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Regulation of HIV‐1 gene expression by histone acetylation and factor recruitment at the LTR promoter

https://doi.org/10.1093/emboj/cdg631
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/j.1460-2075.1993.tb05994.xopenalex-snapshot
Chromatin disruption in the promoter of human immunodeficiency virus type 1 during transcriptional activation.
inferredcites →https://doi.org/10.1002/j.1460-2075.1996.tb00449.xopenalex-snapshot
Transcriptional activation and chromatin remodeling of the HIV‐1 promoter in response to histone acetylation.
inferredcites →https://doi.org/10.1016/0092-8674(89)90289-4openalex-snapshot
Tat trans-activates the human immunodeficiency virus through a nascent RNA target
inferredcites →https://doi.org/10.1016/0092-8674(90)90691-7openalex-snapshot
Cells nonproductively infected with HIV-1 exhibit an aberrant pattern of viral RNA expression: A molecular model for latency
inferredcites →https://doi.org/10.1016/0378-1119(92)90220-jopenalex-snapshot
A novel procedure for quantitative polymerase chain reaction by coamplification of competitive templates
inferredcites →https://doi.org/10.1016/s0006-2952(02)01152-8openalex-snapshot
Diversity of acetylation targets and roles in transcriptional regulation: the human immunodeficiency virus type 1 promoter as a model system
inferredcites →https://doi.org/10.1016/s0021-9258(19)74555-4openalex-snapshot
Effects of integration and replication on transcription of the HIV-1 long terminal repeat.
inferredcites →https://doi.org/10.1016/s0092-8674(00)00169-0openalex-snapshot
Ordered Recruitment of Chromatin Modifying and General Transcription Factors to the IFN-β Promoter
inferredcites →https://doi.org/10.1016/s0092-8674(00)00188-4openalex-snapshot
Cofactor Dynamics and Sufficiency in Estrogen Receptor–Regulated Transcription
inferredcites →https://doi.org/10.1016/s0092-8674(02)00654-2openalex-snapshot
Cooperation between Complexes that Regulate Chromatin Structure and Transcription
inferredcites →https://doi.org/10.1016/s0092-8674(02)01005-xopenalex-snapshot
Function and Selectivity of Bromodomains in Anchoring Chromatin-Modifying Complexes to Promoter Nucleosomes
inferredcites →https://doi.org/10.1016/s0092-8674(02)01077-2openalex-snapshot
Deciphering the Transcriptional Histone Acetylation Code for a Human Gene
inferredcites →https://doi.org/10.1016/s0959-437x(02)00279-4openalex-snapshot
Histone modifications in transcriptional regulation
inferredcites →https://doi.org/10.1016/s1097-2765(01)00200-3openalex-snapshot
A Transient Histone Hyperacetylation Signal Marks Nucleosomes for Remodeling at the PHO8 Promoter In Vivo
inferredcites →https://doi.org/10.1016/s1097-2765(02)00483-5openalex-snapshot
Structural Basis of Lysine-Acetylated HIV-1 Tat Recognition by PCAF Bromodomain
inferredcites →https://doi.org/10.1016/s1097-2765(02)00604-4openalex-snapshot
Ordered Recruitment
inferredcites →https://doi.org/10.1023/a:1003797332379openalex-snapshot
Chromatin control of HIV‐1 gene expression
inferredcites →https://doi.org/10.1038/47412openalex-snapshot
The language of covalent histone modifications
inferredcites →https://doi.org/10.1073/pnas.91.15.7119openalex-snapshot
Fine mapping of a replication origin of human DNA.
inferredcites →https://doi.org/10.1073/pnas.95.23.13519openalex-snapshot
HIV-1 Tat transactivator recruits p300 and CREB-binding protein histone acetyltransferases to the viral promoter
inferredcites →https://doi.org/10.1074/jbc.273.38.24898openalex-snapshot
Activation of Integrated Provirus Requires Histone Acetyltransferase
inferredcites →https://doi.org/10.1074/jbc.m006701200openalex-snapshot
Internalization of HIV-1 Tat Requires Cell Surface Heparan Sulfate Proteoglycans
inferredcites →https://doi.org/10.1074/jbc.m101385200openalex-snapshot
The Histone Acetyltransferase, hGCN5, Interacts with and Acetylates the HIV Transactivator, Tat
inferredcites →https://doi.org/10.1074/jbc.m104830200openalex-snapshot
Visualization of in Vivo Direct Interaction between HIV-1 TAT and Human Cyclin T1 in Specific Subcellular Compartments by Fluorescence Resonance Energy Transfer
inferredcites →https://doi.org/10.1074/jbc.m303045200openalex-snapshot
Cell Membrane Lipid Rafts Mediate Caveolar Endocytosis of HIV-1 Tat Fusion Proteins
inferredcites →https://doi.org/10.1080/152165401753544241openalex-snapshot
Multiple Modes of Transcriptional Regulation by the HIV‐1 Tat Transactivator
inferredcites →https://doi.org/10.1089/aid.1996.12.117openalex-snapshot
Quantitative Dynamics of HIV Type 1 Expression
inferredcites →https://doi.org/10.1093/emboj/cdf669openalex-snapshot
Differential acetylation of Tat coordinates its interaction with the co‐activators cyclin T1 and PCAF
inferredcites →https://doi.org/10.1093/emboj/cdg205openalex-snapshot
Recruitment of human cyclin T1 to nuclear bodies through direct interaction with the PML protein
inferredcites →https://doi.org/10.1101/gad.10.1.37openalex-snapshot
NF-kappa B-mediated chromatin reconfiguration and transcriptional activation of the HIV-1 enhancer in vitro.
