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The role of mitochondrial dysfunction in periodontitis: From mechanisms to therapeutic strategy
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Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/advs.201800873openalex-snapshot
Contribution of Mitophagy to Cell‐Mediated Mineralization: Revisiting a 50‐Year‐Old Conundrum
inferredcites →https://doi.org/10.1007/s00005-009-0026-9openalex-snapshot
Increased salivary level of 8-hydroxydeoxyguanosine is a marker of premature oxidative mitochondrial DNA damage in gingival tissue of patients with periodontitis
inferredcites →https://doi.org/10.1007/s00774-020-01165-3openalex-snapshot
RANKL and osteoimmunology in periodontitis
inferredcites →https://doi.org/10.1007/s10541-005-0161-9openalex-snapshot
Reactive Oxygen Species and Human Inflammatory Periodontal Diseases
inferredcites →https://doi.org/10.1007/s11906-022-01184-7openalex-snapshot
Mitochondrial Dysfunction in the Pathogenesis of Preeclampsia
inferredcites →https://doi.org/10.1016/j.actbio.2015.10.031openalex-snapshot
Resveratrol prevents alveolar bone loss in an experimental rat model of periodontitis
inferredcites →https://doi.org/10.1016/j.archoralbio.2011.03.020openalex-snapshot
Expression of HMGB1 and HMGN2 in gingival tissues, GCF and PICF of periodontitis patients and peri-implantitis
inferredcites →https://doi.org/10.1016/j.archoralbio.2017.01.019openalex-snapshot
Baicalein enhances the osteogenic differentiation of human periodontal ligament cells by activating the Wnt/β-catenin signaling pathway
inferredcites →https://doi.org/10.1016/j.archoralbio.2021.105173openalex-snapshot
Gene expression profiles of mitochondria-endoplasmic reticulum tethering in human gingival fibroblasts in response to periodontal pathogens
inferredcites →https://doi.org/10.1016/j.bbadis.2016.11.010openalex-snapshot
Mitochondrial dysfunction and oxidative stress in metabolic disorders — A step towards mitochondria based therapeutic strategies
inferredcites →https://doi.org/10.1016/j.bbrc.2015.11.022openalex-snapshot
Blockade of Drp1 rescues oxidative stress-induced osteoblast dysfunction
inferredcites →https://doi.org/10.1016/j.bbrc.2018.12.143openalex-snapshot
Mitochondrial abnormalities are involved in periodontal ligament fibroblast apoptosis induced by oxidative stress
inferredcites →https://doi.org/10.1016/j.cell.2015.12.057openalex-snapshot
NF-κB Restricts Inflammasome Activation via Elimination of Damaged Mitochondria
inferredcites →https://doi.org/10.1016/j.cmet.2019.08.003openalex-snapshot
Mitochondrial Damage and Activation of the STING Pathway Lead to Renal Inflammation and Fibrosis
inferredcites →https://doi.org/10.1016/j.cmet.2023.01.003openalex-snapshot
Mitochondrial fragmentation and donut formation enhance mitochondrial secretion to promote osteogenesis
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2014.07.034openalex-snapshot
Resveratrol improves oxidative stress and prevents the progression of periodontitis via the activation of the Sirt1/AMPK and the Nrf2/antioxidant defense pathways in a rat periodontitis model
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2021.05.031openalex-snapshot
Crosstalk between reactive oxygen species and Dynamin-related protein 1 in periodontitis
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2021.09.027openalex-snapshot
Hydroxytyrosol prevents periodontitis-induced bone loss by regulating mitochondrial function and mitogen-activated protein kinase signaling of bone cells
inferredcites →https://doi.org/10.1016/j.ijcard.2018.05.019openalex-snapshot
Mitochondrial oxidative stress, endothelial function and metabolic control in patients with type II diabetes and periodontitis: A randomised controlled clinical trial
inferredcites →https://doi.org/10.1016/j.it.2019.10.008openalex-snapshot
Emerging Role of Mitochondrial DNA as a Major Driver of Inflammation and Disease Progression
inferredcites →https://doi.org/10.1016/j.lfs.2019.05.027openalex-snapshot
