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An Autoinflammatory Disease Due to Homozygous Deletion of the IL1RN Locus
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/art.10688openalex-snapshot
De novo CIAS1 mutations, cytokine activation, and evidence for genetic heterogeneity in patients with neonatal‐onset multisystem inflammatory disease (NOMID): A new member of the expanding family of pyrin‐associated autoinflammatory diseases
inferredcites →https://doi.org/10.1002/art.20033openalex-snapshot
Spectrum of clinical features in Muckle‐Wells syndrome and response to anakinra
inferredcites →https://doi.org/10.1006/geno.2002.6751openalex-snapshot
A Sequence-Based Map of the Nine Genes of the Human Interleukin-1 Cluster
inferredcites →https://doi.org/10.1006/geno.2002.6752openalex-snapshot
Genomic Organization of the Interleukin-1 Locus
inferredcites →https://doi.org/10.1016/j.immuni.2004.09.002openalex-snapshot
Human T Regulatory Cells Can Use the Perforin Pathway to Cause Autologous Target Cell Death
inferredcites →https://doi.org/10.1016/s0022-3476(81)80961-4openalex-snapshot
Arthropathy with rash, chronic meningitis, eye lesions, and mental retardation
inferredcites →https://doi.org/10.1016/s1074-7613(04)00046-9openalex-snapshot
NALP3 Forms an IL-1β-Processing Inflammasome with Increased Activity in Muckle-Wells Autoinflammatory Disorder
inferredcites →https://doi.org/10.1038/ng756openalex-snapshot
Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle–Wells syndrome
inferredcites →https://doi.org/10.1056/nejmoa0807865openalex-snapshot
An Autoinflammatory Disease with Deficiency of the Interleukin-1–Receptor Antagonist
inferredcites →https://doi.org/10.1074/jbc.m400117200openalex-snapshot
Interleukin (IL)-1F6, IL-1F8, and IL-1F9 Signal through IL-1Rrp2 and IL-1RAcP to Activate the Pathway Leading to NF-κB and MAPKs
inferredcites →https://doi.org/10.1084/jem.191.2.303openalex-snapshot
Arterial Inflammation in Mice Lacking the Interleukin 1 Receptor Antagonist Gene
inferredcites →https://doi.org/10.1084/jem.191.2.313openalex-snapshot
Development of Chronic Inflammatory Arthropathy Resembling Rheumatoid Arthritis in Interleukin 1 Receptor Antagonist–Deficient Mice
inferredcites →https://doi.org/10.1084/jem.20070157openalex-snapshot
Opposing activities of two novel members of the IL-1 ligand family regulate skin inflammation
inferredcites →https://doi.org/10.1093/bioinformatics/18.1.207openalex-snapshot
Genesis: cluster analysis of microarray data
inferredcites →https://doi.org/10.1093/rheumatology/keh202openalex-snapshot
The role of interleukin-1 in bone resorption in rheumatoid arthritis
inferredcites →https://doi.org/10.1097/00002281-200301000-00011openalex-snapshot
The expanding spectrum of systemic autoinflammatory disorders and their rheumatic manifestations
inferredcites →https://doi.org/10.1111/j.0022-202x.2004.22305.xopenalex-snapshot
Psoriasis-Like Cutaneous Inflammation in Mice Lacking Interleukin-1 Receptor Antagonist
inferredcites →https://doi.org/10.1111/j.1753-4887.2007.tb00362.xopenalex-snapshot
Inflammation in Pregnancy: Its Roles in Reproductive Physiology, Obstetrical Complications, and Fetal Injury
inferredcites →https://doi.org/10.1182/blood-2007-06-094201openalex-snapshot
Disease-associated CIAS1 mutations induce monocyte death, revealing low-level mosaicism in mutation-negative cryopyrin-associated periodic syndrome patients
inferredcites →https://doi.org/10.1301/nr.2007.dec.s194-s202openalex-snapshot
Inflammation in Pregnancy: Its Roles in Reproductive Physiology, Obstetrical Complications, and Fetal Injury
inferredcites →https://doi.org/10.3109/03009748709102523openalex-snapshot
A Chronic, Infantile, Neurological, Cutaneous and Articular (CINCA) Syndrome. A Specific Entity Analysed in 30 Patients
inferredcites →https://doi.org/10.4049/jimmunol.167.3.1440openalex-snapshot
Two Novel IL-1 Family Members, IL-1δ and IL-1ε, Function as an Antagonist and Agonist of NF-κB Activation Through the Orphan IL-1 Receptor-Related Protein 2
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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P2RX7 Gene Variants Associate with Altered Inflammasome Assembly and Reduced Pyroptosis in Chronic Nonbacterial Osteomyelitis (CNO)