inferredcites →https://doi.org/10.1101/gad.11.24.3327openalex-snapshot
Histone acetyltransferases regulate HIV-1 enhancer activity in vitro
inferredcites →https://doi.org/10.1101/gad.967302openalex-snapshot
Dynamics of global histone acetylation and deacetylation in vivo: rapid restoration of normal histone acetylation status upon removal of activators and repressors
inferredcites →https://doi.org/10.1126/science.287.5460.2023openalex-snapshot
Start Sites of Bidirectional DNA Synthesis at the Human Lamin B2 Origin
inferredcites →https://doi.org/10.1126/science.3313729openalex-snapshot
Cytokine-Induced Expression of HIV-1 in a Chronically Infected Promonocyte Cell Line
inferredcites →https://doi.org/10.1126/science.3422113openalex-snapshot
A Quantitative Bioassay for HIV-1 Based on Trans-Activation
inferredcites →https://doi.org/10.1126/science.3490693openalex-snapshot
Expression and Characterization of the Trans -Activator of HTLV-III/LAV Virus
inferredcites →https://doi.org/10.1128/jvi.67.12.7450-7460.1993openalex-snapshot
In vivo footprinting analysis of constitutive and inducible protein-DNA interactions at the long terminal repeat of human immunodeficiency virus type 1
inferredcites →https://doi.org/10.1128/jvi.69.5.2765-2775.1995openalex-snapshot
Molecular and functional interactions of transcription factor USF with the long terminal repeat of human immunodeficiency virus type 1
inferredcites →https://doi.org/10.1128/jvi.72.10.8252-8256.1998openalex-snapshot
Interaction of Human Immunodeficiency Virus Type 1 Tat with the Transcriptional Coactivators p300 and CREB Binding Protein
inferredcites →https://doi.org/10.1128/jvi.74.15.6790-6799.2000openalex-snapshot
The Human Factors YY1 and LSF Repress the Human Immunodeficiency Virus Type 1 Long Terminal Repeat via Recruitment of Histone Deacetylase 1
inferredcites →https://doi.org/10.1128/mcb.18.5.2535openalex-snapshot
Transcriptional Activation of the Integrated Chromatin-Associated Human Immunodeficiency Virus Type 1 Promoter
inferredcites →https://doi.org/10.1128/mcb.22.9.2965-2973.2002openalex-snapshot
Counterregulation of Chromatin Deacetylation and Histone Deacetylase Occupancy at the Integrated Promoter of Human Immunodeficiency Virus Type 1 (HIV-1) by the HIV-1 Repressor YY1 and HIV-1 Activator Tat
inferredcites →https://doi.org/10.1146/annurev.bi.63.070194.003441openalex-snapshot
CONTROL OF RNA INITIATION AND ELONGATION AT THE HIV-1 PROMOTER
inferredcites →https://doi.org/10.1146/annurev.immunol.18.1.665openalex-snapshot
Reservoirs for HIV-1: Mechanisms for Viral Persistence in the Presence of Antiviral Immune Responses and Antiretroviral Therapy
inferredcites →https://doi.org/10.4049/jimmunol.140.4.1117openalex-snapshot
Characterization of a promonocyte clone chronically infected with HIV and inducible by 13-phorbol-12-myristate acetate.

