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Reprogramming of alveolar macrophages by intestinal segmented filamentous bacteria protects mice from lethal bacterial pneumoniae following influenza infection

https://doi.org/10.1101/2025.01.23.634531

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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1016/j.cell.2020.08.020openalex-snapshot

Patrolling Alveolar Macrophages Conceal Bacteria from the Immune System to Maintain Homeostasis

inferredcites →https://doi.org/10.1016/j.chom.2024.01.002openalex-snapshot

Intestinal microbiota programming of alveolar macrophages influences severity of respiratory viral infection

inferredcites →https://doi.org/10.1016/j.eclinm.2021.100955openalex-snapshot

Outcomes of respiratory viral-bacterial co-infection in adult hospitalized patients

inferredcites →https://doi.org/10.1038/s41590-018-0292-yopenalex-snapshot

Metabolic rewiring of macrophages by CpG potentiates clearance of cancer cells and overcomes tumor-expressed CD47−mediated ‘don’t-eat-me’ signal

inferredcites →https://doi.org/10.1038/s41590-019-0352-yopenalex-snapshot

The lung environment controls alveolar macrophage metabolism and responsiveness in type 2 inflammation

inferredcites →https://doi.org/10.1038/s41590-020-0673-xopenalex-snapshot

Alveolar macrophages are epigenetically altered after inflammation, leading to long-term lung immunoparalysis

inferredcites →https://doi.org/10.1038/s41590-023-01428-xopenalex-snapshot

Influenza-trained mucosal-resident alveolar macrophages confer long-term antitumor immunity in the lungs

inferredcites →https://doi.org/10.1056/nejmc0908216openalex-snapshot

Bacterial Pathogens and Death during the 1918 Influenza Pandemic

inferredcites →https://doi.org/10.1086/591708openalex-snapshot

Predominant Role of Bacterial Pneumonia as a Cause of Death in Pandemic Influenza: Implications for Pandemic Influenza Preparedness

inferredcites →https://doi.org/10.1093/ofid/ofaa546openalex-snapshot

Respiratory Syncytial Virus Infection: Its Propensity for Bacterial Coinfection and Related Mortality in Elderly Adults

inferredcites →https://doi.org/10.1128/jvi.01269-12openalex-snapshot

Type I Interferon Induction during Influenza Virus Infection Increases Susceptibility to Secondary Streptococcus pneumoniae Infection by Negative Regulation of γδ T Cells

inferredcites →https://doi.org/10.1128/mbio.00810-19openalex-snapshot

Influenza “Trains” the Host for Enhanced Susceptibility to Secondary Bacterial Infection

inferredcites →https://doi.org/10.1152/ajplung.00338.2014openalex-snapshot

Influenza-induced type I interferon enhances susceptibility to gram-negative and gram-positive bacterial pneumonia in mice

inferredcites →https://doi.org/10.1172/jci35412openalex-snapshot

Type I IFNs mediate development of postinfluenza bacterial pneumonia in mice

inferredcites →https://doi.org/10.1172/jci38226openalex-snapshot

Reemergence of antibiotic-resistant Staphylococcus aureus in the genomics era

inferredcites →https://doi.org/10.1186/s12879-022-07192-xopenalex-snapshot

Secondary infection in COVID-19 critically ill patients: a retrospective single-center evaluation

inferredcites →https://doi.org/10.1371/journal.pone.0150606openalex-snapshot

A Protocol for the Comprehensive Flow Cytometric Analysis of Immune Cells in Normal and Inflamed Murine Non-Lymphoid Tissues

inferredcites →https://doi.org/10.1371/journal.ppat.1011993openalex-snapshot

Influenza A virus resistance to 4’-fluorouridine coincides with viral attenuation in vitro and in vivo

inferredcites →https://doi.org/10.4049/jimmunol.1300014openalex-snapshot

Depletion of Alveolar Macrophages during Influenza Infection Facilitates Bacterial Superinfections

inferredcites →https://doi.org/10.4049/jimmunol.1301152openalex-snapshot

Immune Dysfunction and Bacterial Coinfections following Influenza

inferredcites →https://doi.org/10.4049/jimmunol.1701406openalex-snapshot

Effects of Influenza on Alveolar Macrophage Viability Are Dependent on Mouse Genetic Strain

inferredcites →https://doi.org/10.4049/jimmunol.2000094openalex-snapshot

Influenza Infection Induces Alveolar Macrophage Dysfunction and Thereby Enables Noninvasive Streptococcus pneumoniae to Cause Deadly Pneumonia

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