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Bacterial artificial chromosomes establish replication timing and sub-nuclear compartment de novo as extra-chromosomal vectors

https://doi.org/10.1093/nar/gkx1265
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1016/j.ceb.2016.02.014openalex-snapshot
How to rule the nucleus: divide et impera
inferredcites →https://doi.org/10.1016/j.ceb.2016.03.022openalex-snapshot
Replication timing and transcriptional control: beyond cause and effect — part III
inferredcites →https://doi.org/10.1016/j.coviro.2012.09.005openalex-snapshot
EBNA1 and host factors in Epstein–Barr virus latent DNA replication
inferredcites →https://doi.org/10.1016/j.exphem.2017.04.004openalex-snapshot
Stability of patient-specific features of altered DNA replication timing in xenografts of primary human acute lymphoblastic leukemia
inferredcites →https://doi.org/10.1016/j.gde.2013.11.022openalex-snapshot
Complex correlations: replication timing and mutational landscapes during cancer and genome evolution
inferredcites →https://doi.org/10.1016/j.virol.2010.01.026openalex-snapshot
Episomal replication timing of γ-herpesviruses in latently infected cells
inferredcites →https://doi.org/10.1016/j.ymeth.2012.04.009openalex-snapshot
Determining long-range chromatin interactions for selected genomic sites using 4C-seq technology: From fixation to computation
inferredcites →https://doi.org/10.1016/s0140-6736(64)92345-1openalex-snapshot
CYTOLOGY OF BURKITT'S TUMOUR (AFRICAN LYMPHOMA)
inferredcites →https://doi.org/10.1038/313812a0openalex-snapshot
Stable replication of plasmids derived from Epstein–Barr virus in various mammalian cells
inferredcites →https://doi.org/10.1038/82444openalex-snapshot
Stable correction of a genetic deficiency in human cells by an episome carrying a 115 kb genomic transgene
inferredcites →https://doi.org/10.1038/nature06947openalex-snapshot
Domain organization of human chromosomes revealed by mapping of nuclear lamina interactions
inferredcites →https://doi.org/10.1038/nature11049openalex-snapshot
Spatial partitioning of the regulatory landscape of the X-inactivation centre
inferredcites →https://doi.org/10.1038/nature11082openalex-snapshot
Topological domains in mammalian genomes identified by analysis of chromatin interactions
inferredcites →https://doi.org/10.1038/nature12064openalex-snapshot
The haplotype-resolved genome and epigenome of the aneuploid HeLa cancer cell line
inferredcites →https://doi.org/10.1038/nature13986openalex-snapshot
Topologically associating domains are stable units of replication-timing regulation
inferredcites →https://doi.org/10.1038/nature14173openalex-snapshot
Differential DNA mismatch repair underlies mutation rate variation across the human genome
inferredcites →https://doi.org/10.1038/ng0994-33openalex-snapshot
Human artificial episomal chromosomes for cloning large DNA fragments in human cells
inferredcites →https://doi.org/10.1038/sj.gt.3301808openalex-snapshot
Establishment of an oriP/EBNA1-based episomal vector transcribing human genomic β-globin in cultured murine fibroblasts
inferredcites →https://doi.org/10.1073/pnas.71.3.631openalex-snapshot
Replication of the Resident Repressed Epstein-Barr Virus Genome During the Early S Phase (S-1 Period) of Nonproducer Raji Cells
inferredcites →https://doi.org/10.1073/pnas.72.4.1477openalex-snapshot
Epstein-Barr virus genomes with properties of circular DNA molecules in carrier cells.
inferredcites →https://doi.org/10.1073/pnas.72.6.2413openalex-snapshot
Viral and cellular DNA synthesis in nuclei from human lymphocytes transformed by Epstein-Barr virus.
inferredcites →https://doi.org/10.1073/pnas.81.12.3806openalex-snapshot
A cis-acting element from the Epstein-Barr viral genome that permits stable replication of recombinant plasmids in latently infected cells.
inferredcites →https://doi.org/10.1073/pnas.88.23.10875openalex-snapshot
Epstein-Barr nuclear antigen 1 mediates a DNA loop within the latent replication origin of Epstein-Barr virus.
