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Broader Epstein–Barr virus–specific T cell receptor repertoire in patients with multiple sclerosis

https://doi.org/10.1084/jem.20220650
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/acn3.51573openalex-snapshot
Copy number variations across the blood–brain barrier in multiple sclerosis
inferredcites →https://doi.org/10.1002/ana.20049openalex-snapshot
Expression of CCR7 in multiple sclerosis: Implications for CNS immunity
inferredcites →https://doi.org/10.1002/ana.21744openalex-snapshot
Pathogenic CD8+ T cells in multiple sclerosis
inferredcites →https://doi.org/10.1002/ana.21978openalex-snapshot
Primary infection with the Epstein‐Barr virus and risk of multiple sclerosis
inferredcites →https://doi.org/10.1002/eji.201444662openalex-snapshot
High‐throughput sequencing of TCR repertoires in multiple sclerosis reveals intrathecal enrichment of EBV‐reactive CD8+ T cells
inferredcites →https://doi.org/10.1007/s00401-007-0320-8openalex-snapshot
Diagnosis of inflammatory demyelination in biopsy specimens: a practical approach
inferredcites →https://doi.org/10.1016/j.cell.2021.04.048openalex-snapshot
Integrated analysis of multimodal single-cell data
inferredcites →https://doi.org/10.1016/j.celrep.2017.08.078openalex-snapshot
Human Tissue-Resident Memory T Cells Are Defined by Core Transcriptional and Functional Signatures in Lymphoid and Mucosal Sites
inferredcites →https://doi.org/10.1016/j.celrep.2019.09.025openalex-snapshot
T Cell Repertoire Dynamics during Pregnancy in Multiple Sclerosis
inferredcites →https://doi.org/10.1016/j.eclinm.2022.101438openalex-snapshot
Highly multiplexed immune repertoire sequencing links multiple lymphocyte classes with severity of response to COVID-19
inferredcites →https://doi.org/10.1016/j.immuni.2018.02.010openalex-snapshot
Understanding Subset Diversity in T Cell Memory
inferredcites →https://doi.org/10.1016/j.molmed.2019.11.003openalex-snapshot
Epstein–Barr Virus in Multiple Sclerosis: Theory and Emerging Immunotherapies
inferredcites →https://doi.org/10.1016/j.molmed.2021.01.004openalex-snapshot
Epstein–Barr Virus in Multiple Sclerosis: Theory and Emerging Immunotherapies
inferredcites →https://doi.org/10.1016/s1474-4422(22)00086-2openalex-snapshot
Epstein-Barr virus and multiple sclerosis: supporting causality
inferredcites →https://doi.org/10.1038/nature10251openalex-snapshot
Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
inferredcites →https://doi.org/10.1038/ncomms3680openalex-snapshot
Using synthetic templates to design an unbiased multiplex PCR assay
inferredcites →https://doi.org/10.1038/ng.3822openalex-snapshot
Immunosequencing identifies signatures of cytomegalovirus exposure history and HLA-mediated effects on the T cell repertoire
inferredcites →https://doi.org/10.1038/ni.3031openalex-snapshot
Molecular regulation of effector and memory T cell differentiation
inferredcites →https://doi.org/10.1038/s41586-022-04419-4openalex-snapshot
Twin study reveals non-heritable immune perturbations in multiple sclerosis
inferredcites →https://doi.org/10.1038/s41586-022-04432-7openalex-snapshot
Clonally expanded B cells in multiple sclerosis bind EBV EBNA1 and GlialCAM
inferredcites →https://doi.org/10.1073/pnas.2003339117openalex-snapshot
Immune Signatures of Prodromal Multiple Sclerosis in Monozygotic Twins
inferredcites →https://doi.org/10.1074/jbc.270.48.28740openalex-snapshot
α4β1Integrin-dependent Cell Adhesion Is Regulated by a Low Affinity Receptor Pool That Is Conformationally Responsive to Ligand
inferredcites →https://doi.org/10.1084/jem.20072397openalex-snapshot
EBNA1-specific T cells from patients with multiple sclerosis cross react with myelin antigens and co-produce IFN-γ and IL-2
inferredcites →https://doi.org/10.1093/brain/awaa117openalex-snapshot
Tissue-resident memory T cells invade the brain parenchyma in multiple sclerosis white matter lesions
inferredcites →https://doi.org/10.1093/brain/awab147openalex-snapshot