Mitophagy is a protective response against oxidative damage in bone marrow mesenchymal stem cells
inferredcites →https://doi.org/10.1016/j.mito.2011.01.009openalex-snapshot
Mitochondrial dysfunction and genetic heterogeneity in chronic periodontitis
inferredcites →https://doi.org/10.1016/j.phrs.2014.10.007openalex-snapshot
Lipophilic antioxidants prevent lipopolysaccharide-induced mitochondrial dysfunction through mitochondrial biogenesis improvement
inferredcites →https://doi.org/10.1016/j.redox.2015.09.005openalex-snapshot
Reactive oxygen species and mitochondria: A nexus of cellular homeostasis
inferredcites →https://doi.org/10.1016/j.redox.2022.102337openalex-snapshot
Cellular redox imbalance on the crossroad between mitochondrial dysfunction, senescence, and proliferation
inferredcites →https://doi.org/10.1016/j.stem.2021.02.011openalex-snapshot
Mitochondria as Signaling Organelles Control Mammalian Stem Cell Fate
inferredcites →https://doi.org/10.1016/j.tox.2017.07.019openalex-snapshot
Mitochondrial fusion, fission, and mitochondrial toxicity
inferredcites →https://doi.org/10.1016/j.trsl.2018.07.014openalex-snapshot
Mitochondria in innate immune signaling
inferredcites →https://doi.org/10.1016/j.yexcr.2016.08.007openalex-snapshot
Mitochondrial reactive oxygen species mediate the lipopolysaccharide-induced pro-inflammatory response in human gingival fibroblasts
inferredcites →https://doi.org/10.1038/nature01658openalex-snapshot
Osteoclast differentiation and activation
inferredcites →https://doi.org/10.1038/nrendo.2011.106openalex-snapshot
Diabetes mellitus and periodontitis: a tale of two common interrelated diseases
inferredcites →https://doi.org/10.1038/s41569-018-0074-0openalex-snapshot
Targeting mitochondria for cardiovascular disorders: therapeutic potential and obstacles
inferredcites →https://doi.org/10.1038/s41576-020-00284-xopenalex-snapshot
Extreme heterogeneity of human mitochondrial DNA from organelles to populations
inferredcites →https://doi.org/10.1038/s41577-020-00488-6openalex-snapshot
Local and systemic mechanisms linking periodontal disease and inflammatory comorbidities
inferredcites →https://doi.org/10.1038/s41586-018-0372-zopenalex-snapshot
New mitochondrial DNA synthesis enables NLRP3 inflammasome activation
inferredcites →https://doi.org/10.1038/sj.cdd.4400779openalex-snapshot
Mitochondria-dependent apoptosis and cellular pH regulation
inferredcites →https://doi.org/10.1042/bse0470069openalex-snapshot
Regulation of mitochondrial biogenesis
inferredcites →https://doi.org/10.1055/s-0042-118458openalex-snapshot
Bone Marrow Stromal Stem Cells in Tissue Engineering and Regenerative Medicine
inferredcites →https://doi.org/10.1080/15548627.2020.1725377openalex-snapshot
Organelle-specific autophagy in inflammatory diseases: a potential therapeutic target underlying the quality control of multiple organelles
inferredcites →https://doi.org/10.1089/ten.tea.2020.0141openalex-snapshot
Bone Marrow Mesenchymal Stem Cell-Derived Small Extracellular Vesicles Promote Periodontal Regeneration
inferredcites →https://doi.org/10.1096/fj.202100067ropenalex-snapshot
Mitochondrial fusion and fission: The fine‐tune balance for cellular homeostasis
inferredcites →https://doi.org/10.1111/cpr.12973openalex-snapshot
NLRP3 regulates alveolar bone loss in ligature‐induced periodontitis by promoting osteoclastic differentiation
inferredcites →https://doi.org/10.1111/iej.13055openalex-snapshot
Simvastatin alleviates bone resorption in apical periodontitis possibly by inhibition of mitophagy‐related osteoblast apoptosis
inferredcites →https://doi.org/10.1111/imcb.12363openalex-snapshot
Lipopolysaccharide promotes Drp1‐dependent mitochondrial fission and associated inflammatory responses in macrophages
inferredcites →https://doi.org/10.1111/j.1600-0757.2012.00455.xopenalex-snapshot
Obesity, diabetes mellitus, atherosclerosis and chronic periodontitis: a shared pathology via oxidative stress and mitochondrial dysfunction?
