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Allergic Skin Inflammation Induced by Chemical Sensitizers Is Controlled by the Transcription Factor Nrf2
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/art.33328openalex-snapshot
Antroquinonol differentially modulates T cell activity and reduces interleukin‐18 production, but enhances Nrf2 activation, in murine accelerated severe lupus nephritis
inferredcites →https://doi.org/10.1006/bbrc.1997.6943openalex-snapshot
An Nrf2/Small Maf Heterodimer Mediates the Induction of Phase II Detoxifying Enzyme Genes through Antioxidant Response Elements
inferredcites →https://doi.org/10.1007/s00204-012-0976-2openalex-snapshot
Induction of skin sensitization is augmented in Nrf2-deficient mice
inferredcites →https://doi.org/10.1016/j.coi.2012.08.003openalex-snapshot
Allergic contact dermatitis: xenoinflammation of the skin
inferredcites →https://doi.org/10.1016/j.immuni.2005.04.004openalex-snapshot
Dynamics and Function of Langerhans Cells In Vivo
inferredcites →https://doi.org/10.1016/j.jaci.2008.01.016openalex-snapshot
Nrf2 activation by sulforaphane restores the age-related decrease of TH1 immunity: Role of dendritic cells
inferredcites →https://doi.org/10.1016/j.taap.2010.03.009openalex-snapshot
Performance of a novel keratinocyte-based reporter cell line to screen skin sensitizers in vitro
inferredcites →https://doi.org/10.1016/j.tox.2010.04.016openalex-snapshot
Sensitization effect of thimerosal is mediated in vitro via reactive oxygen species and calcium signaling
inferredcites →https://doi.org/10.1016/j.toxlet.2004.02.002openalex-snapshot
Gene expression changes in peripheral blood-derived dendritic cells following exposure to a contact allergen
inferredcites →https://doi.org/10.1016/j.yrtph.2009.08.016openalex-snapshot
Potency values from the local lymph node assay: Application to classification, labelling and risk assessment
inferredcites →https://doi.org/10.1038/nm.2896openalex-snapshot
Early infection with respiratory syncytial virus impairs regulatory T cell function and increases susceptibility to allergic asthma
inferredcites →https://doi.org/10.1038/nri3150openalex-snapshot
Early immune events in the induction of allergic contact dermatitis
inferredcites →https://doi.org/10.1038/sj.jid.5700174openalex-snapshot
CD8+ T Cells Are Effector Cells of Contact Dermatitis to Common Skin Allergens in Mice
inferredcites →https://doi.org/10.1038/sj.jid.5700726openalex-snapshot
Skin Contact Irritation Conditions the Development and Severity of Allergic Contact Dermatitis
inferredcites →https://doi.org/10.1046/j.1523-1747.2003.12510.xopenalex-snapshot
Thiol Antioxidants Block the Activation of Antigen-Presenting Cells by Contact Sensitizers
inferredcites →https://doi.org/10.1056/nejmoa1105143openalex-snapshot
Regulatory T-Cell Responses to Low-Dose Interleukin-2 in HCV-Induced Vasculitis
inferredcites →https://doi.org/10.1074/jbc.m111.322420openalex-snapshot
Loss of Transcription Factor Nuclear Factor-Erythroid 2 (NF-E2) p45-related Factor-2 (Nrf2) Leads to Dysregulation of Immune Functions, Redox Homeostasis, and Intracellular Signaling in Dendritic Cells
inferredcites →https://doi.org/10.1084/jem.20050538openalex-snapshot
Disruption of Nrf2 enhances susceptibility to severe airway inflammation and asthma in mice
inferredcites →https://doi.org/10.1084/jem.20070509openalex-snapshot
Toll-like receptor and IL-12 signaling control susceptibility to contact hypersensitivity
inferredcites →https://doi.org/10.1089/ars.2005.7.1664openalex-snapshot
Molecular Mechanism of Nrf2 Activation by Oxidative Stress
inferredcites →https://doi.org/10.1093/toxsci/kfn243openalex-snapshot
HMOX1 and NQO1 Genes are Upregulated in Response to Contact Sensitizers in Dendritic Cells and THP-1 Cell Line: Role of the Keap1/Nrf2 Pathway
inferredcites →https://doi.org/10.1097/01.all.0000182548.60595.80openalex-snapshot
Clinical update on contact allergy
inferredcites →https://doi.org/10.1111/j.0022-202x.2005.23624.xopenalex-snapshot
Redox Imbalance Induced by Contact Sensitizers Triggers the Maturation of Dendritic Cells
inferredcites →https://doi.org/10.1111/j.0105-1873.2005.00683.xopenalex-snapshot
Hapten–protein binding: from theory to practical application in the in vitro prediction of skin sensitization
inferredcites →https://doi.org/10.1111/j.0105-1873.2006.00795.xopenalex-snapshot
Metabolism versus chemical transformation or pro‐ versus prehaptens?
