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miR‐155 deficiency protects mice from experimental colitis by reducing T helper type 1/type 17 responses
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/art.24436openalex-snapshot
MicroRNA‐146a contributes to abnormal activation of the type I interferon pathway in human lupus by targeting the key signaling proteins
inferredcites →https://doi.org/10.1002/eji.200939381openalex-snapshot
Micro‐RNA‐155 inhibits IFN‐γ signaling in CD4 + T cells
inferredcites →https://doi.org/10.1002/eji.201242569openalex-snapshot
IL ‐17‐producing γδ T cells and innate lymphoid cells
inferredcites →https://doi.org/10.1002/immu.200390017openalex-snapshot
Increased expression of DC‐SIGN+IL‐12+IL‐18+ and CD83+IL‐12–IL‐18– dendritic cell populations in the colonic mucosa of patients with Crohn's disease
inferredcites →https://doi.org/10.1016/1074-7613(94)90045-0openalex-snapshot
Inhibition of Thl responses prevents inflammatory bowel disease in scid mice reconstituted with CD45RBhi CD4+ T cells
inferredcites →https://doi.org/10.1016/j.bbi.2011.07.236openalex-snapshot
Role of resveratrol-induced CD11b+ Gr-1+ myeloid derived suppressor cells (MDSCs) in the reduction of CXCR3+ T cells and amelioration of chronic colitis in IL-10−/− mice
inferredcites →https://doi.org/10.1016/j.clim.2003.09.013openalex-snapshot
Neutralization of interleukin-17 aggravates dextran sulfate sodium-induced colitis in mice
inferredcites →https://doi.org/10.1016/j.immuni.2010.09.009openalex-snapshot
MicroRNA-155 Promotes Autoimmune Inflammation by Enhancing Inflammatory T Cell Development
inferredcites →https://doi.org/10.1016/s0952-7915(97)80153-7openalex-snapshot
Origin, maturation and antigen presenting function of dendritic cells
inferredcites →https://doi.org/10.1016/s1074-7613(00)80170-3openalex-snapshot
Abrogation of TGFβ Signaling in T Cells Leads to Spontaneous T Cell Differentiation and Autoimmune Disease
inferredcites →https://doi.org/10.1038/mi.2013.82openalex-snapshot
IL-27, targeting antigen-presenting cells, promotes Th17 differentiation and colitis in mice
inferredcites →https://doi.org/10.1038/nri2708openalex-snapshot
Physiological and pathological roles for microRNAs in the immune system
inferredcites →https://doi.org/10.1053/gast.2000.16500openalex-snapshot
Mice deficient in Th1- and Th2-type cytokines develop distinct forms of hapten-induced colitis
inferredcites →https://doi.org/10.1053/j.gastro.2008.07.068openalex-snapshot
MicroRNAs Are Differentially Expressed in Ulcerative Colitis and Alter Expression of Macrophage Inflammatory Peptide-2α
inferredcites →https://doi.org/10.1056/nejm199109263251306openalex-snapshot
Inflammatory Bowel Disease
inferredcites →https://doi.org/10.1073/pnas.0605298103openalex-snapshot
NF-κB-dependent induction of microRNA miR-146, an inhibitor targeted to signaling proteins of innate immune responses
inferredcites →https://doi.org/10.1084/jem.182.5.1281openalex-snapshot
Antibodies to interleukin 12 abrogate established experimental colitis in mice.