inferredcites →https://doi.org/10.1111/j.1749-6632.2010.05848.xopenalex-snapshot
Anti‐inflammatory effects of resveratrol: possible role in prevention of age‐related cardiovascular disease
inferredcites →https://doi.org/10.1111/jcpe.12711openalex-snapshot
Mitochondrial dysfunction is involved in the aggravation of periodontitis by diabetes
inferredcites →https://doi.org/10.1111/jcpe.13056openalex-snapshot
Osteoimmunology: Inflammatory osteolysis and regeneration of the alveolar bone
inferredcites →https://doi.org/10.1111/jcpe.13155openalex-snapshot
Association between periodontitis and cognitive impairment: Analysis of national health and nutrition examination survey (NHANES) III
inferredcites →https://doi.org/10.1111/jcpe.13811openalex-snapshot
Activation of PGC ‐1α‐dependent mitochondrial biogenesis supports therapeutic effects of silibinin against type I diabetic periodontitis
inferredcites →https://doi.org/10.1111/jdi.13477openalex-snapshot
Epigenetic changes caused by diabetes and their potential role in the development of periodontitis
inferredcites →https://doi.org/10.1111/jre.12733openalex-snapshot
Inflammasomes and their regulation in periodontal disease: A review
inferredcites →https://doi.org/10.1111/jre.12930openalex-snapshot
Sirtuin 3 deficiency exacerbates age‐related periodontal disease
inferredcites →https://doi.org/10.1111/jre.13042openalex-snapshot
PGC ‐1α attenuates TNF ‐α‐induced inflammatory responses in OCCM ‐30 cells
inferredcites →https://doi.org/10.1111/nyas.14872openalex-snapshot
PGC‐1 alpha regulates mitochondrial biogenesis to ameliorate hypoxia‐inhibited cementoblast mineralization
inferredcites →https://doi.org/10.1111/odi.12468openalex-snapshot
The role of Toll‐like receptors in periodontitis
inferredcites →https://doi.org/10.1111/odi.12574openalex-snapshot
Programmed cell death in periodontitis: recent advances and future perspectives
inferredcites →https://doi.org/10.1111/odi.13231openalex-snapshot
Correlation study on mtDNA polymorphisms as potential risk factors in aggressive periodontitis by NGS
inferredcites →https://doi.org/10.1111/odi.14286openalex-snapshot
PINK1 ‐mediated mitophagy reduced inflammatory responses to Porphyromonas gingivalis in macrophages
inferredcites →https://doi.org/10.1111/prd.12233openalex-snapshot
Effect of micronutrient malnutrition on periodontal disease and periodontal therapy
inferredcites →https://doi.org/10.1111/prd.12342openalex-snapshot
Periodontitis is an inflammatory disease of oxidative stress: We should treat it that way
inferredcites →https://doi.org/10.1126/sciadv.aax0672openalex-snapshot
Osteoblastic lysosome plays a central role in mineralization
inferredcites →https://doi.org/10.1128/iai.00111-18openalex-snapshot
Anti-HMGB1 Neutralizing Antibody Attenuates Periodontal Inflammation and Bone Resorption in a Murine Periodontitis Model
inferredcites →https://doi.org/10.1146/annurev-pathmechdis-012419-032711openalex-snapshot
Mitochondrial Dynamics and Its Involvement in Disease
inferredcites →https://doi.org/10.1146/annurev-pharmtox-010716-104908openalex-snapshot
Repairing Mitochondrial Dysfunction in Disease
inferredcites →https://doi.org/10.1152/ajpendo.00292.2010openalex-snapshot
The importance of PGC-1α in contractile activity-induced mitochondrial adaptations
inferredcites →https://doi.org/10.1152/physrev.00026.2013openalex-snapshot
Mitochondrial Reactive Oxygen Species (ROS) and ROS-Induced ROS Release
inferredcites →https://doi.org/10.1155/2015/615486openalex-snapshot
Mechanisms of Bone Resorption in Periodontitis
inferredcites →https://doi.org/10.1155/2018/7514383openalex-snapshot