Classification of neurological diseases using multi-dimensional CSF analysis
inferredcites →https://doi.org/10.1093/brain/awl067openalex-snapshot
Increased frequency and broadened specificity of latent EBV nuclear antigen-1-specific T cells in multiple sclerosis
inferredcites →https://doi.org/10.1093/brain/awr197openalex-snapshot
Epstein-Barr virus in the multiple sclerosis brain: a controversial issue--report on a focused workshop held in the Centre for Brain Research of the Medical University of Vienna, Austria
inferredcites →https://doi.org/10.1093/brain/awy151openalex-snapshot
The compartmentalized inflammatory response in the multiple sclerosis brain is composed of tissue-resident CD8+ T lymphocytes and B cells
inferredcites →https://doi.org/10.1093/nar/gky1006openalex-snapshot
The Immune Epitope Database (IEDB): 2018 update
inferredcites →https://doi.org/10.1093/nar/gkz874openalex-snapshot
VDJdb in 2019: database extension, new analysis infrastructure and a T-cell receptor motif compendium
inferredcites →https://doi.org/10.1111/bpa.12969openalex-snapshot
CNS inflammation after natalizumab therapy for multiple sclerosis: A retrospective histopathological and CSF cohort study
inferredcites →https://doi.org/10.1111/ene.12204openalex-snapshot
A prospective observational post‐marketing study of natalizumab‐treated multiple sclerosis patients: clinical, radiological and biological features and adverse events. The BIONAT cohort
inferredcites →https://doi.org/10.1111/imm.12808openalex-snapshot
Programmed death 1 is highly expressed on CD 8+ CD 57+ T cells in patients with stable multiple sclerosis and inhibits their cytotoxic response to Epstein–Barr virus
inferredcites →https://doi.org/10.1126/science.abj8222openalex-snapshot
Longitudinal analysis reveals high prevalence of Epstein-Barr virus associated with multiple sclerosis
inferredcites →https://doi.org/10.1126/sciimmunol.abb8786openalex-snapshot
Transcriptomic and clonal characterization of T cells in the human central nervous system
inferredcites →https://doi.org/10.1128/jvi.00980-19openalex-snapshot
Epstein-Barr Virus-Specific CD8 T Cells Selectively Infiltrate the Brain in Multiple Sclerosis and Interact Locally with Virus-Infected Cells: Clue for a Virus-Driven Immunopathological Mechanism
inferredcites →https://doi.org/10.1136/jclinpath-2019-205822openalex-snapshot
Epstein-Barr virus (EBV) reactivation and therapeutic inhibitors
inferredcites →https://doi.org/10.1136/jnnp-2020-322941openalex-snapshot
Complete Epstein-Barr virus seropositivity in a large cohort of patients with early multiple sclerosis
inferredcites →https://doi.org/10.1136/jnnp.2008.161018openalex-snapshot
Decreased T cell reactivity to Epstein-Barr virus infected lymphoblastoid cell lines in multiple sclerosis
inferredcites →https://doi.org/10.1159/000444256openalex-snapshot
Modulating Innate and Adaptive Immunity by (R)-Roscovitine: Potential Therapeutic Opportunity in Cystic Fibrosis
inferredcites →https://doi.org/10.1177/1352458515588581openalex-snapshot
Intrathecal CD8 T-cells of multiple sclerosis patients recognize lytic Epstein-Barr virus proteins
inferredcites →https://doi.org/10.1177/1352458520969145openalex-snapshot
BEST-MS: A prospective head-to-head comparative study of natalizumab and fingolimod in active relapsing MS
inferredcites →https://doi.org/10.1177/1352458520970103openalex-snapshot
High anti-JCPyV serum titers coincide with high CSF cell counts in RRMS patients
inferredcites →https://doi.org/10.1182/blood-2009-04-217604openalex-snapshot
Comprehensive assessment of T-cell receptor β-chain diversity in αβ T cells
inferredcites →https://doi.org/10.1182/blood.2020009482openalex-snapshot
CD27 is required for protective lytic EBV antigen–specific CD8+ T-cell expansion
inferredcites →https://doi.org/10.1212/nxi.0000000000000466openalex-snapshot
Molecular signature of Epstein-Barr virus infection in MS brain lesions
inferredcites →https://doi.org/10.1371/journal.ppat.1003220openalex-snapshot
