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EAT-2, a SAP-like adaptor, controls NK cell activation through phospholipase Cγ, Ca++, and Erk, leading to granule polarization
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/s00251-005-0056-3openalex-snapshot
Identification and characterization of two related murine genes, Eat2a and Eat2b, encoding single SH2-domain adapters
inferredcites →https://doi.org/10.1007/s00281-009-0193-0openalex-snapshot
SLAM family receptors and the SLAM-associated protein (SAP) modulate T cell functions
inferredcites →https://doi.org/10.1016/0092-8674(88)90053-0openalex-snapshot
The CD4 and CD8 T cell surface antigens are associated with the internal membrane tyrosine-protein kinase p56lck
inferredcites →https://doi.org/10.1016/0092-8674(93)90404-eopenalex-snapshot
SH2 domains recognize specific phosphopeptide sequences
inferredcites →https://doi.org/10.1016/j.coi.2008.03.005openalex-snapshot
Line of attack: NK cell specificity and integration of signals
inferredcites →https://doi.org/10.1016/j.immuni.2012.03.023openalex-snapshot
The Adaptor SAP Controls NK Cell Activation by Regulating the Enzymes Vav-1 and SHIP-1 and by Enhancing Conjugates with Target Cells
inferredcites →https://doi.org/10.1016/j.immuni.2012.04.006openalex-snapshot
Gene Deregulation and Chronic Activation in Natural Killer Cells Deficient in the Transcription Factor ETS1
inferredcites →https://doi.org/10.1016/j.immuni.2012.05.016openalex-snapshot
The Receptor Ly108 Functions as a SAP Adaptor-Dependent On-Off Switch for T Cell Help to B Cells and NKT Cell Development
inferredcites →https://doi.org/10.1016/j.immuni.2012.05.017openalex-snapshot
Positive and Negative Signaling through SLAM Receptors Regulate Synapse Organization and Thresholds of Cytolysis
inferredcites →https://doi.org/10.1016/j.it.2010.05.008openalex-snapshot
How do SAP family deficiencies compromise immunity?
inferredcites →https://doi.org/10.1016/s0960-9822(00)00221-9openalex-snapshot
Use of an oriented peptide library to determine the optimal substrates of protein kinases
inferredcites →https://doi.org/10.1038/26683openalex-snapshot
The X-linked lymphoproliferative-disease gene product SAP regulates signals induced through the co-receptor SLAM
inferredcites →https://doi.org/10.1038/373536a0openalex-snapshot
Catalytic specificity of protein-tyrosine kinases is critical for selective signalling
inferredcites →https://doi.org/10.1038/75973openalex-snapshot
High-efficiency deleter mice show that FLPe is an alternative to Cre-loxP
inferredcites →https://doi.org/10.1038/90615openalex-snapshot
Regulation of SLAM-mediated signal transduction by SAP, the X-linked lymphoproliferative gene product
inferredcites →https://doi.org/10.1038/nature07345openalex-snapshot
SAP-controlled T–B cell interactions underlie germinal centre formation
inferredcites →https://doi.org/10.1038/ncb919openalex-snapshot
Binding of SAP SH2 domain to FynT SH3 domain reveals a novel mechanism of receptor signalling in immune regulation
inferredcites →https://doi.org/10.1038/ncb920openalex-snapshot
SAP couples Fyn to SLAM immune receptors
inferredcites →https://doi.org/10.1038/ni.1693openalex-snapshot
Influence of CRACC, a SLAM family receptor coupled to the adaptor EAT-2, on natural killer cell function
inferredcites →https://doi.org/10.1038/ni.1763openalex-snapshot
Essential function for SAP family adaptors in the surveillance of hematopoietic cells by natural killer cells
inferredcites →https://doi.org/10.1038/ni.2016openalex-snapshot
STIM1, PKC-δ and RasGRP set a threshold for proapoptotic Erk signaling during B cell development
