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Significant enrichment of Herpesvirus interactors in GWAS data suggests causal inferences for the association between Epstein Barr virus and multiple sclerosis

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/ana.21978openalex-snapshot

Primary infection with the Epstein‐Barr virus and risk of multiple sclerosis

inferredcites →https://doi.org/10.1007/s12185-018-2475-6openalex-snapshot

Comprehensive molecular diagnosis of Epstein–Barr virus-associated lymphoproliferative diseases using next-generation sequencing

inferredcites →https://doi.org/10.1016/j.ajhg.2009.05.011openalex-snapshot

Gene Ontology Analysis of GWA Study Data Sets Provides Insights into the Biology of Bipolar Disorder

inferredcites →https://doi.org/10.1016/j.ajhg.2017.06.005openalex-snapshot

10 Years of GWAS Discovery: Biology, Function, and Translation

inferredcites →https://doi.org/10.1016/j.ajhg.2018.04.002openalex-snapshot

The Post-GWAS Era: From Association to Function

inferredcites →https://doi.org/10.1016/j.cell.2009.12.018openalex-snapshot

A Physical and Regulatory Map of Host-Influenza Interactions Reveals Pathways in H1N1 Infection

inferredcites →https://doi.org/10.1016/j.cell.2018.05.051openalex-snapshot

Common Disease Is More Complex Than Implied by the Core Gene Omnigenic Model

inferredcites →https://doi.org/10.1016/j.cell.2018.07.016openalex-snapshot

Cooperative Metabolic Adaptations in the Host Can Favor Asymptomatic Infection and Select for Attenuated Virulence in an Enteric Pathogen

inferredcites →https://doi.org/10.1016/j.cell.2018.07.038openalex-snapshot

Microbiome: Focus on Causation and Mechanism

inferredcites →https://doi.org/10.1016/j.cell.2018.08.011openalex-snapshot

Memory B Cells Activate Brain-Homing, Autoreactive CD4+ T Cells in Multiple Sclerosis

inferredcites →https://doi.org/10.1016/j.cell.2018.09.049openalex-snapshot

Low-Frequency and Rare-Coding Variation Contributes to Multiple Sclerosis Risk

inferredcites →https://doi.org/10.1016/j.chom.2014.12.013openalex-snapshot

Epstein-Barr Virus Oncoprotein Super-enhancers Control B Cell Growth

inferredcites →https://doi.org/10.1016/j.chom.2015.06.004openalex-snapshot

Global Mapping of the Inc-Human Interactome Reveals that Retromer Restricts Chlamydia Infection

inferredcites →https://doi.org/10.1016/j.cytogfr.2017.11.001openalex-snapshot

Nuclear sirtuins and inflammatory signaling pathways

inferredcites →https://doi.org/10.1016/j.immuni.2011.06.014openalex-snapshot

Mapping a Dynamic Innate Immunity Protein Interaction Network Regulating Type I Interferon Production

inferredcites →https://doi.org/10.1016/j.it.2011.03.008openalex-snapshot

Interferon-β exacerbates Th17-mediated inflammatory disease

inferredcites →https://doi.org/10.1016/j.neuron.2018.05.023openalex-snapshot

Multiscale Analysis of Independent Alzheimer’s Cohorts Finds Disruption of Molecular, Genetic, and Clinical Networks by Human Herpesvirus

inferredcites →https://doi.org/10.1017/s0266462314000221openalex-snapshot

RITUXIMAB AND TOCILIZUMAB FOR THE TREATMENT OF RHEUMATOID ARTHRITIS

inferredcites →https://doi.org/10.1038/msb.2008.66openalex-snapshot

Hepatitis C virus infection protein network

inferredcites →https://doi.org/10.1038/msb.2013.26openalex-snapshot

The functional interactome landscape of the human histone deacetylase family

inferredcites →https://doi.org/10.1038/nature10251openalex-snapshot

Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis

inferredcites →https://doi.org/10.1038/nature10719openalex-snapshot

Global landscape of HIV–human protein complexes

inferredcites →https://doi.org/10.1038/nature11289openalex-snapshot

Viral immune modulators perturb the human molecular network by common and unique strategies

inferredcites →https://doi.org/10.1038/nature11307openalex-snapshot

TNF receptor 1 genetic risk mirrors outcome of anti-TNF therapy in multiple sclerosis

inferredcites →https://doi.org/10.1038/ncomms11350openalex-snapshot

Sex bias in CNS autoimmune disease mediated by androgen control of autoimmune regulator

inferredcites →https://doi.org/10.1038/ng.2770openalex-snapshot

Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis

inferredcites →https://doi.org/10.1038/ng.670openalex-snapshot

Meta-analysis of three genome-wide association studies identifies susceptibility loci for colorectal cancer at 1q41, 3q26.2, 12q13.13 and 20q13.33

inferredcites →https://doi.org/10.1038/nri.2016.9openalex-snapshot

AIRE expands: new roles in immune tolerance and beyond

inferredcites →https://doi.org/10.1038/nsmb.2059openalex-snapshot

Genome-wide CTCF distribution in vertebrates defines equivalent sites that aid the identification of disease-associated genes

inferredcites →https://doi.org/10.1038/s41467-019-08666-4openalex-snapshot

Functional genomics reveal gene regulatory mechanisms underlying schizophrenia risk

