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Senescent cells and the dynamics of aging
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/978-1-4419-7002-2openalex-snapshot
Protein Metabolism and Homeostasis in Aging
inferredcites →https://doi.org/10.1007/978-1-4419-7002-2_11openalex-snapshot
Protein Homeostasis in Models of Aging and Age-Related Conformational Disease
inferredcites →https://doi.org/10.1016/j.cell.2012.12.010openalex-snapshot
Monitoring Tumorigenesis and Senescence In Vivo with a p16INK4a-Luciferase Model
inferredcites →https://doi.org/10.1016/j.cell.2017.02.031openalex-snapshot
Targeted Apoptosis of Senescent Cells Restores Tissue Homeostasis in Response to Chemotoxicity and Aging
inferredcites →https://doi.org/10.1016/j.cell.2017.05.015openalex-snapshot
Senescence in Health and Disease
inferredcites →https://doi.org/10.1016/j.cell.2017.09.004openalex-snapshot
Single-Cell Analysis of Human Pancreas Reveals Transcriptional Signatures of Aging and Somatic Mutation Patterns
inferredcites →https://doi.org/10.1016/j.celrep.2018.03.009openalex-snapshot
p53 in Bronchial Club Cells Facilitates Chronic Lung Inflammation by Promoting Senescence
inferredcites →https://doi.org/10.1016/j.devcel.2014.11.012openalex-snapshot
An Essential Role for Senescent Cells in Optimal Wound Healing through Secretion of PDGF-AA
inferredcites →https://doi.org/10.1016/j.exger.2006.01.003openalex-snapshot
Visualizing hidden heterogeneity in isogenic populations of C. elegans
inferredcites →https://doi.org/10.1016/j.exger.2014.10.012openalex-snapshot
Clinical strategies and animal models for developing senolytic agents
inferredcites →https://doi.org/10.1016/j.mad.2004.09.034openalex-snapshot
Human cell senescence as a DNA damage response
inferredcites →https://doi.org/10.1016/j.mad.2012.05.005openalex-snapshot
Frailty and the role of inflammation, immunosenescence and cellular ageing in the very old: Cross-sectional findings from the Newcastle 85+ Study
inferredcites →https://doi.org/10.1016/j.molmed.2010.03.003openalex-snapshot
Inflammatory networks during cellular senescence: causes and consequences
inferredcites →https://doi.org/10.1016/j.smim.2012.04.008openalex-snapshot
Innate immunosenescence: Effect of aging on cells and receptors of the innate immune system in humans
inferredcites →https://doi.org/10.1038/366461a0openalex-snapshot
A C. elegans mutant that lives twice as long as wild type
inferredcites →https://doi.org/10.1038/jid.2015.108openalex-snapshot
Cell Autonomous and Non-Autonomous Effects of Senescent Cells in the Skin
inferredcites →https://doi.org/10.1038/nature02118openalex-snapshot
A DNA damage checkpoint response in telomere-initiated senescence
inferredcites →https://doi.org/10.1038/nature05092openalex-snapshot
p16INK4a induces an age-dependent decline in islet regenerative potential
inferredcites →https://doi.org/10.1038/nature08227openalex-snapshot
Early-warning signals for critical transitions
inferredcites →https://doi.org/10.1038/nature10600openalex-snapshot
Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders
inferredcites →https://doi.org/10.1038/nature13193openalex-snapshot
The role of senescent cells in ageing
inferredcites →https://doi.org/10.1038/nature16550openalex-snapshot
The temporal scaling of Caenorhabditis elegans ageing
inferredcites →https://doi.org/10.1038/nature16932openalex-snapshot
Naturally occurring p16Ink4a-positive cells shorten healthy lifespan
inferredcites →https://doi.org/10.1038/ncb2784openalex-snapshot
A complex secretory program orchestrated by the inflammasome controls paracrine senescence
inferredcites →https://doi.org/10.1038/ncomms11190openalex-snapshot
Directed elimination of senescent cells by inhibition of BCL-W and BCL-XL
inferredcites →https://doi.org/10.1038/ncomms14532openalex-snapshot
Cellular senescence mediates fibrotic pulmonary disease
inferredcites →https://doi.org/10.1038/ncomms15691openalex-snapshot
Cellular senescence drives age-dependent hepatic steatosis
inferredcites →https://doi.org/10.1038/ng1608openalex-snapshot
A stress-sensitive reporter predicts longevity in isogenic populations of Caenorhabditis elegans
inferredcites →https://doi.org/10.1038/nm.4000openalex-snapshot
Cellular senescence in aging and age-related disease: from mechanisms to therapy
inferredcites →https://doi.org/10.1038/nm.4010openalex-snapshot
Clearance of senescent cells by ABT263 rejuvenates aged hematopoietic stem cells in mice