inferredcites →https://doi.org/10.1038/ni1242openalex-snapshot
Negative regulation of natural killer cell function by EAT-2, a SAP-related adaptor
inferredcites →https://doi.org/10.1038/nri1199openalex-snapshot
Roles of the NKG2D immunoreceptor and its ligands
inferredcites →https://doi.org/10.1038/nri2381openalex-snapshot
Formation and function of the lytic NK-cell immunological synapse
inferredcites →https://doi.org/10.1042/bj20050892openalex-snapshot
c-Cbl and Cbl-b ubiquitin ligases: substrate diversity and the negative regulation of signalling responses
inferredcites →https://doi.org/10.1073/pnas.0710698105openalex-snapshot
SAP expression in T cells, not in B cells, is required for humoral immunity
inferredcites →https://doi.org/10.1074/jbc.273.25.15765openalex-snapshot
Regulation of Mouse PECAM-1 Tyrosine Phosphorylation by the Src and Csk Families of Protein-tyrosine Kinases
inferredcites →https://doi.org/10.1074/jbc.m109.028209openalex-snapshot
Inhibition and Activation by CD244 Depends on CD2 and Phospholipase C-γ1
inferredcites →https://doi.org/10.1074/jbc.m704483200openalex-snapshot
Direct and Indirect Interactions of the Cytoplasmic Region of CD244 (2B4) in Mice and Humans with FYN Kinase
inferredcites →https://doi.org/10.1074/jbc.m800209200openalex-snapshot
Control of T Lymphocyte Signaling by Ly108, a Signaling Lymphocytic Activation Molecule Family Receptor Implicated in Autoimmunity
inferredcites →https://doi.org/10.1084/jem.176.6.1751openalex-snapshot
Fc gamma receptor activation induces the tyrosine phosphorylation of both phospholipase C (PLC)-gamma 1 and PLC-gamma 2 in natural killer cells.
inferredcites →https://doi.org/10.1084/jem.192.3.337openalex-snapshot
X-Linked Lymphoproliferative Disease
inferredcites →https://doi.org/10.1084/jem.194.3.235openalex-snapshot
Gntb-A, a Novel Sh2d1a-Associated Surface Molecule Contributing to the Inability of Natural Killer Cells to Kill Epstein-Barr Virus–Infected B Cells in X-Linked Lymphoproliferative Disease
inferredcites →https://doi.org/10.1084/jem.20112391openalex-snapshot
Expansion of somatically reverted memory CD8+ T cells in patients with X-linked lymphoproliferative disease caused by selective pressure from Epstein-Barr virus
inferredcites →https://doi.org/10.1093/emboj/20.21.5840openalex-snapshot
Structural basis for the interaction of the free SH2 domain EAT‐2 with SLAM receptors in hematopoietic cells
inferredcites →https://doi.org/10.1101/cshperspect.a002469openalex-snapshot
SLAM-Family Receptors: Immune Regulators with or without SAP-Family Adaptors
inferredcites →https://doi.org/10.1128/mcb.00357-06openalex-snapshot
Association between SAP and FynT: Inducible SH3 Domain-Mediated Interaction Controlled by Engagement of the SLAM Receptor
inferredcites →https://doi.org/10.1128/mcb.14.4.2777openalex-snapshot
Specific Motifs Recognized by the SH2 Domains of Csk, 3BP2, fps/fes, GRB-2, HCP, SHC, Syk, and Vav
inferredcites →https://doi.org/10.1128/mcb.17.8.4434openalex-snapshot
Regulation of T-Cell Antigen Receptor Signalling by Syk Tyrosine Protein Kinase
inferredcites →https://doi.org/10.1128/mcb.24.12.5144-5156.2004openalex-snapshot
Molecular Dissection of 2B4 Signaling: Implications for Signal Transduction by SLAM-Related Receptors
inferredcites →https://doi.org/10.1146/annurev-immunol-030409-101302openalex-snapshot
SLAM Family Receptors and SAP Adaptors in Immunity
inferredcites →https://doi.org/10.1146/annurev.immunol.23.021704.115526openalex-snapshot
NK CELL RECOGNITION
inferredcites →https://doi.org/10.1182/blood-2004-07-2731openalex-snapshot