inferredcites →https://doi.org/10.1111/j.1398-9995.2011.02652.xopenalex-snapshot
Mechanisms of chemical-induced innate immunity in allergic contact dermatitis
inferredcites →https://doi.org/10.1111/j.1600-0536.2007.01220.xopenalex-snapshot
The epidemiology of contact allergy in the general population – prevalence and main findings
inferredcites →https://doi.org/10.1111/j.1600-0625.2008.00717.xopenalex-snapshot
Anti‐inflammatory and immune‐regulatory mechanisms prevent contact hypersensitivity to Arnica montana L.
inferredcites →https://doi.org/10.1128/mcb.01704-07openalex-snapshot
Physiological Significance of Reactive Cysteine Residues of Keap1 in Determining Nrf2 Activity
inferredcites →https://doi.org/10.1146/annurev.immunol.25.022106.141623openalex-snapshot
Regulatory T Cells: Mechanisms of Differentiation and Function
inferredcites →https://doi.org/10.1146/annurev.pharmtox.46.120604.141046openalex-snapshot
Cell Survival Responses to Environmental Stresses Via the Keap1-Nrf2-ARE Pathway
inferredcites →https://doi.org/10.1165/rcmb.2008-0438ocopenalex-snapshot
Nuclear Erythroid 2 p45-Related Factor 2 Inhibits the Maturation of Murine Dendritic Cells by Ragweed Extract
inferredcites →https://doi.org/10.1172/jci25790openalex-snapshot
Nrf2 is a critical regulator of the innate immune response and survival during experimental sepsis
inferredcites →https://doi.org/10.1371/journal.pone.0041340openalex-snapshot
Contact Sensitizers Induce Skin Inflammation via ROS Production and Hyaluronic Acid Degradation
inferredcites →https://doi.org/10.3109/1547691x.2012.711782openalex-snapshot
Danger, intracellular signaling, and the orchestration of dendritic cell function in skin sensitization
inferredcites →https://doi.org/10.5021/ad.2010.22.3.269openalex-snapshot
The Roles of Reactive Oxygen Species Produced by Contact Allergens and Irritants in Monocyte-derived Dendritic Cells
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4822668openalex-extracted
An Integrated In-Silico-In-Chemico-In-Vitro (Isiciv) Approach to Identify Biomarkers to Predict the Skin Sensitization Potential of Phytochemicals
inferred← citeshttps://doi.org/10.1002/9783527676965.ch5openalex-snapshot
Chemical Allergen‐Induced Skin Cell Activation
inferred← citeshttps://doi.org/10.1002/biof.1907openalex-snapshot
NRF2 in dermo‐cosmetic: From scientific knowledge to skin care products
inferred← citeshttps://doi.org/10.1002/biof.70013openalex-snapshot
The role of NRF2 transcription factor in inflammatory skin diseases
inferred← citeshttps://doi.org/10.1002/eji.202249984openalex-snapshot
Endoplasmic reticulum stress and the inflammatory response in allergic contact dermatitis
inferred← citeshttps://doi.org/10.1002/rcm.9891openalex-snapshot
Metabolipidomic changes induced by dermal nickel penetration determined in an ex vivo porcine ear skin model
inferred← citeshttps://doi.org/10.1007/164_2021_482openalex-snapshot
Innate Immune Mechanisms in Contact Dermatitis
inferred← citeshttps://doi.org/10.1007/978-3-030-36335-2_59openalex-snapshot
Mechanisms of Irritant and Allergic Contact Dermatitis
inferred← citeshttps://doi.org/10.1007/978-3-319-50353-0_17openalex-snapshot
The KeratinoSens™ Assay for Skin Sensitization Screening
inferred← citeshttps://doi.org/10.1007/978-3-319-72451-5_59-1openalex-snapshot
Mechanisms of Irritant and Allergic Contact Dermatitis
inferred← citeshttps://doi.org/10.1007/978-4-431-56068-5_20openalex-snapshot
Chronicity of Immune Abnormality in Atopic Dermatitis: Interacting Surface Between Environment and Immune System
inferred← citeshttps://doi.org/10.1007/s00018-014-1676-2openalex-snapshot
Adaptation in the innate immune system and heterologous innate immunity