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Connecting HIV ‐1 integration and transcription: a step toward new treatments
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HDAC1/NFκB pathway is involved in curcumin inhibiting of Tat-mediated long terminal repeat transactivation
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Differences in Activation of HIV‐1 Replication by Superinfection With HIV‐1 and HIV‐2 in U1 Cells
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Chromatin-Associated Regulation Of Hiv-1 Transcription
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The Renaissance of Competitive PCR as an Accurate Tool for Precise Nucleic Acid Quantification
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Acetylation in Viral Infection and Disease
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Activating Latent HIV by Inhibiting Bromodomain Proteins
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Microbe-Mediated Genetic Engineering for Enhancement of Nutritional Value in Food Crops
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Subcellular localization of the interaction between the human immunodeficiency virus transactivator Tat and the nucleosome assembly protein 1
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Underlying mechanisms of HIV-1 latency
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Lysine-specific demethylase 1 cooperates with BRAF–histone deacetylase complex 80 to enhance HIV-1 Tat-mediated transactivation
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The transposon-driven evolutionary origin and basis of histone deacetylase functions and limitations in disease prevention
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Experimental Approaches Toward Histone Acetyltransferase Inhibitors as Therapeutics
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Experimental approaches toward histone acetyltransferase modulators as therapeutics
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Post-transcriptional regulation of HIV-1 gene expression
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Transcription: Insights From the HIV-1 Promoter
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Transcellular protein transduction using the Tat protein of HIV-1
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KDM5A/B contribute to HIV-1 latent infection and survival of HIV-1 infected cells
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Chromatin dynamics associated with HIV-1 Tat-activated transcription
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Tat-dependent repression of human immunodeficiency virus type 1 long terminal repeat promoter activity by fusion of cellular transcription factors
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Implications of small molecule activators and inhibitors of histone acetyltransferases in chromatin therapy
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Reversal of HIV-1 latency with anti-microRNA inhibitors
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Inhibitory effect of polysaccharide of Sargassum weizhouense on PCV2 induced inflammation in mice by suppressing histone acetylation
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Microbiota-derived short chain fatty acids: Their role and mechanisms in viral infections
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Sargassum polysaccharide inhibits inflammatory response in PCV2 infected-RAW264.7 cells by regulating histone acetylation
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Heterochromatin Silencing and Locus Repositioning Linked to Regulation of Virulence Genes in Plasmodium falciparum
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Toll-like receptor-3 mediates HIV-1 transactivation via NFκB and JNK pathways and histone acetylation, but prolonged activation suppresses Tat and HIV-1 replication
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NF-κB-Chromatin Interactions Drive Diverse Phenotypes by Modulating Transcriptional Noise
inferred← citeshttps://doi.org/10.1016/j.celrep.2023.112234openalex-snapshot
The lysine methyltransferase SMYD5 amplifies HIV-1 transcription and is post-transcriptionally upregulated by Tat and USP11
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Proximity to PML Nuclear Bodies Regulates HIV-1 Latency in CD4+ T Cells
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The development of immune-modulating compounds to disrupt HIV latency
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Pharmacological targeting of lysine acetyltransferases in human disease: a progress report
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Targeting HIV latency: pharmacologic strategies toward eradication
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Inhibition of Non Canonical HIV-1 Tat Secretion Through the Cellular Na + ,K + -ATPase Blocks HIV-1 Infection
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TFII-I and USF (RBF-2) regulate Ras/MAPK-responsive HIV-1 transcription in T cells
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Novel histone deacetylase inhibitor NCH-51 activates latent HIV-1 gene expression
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A dual selection based, targeted gene replacement tool for Magnaporthe grisea and Fusarium oxysporum
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Nuclear organization and the control of HIV-1 transcription
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CBP/p300 lysine acetyltransferases inhibit HIV-1 expression in latently infected T cells
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Nuclear translocation of triosephosphate isomerase 1 under aerobic glycolytic conditions is associated with HIV-1 transcription
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Effects of butyric acid on the periodontal tissue
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Examining the levels of acetylation, DNA methylation and phosphorylation in HIV-1 positive and multidrug-resistant TB-HIV patients
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Negative Regulation of HIV-1 Transcription by a Heterodimeric NF-κB1/p50 and C-Terminally Truncated STAT5 Complex
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HIV-1 Infection Induces Acetylation of NPM1 That Facilitates Tat Localization and Enhances Viral Transactivation
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Regulation of HIV-1 Latency by Chromatin Structure and Nuclear Architecture
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The HIV-1 Transcriptional Program: From Initiation to Elongation Control
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Human Origins of DNA Replication Selected from a Library of Nascent DNA
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Transcription-Dependent Gene Looping of the HIV-1 Provirus Is Dictated by Recognition of Pre-mRNA Processing Signals
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A pervasive role of histone acetyltransferases and deacetylases in an NF-κB-signaling code
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Viral cis-regulatory elements as sensors of cellular states and environmental cues
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Chromatin regulation of virus infection
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Proteomic profiling identifies nucleolin as a U3-interacting protein that inhibits avian leukosis virus replication
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BCL11B is a general transcriptional repressor of the HIV-1 long terminal repeat in T lymphocytes through recruitment of the NuRD complex
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Epigenetic regulation of CD8+ T-lymphocyte mediated suppression of HIV-1 replication
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IFI44 suppresses HIV-1 LTR promoter activity and facilitates its latency
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SIRT1 regulates Tat-induced HIV-1 transactivation through activating AMP-activated protein kinase
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Anti-viral opportunities during transcriptional activation of latent HIV in the host chromatin
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Waking Up the Sleepers: HIV Latency and Reactivation
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Transcription of HIV: Tat and Cellular Chromatin
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Butyric Acid Effects in the Development of Periodontitis and Systemic Diseases
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Lysine Acetylation Goes Global: From Epigenetics to Metabolism and Therapeutics
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Evaluation of EED Inhibitors as a Class of PRC2-Targeted Small Molecules for HIV Latency Reversal
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The Multifunctional Protein Nucleophosmin (NPM1) Is a Human Linker Histone H1 Chaperone
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Viral–Host Interactions That Control HIV-1 Transcriptional Elongation
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Transcriptional competence of the integrated HIV‐1 provirus at the nuclear periphery
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Fast transcription rates of RNA polymerase II in human cells
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Genetically Modified Hematopoietic Stem Cell Transplantation for HIV-1–infected Patients: Can We Achieve a Cure?
