On the public citation graph
Type 1 diabetes genome-wide association analysis with imputation identifies five new risk regions
0 signed · 68 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/gepi.20297openalex-snapshot
Estimation of significance thresholds for genomewide association scans
inferredcites →https://doi.org/10.1002/gepi.21853openalex-snapshot
A Method for Gene‐Based Pathway Analysis Using Genomewide Association Study Summary Statistics Reveals Nine New Type 1 Diabetes Associations
inferredcites →https://doi.org/10.1007/s00251-012-0650-0openalex-snapshot
Association of AFF1 rs340630 and AFF3 rs10865035 polymorphisms with systemic lupus erythematosus in a Chinese population
inferredcites →https://doi.org/10.1016/j.cell.2016.09.037openalex-snapshot
Lineage-Specific Genome Architecture Links Enhancers and Non-coding Disease Variants to Target Gene Promoters
inferredcites →https://doi.org/10.1016/j.immuni.2009.01.001openalex-snapshot
The Many Roles of FAS Receptor Signaling in the Immune System
inferredcites →https://doi.org/10.1016/s0301-0082(02)00016-3openalex-snapshot
Neurotrophin signaling through the p75 neurotrophin receptor
inferredcites →https://doi.org/10.1038/nature04226openalex-snapshot
A haplotype map of the human genome
inferredcites →https://doi.org/10.1038/nature05911openalex-snapshot
Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls
inferredcites →https://doi.org/10.1038/nature06258openalex-snapshot
A second generation human haplotype map of over 3.1 million SNPs
inferredcites →https://doi.org/10.1038/nature11582openalex-snapshot
Host–microbe interactions have shaped the genetic architecture of inflammatory bowel disease
inferredcites →https://doi.org/10.1038/nature12873openalex-snapshot
Genetics of rheumatoid arthritis contributes to biology and drug discovery
inferredcites →https://doi.org/10.1038/ncomms10069openalex-snapshot
Capture Hi-C reveals novel candidate genes and complex long-range interactions with related autoimmune risk loci
inferredcites →https://doi.org/10.1038/ng.102openalex-snapshot
Newly identified genetic risk variants for celiac disease related to the immune response
inferredcites →https://doi.org/10.1038/ng.2354openalex-snapshot
Fast and accurate genotype imputation in genome-wide association studies through pre-phasing
inferredcites →https://doi.org/10.1038/ng.2462openalex-snapshot
High-density genetic mapping identifies new susceptibility loci for rheumatoid arthritis
inferredcites →https://doi.org/10.1038/ng.249openalex-snapshot
Meta-analysis of genome-wide association study data identifies additional type 1 diabetes risk loci
inferredcites →https://doi.org/10.1038/ng.2770openalex-snapshot
Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis
inferredcites →https://doi.org/10.1038/ng.3245openalex-snapshot
Fine mapping of type 1 diabetes susceptibility loci and evidence for colocalization of causal variants with lymphoid gene enhancers
inferredcites →https://doi.org/10.1038/ng.3330openalex-snapshot
Statistical colocalization of genetic risk variants for related autoimmune diseases in the context of common controls
inferredcites →https://doi.org/10.1038/ng.3434openalex-snapshot
Genetic association analyses implicate aberrant regulation of innate and adaptive immunity genes in the pathogenesis of systemic lupus erythematosus
inferredcites →https://doi.org/10.1038/ng.381openalex-snapshot
Genome-wide association study and meta-analysis find that over 40 loci affect risk of type 1 diabetes
inferredcites →https://doi.org/10.1038/ng.493openalex-snapshot
The imprinted DLK1-MEG3 gene region on chromosome 14q32.2 alters susceptibility to type 1 diabetes
inferredcites →https://doi.org/10.1038/ng.543openalex-snapshot
Multiple common variants for celiac disease influencing immune gene expression