inferredcites →https://doi.org/10.1089/107999003322485099openalex-snapshot
IFN-γ-Inducible Chemokines Enhance Adaptive Immunity and Colitis
inferredcites →https://doi.org/10.1093/jn/132.5.893openalex-snapshot
The Leptin Defense against Wasting Is Abolished in the IL-2–Deficient Mouse Model of Inflammatory Bowel Disease
inferredcites →https://doi.org/10.1097/01.mib.0000218764.06959.91openalex-snapshot
Critical role of IL-17 receptor signaling in acute TNBS-induced colitis
inferredcites →https://doi.org/10.1111/j.1440-1746.2009.06216.xopenalex-snapshot
Increased expression of microRNA in the inflamed colonic mucosa of patients with active ulcerative colitis
inferredcites →https://doi.org/10.1124/jpet.109.160838openalex-snapshot
Resveratrol (Trans-3,5,4′-trihydroxystilbene) Induces Silent Mating Type Information Regulation-1 and Down-Regulates Nuclear Transcription Factor-κB Activation to Abrogate Dextran Sulfate Sodium-Induced Colitis
inferredcites →https://doi.org/10.1126/science.1139253openalex-snapshot
Requirement of bic/microRNA-155 for Normal Immune Function
inferredcites →https://doi.org/10.1136/gut.39.5.684openalex-snapshot
Increased production of tumour necrosis factor-alpha interleukin-1 beta, and interleukin-6 by morphologically normal intestinal biopsies from patients with Crohn's disease.
inferredcites →https://doi.org/10.1136/gut.41.6.793openalex-snapshot
Cytokine mRNA expression in intestinal tissue of interleukin-2 deficient mice with bowel inflammation
inferredcites →https://doi.org/10.1136/gut.52.1.65openalex-snapshot
Increased expression of interleukin 17 in inflammatory bowel disease
inferredcites →https://doi.org/10.1172/jci21404openalex-snapshot
IL-23 is essential for T cell–mediated colitis and promotes inflammation via IL-17 and IL-6
inferredcites →https://doi.org/10.1182/blood-2005-07-2860openalex-snapshot
Gut-associated lymphoid tissue–primed CD4+ T cells display CCR9-dependent and -independent homing to the small intestine
inferredcites →https://doi.org/10.1182/blood-2010-04-280156openalex-snapshot
miR-155 and its star-form partner miR-155* cooperatively regulate type I interferon production by human plasmacytoid dendritic cells
inferredcites →https://doi.org/10.1182/blood-2010-12-322503openalex-snapshot
miR-221 and miR-155 regulate human dendritic cell development, apoptosis, and IL-12 production through targeting of p27kip1, KPC1, and SOCS-1
inferredcites →https://doi.org/10.1182/blood.v84.12.4008.bloodjournal84124008openalex-snapshot
Interleukin-12: a cytokine produced by antigen-presenting cells with immunoregulatory functions in the generation of T-helper cells type 1 and cytotoxic lymphocytes
inferredcites →https://doi.org/10.1371/journal.pone.0013160openalex-snapshot
Identification of Restricted Subsets of Mature microRNA Abnormally Expressed in Inactive Colonic Mucosa of Patients with Inflammatory Bowel Disease
inferredcites →https://doi.org/10.1371/journal.pone.0073757openalex-snapshot
Effects of Sinomenine on the Expression of microRNA-155 in 2,4,6-Trinitrobenzenesulfonic Acid-Induced Colitis in Mice
inferredcites →https://doi.org/10.4049/jimmunol.1101772openalex-snapshot
MicroRNA-155 Is Essential for the T Cell-Mediated Control of Helicobacter pylori Infection and for the Induction of Chronic Gastritis and Colitis
inferredcites →https://doi.org/10.4049/jimmunol.1300351openalex-snapshot
MicroRNA-155 Confers Encephalogenic Potential to Th17 Cells by Promoting Effector Gene Expression
inferredcites →https://doi.org/10.4049/jimmunol.157.5.2174openalex-snapshot