inferredcites →https://doi.org/10.1038/s41586-018-0571-7openalex-snapshot

Genome-wide association studies of brain imaging phenotypes in UK Biobank

inferredcites →https://doi.org/10.1038/s41586-018-0579-zopenalex-snapshot

The UK Biobank resource with deep phenotyping and genomic data

inferredcites →https://doi.org/10.1038/s41588-018-0102-3openalex-snapshot

Transcription factors operate across disease loci, with EBNA2 implicated in autoimmunity

inferredcites →https://doi.org/10.1038/s41588-018-0255-0openalex-snapshot

Distinguishing genetic correlation from causation across 52 diseases and complex traits

inferredcites →https://doi.org/10.1038/s41598-017-09286-yopenalex-snapshot

Transcriptional dysregulation of Interferome in experimental and human Multiple Sclerosis

inferredcites →https://doi.org/10.1056/nejmoa1610528openalex-snapshot

Overexpression of the Cytokine BAFF and Autoimmunity Risk

inferredcites →https://doi.org/10.1073/pnas.1811974115openalex-snapshot

Herpesvirus trigger accelerates neuroinflammation in a nonhuman primate model of multiple sclerosis

inferredcites →https://doi.org/10.1093/brain/awx372openalex-snapshot

Multiple sclerosis risk variants alter expression of co-stimulatory genes in B cells

inferredcites →https://doi.org/10.1093/brain/awy239openalex-snapshot

Learning from other autoimmunities to understand targeting of B cells to control multiple sclerosis

inferredcites →https://doi.org/10.1093/hmg/ddr250openalex-snapshot

Interrogating the complex role of chromosome 16p13.13 in multiple sclerosis susceptibility: independent genetic signals in the CIITA–CLEC16A–SOCS1 gene complex

inferredcites →https://doi.org/10.1093/hmg/ddu328openalex-snapshot

Mendelian randomization: genetic anchors for causal inference in epidemiological studies

inferredcites →https://doi.org/10.1093/nar/gkn851openalex-snapshot

miRecords: an integrated resource for microRNA-target interactions

inferredcites →https://doi.org/10.1093/nar/gkr1099openalex-snapshot

MetaBase--the wiki-database of biological databases

inferredcites →https://doi.org/10.1093/nar/gku1204openalex-snapshot

The BioGRID interaction database: 2015 update

inferredcites →https://doi.org/10.1093/nar/gku830openalex-snapshot

VirusMentha: a new resource for virus-host protein interactions

inferredcites →https://doi.org/10.1101/gr.137323.112openalex-snapshot

Annotation of functional variation in personal genomes using RegulomeDB

inferredcites →https://doi.org/10.1111/2049-632x.12077openalex-snapshot

Susceptibility genes are enriched in those of the herpes simplex virus 1/host interactome in psychiatric and neurological disorders

inferredcites →https://doi.org/10.1126/science.1257601openalex-snapshot

Uncovering disease-disease relationships through the incomplete interactome

inferredcites →https://doi.org/10.1128/jvi.01570-06openalex-snapshot

The Epstein-Barr Virus-Encoded LMP-1 Oncoprotein Negatively Affects Tyk2 Phosphorylation and Interferon Signaling in Human B Cells

inferredcites →https://doi.org/10.1136/ard.2003.020347openalex-snapshot

A multicentre, randomised, double blind, placebo controlled phase II study of subcutaneous interferon beta-1a in the treatment of patients with active rheumatoid arthritis

inferredcites →https://doi.org/10.1155/2013/535738openalex-snapshot

Epstein-Barr Virus in Systemic Autoimmune Diseases

inferredcites →https://doi.org/10.1172/jci.insight.124714openalex-snapshot

Epstein-Barr virus–specific T cell therapy for progressive multiple sclerosis

inferredcites →https://doi.org/10.1177/1352458518823940openalex-snapshot

The role of infections in multiple sclerosis

inferredcites →https://doi.org/10.1186/ar1026openalex-snapshot

A randomized, controlled trial of interferon-β-1a (Avonex®) in patients with rheumatoid arthritis: a pilot study [ISRCTN03626626]

inferredcites →https://doi.org/10.1212/nxi.0000000000000337openalex-snapshot

Dysregulation of MS risk genes and pathways at distinct stages of disease

inferredcites →https://doi.org/10.1212/wnl.0000000000001420openalex-snapshot

Epstein-Barr virus genetic variants are associated with multiple sclerosis

inferredcites →https://doi.org/10.1371/journal.pcbi.1002531openalex-snapshot

Viral Perturbations of Host Networks Reflect Disease Etiology

inferredcites →https://doi.org/10.1371/journal.pone.0063300openalex-snapshot

A “Candidate-Interactome” Aggregate Analysis of Genome-Wide Association Data in Multiple Sclerosis

inferredcites →https://doi.org/10.1371/journal.pone.0119605openalex-snapshot

EBNA2 Binds to Genomic Intervals Associated with Multiple Sclerosis and Overlaps with Vitamin D Receptor Occupancy

inferredcites →https://doi.org/10.1523/jneurosci.4507-10.2011openalex-snapshot

Changed Histone Acetylation Patterns in Normal-Appearing White Matter and Early Multiple Sclerosis Lesions

inferredcites →https://doi.org/10.3389/fneur.2019.00095openalex-snapshot

Genome-Wide Multiple Sclerosis Association Data and Coagulation

inferredcites →https://doi.org/10.3892/or.2015.4134openalex-snapshot

GALC gene is downregulated by promoter hypermethylation in Epstein-Barr virus-associated nasopharyngeal carcinoma

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1016/j.msard.2022.103563openalex-snapshot

Identification of multiple sclerosis-related genes regulated by EBV-encoded microRNAs in B cells

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