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Mechanistic insight into female predominance in Alzheimer’s disease based on aberrant protein S-nitrosylation of C3
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1 of the 95 references listed on this page is in the Retraction Watch database as retracted (export 2026-11-04): 10.1038/mp.2016.139 (notice 2020-08-11).
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/ana.410300410openalex-snapshotPhysical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment
inferredcites →https://doi.org/10.1002/cpt.819openalex-snapshotCritical Issues and Optimized Practices in Quantification of Protein Abundance Level to Determine Interindividual Variability in DMET Proteins by LC‐MS/MS Proteomics
inferredcites →https://doi.org/10.1007/s00018-018-2752-9openalex-snapshotAnalysis of novel endosome-to-Golgi retrieval genes reveals a role for PLD3 in regulating endosomal protein sorting and amyloid precursor protein processing
inferredcites →https://doi.org/10.1016/j.jalz.2011.03.005openalex-snapshotThe diagnosis of dementia due to Alzheimer's disease: Recommendations from the National Institute on Aging‐Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease
inferredcites →https://doi.org/10.1038/336385a0openalex-snapshotEndothelium-derived relaxing factor release on activation of NMDA receptors suggests role as intercellular messenger in the brain
inferredcites →https://doi.org/10.1038/ncomms10242openalex-snapshotElevated glucose and oligomeric β-amyloid disrupt synapses via a common pathway of aberrant protein S-nitrosylation
inferredcites →https://doi.org/10.1038/nrm3143openalex-snapshotUbiquitylation in apoptosis: a post-translational modification at the edge of life and death
inferredcites →https://doi.org/10.1038/s41380-018-0113-6openalex-snapshotShank3 mutation in a mouse model of autism leads to changes in the S-nitroso-proteome and affects key proteins involved in vesicle release and synaptic function
inferredcites →https://doi.org/10.1038/s41467-019-10182-4openalex-snapshotProteome-wide detection of S-nitrosylation targets and motifs using bioorthogonal cleavable-linker-based enrichment and switch technique
inferredcites →https://doi.org/10.1038/s41591-020-0815-6openalex-snapshotLarge-scale proteomic analysis of Alzheimer’s disease brain and cerebrospinal fluid reveals early changes in energy metabolism associated with microglia and astrocyte activation
inferredcites →https://doi.org/10.1111/jnc.14635openalex-snapshotMass spectrometry: A platform for biomarker discovery and validation for Alzheimer's and Parkinson's diseases
inferredcites →https://doi.org/10.1523/jneurosci.0829-08.2008openalex-snapshotComplement C3 Deficiency Leads to Accelerated Amyloid Plaque Deposition and Neurodegeneration and Modulation of the Microglia/Macrophage Phenotype in Amyloid Precursor Protein Transgenic Mice
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/advs.202306469openalex-snapshotMetabolic Bypass Rescues Aberrant S‐nitrosylation‐Induced TCA Cycle Inhibition and Synapse Loss in Alzheimer's Disease Human Neurons
inferred← citeshttps://doi.org/10.1002/alz.70444openalex-snapshotAlzheimer's disease and dementia in Japan: Epidemiological trends, regional disparities, and future projections
inferred← citeshttps://doi.org/10.1002/alz.71185openalex-snapshotSpatial heterogeneity in microglia‐complement crosstalk: Implications for synaptic pruning in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/cam4.72067openalex-snapshotTargeting Temozolomide‐Resistant Glioblastoma: Therapeutic Potential of Neuronal Nitric Oxide Synthase Inhibitor
inferred← citeshttps://doi.org/10.1002/jsfa.13394openalex-snapshotOligosaccharides from black ginseng innovatively prepared by low‐temperature steam‐heating process ameliorate cognitive impairment in Alzheimer's disease mice via the Keap‐1/Nrf2 pathway
inferred← citeshttps://doi.org/10.1007/s00702-025-02952-wopenalex-snapshotSpecies-specific sensitivity to intracerebroventricular streptozotocin in rats and mice highlights pathways and proteins relevant to Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.mtbio.2025.101714openalex-snapshotAstragalin-functionalized ultrasmall nanoparticles modulate the complement pathway to inhibit microglial synaptic phagocytosis for reducing anesthetic neurotoxicity