Hapten-induced model of murine inflammatory bowel disease: mucosa immune responses and protection by tolerance
inferredcites →https://doi.org/10.4049/jimmunol.158.2.566openalex-snapshot
Induction and prevention of colonic inflammation in IL-2-deficient mice
inferredcites →https://doi.org/10.4049/jimmunol.164.6.2890openalex-snapshot
A Bone Marrow-Derived APC in the Gut-Associated Lymphoid Tissue Captures Oral Antigens and Presents Them to Both CD4+ and CD8+ T Cells
inferredcites →https://doi.org/10.4049/jimmunol.171.3.1401openalex-snapshot
Inhibition of IFN-γ-Inducible Protein-10 Abrogates Colitis in IL-10−/− Mice
inferredcites →https://doi.org/10.4049/jimmunol.179.8.5082openalex-snapshot
Modulation of miR-155 and miR-125b Levels following Lipopolysaccharide/TNF-α Stimulation and Their Possible Roles in Regulating the Response to Endotoxin Shock
inferredcites →https://doi.org/10.4049/jimmunol.180.11.7423openalex-snapshot
Chronically Inflamed Human Tissues Are Infiltrated by Highly Differentiated Th17 Lymphocytes
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4834005openalex-extracted
DJ-X-013 Reduces LPS-Induced Inflammation, Modulates Th17/ Myeloid-Derived Suppressor Cells, and Alters NF-kB Expression to Ameliorate Experimental Colitis
inferred← citeshttps://doi.org/10.1002/bies.201600200openalex-snapshot
MicroRNAs at the epicenter of intestinal homeostasis
inferred← citeshttps://doi.org/10.1002/eji.201545487openalex-snapshot
Cross‐regulation between cytokine and microRNA pathways in T cells
inferred← citeshttps://doi.org/10.1002/jcb.26205openalex-snapshot
Altered Expression of MicroRNAs in Rheumatoid Arthritis
inferred← citeshttps://doi.org/10.1002/jcp.27173openalex-snapshot
MiRNAs and inflammatory bowel disease: An interesting new story
inferred← citeshttps://doi.org/10.1002/jlb.3mir0817-346rrropenalex-snapshot
Modulating inflammation through the negative regulation of NF-κB signaling
inferred← citeshttps://doi.org/10.1002/med.22001openalex-snapshot
Artemisinins: Promising drug candidates for the treatment of autoimmune diseases
inferred← citeshttps://doi.org/10.1002/mnfr.201700144openalex-snapshot
Differential intestinal anti‐inflammatory effects of Lactobacillus fermentum and Lactobacillus salivarius in DSS mouse colitis: impact on microRNAs expression and microbiota composition
inferred← citeshttps://doi.org/10.1002/mnfr.201900633openalex-snapshot
Resveratrol Downregulates miR‐31 to Promote T Regulatory Cells during Prevention of TNBS‐Induced Colitis
inferred← citeshttps://doi.org/10.1007/978-1-4939-3335-8_24openalex-snapshot
The Use of MiRNA Antagonists in the Alleviation of Inflammatory Disorders
inferred← citeshttps://doi.org/10.1007/978-3-030-28703-0_10openalex-snapshot
MicroRNAs and Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1007/978-3-319-42059-2_3openalex-snapshot
MicroRNAs and Inflammation in Colorectal Cancer
inferred← citeshttps://doi.org/10.1007/s00011-022-01643-6openalex-snapshot
Role of miR-155 in inflammatory autoimmune diseases: a comprehensive review
inferred← citeshttps://doi.org/10.1007/s00018-016-2167-4openalex-snapshot
Contribution of MicroRNAs to autoimmune diseases
inferred← citeshttps://doi.org/10.1007/s00281-019-00732-9openalex-snapshot
Epigenetic regulation of T helper cells and intestinal pathogenicity
inferred← citeshttps://doi.org/10.1007/s10238-022-00935-9openalex-snapshot
Updated immunomodulatory roles of gut flora and microRNAs in inflammatory bowel diseases
inferred← citeshttps://doi.org/10.1007/s10620-017-4567-1openalex-snapshot
