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PLD3 is a neuronal lysosomal phospholipase D associated with β-amyloid plaques and cognitive function in Alzheimer’s disease

https://doi.org/10.1101/746222
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/ana.21142openalex-snapshot
Subcortical infarcts, Alzheimer's disease pathology, and memory function in older persons
inferredcites →https://doi.org/10.1002/ana.21154openalex-snapshot
TDP‐43 immunoreactivity in hippocampal sclerosis and Alzheimer's disease
inferredcites →https://doi.org/10.1016/0006-291x(73)90674-8openalex-snapshot
Solubilization and properties of a membrane-bound enzyme from rat brain catalyzing a base-exchange reaction
inferredcites →https://doi.org/10.1016/0006-8993(94)91858-9openalex-snapshot
Lysosomal abnormalities in degenerating neurons link neuronal compromise to senile plaque development in Alzheimer disease
inferredcites →https://doi.org/10.1016/j.bcp.2014.01.011openalex-snapshot
Behavioral assays with mouse models of Alzheimer's disease: Practical considerations and guidelines
inferredcites →https://doi.org/10.1016/j.celrep.2017.12.100openalex-snapshot
Unconventional Trafficking of Mammalian Phospholipase D3 to Lysosomes
inferredcites →https://doi.org/10.1016/j.jalz.2017.09.012openalex-snapshot
Conserved brain myelination networks are altered in Alzheimer's and other neurodegenerative diseases
inferredcites →https://doi.org/10.1016/j.mex.2017.03.003openalex-snapshot
Method for organotypic tissue culture in the aged animal
inferredcites →https://doi.org/10.1016/j.nbd.2019.05.015openalex-snapshot
V232M substitution restricts a distinct O-glycosylation of PLD3 and its neuroprotective function
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2017.12.025openalex-snapshot
The effect of rare variants in TREM2 and PLD3 on longitudinal cognitive function in the Wisconsin Registry for Alzheimer's Prevention
inferredcites →https://doi.org/10.1016/j.neuron.2018.11.040openalex-snapshot
Harnessing Genetic Complexity to Enhance Translatability of Alzheimer’s Disease Mouse Models: A Path toward Precision Medicine
inferredcites →https://doi.org/10.1016/s0014-5793(98)01578-6openalex-snapshot
Application of magnetic chromatography to the isolation of lysosomes from fibroblasts of patients with lysosomal storage disorders
inferredcites →https://doi.org/10.1038/nature11502openalex-snapshot
The structural biochemistry of Zucchini implicates it as a nuclease in piRNA biogenesis
inferredcites →https://doi.org/10.1038/nature12825openalex-snapshot
Rare coding variants in the phospholipase D3 gene confer risk for Alzheimer’s disease
inferredcites →https://doi.org/10.1038/nature14036openalex-snapshot
PLD3 and sporadic Alzheimer's disease risk
inferredcites →https://doi.org/10.1038/nature14038openalex-snapshot
PLD3 variants in population studies
inferredcites →https://doi.org/10.1038/nature14039openalex-snapshot
PLD3 in non-familial Alzheimer's disease
inferredcites →https://doi.org/10.1038/nature14040openalex-snapshot
PLD3 gene variants and Alzheimer's disease
inferredcites →https://doi.org/10.1038/nature21030openalex-snapshot
PLD3 gene and processing of APP
inferredcites →https://doi.org/10.1038/ng.2231openalex-snapshot
Meta-analysis identifies nine new loci associated with rheumatoid arthritis in the Japanese population
inferredcites →https://doi.org/10.1038/nrd.2016.252openalex-snapshot
Targeting phospholipase D in cancer, infection and neurodegenerative disorders
inferredcites →https://doi.org/10.1038/s41380-019-0458-5openalex-snapshot
Brain expression of the vascular endothelial growth factor gene family in cognitive aging and alzheimer’s disease
inferredcites →https://doi.org/10.1038/s41590-018-0179-yopenalex-snapshot
PLD3 and PLD4 are single-stranded acid exonucleases that regulate endosomal nucleic-acid sensing
inferredcites →https://doi.org/10.1038/s41593-018-0154-9openalex-snapshot
A molecular network of the aging human brain provides insights into the pathology and cognitive decline of Alzheimer’s disease
inferredcites →https://doi.org/10.1038/sdata.2016.89openalex-snapshot