Increased CD8+ T Cell Response to Epstein-Barr Virus Lytic Antigens in the Active Phase of Multiple Sclerosis
inferredcites →https://doi.org/10.18637/jss.v039.i05openalex-snapshot
Regularization Paths for Cox's Proportional Hazards Model via Coordinate Descent
inferredcites →https://doi.org/10.32614/rj-2014-011openalex-snapshot
The stringdist Package for Approximate String Matching
inferredcites →https://doi.org/10.3389/fimmu.2024.1488860openalex-snapshot
Magnitude and dynamics of the T-cell response to SARS-CoV-2 infection at both individual and population levels
inferredcites →https://doi.org/10.4049/jimmunol.202.supp.131.4openalex-snapshot
Scalable and comprehensive characterization of antigen-specific CD8 T cells using multi-omics single cell analysis
inferredcites →https://doi.org/10.7554/elife.38358openalex-snapshot
Human T cell receptor occurrence patterns encode immune history, genetic background, and receptor specificity

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4576546openalex-extracted
inferred← citeshttps://doi.org/10.1001/jamaneurol.2024.0272openalex-snapshot
Serologic Response to the Epstein-Barr Virus Peptidome and the Risk for Multiple Sclerosis
inferred← citeshttps://doi.org/10.1002/cti2.1437openalex-snapshot
From bedside to bench: how existing therapies inform the relationship between Epstein–Barr virus and multiple sclerosis
inferred← citeshttps://doi.org/10.1002/cti2.1451openalex-snapshot
Epstein–Barr virus and multiple sclerosis: moving from questions of association to questions of mechanism
inferred← citeshttps://doi.org/10.1002/eji.202451233openalex-snapshot
Narrowing Down Key Players in Autoimmunity via Single‐Cell Multiomics
inferred← citeshttps://doi.org/10.1002/eji.70153openalex-snapshot
Latent, Lytic, and Linked to Multiple Sclerosis—How EBV Drives Autoimmunity
inferred← citeshttps://doi.org/10.1007/82_2025_304openalex-snapshot
EBV and Autoimmunity
inferred← citeshttps://doi.org/10.1007/82_2025_339openalex-snapshot
The T Cell Response to Epstein-Barr Virus
inferred← citeshttps://doi.org/10.1007/s00415-025-13248-0openalex-snapshot
Environmental risk factors for multiple sclerosis: a comprehensive systematic review and meta-analysis
inferred← citeshttps://doi.org/10.1007/s10753-024-02079-2openalex-snapshot
Deciphering Autoimmune Diseases: Unveiling the Diagnostic, Therapeutic, and Prognostic Potential of Immune Repertoire Sequencing
inferred← citeshttps://doi.org/10.1007/s42452-025-07346-xopenalex-snapshot
Deciphering the role of CircRNA-miRNA networks in multiple sclerosis pathogenesis through minimal cut-set analysis
inferred← citeshttps://doi.org/10.1016/b978-0-12-824465-4.00271-4openalex-snapshot
The Immunobiology of Multiple Sclerosis
inferred← citeshttps://doi.org/10.1016/j.clim.2023.109733openalex-snapshot
High Epstein-Barr virus capsid antigen IgG level associates with the carriership of CD8+ T cell somatic mutations in the STAT3 SH2 domain
inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2024.136502openalex-snapshot
Investigating TCR-pMHC interactions for TCRs without identified epitopes by constructing a computational pipeline
inferred← citeshttps://doi.org/10.1016/j.isci.2026.115744openalex-snapshot
Multiple sclerosis-associated HLA demarcates EBV-specific CD8+ T cells with an exhausted and brain residency phenotype
inferred← citeshttps://doi.org/10.1016/j.jneuroim.2023.578036openalex-snapshot
Tissue-resident memory T cells in the multiple sclerosis brain and their relationship to Epstein-Barr virus infected B cells
inferred← citeshttps://doi.org/10.1016/j.jneuroim.2024.578314openalex-snapshot
EBV infected cells in the multiple sclerosis brain express PD-L1: How the virus and its niche may escape immune surveillance
inferred← citeshttps://doi.org/10.1016/j.jneuroim.2024.578343openalex-snapshot
Altered EBV specific immune control in multiple sclerosis
inferred← citeshttps://doi.org/10.1016/j.msard.2022.104434openalex-snapshot
What are T-cells telling us about how EBV causes MS?
