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Transcriptional Activation of the Integrated Chromatin-Associated Human Immunodeficiency Virus Type 1 Promoter

https://doi.org/10.1128/mcb.18.5.2535
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/bies.950170510openalex-snapshot
Trichostatin A and trapoxin: Novel chemical probes for the role of histone acetylation in chromatin structure and function
inferredcites →https://doi.org/10.1002/j.1460-2075.1991.tb04997.xopenalex-snapshot
HIV‐1 Tat protein promotes formation of more‐processive elongation complexes.
inferredcites →https://doi.org/10.1002/j.1460-2075.1993.tb05994.xopenalex-snapshot
Chromatin disruption in the promoter of human immunodeficiency virus type 1 during transcriptional activation.
inferredcites →https://doi.org/10.1002/j.1460-2075.1993.tb06029.xopenalex-snapshot
A cooperative interaction between NF‐kappa B and Sp1 is required for HIV‐1 enhancer activation.
inferredcites →https://doi.org/10.1002/j.1460-2075.1996.tb00449.xopenalex-snapshot
Transcriptional activation and chromatin remodeling of the HIV‐1 promoter in response to histone acetylation.
inferredcites →https://doi.org/10.1002/j.1460-2075.1996.tb00607.xopenalex-snapshot
Tissue‐specific factors additively increase the probability of the all‐or‐none formation of a hypersensitive site.
inferredcites →https://doi.org/10.1006/scel.1995.0031openalex-snapshot
Histone acetylation in chromatin and chromosomes
inferredcites →https://doi.org/10.1006/viro.1993.1505openalex-snapshot
Sodium Butyrate Treatment of Cells Latently Infected with HIV-1 Results in the Expression of Unspliced Viral RNA
inferredcites →https://doi.org/10.1007/978-3-642-78929-8_3openalex-snapshot
Regulation of HIV-1 Gene Expression by the Transactivator Protein Tat
inferredcites →https://doi.org/10.1007/bf00337382openalex-snapshot
Histone acetylation: facts and questions
inferredcites →https://doi.org/10.1016/0092-8674(89)90289-4openalex-snapshot
Tat trans-activates the human immunodeficiency virus through a nascent RNA target
inferredcites →https://doi.org/10.1016/0092-8674(90)90120-4openalex-snapshot
TAR-independent activation of the HIV-1 LTR: Evidence that Tat requires specific regions of the promoter
inferredcites →https://doi.org/10.1016/0092-8674(91)90237-sopenalex-snapshot
Activation domains of stably bound GAL4 derivatives alleviate repression of promoters by nucleosomes
inferredcites →https://doi.org/10.1016/0955-0674(93)90006-copenalex-snapshot
Role of chromatin structure in the regulation of transcription by RNA polymerase II
inferredcites →https://doi.org/10.1016/s0021-9258(17)32107-5openalex-snapshot
Protein-DNA interactions within DNase I-hypersensitive sites located downstream of the HIV-1 promoter.
inferredcites →https://doi.org/10.1016/s0021-9258(17)32475-4openalex-snapshot
Mutational analysis of interferon (IFN) regulatory factors 1 and 2. Effects on the induction of IFN-beta gene expression
inferredcites →https://doi.org/10.1016/s0092-8674(00)80978-2openalex-snapshot
RNA Polymerase II Holoenzyme Contains SWI/SNF Regulators Involved in Chromatin Remodeling
inferredcites →https://doi.org/10.1016/s0092-8674(88)91087-2openalex-snapshot
The RNA polymerase II molecule at the 5′ end of the uninduced hsp70 gene of D. melanogaster is transcriptionally engaged
inferredcites →https://doi.org/10.1016/s0959-437x(97)80127-xopenalex-snapshot
Chromatin remodeling and transcription
inferredcites →https://doi.org/10.1016/s0959-437x(97)80129-3openalex-snapshot
Mechanism and regulation of transcriptional elongation and termination by RNA polymerase II
inferredcites →https://doi.org/10.1038/330489a0openalex-snapshot
Anti-termination of transcription within the long terminal repeat of HIV-1 by tat gene product
inferredcites →https://doi.org/10.1038/370075a0openalex-snapshot
Phosphorylation of RNA polymerase II C-terminal domain and transcriptional elongation
inferredcites →https://doi.org/10.1038/384375a0openalex-snapshot
Enhanced processivity of RNA polymerase II triggered by Tat-induced phosphorylation of its carboxy-terminal domain
inferredcites →https://doi.org/10.1038/387183a0openalex-snapshot
Quantification of latent tissue reservoirs and total body viral load in HIV-1 infection
inferredcites →https://doi.org/10.1038/nm1295-1284openalex-snapshot
In vivo fate of HIV-1-infected T cells: Quantitative analysis of the transition to stable latency
inferredcites →https://doi.org/10.1073/pnas.80.24.7551openalex-snapshot
Human fetal to adult hemoglobin switching: changes in chromatin structure of the beta-globin gene locus.