The Causal Role of Mitochondrial Dynamics in Regulating Insulin Resistance in Diabetes: Link through Mitochondrial Reactive Oxygen Species
inferredcites →https://doi.org/10.1155/2022/1000213openalex-snapshot
Mitochondrial DNA Efflux Maintained in Gingival Fibroblasts of Patients with Periodontitis through ROS/mPTP Pathway
inferredcites →https://doi.org/10.1161/circulationaha.115.020869openalex-snapshot
Increasing Evidence for an Association Between Periodontitis and Cardiovascular Disease
inferredcites →https://doi.org/10.1161/hypertensionaha.120.14686openalex-snapshot
Mitochondrial Fission Mediates Endothelial Inflammation
inferredcites →https://doi.org/10.1177/0022034521995157openalex-snapshot
Host Modulation and Treatment of Periodontal Disease
inferredcites →https://doi.org/10.1177/00220345221103145openalex-snapshot
Matrix Vesicle–Mediated Mineralization and Potential Applications
inferredcites →https://doi.org/10.1186/1741-7015-10-122openalex-snapshot
Autophagy in periodontitis patients and gingival fibroblasts: unraveling the link between chronic diseases and inflammation
inferredcites →https://doi.org/10.1186/s12903-022-02167-5openalex-snapshot
PINK1 mediated mitophagy attenuates early apoptosis of gingival epithelial cells induced by high glucose
inferredcites →https://doi.org/10.1186/s13046-022-02262-zopenalex-snapshot
Increased Drp1 promotes autophagy and ESCC progression by mtDNA stress mediated cGAS-STING pathway
inferredcites →https://doi.org/10.1186/s13567-021-01014-9openalex-snapshot
Prototheca spp. induce an inflammatory response via mtROS-mediated activation of NF-κB and NLRP3 inflammasome pathways in bovine mammary epithelial cell cultures
inferredcites →https://doi.org/10.1371/journal.pone.0173270openalex-snapshot
Blockade of cyclophilin D rescues dexamethasone-induced oxidative stress in gingival tissue
inferredcites →https://doi.org/10.1515/hsz-2020-0231openalex-snapshot
Kill one or kill the many: interplay between mitophagy and apoptosis
inferredcites →https://doi.org/10.15252/embj.2020104705openalex-snapshot
Molecular mechanisms and physiological functions of mitophagy
inferredcites →https://doi.org/10.1902/jop.2006.060108openalex-snapshot
New Evidence of Premature Oxidative DNA Damage: Mitochondrial DNA Deletion in Gingival Tissue of Patients With Periodontitis
inferredcites →https://doi.org/10.1902/jop.2013.120746openalex-snapshot
Resveratrol Decreases Periodontal Breakdown and Modulates Local Levels of Cytokines During Periodontitis in Rats
inferredcites →https://doi.org/10.21203/rs.3.rs-954052/v1openalex-snapshot
PINK1 Mediated Mitophagy Attenuates Early Apoptosis of Gingival Epithelial Cells Induced by High Glucose
inferredcites →https://doi.org/10.2147/ijn.s232986openalex-snapshot
Silver Nanoparticle Exposure Causes Pulmonary Structural Damage and Mitochondrial Dynamic Imbalance in the Rat: Protective Effects of Sodium Selenite
inferredcites →https://doi.org/10.3389/fcell.2021.687258openalex-snapshot
Exosomes Regulate Interclonal Communication on Osteogenic Differentiation Among Heterogeneous Osteogenic Single-Cell Clones Through PINK1/Parkin-Mediated Mitophagy
inferredcites →https://doi.org/10.3389/fcimb.2021.766944openalex-snapshot
Periodontal Disease: The Good, The Bad, and The Unknown
inferredcites →https://doi.org/10.3389/fimmu.2021.624919openalex-snapshot
Unraveling the Link Between Mitochondrial Dynamics and Neuroinflammation
inferredcites →https://doi.org/10.3389/fphys.2017.00351openalex-snapshot
Pathways that Regulate ROS Scavenging Enzymes, and Their Role in Defense Against Tissue Destruction in Periodontitis
inferredcites →https://doi.org/10.3389/fphys.2017.00439openalex-snapshot