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CRISPR-mediated Activation of Latent HIV-1 Expression
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Competitive PCR for precise nucleic acid quantification
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Understanding HIV-1 latency provides clues for the eradication of long-term reservoirs
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The flow of gene expression
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Epigenetics, N-myrystoyltransferase-1 and casein kinase-2-alpha modulates the increased replication of HIV-1 CRF02_AG, compared to subtype-B viruses
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Butyrate administration is not sufficient to improve immune reconstitution in antiretroviral-treated SIV-infected macaques
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Acetylation of HIV‐1 integrase by p300 regulates viral integration
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Requirement for SWI/SNF chromatin‐remodeling complex in Tat‐mediated activation of the HIV‐1 promoter
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Suv39H1 and HP1γ are responsible for chromatin‐mediated HIV‐1 transcriptional silencing and post‐integration latency
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CBF‐1 promotes transcriptional silencing during the establishment of HIV‐1 latency
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Illegitimate DNA integration in mammalian cells
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Stable gene expression occurs from a minority of integrated HIV-1-based vectors: transcriptional silencing is present in the majority
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HIV-1 Vpr reactivates latent HIV-1 provirus by inducing depletion of class I HDACs on chromatin
inferred← citeshttps://doi.org/10.1073/pnas.1202174109openalex-snapshot
Amino acid starvation induces reactivation of silenced transgenes and latent HIV-1 provirus via down-regulation of histone deacetylase 4 (HDAC4)
inferred← citeshttps://doi.org/10.1074/jbc.m109.085324openalex-snapshot
Combinatorial Signals from CD28 Differentially Regulate Human Immunodeficiency Virus Transcription in T Cells
inferred← citeshttps://doi.org/10.1074/jbc.m110.103531openalex-snapshot
Involvement of Histone H3 Lysine 9 (H3K9) Methyltransferase G9a in the Maintenance of HIV-1 Latency and Its Reactivation by BIX01294
inferred← citeshttps://doi.org/10.1074/jbc.m110.185637openalex-snapshot
Recruitment of SWI/SNF Complex Is Required for Transcriptional Activation of the SLC11A1 Gene during Macrophage Differentiation of HL-60 Cells
inferred← citeshttps://doi.org/10.1074/jbc.m402839200openalex-snapshot
Polyisoprenylated Benzophenone, Garcinol, a Natural Histone Acetyltransferase Inhibitor, Represses Chromatin Transcription and Alters Global Gene Expression
inferred← citeshttps://doi.org/10.1074/jbc.m408069200openalex-snapshot
Retinoic Acid Inhibition of Chromatin Remodeling at the Human Immunodeficiency Virus Type 1 Promoter
inferred← citeshttps://doi.org/10.1074/jbc.m603336200openalex-snapshot
The SWI/SNF Chromatin-remodeling Complex Is a Cofactor for Tat Transactivation of the HIV Promoter
inferred← citeshttps://doi.org/10.1074/jbc.m608104200openalex-snapshot
The Werner Syndrome Helicase Is a Cofactor for HIV-1 Long Terminal Repeat Transactivation and Retroviral Replication
inferred← citeshttps://doi.org/10.1074/jbc.m610688200openalex-snapshot
Negative Elongation Factor NELF Represses Human Immunodeficiency Virus Transcription by Pausing the RNA Polymerase II Complex
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Genetic and epigenetic regulation of natural resistance to HIV-1 infection: new approaches to unveil the HESN secret
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FBXO34 promotes latent HIV-1 activation by post-transcriptional modulation
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The transcriptional cycle of HIV-1 in real-time and live cells
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Mechanism for the Establishment of Transcriptional HIV Latency in the Brain in a Simian Immunodeficiency Virus–Macaque Model
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Transcriptional Control of HIV Replication by Multiple Modulators and Their Implication for a Novel Antiviral Therapy
inferred← citeshttps://doi.org/10.1089/aid.2017.0180openalex-snapshot
Host Methyltransferases and Demethylases: Potential New Epigenetic Targets for HIV Cure Strategies and Beyond
inferred← citeshttps://doi.org/10.1089/aid.2018.0118openalex-snapshot
Barriers for HIV Cure: The Latent Reservoir
inferred← citeshttps://doi.org/10.1093/jac/dkr599openalex-snapshot
Achieving a cure for HIV infection: do we have reasons to be optimistic?