inferredcites →https://doi.org/10.1073/pnas.88.9.4045openalex-snapshot
The role of Tat in the human immunodeficiency virus life cycle indicates a primary effect on transcriptional elongation.
inferredcites →https://doi.org/10.1073/pnas.91.9.3862openalex-snapshot
Cellular latency in human immunodeficiency virus-infected individuals with high CD4 levels can be detected by the presence of promoter-proximal transcripts.
inferredcites →https://doi.org/10.1073/pnas.93.21.11575openalex-snapshot
Trans-activation by human immunodeficiency virus Tat protein requires the C-terminal domain of RNA polymerase II.
inferredcites →https://doi.org/10.1073/pnas.93.5.2089openalex-snapshot
Human immunodeficiency virus type 1 and 2 Tat proteins specifically interact with RNA polymerase II.
inferredcites →https://doi.org/10.1074/jbc.271.43.27176openalex-snapshot
Control of RNA Polymerase II Elongation Potential by a Novel Carboxyl-terminal Domain Kinase
inferredcites →https://doi.org/10.1093/emboj/16.9.2463openalex-snapshot
Stable co‐occupancy of transcription factors and histones at the HIV‐1 enhancer
inferredcites →https://doi.org/10.1093/nar/15.24.10159openalex-snapshot
Evidence for a locus activation region: the formation of developmentally stable hypersensitive sites in globin-expressing hybrids
inferredcites →https://doi.org/10.1093/nar/17.9.3551openalex-snapshot
Multiple functional domains of Tat, thetrans-activator of HIV-1, defined by mutational analysis
inferredcites →https://doi.org/10.1097/00042560-199508000-00002openalex-snapshot
Sodium Butyrate Stimulation of HIV-1 Gene Expression
inferredcites →https://doi.org/10.1101/gad.10.1.37openalex-snapshot
NF-kappa B-mediated chromatin reconfiguration and transcriptional activation of the HIV-1 enhancer in vitro.
inferredcites →https://doi.org/10.1101/gad.10.12.1479openalex-snapshot
Activator-dependent regulation of transcriptional pausing on nucleosomal templates.
inferredcites →https://doi.org/10.1101/gad.3.4.547openalex-snapshot
Structure, sequence, and position of the stem-loop in tar determine transcriptional elongation by tat through the HIV-1 long terminal repeat.
inferredcites →https://doi.org/10.1101/gad.4.4.659openalex-snapshot
SV40 stimulates expression of the transacting factor Sp1 at the mRNA level.
inferredcites →https://doi.org/10.1101/gad.4.8.1365openalex-snapshot
A bulge structure in HIV-1 TAR RNA is required for Tat binding and Tat-mediated trans-activation.
inferredcites →https://doi.org/10.1101/gad.5.4.683openalex-snapshot
Transcription on nucleosomal templates by RNA polymerase II in vitro: inhibition of elongation with enhancement of sequence-specific pausing.
inferredcites →https://doi.org/10.1101/gad.6.4.655openalex-snapshot
HIV-1 Tat acts as a processivity factor in vitro in conjunction with cellular elongation factors.
inferredcites →https://doi.org/10.1101/gad.9.17.2090openalex-snapshot
Activation of the HIV-1 enhancer by the LEF-1 HMG protein on nucleosome-assembled DNA in vitro.
inferredcites →https://doi.org/10.1101/gad.9.5.559openalex-snapshot
Promoter-proximal pausing of RNA polymerase II defines a general rate-limiting step after transcription initiation.