The Role of Reactive Oxygen Species and Autophagy in Periodontitis and Their Potential Linkage
inferredcites →https://doi.org/10.3390/antiox9121211openalex-snapshot
The Promising Role of Antioxidant Phytochemicals in the Prevention and Treatment of Periodontal Disease via the Inhibition of Oxidative Stress Pathways: Updated Insights
inferredcites →https://doi.org/10.3390/ijms222111338openalex-snapshot
Mitochondria as a Cellular Hub in Infection and Inflammation
inferredcites →https://doi.org/10.3390/jpm11010058openalex-snapshot
Effect of Aging on Homeostasis in the Soft Tissue of the Periodontium: A Narrative Review
inferredcites →https://doi.org/10.3390/molecules24183217openalex-snapshot
Moringin Pretreatment Inhibits the Expression of Genes Involved in Mitophagy in the Stem Cell of the Human Periodontal Ligament
inferredcites →https://doi.org/10.3760/cma.j.issn.1002-0098.2018.07.005openalex-snapshot
[Endoplasmic reticulum-mitochondrial contact regulates osteogenic differentiation of periodontal ligament stem cells via mitofusion 2 in inflammatory microenvironment].
inferredcites →https://doi.org/10.4049/jimmunol.2100846openalex-snapshot
Correction: Kavain Reduces Porphyromonas gingivalis–Induced Adipocyte Inflammation: Role of PGC-1α Signaling
inferredcites →https://doi.org/10.7150/ijbs.46627openalex-snapshot
The Role of Autophagy and Mitophagy in Bone Metabolic Disorders
inferredcites →https://doi.org/10.7717/peerj.8663openalex-snapshot
Suppression of osteogenic differentiation and mitochondrial function change in human periodontal ligament stem cells by melatonin at physiological levels
inferredcites →https://doi.org/10.7860/jcdr/2015/13486.6291openalex-snapshot
Effectiveness of CoQ10 Oral Supplements as an Adjunct to Scaling and Root Planing in Improving Periodontal Health
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/adfm.202416336openalex-snapshot
Neutrophil Airfreighter Efficiently Delivers siRNA‐Loaded Nanocomplex to Mononuclear Phagocytes for Inhibition of mtDNA‐Induced Inflammation
inferred← citeshttps://doi.org/10.1002/adhm.202400533openalex-snapshot
Injectable Thermosensitive Gel CH‐BPNs‐NBP for Effective Periodontitis Treatment through ROS‐Scavenging and Jaw Vascular Unit Protection
inferred← citeshttps://doi.org/10.1002/adhm.71377openalex-snapshot
Pathology‐Targeted Nanoparticles Guided by Peptide Remodel the Periodontal Microenvironment for Periodontitis Therapy
inferred← citeshttps://doi.org/10.1002/adtp.202400078openalex-snapshot
Targeting Mitophagy as a Potential Therapeutic Approach for Age‐Related Bone Diseases
inferred← citeshttps://doi.org/10.1002/pat.70209openalex-snapshot
Injectable Thermosensitive Gel Loaded Salvianolic Acid B Alleviates Periodontitis by Down‐Regulating Oxidative Stress and Protecting Mitochondrial Function
inferred← citeshttps://doi.org/10.1002/smm2.70031openalex-snapshot
Formulation Redesigning of Itaconate Treats Periodontitis via Nrf2/TFAM‐Mediated Reprogramming of Mitochondrial Metabolism
inferred← citeshttps://doi.org/10.1007/s10266-026-01487-0openalex-snapshot
Mitochondrial dysfunction in peri-implantitis: bioinformatics and machine learning analysis with in vivo experiment
inferred← citeshttps://doi.org/10.1007/s10753-025-02390-6openalex-snapshot
Impaired Mitophagy-Induced Apoptosis of Gingival Fibroblasts Exacerbates Diabetic Periodontitis Via THBS-1/CD36-Dependent Macrophage Activation
inferred← citeshttps://doi.org/10.1007/s40496-024-00390-3openalex-snapshot
Epicatechin: Potential Use as Anti-Obese and Anti-Periodontal Nutrient
inferred← citeshttps://doi.org/10.1016/j.biochi.2025.07.014openalex-snapshot