inferredcites →https://doi.org/10.1073/pnas.95.14.8245openalex-snapshot
Propagation and recovery of intact, infectious Epstein–Barr virus from prokaryotic to human cells
inferredcites →https://doi.org/10.1083/jcb.200912167openalex-snapshot
Dynamic plasticity of large-scale chromatin structure revealed by self-assembly of engineered chromosome regions
inferredcites →https://doi.org/10.1083/jcb.201109105openalex-snapshot
Open chromatin structures regulate the efficiencies of pre-RC formation and replication initiation in Epstein-Barr virus
inferredcites →https://doi.org/10.1083/jcb.201305027openalex-snapshot
β-Globin cis-elements determine differential nuclear targeting through epigenetic modifications
inferredcites →https://doi.org/10.1083/jcb.201405110openalex-snapshot
Directed targeting of chromatin to the nuclear lamina is mediated by chromatin state and A-type lamins
inferredcites →https://doi.org/10.1089/hum.1998.9.13-1863openalex-snapshot
A System for Shuttling 200-kb BAC/PAC Clones into Human Cells: Stable Extrachromosomal Persistence and Long-Term Ectopic Gene Activation
inferredcites →https://doi.org/10.1093/hmg/dds232openalex-snapshot
Replication-timing boundaries facilitate cell-type and species-specific regulation of a rearranged human chromosome in mouse
inferredcites →https://doi.org/10.1093/nar/19.18.5053openalex-snapshot
Replication control of autonomously replicating human sequence
inferredcites →https://doi.org/10.1093/nar/gku835openalex-snapshot
Replication of alpha-satellite DNA arrays in endogenous human centromeric regions and in human artificial chromosome
inferredcites →https://doi.org/10.1101/cshperspect.a010132openalex-snapshot
DNA Replication Timing
inferredcites →https://doi.org/10.1101/cshperspect.a013029openalex-snapshot
Replication of Epstein-Barr Viral DNA
inferredcites →https://doi.org/10.1101/gr.099655.109openalex-snapshot
Evolutionarily conserved replication timing profiles predict long-range chromatin interactions and distinguish closely related cell types
inferredcites →https://doi.org/10.1101/gr.099796.109openalex-snapshot
Genome-wide dynamics of replication timing revealed by in vitro models of mouse embryogenesis
inferredcites →https://doi.org/10.1101/gr.138511.112openalex-snapshot
Abnormal developmental control of replication-timing domains in pediatric acute lymphoblastic leukemia
inferredcites →https://doi.org/10.1101/gr.141028.112openalex-snapshot
Constitutive nuclear lamina–genome interactions are highly conserved and associated with A/T-rich sequence
inferredcites →https://doi.org/10.1101/gr.183699.114openalex-snapshot
Topologically associating domains and their long-range contacts are established during early G1 coincident with the establishment of the replication-timing program
inferredcites →https://doi.org/10.1101/gr.213751.116openalex-snapshot
Small chromosomal regions position themselves autonomously according to their chromatin class
inferredcites →https://doi.org/10.1101/sqb.2015.80.027284openalex-snapshot
Large-Scale Chromatin Structure–Function Relationships during the Cell Cycle and Development: Insights from Replication Timing
inferredcites →https://doi.org/10.1126/science.1064603openalex-snapshot
Visualization of DNA Replication on Individual Epstein-Barr Virus Episomes
inferredcites →https://doi.org/10.1126/science.281.5379.1005openalex-snapshot
Genetic Dissection of a Mammalian Replicator in the Human β-Globin Locus
inferredcites →https://doi.org/10.1126/science.aaf8084openalex-snapshot
Spatial organization of chromatin domains and compartments in single chromosomes
inferredcites →https://doi.org/10.1128/jvi.01909-09openalex-snapshot
The Latent Origin of Replication of Epstein-Barr Virus Directs Viral Genomes to Active Regions of the Nucleus
inferredcites →https://doi.org/10.1128/jvi.02115-08openalex-snapshot
Epstein-Barr Virus Episome Stability Is Coupled to a Delay in Replication Timing