inferred← citeshttps://doi.org/10.1007/s00105-019-04527-9openalex-snapshot
Erweitertes Verständnis von Pathogenese und Therapie der Kontaktallergie
inferred← citeshttps://doi.org/10.1007/s00204-015-1555-0openalex-snapshot
Reporter cell lines for skin sensitization testing
inferred← citeshttps://doi.org/10.1007/s00204-015-1634-2openalex-snapshot
Bayesian integrated testing strategy (ITS) for skin sensitization potency assessment: a decision support system for quantitative weight of evidence and adaptive testing strategy
inferred← citeshttps://doi.org/10.1007/s00403-015-1554-2openalex-snapshot
The role of transcription factor Nrf2 in skin cells metabolism
inferred← citeshttps://doi.org/10.1007/s10753-017-0693-3openalex-snapshot
JNJ7777120 Ameliorates Inflammatory and Oxidative Manifestations in a Murine Model of Contact Hypersensitivity via Modulation of TLR and Nrf2 Signaling
inferred← citeshttps://doi.org/10.1007/s10787-021-00834-7openalex-snapshot
Ketoconazole-p aminobenzoic cocrystal, an improved antimycotic drug formulation, does not induce skin sensitization on the skin of BALBc mice
inferred← citeshttps://doi.org/10.1007/s11882-017-0752-8openalex-snapshot
Pathomechanisms of Contact Sensitization
inferred← citeshttps://doi.org/10.1016/j.biopha.2017.05.126openalex-snapshot
Inflammasomes, the cardinal pathology mediators are activated by pathogens, allergens and mutagens: A critical review with focus on NLRP3
inferred← citeshttps://doi.org/10.1016/j.biopha.2022.113727openalex-snapshot
RETRACTED: Dihydroquercetin composite nanofibrous membrane prevents UVA radiation-mediated inflammation, apoptosis and oxidative stress by modulating MAPKs/Nrf2 signaling in human epidermal keratinocytes
inferred← citeshttps://doi.org/10.1016/j.biotechadv.2017.12.012openalex-snapshot
Nrf2 as regulator of innate immunity: A molecular Swiss army knife!
inferred← citeshttps://doi.org/10.1016/j.cotox.2016.10.004openalex-snapshot
Roles of Nrf2 in drug and chemical toxicity
inferred← citeshttps://doi.org/10.1016/j.cotox.2018.11.002openalex-snapshot
Immune-mediated skin diseases induced by chemicals and drugs
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2023.114841openalex-snapshot
Nrf2 regulates the activation of THP-1 cells induced by chloral hydrate
inferred← citeshttps://doi.org/10.1016/j.etp.2013.10.001openalex-snapshot
Inhibitory effects of Juglans mandshurica leaf on allergic dermatitis-like skin lesions-induced by 2,4-dinitrochlorobenzene in mice
inferred← citeshttps://doi.org/10.1016/j.fct.2024.114725openalex-snapshot
Deriving a point of departure for assessing the skin sensitization risk of wearable device constituents with in vitro methods
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2014.03.006openalex-snapshot
Nrf2 expression and activity in human T lymphocytes: stimulation by T cell receptor activation and priming by inorganic arsenic and tert-butylhydroquinone
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2015.02.003openalex-snapshot
Nrf2-dependent repression of interleukin-12 expression in human dendritic cells exposed to inorganic arsenic
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2015.04.018openalex-snapshot
Nrf2—A regulator of keratinocyte redox signaling
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2025.02.005openalex-snapshot
Skin sensitizers enhance superoxide formation by polycyclic aromatic hydrocarbons via the aldo-keto reductase pathway
inferred← citeshttps://doi.org/10.1016/j.jaci.2022.02.002openalex-snapshot
Novel insights into contact dermatitis
inferred← citeshttps://doi.org/10.1016/j.jpap.2024.100227openalex-snapshot