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Diragan: Synthetic Data Generation for the Deficiency of Interleukin-1 Receptor Antagonist Using Generative Adversarial Networks
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Deficiency of Interleukin 1 Receptor Antagonist (DIRA)
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inferred← citeshttps://doi.org/10.1016/b978-1-4377-0412-9.00025-3openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/b978-1-4377-0755-7.00157-3openalex-snapshot
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Transgenic over-expression of interleukin-33 in osteoblasts results in decreased osteoclastogenesis
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Recombinant interleukin-1 receptor antagonist attenuates the severity of chronic pancreatitis induced by TNBS in rats
inferred← citeshttps://doi.org/10.1016/j.berh.2012.07.009openalex-snapshot
Autoinflammatory syndromes and cellular responses to stress: pathophysiology, diagnosis and new treatment perspectives
inferred← citeshttps://doi.org/10.1016/j.canlet.2011.10.001openalex-snapshot
Inflammasomes as molecular mediators of inflammation and cancer: Potential role in melanoma
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p65-Dependent production of interleukin-1β by osteolytic prostate cancer cells causes an induction of chemokine expression in osteoblasts
inferred← citeshttps://doi.org/10.1016/j.ccol.2014.10.030openalex-snapshot
10.1016/j.ccol.2014.10.030
inferred← citeshttps://doi.org/10.1016/j.cell.2010.02.043openalex-snapshot
Anti-inflammatory Agents: Present and Future
inferred← citeshttps://doi.org/10.1016/j.cell.2010.03.002openalex-snapshot
Autoinflammatory Disease Reloaded: A Clinical Perspective
inferred← citeshttps://doi.org/10.1016/j.celrep.2016.11.052openalex-snapshot
An NLRP3 Mutation Causes Arthropathy and Osteoporosis in Humanized Mice
inferred← citeshttps://doi.org/10.1016/j.clicom.2024.09.001openalex-snapshot
Deficiency of interleukin-1 receptor antagonist: An updated review of the pathogenesis, clinical characteristics, and treatments
inferred← citeshttps://doi.org/10.1016/j.clicom.2025.05.001openalex-snapshot
Deficiency of the interleukin-1 receptor antagonist: Characterizing the molecular consequences of loss-of-function IL1RN variants from structural and biochemical evidence
inferred← citeshttps://doi.org/10.1016/j.clim.2010.02.013openalex-snapshot
Monogenic IL-1 mediated autoinflammatory and immunodeficiency syndromes: Finding the right balance in response to danger signals
inferred← citeshttps://doi.org/10.1016/j.clim.2012.08.003openalex-snapshot
A novel mutation in the interleukin-1 receptor antagonist associated with intrauterine disease onset
inferred← citeshttps://doi.org/10.1016/j.clim.2012.12.012openalex-snapshot
Autoinflammatory bone disorders
inferred← citeshttps://doi.org/10.1016/j.clim.2013.03.016openalex-snapshot
Monogenic autoinflammatory diseases: Concept and clinical manifestations
inferred← citeshttps://doi.org/10.1016/j.clim.2013.04.008openalex-snapshot
Inflammasome and cytokine blocking strategies in autoinflammatory disorders
inferred← citeshttps://doi.org/10.1016/j.clim.2018.03.009openalex-snapshot
TNFAIP3 haploinsufficiency is the cause of autoinflammatory manifestations in a patient with a deletion of 13Mb on chromosome 6
inferred← citeshttps://doi.org/10.1016/j.clim.2020.108458openalex-snapshot
Chronic non-bacterial osteomyelitis and autoinflammatory bone diseases
inferred← citeshttps://doi.org/10.1016/j.clim.2020.108540openalex-snapshot
Cutibacterium acnes and autoinflammatory bone disease: Case series of three patients
inferred← citeshttps://doi.org/10.1016/j.clindermatol.2014.02.004openalex-snapshot
Autoinflammatory syndromes for the dermatologist
inferred← citeshttps://doi.org/10.1016/j.clp.2019.10.007openalex-snapshot
Autoinflammatory Disorders with Perinatal Onset
inferred← citeshttps://doi.org/10.1016/j.cmrp.2014.08.004openalex-snapshot
Rheumatological disorders and fever
inferred← citeshttps://doi.org/10.1016/j.coi.2012.09.005openalex-snapshot
After GWAS: mice to the rescue?