inferredcites →https://doi.org/10.1126/science.1347958openalex-snapshot
Transcription Factor Loading on the MMTV Promoter: A Bimodal Mechanism for Promoter Activation
inferredcites →https://doi.org/10.1126/science.274.5287.605openalex-snapshot
Tat-SF1: Cofactor for Stimulation of Transcriptional Elongation by HIV-1 Tat
inferredcites →https://doi.org/10.1126/science.3313729openalex-snapshot
Cytokine-Induced Expression of HIV-1 in a Chronically Infected Promonocyte Cell Line
inferredcites →https://doi.org/10.1128/jvi.65.8.4350-4358.1991openalex-snapshot
Contribution of NF-kappa B and Sp1 binding motifs to the replicative capacity of human immunodeficiency virus type 1: distinct patterns of viral growth are determined by T-cell types
inferredcites →https://doi.org/10.1128/jvi.67.12.7450-7460.1993openalex-snapshot
In vivo footprinting analysis of constitutive and inducible protein-DNA interactions at the long terminal repeat of human immunodeficiency virus type 1
inferredcites →https://doi.org/10.1128/jvi.67.3.1658-1662.1993openalex-snapshot
Replication of type 1 human immunodeficiency viruses containing linker substitution mutations in the -201 to -130 region of the long terminal repeat
inferredcites →https://doi.org/10.1128/jvi.69.3.1612-1620.1995openalex-snapshot
Lentivirus Tat proteins specifically associate with a cellular protein kinase, TAK, that hyperphosphorylates the carboxyl-terminal domain of the large subunit of RNA polymerase II: candidate for a Tat cofactor
inferredcites →https://doi.org/10.1128/jvi.70.7.4576-4584.1996openalex-snapshot
The human immunodeficiency virus Tat proteins specifically associate with TAK in vivo and require the carboxyl-terminal domain of RNA polymerase II for function
inferredcites →https://doi.org/10.1128/jvi.71.7.5495-5504.1997openalex-snapshot
The kappaB sites in the human immunodeficiency virus type 1 long terminal repeat enhance virus replication yet are not absolutely required for viral growth
inferredcites →https://doi.org/10.1128/mcb.12.2.685openalex-snapshot
A novel mitogen-inducible gene product related to p50/p105-NF-kappa B participates in transactivation through a kappa B site.
inferredcites →https://doi.org/10.1128/mcb.16.5.2044openalex-snapshot
Three Functional Classes of Transcriptional Activation Domains
inferredcites →https://doi.org/10.1128/mcb.16.6.2958openalex-snapshot
cis-Acting Sequences Located Downstream of the Human Immunodeficiency Virus Type 1 Promoter Affect Its Chromatin Structure and Transcriptional Activity
inferredcites →https://doi.org/10.1128/mcb.17.4.1817openalex-snapshot
The Human Immunodeficiency Virus Transactivator Tat Interacts with the RNA Polymerase II Holoenzyme
inferredcites →https://doi.org/10.1146/annurev.bi.57.070188.001111openalex-snapshot
NUCLEASE HYPERSENSITIVE SITES IN CHROMATIN
inferredcites →https://doi.org/10.1146/annurev.bi.63.070194.001405openalex-snapshot
ROLE OF CHROMATIN STRUCTURE IN THE REGULATION OF TRANSCRIPTION BY RNA POLYMERASE II
inferredcites →https://doi.org/10.1146/annurev.bi.63.070194.003441openalex-snapshot
CONTROL OF RNA INITIATION AND ELONGATION AT THE HIV-1 PROMOTER
inferredcites →https://doi.org/10.1146/annurev.cb.08.110192.003023openalex-snapshot
Chromatin Structure and Transcription
inferredcites →https://doi.org/10.4049/jimmunol.142.2.431openalex-snapshot
Monokine regulation of human immunodeficiency virus-1 expression in a chronically infected human T cell clone.

