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A Novel Thioredoxin-Mediated Mechanism in Autism Spectrum Disorder

https://doi.org/10.2139/ssrn.4844704
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Nuclear and cytoplasmic peroxiredoxin-1 differentially regulate NF-κB activities
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The thioredoxin antioxidant system
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Protective effects of the thioredoxin and glutaredoxin systems in dopamine-induced cell death
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A cross-talk between nitric oxide and the glutamatergic system in a Shank3 mouse model of autism
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SHANK3 mutations identified in autism lead to modification of dendritic spine morphology via an actin-dependent mechanism
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Neurodegenerative diseases and oxidative stress
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Iron, brain ageing and neurodegenerative disorders
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Deficiency of peroxiredoxin 2 exacerbates angiotensin II-induced abdominal aortic aneurysm
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Shank3 mutation in a mouse model of autism leads to changes in the S-nitroso-proteome and affects key proteins involved in vesicle release and synaptic function
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S-nitrosylation of E3 ubiquitin-protein ligase RNF213 alters non-canonical Wnt/Ca+2 signaling in the P301S mouse model of tauopathy
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Proteomics of autism and Alzheimer’s mouse models reveal common alterations in mTOR signaling pathway
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Arsenic alters nitric oxide signaling similar to autism spectrum disorder and Alzheimer’s disease-associated mutations
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Inhibition of the thioredoxin system by PX-12 (1-methylpropyl 2-imidazolyl disulfide) impedes HIV-1 infection in TZM-bl cells
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Systems biology reveals reprogramming of the S-nitroso-proteome in the cortical and striatal regions of mice during aging process
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Mutations associated with autism lead to similar synaptic and behavioral alterations in both sexes of male and female mouse brain
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Symptom dimensions of resting-state electroencephalographic functional connectivity in autism
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Involvement of glutaredoxin-1 and thioredoxin-1 in β-amyloid toxicity and Alzheimer's disease
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Impaired GABA synthesis, uptake and release are associated with depression-like behaviors induced by chronic mild stress
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Glutathione metabolism in brain
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AP-1 transcriptional activity is regulated by a direct association between thioredoxin and Ref-1
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S-Nitrosoglutathione Is Cleaved by the Thioredoxin System with Liberation of Glutathione and Redox Regulating Nitric Oxide
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Thioredoxin Facilitates the Induction of Heme Oxygenase-1 in Response to Inflammatory Mediators
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Thioredoxin Reductase Linked to Cytoskeleton by Focal Adhesion Kinase Reverses Actin S-Nitrosylation and Restores Neutrophil β2 Integrin Function
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3-Nitrotyrosine: a versatile oxidative stress biomarker for major neurodegenerative diseases
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Thioredoxin-1 and Posttranslational Modifications
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Significance of Brain Tissue Oxygenation and the Arachidonic Acid Cascade in Stroke
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Thioredoxin 1-Mediated Post-Translational Modifications: Reduction, Transnitrosylation, Denitrosylation, and Related Proteomics Methodologies
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TrxR1 as a Potent Regulator of the Nrf2-Keap1 Response System
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Redox Signaling Mediated by Thioredoxin and Glutathione Systems in the Central Nervous System
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Thioredoxin and Its Role in Toxicology
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Increased Nuclear Thioredoxin-1 Potentiates Cadmium-Induced Cytotoxicity
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Oxidative stress and neurodegeneration: where are we now?
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Why is nitric oxide important for our brain?
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Mechanistic insight into female predominance in Alzheimer’s disease based on aberrant protein S-nitrosylation of C3
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Regulated Protein Denitrosylation by Cytosolic and Mitochondrial Thioredoxins
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Cytoplasmic Thioredoxin Reductase Is Essential for Embryogenesis but Dispensable for Cardiac Development
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Autistic phenotypes and genetic testing: state-of-the-art for the clinical geneticist
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SUPEROXIDE RADICAL AND SUPEROXIDE DISMUTASES
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Potential Role of Selenoenzymes and Antioxidant Metabolism in relation to Autism Etiology and Pathology
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Thioredoxin System Regulation in the Central Nervous System: Experimental Models and Clinical Evidence
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Oxidative Stress as a Common Key Event in Developmental Neurotoxicity
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Oxidative stress and inflammation biomarkers in the blood of patients with Huntington’s disease
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Inhibition of thioredoxin activates mitophagy and overcomes adaptive bortezomib resistance in multiple myeloma
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Thioredoxin level and inflammatory markers in children with autism spectrum disorders
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N-Methyl-D-Aspartate Receptor-Dependent Denitrosylation of Neuronal Nitric Oxide Synthase Increase the Enzyme Activity
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Disturbed Flow Enhances Inflammatory Signaling and Atherogenesis by Increasing Thioredoxin-1 Level in Endothelial Cell Nuclei
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A metabolic map of the DNA damage response identifies PRDX1 in the control of nuclear ROS scavenging and aspartate availability
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Expression of the Excitatory Postsynaptic Scaffolding Protein, Shank3, in Human Brain: Effect of Age and Alzheimer’s Disease
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The Importance of Thioredoxin-1 in Health and Disease
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Systems Biology Reveals S-Nitrosylation-Dependent Regulation of Mitochondrial Functions in Mice with Shank3 Mutation Associated with Autism Spectrum Disorder
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Thioredoxin and Cancer: A Role for Thioredoxin in all States of Tumor Oxygenation
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Nrf2, the Master Regulator of Anti-Oxidative Responses
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Low Doses of Arsenic in a Mouse Model of Human Exposure and in Neuronal Culture Lead to S-Nitrosylation of Synaptic Proteins and Apoptosis via Nitric Oxide
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Oxidative Stress: A Key Modulator in Neurodegenerative Diseases
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The Role of Oxidative Stress in Autism Spectrum Disorder: A Narrative Literature Review
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Novel Object Recognition Test for the Investigation of Learning and Memory in Mice
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Oxidative stress in neurodegenerative diseases.
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Nuclear translocation of thioredoxin-1 promotes colorectal cancer development via modulation of the IL-6/STAT3 signaling axis through interaction with STAT3
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