Impaired Ig class switch in mice deficient for the X-linked lymphoproliferative disease gene Sap
inferredcites →https://doi.org/10.1182/blood-2004-08-3269openalex-snapshot
SAP controls the cytolytic activity of CD8+ T cells against EBV-infected cells
inferredcites →https://doi.org/10.1182/blood-2005-06-2428openalex-snapshot
Phospholipase C-γ2 is essential for NK cell cytotoxicity and innate immunity to malignant and virally infected cells
inferredcites →https://doi.org/10.1182/blood-2009-09-238832openalex-snapshot
Impaired Epstein-Barr virus–specific CD8+ T-cell function in X-linked lymphoproliferative disease is restricted to SLAM family–positive B-cell targets
inferredcites →https://doi.org/10.1371/journal.pbio.1001187openalex-snapshot
Molecular Pathogenesis of EBV Susceptibility in XLP as Revealed by Analysis of Female Carriers with Heterozygous Expression of SAP
inferredcites →https://doi.org/10.4049/jimmunol.1001974openalex-snapshot
Cutting Edge: The Adapters EAT-2A and -2B Are Positive Regulators of CD244- and CD84-Dependent NK Cell Functions in the C57BL/6 Mouse
inferredcites →https://doi.org/10.4049/jimmunol.175.12.7996openalex-snapshot
The Cytotoxicity Receptor CRACC (CS-1) Recruits EAT-2 and Activates the PI3K and Phospholipase Cγ Signaling Pathways in Human NK Cells
inferredcites →https://doi.org/10.4049/jimmunol.175.2.749openalex-snapshot
Phospholipase C-γ2 Is a Critical Signaling Mediator for Murine NK Cell Activating Receptors
inferredcites →https://doi.org/10.4049/jimmunol.177.8.5365openalex-snapshot
Differential and Nonredundant Roles of Phospholipase Cγ2 and Phospholipase Cγ1 in the Terminal Maturation of NK Cells
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/cac2.12579openalex-snapshot
“Find Me” and “Eat Me” signals: tools to drive phagocytic processes for modulating antitumor immunity
inferred← citeshttps://doi.org/10.1002/ctm2.1065openalex-snapshot
Single‐cell transcriptome profiling reveals the key role of ZNF683 in natural killer cell exhaustion in multiple myeloma
inferred← citeshttps://doi.org/10.1002/eji.202149406openalex-snapshot
SLAMF7 selectively favors degranulation to promote cytotoxicity in human NK cells
inferred← citeshttps://doi.org/10.1002/eji.70014openalex-snapshot
Influence of SLAM Family Receptors in the NK‐Cell‐Mediated Surveillance of Lymphoblastic Acute Leukemia
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IgG4-related disease
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Regulation of MHC class I-independent NK cell education by SLAM family receptors
inferred← citeshttps://doi.org/10.1016/j.ccm.2019.05.005openalex-snapshot
Immunoglobulin G4–related Disease
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Inflammatory macrophages exploit unconventional pro-phagocytic integrins for phagocytosis and anti-tumor immunity
inferred← citeshttps://doi.org/10.1016/j.clim.2018.10.011openalex-snapshot
SLAM family receptors in natural killer cells – Mediators of adhesion, activation and inhibition via cis and trans interactions
inferred← citeshttps://doi.org/10.1016/j.clim.2025.110574openalex-snapshot
The role of signaling lymphocytic activation molecule (SLAM) family receptors in health and disease progression: Focusing on cancer and therapy
inferred← citeshttps://doi.org/10.1016/j.coi.2015.11.003openalex-snapshot
SLAM family receptors in normal immunity and immune pathologies
inferred← citeshttps://doi.org/10.1016/j.immuni.2015.02.013openalex-snapshot
Epigenetic Modification and Antibody-Dependent Expansion of Memory-like NK Cells in Human Cytomegalovirus-Infected Individuals
inferred← citeshttps://doi.org/10.1016/j.intimp.2018.12.020openalex-snapshot