inferred← citeshttps://doi.org/10.1016/j.msard.2023.105037openalex-snapshot
Humoral response to Epstein-Barr virus in patients with multiple sclerosis treated with B cell depletion therapy
inferred← citeshttps://doi.org/10.1016/j.msard.2024.105597openalex-snapshot
Ocrelizumab B cell depletion has no effect on HERV RNA expression in PBMC in MS patients
inferred← citeshttps://doi.org/10.1016/j.nicl.2024.103597openalex-snapshot
Impact of adult-onset multiple sclerosis on MRI-based intracranial volume: A study in clinically discordant monozygotic twins
inferred← citeshttps://doi.org/10.1016/j.phrs.2023.106797openalex-snapshot
T-cell receptor signaling modulated by the co-receptors: Potential targets for stroke treatment
inferred← citeshttps://doi.org/10.1016/j.retram.2024.103439openalex-snapshot
The clinical applications of immunosequencing
inferred← citeshttps://doi.org/10.1016/j.smim.2025.101947openalex-snapshot
The role of the adaptive immune system in the initiation and persistence of multiple sclerosis
inferred← citeshttps://doi.org/10.1016/j.smim.2025.101962openalex-snapshot
Unraveling the pathophysiology of narcolepsy type 1 through hypothesis-driven and hypothesis-generating approaches
inferred← citeshttps://doi.org/10.1016/s1474-4422(22)00471-9openalex-snapshot
Epstein-Barr virus as a cause of multiple sclerosis: opportunities for prevention and therapy
inferred← citeshttps://doi.org/10.1038/s41579-025-01181-yopenalex-snapshot
Epstein–Barr virus pathogenesis and emerging control strategies
inferred← citeshttps://doi.org/10.1038/s41582-023-00775-5openalex-snapshot
Epstein–Barr virus as a leading cause of multiple sclerosis: mechanisms and implications
inferred← citeshttps://doi.org/10.1038/s41582-024-01025-yopenalex-snapshot
Coeliac disease as a model for understanding multiple sclerosis
inferred← citeshttps://doi.org/10.1038/s41590-023-01664-1openalex-snapshot
Non-mutational neoantigens in disease
inferred← citeshttps://doi.org/10.1038/s41590-025-02412-3openalex-snapshot
Antigen specificity of clonally enriched CD8+ T cells in multiple sclerosis
inferred← citeshttps://doi.org/10.1073/pnas.2416097121openalex-snapshot
CD4 T cells restricted to DRB1*15:01 recognize two Epstein–Barr virus glycoproteins capable of intracellular antigen presentation
inferred← citeshttps://doi.org/10.1073/pnas.2419689122openalex-snapshot
Multiple sclerosis and gut microbiota: Lachnospiraceae from the ileum of MS twins trigger MS-like disease in germfree transgenic mice—An unbiased functional study
inferred← citeshttps://doi.org/10.1073/pnas.2420070121openalex-snapshot
Connecting the dots: Presentation of EBV antigens on HLA class II risk alleles connects the two main risk factors of multiple sclerosis
inferred← citeshttps://doi.org/10.1073/pnas.2424986122openalex-snapshot
Antibody reactivity against EBNA1 and GlialCAM differentiates multiple sclerosis patients from healthy controls
inferred← citeshttps://doi.org/10.1080/08923973.2024.2330642openalex-snapshot
Immunopathogenesis of multiple sclerosis: molecular and cellular mechanisms and new immunotherapeutic approaches
inferred← citeshttps://doi.org/10.1080/13543784.2026.2705509openalex-snapshot
Vidofludimus calcium for the treatment of multiple sclerosis: development rationale, mechanism of action, and early trial data
inferred← citeshttps://doi.org/10.1080/1744666x.2023.2248391openalex-snapshot
Interferon beta-1a sc at 25 years: a mainstay in the treatment of multiple sclerosis over the period of one generation
inferred← citeshttps://doi.org/10.1080/17460794.2025.2595851openalex-snapshot
The role of co-infections and the microbiome in the brain and mental health of people living with HIV
inferred← citeshttps://doi.org/10.1089/vim.2023.0109openalex-snapshot
Epstein Barr Virus and Multiple Sclerosis: Is a Cure Possible?