inferred← citeshttps://doi.org/10.1093/nar/gkaf1323openalex-snapshot
The human chromatin remodeling complex p400 restricts HIV-1 transcription in a Tat-dependent manner
inferred← citeshttps://doi.org/10.1093/nar/gkag631openalex-snapshot
Quantitative limits of host-driven HIV transcription and host gene control by the viral transactivator Tat
inferred← citeshttps://doi.org/10.1093/nar/gkh289openalex-snapshot
HIV-1 Tat directly binds to NF B enhancer sequence: role in viral and cellular gene expression
inferred← citeshttps://doi.org/10.1097/coh.0b013e328340ffbbopenalex-snapshot
The molecular biology of HIV latency: breaking and restoring the Tat-dependent transcriptional circuit
inferred← citeshttps://doi.org/10.1097/coh.0b013e3283412384openalex-snapshot
Epigenetic regulation of HIV latency
inferred← citeshttps://doi.org/10.1097/coh.0b013e328341242dopenalex-snapshot
Histone deacetylase inhibitors and HIV latency
inferred← citeshttps://doi.org/10.1097/qad.0000000000001159openalex-snapshot
Extracellular histones identified in crocodile blood inhibit in-vitro HIV-1 infection
inferred← citeshttps://doi.org/10.1097/qad.0b013e328361d059openalex-snapshot
M1 polarization of human monocyte-derived macrophages restricts pre and postintegration steps of HIV-1 replication
inferred← citeshttps://doi.org/10.1097/qai.0b013e3181d3dca3openalex-snapshot
The Histone Deacetylase Inhibitor ITF2357 Decreases Surface CXCR4 and CCR5 Expression on CD4+ T-Cells and Monocytes and is Superior to Valproic Acid for Latent HIV-1 Expression in Vitro
inferred← citeshttps://doi.org/10.1099/vir.0.013656-0openalex-snapshot
Integrase interactor 1 (Ini1/hSNF5) is a repressor of basal human immunodeficiency virus type 1 promoter activity
inferred← citeshttps://doi.org/10.1099/vir.0.041186-0openalex-snapshot
Deployment of the human immunodeficiency virus type 1 protein arsenal: combating the host to enhance viral transcription and providing targets for therapeutic development
inferred← citeshttps://doi.org/10.1099/vir.0.049296-0openalex-snapshot
Recent developments in human immunodeficiency virus-1 latency research
inferred← citeshttps://doi.org/10.1101/2022.11.17.516956openalex-snapshot
KDM5A/B promotes HIV-1 latency and KDM5 inhibitors promote HIV-1 lytic reactivation
inferred← citeshttps://doi.org/10.1101/2025.01.14.632503openalex-snapshot
Extreme high-diversity barcoded HIV library enables single cell multi-omics analysis of viral expression and fate determination
inferred← citeshttps://doi.org/10.1101/2025.01.31.635859openalex-snapshot
HIV-1 Vpr drives epigenetic remodeling to enhance virus transcription and latency reactivation
inferred← citeshttps://doi.org/10.1101/2025.11.26.690466openalex-snapshot
Single-Molecule DNA Footprinting and Transcription Imaging Reveal the Molecular Mechanisms of Promoter Dynamics
inferred← citeshttps://doi.org/10.1101/cshperspect.a039446openalex-snapshot
Viruses in the Nucleus
inferred← citeshttps://doi.org/10.1101/gad.1291705openalex-snapshot
A human splicing factor, SKIP, associates with P-TEFb and enhances transcription elongation by HIV-1 Tat
inferred← citeshttps://doi.org/10.1111/febs.17264openalex-snapshot
MOF ‐mediated acetylation of CDK 9 promotes global transcription by modulating P‐ TEF b complex formation
inferred← citeshttps://doi.org/10.1111/j.1574-6976.2012.00335.xopenalex-snapshot
HIV latency: experimental systems and molecular models
inferred← citeshttps://doi.org/10.1126/sciadv.adi4162openalex-snapshot
Enhanced NF-κB activation via HIV-1 Tat-TRAF6 cross-talk
inferred← citeshttps://doi.org/10.1126/scitranslmed.abh3351openalex-snapshot
Genome-wide CRISPR screens identify combinations of candidate latency reversing agents for targeting the latent HIV-1 reservoir
inferred← citeshttps://doi.org/10.1128/aac.01815-20openalex-snapshot
Epigenetic Compound Screening Uncovers Small Molecules for Reactivation of Latent HIV-1
inferred← citeshttps://doi.org/10.1128/aac.50.2.547-555.2006openalex-snapshot
Inhibition of Human Immunodeficiency Virus Type 1 Replication in Latently Infected Cells by a Novel IκB Kinase Inhibitor
