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Targeting Wnt Signaling To Overcome PARP Inhibitor Resistance
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/cpt1976196802openalex-snapshot
Absorption of pyrvinium pamoate
inferredcites →https://doi.org/10.1002/ijc.25083openalex-snapshot
Epigenetic silencing of SFRP5 is related to malignant phenotype and chemoresistance of ovarian cancer through Wnt signaling pathway
inferredcites →https://doi.org/10.1002/path.4140openalex-snapshot
Secondary mutations in BRCA2 associated with clinical resistance to a PARP inhibitor
inferredcites →https://doi.org/10.1002/pros.20117openalex-snapshot
Inhibition of Wnt signaling downregulates Akt activity and induces chemosensitivity in PTEN-mutated prostate cancer cells
inferredcites →https://doi.org/10.1007/978-1-61779-998-3_27openalex-snapshot
I-SceI-Based Assays to Examine Distinct Repair Outcomes of Mammalian Chromosomal Double Strand Breaks
inferredcites →https://doi.org/10.1016/j.bbrc.2012.07.127openalex-snapshot
Upregulated miR-130a increases drug resistance by regulating RUNX3 and Wnt signaling in cisplatin-treated HCC cell
inferredcites →https://doi.org/10.1016/j.dnarep.2014.02.006openalex-snapshot
Non-homologous end joining often uses microhomology: Implications for alternative end joining
inferredcites →https://doi.org/10.1016/j.molcel.2017.09.036openalex-snapshot
Restoration of Replication Fork Stability in BRCA1- and BRCA2-Deficient Cells by Inactivation of SNF2-Family Fork Remodelers
inferredcites →https://doi.org/10.1016/j.ygyno.2015.12.020openalex-snapshot
Efficacy and safety of olaparib monotherapy in germline BRCA1 / 2 mutation carriers with advanced ovarian cancer and three or more lines of prior therapy
inferredcites →https://doi.org/10.1016/j.ygyno.2017.10.003openalex-snapshot
PARP inhibitors: Clinical utility and possibilities of overcoming resistance
inferredcites →https://doi.org/10.1038/nature03445openalex-snapshot
Targeting the DNA repair defect in BRCA mutant cells as a therapeutic strategy
inferredcites →https://doi.org/10.1038/nature06548openalex-snapshot
Resistance to therapy caused by intragenic deletion in BRCA2
inferredcites →https://doi.org/10.1038/nature18325openalex-snapshot
Replication fork stability confers chemoresistance in BRCA-deficient cells
inferredcites →https://doi.org/10.1038/nchembio.453openalex-snapshot
Small-molecule inhibition of Wnt signaling through activation of casein kinase 1α
inferredcites →https://doi.org/10.1038/ng1180openalex-snapshot
PGC-1α-responsive genes involved in oxidative phosphorylation are coordinately downregulated in human diabetes
inferredcites →https://doi.org/10.1038/nm.3799openalex-snapshot
Synthetic lethality by targeting EZH2 methyltransferase activity in ARID1A-mutated cancers
inferredcites →https://doi.org/10.1038/nmeth.3317openalex-snapshot
HISAT: a fast spliced aligner with low memory requirements
inferredcites →https://doi.org/10.1038/nprot.2009.28openalex-snapshot
Gaussia luciferase reporter assay for monitoring biological processes in culture and in vivo
inferredcites →https://doi.org/10.1038/onc.2016.10openalex-snapshot
Pygo2 activates MDR1 expression and mediates chemoresistance in breast cancer via the Wnt/β-catenin pathway
inferredcites →https://doi.org/10.1042/bj20121140openalex-snapshot
Pyrvinium pamoate does not activate protein kinase CK1, but promotes Akt/PKB down-regulation and GSK3 activation
inferredcites →https://doi.org/10.1056/nejmoa1611310openalex-snapshot
Niraparib Maintenance Therapy in Platinum-Sensitive, Recurrent Ovarian Cancer
inferredcites →https://doi.org/10.1073/pnas.0506580102openalex-snapshot
Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles
inferredcites →https://doi.org/10.1073/pnas.92.6.2298openalex-snapshot
Nuclear foci of mammalian Rad51 recombination protein in somatic cells after DNA damage and its localization in synaptonemal complexes.