Pivotal roles of TRPV1 channel and Nrf2 factor in green light modulation of keratinocyte inflammatory response
inferred← citeshttps://doi.org/10.1016/j.jphotobiol.2019.111518openalex-snapshot
6-shogaol, a active constiuents of ginger prevents UVB radiation mediated inflammation and oxidative stress through modulating NrF2 signaling in human epidermal keratinocytes (HaCaT cells)
inferred← citeshttps://doi.org/10.1016/j.jphs.2019.08.004openalex-snapshot
Effects of 2′-3′-dihydroxy-4′,6′-dimethoxychalcone derived from green perilla on auricle thickness in chronic contact dermatitis model mice
inferred← citeshttps://doi.org/10.1016/j.mad.2020.111251openalex-snapshot
The role of Sestrins in the regulation of the aging process
inferred← citeshttps://doi.org/10.1016/j.pharma.2015.06.002openalex-snapshot
Perspectives and strategies of alternative methods used in the risk assessment of personal care products
inferred← citeshttps://doi.org/10.1016/j.phyplu.2024.100650openalex-snapshot
An integrated In-Silico-In-Chemico-In-Vitro (iSiCiV) Approach to identify biomarkers to predict the skin sensitisation potential of phytochemicals
inferred← citeshttps://doi.org/10.1016/j.taap.2016.08.019openalex-snapshot
6-Shogaol, an active compound of ginger, alleviates allergic dermatitis-like skin lesions via cytokine inhibition by activating the Nrf2 pathway
inferred← citeshttps://doi.org/10.1016/j.taap.2016.11.001openalex-snapshot
Proteomics analysis of dendritic cell activation by contact allergens reveals possible biomarkers regulated by Nrf2
inferred← citeshttps://doi.org/10.1016/j.taap.2017.02.014openalex-snapshot
Dendritic cells' death induced by contact sensitizers is controlled by Nrf2 and depends on glutathione levels
inferred← citeshttps://doi.org/10.1016/j.taap.2022.116163openalex-snapshot
NRF2-ARE signaling is responsive to haloacetonitrile-induced oxidative stress in human keratinocytes
inferred← citeshttps://doi.org/10.1016/j.taap.2023.116650openalex-snapshot
A comparative proteomics analysis of four contact allergens in THP-1 cells shows distinct alterations in key metabolic pathways
inferred← citeshttps://doi.org/10.1016/j.tiv.2015.02.012openalex-snapshot
Genes specifically modulated in sensitized skins allow the detection of sensitizers in a reconstructed human skin model. Development of the SENS-IS assay
inferred← citeshttps://doi.org/10.1016/j.tiv.2016.12.005openalex-snapshot
An in vitro skin sensitization assay termed EpiSensA for broad sets of chemicals including lipophilic chemicals and pre/pro-haptens
inferred← citeshttps://doi.org/10.1016/j.tiv.2020.104955openalex-snapshot
Enhanced predictive capacity using dual-parameter chip model that simulates physiological skin irritation
inferred← citeshttps://doi.org/10.1016/j.toxlet.2019.07.023openalex-snapshot
Skin sensitizing effects of sulfur mustard and other alkylating agents in accordance to OECD guidelines
inferred← citeshttps://doi.org/10.1016/j.toxlet.2022.07.312openalex-snapshot
P05-03 Photobiomodulation-induced Nrf2 partially controls the inflammatory response in keratinocytes
inferred← citeshttps://doi.org/10.1016/j.toxlet.2022.07.441openalex-snapshot
P10-10 Accumulation of Nrf2 controlled by CK2 in dendritic cells plays a role in the chemical sensitizer-induced inflammation response
inferred← citeshttps://doi.org/10.1016/s0378-4274(23)00434-4openalex-snapshot
OS03-07: Photobiomodulation Controls Keratinocytes Inflammatory Response through Nrf2 and Reduces Langerhans Cells Activation
inferred← citeshttps://doi.org/10.1021/acs.chemrestox.6b00147openalex-snapshot
Mass Spectrometric and Functional Aspects of Drug–Protein Conjugation
inferred← citeshttps://doi.org/10.1021/acs.jmedchem.5c02838openalex-snapshot