Role of MiRNAs in Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1007/s11064-014-1500-2openalex-snapshot
Suppression of MicroRNA-155 Attenuates Neuropathic Pain by Regulating SOCS1 Signalling Pathway
inferred← citeshttps://doi.org/10.1007/s11302-022-09840-yopenalex-snapshot
MicroRNA: Crucial modulator in purinergic signalling involved diseases
inferred← citeshttps://doi.org/10.1007/s13238-017-0398-2openalex-snapshot
The development and function of dendritic cell populations and their regulation by miRNAs
inferred← citeshttps://doi.org/10.1016/b978-0-12-809912-4.00004-0openalex-snapshot
MicroRNA and T Helper Cell-Mediated Immune Responses
inferred← citeshttps://doi.org/10.1016/b978-0-12-816843-1.00015-1openalex-snapshot
The role for the microbiome in the regulation of the circadian clock and metabolism
inferred← citeshttps://doi.org/10.1016/j.ajpath.2021.01.012openalex-snapshot
MicroRNA155 Plays a Critical Role in the Pathogenesis of Cutaneous Leishmania major Infection by Promoting a Th2 Response and Attenuating Dendritic Cell Activity
inferred← citeshttps://doi.org/10.1016/j.banm.2024.03.013openalex-snapshot
Rôle des miARN dans les maladies inflammatoires chroniques de l’intestin et potentiel diagnostique et thérapeutique
inferred← citeshttps://doi.org/10.1016/j.bbi.2016.06.008openalex-snapshot
Fatty acid amide hydrolase (FAAH) blockade ameliorates experimental colitis by altering microRNA expression and suppressing inflammation
inferred← citeshttps://doi.org/10.1016/j.biopha.2024.117379openalex-snapshot
DJ-X-013 reduces LPS-induced inflammation, modulates Th17/ myeloid-derived suppressor cells, and alters NF-κB expression to ameliorate experimental colitis
inferred← citeshttps://doi.org/10.1016/j.biopha.2025.117939openalex-snapshot
Modulation of occludin, NF-κB, p-STAT3, and Th17 response by DJ-X-025 decreases inflammation and ameliorates experimental colitis
inferred← citeshttps://doi.org/10.1016/j.celrep.2015.09.073openalex-snapshot
MicroRNA-223 Regulates the Differentiation and Function of Intestinal Dendritic Cells and Macrophages by Targeting C/EBPβ
inferred← citeshttps://doi.org/10.1016/j.isci.2022.105176openalex-snapshot
MYC-mediated silencing of miR-181a-5p promotes pathogenic Th17 responses by modulating AKT3-FOXO3 signaling
inferred← citeshttps://doi.org/10.1016/j.it.2022.09.003openalex-snapshot
miRNA effects on gut homeostasis: therapeutic implications for inflammatory bowel disease
inferred← citeshttps://doi.org/10.1016/j.jaut.2019.04.014openalex-snapshot
MicroRNA-125a suppresses intestinal mucosal inflammation through targeting ETS-1 in patients with inflammatory bowel diseases
inferred← citeshttps://doi.org/10.1016/j.jbc.2022.102461openalex-snapshot
MicroRNA-16 inhibits the TLR4/NF-κB pathway and maintains tight junction integrity in irritable bowel syndrome with diarrhea
inferred← citeshttps://doi.org/10.1016/j.jnutbio.2018.08.005openalex-snapshot
Intestinal anti-inflammatory effect of the probiotic Saccharomyces boulardii in DSS-induced colitis in mice: Impact on microRNAs expression and gut microbiota composition
inferred← citeshttps://doi.org/10.1016/j.jnutbio.2022.109101openalex-snapshot
Resveratrol and resveratrol nano-delivery systems in the treatment of inflammatory bowel disease
inferred← citeshttps://doi.org/10.1016/j.lfs.2023.122317openalex-snapshot
Inhibition of microRNA-34c reduces detrusor ROCK2 expression and urinary bladder inflammation in experimental cystitis
inferred← citeshttps://doi.org/10.1016/j.phrs.2021.105753openalex-snapshot