inferred← citeshttps://doi.org/10.1128/jvi.00161-10openalex-snapshot
Combinatorial Latency Reactivation for HIV-1 Subtypes and Variants
inferred← citeshttps://doi.org/10.1128/jvi.00379-22openalex-snapshot
Heterogeneity of Latency Establishment in the Different Human CD4 + T Cell Subsets Stimulated with IL-15
inferred← citeshttps://doi.org/10.1128/jvi.00789-11openalex-snapshot
Nuclear Receptor Signaling Inhibits HIV-1 Replication in Macrophages through Multipletrans-Repression Mechanisms
inferred← citeshttps://doi.org/10.1128/jvi.00836-11openalex-snapshot
Epigenetic Silencing of HIV-1 by the Histone H3 Lysine 27 Methyltransferase Enhancer of Zeste 2
inferred← citeshttps://doi.org/10.1128/jvi.01208-07openalex-snapshot
c-Myc and Sp1 Contribute to Proviral Latency by Recruiting Histone Deacetylase 1 to the Human Immunodeficiency Virus Type 1 Promoter
inferred← citeshttps://doi.org/10.1128/jvi.01334-25openalex-snapshot
RNA splicing patterns contribute to burst size variation among HIV-1-infected Jurkat cell clones
inferred← citeshttps://doi.org/10.1128/jvi.01920-10openalex-snapshot
Chromatin Reassembly Factors Are Involved in Transcriptional Interference Promoting HIV Latency
inferred← citeshttps://doi.org/10.1128/jvi.02665-13openalex-snapshot
Histone Deacetylase Classes I and II Regulate Kaposi's Sarcoma-Associated Herpesvirus Reactivation
inferred← citeshttps://doi.org/10.1128/jvi.05972-11openalex-snapshot
Combination of Biological Screening in a Cellular Model of Viral Latency and Virtual Screening Identifies Novel Compounds That Reactivate HIV-1
inferred← citeshttps://doi.org/10.1128/jvi.78.17.9105-9114.2004openalex-snapshot
Analysis of Human Immunodeficiency Virus Type 1 Transcriptional Elongation in Resting CD4+T Cells In Vivo
inferred← citeshttps://doi.org/10.1128/jvi.78.22.12198-12206.2004openalex-snapshot
Cell Cycle Arrest in G 2 Induces Human Immunodeficiency Virus Type 1 Transcriptional Activation through Histone Acetylation and Recruitment of CBP, NF-κB, and c-Jun to the Long Terminal Repeat Promoter
inferred← citeshttps://doi.org/10.1128/jvi.79.7.4396-4406.2005openalex-snapshot
TFII-I Regulates Induction of Chromosomally Integrated Human Immunodeficiency Virus Type 1 Long Terminal Repeat in Cooperation with USF
inferred← citeshttps://doi.org/10.1128/mbio.00795-21openalex-snapshot
FKBP3 Induces Human Immunodeficiency Virus Type 1 Latency by Recruiting Histone Deacetylase 1/2 to the Viral Long Terminal Repeat
inferred← citeshttps://doi.org/10.1128/mcb.01557-07openalex-snapshot
Acetylation of Conserved Lysines in the Catalytic Core of Cyclin-Dependent Kinase 9 Inhibits Kinase Activity and Regulates Transcription
inferred← citeshttps://doi.org/10.1142/s0219720010004677openalex-snapshot
SNPS IN THE HIV-1 TATA BOX AND THE AIDS PANDEMIC
inferred← citeshttps://doi.org/10.1146/annurev-pharmtox-010510-100237openalex-snapshot
Curing HIV: Pharmacologic Approaches to Target HIV-1 Latency
inferred← citeshttps://doi.org/10.1172/jci.insight.123052openalex-snapshot
Determinants of the efficacy of HIV latency-reversing agents and implications for drug and treatment design
inferred← citeshttps://doi.org/10.1172/jci.insight.179680openalex-snapshot
The HIV latency reversing agent HODHBt inhibits the phosphatases PTPN1 and PTPN2
inferred← citeshttps://doi.org/10.1172/jci.insight.99127openalex-snapshot
HIV infection results in clonal expansions containing integrations within pathogenesis-related biological pathways
inferred← citeshttps://doi.org/10.1172/jci136227openalex-snapshot
The multifaceted nature of HIV latency
inferred← citeshttps://doi.org/10.1172/jci151380openalex-snapshot
The multifaceted nature of HIV latency
inferred← citeshttps://doi.org/10.1177/11779322211072333openalex-snapshot
Decoding Human Genome Regulatory Features That Influence HIV-1 Proviral Expression and Fate Through an Integrated Genomics Approach
inferred← citeshttps://doi.org/10.1177/1179555717694556openalex-snapshot
Functional Studies of CCAAT/Enhancer Binding Protein Site Located Downstream of the Transcriptional Start Site