Human whole genome genotype and transcriptome data for Alzheimer’s and other neurodegenerative diseases
inferredcites →https://doi.org/10.1038/sdata.2018.185openalex-snapshot
The Mount Sinai cohort of large-scale genomic, transcriptomic and proteomic data in Alzheimer's disease
inferredcites →https://doi.org/10.1073/pnas.1510329112openalex-snapshot
Massive accumulation of luminal protease-deficient axonal lysosomes at Alzheimer’s disease amyloid plaques
inferredcites →https://doi.org/10.1074/jbc.m508800200openalex-snapshot
Kinetic Analysis of a Mammalian Phospholipase D
inferredcites →https://doi.org/10.1093/brain/awy258openalex-snapshot
PLD3 and spinocerebellar ataxia
inferredcites →https://doi.org/10.1101/225714openalex-snapshot
Systems genetics identifies modifiers of Alzheimer’s disease risk and resilience
inferredcites →https://doi.org/10.1111/bpa.12365openalex-snapshot
The Relationship of Cerebral Vessel Pathology to Brain Microinfarcts
inferredcites →https://doi.org/10.1136/annrheumdis-2018-214116openalex-snapshot
PLD4 is a genetic determinant to systemic lupus erythematosus and involved in murine autoimmune phenotypes
inferredcites →https://doi.org/10.1161/strokeaha.110.595082openalex-snapshot
Microinfarct Pathology, Dementia, and Cognitive Systems
inferredcites →https://doi.org/10.1161/strokeaha.111.623462openalex-snapshot
Cerebrovascular Disease Pathology and Parkinsonian Signs in Old Age
inferredcites →https://doi.org/10.1186/s13195-014-0070-5openalex-snapshot
PLD3 is accumulated on neuritic plaques in Alzheimer’s disease brains
inferredcites →https://doi.org/10.1212/01.wnl.0000055863.87435.b2openalex-snapshot
Relation of cerebral infarctions to dementia and cognitive function in older persons
inferredcites →https://doi.org/10.1212/01.wnl.0000271090.28148.24openalex-snapshot
Mixed brain pathologies account for most dementia cases in community-dwelling older persons
inferredcites →https://doi.org/10.1212/wnl.0000000000001935openalex-snapshot
Temporal course and pathologic basis of unawareness of memory loss in dementia
inferredcites →https://doi.org/10.1212/wnl.0000000000002175openalex-snapshot
Cerebral amyloid angiopathy and cognitive outcomes in community-based older persons
inferredcites →https://doi.org/10.1371/journal.pone.0033341openalex-snapshot
A Role for Phospholipase D3 in Myotube Formation
inferredcites →https://doi.org/10.1523/jneurosci.1202-06.2006openalex-snapshot
Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation
inferredcites →https://doi.org/10.18632/oncotarget.19419openalex-snapshot
rs2841277 (PLD4) is associated with susceptibility and rs4672495 is associated with disease activity in rheumatoid arthritis
inferredcites →https://doi.org/10.2174/156720512801322573openalex-snapshot
Overview and Findings from the Religious Orders Study
inferredcites →https://doi.org/10.2174/156720512801322663openalex-snapshot
Overview and Findings from the Rush Memory and Aging Project
inferredcites →https://doi.org/10.25405/ncl.34306728openalex-snapshot
Development, appraisal, validation and implementation of a consensus protocol for the assessment of cerebral amyloid angiopathy in post-mortem brain tissue
inferredcites →https://doi.org/10.3233/jad-179939openalex-snapshot
Religious Orders Study and Rush Memory and Aging Project
inferredcites →https://doi.org/10.3389/fgene.2014.00088openalex-snapshot
Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1002/cbf.3848openalex-snapshot
OGD/R‐induced ferroptosis and pyroptosis in retinal pigment epithelium cells: Role of PLD1 and PLD2 modulation
inferred← citeshttps://doi.org/10.1016/j.heliyon.2024.e39378openalex-snapshot
Unveiling mitochondria as central components driving cognitive decline in alzheimer's disease through cross-transcriptomic analysis of hippocampus and entorhinal cortex microarray datasets
inferred← citeshttps://doi.org/10.1038/s41586-022-05491-6openalex-snapshot
PLD3 affects axonal spheroids and network defects in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1038/s43587-025-00823-3openalex-snapshot
Subcellular proteomics and iPSC modeling uncover reversible mechanisms of axonal pathology in Alzheimer’s disease
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