Mastiha has efficacy in immune-mediated inflammatory diseases through a microRNA-155 Th17 dependent action
inferred← citeshttps://doi.org/10.1016/j.prp.2017.08.001openalex-snapshot
miR-155 targets Est-1 and induces ulcerative colitis via the IL-23/17/6-mediated Th17 pathway
inferred← citeshttps://doi.org/10.1016/j.tips.2019.09.007openalex-snapshot
miRNAs as Therapeutic Targets in Inflammatory Disease
inferred← citeshttps://doi.org/10.1016/s1995-7645(14)60318-7openalex-snapshot
Expression level of miR–155 in peripheral blood
inferred← citeshttps://doi.org/10.1038/cddis.2016.165openalex-snapshot
MicroRNA-155 facilitates skeletal muscle regeneration by balancing pro- and anti-inflammatory macrophages
inferred← citeshttps://doi.org/10.1038/emm.2015.21openalex-snapshot
MiR-155 modulates the inflammatory phenotype of intestinal myofibroblasts by targeting SOCS1 in ulcerative colitis
inferred← citeshttps://doi.org/10.1038/s41366-018-0114-1openalex-snapshot
MicroRNA-30 modulates metabolic inflammation by regulating Notch signaling in adipose tissue macrophages
inferred← citeshttps://doi.org/10.1038/s41385-019-0216-7openalex-snapshot
MicroRNA-219a-5p suppresses intestinal inflammation through inhibiting Th1/Th17-mediated immune responses in inflammatory bowel disease
inferred← citeshttps://doi.org/10.1038/s41385-019-0241-6openalex-snapshot
The role of miR-155 in cigarette smoke-induced pulmonary inflammation and COPD
inferred← citeshttps://doi.org/10.1038/s41525-016-0001-4openalex-snapshot
Relation between NOD2 genotype and changes in innate signaling in Crohn’s disease on mRNA and miRNA levels
inferred← citeshttps://doi.org/10.1038/srep30824openalex-snapshot
MicroRNA-16 is putatively involved in the NF-κB pathway regulation in ulcerative colitis through adenosine A2a receptor (A2aAR) mRNA targeting
inferred← citeshttps://doi.org/10.1039/d0fo01724hopenalex-snapshot
Alleviation of DSS-induced colitis via Lactobacillus acidophilus treatment in mice
inferred← citeshttps://doi.org/10.1053/j.gastro.2015.09.049openalex-snapshot
Activity of SHIP, Which Prevents Expression of Interleukin 1β, Is Reduced in Patients With Crohn’s Disease
inferred← citeshttps://doi.org/10.1080/08830185.2019.1648454openalex-snapshot
Emerging roles of noncoding RNAs in T cell differentiation and functions in autoimmune diseases
inferred← citeshttps://doi.org/10.1080/13880209.2020.1867196openalex-snapshot
Artesunate alleviates the inflammatory response of ulcerative colitis by regulating the expression of miR-155
inferred← citeshttps://doi.org/10.1080/14728222.2024.2408423openalex-snapshot
Target potential of miRNAs in ulcerative colitis: what do we know?
inferred← citeshttps://doi.org/10.1080/2162402x.2020.1724761openalex-snapshot
Overexpression of microRNA-155 enhances the efficacy of dendritic cell vaccine against breast cancer
inferred← citeshttps://doi.org/10.1080/25787489.2021.1955196openalex-snapshot
Expression of microRNA-155 in circulating T cells is an indicator of immune activation levels in HIV-1 infected patients
inferred← citeshttps://doi.org/10.1089/bio.2022.0003openalex-snapshot
Genetically Engineered Exosomes as a Potential Regulator of Th1 Cells Response in Rheumatoid Arthritis
inferred← citeshttps://doi.org/10.1093/ecco-jcc/jjab223openalex-snapshot
MicroRNA and Gut Microbiota: Tiny but Mighty—Novel Insights into Their Cross-talk in Inflammatory Bowel Disease Pathogenesis and Therapeutics
inferred← citeshttps://doi.org/10.1093/ibd/izac122openalex-snapshot
MiRNA-Based Therapies for the Treatment of Inflammatory Bowel Disease: What Are We Still Missing?