Design, Synthesis, and Biological Evaluation of Novel Vinyl Selenone Derivatives as Potent Nrf2 Activators for Atopic Dermatitis
inferred← citeshttps://doi.org/10.1038/s41598-019-41493-7openalex-snapshot
Amorphous silicon dioxide nanoparticles modulate immune responses in a model of allergic contact dermatitis
inferred← citeshttps://doi.org/10.1039/c7tx00058hopenalex-snapshot
Application of proteomics in the elucidation of chemical-mediated allergic contact dermatitis
inferred← citeshttps://doi.org/10.1055/s-0042-103772openalex-snapshot
Mechanismen der Hautentzündung bei der Kontaktdermatitis
inferred← citeshttps://doi.org/10.1070/qe2014v044n08abeh015489openalex-snapshot
Optical diagnostics of vascular reactions triggered by weak allergens using laser speckle-contrast imaging technique
inferred← citeshttps://doi.org/10.1074/jbc.m117.802157openalex-snapshot
The nuclear factor-erythroid 2-related factor/heme oxygenase-1 axis is critical for the inflammatory features of type 2 diabetes–associated osteoarthritis
inferred← citeshttps://doi.org/10.1093/toxsci/kfv148openalex-snapshot
Activation of the Endoperoxide Ascaridole Modulates Its Sensitizing Capacity
inferred← citeshttps://doi.org/10.1097/aci.0000000000000142openalex-snapshot
Immunological mechanisms in allergic contact dermatitis
inferred← citeshttps://doi.org/10.1111/all.13762openalex-snapshot
Dietary coconut oil ameliorates skin contact hypersensitivity through mead acid production in mice
inferred← citeshttps://doi.org/10.1111/all.15024openalex-snapshot
IRE1 and PERK signaling regulates inflammatory responses in a murine model of contact hypersensitivity
inferred← citeshttps://doi.org/10.1111/cod.12789openalex-snapshot
Current knowledge on biomarkers for contact sensitization and allergic contact dermatitis
inferred← citeshttps://doi.org/10.1111/cod.13153openalex-snapshot
Mice with epidermal filaggrin deficiency show increased immune reactivity to nickel
inferred← citeshttps://doi.org/10.1111/cod.14073openalex-snapshot
Regulation of the immune response to contact sensitizers by Nrf2
inferred← citeshttps://doi.org/10.1111/exd.12622openalex-snapshot
FcR γ promotes contact hypersensitivity to oxazolone without affecting the contact sensitisation process in B6 mice
inferred← citeshttps://doi.org/10.1124/dmd.121.000414openalex-snapshot
The Complicated Role of Nuclear Factor Erythroid-Derived 2-Like 2 in Allergy and Asthma
inferred← citeshttps://doi.org/10.1159/000499432openalex-snapshot
Nrf2 Activation by SK-119 Attenuates Oxidative Stress, UVB, and LPS-Induced Damage
inferred← citeshttps://doi.org/10.1186/s13075-018-1801-yopenalex-snapshot
Impairment of glyoxalase-1, an advanced glycation end-product detoxifying enzyme, induced by inflammation in age-related osteoarthritis
inferred← citeshttps://doi.org/10.12659/msmbr.897771openalex-snapshot
Anti-Inflammatory and Antioxidant Effects of Repeated Exposure to Cruciferous Allyl Nitrile in Sensitizer-Induced Ear Edema in Mice
inferred← citeshttps://doi.org/10.12932/ap-200122-1309openalex-snapshot
Oleuropein attenuates inflammation and regulates immune responses in a 2,4-dinitrochlorobenzene-induced atopic dermatitis mouse model
inferred← citeshttps://doi.org/10.1371/journal.pone.0216465openalex-snapshot
Nrf2 downregulates zymosan-induced neutrophil activation and modulates migration
inferred← citeshttps://doi.org/10.14670/hh-18-293openalex-snapshot
Shufeng Jiedu capsules protect rats against LPS-induced acute lung injury via activating NRF2-associated antioxidant pathway.
inferred← citeshttps://doi.org/10.1539/sangyoeisei.wadai15001openalex-snapshot
Can a sensitizer potency be attenuated?