inferredcites →https://doi.org/10.1038/ng.717openalex-snapshot
Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci
inferredcites →https://doi.org/10.1038/ng.764openalex-snapshot
Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47
inferredcites →https://doi.org/10.1038/ng.998openalex-snapshot
Dense genotyping identifies and localizes multiple common and rare variant association signals in celiac disease
inferredcites →https://doi.org/10.1038/ng1333openalex-snapshot
Assessing the impact of population stratification on genetic association studies
inferredcites →https://doi.org/10.1038/ng2068openalex-snapshot
Robust associations of four new chromosome regions from genome-wide analyses of type 1 diabetes
inferredcites →https://doi.org/10.1038/ng2088openalex-snapshot
A new multipoint method for genome-wide association studies by imputation of genotypes
inferredcites →https://doi.org/10.1038/ng2102openalex-snapshot
Large-scale genetic fine mapping and genotype-phenotype associations implicate polymorphism in the IL2RA region in type 1 diabetes
inferredcites →https://doi.org/10.1056/nejme0809719openalex-snapshot
Shared Genetic Risk Factors for Type 1 Diabetes and Celiac Disease
inferredcites →https://doi.org/10.1073/pnas.0508122102openalex-snapshot
The role of TNF-α in the pathogenesis of type 1 diabetes in the nonobese diabetic mouse: Analysis of dendritic cell maturation
inferredcites →https://doi.org/10.1093/hmg/ddn288openalex-snapshot
Practical aspects of imputation-driven meta-analysis of genome-wide association studies
inferredcites →https://doi.org/10.1093/hmg/ddp177openalex-snapshot
Identification of AF4/FMR2 family, member 3 (AFF3) as a novel rheumatoid arthritis susceptibility locus and confirmation of two further pan-autoimmune susceptibility genes
inferredcites →https://doi.org/10.1093/ije/dyi183openalex-snapshot
Cohort profile: 1958 British birth cohort (National Child Development Study)
inferredcites →https://doi.org/10.1093/rheumatology/kel322openalex-snapshot
Unstable diabetes in a patient receiving anti-TNF- for rheumatoid arthritis
inferredcites →https://doi.org/10.1111/pedi.12305openalex-snapshot
The role for gut permeability in the pathogenesis of type 1 diabetes - a solid or leaky concept?
inferredcites →https://doi.org/10.1126/science.7542801openalex-snapshot
A Role for CD5 in TCR-Mediated Signal Transduction and Thymocyte Selection
inferredcites →https://doi.org/10.1136/ard.2009.118406openalex-snapshot
Investigation of rheumatoid arthritis susceptibility genes identifies association of AFF3 and CD226 variants with response to anti-tumour necrosis factor treatment
inferredcites →https://doi.org/10.1146/annurev.genom.9.081307.164242openalex-snapshot
Genotype Imputation
inferredcites →https://doi.org/10.1186/2251-6581-13-46openalex-snapshot
TNF-α and IFN-γ gene variation and genetic susceptibility to type 1 diabetes and its microangiopathic complications
inferredcites →https://doi.org/10.1186/ar3204openalex-snapshot
Promise and pitfalls of the Immunochip
inferredcites →https://doi.org/10.1371/journal.pgen.1000477openalex-snapshot
Designing Genome-Wide Association Studies: Sample Size, Power, Imputation, and the Choice of Genotyping Chip
inferredcites →https://doi.org/10.1371/journal.pgen.1002293openalex-snapshot
A Genome-Wide Meta-Analysis of Six Type 1 Diabetes Cohorts Identifies Multiple Associated Loci
inferredcites →https://doi.org/10.1371/journal.pone.0038539openalex-snapshot
CD6 and Syntaxin Binding Protein 6 Variants and Response to Tumor Necrosis Factor Alpha Inhibitors in Danish Patients with Rheumatoid Arthritis
inferredcites →https://doi.org/10.1534/g3.111.001198openalex-snapshot
Genotype Imputation with Thousands of Genomes
inferredcites →https://doi.org/10.2337/db11-0638openalex-snapshot
