On the public citation graph

Abl1 deletion in gut stem cells suppresses p53 induction and promotes colitis-associated tumor formation

0 signed · 12 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/s00018-014-1636-xopenalex-snapshot

Colitis-associated neoplasia: molecular basis and clinical translation

inferredcites →https://doi.org/10.1038/nature06196openalex-snapshot

Identification of stem cells in small intestine and colon by marker gene Lgr5

inferredcites →https://doi.org/10.1038/nm.2033openalex-snapshot

Inhibition of the c-Abl–TAp63 pathway protects mouse oocytes from chemotherapy-induced death

inferredcites →https://doi.org/10.1038/nrgastro.2017.1openalex-snapshot

Clonal evolution of colorectal cancer in IBD

inferredcites →https://doi.org/10.1053/j.gastro.2010.01.005openalex-snapshot

Distinct Effects of p38α Deletion in Myeloid Lineage and Gut Epithelia in Mouse Models of Inflammatory Bowel Disease

inferredcites →https://doi.org/10.1126/science.1260419openalex-snapshot

Tissue-based map of the human proteome

inferredcites →https://doi.org/10.1126/scitranslmed.3010567openalex-snapshot

An EphB-Abl signaling pathway is associated with intestinal tumor initiation and growth

inferredcites →https://doi.org/10.1128/mcb.01454-13openalex-snapshot

The Capable ABL: What Is Its Biological Function?

inferredcites →https://doi.org/10.1158/2159-8290.cd-14-0595openalex-snapshot

Promotion of Colorectal Cancer Invasion and Metastasis through Activation of NOTCH–DAB1–ABL–RHOGEF Protein TRIO

inferredcites →https://doi.org/10.1242/jcs.175521openalex-snapshot

Multifunctional Abl kinases in health and disease

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4781335openalex-extracted

The Abl1 Tyrosine Kinase and p53 are Key Players in Doxorubicin-Induced Cardiomyopathy and Their Cell Death Signaling Differs in Atrial and Ventricular Cardiomyocytes

inferred← citeshttps://doi.org/10.1186/s12967-024-05623-8openalex-snapshot

The Abl1 tyrosine kinase is a key player in doxorubicin-induced cardiomyopathy and its p53/p73 cell death mediated signaling differs in atrial and ventricular cardiomyocytes

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