Direct potentiation of NK cell cytotoxicity by 8-azaguanine with potential antineoplastic activity
inferred← citeshttps://doi.org/10.1016/j.isci.2024.111669openalex-snapshot
Imaging of biphasic signalosomes constructed by checkpoint receptor 2B4 in conventional and chimeric antigen receptor-T cells
inferred← citeshttps://doi.org/10.1016/j.omto.2021.12.004openalex-snapshot
Adenoviral delivery of an immunomodulatory protein to the tumor microenvironment controls tumor growth
inferred← citeshttps://doi.org/10.1016/j.pan.2023.08.005openalex-snapshot
Increased SLAMF7+CD8+ T cells are associated with the pathogenesis of experimental autoimmune pancreatitis in mice
inferred← citeshttps://doi.org/10.1016/j.trim.2017.03.001openalex-snapshot
Steroid free immunosuppression is associated with enhanced Th1 transcripts in kidney transplantation
inferred← citeshttps://doi.org/10.1016/j.vaccine.2015.05.098openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.yexcr.2025.114516openalex-snapshot
The multifaceted role of CS1 (SLAMF7) in immunoregulation: Implications for cancer therapy and autoimmune disorders
inferred← citeshttps://doi.org/10.1038/cddis.2016.212openalex-snapshot
WASH has a critical role in NK cell cytotoxicity through Lck-mediated phosphorylation
inferred← citeshttps://doi.org/10.1038/ncomms10183openalex-snapshot
Natural cytotoxicity receptor splice variants orchestrate the distinct functions of human natural killer cell subtypes
inferred← citeshttps://doi.org/10.1038/ni.3369openalex-snapshot
A hematopoietic cell–driven mechanism involving SLAMF6 receptor, SAP adaptors and SHP-1 phosphatase regulates NK cell education
inferred← citeshttps://doi.org/10.1038/s41375-021-01172-xopenalex-snapshot
Selective elimination of immunosuppressive T cells in patients with multiple myeloma
inferred← citeshttps://doi.org/10.1038/s41423-019-0222-4openalex-snapshot
NK cell recognition of hematopoietic cells by SLAM-SAP families
inferred← citeshttps://doi.org/10.1038/s41435-022-00172-wopenalex-snapshot
Macrophage-mediated anti-tumor immunity against high-risk neuroblastoma
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NKG2D upregulation sensitizes tumors to combined anti-PD1 and anti-VEGF therapy and prevents hearing loss
inferred← citeshttps://doi.org/10.1038/s41597-023-02348-zopenalex-snapshot
Single cell transcriptome sequencing of stimulated and frozen human peripheral blood mononuclear cells
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Human lncRNAs NEAT1 and MALAT1 regulate the tumor microenvironment in lung cancer PDX models in athymic nude mice
inferred← citeshttps://doi.org/10.1056/nejmoa1505654openalex-snapshot
Elotuzumab Therapy for Relapsed or Refractory Multiple Myeloma
inferred← citeshttps://doi.org/10.1080/14740338.2017.1279603openalex-snapshot
Efficacy and safety of elotuzumab for the treatment of multiple myeloma
inferred← citeshttps://doi.org/10.1080/2162402x.2017.1339853openalex-snapshot
The anti-SLAMF7 antibody elotuzumab mediates NK cell activation through both CD16-dependent and –independent mechanisms
inferred← citeshttps://doi.org/10.1083/jcb.2051oia70openalex-snapshot
EAT-2, a SAP-like adaptor, controls NK cell activation through phospholipase Cγ, Ca++, and Erk, leading to granule polarization
inferred← citeshttps://doi.org/10.1084/jem.20150792openalex-snapshot
DNAM-1 controls NK cell activation via an ITT-like motif
inferred← citeshttps://doi.org/10.1084/jem.20160552openalex-snapshot
Deletion of Slam locus in mice reveals inhibitory role of SLAM family in NK cell responses regulated by cytokines and LFA-1