inferred← citeshttps://doi.org/10.1093/ibd/izy046openalex-snapshot
Circulating and Fecal microRNAs as Biomarkers for Inflammatory Bowel Diseases
inferred← citeshttps://doi.org/10.1093/intimm/dxx019openalex-snapshot
MicroRNA-mediated dynamic control of mucosal immunity
inferred← citeshttps://doi.org/10.1101/2024.08.15.608161openalex-snapshot
Human-derived microRNA 21 regulates indole and L-tryptophan biosynthesis transcripts in a prominent gut symbiont
inferred← citeshttps://doi.org/10.1111/andr.70157openalex-snapshot
Histopathological Characterization and Differential Expression of miRNAs in Male Pediatric Patients With Lichen Sclerosus
inferred← citeshttps://doi.org/10.1111/hiv.12470openalex-snapshot
Micro RNA ‐155 is a biomarker of T‐cell activation and immune dysfunction in HIV ‐1‐infected patients
inferred← citeshttps://doi.org/10.1111/ijlh.13245openalex-snapshot
Differential expression of miRNAs and their target genes: Exploring a new perspective of acquired aplastic anemia pathogenesis
inferred← citeshttps://doi.org/10.1128/mbio.03928-24openalex-snapshot
Human-derived microRNA 21 regulates indole and L-tryptophan biosynthesis transcripts in the gut commensal Bacteroides thetaiotaomicron
inferred← citeshttps://doi.org/10.1136/gutjnl-2014-307891openalex-snapshot
MicroRNAs: new players in IBD
inferred← citeshttps://doi.org/10.1136/gutjnl-2015-309389openalex-snapshot
miR-301a promotes intestinal mucosal inflammation through induction of IL-17A and TNF-α in IBD
inferred← citeshttps://doi.org/10.1152/ajpgi.00326.2015openalex-snapshot
MicroRNA-155 deletion promotes tumorigenesis in the azoxymethane-dextran sulfate sodium model of colon cancer
inferred← citeshttps://doi.org/10.1155/2018/7565076openalex-snapshot
The Role of Autophagy and Related MicroRNAs in Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1155/2020/8811477openalex-snapshot
MiR-155-Mediated Deregulation of GPER1 Plays an Important Role in the Gender Differences Related to Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1166/mex.2022.2134openalex-snapshot
Liposomal nanoparticle-based miR-155 antagonist regulates Th17/Treg signaling pathway and its inhibitory effect against myocarditis
inferred← citeshttps://doi.org/10.1172/jci.insight.127551openalex-snapshot
Indole-3-carbinol prevents colitis and associated microbial dysbiosis in an IL-22–dependent manner
inferred← citeshttps://doi.org/10.1186/s12876-022-02533-1openalex-snapshot
MiRNA-374b-5p and miRNA-106a-5p are related to inflammatory bowel disease via regulating IL-10 and STAT3 signaling pathways
inferred← citeshttps://doi.org/10.1186/s13075-018-1550-yopenalex-snapshot
miR-155 is dispensable in monosodium urate-induced gouty inflammation in mice
inferred← citeshttps://doi.org/10.1186/s41544-019-0026-9openalex-snapshot
microRNAs as therapeutic targets in intestinal diseases
inferred← citeshttps://doi.org/10.14218/erhm.2015.00003openalex-snapshot
Intestinal Fibrosis in Inflammatory Bowel Disease: The Role of MicroRNAs
inferred← citeshttps://doi.org/10.14715/cmb/2023.69.7.29openalex-snapshot
LncRNA-CBR3-AS1 promotes and enhances the malignancy of ulcerative colitis via targeting miRNA-145-5p/FN1
inferred← citeshttps://doi.org/10.14748/vmf.v9i2.6752openalex-snapshot
The influence of miRNAs on the regulation of the immune system in inflammatory bowel disease
inferred← citeshttps://doi.org/10.18632/aging.202715openalex-snapshot
Mucosal microRNAs relate to age and severity of disease in ulcerative colitis
inferred← citeshttps://doi.org/10.18632/oncotarget.7324openalex-snapshot
Exosomes released by granulocytic myeloid-derived suppressor cells attenuate DSS-induced colitis in mice
inferred← citeshttps://doi.org/10.21037/atm-22-4859openalex-snapshot
MicroRNA-155-5p inhibition alleviates irritable bowel syndrome by increasing claudin-1 and ZO-1 expression
inferred← citeshttps://doi.org/10.2174/1871530320666200825184924openalex-snapshot
Role of MicroRNAs in the Progression and Metastasis of Colon Cancer
inferred← citeshttps://doi.org/10.31925/farmacia.2018.6.15openalex-snapshot