inferredcites →https://doi.org/10.1038/nm.4054openalex-snapshot
p16Ink4a-induced senescence of pancreatic beta cells enhances insulin secretion
inferredcites →https://doi.org/10.1038/nm.4324openalex-snapshot
Local clearance of senescent cells attenuates the development of post-traumatic osteoarthritis and creates a pro-regenerative environment
inferredcites →https://doi.org/10.1038/nprot.2009.191openalex-snapshot
Protocols to detect senescence-associated beta-galactosidase (SA-βgal) activity, a biomarker of senescent cells in culture and in vivo
inferredcites →https://doi.org/10.1038/nrc2440openalex-snapshot
Living on a break: cellular senescence as a DNA-damage response
inferredcites →https://doi.org/10.1038/nrm2233openalex-snapshot
Cellular senescence: when bad things happen to good cells
inferredcites →https://doi.org/10.1038/nrm3823openalex-snapshot
Cellular senescence: from physiology to pathology
inferredcites →https://doi.org/10.1038/s41467-017-00314-zopenalex-snapshot
Identification of HSP90 inhibitors as a novel class of senolytics
inferredcites →https://doi.org/10.1038/s41586-018-0543-yopenalex-snapshot
Clearance of senescent glial cells prevents tau-dependent pathology and cognitive decline
inferredcites →https://doi.org/10.1038/s41591-018-0092-9openalex-snapshot
Senolytics improve physical function and increase lifespan in old age
inferredcites →https://doi.org/10.1073/pnas.261228098openalex-snapshot
Explaining mortality rate plateaus
inferredcites →https://doi.org/10.1073/pnas.71.6.2169openalex-snapshot
Correlation Between Deoxyribonucleic Acid Excision-Repair and Life-Span in a Number of Mammalian Species
inferredcites →https://doi.org/10.1073/pnas.92.20.9363openalex-snapshot
A biomarker that identifies senescent human cells in culture and in aging skin in vivo.
inferredcites →https://doi.org/10.1074/jbc.ra117.001068openalex-snapshot
The invasin D protein from Yersinia pseudotuberculosis selectively binds the Fab region of host antibodies and affects colonization of the intestine
inferredcites →https://doi.org/10.1083/jcb.201206006openalex-snapshot
p53-dependent release of Alarmin HMGB1 is a central mediator of senescent phenotypes
inferredcites →https://doi.org/10.1093/carcin/bgh296openalex-snapshot
Senescence and immortalization: role of telomeres and telomerase
inferredcites →https://doi.org/10.1093/gerona/glu057openalex-snapshot
Chronic Inflammation (Inflammaging) and Its Potential Contribution to Age-Associated Diseases
inferredcites →https://doi.org/10.1093/ije/dyv105openalex-snapshot
Data Resource Profile: The Human Mortality Database (HMD)
inferredcites →https://doi.org/10.1098/rstb.2014.0379openalex-snapshot
Deciphering death: a commentary on Gompertz (1825) ‘On the nature of the function expressive of the law of human mortality, and on a new mode of determining the value of life contingencies’
inferredcites →https://doi.org/10.1098/rstl.1825.0026openalex-snapshot
XXIV. On the nature of the function expressive of the law of human mortality, and on a new mode of determining the value of life contingencies. In a letter to Francis Baily, Esq. F. R. S. &c
inferredcites →https://doi.org/10.1101/gad.235184.113openalex-snapshot
Cellular senescence and its effector programs
inferredcites →https://doi.org/10.1111/acel.12344openalex-snapshot
The Achilles’ heel of senescent cells: from transcriptome to senolytic drugs
inferredcites →https://doi.org/10.1111/acel.12445openalex-snapshot
Identification of a novel senolytic agent, navitoclax, targeting the Bcl‐2 family of anti‐apoptotic factors
inferredcites →https://doi.org/10.1111/acel.12458openalex-snapshot
Chronic senolytic treatment alleviates established vasomotor dysfunction in aged or atherosclerotic mice
inferredcites →https://doi.org/10.1111/acel.12592openalex-snapshot
Quantitative identification of senescent cells in aging and disease
inferredcites →https://doi.org/10.1111/acel.12840openalex-snapshot
Tau protein aggregation is associated with cellular senescence in the brain
inferredcites →https://doi.org/10.1111/acel.12841openalex-snapshot
Integrating cellular senescence with the concept of damage accumulation in aging: Relevance for clearance of senescent cells
inferredcites →https://doi.org/10.1111/j.1365-2567.2007.02555.xopenalex-snapshot
Immunosenescence: emerging challenges for an ageing population
inferredcites →https://doi.org/10.1111/j.1474-9726.2008.00377.xopenalex-snapshot