inferred← citeshttps://doi.org/10.1182/blood-2006-08-042556openalex-snapshot
Naturally occurring C-terminally truncated STAT5 is a negative regulator of HIV-1 expression
inferred← citeshttps://doi.org/10.1186/1471-2199-8-63openalex-snapshot
HIV-1 TAR element is processed by Dicer to yield a viral micro-RNA involved in chromatin remodeling of the viral LTR
inferred← citeshttps://doi.org/10.1186/1742-4690-10-124openalex-snapshot
Transcription elongation regulator 1 (TCERG1) regulates competent RNA polymerase II-mediated elongation of HIV-1 transcription and facilitates efficient viral replication
inferred← citeshttps://doi.org/10.1186/1742-4690-10-67openalex-snapshot
HIV-1 transcription and latency: an update
inferred← citeshttps://doi.org/10.1186/1742-4690-2-69openalex-snapshot
Tat gets the "green" light on transcription initiation
inferred← citeshttps://doi.org/10.1186/1742-4690-2-73openalex-snapshot
Characterization of two candidate genes, NCoA3 and IRF8, potentially involved in the control of HIV-1 latency
inferred← citeshttps://doi.org/10.1186/1742-4690-3-7openalex-snapshot
Latency: the hidden HIV-1 challenge
inferred← citeshttps://doi.org/10.1186/1742-4690-5-8openalex-snapshot
The histone chaperone protein Nucleosome Assembly Protein-1 (hNAP-1) binds HIV-1 Tat and promotes viral transcription
inferred← citeshttps://doi.org/10.1186/1742-4690-5-98openalex-snapshot
Intragenic transcriptional cis-activation of the human immunodeficiency virus 1 does not result in allele-specific inhibition of the endogenous gene
inferred← citeshttps://doi.org/10.1186/1742-4690-6-111openalex-snapshot
Molecular control of HIV-1 postintegration latency: implications for the development of new therapeutic strategies
inferred← citeshttps://doi.org/10.1186/1742-4690-6-2openalex-snapshot
Human cyclin T1 expression ameliorates a T-cell-specific transcriptional limitation for HIV in transgenic rats, but is not sufficient for a spreading infection of prototypic R5 HIV-1 strains ex vivo
inferred← citeshttps://doi.org/10.1186/1743-422x-7-266openalex-snapshot
Cell line-dependent variability in HIV activation employing DNMT inhibitors
inferred← citeshttps://doi.org/10.1186/gb-2007-8-11-228openalex-snapshot
Chromatin, gene silencing and HIV latency
inferred← citeshttps://doi.org/10.1186/s12964-025-02221-zopenalex-snapshot
RYBP promotes HIV-1 latency through promoting H2AK119ub and decreasing H3K4me3
inferred← citeshttps://doi.org/10.1186/s12977-015-0169-1openalex-snapshot
Apoptosis-induced activation of HIV-1 in latently infected cell lines
inferred← citeshttps://doi.org/10.1186/s12977-018-0447-9openalex-snapshot
Cellular RelB interacts with the transactivator Tat and enhance HIV-1 expression
inferred← citeshttps://doi.org/10.1186/s12977-024-00639-wopenalex-snapshot
The cell biology of HIV-1 latency and rebound
inferred← citeshttps://doi.org/10.1186/s12985-016-0637-9openalex-snapshot
Activation of HIV-1 expression in latently infected CD4+ T cells by the small molecule PKC412
inferred← citeshttps://doi.org/10.1186/s13068-018-1296-4openalex-snapshot
Constitutive hyperproduction of sorbicillinoids in Trichoderma reesei ZC121
inferred← citeshttps://doi.org/10.1186/s13072-019-0267-8openalex-snapshot
Tat inhibition by didehydro-Cortistatin A promotes heterochromatin formation at the HIV-1 long terminal repeat
inferred← citeshttps://doi.org/10.1186/s13072-025-00573-xopenalex-snapshot
Impact of chromatin on HIV-1 latency: a multi-dimensional perspective
inferred← citeshttps://doi.org/10.1186/s13072-025-00582-wopenalex-snapshot
Human chromatin remodelers regulating HIV-1 transcription: a target for small molecule inhibitors
inferred← citeshttps://doi.org/10.1186/s13148-015-0137-6openalex-snapshot
Epigenetic control of HIV-1 post integration latency: implications for therapy
inferred← citeshttps://doi.org/10.1186/s13148-018-0448-5openalex-snapshot
Expression profiling of chromatin-modifying enzymes and global DNA methylation in CD4+ T cells from patients with chronic HIV infection at different HIV control and progression states
inferred← citeshttps://doi.org/10.1189/jlb.0409264openalex-snapshot