The role of mitochondrial DNA in bone/cartilage diseases
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2025.123588openalex-snapshot
Core-shell structured CeO2@ZIF-8 nanohybrids regulating the Ce(III)/Ce(IV) valence conversion to enhance ROS-scavenging capacity for periodontitis treatment
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2025.123748openalex-snapshot
Advanced biomaterials regulating mitochondria for treatment of peri-implantitis
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2026.124307openalex-snapshot
A ZIF90-tungsten-based nanoparticle hydrogel system with antibacterial and antioxidant properties promotes periodontal bone regeneration by modulating the ROS/Plagl1/Wnt pathway
inferred← citeshttps://doi.org/10.1016/j.cpan.2026.09.013openalex-snapshot
Metabolic Reprogramming in Periodontitis: Evidence Stratification, Pharmacological Modulators, and Translational Challenges
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2026.01.010openalex-snapshot
Non-surgical periodontal treatment improves mitochondrial bioenergetics in circulating immune cells of patients with chronic periodontitis
inferred← citeshttps://doi.org/10.1016/j.gene.2024.148476openalex-snapshot
Regulatory role of PDK1 via integrated gene analysis of mitochondria-immune response in periodontitis
inferred← citeshttps://doi.org/10.1016/j.identj.2026.109682openalex-snapshot
Mitochondrial Metabolic Biomarkers in Periodontitis: Discovery and Clinical Validation
inferred← citeshttps://doi.org/10.1016/j.identj.2026.111194openalex-snapshot
Mitochondrial Dysfunction in Periodontitis: Oxidative Stress, Metabolism and Alveolar Bone Loss
inferred← citeshttps://doi.org/10.1016/j.identj.2026.111244openalex-snapshot
Mitochondrial Dysfunction in Oral Diseases: Shared Pathogenic Mechanisms and Therapeutic Opportunities
inferred← citeshttps://doi.org/10.1016/j.jconrel.2026.114776openalex-snapshot
An osteoimmunomodulatory microneedle patch targeting ER-mitochondria calcium crosstalk for periodontal tissue regeneration
inferred← citeshttps://doi.org/10.1016/j.micpath.2024.106570openalex-snapshot
A high concentrate diet inhibits forkhead box protein A2 expression, and induces oxidative stress, mitochondrial dysfunction and mitochondrial unfolded protein response in the liver of dairy cows
inferred← citeshttps://doi.org/10.1016/j.mito.2024.101942openalex-snapshot
The Interplay of Mitochondrial Dysfunction in Oral Diseases: Recent Updates in Pathogenesis and Therapeutic Implications
inferred← citeshttps://doi.org/10.1021/acsomega.5c13610openalex-snapshot
Research on the Improvement of Oxidative Stress Regulation in Periodontal Ligament Stem Cells and the Promotion of Periodontal Bone Tissue Regeneration by GDF5/ROS-Responsive Degradable Hydrogel
inferred← citeshttps://doi.org/10.1038/s41368-025-00360-0openalex-snapshot
Dimethyl fumarate modulates M1/M2 macrophage polarization to ameliorate periodontal destruction by increasing TUFM-mediated mitophagy
inferred← citeshttps://doi.org/10.1038/s41598-026-55765-6openalex-snapshot
Bioinformatics-based identification of mitophagy related biomarkers in periodontitis
inferred← citeshttps://doi.org/10.1039/d4mh01137fopenalex-snapshot
Two birds with one stone: natural polyphenols boosted periodontitis treatment of chlorhexidine via reducing toxicity and regulating microenvironments
inferred← citeshttps://doi.org/10.1080/13510002.2025.2475691openalex-snapshot
Hyperhomocysteinaemia aggravates periodontitis by suppressing the Nrf2/HO-1 signalling pathway
inferred← citeshttps://doi.org/10.1080/21505594.2025.2573660openalex-snapshot
Ferroptosis and macrophage efferocytosis in periodontitis: Biological insights and therapeutic advances