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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HIV-1-induced type I IFNs promote viral latency in macrophages
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Histone deacetylase inhibitor chidamide promotes reactivation of latent human immunodeficiency virus by introducing histone acetylation
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Acetylation of HIV-1 Tat by CBP/P300 Increases Transcription of Integrated HIV-1 Genome and Enhances Binding to Core Histones
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Enhancement of the p300 HAT Activity by HIV-1 Tat on Chromatin DNA
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CCAAT/Enhancer Binding Proteins Are Not Required for HIV-1 Entry but Regulate Proviral Transcription by Recruiting Coactivators to the Long-Terminal Repeat in Monocytic Cells
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Transcriptional Control of Proteases
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Chromatin-Associated Regulation Of Hiv-1 Transcription
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Induction and Repression of NF-κB-Driven Inflammatory Genes
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Genetic variation and function of the HIV-1 Tat protein
inferred← citeshttps://doi.org/10.1007/s11033-010-0653-6openalex-snapshot
Histonedeacetylase inhibitor Oxamflatin increase HIV-1 transcription by inducing histone modification in latently infected cells
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Induction of chromosomally integrated HIV-1 LTR requires RBF-2 (USF/TFII-I) and RAS/MAPK signaling
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Underlying mechanisms of HIV-1 latency
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Silencing suppressors: viral weapons for countering host cell defenses
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Tat-dependent repression of human immunodeficiency virus type 1 long terminal repeat promoter activity by fusion of cellular transcription factors
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Regulation at multiple levels of NF-κB-mediated transactivation by protein acetylation
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Administration of HDAC inhibitors to reactivate HIV-1 expression in latent cellular reservoirs: implications for the development of therapeutic strategies
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HIV-1 protease inhibitors do not interfere with provirus transcription and host cell apoptosis induced by combined treatment TNF-α+TSA
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Influenza virus activates human immunodeficiency virus type-1 gene expression in human CD4-expressing T cells through an NF-κB-dependent pathway
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Regulation of HIV-1 latency by T-cell activation
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Nuclear organization and the control of HIV-1 transcription
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Lineage-specific and ubiquitous biological roles of the mammalian transcription factor LSF
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Targeting the Latent Reservoir for HIV-1
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Regulation of HIV-1 Latency by Chromatin Structure and Nuclear Architecture
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The HIV-1 Transcriptional Program: From Initiation to Elongation Control
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Epigenetic regulation of high glucose-induced proinflammatory cytokine production in monocytes by curcumin
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Differential expression and interaction of host factors augment HIV-1 gene expression in neonatal mononuclear cells
inferred← citeshttps://doi.org/10.1016/j.virol.2010.06.001openalex-snapshot
Epigenetic regulation of CD8+ T-lymphocyte mediated suppression of HIV-1 replication
inferred← citeshttps://doi.org/10.1016/s0002-9440(10)63675-9openalex-snapshot
Transgenic Mice Demonstrate Novel Promoter Regions for Tissue-Specific Expression of the Urokinase Receptor Gene
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Signal transduction by tumor necrosis factor and gene regulation of the inflammatory cytokine interleukin-6
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Diversity of acetylation targets and roles in transcriptional regulation: the human immunodeficiency virus type 1 promoter as a model system