inferred← citeshttps://doi.org/10.1093/brain/awae006openalex-snapshot
Transcriptomics reveals CSF cellular composition in multiple sclerosis but detects no viral RNA
inferred← citeshttps://doi.org/10.1093/brain/awae244openalex-snapshot
Broader anti-EBV TCR repertoire in multiple sclerosis: disease specificity and treatment modulation
inferred← citeshttps://doi.org/10.1093/brain/awaf027openalex-snapshot
EBV-specific T-cell responses are telling us something important about multiple sclerosis
inferred← citeshttps://doi.org/10.1093/brain/awaf170openalex-snapshot
The case for targeting latent and lytic Epstein-Barr virus infection in multiple sclerosis
inferred← citeshttps://doi.org/10.1093/noajnl/vdad035openalex-snapshot
Systemic immune derangements are shared across various CNS pathologies and reflect novel mechanisms of immune privilege
inferred← citeshttps://doi.org/10.1097/wco.0000000000001266openalex-snapshot
Targeting Epstein–Barr virus in multiple sclerosis: when and how?
inferred← citeshttps://doi.org/10.1101/2022.02.23.481716openalex-snapshot
Epstein-Barr virus promotes T cell dysregulation in a humanized mouse model of multiple sclerosis
inferred← citeshttps://doi.org/10.1101/2023.02.02.526761openalex-snapshot
Discovering SARS-CoV-2 neoepitopes and the associated TCR-pMHC recognition mechanisms by combining single-cell sequencing, deep learning, and molecular dynamics simulation techniques
inferred← citeshttps://doi.org/10.1101/2023.10.22.23297386openalex-snapshot
Deep cervical lymph nodes of patients with multiple sclerosis show dysregulated B cells in the presence of Epstein-Barr virus
inferred← citeshttps://doi.org/10.1101/2024.07.30.605745openalex-snapshot
Regulatory T-cells in multiple sclerosis produce IL-10 in the central 1 nervous system but are activated by Epstein-Barr Virus
inferred← citeshttps://doi.org/10.1101/2024.10.23.619834openalex-snapshot
Linking myelin and Epstein-Barr virus specific immune responses in multiple sclerosis: insights from integrated public T cell receptor repertoires
inferred← citeshttps://doi.org/10.1101/2025.08.03.668342openalex-snapshot
De novo identification of the specificities of recurrent human T cell receptors
inferred← citeshttps://doi.org/10.1111/imcb.12825openalex-snapshot
Immune repertoire profiling in myasthenia gravis
inferred← citeshttps://doi.org/10.1126/sciadv.adg3032openalex-snapshot
Cross-reactive EBNA1 immunity targets alpha-crystallin B and is associated with multiple sclerosis
inferred← citeshttps://doi.org/10.1126/sciadv.adu5110openalex-snapshot
Epstein-Barr virus infection promotes T cell dysregulation in a humanized mouse model of multiple sclerosis
inferred← citeshttps://doi.org/10.1126/sciadv.aeb1732openalex-snapshot
De novo identification of the specificities of recurrently identified human T cell receptors
inferred← citeshttps://doi.org/10.1126/sciimmunol.adf0763openalex-snapshot
EBVerified TCRs and multiple sclerosis
inferred← citeshttps://doi.org/10.1126/scitranslmed.adg7863openalex-snapshot
Learning CNS immunopathology from therapeutic interventions
inferred← citeshttps://doi.org/10.1126/scitranslmed.adz6566openalex-snapshot
CD4 + T cells reactive to Epstein-Barr virus late lytic antigens are enriched in individuals with multiple sclerosis
inferred← citeshttps://doi.org/10.1128/mmbr.00119-23openalex-snapshot
Multiple sclerosis and infection: history, EBV, and the search for mechanism
inferred← citeshttps://doi.org/10.1177/13524585241235825openalex-snapshot
EBV infection drives MS pathology: Yes
inferred← citeshttps://doi.org/10.1182/blood.2023023142openalex-snapshot
Virus-reactive T cells expanded in aplastic anemia eliminate hematopoietic progenitor cells by molecular mimicry
inferred← citeshttps://doi.org/10.1186/s12985-026-03143-zopenalex-snapshot
Viral associations in multiple sclerosis: pathogenetic mechanisms and therapeutic implications
inferred← citeshttps://doi.org/10.1186/s12993-025-00286-xopenalex-snapshot
Disruption of circadian rhythms and behavior in Drosophila melanogaster induced by Epstein-Barr virus: insights into viral infections and host physiology