inferred← citeshttps://doi.org/10.1111/cpr.13781openalex-snapshot
Ubiquitin‐Proteasome System in Periodontitis: Mechanisms and Clinical Implications
inferred← citeshttps://doi.org/10.1111/febs.17352openalex-snapshot
Attenuated NIX in impaired mitophagy contributes to exacerbating cellular senescence in experimental periodontitis under hyperglycemic conditions
inferred← citeshttps://doi.org/10.1111/jcpe.14184openalex-snapshot
Exosomes Derived From Human Gingival Mesenchymal Stem Cells Induce Metabolic Reprogramming of Inflammatory Macrophages
inferred← citeshttps://doi.org/10.1111/jre.13333openalex-snapshot
Insulin resistance and periodontitis: Mediation by blood pressure
inferred← citeshttps://doi.org/10.1111/jre.70016openalex-snapshot
The Role of Oxidative Stress in Periodontitis
inferred← citeshttps://doi.org/10.1111/jre.70099openalex-snapshot
Periodontal Medicine Rewired: Mechanisms Linking Periodontitis to Systemic Diseases
inferred← citeshttps://doi.org/10.1111/odi.70019openalex-snapshot
LIPUS Protects PDLSCs From Oxidative Damage and Promotes Alveolar Bone Regeneration via FOXO1
inferred← citeshttps://doi.org/10.1177/00220345251315723openalex-snapshot
Role of Pink1 in Regulating Osteoclast Differentiation during Periodontitis
inferred← citeshttps://doi.org/10.1177/00220345261416506openalex-snapshot
Dual-Network Silk Fibroin/Quercetin Hydrogel for Periodontitis Treatment
inferred← citeshttps://doi.org/10.1177/00220345261450582openalex-snapshot
Restoring Cellular Homeostasis as a Therapeutic Strategy for Periodontitis
inferred← citeshttps://doi.org/10.1186/s12302-024-00949-yopenalex-snapshot
Association between metal exposures and periodontitis among U.S. adults: the potential mediating role of biological aging
inferred← citeshttps://doi.org/10.1186/s12951-025-03907-9openalex-snapshot
Fisetin carbon dots alleviate periodontitis by enhancing mitophagy through regulation of sirtuin 3 SUMOylation
inferred← citeshttps://doi.org/10.1186/s12951-026-04660-3openalex-snapshot
Size-dependent ruthenium/ceria nanozymes synchronize catalytic ROS scavenging and electrostatic mtDNA sequestration for periodontitis therapy
inferred← citeshttps://doi.org/10.1186/s13287-025-04860-yopenalex-snapshot
Exosomes from LPS-pretreated BMSCs treated periodontitis via improving oxidative stress
inferred← citeshttps://doi.org/10.1186/s13619-026-00283-zopenalex-snapshot
FRZB regulates the osteogenic differentiation of periodontal ligament stem cells in an inflammatory microenvironment through Wnt5a-mitochondrial axis
inferred← citeshttps://doi.org/10.1186/s40001-026-04394-wopenalex-snapshot
A novel mitochondrial DNA deletion in molar–incisor periodontitis
inferred← citeshttps://doi.org/10.1186/s40001-026-04846-3openalex-snapshot
Ageing-driven periodontitis: molecular mechanisms and emerging anti-ageing interventions
inferred← citeshttps://doi.org/10.12677/acm.2026.1641680openalex-snapshot
Mitochondrial Basis of Resolution-Regeneration Uncoupling in Periodontitis and Its Targeted Therapy
inferred← citeshttps://doi.org/10.1631/jzus.b2300928openalex-snapshot
Mitochondria derived from human embryonic stem cell-derived mesenchymal stem cells alleviate the inflammatory response in human gingival fibroblasts
inferred← citeshttps://doi.org/10.17816/dent632944openalex-snapshot
Mitochondrial dysfunction in the pathogenesis and treatment of oral inflammatory diseases
inferred← citeshttps://doi.org/10.21203/rs.3.rs-3534702/v1openalex-snapshot
Integrated analyses revealed the potential role and immune link of mitochondrial dysfunction between periodontitis and type 2 diabetes mellitus.