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Glucocorticoids suppress human immunodeficiency virus type-1 long terminal repeat activity in a cell type-specific, glucocorticoid receptor-mediated fashion: direct protective effects at variance with clinical phenomenology
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Role of chromatin in HIV-1 transcriptional regulation
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NF-κB and HIV: Linking viral and immune activation
inferred← citeshttps://doi.org/10.1016/s1097-2765(02)00477-xopenalex-snapshot
The Phosphorylation Status of Nuclear NF-ΚB Determines Its Association with CBP/p300 or HDAC-1
inferred← citeshttps://doi.org/10.1016/s1097-2765(04)00004-8openalex-snapshot
HIV-1 Nef Mimics an Integrin Receptor Signal that Recruits the Polycomb Group Protein Eed to the Plasma Membrane
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HIV basic virology for clinicians
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Chromatin control of HIV‐1 gene expression
inferred← citeshttps://doi.org/10.1038/sj.emboj.7601074openalex-snapshot
Requirement for SWI/SNF chromatin‐remodeling complex in Tat‐mediated activation of the HIV‐1 promoter
inferred← citeshttps://doi.org/10.1038/sj.emboj.7601517openalex-snapshot
Suv39H1 and HP1γ are responsible for chromatin‐mediated HIV‐1 transcriptional silencing and post‐integration latency
inferred← citeshttps://doi.org/10.1038/srep43221openalex-snapshot
Repression of Human T-lymphotropic virus type 1 Long Terminal Repeat sense transcription by Sp1 recruitment to novel Sp1 binding sites
inferred← citeshttps://doi.org/10.1046/j.1523-1755.2001.00732.xopenalex-snapshot
Nuclear factor-κB binding to the HIV-1 LTR in kidney: Implications for HIV-associated nephropathy
inferred← citeshttps://doi.org/10.1073/pnas.95.23.13519openalex-snapshot
HIV-1 Tat transactivator recruits p300 and CREB-binding protein histone acetyltransferases to the viral promoter
inferred← citeshttps://doi.org/10.1074/jbc.274.3.1635openalex-snapshot
HEED, the Product of the Human Homolog of the Murineeed Gene, Binds to the Matrix Protein of HIV-1
inferred← citeshttps://doi.org/10.1074/jbc.m102441200openalex-snapshot
Initiation of HIV-1 Reverse Transcription Is Regulated by a Primer Activation Signal
inferred← citeshttps://doi.org/10.1074/jbc.m201895200openalex-snapshot
Tat Acetyl-acceptor Lysines Are Important for Human Immunodeficiency Virus Type-1 Replication
inferred← citeshttps://doi.org/10.1074/jbc.m210470200openalex-snapshot
The Low Viral Production in Trophoblastic Cells Is Due to a High Endocytic Internalization of the Human Immunodeficiency Virus Type 1 and Can Be Overcome by the Pro-inflammatory Cytokines Tumor Necrosis Factor-α and Interleukin-1
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Prostratin Antagonizes HIV Latency by Activating NF-κB
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Retinoic Acid Inhibition of Chromatin Remodeling at the Human Immunodeficiency Virus Type 1 Promoter
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Genetic and epigenetic regulation of natural resistance to HIV-1 infection: new approaches to unveil the HESN secret
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Polyamides Reveal a Role for Repression in Latency within Resting T Cells of HIV‐Infected Donors
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Mechanism for the Establishment of Transcriptional HIV Latency in the Brain in a Simian Immunodeficiency Virus–Macaque Model
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The Regulation of HIV-1 Gene Expression: The Emerging Role of Chromatin
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Expression of Latent HIV Induced by the Potent HDAC Inhibitor Suberoylanilide Hydroxamic Acid
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Short Communication: Activation of Latent HIV Type 1 Gene Expression by Suberoylanilide Hydroxamic Acid (SAHA), an HDAC Inhibitor Approved for Use to Treat Cutaneous T Cell Lymphoma
inferred← citeshttps://doi.org/10.1093/emboj/20.7.1726openalex-snapshot
The site of HIV‐1 integration in the human genome determines basal transcriptional activity and response to Tat transactivation
inferred← citeshttps://doi.org/10.1093/emboj/cdg631openalex-snapshot
Regulation of HIV‐1 gene expression by histone acetylation and factor recruitment at the LTR promoter
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Achieving a cure for HIV infection: do we have reasons to be optimistic?