THE ADMINISTRATION OF TRICHURIS SUIS OVA EXTRACT DECREASES IL-17/IL-23 LEVELS IN EXPERIMENTAL INFLAMMATORY BOWEL DISEASE
inferred← citeshttps://doi.org/10.3389/fimmu.2018.00904openalex-snapshot
Critical Role of Alternative M2 Skewing in miR-155 Deletion-Mediated Protection of Colitis
inferred← citeshttps://doi.org/10.3389/fimmu.2021.669382openalex-snapshot
Implications the Role of miR-155 in the Pathogenesis of Autoimmune Diseases
inferred← citeshttps://doi.org/10.3389/fimmu.2021.716564openalex-snapshot
Differential Expression of microRNAs Correlates With the Severity of Experimental Autoimmune Cystitis
inferred← citeshttps://doi.org/10.3389/fmicb.2018.02910openalex-snapshot
Resveratrol-Mediated Attenuation of Staphylococcus aureus Enterotoxin B-Induced Acute Liver Injury Is Associated With Regulation of microRNA and Induction of Myeloid-Derived Suppressor Cells
inferred← citeshttps://doi.org/10.3389/fmicb.2024.1343891openalex-snapshot
Lacticaseibacillusparacasei BNCC345679 revolutionizes DSS-induced colitis and modulates gut microbiota
inferred← citeshttps://doi.org/10.3389/fonc.2020.00416openalex-snapshot
Inhibition of microRNA-155 Reduces Neuropathic Pain During Chemotherapeutic Bortezomib via Engagement of Neuroinflammation
inferred← citeshttps://doi.org/10.3389/fphar.2018.00468openalex-snapshot
The Administration of Escherichia coli Nissle 1917 Ameliorates Development of DSS-Induced Colitis in Mice
inferred← citeshttps://doi.org/10.3389/fphar.2022.813659openalex-snapshot
New Insights Into the Epigenetic Regulation of Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.3389/fphys.2018.00686openalex-snapshot
MiRNA-155 Regulates the Th17/Treg Ratio by Targeting SOCS1 in Severe Acute Pancreatitis
inferred← citeshttps://doi.org/10.3390/ani13121979openalex-snapshot
Expression Profile of miR-199a and Its Role in the Regulation of Intestinal Inflammation
inferred← citeshttps://doi.org/10.3390/biomedicines12020422openalex-snapshot
microRNA–mRNA Networks Linked to Inflammation and Immune System Regulation in Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.3390/cancers14153613openalex-snapshot
Apigenin Targets MicroRNA-155, Enhances SHIP-1 Expression, and Augments Anti-Tumor Responses in Pancreatic Cancer
inferred← citeshttps://doi.org/10.3390/cells10092204openalex-snapshot
Role of MicroRNA in Inflammatory Bowel Disease: Clinical Evidence and the Development of Preclinical Animal Models
inferred← citeshttps://doi.org/10.3390/cells10123358openalex-snapshot
The Impact of MicroRNAs during Inflammatory Bowel Disease: Effects on the Mucus Layer and Intercellular Junctions for Gut Permeability
inferred← citeshttps://doi.org/10.3390/cells7020012openalex-snapshot
MiRNAs at the Crossroads between Innate Immunity and Cancer: Focus on Macrophages
inferred← citeshttps://doi.org/10.3390/foods12183434openalex-snapshot
Sea Cucumber Peptide Alleviates Ulcerative Colitis Induced by Dextran Sulfate Sodium by Alleviating Gut Microbiota Imbalance and Regulating miR-155/SOCS1 Axis in Mice
inferred← citeshttps://doi.org/10.3390/ijms17050709openalex-snapshot
Expression and Function of miR-155 in Diseases of the Gastrointestinal Tract
inferred← citeshttps://doi.org/10.3390/ijms22010362openalex-snapshot
Vitamin D Modulates Intestinal Microbiota in Inflammatory Bowel Diseases
inferred← citeshttps://doi.org/10.3390/ijms22042213openalex-snapshot
Cell-to-Cell Communication by Host-Released Extracellular Vesicles in the Gut: Implications in Health and Disease
inferred← citeshttps://doi.org/10.3390/ijms22083967openalex-snapshot
The Role of microRNAs in Development of Colitis-Associated Colorectal Cancer
inferred← citeshttps://doi.org/10.3390/ijms222111382openalex-snapshot
A Combined mRNA- and miRNA-Sequencing Approach Reveals miRNAs as Potential Regulators of the Small Intestinal Transcriptome in Celiac Disease
inferred← citeshttps://doi.org/10.3390/ijms24032233openalex-snapshot
miRNA Molecules—Late Breaking Treatment for Inflammatory Bowel Diseases?