inferred← citeshttps://doi.org/10.1016/j.neurot.2025.e00708openalex-snapshotAberrant S-nitrosylation in the TCA cycle contributes to mitochondrial dysfunction, energy compromise, and synapse loss in neurodegenerative diseases
inferred← citeshttps://doi.org/10.1089/ars.2023.0395openalex-snapshotIncreased Neuronal Nitric Oxide Synthase in Alzheimer's Disease Mediates Spontaneous Calcium Signaling and Divergent Glutamatergic Calcium Responses
inferred← citeshttps://doi.org/10.1111/jnc.70365openalex-snapshotAberrant Protein S‐Nitrosylation Mimics the Effect of Rare Genetic Mutations in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.1159/000549913openalex-snapshotA Global, Regional, and National Survey on Burden and Quality of Care Index of Alzheimer’s Disease and Other Dementias in Adults Aged 55 and Above: The Global Burden of Disease Study 1990–2021
inferred← citeshttps://doi.org/10.1172/jci167501openalex-snapshotComplement C3aR depletion reverses HIF-1α–induced metabolic impairment and enhances microglial response to Aβ pathology
inferred← citeshttps://doi.org/10.1186/s12916-023-03159-0openalex-snapshotMale- and female-specific reproductive risk factors across the lifespan for dementia or cognitive decline: a systematic review and meta-analysis
inferred← citeshttps://doi.org/10.1186/s13195-024-01492-xopenalex-snapshotAge, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.1186/s40035-025-00514-4openalex-snapshotSIK2-mediated phosphorylation of GABARAPL2 facilitates autophagosome–lysosome fusion and rescues neurodegeneration in an Alzheimer’s disease model
inferred← citeshttps://doi.org/10.1371/journal.pone.0331204openalex-snapshotGlobal trends in Alzheimer’s disease and other dementias in adults aged 55 and above (1992–2021): An age-period-cohort analysis based on the GBD 2021
inferred← citeshttps://doi.org/10.14283/jpad.2024.21openalex-snapshotGlobal Burden of Dementia Death from 1990 to 2019, with Projections to 2050: An Analysis of 2019 Global Burden of Disease Study
inferred← citeshttps://doi.org/10.3389/fncir.2023.1099467openalex-snapshotAberrant protein S-nitrosylation contributes to hyperexcitability-induced synaptic damage in Alzheimer’s disease: Mechanistic insights and potential therapies
inferred← citeshttps://doi.org/10.6084/m9.figshare.24630273openalex-snapshotAdditional file 1 of Male- and female-specific reproductive risk factors across the lifespan for dementia or cognitive decline: a systematic review and meta-analysis
inferred← citeshttps://doi.org/10.6084/m9.figshare.26085125openalex-snapshotAdditional file 1 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26085212openalex-snapshotAdditional file 2 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737956openalex-snapshotAdditional file 3 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737959openalex-snapshotAdditional file 4 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737962openalex-snapshotAdditional file 5 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737965openalex-snapshotAdditional file 6 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737968openalex-snapshotAdditional file 7 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737971openalex-snapshotAdditional file 8 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737974openalex-snapshotAdditional file 9 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737977openalex-snapshotAdditional file 10 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737980openalex-snapshotAdditional file 11 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737983openalex-snapshotAdditional file 12 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
inferred← citeshttps://doi.org/10.6084/m9.figshare.26737986openalex-snapshotAdditional file 13 of Age, sex and Alzheimer’s disease: a longitudinal study of 3xTg-AD mice reveals sex-specific disease trajectories and inflammatory responses mirrored in postmortem brains from Alzheimer’s patients
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