inferred← citeshttps://doi.org/10.21203/rs.3.rs-10234125/v1openalex-snapshot
Dimethyl fumarate improves imiquimod-induced psoriasiform inflammation and melanogenesis
inferred← citeshttps://doi.org/10.3109/10408444.2014.925425openalex-snapshot
Anchoring molecular mechanisms to the adverse outcome pathway for skin sensitization: Analysis of existing data
inferred← citeshttps://doi.org/10.3109/10408444.2014.940445openalex-snapshot
A critical appraisal of the process of regulatory implementation of novelin vivoandin vitromethods for chemical hazard and risk assessment
inferred← citeshttps://doi.org/10.3109/1547691x.2014.975897openalex-snapshot
The involvement of the Toll-like receptor signaling and Nrf2-Keap1 pathways in thein vitroregulation of IL-8 and HMOX1 for skin sensitization
inferred← citeshttps://doi.org/10.3389/fimmu.2019.01004openalex-snapshot
Nrf2 Involvement in Chemical-Induced Skin Innate Immunity
inferred← citeshttps://doi.org/10.3389/fimmu.2024.1374425openalex-snapshot
The gut lactic acid bacteria metabolite, 10-oxo-cis-6,trans-11-octadecadienoic acid, suppresses inflammatory bowel disease in mice by modulating the NRF2 pathway and GPCR-signaling
inferred← citeshttps://doi.org/10.3389/fnut.2023.1081263openalex-snapshot
A rose flavor compound activating the NRF2 pathway in dendritic cells ameliorates contact hypersensitivity in mice
inferred← citeshttps://doi.org/10.3389/ftox.2023.1132020openalex-snapshot
Ethylhexadecyldimethylammonium bromide, a quaternary ammonium compound, controls inflammatory response through NRF2 pathway in a human immortalized keratinocyte cell line
inferred← citeshttps://doi.org/10.3390/antiox11030575openalex-snapshot
The Inflammatory Response in Human Keratinocytes Exposed to Cinnamaldehyde Is Regulated by Nrf2
inferred← citeshttps://doi.org/10.3390/antiox12030766openalex-snapshot
Photobiomodulation Controls Keratinocytes Inflammatory Response through Nrf2 and Reduces Langerhans Cells Activation
inferred← citeshttps://doi.org/10.3390/cosmetics3010008openalex-snapshot
Mechanistic Understanding of Contact Allergy
inferred← citeshttps://doi.org/10.3390/jcm14207175openalex-snapshot
Allergic Contact Dermatitis: Immunopathology and Potential Therapeutic Strategies
inferred← citeshttps://doi.org/10.4049/jimmunol.1501447openalex-snapshot
Autocrine and Paracrine Regulation of Keratinocyte Proliferation through a Novel Nrf2–IL-36γ Pathway
inferred← citeshttps://doi.org/10.4049/jimmunol.1801065openalex-snapshot
Cutting Edge: Nrf2 Regulates Neutrophil Recruitment and Accumulation in Skin during Contact Hypersensitivity
inferred← citeshttps://doi.org/10.4049/jimmunol.2100577openalex-snapshot
Aberrant HO-1/NQO1–Reactive Oxygen Species–ERK Signaling Pathway Contributes to Aggravation of TPA-Induced Irritant Contact Dermatitis in Nrf2-Deficient Mice
inferred← citeshttps://doi.org/10.70675/0958a7daze8d7z4adaz9db4z01df5bdd3bc8openalex-snapshot
Evaluation in vitro de la sensibilisation cutanée aux xénobiotiques : Pertinence d’un modèle de co-culture épiderme reconstruit humain/cellules THP-1
inferred← citeshttps://doi.org/10.70675/0c979a0ez843cz4002zabcczdf942e54dc18openalex-snapshot
Rôle des lymphocytes T CD8+ dans les hypersensibilités retardées cutanées médicamenteuses
inferred← citeshttps://doi.org/10.70675/158fdf52zf391z4345za623z1930dc13404dopenalex-snapshot
L'apport de l'étude des mécanismes intracellulaires pour l'évaluation du pouvoir sensibilisant de substances industrielles
inferred← citeshttps://doi.org/10.70675/4900e006zd72bz460czbce9z30cd5e7312bcopenalex-snapshot
Les allergies cutanées aux fragrances : mécanisme d'action et rôle du facteur de transcription Nrf2. Du modèle 2D au modèle 3D.
inferred← citeshttps://doi.org/10.70675/a019e3ccz77b1z4f0dz8000z6c13a5462c44openalex-snapshot
Rôle du facteur de transcription Nrf2 dans la régulation des fonctions du neutrophile in vitro et dans l’allergie cutanée
inferred← citeshttps://doi.org/10.70675/fed3e8c8z64c2z4e1ez81a9z054e2d240b5fopenalex-snapshot
Rôle de la CK2 dans l’activation de la réponse immunitaire induite par les molécules allergisantes et son lien avec Nrf2
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