inferred← citeshttps://doi.org/10.3390/ijms27188359openalex-snapshot
Regulation of Inducible Nitric Oxide Synthase (NOS2) Expression in Healthy and Inflamed Bowel: A Narrative Review
inferred← citeshttps://doi.org/10.3390/immuno1040039openalex-snapshot
Role of microRNAs in the Pathophysiology of Ulcerative Colitis
inferred← citeshttps://doi.org/10.3390/nu16244411openalex-snapshot
Importance of Fecal Microbiota Transplantation and Molecular Regulation as Therapeutic Strategies in Inflammatory Bowel Diseases
inferred← citeshttps://doi.org/10.3748/wjg.v22.i35.7882openalex-snapshot
Biomarkers for colitis-associated colorectal cancer
inferred← citeshttps://doi.org/10.3748/wjg.v23.i6.976openalex-snapshot
MicroRNA-155 promotes the pathogenesis of experimental colitis by repressing SHIP-1 expression
inferred← citeshttps://doi.org/10.3748/wjg.v26.i32.4763openalex-snapshot
Immune and microRNA responses toHelicobacter muridaruminfection and indole-3-carbinol during colitis
inferred← citeshttps://doi.org/10.3892/mmr.2017.6755openalex-snapshot
Elevated expression of miR-155 is associated with the differentiation of CD8+ T cells in patients with HIV-1
inferred← citeshttps://doi.org/10.4049/jimmunol.1501727openalex-snapshot
Dietary Indoles Suppress Delayed-Type Hypersensitivity by Inducing a Switch from Proinflammatory Th17 Cells to Anti-Inflammatory Regulatory T Cells through Regulation of MicroRNA
inferred← citeshttps://doi.org/10.4161/21688362.2014.944446openalex-snapshot
MicroRNAs regulate tight junction proteins and modulate epithelial/endothelial barrier functions
inferred← citeshttps://doi.org/10.4172/0974-8369.1000231openalex-snapshot
Micro RNA as Biomarkers and Tool for Target-Based Treatment in Patients with Inflammatory Bowel Diseases
inferred← citeshttps://doi.org/10.4251/wjgo.v13.i9.995openalex-snapshot
MicroRNA expression in inflammatory bowel disease-associated colorectal cancer
inferred← citeshttps://doi.org/10.48087/bjmstf.2015.2111openalex-snapshot
Crohn’s disease: between the genetic determinism and the epigenetic hope
inferred← citeshttps://doi.org/10.5152/tjg.2019.18159openalex-snapshot
Up-regulation of miR-155 contributes to TNF-mediated hepatocyte apoptosis in acute liver failure
inferred← citeshttps://doi.org/10.5483/bmbrep.2016.49.5.041openalex-snapshot
MicroRNA-orchestrated pathophysiologic control in gut homeostasis and inflammation
inferred← citeshttps://doi.org/10.6084/m9.figshare.21629437openalex-snapshot
Additional file 1 of MiRNA-374b-5p and miRNA-106a-5p are related to inflammatory bowel disease via regulating IL-10 and STAT3 signaling pathways
inferred← citeshttps://doi.org/10.70675/4ac13bd9zbf56z4ff7za4b8zd2904548ff51openalex-snapshot
Les exosomes, médiateurs fondamentaux de la communication intercellulaire au cours de l'infection par les bactéries AIEC impliquées dans la maladie de Crohn
inferred← citeshttps://doi.org/10.70675/f2410d71zcb6az4796z8c99z98b53da92cd8openalex-snapshot
Etude du dysfonctionnement des macrophages de patients atteints de maladie de Crohn vis-à-vis de l'infection par les bactéries Escherichia coli adhérentes et invasives
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