On the public citation graph

Gnas R201C Induces Murine Pancreatic Cystic Neoplasms through Suppression of YAP1 Signaling and Transcriptional Reprogramming

https://doi.org/10.1101/310292
0 signed · 39 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.
Claim with ORCID
Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.
See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1016/j.ccr.2005.04.023openalex-snapshot
Trp53R172H and KrasG12D cooperate to promote chromosomal instability and widely metastatic pancreatic ductal adenocarcinoma in mice
inferredcites →https://doi.org/10.1016/j.cell.2007.07.019openalex-snapshot
Elucidation of a Universal Size-Control Mechanism in Drosophila and Mammals
inferredcites →https://doi.org/10.1016/j.cell.2011.02.031openalex-snapshot
Yap1 Acts Downstream of α-Catenin to Control Epidermal Proliferation
inferredcites →https://doi.org/10.1016/j.cell.2014.06.003openalex-snapshot
Yap1 Activation Enables Bypass of Oncogenic Kras Addiction in Pancreatic Cancer
inferredcites →https://doi.org/10.1016/j.cub.2011.09.019openalex-snapshot
Cell–Cell Junctions: α-Catenin and E-Cadherin Help Fence In Yap1
inferredcites →https://doi.org/10.1016/s1535-6108(03)00309-xopenalex-snapshot
Preinvasive and invasive ductal pancreatic cancer and its early detection in the mouse
inferredcites →https://doi.org/10.1038/340692a0openalex-snapshot
GTPase inhibiting mutations activate the α chain of Gs and stimulate adenylyl cyclase in human pituitary tumours
inferredcites →https://doi.org/10.1038/ncb3160openalex-snapshot
TEAD and YAP regulate the enhancer network of human embryonic pancreatic progenitors
inferredcites →https://doi.org/10.1038/ncb3164openalex-snapshot
Inactivation of a Gαs–PKA tumour suppressor pathway in skin stem cells initiates basal-cell carcinogenesis
inferredcites →https://doi.org/10.1038/ng1180openalex-snapshot
PGC-1α-responsive genes involved in oxidative phosphorylation are coordinately downregulated in human diabetes
inferredcites →https://doi.org/10.1038/nrgastro.2011.2openalex-snapshot
Cystic precursors to invasive pancreatic cancer
inferredcites →https://doi.org/10.1038/onc.2010.202openalex-snapshot
The activating mutation R201C in GNAS promotes intestinal tumourigenesis in ApcMin/+ mice through activation of Wnt and ERK1/2 MAPK pathways
inferredcites →https://doi.org/10.1038/onc.2015.294openalex-snapshot
GNASR201H and KrasG12D cooperate to promote murine pancreatic tumorigenesis recapitulating human intraductal papillary mucinous neoplasm
inferredcites →https://doi.org/10.1038/onc.2016.288openalex-snapshot
Yes-associated protein mediates immune reprogramming in pancreatic ductal adenocarcinoma
inferredcites →https://doi.org/10.1053/j.gastro.2013.01.073openalex-snapshot
Pancreatic Cystic Neoplasms: Management and Unanswered Questions
inferredcites →https://doi.org/10.1053/j.gastro.2013.11.052openalex-snapshot
MAPK Signaling Is Required for Dedifferentiation of Acinar Cells and Development of Pancreatic Intraepithelial Neoplasia in Mice
inferredcites →https://doi.org/10.1053/j.gastro.2016.05.006openalex-snapshot
YAP1 and TAZ Control Pancreatic Cancer Initiation in Mice by Direct Up-regulation of JAK–STAT3 Signaling
inferredcites →https://doi.org/10.1056/nejm199112123252403openalex-snapshot
Activating Mutations of the Stimulatory G Protein in the McCune–Albright Syndrome
inferredcites →https://doi.org/10.1073/pnas.0506580102openalex-snapshot
Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles
inferredcites →https://doi.org/10.1073/pnas.1103345108openalex-snapshot