inferred← citeshttps://doi.org/10.1212/nxi.0000000000200208openalex-snapshot
Safety and Dose-Response of Vidofludimus Calcium in Relapsing Multiple Sclerosis
inferred← citeshttps://doi.org/10.1212/nxi.0000000000200217openalex-snapshot
Disease-Modifying Treatments for Multiple Sclerosis Affect Measures of Cellular Immune Responses to EBNA-1 Peptides
inferred← citeshttps://doi.org/10.1212/nxi.0000000000200545openalex-snapshot
EBV Dysregulation Is Associated With Immune Imbalance in Multiple Sclerosis
inferred← citeshttps://doi.org/10.1212/nxi.0000000000200636openalex-snapshot
Distinct Multiomic Signatures of Environmental Exposure Drive Immune Dysregulation and Disease Activity in Early Multiple Sclerosis
inferred← citeshttps://doi.org/10.1212/nxi.0000000000200654openalex-snapshot
EBNA1 381-452 and Cross-Reactive Antibody Responses Reveal Immune Signatures in Multiple Sclerosis
inferred← citeshttps://doi.org/10.17879/freeneuropathology-2023-4675openalex-snapshot
Multiple sclerosis: 2023 update
inferred← citeshttps://doi.org/10.21203/rs.3.rs-2146712/v1openalex-snapshot
CD8+ T cell clonotypes from prior SARS-CoV-2 infection predominate during the cellular immune response to mRNA vaccination
inferred← citeshttps://doi.org/10.33588/rn.7701.2023167openalex-snapshot
XV Reunión Post-ECTRIMS: revisión de las novedades presentadas en el Congreso ECTRIMS 2022 (I)
inferred← citeshttps://doi.org/10.3389/fimmu.2023.1004795openalex-snapshot
Anti-CD20 therapies in multiple sclerosis: From pathology to the clinic
inferred← citeshttps://doi.org/10.3389/fimmu.2023.1127470openalex-snapshot
Antigen-specificity measurements are the key to understanding T cell responses
inferred← citeshttps://doi.org/10.3389/fimmu.2023.1212546openalex-snapshot
The good and the bad of T cell cross-reactivity: challenges and opportunities for novel therapeutics in autoimmunity and cancer
inferred← citeshttps://doi.org/10.3389/fimmu.2023.1289313openalex-snapshot
Mechanisms of T cell evasion by Epstein-Barr virus and implications for tumor survival
inferred← citeshttps://doi.org/10.3389/fimmu.2024.1401156openalex-snapshot
High heterogeneity of cross-reactive immunoglobulins in multiple sclerosis presumes combining of B-cell epitopes for diagnostics: a case-control study
inferred← citeshttps://doi.org/10.3389/fimmu.2024.1503808openalex-snapshot
Epstein-Barr virus, vitamin D and the immune response: connections with consequences for multiple sclerosis
inferred← citeshttps://doi.org/10.3389/fimmu.2024.1509927openalex-snapshot
EBV-specific T-cell immunity: relevance for multiple sclerosis
inferred← citeshttps://doi.org/10.3389/fimmu.2026.1836543openalex-snapshot
HuBIE: The human blood immunome encyclopedia of TCRs and BCRs in bloodstream infections and cancer
inferred← citeshttps://doi.org/10.3389/fimmu.2026.1929166openalex-snapshot
High EBV-reactive IgG levels in serum are linked to typical inflammatory cerebrospinal fluid features in multiple sclerosis
inferred← citeshttps://doi.org/10.3390/cells11223619openalex-snapshot
Epstein-Barr Virus-Induced Genes and Endogenous Retroviruses in Immortalized B Cells from Patients with Multiple Sclerosis
inferred← citeshttps://doi.org/10.3390/sclerosis2030009openalex-snapshot
Multiple Sclerosis: Immune Cells, Histopathology, and Therapeutics
inferred← citeshttps://doi.org/10.55788/34653a88openalex-snapshot
MS associated with a broader Epstein-Barr virus specific T-cell receptor repertoire
inferred← citeshttps://doi.org/10.55788/fb36add0openalex-snapshot
Ineffective response to EBV in MS not seen in similar diseases
inferred← citeshttps://doi.org/10.64898/2025.12.11.25342083openalex-snapshot
A Genetically Driven Immunologic Mechanism Underlying the Link between EBV and Multiple Sclerosis
inferred← citeshttps://doi.org/10.64898/2026.03.17.712532openalex-snapshot
immunoPETE: A DNA-based integrated B-cell and T-cell receptor profiling platform
inferred← citeshttps://doi.org/10.64898/2026.09.28.753741openalex-snapshot
A pro-inflammatory effector-memory T cell program links peripheral EBV priming to CNS autoimmunity
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