FUT2 Nonsecretor Status Links Type 1 Diabetes Susceptibility and Resistance to Infection
inferredcites →https://doi.org/10.2337/db14-1847openalex-snapshot
Alterations in Intestinal Microbiota Correlate With Susceptibility to Type 1 Diabetes
inferredcites →https://doi.org/10.7326/0003-4819-130-12-199906150-00019openalex-snapshot
Toward Evidence-Based Medical Statistics. 2: The Bayes Factor
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/gepi.22521openalex-snapshot
Evaluation of polygenic risk scores to differentiate between type 1 and type 2 diabetes
inferred← citeshttps://doi.org/10.1002/gepi.22608openalex-snapshot
Bayesian Effect Size Ranking to Prioritise Genetic Risk Variants in Common Diseases for Follow‐Up Studies
inferred← citeshttps://doi.org/10.1007/s00125-026-06721-6openalex-snapshot
Multi-tissue multi-omics integration reveals tissue-specific pathways, gene networks and drug candidates for type 1 diabetes
inferred← citeshttps://doi.org/10.1016/j.gde.2018.01.006openalex-snapshot
Genetics of type 1 diabetes
inferred← citeshttps://doi.org/10.1016/j.jaut.2018.06.005openalex-snapshot
Identification of loci where DNA methylation potentially mediates genetic risk of type 1 diabetes
inferred← citeshttps://doi.org/10.1016/j.lfs.2023.121701openalex-snapshot
The role of endoplasmic reticulum aminopeptidases in type 1 diabetes mellitus
inferred← citeshttps://doi.org/10.1016/j.xhgg.2024.100336openalex-snapshot
Discovery of new myositis genetic associations through leveraging other immune-mediated diseases
inferred← citeshttps://doi.org/10.1038/s41431-018-0176-5openalex-snapshot
Comparison of methods for transcriptome imputation through application to two common complex diseases
inferred← citeshttps://doi.org/10.1038/s41467-020-16327-0openalex-snapshot
An integrated multi-omics approach identifies the landscape of interferon-α-mediated responses of human pancreatic beta cells
inferred← citeshttps://doi.org/10.1038/s41581-020-0278-5openalex-snapshot
Genetics of diabetes mellitus and diabetes complications
inferred← citeshttps://doi.org/10.1038/s41588-019-0524-6openalex-snapshot
The impact of proinflammatory cytokines on the β-cell regulatory landscape provides insights into the genetics of type 1 diabetes
inferred← citeshttps://doi.org/10.1038/s41590-018-0129-8openalex-snapshot
Approaches and advances in the genetic causes of autoimmune disease and their implications
inferred← citeshttps://doi.org/10.1093/bioinformatics/btab456openalex-snapshot
RápidoPGS: a rapid polygenic score calculator for summary GWAS data without a test dataset
inferred← citeshttps://doi.org/10.1093/hmg/ddaa097openalex-snapshot
Functional genomics in autoimmune diseases
inferred← citeshttps://doi.org/10.1101/2021.02.05.429962openalex-snapshot
Enhanced genetic analysis of type 1 diabetes by selecting variants on both effect size and significance, and by integration with autoimmune thyroid disease
inferred← citeshttps://doi.org/10.1136/bmjdrc-2022-003167openalex-snapshot
Causal associations between type 1 diabetes and COVID-19 infection and prognosis: a two-sample Mendelian randomization study
inferred← citeshttps://doi.org/10.1371/journal.pmed.1002362openalex-snapshot
Childhood adiposity and risk of type 1 diabetes: A Mendelian randomization study
inferred← citeshttps://doi.org/10.33996/revistavive.v6i16.228openalex-snapshot
Polimorfismo (cag) del gen atxn2 como factor de riesgo relacionado con diabetes mellitus tipo 2
inferred← citeshttps://doi.org/10.70675/8aca4706z3a62z4627za1baze554a54e5359openalex-snapshot
La dérive génétique : un outil pour identifier de nouveaux facteurs impliqués dans la pathogénie des maladies complexes tel le diabète auto-immune de la souris non obèse diabétique
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.