
inferred← citeshttps://doi.org/10.1080/19336950.2015.1048936openalex-snapshot
L-type calcium channels as drug targets in CNS disorders
inferred← citeshttps://doi.org/10.1080/19336950.2018.1499368openalex-snapshot
Mutations in voltage-gated L-type calcium channel: implications in cardiac arrhythmia
inferred← citeshttps://doi.org/10.1080/19336950.2018.1532255openalex-snapshot
Potassium channels in the sinoatrial node and their role in heart rate control
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