inferred← citeshttps://doi.org/10.1016/j.cyto.2015.06.017openalex-snapshot
The interleukin (IL)-1 cytokine family – Balance between agonists and antagonists in inflammatory diseases
inferred← citeshttps://doi.org/10.1016/j.cyto.2016.05.012openalex-snapshot
Interleukin-36 potently stimulates human M2 macrophages, Langerhans cells and keratinocytes to produce pro-inflammatory cytokines
inferred← citeshttps://doi.org/10.1016/j.cyto.2019.154951openalex-snapshot
EFLA 945 restricts AIM2 inflammasome activation by preventing DNA entry for psoriasis treatment
inferred← citeshttps://doi.org/10.1016/j.cyto.2022.155808openalex-snapshot
Multifaceted roles of IL-38 in inflammation and cancer
inferred← citeshttps://doi.org/10.1016/j.cyto.2022.155935openalex-snapshot
IL-1 family cytokines serve as 'activity recognition receptors' for aberrant protease activity indicative of danger
inferred← citeshttps://doi.org/10.1016/j.cytogfr.2016.11.001openalex-snapshot
Cytokine signatures in hereditary fever syndromes (HFS)
inferred← citeshttps://doi.org/10.1016/j.dci.2012.04.014openalex-snapshot
Identification, cloning and characterisation of interleukin-1F5 (IL-36RN) in the chicken
inferred← citeshttps://doi.org/10.1016/j.dci.2014.11.016openalex-snapshot
Molecular and functional characterization of an IL-1β receptor antagonist in grass carp (Ctenopharyngodon idella)
inferred← citeshttps://doi.org/10.1016/j.det.2013.04.001openalex-snapshot
Autoinflammatory Pustular Neutrophilic Diseases
inferred← citeshttps://doi.org/10.1016/j.det.2013.04.002openalex-snapshot
Autoinflammatory Disorders, Pain, and Neural Regulation of Inflammation
inferred← citeshttps://doi.org/10.1016/j.det.2013.04.010openalex-snapshot
Introduction to Autoinflammatory Syndromes and Diseases
inferred← citeshttps://doi.org/10.1016/j.det.2016.07.005openalex-snapshot
Dermatologic Manifestations of Monogenic Autoinflammatory Diseases
inferred← citeshttps://doi.org/10.1016/j.det.2021.12.003openalex-snapshot
Clinical Decisions in Pediatric Psoriasis
inferred← citeshttps://doi.org/10.1016/j.ejcb.2010.04.008openalex-snapshot
Interleukin-1, inflammasomes and the skin
inferred← citeshttps://doi.org/10.1016/j.ejim.2010.03.005openalex-snapshot
Targeting IL-1β in disease; the expanding role of NLRP3 inflammasome
inferred← citeshttps://doi.org/10.1016/j.ijcard.2025.133674openalex-snapshot
High admission levels of interleukin-1 receptor antagonist in acute myocardial infarction patients are associated with increased rates of all-cause mortality and cardiac death at five years follow-up
inferred← citeshttps://doi.org/10.1016/j.imbio.2012.10.007openalex-snapshot
Activated phenotype of circulating neutrophils in familial Mediterranean fever
inferred← citeshttps://doi.org/10.1016/j.imlet.2013.12.013openalex-snapshot
Autoinflammatory diseases
inferred← citeshttps://doi.org/10.1016/j.imlet.2017.11.013openalex-snapshot
New mosaic tiles in childhood hereditary autoinflammatory disorders
inferred← citeshttps://doi.org/10.1016/j.immuni.2013.11.010openalex-snapshot
The Interleukin-1 Family: Back to the Future
inferred← citeshttps://doi.org/10.1016/j.immuni.2023.05.014openalex-snapshot
Identification of an IL-1 receptor mutation driving autoinflammation directs IL-1-targeted drug design
inferred← citeshttps://doi.org/10.1016/j.intimp.2012.02.014openalex-snapshot
Anakinra as an interleukin 1 receptor antagonist, complicated genetics and molecular impacts- from the point of view of mouse genomics
inferred← citeshttps://doi.org/10.1016/j.it.2009.06.001openalex-snapshot
TIR8/SIGIRR: an IL-1R/TLR family member with regulatory functions in inflammation and T cell polarization