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Stabilization of the U5-leader stem in the HIV-1 RNA genome affects initiation and elongation of reverse transcription
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HIV-1 Tat directly binds to NF B enhancer sequence: role in viral and cellular gene expression
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A novel homologous recombination system to study 92 kDa type IV collagenase transcription demonstrates that the NFκB motif drives the transition from a repressed to an activated state of gene expression
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Coaxing HIV-1 from resting CD4 T cells
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Epigenetic regulation of HIV latency
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Expression of latent human immunodeficiency type 1 is induced by novel and selective histone deacetylase inhibitors
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The SMAC Mimetic AZD5582 is a Potent HIV Latency Reversing Agent
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HIV Provirus Stably Reproduces Parental Latent and Induced Transcription Phenotypes Regardless of the Chromosomal Integration Site
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Targeting IκB Proteins for HIV Latency Activation: the Role of Individual IκB and NF-κB Proteins
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Human Immunodeficiency Virus Type 1 (HIV-1) Vpr Functions as an Immediate-Early Protein during HIV-1 Infection
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Human Immunodeficiency Virus Type 1 Tat Protein Activates Transcription Factor NF-κB through the Cellular Interferon-Inducible, Double-Stranded RNA-Dependent Protein Kinase, PKR
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The Human Factors YY1 and LSF Repress the Human Immunodeficiency Virus Type 1 Long Terminal Repeat via Recruitment of Histone Deacetylase 1
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Endothelial Cells Enhance Human Immunodeficiency Virus Type 1 Replication in Macrophages through a C/EBP-Dependent Mechanism
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In Vivo Analysis of Retroviral Enhancer Mutations in Hematopoietic Cells: SP1/EGR1 and ETS/GATA Motifs Contribute to Long Terminal Repeat Specificity
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Synergistic Activation of Human Immunodeficiency Virus Type 1 Promoter Activity by NF-κB and Inhibitors of Deacetylases: Potential Perspectives for the Development of Therapeutic Strategies
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Differential Requirements for Activation of Integrated and Transiently Transfected Human T-Cell Leukemia Virus Type 1 Long Terminal Repeat
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Host Cell Gene Expression during Human Immunodeficiency Virus Type 1 Latency and Reactivation and Effects of Targeting Genes That Are Differentially Expressed in Viral Latency
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Cell Cycle Arrest in G 2 Induces Human Immunodeficiency Virus Type 1 Transcriptional Activation through Histone Acetylation and Recruitment of CBP, NF-κB, and c-Jun to the Long Terminal Repeat Promoter
inferred← citeshttps://doi.org/10.1128/jvi.79.7.4316-4328.2005openalex-snapshot
Tetraspanin CD81 Provides a Costimulatory Signal Resulting in Increased Human Immunodeficiency Virus Type 1 Gene Expression in Primary CD4+T Lymphocytes through NF-κB, NFAT, and AP-1 Transduction Pathways
inferred← citeshttps://doi.org/10.1128/jvi.80.9.4570-4579.2006openalex-snapshot
Hexamethylbisacetamide Remodels the Human Immunodeficiency Virus Type 1 (HIV-1) Promoter and Induces Tat-Independent HIV-1 Expression but Blunts Cell Activation
inferred← citeshttps://doi.org/10.1128/mcb.21.20.7065-7077.2001openalex-snapshot
The p65 (RelA) Subunit of NF-κB Interacts with the Histone Deacetylase (HDAC) Corepressors HDAC1 and HDAC2 To Negatively Regulate Gene Expression
inferred← citeshttps://doi.org/10.1128/mcb.22.12.4043-4052.2002openalex-snapshot
Chromatin Disruption and Histone Acetylation in Regulation of the Human Immunodeficiency Virus Type 1 Long Terminal Repeat by Thyroid Hormone Receptor
inferred← citeshttps://doi.org/10.1128/mcb.22.9.2965-2973.2002openalex-snapshot
Counterregulation of Chromatin Deacetylation and Histone Deacetylase Occupancy at the Integrated Promoter of Human Immunodeficiency Virus Type 1 (HIV-1) by the HIV-1 Repressor YY1 and HIV-1 Activator Tat
inferred← citeshttps://doi.org/10.1128/mcb.23.17.6200-6209.2003openalex-snapshot
Potentiation of Tumor Necrosis Factor-Induced NF-κB Activation by Deacetylase Inhibitors Is Associated with a Delayed Cytoplasmic Reappearance of IκBα
inferred← citeshttps://doi.org/10.1128/mcb.24.15.6890.2004openalex-snapshot
Potentiation of Tumor Necrosis Factor-Induced NF-κB Activation by Deacetylase Inhibitors Is Associated with a Delayed Cytoplasmic Reappearance of IκBα
inferred← citeshttps://doi.org/10.1139/o02-079openalex-snapshot
The promyelocytic leukemia nuclear body: sites of activity?
inferred← citeshttps://doi.org/10.1155/2012/123637openalex-snapshot
HTLV-1 Infection and Its Associated Diseases
inferred← citeshttps://doi.org/10.1155/2012/574967openalex-snapshot
Molecular Understanding of HIV-1 Latency
inferred← citeshttps://doi.org/10.1155/2012/614120openalex-snapshot
Mechanisms of HIV Transcriptional Regulation and Their Contribution to Latency
inferred← citeshttps://doi.org/10.1155/2012/984754openalex-snapshot
Cotranscriptional Chromatin Remodeling by Small RNA Species: An HTLV-1 Perspective
inferred← citeshttps://doi.org/10.1155/2013/484378openalex-snapshot
TNF and TNF Receptor Superfamily Members in HIV infection: New Cellular Targets for Therapy?