inferredcites →https://doi.org/10.1128/jvi.02318-15openalex-snapshot
Identification of MEF2B , EBF1 , and IL6R as Direct Gene Targets of Epstein-Barr Virus (EBV) Nuclear Antigen 1 Critical for EBV-Infected B-Lymphocyte Survival
inferredcites →https://doi.org/10.1128/jvi.40.1.11-19.1981openalex-snapshot
Sequence complexity of circular Epstein-Bar virus DNA in transformed cells
inferredcites →https://doi.org/10.1128/jvi.50.1.248-254.1984openalex-snapshot
Detection of circular and linear herpesvirus DNA molecules in mammalian cells by gel electrophoresis
inferredcites →https://doi.org/10.1128/jvi.74.18.8563-8574.2000openalex-snapshot
Initiation of DNA Replication within oriP Is Dispensable for Stable Replication of the Latent Epstein-Barr Virus Chromosome after Infection of Established Cell Lines
inferredcites →https://doi.org/10.1128/mcb.11.3.1464openalex-snapshot
Replication Initiates at Multiple Locations on an Autonomously Replicating Plasmid in Human Cells
inferredcites →https://doi.org/10.1128/mcb.11.4.2263openalex-snapshot
Autonomous DNA Replication in Human Cells Is Affected by the Size and the Source of the DNA
inferredcites →https://doi.org/10.1128/mcb.11.9.4779openalex-snapshot
Replication Timing Control Can Be Maintained in Extrachromosomally Amplified Genes
inferredcites →https://doi.org/10.1128/mcb.15.5.2893openalex-snapshot
Initiation of Latent DNA Replication in the Epstein-Barr Virus Genome Can Occur at Sites other than the Genetically Defined Origin
inferredcites →https://doi.org/10.1128/mcb.5.8.1822openalex-snapshot
A putative origin of replication of plasmids derived from Epstein-Barr virus is composed of two cis-acting components.
inferredcites →https://doi.org/10.1128/mcb.5.8.1822-1832.1985openalex-snapshot
A Putative Origin of Replication of Plasmids Derived from Epstein-Barr Virus Is Composed of Two cis -Acting Components
inferredcites →https://doi.org/10.1242/jcs.03434openalex-snapshot
Symmetrical localization of extrachromosomally replicating viral genomes on sister chromatids
inferredcites →https://doi.org/10.1371/journal.pbio.0020152openalex-snapshot
Plasticity of DNA Replication Initiation in Epstein-Barr Virus Episomes
inferredcites →https://doi.org/10.1371/journal.pbio.1001277openalex-snapshot
USF Binding Sequences from the HS4 Insulator Element Impose Early Replication Timing on a Vertebrate Replicator
inferredcites →https://doi.org/10.1371/journal.pone.0018609openalex-snapshot
The Dyad Symmetry Element of Epstein-Barr Virus Is a Dominant but Dispensable Replication Origin

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.3188454openalex-extracted
Identification of cis Elements for Spatio-temporal Control of DNA Replication
inferred← citeshttps://doi.org/10.1002/advs.77370openalex-snapshot
In Vivo Direct Reprogramming: Current Progress and Future Prospects from Mechanisms to Therapeutic Application
inferred← citeshttps://doi.org/10.1016/b978-0-12-817495-1.00007-4openalex-snapshot
Exploring bacterial diversity
inferred← citeshttps://doi.org/10.1016/j.cell.2018.11.036openalex-snapshot
Identifying cis Elements for Spatiotemporal Control of Mammalian DNA Replication
inferred← citeshttps://doi.org/10.1101/778423openalex-snapshot
Active transcription regulates ORC/MCM distribution whereas replication timing correlates with ORC density in human cells
inferred← citeshttps://doi.org/10.1371/journal.pcbi.1011138openalex-snapshot
Neural network and kinetic modelling of human genome replication reveal replication origin locations and strengths
inferred← citeshttps://doi.org/10.3410/f.734689559.793556009openalex-snapshot
Faculty Opinions recommendation of Identifying cis elements for spatiotemporal control of mammalian DNA replication.
inferred← citeshttps://doi.org/10.7554/elife.62161openalex-snapshot
Human ORC/MCM density is low in active genes and correlates with replication time but does not delimit initiation zones
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