inferred← citeshttps://doi.org/10.1084/jem.2016055209072016copenalex-snapshot
Correction: Deletion of Slam locus in mice reveals inhibitory role of SLAM family in NK cell responses regulated by cytokines and LFA-1
inferred← citeshttps://doi.org/10.1089/scd.2014.0597openalex-snapshot
The Aryl Hydrocarbon Receptor Antagonist StemRegenin1 Improves In Vitro Generation of Highly Functional Natural Killer Cells from CD34 + Hematopoietic Stem and Progenitor Cells
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Enhancing network activation in natural killer cells: predictions from in silico modeling
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Single Cell Atlas of Human Dura Reveals Cellular Meningeal Landscape and Insights into Meningioma Immune Response
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Natural Killer Cell Dysfunction In Human Bladder Cancer Is Caused By Tissue-Specific Suppression of SLAMF6 Signaling
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Maternal‐Fetal Interface Cell Dysfunction in Patients With Preeclampsia Revealed via Single‐Cell RNA Sequencing
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Loss of PTPN12 Stimulates Progression of ErbB2-Dependent Breast Cancer by Enhancing Cell Survival, Migration, and Epithelial-to-Mesenchymal Transition
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Immune Cell Inhibition by SLAMF7 Is Mediated by a Mechanism Requiring Src Kinases, CD45, and SHIP-1 That Is Defective in Multiple Myeloma Cells
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Research Progress on NK Cell Receptors and Their Signaling Pathways
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Application of Weighted Gene Coexpression Network Analysis to Identify Key Modules and Hub Genes in Systemic Juvenile Idiopathic Arthritis
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Elotuzumab Enhances CD16‐Independent NK Cell‐Mediated Cytotoxicity against Myeloma Cells by Upregulating Several NK Cell‐Enhancing Genes
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Enhanced SLAMF7 Homotypic Interactions by Elotuzumab Improves NK Cell Killing of Multiple Myeloma
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Multifunctional Bispecific Nanovesicles Targeting SLAMF7 Trigger Potent Antitumor Immunity
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2B4 costimulatory domain enhancing cytotoxic ability of anti-CD5 chimeric antigen receptor engineered natural killer cells against T cell malignancies
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2B4 Is Dispensable for T-Dependent B Cell Immune Responses, but Its Deficiency Leads to Enhanced T-Independent Responses Due to an Increase in Peritoneal Cavity B1b Cells
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inferred← citeshttps://doi.org/10.2217/fon-2017-0371openalex-snapshot
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The efficacy of combination treatment with elotuzumab and lenalidomide is dependent on crosstalk between natural killer cells, monocytes and myeloma cells
inferred← citeshttps://doi.org/10.3389/fimmu.2015.00158openalex-snapshot
Negative Regulation of Humoral Immunity Due to Interplay between the SLAMF1, SLAMF5, and SLAMF6 Receptors
inferred← citeshttps://doi.org/10.3389/fimmu.2016.00004openalex-snapshot
Responses to Microbial Challenges by SLAMF Receptors
inferred← citeshttps://doi.org/10.3389/fimmu.2017.01444openalex-snapshot
Restoring Natural Killer Cell Immunity against Multiple Myeloma in the Era of New Drugs
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inferred← citeshttps://doi.org/10.3389/fimmu.2018.00307openalex-snapshot