inferred← citeshttps://doi.org/10.1161/01.atv.0000247247.89787.e7openalex-snapshot
Histone Deacetylase Inhibitor Reduces Monocyte Adhesion to Endothelium Through the Suppression of Vascular Cell Adhesion Molecule-1 Expression
inferred← citeshttps://doi.org/10.1177/1179555717694556openalex-snapshot
Functional Studies of CCAAT/Enhancer Binding Protein Site Located Downstream of the Transcriptional Start Site
inferred← citeshttps://doi.org/10.1186/1742-4690-2-6openalex-snapshot
HIV-1 Tat interacts with LIS1 protein
inferred← citeshttps://doi.org/10.1186/1742-4690-3-48openalex-snapshot
Effect of SWI/SNF chromatin remodeling complex on HIV-1 Tat activated transcription
inferred← citeshttps://doi.org/10.1186/1742-4690-5-8openalex-snapshot
The histone chaperone protein Nucleosome Assembly Protein-1 (hNAP-1) binds HIV-1 Tat and promotes viral transcription
inferred← citeshttps://doi.org/10.1186/1742-4690-6-111openalex-snapshot
Molecular control of HIV-1 postintegration latency: implications for the development of new therapeutic strategies
inferred← citeshttps://doi.org/10.1186/1742-4690-8-73openalex-snapshot
Latency profiles of full length HIV-1 molecular clone variants with a subtype specific promoter
inferred← citeshttps://doi.org/10.1186/1743-422x-10-309openalex-snapshot
Extracts from Acacia catechu suppress HIV-1 replication by inhibiting the activities of the viral protease and Tat
inferred← citeshttps://doi.org/10.1186/1743-422x-7-266openalex-snapshot
Cell line-dependent variability in HIV activation employing DNMT inhibitors
inferred← citeshttps://doi.org/10.1186/s40164-025-00655-2openalex-snapshot
Molecular mechanisms of viral oncogenesis in haematological malignancies: perspectives from metabolic reprogramming, epigenetic regulation and immune microenvironment remodeling
inferred← citeshttps://doi.org/10.1371/journal.pbio.1001206openalex-snapshot
Repressive LTR Nucleosome Positioning by the BAF Complex Is Required for HIV Latency
inferred← citeshttps://doi.org/10.1371/journal.pone.0006093openalex-snapshot
Synergistic Activation of HIV-1 Expression by Deacetylase Inhibitors and Prostratin: Implications for Treatment of Latent Infection
inferred← citeshttps://doi.org/10.1371/journal.pone.0034490openalex-snapshot
HTLV-1 Tax Mediated Downregulation of miRNAs Associated with Chromatin Remodeling Factors in T Cells with Stably Integrated Viral Promoter
inferred← citeshttps://doi.org/10.1371/journal.pone.0088116openalex-snapshot
CCAAT Enhancer Binding Protein and Nuclear Factor of Activated T Cells Regulate HIV-1 LTR via a Novel Conserved Downstream Site in Cells of the Monocyte-Macrophage Lineage
inferred← citeshttps://doi.org/10.1371/journal.ppat.1003338openalex-snapshot
Phosphorylation of CDK9 at Ser175 Enhances HIV Transcription and Is a Marker of Activated P-TEFb in CD4+ T Lymphocytes
inferred← citeshttps://doi.org/10.1371/journal.ppat.1006460openalex-snapshot
Digoxin reveals a functional connection between HIV-1 integration preference and T-cell activation
inferred← citeshttps://doi.org/10.1371/journal.ppat.1009364openalex-snapshot
ZBTB2 represses HIV-1 transcription and is regulated by HIV-1 Vpr and cellular DNA damage responses
inferred← citeshttps://doi.org/10.1385/1-59259-054-3:277openalex-snapshot
The Human Immunodeficiency Virus LTR-Promoter Region as a Reporter of Stress-Induced Gene Expression
inferred← citeshttps://doi.org/10.1517/14712598.2011.546339openalex-snapshot
Tits and bits of HIV Tat protein
inferred← citeshttps://doi.org/10.1677/jme-06-0065openalex-snapshot