inferred← citeshttps://doi.org/10.1016/j.jaad.2013.11.005openalex-snapshot
New insights into pustular dermatoses in pediatric patients
inferred← citeshttps://doi.org/10.1016/j.jaci.2009.11.001openalex-snapshot
Therapy of autoinflammatory syndromes
inferred← citeshttps://doi.org/10.1016/j.jaci.2009.11.016openalex-snapshot
Autoinflammation: The prominent role of IL-1 in monogenic autoinflammatory diseases and implications for common illnesses
inferred← citeshttps://doi.org/10.1016/j.jaci.2015.04.049openalex-snapshot
Practice parameter for the diagnosis and management of primary immunodeficiency
inferred← citeshttps://doi.org/10.1016/j.jaci.2020.08.017openalex-snapshot
Monogenic autoinflammatory disorders: Conceptual overview, phenotype, and clinical approach
inferred← citeshttps://doi.org/10.1016/j.jaci.2022.12.816openalex-snapshot
Biologics and JAK inhibitors for the treatment of monogenic systemic autoinflammatory diseases in children
inferred← citeshttps://doi.org/10.1016/j.jaip.2020.11.055openalex-snapshot
Lifelong Immune Modulation Versus Hematopoietic Cell Therapy for Inborn Errors of Immunity
inferred← citeshttps://doi.org/10.1016/j.jaut.2024.103183openalex-snapshot
P2RX7 gene variants associate with altered inflammasome assembly and reduced pyroptosis in chronic nonbacterial osteomyelitis (CNO)
inferred← citeshttps://doi.org/10.1016/j.jdermsci.2012.01.004openalex-snapshot
Rare hereditary autoinflammatory disorders: Towards an understanding of critical in vivo inflammatory pathways
inferred← citeshttps://doi.org/10.1016/j.jid.2024.07.040openalex-snapshot
The Role of Crosstalk between Nets and Keratinocytes in Skin Immunity
inferred← citeshttps://doi.org/10.1016/j.jtauto.2021.100095openalex-snapshot
Chronic nonbacterial osteomyelitis (CNO) and chronic recurrent multifocal osteomyelitis (CRMO)
inferred← citeshttps://doi.org/10.1016/j.jtauto.2023.100213openalex-snapshot
Accuracy of generative artificial intelligence models in differential diagnoses of familial Mediterranean fever and deficiency of Interleukin-1 receptor antagonist
inferred← citeshttps://doi.org/10.1016/j.lfs.2020.118258openalex-snapshot
The intrinsic and extrinsic elements regulating inflammation
inferred← citeshttps://doi.org/10.1016/j.mucimm.2026.100410openalex-snapshot
The immune system in postnatal lung development
inferred← citeshttps://doi.org/10.1016/j.pcl.2016.08.008openalex-snapshot
When to Suspect Autoinflammatory/Recurrent Fever Syndromes
inferred← citeshttps://doi.org/10.1016/j.piel.2012.10.007openalex-snapshot
Concepto y clasificación de los síndromes autoinflamatorios
inferred← citeshttps://doi.org/10.1016/j.rbr.2017.07.419openalex-snapshot
DEFICIÊNCIA DO ANTAGONISTA DO RECEPTOR DA INTERLEUCINA‐1 (DIRA) – UMA NOVA DOENÇA AUTOINFLAMATÓRIA
inferred← citeshttps://doi.org/10.1016/j.rdc.2013.05.002openalex-snapshot
Autoinflammatory Bone Diseases
inferred← citeshttps://doi.org/10.1016/j.rdc.2013.08.001openalex-snapshot
Monogenic Autoinflammatory Diseases
inferred← citeshttps://doi.org/10.1016/j.siny.2015.12.005openalex-snapshot
Primary immunodeficiency in the neonate: Early diagnosis and management
inferred← citeshttps://doi.org/10.1016/j.smim.2013.10.008openalex-snapshot
Treating inflammation by blocking interleukin-1 in humans
inferred← citeshttps://doi.org/10.1016/j.smim.2013.10.014openalex-snapshot
Interleukin-18, more than a Th1 cytokine
inferred← citeshttps://doi.org/10.1016/j.trsl.2015.06.006openalex-snapshot
Targeting the inflammasome in rheumatic diseases
inferred← citeshttps://doi.org/10.1016/j.xjidi.2022.100176openalex-snapshot
Dermatologic Manifestations of Noninflammasome-Mediated Autoinflammatory Diseases
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