HIV-1 regulation of latency in the monocyte-macrophage lineage and in CD4+ T lymphocytes
inferred← citeshttps://doi.org/10.1201/9780824748401.ch10openalex-snapshot
Therapeutic Strategies
inferred← citeshttps://doi.org/10.1242/jcs.003632openalex-snapshot
Processing of recombinant AAV genomes occurs in specific nuclear structures that overlap with foci of DNA-damage-response proteins
inferred← citeshttps://doi.org/10.12998/wjcc.v9.i7.1513openalex-snapshot
Regulation of the expression of proinflammatory cytokines induced by SARS-CoV-2
inferred← citeshttps://doi.org/10.1371/annotation/45587774-5d7c-4023-97b0-96833be7ad9aopenalex-snapshot
Correction: Yeast Genetic Analysis Reveals the Involvement of Chromatin Reassembly Factors in Repressing HIV-1 Basal Transcription
inferred← citeshttps://doi.org/10.1371/journal.pbio.1001206openalex-snapshot
Repressive LTR Nucleosome Positioning by the BAF Complex Is Required for HIV Latency
inferred← citeshttps://doi.org/10.1371/journal.pbio.3003621openalex-snapshot
DNA damage induced by HIV-1 Vpr triggers epigenetic remodeling and transcriptional programs to enhance virus transcription and latency reactivation
inferred← citeshttps://doi.org/10.1371/journal.pgen.1000339openalex-snapshot
Yeast Genetic Analysis Reveals the Involvement of Chromatin Reassembly Factors in Repressing HIV-1 Basal Transcription
inferred← citeshttps://doi.org/10.1371/journal.pgen.1000927openalex-snapshot
Epigenetic Regulation of a Murine Retrotransposon by a Dual Histone Modification Mark
inferred← citeshttps://doi.org/10.1371/journal.pone.0000151openalex-snapshot
Recruitment and Activation of RSK2 by HIV-1 Tat
inferred← citeshttps://doi.org/10.1371/journal.pone.0006093openalex-snapshot
Synergistic Activation of HIV-1 Expression by Deacetylase Inhibitors and Prostratin: Implications for Treatment of Latent Infection
inferred← citeshttps://doi.org/10.1371/journal.pone.0048832openalex-snapshot
Selective Histonedeacetylase Inhibitor M344 Intervenes in HIV-1 Latency through Increasing Histone Acetylation and Activation of NF-kappaB
inferred← citeshttps://doi.org/10.1371/journal.pone.0102684openalex-snapshot
Selective HDAC Inhibition for the Disruption of Latent HIV-1 Infection
inferred← citeshttps://doi.org/10.1371/journal.ppat.1000260openalex-snapshot
Control of Stochastic Gene Expression by Host Factors at the HIV Promoter
inferred← citeshttps://doi.org/10.1371/journal.ppat.1002620openalex-snapshot
Regulation of Mycobacterium tuberculosis-Dependent HIV-1 Transcription Reveals a New Role for NFAT5 in the Toll-Like Receptor Pathway
inferred← citeshttps://doi.org/10.1371/journal.ppat.1006460openalex-snapshot
Digoxin reveals a functional connection between HIV-1 integration preference and T-cell activation
inferred← citeshttps://doi.org/10.1371/journal.ppat.1009147openalex-snapshot
Mutations altering acetylated residues in the CTD of HIV-1 integrase cause defects in proviral transcription at early times after integration of viral DNA
inferred← citeshttps://doi.org/10.1371/journal.ppat.1009207openalex-snapshot
Three-dimensional chromatin in infectious disease—A role for gene regulation and pathogenicity?
inferred← citeshttps://doi.org/10.1371/journal.ppat.1011101openalex-snapshot
A modular CRISPR screen identifies individual and combination pathways contributing to HIV-1 latency
inferred← citeshttps://doi.org/10.14288/1.0383231openalex-snapshot
Small molecules effective for disruption of HIV-1 latency
inferred← citeshttps://doi.org/10.1517/14728222.11.6.835openalex-snapshot
The therapeutic uses of chromatin-modifying agents
inferred← citeshttps://doi.org/10.15252/embj.201487870openalex-snapshot
K‐Lysine acetyltransferase 2a regulates a hippocampal gene expression network linked to memory formation
inferred← citeshttps://doi.org/10.1586/14787210.3.5.805openalex-snapshot
Molecular mechanisms of HIV-1 proviral latency
inferred← citeshttps://doi.org/10.17615/a6ry-q486openalex-snapshot
Investigation of HIV cure strategies in vivo in BLT humanized mice
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