Activation of the human immunodeficiency virus type I long terminal repeat by 1α,25-dihydroxyvitamin D3
inferred← citeshttps://doi.org/10.17615/q6r8-kt69openalex-snapshot
Regulation of human immunodeficiency virus type 1 latency by histone deacetylases
inferred← citeshttps://doi.org/10.2119/molmed.2011.00076openalex-snapshot
Histone Deacetylase Inhibitors for Purging HIV-1 from the Latent Reservoir
inferred← citeshttps://doi.org/10.2174/157016213804999249openalex-snapshot
Histone Deacetylase Inhibitor MC1293 Induces Latent HIV-1 Reactivation by Histone Modification In Vitro Latency Cell Lines
inferred← citeshttps://doi.org/10.2217/epi.11.99openalex-snapshot
Epigenomic Deregulation in the Immune System
inferred← citeshttps://doi.org/10.2217/fvl.12.26openalex-snapshot
Molecular Mechanisms of HIV-1 Infection in Neonatal Target Cells
inferred← citeshttps://doi.org/10.3389/fcimb.2025.1593445openalex-snapshot
The emerging roles of ubiquitin-like modifications in regulating HIV replication and host defense
inferred← citeshttps://doi.org/10.3389/fmicb.2018.02131openalex-snapshot
Post-translational Modification-Based Regulation of HIV Replication
inferred← citeshttps://doi.org/10.3389/fmicb.2019.00224openalex-snapshot
HUSH, a Link Between Intrinsic Immunity and HIV Latency
inferred← citeshttps://doi.org/10.3389/fphar.2021.720821openalex-snapshot
Effect of Xanthium Strumarium on HIV-1 5′-LTR Transcriptional Activity and Viral Reactivation in Latently Infected Cells
inferred← citeshttps://doi.org/10.3390/biology1020339openalex-snapshot
Transcriptional Gene Silencing (TGS) via the RNAi Machinery in HIV-1 Infections
inferred← citeshttps://doi.org/10.3390/biom13060881openalex-snapshot
HIV-1 Transcriptional Activator Tat Inhibits IL2 Expression by Preventing the Presence of Pol II on the IL2 Promoter
inferred← citeshttps://doi.org/10.3390/nu11010133openalex-snapshot
Daily Nutritional Supplementation with Vitamin D3 and Phenylbutyrate to Treatment-Naïve HIV Patients Tested in a Randomized Placebo-Controlled Trial
inferred← citeshttps://doi.org/10.3390/v11121162openalex-snapshot
Nuclear Transit and HIV LTR Binding of NF-κB Subunits Held by IκB Proteins: Implications for HIV-1 Activation
inferred← citeshttps://doi.org/10.3390/v12050555openalex-snapshot
HIV-1 Proviral Transcription and Latency in the New Era
inferred← citeshttps://doi.org/10.3390/v9040064openalex-snapshot
Targeting TNF and TNF Receptor Pathway in HIV-1 Infection: from Immune Activation to Viral Reservoirs
inferred← citeshttps://doi.org/10.3410/f.1015087.195800openalex-snapshot
Faculty Opinions recommendation of Potentiation of tumor necrosis factor-induced NF-kappa B activation by deacetylase inhibitors is associated with a delayed cytoplasmic reappearance of I kappa B alpha.
inferred← citeshttps://doi.org/10.3892/ijmm_00000461openalex-snapshot
Histone deacetylase inhibitor Scriptaid reactivates latent HIV-1 promoter by inducing histone modification in in vitro latency cell lines
inferred← citeshttps://doi.org/10.6026/97320630008359openalex-snapshot
Relevance of systems biological approach in the differential diagnosis of invasive lobular carcinoma & invasive ductal carcinoma
inferred← citeshttps://doi.org/10.70675/a8f60021z4ac1z485az94c1za6a503874720openalex-snapshot
Antagonism of HUSH restriction by lentiviral Vpx and Vpr proteins
inferred← citeshttps://doi.org/10.7150/ijbs.10366openalex-snapshot
Interaction between HIV-1 Tat and DNA-PKcs modulates HIV transcription and class switch recombination
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