inferred← citeshttps://doi.org/10.2147/ccide.s560013openalex-snapshot
Integrative MultiOmics and Machine Learning Reveal Peroxiredoxin 4 as a Critical Hub Governing Mitochondrial Dysfunction and B Cell Differentiation in Periodontitis
inferred← citeshttps://doi.org/10.2147/ijn.s470419openalex-snapshot
FoxO1-Overexpressed Small Extracellular Vesicles Derived from hPDLSCs Promote Periodontal Tissue Regeneration by Reducing Mitochondrial Dysfunction to Regulate Osteogenesis and Inflammation
inferred← citeshttps://doi.org/10.2147/jir.s498739openalex-snapshot
Single-Cell and Transcriptome Analysis of Periodontitis: Molecular Subtypes and Biomarkers Linked to Mitochondrial Dysfunction and Immunity
inferred← citeshttps://doi.org/10.3389/fcimb.2025.1634909openalex-snapshot
Mechanisms and therapeutic perspectives of mitochondrial dysfunction of macrophages in periodontitis
inferred← citeshttps://doi.org/10.3389/fimmu.2025.1526961openalex-snapshot
Ferroptosis and cuproptosis in periodontitis: recent biological insights and therapeutic advances
inferred← citeshttps://doi.org/10.3389/fphar.2025.1446574openalex-snapshot
Genipin attenuates oxidative damage in periodontal tissues by alleviating mitochondrial dysfunction and abnormal glucose uptake through inhibition of UCP2
inferred← citeshttps://doi.org/10.3389/froh.2025.1540178openalex-snapshot
The role of Mitofusin-1 and Mitofusin-2 in periodontal disease: a comprehensive review
inferred← citeshttps://doi.org/10.3389/froh.2026.1791758openalex-snapshot
Periodontitis and cardiovascular diseases: bridging the gap through mitochondrial dysfunction
inferred← citeshttps://doi.org/10.3390/antiox13121450openalex-snapshot
Modulatory Effects of Photobiomodulation on Oxidative and Inflammatory Responses in a Murine Model of Periodontitis
inferred← citeshttps://doi.org/10.3390/biom16091369openalex-snapshot
Integrative Transcriptomics Reveals a Mitochondria-Related Gene Signature in Periodontitis
inferred← citeshttps://doi.org/10.3390/diseases12010012openalex-snapshot
Impact of Non-Surgical Periodontal Treatment on the Concentration and Level of MRP-8/14 (Calprotectin) as an Inflammatory Biomarker in Women with Periodontitis and Rheumatoid Arthritis: A Quasi-Experimental Study
inferred← citeshttps://doi.org/10.3390/ijms242015483openalex-snapshot
Mitochondrial Dysfunction in the Pathogenesis and Treatment of Oral Inflammatory Diseases
inferred← citeshttps://doi.org/10.3390/ijms25021024openalex-snapshot
Mitochondrial Dysfunction in Periodontitis and Associated Systemic Diseases: Implications for Pathomechanisms and Therapeutic Strategies
inferred← citeshttps://doi.org/10.3390/ijms25052763openalex-snapshot
Matrix Metalloproteinases in the Periodontium—Vital in Tissue Turnover and Unfortunate in Periodontitis
inferred← citeshttps://doi.org/10.3390/ijms25147955openalex-snapshot
Causal Relationship between Mitochondrial Biological Function and Periodontitis: Evidence from a Mendelian Randomization Study
inferred← citeshttps://doi.org/10.3390/jcm15145402openalex-snapshot
Cyclic Altitude Training, Mitochondrial Health, and the Oral–Airway Axis: Intermittent Hypoxia Between Adaptation and Disease
inferred← citeshttps://doi.org/10.59717/j.xinn-med.2026.100255openalex-snapshot
Mitochondrial quality control as a unifying regulatory platform in inflammatory bone diseases
inferred← citeshttps://doi.org/10.6084/m9.figshare.26738589openalex-snapshot
Additional file 1 of Association between metal exposures and periodontitis among U.S. adults: the potential mediating role of biological aging
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
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