E-cadherin mediates contact inhibition of proliferation through Hippo signaling-pathway components
inferredcites →https://doi.org/10.1093/nar/gni051openalex-snapshot
Conditional and inducible transgene expression in mice through the combinatorial use of Cre-mediated recombination and tetracycline induction
inferredcites →https://doi.org/10.1097/01.sla.0000128306.90650.aaopenalex-snapshot
Intraductal Papillary Mucinous Neoplasms of the Pancreas
inferredcites →https://doi.org/10.1097/pas.0000000000000117openalex-snapshot
GNAS Sequencing Identifies IPMN-specific Mutations in a Subgroup of Diminutive Pancreatic Cysts Referred to as “Incipient IPMNs”
inferredcites →https://doi.org/10.1097/pas.0000000000000533openalex-snapshot
A Revised Classification System and Recommendations From the Baltimore Consensus Meeting for Neoplastic Precursor Lesions in the Pancreas
inferredcites →https://doi.org/10.1101/gad.274951.115openalex-snapshot
αE-catenin inhibits a Src–YAP1 oncogenic module that couples tyrosine kinases and the effector of Hippo signaling pathway
inferredcites →https://doi.org/10.1126/scisignal.2001823openalex-snapshot
α-Catenin Is a Tumor Suppressor That Controls Cell Accumulation by Regulating the Localization and Activity of the Transcriptional Coactivator Yap1
inferredcites →https://doi.org/10.1126/scisignal.2005049openalex-snapshot
Downstream of Mutant KRAS, the Transcription Regulator YAP Is Essential for Neoplastic Progression to Pancreatic Ductal Adenocarcinoma
inferredcites →https://doi.org/10.1126/scitranslmed.3002543openalex-snapshot
Recurrent GNAS Mutations Define an Unexpected Pathway for Pancreatic Cyst Development
inferredcites →https://doi.org/10.1152/physrev.00021.2003openalex-snapshot
Localized Effects of cAMP Mediated by Distinct Routes of Protein Kinase A
inferredcites →https://doi.org/10.1158/0008-5472.can-05-2168openalex-snapshot
Pathology of Genetically Engineered Mouse Models of Pancreatic Exocrine Cancer: Consensus Report and Recommendations
inferredcites →https://doi.org/10.1158/0008-5472.can-06-4167openalex-snapshot
Inactivation of Smad4 Accelerates KrasG12D-Mediated Pancreatic Neoplasia
inferredcites →https://doi.org/10.1158/0008-5472.can-16-2594openalex-snapshot
eIF5A-PEAK1 Signaling Regulates YAP1/TAZ Protein Expression and Pancreatic Cancer Cell Growth
inferredcites →https://doi.org/10.1158/1078-0432.ccr-11-2165openalex-snapshot
Molecular Determinants of Retinoic Acid Sensitivity in Pancreatic Cancer
inferredcites →https://doi.org/10.1210/jc.2005-2710openalex-snapshot
Activating Mutations of the Stimulatory G Protein in Juvenile Ovarian Granulosa Cell Tumors: A New Prognostic Factor?
inferredcites →https://doi.org/10.1245/s10434-013-3096-1openalex-snapshot
Clinicopathological Correlates of Activating GNAS Mutations in Intraductal Papillary Mucinous Neoplasm (IPMN) of the Pancreas
inferredcites →https://doi.org/10.1677/joe.1.06894openalex-snapshot
Phenotypic and global gene expression profile changes between low passage and high passage MIN-6 cells
inferredcites →https://doi.org/10.18632/oncotarget.357openalex-snapshot
Disruption of p16 and Activation of Kras in Pancreas Increase Ductal Adenocarcinoma Formation and Metastasis in vivo
inferredcites →https://doi.org/10.3322/caac.21387openalex-snapshot
Cancer statistics, 2017
inferredcites →https://doi.org/10.4161/cbt.12.7.16475openalex-snapshot
Cyclin-dependent kinase inhibitor Dinaciclib (SCH727965) inhibits pancreatic cancer growth and progression in murine xenograft models
Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.