inferredcites →https://doi.org/10.1093/nar/gkt635openalex-snapshot
Transcriptional regulation of human DNA repair genes following genotoxic stress: trigger mechanisms, inducible responses and genotoxic adaptation
inferredcites →https://doi.org/10.1111/febs.12737openalex-snapshot
Long non‐coding RNA UCA 1 increases chemoresistance of bladder cancer cells by regulating Wnt signaling
inferredcites →https://doi.org/10.1158/0008-5472.can-06-0140openalex-snapshot
Deficiency in the Repair of DNA Damage by Homologous Recombination and Sensitivity to Poly(ADP-Ribose) Polymerase Inhibition
inferredcites →https://doi.org/10.1158/0008-5472.can-11-1341openalex-snapshot
Wnt5a Suppresses Epithelial Ovarian Cancer by Promoting Cellular Senescence
inferredcites →https://doi.org/10.1158/0008-5472.can-16-0854openalex-snapshot
BET Inhibitors Suppress ALDH Activity by Targeting ALDH1A1 Super-Enhancer in Ovarian Cancer
inferredcites →https://doi.org/10.1200/jco.2016.70.4627openalex-snapshot
Reversion of BRCA1/2 Germline Mutations Detected in Circulating Tumor DNA From Patients With High-Grade Serous Ovarian Cancer
inferredcites →https://doi.org/10.12659/msm.901667openalex-snapshot
Targeting of Wnt/β-Catenin by Anthelmintic Drug Pyrvinium Enhances Sensitivity of Ovarian Cancer Cells to Chemotherapy
inferredcites →https://doi.org/10.1371/journal.pone.0010001openalex-snapshot
Dynamic Dependence on ATR and ATM for Double-Strand Break Repair in Human Embryonic Stem Cells and Neural Descendants
inferredcites →https://doi.org/10.17615/hvp3-wg08openalex-snapshot
Integrated genomic analyses of ovarian carcinoma
inferredcites →https://doi.org/10.18632/oncotarget.4690openalex-snapshot
Critical role of Wnt/β-catenin signaling in driving epithelial ovarian cancer platinum resistance
inferredcites →https://doi.org/10.3791/54944openalex-snapshot
Labeling of Breast Cancer Patient-derived Xenografts with Traceable Reporters for Tumor Growth and Metastasis Studies
inferredcites →https://doi.org/10.3892/ijo.2016.3337openalex-snapshot
WNT pathway inhibitor pyrvinium pamoate inhibits the self-renewal and metastasis of breast cancer stem cells
inferredcites →https://doi.org/10.4161/cbt.4.9.2141openalex-snapshot
BRCA2-deficient CAPAN-1 cells are extremely sensitive to the inhibition of poly (ADP-ribose) polymerase: An issue of potency
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/mc.23308openalex-snapshot
Inhibiting Wnt/beta‐catenin in CTNNB1‐mutated endometrial cancer
inferred← citeshttps://doi.org/10.1016/b978-0-12-819840-7.00012-1openalex-snapshot
Models of ovarian cancer to test new agents
inferred← citeshttps://doi.org/10.1016/j.annonc.2020.02.008openalex-snapshot
Reversion and non-reversion mechanisms of resistance to PARP inhibitor or platinum chemotherapy in BRCA1/2-mutant metastatic breast cancer
inferred← citeshttps://doi.org/10.1016/j.seminoncol.2025.152375openalex-snapshot
Breaking barriers: Deciphering the mechanisms of Olaparib resistance in prostate cancer
inferred← citeshttps://doi.org/10.1074/jbc.ra119.009615openalex-snapshot
Wnt family member 4 (WNT4) and WNT3A activate cell-autonomous Wnt signaling independent of porcupine O-acyltransferase or Wnt secretion
inferred← citeshttps://doi.org/10.1101/333906openalex-snapshot
WNT4 and WNT3A activate cell autonomous Wnt signaling independent of PORCN or secretion
inferred← citeshttps://doi.org/10.1158/0008-5472.can-18-3592openalex-snapshot
N6-Methylation of Adenosine of FZD10 mRNA Contributes to PARP Inhibitor Resistance
inferred← citeshttps://doi.org/10.4414/smw.2019.20129openalex-snapshot
Targeting the Wnt signalling pathway in cancer: prospects and perils
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