Signals Controlling Lytic Granule Polarization at the Cytotoxic Immune Synapse
inferred← citeshttps://doi.org/10.3389/fimmu.2018.02551openalex-snapshot
Mechanisms of NK Cell Activation and Clinical Activity of the Therapeutic SLAMF7 Antibody, Elotuzumab in Multiple Myeloma
inferred← citeshttps://doi.org/10.3389/fimmu.2021.649311openalex-snapshot
Insights Into Human Intrahepatic NK Cell Function From Single Cell RNA Sequencing Datasets
inferred← citeshttps://doi.org/10.3389/fimmu.2021.792334openalex-snapshot
Filamin A Is Required for NK Cell Cytotoxicity at the Expense of Cytokine Production via Synaptic Filamentous Actin Modulation
inferred← citeshttps://doi.org/10.3389/fimmu.2022.803995openalex-snapshot
A NK Cell Odyssey: From Bench to Therapeutics Against Hematological Malignancies
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SLAM-family receptors come of age as a potential molecular target in cancer immunotherapy
inferred← citeshttps://doi.org/10.3389/fimmu.2025.1620327openalex-snapshot
X-linked lymphoproliferative disease type 1: a clinical and genetic update
inferred← citeshttps://doi.org/10.3389/fonc.2021.684561openalex-snapshot
Mechanisms of Action of the New Antibodies in Use in Multiple Myeloma
inferred← citeshttps://doi.org/10.3390/biomedicines11072086openalex-snapshot
Therapeutic Targets of Monoclonal Antibodies Used in the Treatment of Cancer: Current and Emerging
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Natural Killer Cells: Tumor Surveillance and Signaling
inferred← citeshttps://doi.org/10.3390/cancers13020279openalex-snapshot
Immune Functions of Signaling Lymphocytic Activation Molecule Family Molecules in Multiple Myeloma
inferred← citeshttps://doi.org/10.3390/cancers13071571openalex-snapshot
Monoclonal Antibodies and Antibody Drug Conjugates in Multiple Myeloma
inferred← citeshttps://doi.org/10.3390/ijms18061262openalex-snapshot
Amphotericin B, an Anti-Fungal Medication, Directly Increases the Cytotoxicity of NK Cells
inferred← citeshttps://doi.org/10.4049/jimmunol.1403023openalex-snapshot
B Cell–Intrinsic CD84 and Ly108 Maintain Germinal Center B Cell Tolerance
inferred← citeshttps://doi.org/10.4049/jimmunol.1800847openalex-snapshot
SLAMF7 Is a Critical Negative Regulator of IFN-α–Mediated CXCL10 Production in Chronic HIV Infection
inferred← citeshttps://doi.org/10.4084/mjhid.2018.014openalex-snapshot
ELOTUZUMAB FOR THE TREATMENT OF RELAPSED OR REFRACTORY MULTIPLE MYELOMA, WITH SPECIAL REFERENCE TO ITS MODES OF ACTION AND SLAMF7 SIGNALING
inferred← citeshttps://doi.org/10.6084/m9.figshare.19736402openalex-snapshot
Additional file 1 of Single-cell profiling of human dura and meningioma reveals cellular meningeal landscape and insights into meningioma immune response
inferred← citeshttps://doi.org/10.70675/9a0f10e8zdf71z4feaz991cz3baf64da5229openalex-snapshot
Influence des protéines de signalisation DOK1 et DOK2 sur le devenir et le comportement des cellules Natural Killer
inferred← citeshttps://doi.org/10.70675/caff0fc7z69abz4a91z9ffez4ae67a4969aeopenalex-snapshot
Calcium signature in the pancreatic cancer : role in the activation of the pancreatic stellate cells and in the progression of the pancreatic cancer
inferred← citeshttps://doi.org/10.70675/e8f87c4fz838ez4254zafdazcc06fa9ee7eaopenalex-snapshot
Infections virales par l’Hépatite E et Zika : pathogenèse à l’interface mère-fœtus et rôle de la réponse immune
inferred← citeshttps://doi.org/10.7939/r3-6gfy-s283openalex-snapshot
Murine leupaxin deletion results in spontaneous weight gain, tissue inflammation, and integrin signaling defects
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