Age‐related intrinsic changes in human bone‐marrow‐derived mesenchymal stem cells and their differentiation to osteoblasts
inferredcites →https://doi.org/10.1111/j.1474-9726.2009.00489.xopenalex-snapshot
Expression of p16 INK4a in peripheral blood T‐cells is a biomarker of human aging
inferredcites →https://doi.org/10.1111/j.1474-9726.2012.00795.xopenalex-snapshot
A senescent cell bystander effect: senescence‐induced senescence
inferredcites →https://doi.org/10.1111/j.1523-1755.2004.00438.xopenalex-snapshot
Expression of p16INK4a and other cell cycle regulator and senescence associated genes in aging human kidney
inferredcites →https://doi.org/10.1111/jgs.14969openalex-snapshot
The Clinical Potential of Senolytic Drugs
inferredcites →https://doi.org/10.1126/science.1057991openalex-snapshot
Extension of Life-Span by Loss of CHICO, a Drosophila Insulin Receptor Substrate Protein
inferredcites →https://doi.org/10.1126/science.1069523openalex-snapshot
Senescence Induced by Altered Telomere State, Not Telomere Loss
inferredcites →https://doi.org/10.1126/science.1086016openalex-snapshot
Demography of Dietary Restriction and Death in Drosophila
inferredcites →https://doi.org/10.1126/science.1122446openalex-snapshot
Cellular Senescence in Aging Primates
inferredcites →https://doi.org/10.1126/science.1213491openalex-snapshot
Fitness Trade-Offs and Environmentally Induced Mutation Buffering in Isogenic C. elegans
inferredcites →https://doi.org/10.1126/science.2392681openalex-snapshot
Increased Life-Span of age -1 Mutants in Caenorhabditis elegans and Lower Gompertz Rate of Aging
inferredcites →https://doi.org/10.1126/science.280.5365.855openalex-snapshot
Biodemographic Trajectories of Longevity
inferredcites →https://doi.org/10.1126/science.aaf6659openalex-snapshot
Senescent intimal foam cells are deleterious at all stages of atherosclerosis
inferredcites →https://doi.org/10.1126/science.aam9309openalex-snapshot
Quantitative analysis of population-scale family trees with millions of relatives
inferredcites →https://doi.org/10.1146/annurev-biochem-060614-033955openalex-snapshot
The Biology of Proteostasis in Aging and Disease
inferredcites →https://doi.org/10.1146/annurev-physiol-030212-183712openalex-snapshot
Genetics of Longevity in Model Organisms: Debates and Paradigm Shifts
inferredcites →https://doi.org/10.1159/000485845openalex-snapshot
Tubular Cell Senescence in the Donated Kidney Predicts Allograft Function, but Not Donor Remnant Kidney Function, in Living Donor Kidney Transplantation
inferredcites →https://doi.org/10.1172/jci95149openalex-snapshot
Strategies targeting cellular senescence
inferredcites →https://doi.org/10.1183/09031936.03.00011903openalex-snapshot
Bleomycin induces cellular senescence in alveolar epithelial cells
inferredcites →https://doi.org/10.1371/journal.pone.0190374openalex-snapshot
Investigation of quercetin and hyperoside as senolytics in adult human endothelial cells
inferredcites →https://doi.org/10.1534/genetics.118.301613openalex-snapshot
Estimates of the Heritability of Human Longevity Are Substantially Inflated due to Assortative Mating
inferredcites →https://doi.org/10.18632/aging.100866openalex-snapshot
DNA repair in species with extreme lifespan differences
inferredcites →https://doi.org/10.18632/aging.101202openalex-snapshot
New agents that target senescent cells: the flavone, fisetin, and the BCL-XL inhibitors, A1331852 and A1155463
inferredcites →https://doi.org/10.48550/arxiv.1502.04307openalex-snapshot
Critical dynamics of gene networks is a mechanism behind ageing and Gompertz law
inferredcites →https://doi.org/10.7554/elife.12997openalex-snapshot
Targeting senescent cells enhances adipogenesis and metabolic function in old age
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1007/978-3-030-44903-2_4openalex-snapshot
Senolytics Target Senescent Cells and Improve Aging and Age-Related Diseases
inferred← citeshttps://doi.org/10.1016/j.mad.2021.111550openalex-snapshot
Breathe it in – Spotlight on senescence and regeneration in the lung
inferred← citeshttps://doi.org/10.1073/pnas.1906592116openalex-snapshot
Proteostasis collapse is a driver of cell aging and death
inferred← citeshttps://doi.org/10.70675/f49bc069z5ee6z4c49zb46fz0a1413dbba95openalex-snapshot
Implication of the receptor for advanced glycation end-products (RAGE) during inflammation and ageing
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